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Biological evaluation of transdichloridoplatinum(II) complexes with 3- and 4-acetylpyridine in comparison to cisplatin.

Radiology and oncology

Filipovic L, Arandelovic S, Gligorijevic N, Krivokuca A, Jankovic R, Srdic-Rajic T, Rakic G, Tesic Z, Radulovic S.
PMID: 24294179
Radiol Oncol. 2013 Oct 08;47(4):346-57. doi: 10.2478/raon-2013-0050. eCollection 2013.

BACKGROUND: In our previous study we reported the synthesis and cytotoxicity of two trans-platinum(II) complexes: trans-[PtCl2(3-acetylpyridine)2] (1) and trans-[PtCl2(4-acetylpyridine)2] (2), revealing significant cytotoxic potential of 2. In order to evaluate the mechanism underlying biological activity of both trans-Pt(II) isomers,...

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Filipovic L, Arandelovic S, Gligorijevic N, et al. Biological evaluation of transdichloridoplatinum(II) complexes with 3- and 4-acetylpyridine in comparison to cisplatin. Radiol Oncol. 2013;47(4):346-57doi: 10.2478/raon-2013-0050.
Filipovic, L., Arandelovic, S., Gligorijevic, N., Krivokuca, A., Jankovic, R., Srdic-Rajic, T., Rakic, G., Tesic, Z., & Radulovic, S. (2013). Biological evaluation of transdichloridoplatinum(II) complexes with 3- and 4-acetylpyridine in comparison to cisplatin. Radiology and oncology, 47(4), 346-57. https://doi.org/10.2478/raon-2013-0050
Filipovic, Lana, et al. "Biological evaluation of transdichloridoplatinum(II) complexes with 3- and 4-acetylpyridine in comparison to cisplatin." Radiology and oncology vol. 47,4 (2013): 346-57. doi: https://doi.org/10.2478/raon-2013-0050
Filipovic L, Arandelovic S, Gligorijevic N, Krivokuca A, Jankovic R, Srdic-Rajic T, Rakic G, Tesic Z, Radulovic S. Biological evaluation of transdichloridoplatinum(II) complexes with 3- and 4-acetylpyridine in comparison to cisplatin. Radiol Oncol. 2013 Oct 08;47(4):346-57. doi: 10.2478/raon-2013-0050. eCollection 2013. PMID: 24294179; PMCID: PMC3814279.
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Malic Dehyrogenases in Sea Urchin Eggs.

Science (New York, N.Y.)

Moore RO, Villee CA.
PMID: 17753946
Science. 1962 Oct 26;138(3539):508-9. doi: 10.1126/science.138.3539.508.

Unfertilized Arbacia eggs have five electrophoretically distinct malic dehydrogenases which differ in their rates with pyridine nucleotide analogs. Both the number of peaks and their relative size change during early embryonic development. A D-malic dehydrogenase which utilizes the 3-acetylpyridine...

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Moore RO, Villee CA. Malic Dehyrogenases in Sea Urchin Eggs. Science. 1962;138(3539):508-9doi: 10.1126/science.138.3539.508.
Moore, R. O., & Villee, C. A. (1962). Malic Dehyrogenases in Sea Urchin Eggs. Science (New York, N.Y.), 138(3539), 508-9. https://doi.org/10.1126/science.138.3539.508
Moore, R O, and Villee, C A. "Malic Dehyrogenases in Sea Urchin Eggs." Science (New York, N.Y.) vol. 138,3539 (1962): 508-9. doi: https://doi.org/10.1126/science.138.3539.508
Moore RO, Villee CA. Malic Dehyrogenases in Sea Urchin Eggs. Science. 1962 Oct 26;138(3539):508-9. doi: 10.1126/science.138.3539.508. PMID: 17753946.
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trans-Bis(3-acetylpyridine-kappaN)diiodoplatinum(II).

Acta crystallographica. Section C, Crystal structure communications

Tessier C, Rochon FD.
PMID: 17148903
Acta Crystallogr C. 2006 Dec;62:m620-2. doi: 10.1107/S0108270106045197. Epub 2006 Nov 22.

In the square-planar title complex, [PtI2(C7H7NO)2], the Pt atom lies on a crystallographic inversion center, coinciding with an anti arrangement of the 3-acetylpyridine ligands. The dihedral angles between the pyridine rings and the Pt coordination plane are 67.5 (2)...

