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Coactivation of Local Circuit NMDA Receptor Mediated epsps Induces Lasting Enhancement of Minimal Schaffer Collateral epsps in Slices of Rat Hippocampus.

The European journal of neuroscience

Radpour S, Thomson AM.
PMID: 12106491
Eur J Neurosci. 1991;3(6):602-13. doi: 10.1111/j.1460-9568.1991.tb00846.x.

Lasting enhancement of minimal Schaffer collateral epsps in CA1 pyramidal neurons was induced when these epsps were coactivated with putative local circuit N-methyl-d-aspartate (NMDA) receptor-mediated epsps. In transverse slices of hippocampus, minimal epsps were evoked by stimulating the Schaffer...

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Radpour S, Thomson AM. Coactivation of Local Circuit NMDA Receptor Mediated epsps Induces Lasting Enhancement of Minimal Schaffer Collateral epsps in Slices of Rat Hippocampus. Eur J Neurosci. 1991;3(6):602-13doi: 10.1111/j.1460-9568.1991.tb00846.x.
Radpour, S., & Thomson, A. M. (1991). Coactivation of Local Circuit NMDA Receptor Mediated epsps Induces Lasting Enhancement of Minimal Schaffer Collateral epsps in Slices of Rat Hippocampus. The European journal of neuroscience, 3(6), 602-13. https://doi.org/10.1111/j.1460-9568.1991.tb00846.x
Radpour, S, and Thomson, A M. "Coactivation of Local Circuit NMDA Receptor Mediated epsps Induces Lasting Enhancement of Minimal Schaffer Collateral epsps in Slices of Rat Hippocampus." The European journal of neuroscience vol. 3,6 (1991): 602-13. doi: https://doi.org/10.1111/j.1460-9568.1991.tb00846.x
Radpour S, Thomson AM. Coactivation of Local Circuit NMDA Receptor Mediated epsps Induces Lasting Enhancement of Minimal Schaffer Collateral epsps in Slices of Rat Hippocampus. Eur J Neurosci. 1991;3(6):602-13. doi: 10.1111/j.1460-9568.1991.tb00846.x. PMID: 12106491.
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The Involvement of N-Methyl-D-Aspartate Receptors in Induction and Maintenance of Long-Term Potentiation in Rat Visual Cortex.

The European journal of neuroscience

Artola A, Singer W.
PMID: 12106052
Eur J Neurosci. 1990;2(3):254-269. doi: 10.1111/j.1460-9568.1990.tb00417.x.

Pyramidal neurons from layers II and III of rat visual cortex slices were studied with intracellular recordings. The involvement of N-methyl-D-aspartate (NMDA) receptors was investigated: (1) in the synaptic response to white matter stimulation; (2) in the induction of...

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Artola A, Singer W. The Involvement of N-Methyl-D-Aspartate Receptors in Induction and Maintenance of Long-Term Potentiation in Rat Visual Cortex. Eur J Neurosci. 1990;2(3):254-269doi: 10.1111/j.1460-9568.1990.tb00417.x.
Artola, A., & Singer, W. (1990). The Involvement of N-Methyl-D-Aspartate Receptors in Induction and Maintenance of Long-Term Potentiation in Rat Visual Cortex. The European journal of neuroscience, 2(3), 254-269. https://doi.org/10.1111/j.1460-9568.1990.tb00417.x
Artola, A., and Singer, W.. "The Involvement of N-Methyl-D-Aspartate Receptors in Induction and Maintenance of Long-Term Potentiation in Rat Visual Cortex." The European journal of neuroscience vol. 2,3 (1990): 254-269. doi: https://doi.org/10.1111/j.1460-9568.1990.tb00417.x
Artola A, Singer W. The Involvement of N-Methyl-D-Aspartate Receptors in Induction and Maintenance of Long-Term Potentiation in Rat Visual Cortex. Eur J Neurosci. 1990;2(3):254-269. doi: 10.1111/j.1460-9568.1990.tb00417.x. PMID: 12106052.
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Modulation by NMDA Receptor Antagonists of Glycine Receptor Isoform Expression in Cultured Spinal Cord Neurons.

