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Analysis of single nucleotide polymorphisms in the FAS and CTLA-4 genes of peripheral T-cell lymphomas.

Journal of hematopathology

Bonzheim I, Geissinger E, Chuang WY, Roth S, Ströbel P, Marx A, Reimer P, Wilhelm M, Puppe B, Rosenwald A, Müller-Hermelink HK, Rüdiger T.
PMID: 19669200
J Hematop. 2008 Jul;1(1):11-21. doi: 10.1007/s12308-008-0003-y. Epub 2008 Apr 24.

Angioimmunoblastic T-cell lymphoma (AILT) represents a subset of T-cell lymphomas but resembles an autoimmune disease in many of its clinical aspects. Despite the phenotype of effector T-cells and high expression of FAS and CTLA-4 receptor molecules, tumor cells fail...

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Bonzheim I, Geissinger E, Chuang WY, et al. Analysis of single nucleotide polymorphisms in the FAS and CTLA-4 genes of peripheral T-cell lymphomas. J Hematop. 2008;1(1):11-21doi: 10.1007/s12308-008-0003-y.
Bonzheim, I., Geissinger, E., Chuang, W. Y., Roth, S., Ströbel, P., Marx, A., Reimer, P., Wilhelm, M., Puppe, B., Rosenwald, A., Müller-Hermelink, H. K., & Rüdiger, T. (2008). Analysis of single nucleotide polymorphisms in the FAS and CTLA-4 genes of peripheral T-cell lymphomas. Journal of hematopathology, 1(1), 11-21. https://doi.org/10.1007/s12308-008-0003-y
Bonzheim, Irina, et al. "Analysis of single nucleotide polymorphisms in the FAS and CTLA-4 genes of peripheral T-cell lymphomas." Journal of hematopathology vol. 1,1 (2008): 11-21. doi: https://doi.org/10.1007/s12308-008-0003-y
Bonzheim I, Geissinger E, Chuang WY, Roth S, Ströbel P, Marx A, Reimer P, Wilhelm M, Puppe B, Rosenwald A, Müller-Hermelink HK, Rüdiger T. Analysis of single nucleotide polymorphisms in the FAS and CTLA-4 genes of peripheral T-cell lymphomas. J Hematop. 2008 Jul;1(1):11-21. doi: 10.1007/s12308-008-0003-y. Epub 2008 Apr 24. PMID: 19669200; PMCID: PMC2712330.
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The inflammation in cutaneous lichen planus is dominated by IFN-ϒ and IL-21-A basis for therapeutic JAK1 inhibition.

Experimental dermatology

Pietschke K, Holstein J, Meier K, Schäfer I, Müller-Hermelink E, Gonzalez-Menendez I, Quintanilla-Martinez L, Ghoreschi FC, Solimani F, Ghoreschi K.
PMID: 33113249
Exp Dermatol. 2021 Feb;30(2):262-270. doi: 10.1111/exd.14226. Epub 2020 Nov 12.

Cutaneous lichen planus (CLP) and psoriasis (PSO) are both common chronic inflammatory skin diseases for which development of new treatments requires the identification of key targets. While PSO is a typical Th17/IL-17-disorder, there is some evidence that Th1/IFN-ɣ dominate...