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Tessier C, Rochon FD. trans-Bis(3-acetylpyridine-kappaN)diiodoplatinum(II). Acta Crystallogr C. 2006;62:m620-2doi: 10.1107/S0108270106045197.
Tessier, C., & Rochon, F. D. (2006). trans-Bis(3-acetylpyridine-kappaN)diiodoplatinum(II). Acta crystallographica. Section C, Crystal structure communications, 62m620-2. https://doi.org/10.1107/S0108270106045197
Tessier, Christian, and Rochon, Fernande D. "trans-Bis(3-acetylpyridine-kappaN)diiodoplatinum(II)." Acta crystallographica. Section C, Crystal structure communications vol. 62 (2006): m620-2. doi: https://doi.org/10.1107/S0108270106045197
Tessier C, Rochon FD. trans-Bis(3-acetylpyridine-kappaN)diiodoplatinum(II). Acta Crystallogr C. 2006 Dec;62:m620-2. doi: 10.1107/S0108270106045197. Epub 2006 Nov 22. PMID: 17148903.
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Computational insights into the neuroprotective action of riluzole on 3-acetylpyridine-induced ataxia in rats.

Cell journal

Abbasi S, Edrisi M, Mahnam A, Janahmadi M.
PMID: 23862110
Cell J. 2013;15(2):98-107. Epub 2013 Jul 02.

OBJECTIVE: Intra-peritoneal administration of riluzole has been shown to preserve the membrane properties and firing characteristics of Purkinje neurons in a rat model of cerebellar ataxia induced by 3-acetylpyridine (3-AP). However, the exact mechanism(s) by which riluzole restores the...

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Abbasi S, Edrisi M, Mahnam A, et al. Computational insights into the neuroprotective action of riluzole on 3-acetylpyridine-induced ataxia in rats. Cell J. 2013;15(2):98-107
Abbasi, S., Edrisi, M., Mahnam, A., & Janahmadi, M. (2013). Computational insights into the neuroprotective action of riluzole on 3-acetylpyridine-induced ataxia in rats. Cell journal, 15(2), 98-107.
Abbasi, Samira, et al. "Computational insights into the neuroprotective action of riluzole on 3-acetylpyridine-induced ataxia in rats." Cell journal vol. 15,2 (2013): 98-107.
Abbasi S, Edrisi M, Mahnam A, Janahmadi M. Computational insights into the neuroprotective action of riluzole on 3-acetylpyridine-induced ataxia in rats. Cell J. 2013;15(2):98-107. Epub 2013 Jul 02. PMID: 23862110; PMCID: PMC3712782.
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Melittin administration ameliorates motor function, prevents apoptotic cell death and protects Purkinje neurons in the rat model of cerebellar ataxia induced by 3-Acetylpyridine.

Toxicon : official journal of the International Society on Toxinology

Ghorbani Z, Abdollahifar MA, Vakili K, Moghaddam MH, Mehdizadeh M, Marzban H, Rasoolijazi H, Aliaghaei A.
PMID: 34793821
Toxicon. 2021 Nov 15;205:57-66. doi: 10.1016/j.toxicon.2021.11.008. Epub 2021 Nov 15.

Cerebellar ataxia (CA) is a condition in which cerebellar dysfunction leads to movement disorders such as dysmetria, asynergy and dysdiadochokinesia. This study investigates the therapeutic effects of Melittin (MEL) on 3-acetylpyridine-induced (3-AP) cerebellar ataxia (CA) rat model. Initially, CA...

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Ghorbani Z, Abdollahifar MA, Vakili K, et al. Melittin administration ameliorates motor function, prevents apoptotic cell death and protects Purkinje neurons in the rat model of cerebellar ataxia induced by 3-Acetylpyridine. Toxicon. 2021;205:57-66doi: 10.1016/j.toxicon.2021.11.008.
Ghorbani, Z., Abdollahifar, M. A., Vakili, K., Moghaddam, M. H., Mehdizadeh, M., Marzban, H., Rasoolijazi, H., & Aliaghaei, A. (2021). Melittin administration ameliorates motor function, prevents apoptotic cell death and protects Purkinje neurons in the rat model of cerebellar ataxia induced by 3-Acetylpyridine. Toxicon : official journal of the International Society on Toxinology, 20557-66. https://doi.org/10.1016/j.toxicon.2021.11.008
Ghorbani, Zeynab, et al. "Melittin administration ameliorates motor function, prevents apoptotic cell death and protects Purkinje neurons in the rat model of cerebellar ataxia induced by 3-Acetylpyridine." Toxicon : official journal of the International Society on Toxinology vol. 205 (2021): 57-66. doi: https://doi.org/10.1016/j.toxicon.2021.11.008
Ghorbani Z, Abdollahifar MA, Vakili K, Moghaddam MH, Mehdizadeh M, Marzban H, Rasoolijazi H, Aliaghaei A. Melittin administration ameliorates motor function, prevents apoptotic cell death and protects Purkinje neurons in the rat model of cerebellar ataxia induced by 3-Acetylpyridine. Toxicon. 2021 Nov 15;205:57-66. doi: 10.1016/j.toxicon.2021.11.008. Epub 2021 Nov 15. PMID: 34793821.
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Spontaneous Saccades and Gaze-Holding Ability in the Pigmented Rat. I. Effects of Inferior Olive Lesion.

