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Chemoprevention of tea on colorectal cancer induced by dimethylhydrazine in Wistar rats.

World journal of gastroenterology

Jia XD, Han C.
PMID: 11819677
World J Gastroenterol. 2000 Oct;6(5):699-703. doi: 10.3748/wjg.v6.i5.699.

AIM:To investigate the chemopreventive effects of green tea and tea pigment on 1,2-dimethylhydrazine (DMH)-induced rat colorectal carcinogenesis.METHODS:Male weaning Wistar rats were randomly allocated into four groups.Rats in the positive control group were given a(c)s.c. injection of DMH, once a...

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Jia XD, Han C. Chemoprevention of tea on colorectal cancer induced by dimethylhydrazine in Wistar rats. World J Gastroenterol. 2000;6(5):699-703doi: 10.3748/wjg.v6.i5.699.
Jia, X. D., & Han, C. (2000). Chemoprevention of tea on colorectal cancer induced by dimethylhydrazine in Wistar rats. World journal of gastroenterology, 6(5), 699-703. https://doi.org/10.3748/wjg.v6.i5.699
Jia, Xu-Dong, and Han, Chi. "Chemoprevention of tea on colorectal cancer induced by dimethylhydrazine in Wistar rats." World journal of gastroenterology vol. 6,5 (2000): 699-703. doi: https://doi.org/10.3748/wjg.v6.i5.699
Jia XD, Han C. Chemoprevention of tea on colorectal cancer induced by dimethylhydrazine in Wistar rats. World J Gastroenterol. 2000 Oct;6(5):699-703. doi: 10.3748/wjg.v6.i5.699. PMID: 11819677; PMCID: PMC4688846.
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Chemistry of stable iminopropadienones, RN=C=C=C=O.

The Journal of organic chemistry

Bibas H, Moloney DW, Neumann R, Shtaiwi M, Bernhardt PV, Wentrup C.
PMID: 11950308
J Org Chem. 2002 Apr 19;67(8):2619-31. doi: 10.1021/jo0110552.

The synthesis, spectroscopic properties, and chemical reactions of the stable (neopentylimino)-, (mesitylimino)-, and (o-tert-butylphenylimino)propadienones (6) are reported. Nucleophilic addition of amines affords the malonic amidoamidines 7 and 8. 3,5-Dimethylpyrazole reacts analogously to form 9b. Addition of 1,2-dimethylhydrazine produces pyrazolinones...

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Bibas H, Moloney DW, Neumann R, et al. Chemistry of stable iminopropadienones, RN=C=C=C=O. J Org Chem. 2002;67(8):2619-31doi: 10.1021/jo0110552.
Bibas, H., Moloney, D. W., Neumann, R., Shtaiwi, M., Bernhardt, P. V., & Wentrup, C. (2002). Chemistry of stable iminopropadienones, RN=C=C=C=O. The Journal of organic chemistry, 67(8), 2619-31. https://doi.org/10.1021/jo0110552
Bibas, Hervé, et al. "Chemistry of stable iminopropadienones, RN=C=C=C=O." The Journal of organic chemistry vol. 67,8 (2002): 2619-31. doi: https://doi.org/10.1021/jo0110552
Bibas H, Moloney DW, Neumann R, Shtaiwi M, Bernhardt PV, Wentrup C. Chemistry of stable iminopropadienones, RN=C=C=C=O. J Org Chem. 2002 Apr 19;67(8):2619-31. doi: 10.1021/jo0110552. PMID: 11950308.
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IGF-II transgenic mice display increased aberrant colon crypt multiplicity and tumor volume after 1,2-dimethylhydrazine treatment.

Journal of carcinogenesis

Diehl D, Oesterle D, Elmlinger MW, Hoeflich A, Wolf E, Lahm H.
PMID: 17118177
J Carcinog. 2006 Nov 21;5:24. doi: 10.1186/1477-3163-5-24.

