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Autoradiographic localization of [(3)H]paroxetine specific binding in the rat brain.

Neurochemistry international

Gobbi M, Cervo L, Taddei C, Mennini T.
PMID: 20504564
Neurochem Int. 1990;16(3):247-51. doi: 10.1016/0197-0186(90)90098-e.

Specific binding of [(3)H]paroxetine, a selective serotonin uptake inhibitor, has recently been described. We used the autoradiographic technique to establish the regional distribution of these binding sites in the brain of control and 5,7-dihydroxytryptamine (5,7-DHT) lesioned rats. Binding levels...

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Gobbi M, Cervo L, Taddei C, et al. Autoradiographic localization of [(3)H]paroxetine specific binding in the rat brain. Neurochem Int. 1990;16(3):247-51doi: 10.1016/0197-0186(90)90098-e.
Gobbi, M., Cervo, L., Taddei, C., & Mennini, T. (1990). Autoradiographic localization of [(3)H]paroxetine specific binding in the rat brain. Neurochemistry international, 16(3), 247-51. https://doi.org/10.1016/0197-0186(90)90098-e
Gobbi, M, et al. "Autoradiographic localization of [(3)H]paroxetine specific binding in the rat brain." Neurochemistry international vol. 16,3 (1990): 247-51. doi: https://doi.org/10.1016/0197-0186(90)90098-e
Gobbi M, Cervo L, Taddei C, Mennini T. Autoradiographic localization of [(3)H]paroxetine specific binding in the rat brain. Neurochem Int. 1990;16(3):247-51. doi: 10.1016/0197-0186(90)90098-e. PMID: 20504564.
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Established amyloid-β pathology is unaffected by chronic treatment with the selective serotonin reuptake inhibitor paroxetine.

Alzheimer's & dementia (New York, N. Y.)

Severino M, Sivasaravanaparan M, Olesen LØ, von Linstow CU, Metaxas A, Bouzinova EV, Khan AM, Lambertsen KL, Babcock AA, Gramsbergen JB, Wiborg O, Finsen B.
PMID: 29955664
Alzheimers Dement (N Y). 2018 May 24;4:215-223. doi: 10.1016/j.trci.2018.04.005. eCollection 2018.

INTRODUCTION: Treatment with selective serotonin reuptake inhibitors has been suggested to mitigate amyloid-β (Aβ) pathology in Alzheimer's disease, in addition to an antidepressant mechanism of action.METHODS: We investigated whether chronic treatment with paroxetine, a selective serotonin reuptake inhibitor, mitigates...

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Severino M, Sivasaravanaparan M, Olesen LØ, et al. Established amyloid-β pathology is unaffected by chronic treatment with the selective serotonin reuptake inhibitor paroxetine. Alzheimers Dement (N Y). 2018;4:215-223doi: 10.1016/j.trci.2018.04.005.
Severino, M., Sivasaravanaparan, M., Olesen, L. �. �., von Linstow, C. U., Metaxas, A., Bouzinova, E. V., Khan, A. M., Lambertsen, K. L., Babcock, A. A., Gramsbergen, J. B., Wiborg, O., & Finsen, B. (2018). Established amyloid-β pathology is unaffected by chronic treatment with the selective serotonin reuptake inhibitor paroxetine. Alzheimer's & dementia (New York, N. Y.), 4215-223. https://doi.org/10.1016/j.trci.2018.04.005
Severino, Maurizio, et al. "Established amyloid-β pathology is unaffected by chronic treatment with the selective serotonin reuptake inhibitor paroxetine." Alzheimer's & dementia (New York, N. Y.) vol. 4 (2018): 215-223. doi: https://doi.org/10.1016/j.trci.2018.04.005
Severino M, Sivasaravanaparan M, Olesen LØ, von Linstow CU, Metaxas A, Bouzinova EV, Khan AM, Lambertsen KL, Babcock AA, Gramsbergen JB, Wiborg O, Finsen B. Established amyloid-β pathology is unaffected by chronic treatment with the selective serotonin reuptake inhibitor paroxetine. Alzheimers Dement (N Y). 2018 May 24;4:215-223. doi: 10.1016/j.trci.2018.04.005. eCollection 2018. PMID: 29955664; PMCID: PMC6021554.
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The effect of BIMT 17, a new potential antidepressant, in the forced swimming test in mice.

Behavioural pharmacology

Cesana R, Ciprandi C, Borsini F.
PMID: 11224371
Behav Pharmacol. 1995 Nov;6(7):688-694.

BIMT 17 inhibits cortical electrical activity through the activation of cortical 5-HT(1A) receptors combined with the antagonism of 5-HT(2) receptors. The aim of this study was to evaluate the potential antidepressant activity of BIMT 17 and its mechanism of...