The European journal of neuroscience

Hoch W, Betz H, Schramm M, Wolters I, Becker CM.
PMID: 12106346
Eur J Neurosci. 1992;4(5):389-395. doi: 10.1111/j.1460-9568.1992.tb00887.x.

Two developmentally regulated isoforms of the inhibitory glycine receptor harbouring different alpha subunit variants, GlyRN (neonatal) and GlyRA (adult), have previously been identified in rodent spinal cord. Primary cultures of embyronic spinal neurons, however, express predominantly GlyRN. Here, N-methyl-d-aspartate...

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Hoch W, Betz H, Schramm M, et al. Modulation by NMDA Receptor Antagonists of Glycine Receptor Isoform Expression in Cultured Spinal Cord Neurons. Eur J Neurosci. 1992;4(5):389-395doi: 10.1111/j.1460-9568.1992.tb00887.x.
Hoch, W., Betz, H., Schramm, M., Wolters, I., & Becker, C. M. (1992). Modulation by NMDA Receptor Antagonists of Glycine Receptor Isoform Expression in Cultured Spinal Cord Neurons. The European journal of neuroscience, 4(5), 389-395. https://doi.org/10.1111/j.1460-9568.1992.tb00887.x
Hoch, W., et al. "Modulation by NMDA Receptor Antagonists of Glycine Receptor Isoform Expression in Cultured Spinal Cord Neurons." The European journal of neuroscience vol. 4,5 (1992): 389-395. doi: https://doi.org/10.1111/j.1460-9568.1992.tb00887.x
Hoch W, Betz H, Schramm M, Wolters I, Becker CM. Modulation by NMDA Receptor Antagonists of Glycine Receptor Isoform Expression in Cultured Spinal Cord Neurons. Eur J Neurosci. 1992;4(5):389-395. doi: 10.1111/j.1460-9568.1992.tb00887.x. PMID: 12106346.
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Long-term Potentiation in the Striatum is Unmasked by Removing the Voltage-dependent Magnesium Block of NMDA Receptor Channels.

The European journal of neuroscience

Calabresi P, Pisani A, Mercuri NB, Bernardi G.
PMID: 12106428
Eur J Neurosci. 1992;4(10):929-935. doi: 10.1111/j.1460-9568.1992.tb00119.x.

We have studied the effects of tetanic stimulation of the corticostriatal pathway on the amplitude of striatal excitatory synaptic potentials. Recordings were obtained from a corticostriatal slice preparation by utilizing both extracellular and intracellular techniques. Under the control condition...

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Calabresi P, Pisani A, Mercuri NB, et al. Long-term Potentiation in the Striatum is Unmasked by Removing the Voltage-dependent Magnesium Block of NMDA Receptor Channels. Eur J Neurosci. 1992;4(10):929-935doi: 10.1111/j.1460-9568.1992.tb00119.x.
Calabresi, P., Pisani, A., Mercuri, N. B., & Bernardi, G. (1992). Long-term Potentiation in the Striatum is Unmasked by Removing the Voltage-dependent Magnesium Block of NMDA Receptor Channels. The European journal of neuroscience, 4(10), 929-935. https://doi.org/10.1111/j.1460-9568.1992.tb00119.x
Calabresi, P., et al. "Long-term Potentiation in the Striatum is Unmasked by Removing the Voltage-dependent Magnesium Block of NMDA Receptor Channels." The European journal of neuroscience vol. 4,10 (1992): 929-935. doi: https://doi.org/10.1111/j.1460-9568.1992.tb00119.x
Calabresi P, Pisani A, Mercuri NB, Bernardi G. Long-term Potentiation in the Striatum is Unmasked by Removing the Voltage-dependent Magnesium Block of NMDA Receptor Channels. Eur J Neurosci. 1992;4(10):929-935. doi: 10.1111/j.1460-9568.1992.tb00119.x. PMID: 12106428.
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Onset Characteristics of Long-Term Potentiation in the Guinea-Pig Hippocampal CA1 Region in Vitro.