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Pietschke K, Holstein J, Meier K, et al. The inflammation in cutaneous lichen planus is dominated by IFN-ϒ and IL-21-A basis for therapeutic JAK1 inhibition. Exp Dermatol. 2020;30(2):262-270doi: 10.1111/exd.14226.
Pietschke, K., Holstein, J., Meier, K., Schäfer, I., Müller-Hermelink, E., Gonzalez-Menendez, I., Quintanilla-Martinez, L., Ghoreschi, F. C., Solimani, F., & Ghoreschi, K. (2021). The inflammation in cutaneous lichen planus is dominated by IFN-ϒ and IL-21-A basis for therapeutic JAK1 inhibition. Experimental dermatology, 30(2), 262-270. https://doi.org/10.1111/exd.14226
Pietschke, Katharina, et al. "The inflammation in cutaneous lichen planus is dominated by IFN-ϒ and IL-21-A basis for therapeutic JAK1 inhibition." Experimental dermatology vol. 30,2 (2021): 262-270. doi: https://doi.org/10.1111/exd.14226
Pietschke K, Holstein J, Meier K, Schäfer I, Müller-Hermelink E, Gonzalez-Menendez I, Quintanilla-Martinez L, Ghoreschi FC, Solimani F, Ghoreschi K. The inflammation in cutaneous lichen planus is dominated by IFN-ϒ and IL-21-A basis for therapeutic JAK1 inhibition. Exp Dermatol. 2021 Feb;30(2):262-270. doi: 10.1111/exd.14226. Epub 2020 Nov 12. PMID: 33113249.
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[Opening address of the President].

Verhandlungen der Deutschen Gesellschaft fur Pathologie

Müller-Hermelink HK.
PMID: 9064990
Verh Dtsch Ges Pathol. 1996;80:L-LV.

No abstract available.

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Müller-Hermelink HK. Eröffnungsrede des Vorsitzenden. [Opening address of the President]. Verh Dtsch Ges Pathol. 1996;80:L-LV
Müller-Hermelink, H. K. (1996). Eröffnungsrede des Vorsitzenden. [Opening address of the President]. Verhandlungen der Deutschen Gesellschaft fur Pathologie, 80L-LV.
Müller-Hermelink, H K. "Eröffnungsrede des Vorsitzenden." [Opening address of the President]. Verhandlungen der Deutschen Gesellschaft fur Pathologie vol. 80 (1996): L-LV.
Müller-Hermelink HK. Eröffnungsrede des Vorsitzenden. [Opening address of the President]. Verh Dtsch Ges Pathol. 1996;80:L-LV. German. PMID: 9064990.
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Germinal center B cell-like (GCB) and activated B cell-like (ABC) type of diffuse large B cell lymphoma (DLBCL): analysis of molecular predictors, signatures, cell cycle state and patient survival.

Cancer informatics

Blenk S, Engelmann J, Weniger M, Schultz J, Dittrich M, Rosenwald A, Müller-Hermelink HK, Müller T, Dandekar T.
PMID: 19455257
Cancer Inform. 2007 Dec 12;3:399-420.

Aiming to find key genes and events, we analyze a large data set on diffuse large B-cell lymphoma (DLBCL) gene-expression (248 patients, 12196 spots). Applying the loess normalization method on these raw data yields improved survival predictions, in particular...

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Blenk S, Engelmann J, Weniger M, et al. Germinal center B cell-like (GCB) and activated B cell-like (ABC) type of diffuse large B cell lymphoma (DLBCL): analysis of molecular predictors, signatures, cell cycle state and patient survival. Cancer Inform. 2007;3:399-420
Blenk, S., Engelmann, J., Weniger, M., Schultz, J., Dittrich, M., Rosenwald, A., Müller-Hermelink, H. K., Müller, T., & Dandekar, T. (2007). Germinal center B cell-like (GCB) and activated B cell-like (ABC) type of diffuse large B cell lymphoma (DLBCL): analysis of molecular predictors, signatures, cell cycle state and patient survival. Cancer informatics, 3399-420.
Blenk, S, et al. "Germinal center B cell-like (GCB) and activated B cell-like (ABC) type of diffuse large B cell lymphoma (DLBCL): analysis of molecular predictors, signatures, cell cycle state and patient survival." Cancer informatics vol. 3 (2007): 399-420.
Blenk S, Engelmann J, Weniger M, Schultz J, Dittrich M, Rosenwald A, Müller-Hermelink HK, Müller T, Dandekar T. Germinal center B cell-like (GCB) and activated B cell-like (ABC) type of diffuse large B cell lymphoma (DLBCL): analysis of molecular predictors, signatures, cell cycle state and patient survival. Cancer Inform. 2007 Dec 12;3:399-420. PMID: 19455257; PMCID: PMC2675856.
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Ki-67 as a prognostic marker in mantle cell lymphoma-consensus guidelines of the pathology panel of the European MCL Network.