In colorectal cancer insulin-like growth factor II (IGF-II) is frequently overexpressed. To evaluate, whether IGF-II affects different stages of tumorigenesis, we induced neoplastic alterations in the colon of wild-type and IGF-II transgenic mice using 1,2-dimethylhydrazine (DMH). Aberrant crypt foci...

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Diehl D, Oesterle D, Elmlinger MW, et al. IGF-II transgenic mice display increased aberrant colon crypt multiplicity and tumor volume after 1,2-dimethylhydrazine treatment. J Carcinog. 2006;5:24doi: 10.1186/1477-3163-5-24.
Diehl, D., Oesterle, D., Elmlinger, M. W., Hoeflich, A., Wolf, E., & Lahm, H. (2006). IGF-II transgenic mice display increased aberrant colon crypt multiplicity and tumor volume after 1,2-dimethylhydrazine treatment. Journal of carcinogenesis, 524. https://doi.org/10.1186/1477-3163-5-24
Diehl, Daniela, et al. "IGF-II transgenic mice display increased aberrant colon crypt multiplicity and tumor volume after 1,2-dimethylhydrazine treatment." Journal of carcinogenesis vol. 5 (2006): 24. doi: https://doi.org/10.1186/1477-3163-5-24
Diehl D, Oesterle D, Elmlinger MW, Hoeflich A, Wolf E, Lahm H. IGF-II transgenic mice display increased aberrant colon crypt multiplicity and tumor volume after 1,2-dimethylhydrazine treatment. J Carcinog. 2006 Nov 21;5:24. doi: 10.1186/1477-3163-5-24. PMID: 17118177; PMCID: PMC1660565.
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Effects of a probiotic soy product and physical exercise on formation of pre-neoplastic lesions in rat colons in a short-term model of carcinogenic.

Journal of the International Society of Sports Nutrition

Silva MF, Sivieri K, Rossi EA.
PMID: 19660118
J Int Soc Sports Nutr. 2009 Aug 06;6:17. doi: 10.1186/1550-2783-6-17.

PURPOSE: In this study the influence of moderate or intense physical exercise, alone or in combination with the consumption of a soya product fermented with Enterococcus faecium, on the development of colon cancer induced chemically in rats with 1,2-dimethylhydrazine...

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Silva MF, Sivieri K, Rossi EA. Effects of a probiotic soy product and physical exercise on formation of pre-neoplastic lesions in rat colons in a short-term model of carcinogenic. J Int Soc Sports Nutr. 2009;6:17doi: 10.1186/1550-2783-6-17.
Silva, M. F., Sivieri, K., & Rossi, E. A. (2009). Effects of a probiotic soy product and physical exercise on formation of pre-neoplastic lesions in rat colons in a short-term model of carcinogenic. Journal of the International Society of Sports Nutrition, 617. https://doi.org/10.1186/1550-2783-6-17
Silva, Maicon F, et al. "Effects of a probiotic soy product and physical exercise on formation of pre-neoplastic lesions in rat colons in a short-term model of carcinogenic." Journal of the International Society of Sports Nutrition vol. 6 (2009): 17. doi: https://doi.org/10.1186/1550-2783-6-17
Silva MF, Sivieri K, Rossi EA. Effects of a probiotic soy product and physical exercise on formation of pre-neoplastic lesions in rat colons in a short-term model of carcinogenic. J Int Soc Sports Nutr. 2009 Aug 06;6:17. doi: 10.1186/1550-2783-6-17. PMID: 19660118; PMCID: PMC2731074.
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Chemopreventive effects of mate against mouse mammary and colon carcinogenesis.

Human & experimental toxicology

Zapaterini JR, Bidinotto LT, Rodriguues MA, Barbisan LF.
PMID: 20071474
Hum Exp Toxicol. 2010 Jan 13; doi: 10.1177/0960327109359636. Epub 2010 Jan 13.

The present study evaluated the chemopreventive potential of mate tea-like intake on mammary and colon carcinogenesis initiated by 7,12-dimethylbenz(a)antracene (DMBA) and 1,2-dimethylhydrazine (DMH) in female Swiss mice. After the initiation period, the animals received basal diet and organic mate...