The European journal of neuroscience

Gustafsson B, Asztely F, Hanse E, Wigström H.
PMID: 12106147
Eur J Neurosci. 1989 Jul;1(4):382-394. doi: 10.1111/j.1460-9568.1989.tb00803.x.

The temporal development of long-term potentiation (LTP) was examined in the CA1 region of the hippocampal slice preparation (bath temperature 30 degrees C). LTP was evoked by a single brief afferent tetanus (3 - 40 impulses at 50 Hz)...

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Gustafsson B, Asztely F, Hanse E, et al. Onset Characteristics of Long-Term Potentiation in the Guinea-Pig Hippocampal CA1 Region in Vitro. Eur J Neurosci. 1989;1(4):382-394doi: 10.1111/j.1460-9568.1989.tb00803.x.
Gustafsson, B., Asztely, F., Hanse, E., & Wigström, H. (1989). Onset Characteristics of Long-Term Potentiation in the Guinea-Pig Hippocampal CA1 Region in Vitro. The European journal of neuroscience, 1(4), 382-394. https://doi.org/10.1111/j.1460-9568.1989.tb00803.x
Gustafsson, B., et al. "Onset Characteristics of Long-Term Potentiation in the Guinea-Pig Hippocampal CA1 Region in Vitro." The European journal of neuroscience vol. 1,4 (1989): 382-394. doi: https://doi.org/10.1111/j.1460-9568.1989.tb00803.x
Gustafsson B, Asztely F, Hanse E, Wigström H. Onset Characteristics of Long-Term Potentiation in the Guinea-Pig Hippocampal CA1 Region in Vitro. Eur J Neurosci. 1989 Jul;1(4):382-394. doi: 10.1111/j.1460-9568.1989.tb00803.x. PMID: 12106147.
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Modulation of GABA-mediated Synaptic Potentials by Glutamatergic Agonists in Neonatal CA3 Rat Hippocampal Neurons.

The European journal of neuroscience

Gaiarsa JL, Corradetti R, Cherubini E, Ben-Ari Y.
PMID: 12106187
Eur J Neurosci. 1991;3(4):301-309. doi: 10.1111/j.1460-9568.1991.tb00816.x.

Intracellular recordings were made from slices of adult and neonatal hippocampal neurons. During the first 2 weeks of life the majority of pyramidal cells exhibited spontaneous gamma-aminobutyric acid (GABA)-mediated synaptic potentials, which were depolarizing at birth and became hyperpolarizing...

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Gaiarsa JL, Corradetti R, Cherubini E, et al. Modulation of GABA-mediated Synaptic Potentials by Glutamatergic Agonists in Neonatal CA3 Rat Hippocampal Neurons. Eur J Neurosci. 1991;3(4):301-309doi: 10.1111/j.1460-9568.1991.tb00816.x.
Gaiarsa, J. L., Corradetti, R., Cherubini, E., & Ben-Ari, Y. (1991). Modulation of GABA-mediated Synaptic Potentials by Glutamatergic Agonists in Neonatal CA3 Rat Hippocampal Neurons. The European journal of neuroscience, 3(4), 301-309. https://doi.org/10.1111/j.1460-9568.1991.tb00816.x
Gaiarsa, Jean-Luc, et al. "Modulation of GABA-mediated Synaptic Potentials by Glutamatergic Agonists in Neonatal CA3 Rat Hippocampal Neurons." The European journal of neuroscience vol. 3,4 (1991): 301-309. doi: https://doi.org/10.1111/j.1460-9568.1991.tb00816.x
Gaiarsa JL, Corradetti R, Cherubini E, Ben-Ari Y. Modulation of GABA-mediated Synaptic Potentials by Glutamatergic Agonists in Neonatal CA3 Rat Hippocampal Neurons. Eur J Neurosci. 1991;3(4):301-309. doi: 10.1111/j.1460-9568.1991.tb00816.x. PMID: 12106187.
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Extracellular purine catabolite and amino acid levels in the rat striatum during severe hypoglycemia: Effects of 2-amino-5-phosphonovalerate.