Journal of hematopathology

Klapper W, Hoster E, Determann O, Oschlies I, van der Laak J, Berger F, Bernd HW, Cabeçadas J, Campo E, Cogliatti S, Hansmann ML, Kluin PM, Kodet R, Krivolapov YA, Loddenkemper C, Stein H, Möller P, Barth TE, Müller-Hermelink K, Rosenwald A, Ott G, Pileri S, Ralfkiaer E, Rymkiewicz G, van Krieken JH, Wacker HH, Unterhalt M, Hiddemann W, Dreyling M.
PMID: 19669190
J Hematop. 2009 Jul;2(2):103-11. doi: 10.1007/s12308-009-0036-x. Epub 2009 Jun 16.

Mantle cell lymphoma (MCL) has a heterogeneous clinical course and is mainly an aggressive B cell non-Hodgkin lymphoma; however, there are some indolent cases The Ki-67 index, defined by the percentage of Ki-67-positive lymphoma cells on histopathological slides, has...

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Klapper W, Hoster E, Determann O, et al. Ki-67 as a prognostic marker in mantle cell lymphoma-consensus guidelines of the pathology panel of the European MCL Network. J Hematop. 2009;2(2):103-11doi: 10.1007/s12308-009-0036-x.
Klapper, W., Hoster, E., Determann, O., Oschlies, I., van der Laak, J., Berger, F., Bernd, H. W., Cabeçadas, J., Campo, E., Cogliatti, S., Hansmann, M. L., Kluin, P. M., Kodet, R., Krivolapov, Y. A., Loddenkemper, C., Stein, H., Möller, P., Barth, T. E., Müller-Hermelink, K., Rosenwald, A., Ott, G., Pileri, S., Ralfkiaer, E., Rymkiewicz, G., van Krieken, J. H., Wacker, H. H., Unterhalt, M., Hiddemann, W., Dreyling, M. (2009). Ki-67 as a prognostic marker in mantle cell lymphoma-consensus guidelines of the pathology panel of the European MCL Network. Journal of hematopathology, 2(2), 103-11. https://doi.org/10.1007/s12308-009-0036-x
Klapper, Wolfram, et al. "Ki-67 as a prognostic marker in mantle cell lymphoma-consensus guidelines of the pathology panel of the European MCL Network." Journal of hematopathology vol. 2,2 (2009): 103-11. doi: https://doi.org/10.1007/s12308-009-0036-x
Klapper W, Hoster E, Determann O, Oschlies I, van der Laak J, Berger F, Bernd HW, Cabeçadas J, Campo E, Cogliatti S, Hansmann ML, Kluin PM, Kodet R, Krivolapov YA, Loddenkemper C, Stein H, Möller P, Barth TE, Müller-Hermelink K, Rosenwald A, Ott G, Pileri S, Ralfkiaer E, Rymkiewicz G, van Krieken JH, Wacker HH, Unterhalt M, Hiddemann W, Dreyling M. Ki-67 as a prognostic marker in mantle cell lymphoma-consensus guidelines of the pathology panel of the European MCL Network. J Hematop. 2009 Jul;2(2):103-11. doi: 10.1007/s12308-009-0036-x. Epub 2009 Jun 16. PMID: 19669190; PMCID: PMC2725281.
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[Heat shock protein 90 alpha und beta are overexpressed in multiple myeloma cells and critically contribute to survival].