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Cesana R, Ciprandi C, Borsini F. The effect of BIMT 17, a new potential antidepressant, in the forced swimming test in mice. Behav Pharmacol. 1995;6(7):688-694
Cesana, R., Ciprandi, C., & Borsini, F. (1995). The effect of BIMT 17, a new potential antidepressant, in the forced swimming test in mice. Behavioural pharmacology, 6(7), 688-694.
Cesana, R., et al. "The effect of BIMT 17, a new potential antidepressant, in the forced swimming test in mice." Behavioural pharmacology vol. 6,7 (1995): 688-694.
Cesana R, Ciprandi C, Borsini F. The effect of BIMT 17, a new potential antidepressant, in the forced swimming test in mice. Behav Pharmacol. 1995 Nov;6(7):688-694. PMID: 11224371.
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The Serotonergic System Modulates the Cocaine-Induced Expression of the Immediate Early Genes egr-1 and c-fos in Rat Brain.

Annals of the New York Academy of Sciences

Humblot N, Thiriet N, Gobaille S, Aunis D, Zwiller J.
PMID: 29090823
Ann N Y Acad Sci. 1998 May;844(1):7-20. doi: 10.1111/j.1749-6632.1998.tb08217.x.

Transcription regulatory factors are rapidly induced in brain by a wide variety of stimuli and may be important in coordinating changes in gene expression underlying neuronal plasticity. Using in situ hybridization, we found that acute cocaine administration (20 mg/kg,...

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Humblot N, Thiriet N, Gobaille S, et al. The Serotonergic System Modulates the Cocaine-Induced Expression of the Immediate Early Genes egr-1 and c-fos in Rat Brain. Ann N Y Acad Sci. 1998;844(1):7-20doi: 10.1111/j.1749-6632.1998.tb08217.x.
Humblot, N., Thiriet, N., Gobaille, S., Aunis, D., & Zwiller, J. (1998). The Serotonergic System Modulates the Cocaine-Induced Expression of the Immediate Early Genes egr-1 and c-fos in Rat Brain. Annals of the New York Academy of Sciences, 844(1), 7-20. https://doi.org/10.1111/j.1749-6632.1998.tb08217.x
Humblot, N, et al. "The Serotonergic System Modulates the Cocaine-Induced Expression of the Immediate Early Genes egr-1 and c-fos in Rat Brain." Annals of the New York Academy of Sciences vol. 844,1 (1998): 7-20. doi: https://doi.org/10.1111/j.1749-6632.1998.tb08217.x
Humblot N, Thiriet N, Gobaille S, Aunis D, Zwiller J. The Serotonergic System Modulates the Cocaine-Induced Expression of the Immediate Early Genes egr-1 and c-fos in Rat Brain. Ann N Y Acad Sci. 1998 May;844(1):7-20. doi: 10.1111/j.1749-6632.1998.tb08217.x. PMID: 29090823.
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Molecular mechanism of biological action of the serotonergic neurotoxin 5,7-dihydroxytryptamine.

Neurochemistry international

Sinhababu AK, Borchardt RT.
PMID: 20501230
Neurochem Int. 1988;12(3):273-84. doi: 10.1016/0197-0186(88)90165-9.

5,7-Dihydroxytryptamine (5,7-DHT) is a selective serotonergic neurotoxin by virtue of its selective uptake into 5-hydroxytryptamine neurons and its ability to undergo autoxidation. The mechanism by which 5,7-DHT induces neurodegenerative effects remains enigmatic. The mechanism of autoxidation of 5,7-DHT, which...

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Sinhababu AK, Borchardt RT. Molecular mechanism of biological action of the serotonergic neurotoxin 5,7-dihydroxytryptamine. Neurochem Int. 1988;12(3):273-84doi: 10.1016/0197-0186(88)90165-9.
Sinhababu, A. K., & Borchardt, R. T. (1988). Molecular mechanism of biological action of the serotonergic neurotoxin 5,7-dihydroxytryptamine. Neurochemistry international, 12(3), 273-84. https://doi.org/10.1016/0197-0186(88)90165-9
Sinhababu, A K, and Borchardt, R T. "Molecular mechanism of biological action of the serotonergic neurotoxin 5,7-dihydroxytryptamine." Neurochemistry international vol. 12,3 (1988): 273-84. doi: https://doi.org/10.1016/0197-0186(88)90165-9
Sinhababu AK, Borchardt RT. Molecular mechanism of biological action of the serotonergic neurotoxin 5,7-dihydroxytryptamine. Neurochem Int. 1988;12(3):273-84. doi: 10.1016/0197-0186(88)90165-9. PMID: 20501230.
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A radioautographic and immunocytochemical study of the GABA systems of the habenula complex in the rat.

Neurochemistry international

Belin MF, Aguera M, Nanopoulos D, Gamrani H, Maitre M, Calas A, Pujol JF.
PMID: 20487881
Neurochem Int. 1982;4(4):303-12. doi: 10.1016/0197-0186(82)90067-5.