Neurochemistry international

Butcher SP, Hagberg H, Sandberg M, Hamberger A.
PMID: 20501148
Neurochem Int. 1987;11(1):95-9. doi: 10.1016/0197-0186(87)90154-9.

The effects of d-2-amino-5-phosphonovalerate on severe hypoglycemia-induced alterations in extracellular levels of amino acids and purine catabolites were examined in the rat striatum using the brain microdialysis technique. The extracellular concentrations of purine catabolites (hypoxanthine, xanthine, inosine and adenosine)...

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Butcher SP, Hagberg H, Sandberg M, et al. Extracellular purine catabolite and amino acid levels in the rat striatum during severe hypoglycemia: Effects of 2-amino-5-phosphonovalerate. Neurochem Int. 1987;11(1):95-9doi: 10.1016/0197-0186(87)90154-9.
Butcher, S. P., Hagberg, H., Sandberg, M., & Hamberger, A. (1987). Extracellular purine catabolite and amino acid levels in the rat striatum during severe hypoglycemia: Effects of 2-amino-5-phosphonovalerate. Neurochemistry international, 11(1), 95-9. https://doi.org/10.1016/0197-0186(87)90154-9
Butcher, S P, et al. "Extracellular purine catabolite and amino acid levels in the rat striatum during severe hypoglycemia: Effects of 2-amino-5-phosphonovalerate." Neurochemistry international vol. 11,1 (1987): 95-9. doi: https://doi.org/10.1016/0197-0186(87)90154-9
Butcher SP, Hagberg H, Sandberg M, Hamberger A. Extracellular purine catabolite and amino acid levels in the rat striatum during severe hypoglycemia: Effects of 2-amino-5-phosphonovalerate. Neurochem Int. 1987;11(1):95-9. doi: 10.1016/0197-0186(87)90154-9. PMID: 20501148.
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Corticosterone enhances the potency of ethanol against hippocampal long-term potentiation via local neurosteroid synthesis.

Frontiers in cellular neuroscience

Izumi Y, O'Dell KA, Zorumski CF.
PMID: 26190975
Front Cell Neurosci. 2015 Jul 03;9:254. doi: 10.3389/fncel.2015.00254. eCollection 2015.

Corticosterone is known to accumulate in brain after various stressors including alcohol intoxication. Just as severe alcohol intoxication is typically required to impair memory formation only high concentrations of ethanol (60 mM) acutely inhibit long-term potentiation (LTP), a cellular...

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Izumi Y, O'Dell KA, Zorumski CF. Corticosterone enhances the potency of ethanol against hippocampal long-term potentiation via local neurosteroid synthesis. Front Cell Neurosci. 2015;9:254doi: 10.3389/fncel.2015.00254.
Izumi, Y., O'Dell, K. A., & Zorumski, C. F. (2015). Corticosterone enhances the potency of ethanol against hippocampal long-term potentiation via local neurosteroid synthesis. Frontiers in cellular neuroscience, 9254. https://doi.org/10.3389/fncel.2015.00254
Izumi, Yukitoshi, et al. "Corticosterone enhances the potency of ethanol against hippocampal long-term potentiation via local neurosteroid synthesis." Frontiers in cellular neuroscience vol. 9 (2015): 254. doi: https://doi.org/10.3389/fncel.2015.00254
Izumi Y, O'Dell KA, Zorumski CF. Corticosterone enhances the potency of ethanol against hippocampal long-term potentiation via local neurosteroid synthesis. Front Cell Neurosci. 2015 Jul 03;9:254. doi: 10.3389/fncel.2015.00254. eCollection 2015. PMID: 26190975; PMCID: PMC4490241.
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Glutamate as the Principal Mossy Fibre Transmitter in Rat Cerebellum: pharmacological evidence.