Verhandlungen der Deutschen Gesellschaft fur Pathologie

Andrulis M, Chatterjee M, Jain S, Stühmer T, Ungethüm U, Kuban RJ, Lorentz H, Bommert K, Topp M, Kramer D, Müller-Hermelinks HK, Einsele H, Bargou RC, Greiner A.
PMID: 18314631
Verh Dtsch Ges Pathol. 2007;91:330-7.

HSP90's are overexpressed in different cancer types and they probably are required to sustain aberrant signalling in malignant cells. Recently, pharmacological inhibition of HSP90 was found to suppress growth of myeloma cell lines and in primary myeloma cells. Therefore,...

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Andrulis M, Chatterjee M, Jain S, et al. Die Hitzeschockproteine 90 alpha und beta sind im mutiplen Myelom überexprimiert und tragen zum Tumorzellüberleben bei. [Heat shock protein 90 alpha und beta are overexpressed in multiple myeloma cells and critically contribute to survival]. Verh Dtsch Ges Pathol. 2007;91:330-7
Andrulis, M., Chatterjee, M., Jain, S., Stühmer, T., Ungethüm, U., Kuban, R. J., Lorentz, H., Bommert, K., Topp, M., Kramer, D., Müller-Hermelinks, H. K., Einsele, H., Bargou, R. C., & Greiner, A. (2007). Die Hitzeschockproteine 90 alpha und beta sind im mutiplen Myelom überexprimiert und tragen zum Tumorzellüberleben bei. [Heat shock protein 90 alpha und beta are overexpressed in multiple myeloma cells and critically contribute to survival]. Verhandlungen der Deutschen Gesellschaft fur Pathologie, 91330-7.
Andrulis, M, et al. "Die Hitzeschockproteine 90 alpha und beta sind im mutiplen Myelom überexprimiert und tragen zum Tumorzellüberleben bei." [Heat shock protein 90 alpha und beta are overexpressed in multiple myeloma cells and critically contribute to survival]. Verhandlungen der Deutschen Gesellschaft fur Pathologie vol. 91 (2007): 330-7.
Andrulis M, Chatterjee M, Jain S, Stühmer T, Ungethüm U, Kuban RJ, Lorentz H, Bommert K, Topp M, Kramer D, Müller-Hermelinks HK, Einsele H, Bargou RC, Greiner A. Die Hitzeschockproteine 90 alpha und beta sind im mutiplen Myelom überexprimiert und tragen zum Tumorzellüberleben bei. [Heat shock protein 90 alpha und beta are overexpressed in multiple myeloma cells and critically contribute to survival]. Verh Dtsch Ges Pathol. 2007;91:330-7. German. PMID: 18314631.
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Expression of CD56 isoforms in primary and relapsed adult granulosa cell tumors of the ovary.

Diagnostic pathology

Völker HU, Engert S, Cramer A, Schmidt M, Kämmerer U, Müller-Hermelink HK, Gattenlöhner S.
PMID: 18613980
Diagn Pathol. 2008 Jul 09;3:29. doi: 10.1186/1746-1596-3-29.

BACKGROUND: Adult granulosa cell tumors of the ovary (GCTs) are sex cord stromal tumors of unpredictable behaviour. Up to now, the prediction of the relapsing/malignant potential remains difficult. CD56 (NCAM) in GCTs was previously described in only two studies....

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Völker HU, Engert S, Cramer A, et al. Expression of CD56 isoforms in primary and relapsed adult granulosa cell tumors of the ovary. Diagn Pathol. 2008;3:29doi: 10.1186/1746-1596-3-29.
Völker, H. U., Engert, S., Cramer, A., Schmidt, M., Kämmerer, U., Müller-Hermelink, H. K., & Gattenlöhner, S. (2008). Expression of CD56 isoforms in primary and relapsed adult granulosa cell tumors of the ovary. Diagnostic pathology, 329. https://doi.org/10.1186/1746-1596-3-29
Völker, Hans-Ullrich, et al. "Expression of CD56 isoforms in primary and relapsed adult granulosa cell tumors of the ovary." Diagnostic pathology vol. 3 (2008): 29. doi: https://doi.org/10.1186/1746-1596-3-29
Völker HU, Engert S, Cramer A, Schmidt M, Kämmerer U, Müller-Hermelink HK, Gattenlöhner S. Expression of CD56 isoforms in primary and relapsed adult granulosa cell tumors of the ovary. Diagn Pathol. 2008 Jul 09;3:29. doi: 10.1186/1746-1596-3-29. PMID: 18613980; PMCID: PMC2474830.
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Lethal pneumatosis coli in a 12-month-old child caused by acute intestinal gas gangrene after prolonged artificial nutrition: a case report.