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Zapaterini JR, Bidinotto LT, Rodriguues MA, et al. Chemopreventive effects of mate against mouse mammary and colon carcinogenesis. Hum Exp Toxicol. 2010;doi: 10.1177/0960327109359636.
Zapaterini, J. R., Bidinotto, L. T., Rodriguues, M. A., & Barbisan, L. F. (2010). Chemopreventive effects of mate against mouse mammary and colon carcinogenesis. Human & experimental toxicology, . https://doi.org/10.1177/0960327109359636
Zapaterini, Joyce R, et al. "Chemopreventive effects of mate against mouse mammary and colon carcinogenesis." Human & experimental toxicology vol. (2010). doi: https://doi.org/10.1177/0960327109359636
Zapaterini JR, Bidinotto LT, Rodriguues MA, Barbisan LF. Chemopreventive effects of mate against mouse mammary and colon carcinogenesis. Hum Exp Toxicol. 2010 Jan 13; doi: 10.1177/0960327109359636. Epub 2010 Jan 13. PMID: 20071474.
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Chemopreventive effects of nonsteroidal anti-inflammatory drugs on 1,2-dimethylhydrazine-induced colon carcinogenesis in rats.

Toxicology mechanisms and methods

Kanwar SS, Vaiphei K, Nehru B, Sanyal SN.
PMID: 20020969
Toxicol Mech Methods. 2007;17(4):197-204. doi: 10.1080/15376510600920138.

ABSTRACT The anticancer efficacy of two different classes of NSAIDs, the nonspecific cyclooxygenase (COX) inhibitor aspirin and the specific COX-2 inhibitor celecoxib, was examined at their therapeutic anti-inflammatory doses during 1,2-dimethylhydrazine (DMH)-induced colon carcinogenesis in a rat model. Eight...

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Kanwar SS, Vaiphei K, Nehru B, et al. Chemopreventive effects of nonsteroidal anti-inflammatory drugs on 1,2-dimethylhydrazine-induced colon carcinogenesis in rats. Toxicol Mech Methods. 2007;17(4):197-204doi: 10.1080/15376510600920138.
Kanwar, S. S., Vaiphei, K., Nehru, B., & Sanyal, S. N. (2007). Chemopreventive effects of nonsteroidal anti-inflammatory drugs on 1,2-dimethylhydrazine-induced colon carcinogenesis in rats. Toxicology mechanisms and methods, 17(4), 197-204. https://doi.org/10.1080/15376510600920138
Kanwar, S S, et al. "Chemopreventive effects of nonsteroidal anti-inflammatory drugs on 1,2-dimethylhydrazine-induced colon carcinogenesis in rats." Toxicology mechanisms and methods vol. 17,4 (2007): 197-204. doi: https://doi.org/10.1080/15376510600920138
Kanwar SS, Vaiphei K, Nehru B, Sanyal SN. Chemopreventive effects of nonsteroidal anti-inflammatory drugs on 1,2-dimethylhydrazine-induced colon carcinogenesis in rats. Toxicol Mech Methods. 2007;17(4):197-204. doi: 10.1080/15376510600920138. PMID: 20020969.
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High tumor-preventive effects of polyclonal IgG generated against p53 tumor-associated protein obtained from benign-tumor bearing rats.

Oncology reports

Zusman I, Zusman R, Gurevich P, Korol D, Tendler Y.
PMID: 21594494
Oncol Rep. 1996 Sep;3(5):975-9. doi: 10.3892/or.3.5.975.

We have shown the different effects of rabbit IgG generated against various types of p53 tumor-associated protein on chemically induced colon cancer in rats. p53 protein was isolated in the form of cytoplasmic, soluble, protein from sera obtained from:...