The European journal of neuroscience

Garthwaite J, Brodbelt AR.
PMID: 12106061
Eur J Neurosci. 1990 Feb;2(2):177-180. doi: 10.1111/j.1460-9568.1990.tb00410.x.

To examine the possibility that glutamate may be widely used as the transmitter for cerebellar mossy fibres, population responses of granule cells following electrical stimulation of these fibres were recorded in rat cerebellar slices using a gap technique. Several...

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Garthwaite J, Brodbelt AR. Glutamate as the Principal Mossy Fibre Transmitter in Rat Cerebellum: pharmacological evidence. Eur J Neurosci. 1990;2(2):177-180doi: 10.1111/j.1460-9568.1990.tb00410.x.
Garthwaite, J., & Brodbelt, A. R. (1990). Glutamate as the Principal Mossy Fibre Transmitter in Rat Cerebellum: pharmacological evidence. The European journal of neuroscience, 2(2), 177-180. https://doi.org/10.1111/j.1460-9568.1990.tb00410.x
Garthwaite, J., and Brodbelt, A. R.. "Glutamate as the Principal Mossy Fibre Transmitter in Rat Cerebellum: pharmacological evidence." The European journal of neuroscience vol. 2,2 (1990): 177-180. doi: https://doi.org/10.1111/j.1460-9568.1990.tb00410.x
Garthwaite J, Brodbelt AR. Glutamate as the Principal Mossy Fibre Transmitter in Rat Cerebellum: pharmacological evidence. Eur J Neurosci. 1990 Feb;2(2):177-180. doi: 10.1111/j.1460-9568.1990.tb00410.x. PMID: 12106061.
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Activation of N-methyl-d-aspartate Receptors Induces Endogenous Rhythmic Bursting Activities in Nucleus Tractus Solitarii Neurons: An Intracellular Study on Adult Rat Brainstem Slices.

The European journal of neuroscience

Tell F, Jean A.
PMID: 12106233
Eur J Neurosci. 1991;3(12):1353-1365. doi: 10.1111/j.1460-9568.1991.tb00068.x.

A brainstem slice preparation and intracellular recording techniques were used to examine the effects of N-methyl-d-aspartate (NMDA) application on neurons within the swallowing area of the nucleus tractus solitarii (NTS). According to their cellular properties, NTS neurons were classified...

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Tell F, Jean A. Activation of N-methyl-d-aspartate Receptors Induces Endogenous Rhythmic Bursting Activities in Nucleus Tractus Solitarii Neurons: An Intracellular Study on Adult Rat Brainstem Slices. Eur J Neurosci. 1991;3(12):1353-1365doi: 10.1111/j.1460-9568.1991.tb00068.x.
Tell, F., & Jean, A. (1991). Activation of N-methyl-d-aspartate Receptors Induces Endogenous Rhythmic Bursting Activities in Nucleus Tractus Solitarii Neurons: An Intracellular Study on Adult Rat Brainstem Slices. The European journal of neuroscience, 3(12), 1353-1365. https://doi.org/10.1111/j.1460-9568.1991.tb00068.x
Tell, Fabien, and Jean, André. "Activation of N-methyl-d-aspartate Receptors Induces Endogenous Rhythmic Bursting Activities in Nucleus Tractus Solitarii Neurons: An Intracellular Study on Adult Rat Brainstem Slices." The European journal of neuroscience vol. 3,12 (1991): 1353-1365. doi: https://doi.org/10.1111/j.1460-9568.1991.tb00068.x
Tell F, Jean A. Activation of N-methyl-d-aspartate Receptors Induces Endogenous Rhythmic Bursting Activities in Nucleus Tractus Solitarii Neurons: An Intracellular Study on Adult Rat Brainstem Slices. Eur J Neurosci. 1991;3(12):1353-1365. doi: 10.1111/j.1460-9568.1991.tb00068.x. PMID: 12106233.
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Characterization of l-2-Amino-4-Phosphonobutanoate Action Following Sensitization by Quisqualate in Rat Hippocampal Slice Cultures.