Journal of medical case reports

Kircher S, Wössner R, Müller-Hermelink HK, Völker HU.
PMID: 18652650
J Med Case Rep. 2008 Jul 24;2:238. doi: 10.1186/1752-1947-2-238.

INTRODUCTION: Pneumatosis coli is a rare disease with heterogeneous symptoms which can be detected in the course of various acute and chronic intestinal diseases in children, such as necrotizing enterocolitis, intestinal obstruction and intestinal bacteriological infections.CASE PRESENTATION: We report...

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Kircher S, Wössner R, Müller-Hermelink HK, et al. Lethal pneumatosis coli in a 12-month-old child caused by acute intestinal gas gangrene after prolonged artificial nutrition: a case report. J Med Case Rep. 2008;2:238doi: 10.1186/1752-1947-2-238.
Kircher, S., Wössner, R., Müller-Hermelink, H. K., & Völker, H. U. (2008). Lethal pneumatosis coli in a 12-month-old child caused by acute intestinal gas gangrene after prolonged artificial nutrition: a case report. Journal of medical case reports, 2238. https://doi.org/10.1186/1752-1947-2-238
Kircher, Stefan, et al. "Lethal pneumatosis coli in a 12-month-old child caused by acute intestinal gas gangrene after prolonged artificial nutrition: a case report." Journal of medical case reports vol. 2 (2008): 238. doi: https://doi.org/10.1186/1752-1947-2-238
Kircher S, Wössner R, Müller-Hermelink HK, Völker HU. Lethal pneumatosis coli in a 12-month-old child caused by acute intestinal gas gangrene after prolonged artificial nutrition: a case report. J Med Case Rep. 2008 Jul 24;2:238. doi: 10.1186/1752-1947-2-238. PMID: 18652650; PMCID: PMC2503997.
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Differential diagnosis of laryngeal spindle cell carcinoma and inflammatory myofibroblastic tumor--report of two cases with similar morphology.

Diagnostic pathology

Völker HU, Scheich M, Höller S, Ströbel P, Hagen R, Müller-Hermelink HK, Eck M.
PMID: 17212821
Diagn Pathol. 2007 Jan 09;2:1. doi: 10.1186/1746-1596-2-1.

BACKGROUND: Spindle cell tumors of the larynx are rare. In some cases, the dignity is difficult to determine. We report two cases of laryngeal spindle cell tumors.CASE PRESENTATION: Case 1 is a spindle cell carcinoma (SPC) in a 55...

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Völker HU, Scheich M, Höller S, et al. Differential diagnosis of laryngeal spindle cell carcinoma and inflammatory myofibroblastic tumor--report of two cases with similar morphology. Diagn Pathol. 2007;2:1doi: 10.1186/1746-1596-2-1.
Völker, H. U., Scheich, M., Höller, S., Ströbel, P., Hagen, R., Müller-Hermelink, H. K., & Eck, M. (2007). Differential diagnosis of laryngeal spindle cell carcinoma and inflammatory myofibroblastic tumor--report of two cases with similar morphology. Diagnostic pathology, 21. https://doi.org/10.1186/1746-1596-2-1
Völker, Hans-Ullrich, et al. "Differential diagnosis of laryngeal spindle cell carcinoma and inflammatory myofibroblastic tumor--report of two cases with similar morphology." Diagnostic pathology vol. 2 (2007): 1. doi: https://doi.org/10.1186/1746-1596-2-1
Völker HU, Scheich M, Höller S, Ströbel P, Hagen R, Müller-Hermelink HK, Eck M. Differential diagnosis of laryngeal spindle cell carcinoma and inflammatory myofibroblastic tumor--report of two cases with similar morphology. Diagn Pathol. 2007 Jan 09;2:1. doi: 10.1186/1746-1596-2-1. PMID: 17212821; PMCID: PMC1779261.
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A cytomorphological and immunohistochemical profile of aggressive B-cell lymphoma: high clinical impact of a cumulative immunohistochemical outcome predictor score.

