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Quinolinate-like neurotoxicity produced by aminooxyacetic acid in rat striatum.

Amino acids

Turski WA, Urbańska E, Sieklucka M, Ikonomidou C.
PMID: 24192903
Amino Acids. 1992 Oct;2(3):245-53. doi: 10.1007/BF00805946.

The endogenous tryptophan metabolite quinolinic acid elicits in rodent brain a pattern of neuronal degeneration which resembles that caused by L-glutamate. Its qualities as a neurotoxic agent raised the hypothesis that quinolinic acid might be involved in the pathogenesis...

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Turski WA, Urbańska E, Sieklucka M, et al. Quinolinate-like neurotoxicity produced by aminooxyacetic acid in rat striatum. Amino Acids. 1992;2(3):245-53doi: 10.1007/BF00805946.
Turski, W. A., Urbańska, E., Sieklucka, M., & Ikonomidou, C. (1992). Quinolinate-like neurotoxicity produced by aminooxyacetic acid in rat striatum. Amino acids, 2(3), 245-53. https://doi.org/10.1007/BF00805946
Turski, W A, et al. "Quinolinate-like neurotoxicity produced by aminooxyacetic acid in rat striatum." Amino acids vol. 2,3 (1992): 245-53. doi: https://doi.org/10.1007/BF00805946
Turski WA, Urbańska E, Sieklucka M, Ikonomidou C. Quinolinate-like neurotoxicity produced by aminooxyacetic acid in rat striatum. Amino Acids. 1992 Oct;2(3):245-53. doi: 10.1007/BF00805946. PMID: 24192903.
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l-Homocysteate Preferentially Activates N-methyl-D-aspartate Receptors to CA1 Rat Hippocampal Neurons.

The European journal of neuroscience

Provini L, Ito S, Ben Ari Y, Cherubini E.
PMID: 12106254
Eur J Neurosci. 1991;3(10):962-970. doi: 10.1111/j.1460-9568.1991.tb00032.x.

Intracellular recordings and current and single-electrode voltage-clamp techniques were used to study the membrane responses of CA1 pyramidal neurons to bath application of l-homocysteic acid (l-HC) in the rat hippocampal slice preparation. In control artificial cerebrospinal fluid (ACSF), l-HC...

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Provini L, Ito S, Ben Ari Y, et al. l-Homocysteate Preferentially Activates N-methyl-D-aspartate Receptors to CA1 Rat Hippocampal Neurons. Eur J Neurosci. 1991;3(10):962-970doi: 10.1111/j.1460-9568.1991.tb00032.x.
Provini, L., Ito, S., Ben Ari, Y., & Cherubini, E. (1991). l-Homocysteate Preferentially Activates N-methyl-D-aspartate Receptors to CA1 Rat Hippocampal Neurons. The European journal of neuroscience, 3(10), 962-970. https://doi.org/10.1111/j.1460-9568.1991.tb00032.x
Provini, L., et al. "l-Homocysteate Preferentially Activates N-methyl-D-aspartate Receptors to CA1 Rat Hippocampal Neurons." The European journal of neuroscience vol. 3,10 (1991): 962-970. doi: https://doi.org/10.1111/j.1460-9568.1991.tb00032.x
Provini L, Ito S, Ben Ari Y, Cherubini E. l-Homocysteate Preferentially Activates N-methyl-D-aspartate Receptors to CA1 Rat Hippocampal Neurons. Eur J Neurosci. 1991;3(10):962-970. doi: 10.1111/j.1460-9568.1991.tb00032.x. PMID: 12106254.
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The specificity of glutamate inhibition of protein synthesis in synaptosomal fractions from rat cerebral cortex.

Neurochemistry international

Cheung MK.
PMID: 20504497
Neurochem Int. 1989;15(3):293-300. doi: 10.1016/0197-0186(89)90135-6.

In vitro addition of glutamate (GLU) resulted in a dose-dependent inhibition of protein synthesis in synaptosomes from adult rat brain cortex. There was significant (20-25%) inhibition at 25 ?M GLU and a maximum (30-50%) inhibition was observed at [GLU]...

Cite
Cheung MK. The specificity of glutamate inhibition of protein synthesis in synaptosomal fractions from rat cerebral cortex. Neurochem Int. 1989;15(3):293-300doi: 10.1016/0197-0186(89)90135-6.
Cheung, M. K. (1989). The specificity of glutamate inhibition of protein synthesis in synaptosomal fractions from rat cerebral cortex. Neurochemistry international, 15(3), 293-300. https://doi.org/10.1016/0197-0186(89)90135-6
Cheung, M K. "The specificity of glutamate inhibition of protein synthesis in synaptosomal fractions from rat cerebral cortex." Neurochemistry international vol. 15,3 (1989): 293-300. doi: https://doi.org/10.1016/0197-0186(89)90135-6
Cheung MK. The specificity of glutamate inhibition of protein synthesis in synaptosomal fractions from rat cerebral cortex. Neurochem Int. 1989;15(3):293-300. doi: 10.1016/0197-0186(89)90135-6. PMID: 20504497.
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