The European journal of neuroscience

Charpak S, Thompson SM, Gähwiler BH, Gerber U.
PMID: 12106335
Eur J Neurosci. 1992;4(6):491-499. doi: 10.1111/j.1460-9568.1992.tb00899.x.

An excitatory action of l-2-amino-4-phosphonobutanoate (l-AP4), a glutamate analogue, is observed following pre-exposure of tissue to quisqualate. We have studied the mechanism of sensitization of l-AP4 responses by quisqualate in voltage-clamped CA3 pyramidal cells in rat hippocampal slice cultures...

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Charpak S, Thompson SM, Gähwiler BH, et al. Characterization of l-2-Amino-4-Phosphonobutanoate Action Following Sensitization by Quisqualate in Rat Hippocampal Slice Cultures. Eur J Neurosci. 1992;4(6):491-499doi: 10.1111/j.1460-9568.1992.tb00899.x.
Charpak, S., Thompson, S. M., Gähwiler, B. H., & Gerber, U. (1992). Characterization of l-2-Amino-4-Phosphonobutanoate Action Following Sensitization by Quisqualate in Rat Hippocampal Slice Cultures. The European journal of neuroscience, 4(6), 491-499. https://doi.org/10.1111/j.1460-9568.1992.tb00899.x
Charpak, Serge, et al. "Characterization of l-2-Amino-4-Phosphonobutanoate Action Following Sensitization by Quisqualate in Rat Hippocampal Slice Cultures." The European journal of neuroscience vol. 4,6 (1992): 491-499. doi: https://doi.org/10.1111/j.1460-9568.1992.tb00899.x
Charpak S, Thompson SM, Gähwiler BH, Gerber U. Characterization of l-2-Amino-4-Phosphonobutanoate Action Following Sensitization by Quisqualate in Rat Hippocampal Slice Cultures. Eur J Neurosci. 1992;4(6):491-499. doi: 10.1111/j.1460-9568.1992.tb00899.x. PMID: 12106335.
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Expression of the Neural Recognition Molecule L1 by Cultured Neural Cells is influenced by K+ and the Glutamate Receptor Agonist NMDA.

The European journal of neuroscience

Scherer M, Heller M, Schachner M.
PMID: 12106341
Eur J Neurosci. 1992;4(6):554-562. doi: 10.1111/j.1460-9568.1992.tb00905.x.

To investigate the influence of neuronal activity on the expression of neural recognition molecules, cultures of neural cell lines and dissociated cells of early postnatal mouse cerebellum were maintained in the presence of elevated concentrations of K+ and the...

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Scherer M, Heller M, Schachner M. Expression of the Neural Recognition Molecule L1 by Cultured Neural Cells is influenced by K+ and the Glutamate Receptor Agonist NMDA. Eur J Neurosci. 1992;4(6):554-562doi: 10.1111/j.1460-9568.1992.tb00905.x.
Scherer, M., Heller, M., & Schachner, M. (1992). Expression of the Neural Recognition Molecule L1 by Cultured Neural Cells is influenced by K+ and the Glutamate Receptor Agonist NMDA. The European journal of neuroscience, 4(6), 554-562. https://doi.org/10.1111/j.1460-9568.1992.tb00905.x
Scherer, Martin, et al. "Expression of the Neural Recognition Molecule L1 by Cultured Neural Cells is influenced by K+ and the Glutamate Receptor Agonist NMDA." The European journal of neuroscience vol. 4,6 (1992): 554-562. doi: https://doi.org/10.1111/j.1460-9568.1992.tb00905.x
Scherer M, Heller M, Schachner M. Expression of the Neural Recognition Molecule L1 by Cultured Neural Cells is influenced by K+ and the Glutamate Receptor Agonist NMDA. Eur J Neurosci. 1992;4(6):554-562. doi: 10.1111/j.1460-9568.1992.tb00905.x. PMID: 12106341.
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Showing 1 to 12 of 19 entries