Journal of hematopathology

Höller S, Horn H, Lohr A, Mäder U, Katzenberger T, Kalla J, Bernd HW, Went P, Ott MM, Müller-Hermelink HK, Rosenwald A, Ott G.
PMID: 20309427
J Hematop. 2009 Sep 02;2(4):187-94. doi: 10.1007/s12308-009-0044-x.

We analyzed morphological and immunohistochemical features in 174 aggressive B-cell lymphomas of nodal and extranodal origin. Morphological features included presence or absence of a follicular component and cytologic criteria according to the Kiel classification, whereas immunohistochemical studies included expression...

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Höller S, Horn H, Lohr A, et al. A cytomorphological and immunohistochemical profile of aggressive B-cell lymphoma: high clinical impact of a cumulative immunohistochemical outcome predictor score. J Hematop. 2009;2(4):187-94doi: 10.1007/s12308-009-0044-x.
Höller, S., Horn, H., Lohr, A., Mäder, U., Katzenberger, T., Kalla, J., Bernd, H. W., Went, P., Ott, M. M., Müller-Hermelink, H. K., Rosenwald, A., & Ott, G. (2009). A cytomorphological and immunohistochemical profile of aggressive B-cell lymphoma: high clinical impact of a cumulative immunohistochemical outcome predictor score. Journal of hematopathology, 2(4), 187-94. https://doi.org/10.1007/s12308-009-0044-x
Höller, Sylvia, et al. "A cytomorphological and immunohistochemical profile of aggressive B-cell lymphoma: high clinical impact of a cumulative immunohistochemical outcome predictor score." Journal of hematopathology vol. 2,4 (2009): 187-94. doi: https://doi.org/10.1007/s12308-009-0044-x
Höller S, Horn H, Lohr A, Mäder U, Katzenberger T, Kalla J, Bernd HW, Went P, Ott MM, Müller-Hermelink HK, Rosenwald A, Ott G. A cytomorphological and immunohistochemical profile of aggressive B-cell lymphoma: high clinical impact of a cumulative immunohistochemical outcome predictor score. J Hematop. 2009 Sep 02;2(4):187-94. doi: 10.1007/s12308-009-0044-x. PMID: 20309427; PMCID: PMC2798934.
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Anti-Apoptotic Signature in Thymic Squamous Cell Carcinomas - Functional Relevance of Anti-Apoptotic BIRC3 Expression in the Thymic Carcinoma Cell Line 1889c.

Frontiers in oncology

Huang B, Belharazem D, Li L, Kneitz S, Schnabel PA, Rieker RJ, Körner D, Nix W, Schalke B, Müller-Hermelink HK, Ott G, Rosenwald A, Ströbel P, Marx A.
PMID: 24427739
Front Oncol. 2013 Dec 31;3:316. doi: 10.3389/fonc.2013.00316. eCollection 2013.

The molecular pathogenesis of thymomas and thymic carcinomas (TCs) is poorly understood and results of adjuvant therapy are unsatisfactory in case of metastatic disease and tumor recurrence. For these clinical settings, novel therapeutic strategies are urgently needed. Recently, limited...