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Zusman I, Zusman R, Gurevich P, et al. High tumor-preventive effects of polyclonal IgG generated against p53 tumor-associated protein obtained from benign-tumor bearing rats. Oncol Rep. 1996;3(5):975-9doi: 10.3892/or.3.5.975.
Zusman, I., Zusman, R., Gurevich, P., Korol, D., & Tendler, Y. (1996). High tumor-preventive effects of polyclonal IgG generated against p53 tumor-associated protein obtained from benign-tumor bearing rats. Oncology reports, 3(5), 975-9. https://doi.org/10.3892/or.3.5.975
Zusman, I, et al. "High tumor-preventive effects of polyclonal IgG generated against p53 tumor-associated protein obtained from benign-tumor bearing rats." Oncology reports vol. 3,5 (1996): 975-9. doi: https://doi.org/10.3892/or.3.5.975
Zusman I, Zusman R, Gurevich P, Korol D, Tendler Y. High tumor-preventive effects of polyclonal IgG generated against p53 tumor-associated protein obtained from benign-tumor bearing rats. Oncol Rep. 1996 Sep;3(5):975-9. doi: 10.3892/or.3.5.975. PMID: 21594494.
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Abnormal Savda Munziq, an Herbal Preparation of Traditional Uighur Medicine, May Prevent 1,2-dimethylhydrazine-Induced Rat Colon Carcinogenesis.

Evidence-based complementary and alternative medicine : eCAM

Yusup A, Upur H, Umar A, Berke B, Yimit D, Lapham JC, Moore N, Cassand P.
PMID: 19561161
Evid Based Complement Alternat Med. 2011;2011:152015. doi: 10.1093/ecam/nep059. Epub 2011 Jun 16.

The study tried to assess the chemoprotective effect of abnormal Savda Munziq (ASMq) on 1,2-dimethylhydrazine (DMH)-induced rat colon carcinogenesis. Male F344 rats were randomized into eight groups: Group 1 was served as control, no DMH injection was given and...

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Yusup A, Upur H, Umar A, et al. Abnormal Savda Munziq, an Herbal Preparation of Traditional Uighur Medicine, May Prevent 1,2-dimethylhydrazine-Induced Rat Colon Carcinogenesis. Evid Based Complement Alternat Med. 2011;2011:152015doi: 10.1093/ecam/nep059.
Yusup, A., Upur, H., Umar, A., Berke, B., Yimit, D., Lapham, J. C., Moore, N., & Cassand, P. (2011). Abnormal Savda Munziq, an Herbal Preparation of Traditional Uighur Medicine, May Prevent 1,2-dimethylhydrazine-Induced Rat Colon Carcinogenesis. Evidence-based complementary and alternative medicine : eCAM, 2011152015. https://doi.org/10.1093/ecam/nep059
Yusup, Abdiryim, et al. "Abnormal Savda Munziq, an Herbal Preparation of Traditional Uighur Medicine, May Prevent 1,2-dimethylhydrazine-Induced Rat Colon Carcinogenesis." Evidence-based complementary and alternative medicine : eCAM vol. 2011 (2011): 152015. doi: https://doi.org/10.1093/ecam/nep059
Yusup A, Upur H, Umar A, Berke B, Yimit D, Lapham JC, Moore N, Cassand P. Abnormal Savda Munziq, an Herbal Preparation of Traditional Uighur Medicine, May Prevent 1,2-dimethylhydrazine-Induced Rat Colon Carcinogenesis. Evid Based Complement Alternat Med. 2011;2011:152015. doi: 10.1093/ecam/nep059. Epub 2011 Jun 16. PMID: 19561161; PMCID: PMC3136791.
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Soy Saponins Meditate the Progression of Colon Cancer in Rats by Inhibiting the Activity of β -Glucuronidase and the Number of Aberrant Crypt Foci but Not Cyclooxygenase-2 Activity.

ISRN oncology

Guo YW, Chen YH, Chiu WC, Liao H, Lin SH.
PMID: 24224098
ISRN Oncol. 2013 Oct 02;2013:645817. doi: 10.1155/2013/645817. eCollection 2013.