The European journal of neuroscience

Strata P, Chelazzi L, Ghirardi M, Rossi F, Tempia F.
PMID: 12106068
Eur J Neurosci. 1990;2(12):1074-1084. doi: 10.1111/j.1460-9568.1990.tb00019.x.

We have studied the effects of lesion of the inferior olive on the spontaneous eye movements performed both in the light and dark in head restrained pigmented rats. The inferior olive lesion was made at least 1 month before...

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Strata P, Chelazzi L, Ghirardi M, et al. Spontaneous Saccades and Gaze-Holding Ability in the Pigmented Rat. I. Effects of Inferior Olive Lesion. Eur J Neurosci. 1990;2(12):1074-1084doi: 10.1111/j.1460-9568.1990.tb00019.x.
Strata, P., Chelazzi, L., Ghirardi, M., Rossi, F., & Tempia, F. (1990). Spontaneous Saccades and Gaze-Holding Ability in the Pigmented Rat. I. Effects of Inferior Olive Lesion. The European journal of neuroscience, 2(12), 1074-1084. https://doi.org/10.1111/j.1460-9568.1990.tb00019.x
Strata, P., et al. "Spontaneous Saccades and Gaze-Holding Ability in the Pigmented Rat. I. Effects of Inferior Olive Lesion." The European journal of neuroscience vol. 2,12 (1990): 1074-1084. doi: https://doi.org/10.1111/j.1460-9568.1990.tb00019.x
Strata P, Chelazzi L, Ghirardi M, Rossi F, Tempia F. Spontaneous Saccades and Gaze-Holding Ability in the Pigmented Rat. I. Effects of Inferior Olive Lesion. Eur J Neurosci. 1990;2(12):1074-1084. doi: 10.1111/j.1460-9568.1990.tb00019.x. PMID: 12106068.
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TGF-β1 Provides Neuroprotection via Inhibition of Microglial Activation in 3-Acetylpyridine-Induced Cerebellar Ataxia Model Rats.

Frontiers in neuroscience

Cao BB, Zhang XX, Du CY, Liu Z, Qiu YH, Peng YP.
PMID: 32265625
Front Neurosci. 2020 Mar 20;14:187. doi: 10.3389/fnins.2020.00187. eCollection 2020.

Cerebellar ataxias (CAs) consist of a heterogeneous group of neurodegenerative diseases hallmarked by motor deficits and deterioration of the cerebellum and its associated circuitries. Neuroinflammatory responses are present in CA brain, but how neuroinflammation may contribute to CA pathogenesis...

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Cao BB, Zhang XX, Du CY, et al. TGF-β1 Provides Neuroprotection via Inhibition of Microglial Activation in 3-Acetylpyridine-Induced Cerebellar Ataxia Model Rats. Front Neurosci. 2020;14:187doi: 10.3389/fnins.2020.00187.
Cao, B. B., Zhang, X. X., Du, C. Y., Liu, Z., Qiu, Y. H., & Peng, Y. P. (2020). TGF-β1 Provides Neuroprotection via Inhibition of Microglial Activation in 3-Acetylpyridine-Induced Cerebellar Ataxia Model Rats. Frontiers in neuroscience, 14187. https://doi.org/10.3389/fnins.2020.00187
Cao, Bei-Bei, et al. "TGF-β1 Provides Neuroprotection via Inhibition of Microglial Activation in 3-Acetylpyridine-Induced Cerebellar Ataxia Model Rats." Frontiers in neuroscience vol. 14 (2020): 187. doi: https://doi.org/10.3389/fnins.2020.00187
Cao BB, Zhang XX, Du CY, Liu Z, Qiu YH, Peng YP. TGF-β1 Provides Neuroprotection via Inhibition of Microglial Activation in 3-Acetylpyridine-Induced Cerebellar Ataxia Model Rats. Front Neurosci. 2020 Mar 20;14:187. doi: 10.3389/fnins.2020.00187. eCollection 2020. PMID: 32265625; PMCID: PMC7099147.
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Increase in the Urinary Excretion of Nicotinamide and Its Metabolites in Rats Administered 3-Acetylpyridine, an Antagonist of Niacin.