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Huang B, Belharazem D, Li L, et al. Anti-Apoptotic Signature in Thymic Squamous Cell Carcinomas - Functional Relevance of Anti-Apoptotic BIRC3 Expression in the Thymic Carcinoma Cell Line 1889c. Front Oncol. 2013;3:316doi: 10.3389/fonc.2013.00316.
Huang, B., Belharazem, D., Li, L., Kneitz, S., Schnabel, P. A., Rieker, R. J., Körner, D., Nix, W., Schalke, B., Müller-Hermelink, H. K., Ott, G., Rosenwald, A., Ströbel, P., & Marx, A. (2013). Anti-Apoptotic Signature in Thymic Squamous Cell Carcinomas - Functional Relevance of Anti-Apoptotic BIRC3 Expression in the Thymic Carcinoma Cell Line 1889c. Frontiers in oncology, 3316. https://doi.org/10.3389/fonc.2013.00316
Huang, Bei, et al. "Anti-Apoptotic Signature in Thymic Squamous Cell Carcinomas - Functional Relevance of Anti-Apoptotic BIRC3 Expression in the Thymic Carcinoma Cell Line 1889c." Frontiers in oncology vol. 3 (2013): 316. doi: https://doi.org/10.3389/fonc.2013.00316
Huang B, Belharazem D, Li L, Kneitz S, Schnabel PA, Rieker RJ, Körner D, Nix W, Schalke B, Müller-Hermelink HK, Ott G, Rosenwald A, Ströbel P, Marx A. Anti-Apoptotic Signature in Thymic Squamous Cell Carcinomas - Functional Relevance of Anti-Apoptotic BIRC3 Expression in the Thymic Carcinoma Cell Line 1889c. Front Oncol. 2013 Dec 31;3:316. doi: 10.3389/fonc.2013.00316. eCollection 2013. PMID: 24427739; PMCID: PMC3876280.
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Fatal Human Meningoencephalitis due to Halicephalobus Nematodes, Germany.

Open forum infectious diseases

Monoranu CM, Müllges W, Keppler M, Brehm K, Ondrejka SL, Muntau B, Tannich E, Müller-Hermelink HK, Tappe D.
PMID: 26125032
Open Forum Infect Dis. 2015 May 22;2(2):ofv061. doi: 10.1093/ofid/ofv061. eCollection 2015 Apr.

Infections with Halicephalobus nematodes, causative agents of severe meningoencephalitis in horses, have rarely been reported in humans. In this study, the clinical, serological, cytokine, and histopathological findings of a rapidly progressive and eventually fatal meningoencephalitis in a previously healthy...

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Monoranu CM, Müllges W, Keppler M, et al. Fatal Human Meningoencephalitis due to Halicephalobus Nematodes, Germany. Open Forum Infect Dis. 2015;2(2):ofv061doi: 10.1093/ofid/ofv061.
Monoranu, C. M., Müllges, W., Keppler, M., Brehm, K., Ondrejka, S. L., Muntau, B., Tannich, E., Müller-Hermelink, H. K., & Tappe, D. (2015). Fatal Human Meningoencephalitis due to Halicephalobus Nematodes, Germany. Open forum infectious diseases, 2(2), ofv061. https://doi.org/10.1093/ofid/ofv061
Monoranu, Camelia-Maria, et al. "Fatal Human Meningoencephalitis due to Halicephalobus Nematodes, Germany." Open forum infectious diseases vol. 2,2 (2015): ofv061. doi: https://doi.org/10.1093/ofid/ofv061
Monoranu CM, Müllges W, Keppler M, Brehm K, Ondrejka SL, Muntau B, Tannich E, Müller-Hermelink HK, Tappe D. Fatal Human Meningoencephalitis due to Halicephalobus Nematodes, Germany. Open Forum Infect Dis. 2015 May 22;2(2):ofv061. doi: 10.1093/ofid/ofv061. eCollection 2015 Apr. PMID: 26125032; PMCID: PMC4462890.
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