Objective. The effect of extracted crude soybean saponins on preneoplastic lesions, aberrant crypt foci (ACF), and the related mechanism were investigated. Research Methods and Procedures. Rats were assigned into five groups according to different doses of extracted crude soybean...

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Guo YW, Chen YH, Chiu WC, et al. Soy Saponins Meditate the Progression of Colon Cancer in Rats by Inhibiting the Activity of β -Glucuronidase and the Number of Aberrant Crypt Foci but Not Cyclooxygenase-2 Activity. ISRN Oncol. 2013;2013:645817doi: 10.1155/2013/645817.
Guo, Y. W., Chen, Y. H., Chiu, W. C., Liao, H., & Lin, S. H. (2013). Soy Saponins Meditate the Progression of Colon Cancer in Rats by Inhibiting the Activity of β -Glucuronidase and the Number of Aberrant Crypt Foci but Not Cyclooxygenase-2 Activity. ISRN oncology, 2013645817. https://doi.org/10.1155/2013/645817
Guo, Yu-Wei, et al. "Soy Saponins Meditate the Progression of Colon Cancer in Rats by Inhibiting the Activity of β -Glucuronidase and the Number of Aberrant Crypt Foci but Not Cyclooxygenase-2 Activity." ISRN oncology vol. 2013 (2013): 645817. doi: https://doi.org/10.1155/2013/645817
Guo YW, Chen YH, Chiu WC, Liao H, Lin SH. Soy Saponins Meditate the Progression of Colon Cancer in Rats by Inhibiting the Activity of β -Glucuronidase and the Number of Aberrant Crypt Foci but Not Cyclooxygenase-2 Activity. ISRN Oncol. 2013 Oct 02;2013:645817. doi: 10.1155/2013/645817. eCollection 2013. PMID: 24224098; PMCID: PMC3808719.
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Predicting the progress of colon cancer by DNA methylation markers of the p16 gene in feces - Evidence from an animal model.

Genetics and molecular biology

Wu WC, Hsu CH, Kuan JC, Hsieh JF, Sun CA, Yang T, Wu CC, Chou YC.
PMID: 24130437
Genet Mol Biol. 2013 Sep;36(3):323-8. doi: 10.1590/S1415-47572013000300004. Epub 2013 Aug 30.

A new noninvasive screening tool for colorectal neoplasia detects epigenetic alterations exhibited by gastrointestinal tumor cells shed into stool. There is insufficient existing data to determine temporal associations between colorectal cancer (CRC) progression and aberrant DNA methylation. To evaluate...

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Wu WC, Hsu CH, Kuan JC, et al. Predicting the progress of colon cancer by DNA methylation markers of the p16 gene in feces - Evidence from an animal model. Genet Mol Biol. 2013;36(3):323-8doi: 10.1590/S1415-47572013000300004.
Wu, W. C., Hsu, C. H., Kuan, J. C., Hsieh, J. F., Sun, C. A., Yang, T., Wu, C. C., & Chou, Y. C. (2013). Predicting the progress of colon cancer by DNA methylation markers of the p16 gene in feces - Evidence from an animal model. Genetics and molecular biology, 36(3), 323-8. https://doi.org/10.1590/S1415-47572013000300004
Wu, Wen-Chih, et al. "Predicting the progress of colon cancer by DNA methylation markers of the p16 gene in feces - Evidence from an animal model." Genetics and molecular biology vol. 36,3 (2013): 323-8. doi: https://doi.org/10.1590/S1415-47572013000300004
Wu WC, Hsu CH, Kuan JC, Hsieh JF, Sun CA, Yang T, Wu CC, Chou YC. Predicting the progress of colon cancer by DNA methylation markers of the p16 gene in feces - Evidence from an animal model. Genet Mol Biol. 2013 Sep;36(3):323-8. doi: 10.1590/S1415-47572013000300004. Epub 2013 Aug 30. PMID: 24130437; PMCID: PMC3795160.
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Estradiol and progesterone regulate proliferation and apoptosis in colon cancer.