Bioscience, biotechnology, and biochemistry

Shibata K, Sumizaki J, Onodera M.
PMID: 27286219
Biosci Biotechnol Biochem. 1992 Jan;56(5):825. doi: 10.1271/bbb.56.825.

No abstract available.

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Shibata K, Sumizaki J, Onodera M. Increase in the Urinary Excretion of Nicotinamide and Its Metabolites in Rats Administered 3-Acetylpyridine, an Antagonist of Niacin. Biosci Biotechnol Biochem. 1992;56(5):825doi: 10.1271/bbb.56.825.
Shibata, K., Sumizaki, J., & Onodera, M. (1992). Increase in the Urinary Excretion of Nicotinamide and Its Metabolites in Rats Administered 3-Acetylpyridine, an Antagonist of Niacin. Bioscience, biotechnology, and biochemistry, 56(5), 825. https://doi.org/10.1271/bbb.56.825
Shibata, K, et al. "Increase in the Urinary Excretion of Nicotinamide and Its Metabolites in Rats Administered 3-Acetylpyridine, an Antagonist of Niacin." Bioscience, biotechnology, and biochemistry vol. 56,5 (1992): 825. doi: https://doi.org/10.1271/bbb.56.825
Shibata K, Sumizaki J, Onodera M. Increase in the Urinary Excretion of Nicotinamide and Its Metabolites in Rats Administered 3-Acetylpyridine, an Antagonist of Niacin. Biosci Biotechnol Biochem. 1992 Jan;56(5):825. doi: 10.1271/bbb.56.825. PMID: 27286219.
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Melittin administration ameliorates motor function, prevents apoptotic cell death and protects Purkinje neurons in the rat model of cerebellar ataxia induced by 3-Acetylpyridine.

Toxicon : official journal of the International Society on Toxinology

Ghorbani Z, Abdollahifar MA, Vakili K, Moghaddam MH, Mehdizadeh M, Marzban H, Rasoolijazi H, Aliaghaei A.
PMID: 34793821
Toxicon. 2022 Jan 15;205:57-66. doi: 10.1016/j.toxicon.2021.11.008. Epub 2021 Nov 15.

Cerebellar ataxia (CA) is a condition in which cerebellar dysfunction leads to movement disorders such as dysmetria, asynergy and dysdiadochokinesia. This study investigates the therapeutic effects of Melittin (MEL) on 3-acetylpyridine-induced (3-AP) cerebellar ataxia (CA) rat model. Initially, CA...

Cite
Ghorbani Z, Abdollahifar MA, Vakili K, et al. Melittin administration ameliorates motor function, prevents apoptotic cell death and protects Purkinje neurons in the rat model of cerebellar ataxia induced by 3-Acetylpyridine. Toxicon. 2021;205:57-66doi: 10.1016/j.toxicon.2021.11.008.
Ghorbani, Z., Abdollahifar, M. A., Vakili, K., Moghaddam, M. H., Mehdizadeh, M., Marzban, H., Rasoolijazi, H., & Aliaghaei, A. (2022). Melittin administration ameliorates motor function, prevents apoptotic cell death and protects Purkinje neurons in the rat model of cerebellar ataxia induced by 3-Acetylpyridine. Toxicon : official journal of the International Society on Toxinology, 20557-66. https://doi.org/10.1016/j.toxicon.2021.11.008
Ghorbani, Zeynab, et al. "Melittin administration ameliorates motor function, prevents apoptotic cell death and protects Purkinje neurons in the rat model of cerebellar ataxia induced by 3-Acetylpyridine." Toxicon : official journal of the International Society on Toxinology vol. 205 (2022): 57-66. doi: https://doi.org/10.1016/j.toxicon.2021.11.008
Ghorbani Z, Abdollahifar MA, Vakili K, Moghaddam MH, Mehdizadeh M, Marzban H, Rasoolijazi H, Aliaghaei A. Melittin administration ameliorates motor function, prevents apoptotic cell death and protects Purkinje neurons in the rat model of cerebellar ataxia induced by 3-Acetylpyridine. Toxicon. 2022 Jan 15;205:57-66. doi: 10.1016/j.toxicon.2021.11.008. Epub 2021 Nov 15. PMID: 34793821.
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Effect of Climbing Fibre Deprivation on the K+-evoked Release of Endogenous Adenosine from Rat Cerebellar Slices.

The European journal of neuroscience

Do KQ, Vollenweider FX, Zollinger M, Cuénod M.
PMID: 12106218
Eur J Neurosci. 1991;3(2):201-208. doi: 10.1111/j.1460-9568.1991.tb00080.x.

We report the identification of a compound whose K+-induced Ca2+-dependent release in rat cerebellar slices was reduced following climbing fibre deprivation by 3-acetylpyridine (3-AP) treatment. Based on HPLC retention time, UV absorption spectrum, and mass spectrometry, this compound was...

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Do KQ, Vollenweider FX, Zollinger M, et al. Effect of Climbing Fibre Deprivation on the K+-evoked Release of Endogenous Adenosine from Rat Cerebellar Slices. Eur J Neurosci. 1991;3(2):201-208doi: 10.1111/j.1460-9568.1991.tb00080.x.
Do, K. Q., Vollenweider, F. X., Zollinger, M., & Cuénod, M. (1991). Effect of Climbing Fibre Deprivation on the K+-evoked Release of Endogenous Adenosine from Rat Cerebellar Slices. The European journal of neuroscience, 3(2), 201-208. https://doi.org/10.1111/j.1460-9568.1991.tb00080.x
Do, Kim Quang, et al. "Effect of Climbing Fibre Deprivation on the K+-evoked Release of Endogenous Adenosine from Rat Cerebellar Slices." The European journal of neuroscience vol. 3,2 (1991): 201-208. doi: https://doi.org/10.1111/j.1460-9568.1991.tb00080.x
Do KQ, Vollenweider FX, Zollinger M, Cuénod M. Effect of Climbing Fibre Deprivation on the K+-evoked Release of Endogenous Adenosine from Rat Cerebellar Slices. Eur J Neurosci. 1991;3(2):201-208. doi: 10.1111/j.1460-9568.1991.tb00080.x. PMID: 12106218.
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An apparent oligomer of malate dehydrogenase from bean leaves.

Plant physiology

Habig W, Racusen D.
PMID: 16658714
Plant Physiol. 1974 Mar;53(3):402-10. doi: 10.1104/pp.53.3.402.

Two forms of malate dehydrogenase of widely differing molecular weight have been examined from primary leaves of Phaseolus vulgaris. In addition to the normal 69,000 molecular weight enzyme, an unusual form of 280,000 molecular weight may be detected by...

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Habig W, Racusen D. An apparent oligomer of malate dehydrogenase from bean leaves. Plant Physiol. 1974;53(3):402-10doi: 10.1104/pp.53.3.402.
Habig, W., & Racusen, D. (1974). An apparent oligomer of malate dehydrogenase from bean leaves. Plant physiology, 53(3), 402-10. https://doi.org/10.1104/pp.53.3.402
Habig, W, and Racusen, D. "An apparent oligomer of malate dehydrogenase from bean leaves." Plant physiology vol. 53,3 (1974): 402-10. doi: https://doi.org/10.1104/pp.53.3.402
Habig W, Racusen D. An apparent oligomer of malate dehydrogenase from bean leaves. Plant Physiol. 1974 Mar;53(3):402-10. doi: 10.1104/pp.53.3.402. PMID: 16658714; PMCID: PMC543231.
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On the presence of a nicotinamide nucleotide transhydrogenase in mitochondria from potato tuber.

Plant physiology

Carlenor E, Persson B, Glaser E, Andersson B, Rydström J.
PMID: 16666299
Plant Physiol. 1988 Oct;88(2):303-8. doi: 10.1104/pp.88.2.303.

Mitochondria isolated from potato (Solanum tuberosum L.) tuber were investigated for the presence of a nicotinamide nucleotide transhydrogenase activity. Submitochondrial particles derived from these mitochondria by sonication catalyzed a reduction of NAD(+) or 3-acetylpyridine-NAD(+) by NADPH, which showed a...

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Carlenor E, Persson B, Glaser E, et al. On the presence of a nicotinamide nucleotide transhydrogenase in mitochondria from potato tuber. Plant Physiol. 1988;88(2):303-8doi: 10.1104/pp.88.2.303.
Carlenor, E., Persson, B., Glaser, E., Andersson, B., & Rydström, J. (1988). On the presence of a nicotinamide nucleotide transhydrogenase in mitochondria from potato tuber. Plant physiology, 88(2), 303-8. https://doi.org/10.1104/pp.88.2.303
Carlenor, E, et al. "On the presence of a nicotinamide nucleotide transhydrogenase in mitochondria from potato tuber." Plant physiology vol. 88,2 (1988): 303-8. doi: https://doi.org/10.1104/pp.88.2.303
Carlenor E, Persson B, Glaser E, Andersson B, Rydström J. On the presence of a nicotinamide nucleotide transhydrogenase in mitochondria from potato tuber. Plant Physiol. 1988 Oct;88(2):303-8. doi: 10.1104/pp.88.2.303. PMID: 16666299; PMCID: PMC1055572.
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