Endocrine connections

Sasso CV, Santiano FE, Campo Verde Arboccó F, Zyla LE, Semino SN, Guerrero-Gimenez ME, Pistone Creydt V, López Fontana CM, Carón RW.
PMID: 30738018
Endocr Connect. 2019 Mar 01;8(3):217-229. doi: 10.1530/EC-18-0374.

Epidemiological studies describe estrogens as protectors in the development of colon cancer in postmenopausal women treated with hormone replacement therapy. However, the role of progesterone in colon cancer has been minimally studied and the results are controversial. For the...

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Sasso CV, Santiano FE, Campo Verde Arboccó F, et al. Estradiol and progesterone regulate proliferation and apoptosis in colon cancer. Endocr Connect. 2019;8(3):217-229doi: 10.1530/EC-18-0374.
Sasso, C. V., Santiano, F. E., Campo Verde Arboccó, F., Zyla, L. E., Semino, S. N., Guerrero-Gimenez, M. E., Pistone Creydt, V., López Fontana, C. M., & Carón, R. W. (2019). Estradiol and progesterone regulate proliferation and apoptosis in colon cancer. Endocrine connections, 8(3), 217-229. https://doi.org/10.1530/EC-18-0374
Sasso, Corina Verónica, et al. "Estradiol and progesterone regulate proliferation and apoptosis in colon cancer." Endocrine connections vol. 8,3 (2019): 217-229. doi: https://doi.org/10.1530/EC-18-0374
Sasso CV, Santiano FE, Campo Verde Arboccó F, Zyla LE, Semino SN, Guerrero-Gimenez ME, Pistone Creydt V, López Fontana CM, Carón RW. Estradiol and progesterone regulate proliferation and apoptosis in colon cancer. Endocr Connect. 2019 Mar 01;8(3):217-229. doi: 10.1530/EC-18-0374. PMID: 30738018; PMCID: PMC6391933.
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Arsenic trioxide exposure accelerates colon preneoplasic aberrant crypt foci induction regionally through mitochondrial dysfunction.

Toxicology research

Moulahoum H, Boumaza BMA, Ferrat M, Djerdjouri B.
PMID: 30090573
Toxicol Res (Camb). 2017 Sep 27;7(2):182-190. doi: 10.1039/c7tx00213k. eCollection 2018 Mar 01.

Arsenic poisoning is a worldwide problem. Thus, we studied the effects of arsenic trioxide (ATO) administration on a 1,2-dimethylhydrazine (DMH)-induced preneoplasic colon carcinogenesis model. Mice were separated into four study groups; the control group received only vehicles. The ATO...

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Moulahoum H, Boumaza BMA, Ferrat M, et al. Arsenic trioxide exposure accelerates colon preneoplasic aberrant crypt foci induction regionally through mitochondrial dysfunction. Toxicol Res (Camb). 2017;7(2):182-190doi: 10.1039/c7tx00213k.
Moulahoum, H., Boumaza, B. M. A., Ferrat, M., & Djerdjouri, B. (2018). Arsenic trioxide exposure accelerates colon preneoplasic aberrant crypt foci induction regionally through mitochondrial dysfunction. Toxicology research, 7(2), 182-190. https://doi.org/10.1039/c7tx00213k
Moulahoum, Hichem, et al. "Arsenic trioxide exposure accelerates colon preneoplasic aberrant crypt foci induction regionally through mitochondrial dysfunction." Toxicology research vol. 7,2 (2018): 182-190. doi: https://doi.org/10.1039/c7tx00213k
Moulahoum H, Boumaza BMA, Ferrat M, Djerdjouri B. Arsenic trioxide exposure accelerates colon preneoplasic aberrant crypt foci induction regionally through mitochondrial dysfunction. Toxicol Res (Camb). 2017 Sep 27;7(2):182-190. doi: 10.1039/c7tx00213k. eCollection 2018 Mar 01. PMID: 30090573; PMCID: PMC6062324.
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Showing 1 to 12 of 82 entries