Radioautography of [(3)H]GABA accumulation and immunocytochemistry of glutamate decarboxylase have been used to study anatomically and morphologically the GABA system of the rat habenular (Hb) complex. Radioautographic visualisation of GABA specific neurons show a very high innervation of the...

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Belin MF, Aguera M, Nanopoulos D, et al. A radioautographic and immunocytochemical study of the GABA systems of the habenula complex in the rat. Neurochem Int. 1982;4(4):303-12doi: 10.1016/0197-0186(82)90067-5.
Belin, M. F., Aguera, M., Nanopoulos, D., Gamrani, H., Maitre, M., Calas, A., & Pujol, J. F. (1982). A radioautographic and immunocytochemical study of the GABA systems of the habenula complex in the rat. Neurochemistry international, 4(4), 303-12. https://doi.org/10.1016/0197-0186(82)90067-5
Belin, M F, et al. "A radioautographic and immunocytochemical study of the GABA systems of the habenula complex in the rat." Neurochemistry international vol. 4,4 (1982): 303-12. doi: https://doi.org/10.1016/0197-0186(82)90067-5
Belin MF, Aguera M, Nanopoulos D, Gamrani H, Maitre M, Calas A, Pujol JF. A radioautographic and immunocytochemical study of the GABA systems of the habenula complex in the rat. Neurochem Int. 1982;4(4):303-12. doi: 10.1016/0197-0186(82)90067-5. PMID: 20487881.
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Changes in activities of tryptophan hydroxylase and cyclic AMP-dependent and calcium-calmodulin-dependent protein kinases in raphe serotonergic neurons of 5,7-dihydroxytryptamine-treated rats.

Neurochemistry international

Sawada M, Kanamori T, Hayakawa T, Nagatsu T.
PMID: 20492985
Neurochem Int. 1985;7(5):761-3. doi: 10.1016/0197-0186(85)90030-0.

It was reported that tryptophan hydroxylase was activated in vitro only under calcium-calmodulin-dependent phosphorylating condition, but not under cyclic AMP-dependent phosphorylating condition. This is attributed to the absence of cyclic AMP-dependent protein kinase in serotonergic neurons. Since we had...

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Sawada M, Kanamori T, Hayakawa T, et al. Changes in activities of tryptophan hydroxylase and cyclic AMP-dependent and calcium-calmodulin-dependent protein kinases in raphe serotonergic neurons of 5,7-dihydroxytryptamine-treated rats. Neurochem Int. 1985;7(5):761-3doi: 10.1016/0197-0186(85)90030-0.
Sawada, M., Kanamori, T., Hayakawa, T., & Nagatsu, T. (1985). Changes in activities of tryptophan hydroxylase and cyclic AMP-dependent and calcium-calmodulin-dependent protein kinases in raphe serotonergic neurons of 5,7-dihydroxytryptamine-treated rats. Neurochemistry international, 7(5), 761-3. https://doi.org/10.1016/0197-0186(85)90030-0
Sawada, M, et al. "Changes in activities of tryptophan hydroxylase and cyclic AMP-dependent and calcium-calmodulin-dependent protein kinases in raphe serotonergic neurons of 5,7-dihydroxytryptamine-treated rats." Neurochemistry international vol. 7,5 (1985): 761-3. doi: https://doi.org/10.1016/0197-0186(85)90030-0
Sawada M, Kanamori T, Hayakawa T, Nagatsu T. Changes in activities of tryptophan hydroxylase and cyclic AMP-dependent and calcium-calmodulin-dependent protein kinases in raphe serotonergic neurons of 5,7-dihydroxytryptamine-treated rats. Neurochem Int. 1985;7(5):761-3. doi: 10.1016/0197-0186(85)90030-0. PMID: 20492985.
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Analysis of the Mechanism underlying the Rhythm Reversal from Diurnal to Nocturnal in the Cricket Gryllus bimaculatus, with Special Reference to the Role of Serotonin.

Zoological science

Nishinokubi I, Tomioka K.
PMID: 18522461
Zoolog Sci. 2000 Nov 01;17(8):1075-80. doi: 10.2108/zsj.17.1075.

The cricket, Gryllus bimaculatus, shows a rhythm reversal from diurnal to nocturnal in about a week after the imaginal molt. In the present study, we investigated the role of serotonin (5-HT) in the rhythm reversal. The 5-HT content in...

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Nishinokubi I, Tomioka K. Analysis of the Mechanism underlying the Rhythm Reversal from Diurnal to Nocturnal in the Cricket Gryllus bimaculatus, with Special Reference to the Role of Serotonin. Zoolog Sci. 2000;17(8):1075-80doi: 10.2108/zsj.17.1075.
Nishinokubi, I., & Tomioka, K. (2000). Analysis of the Mechanism underlying the Rhythm Reversal from Diurnal to Nocturnal in the Cricket Gryllus bimaculatus, with Special Reference to the Role of Serotonin. Zoological science, 17(8), 1075-80. https://doi.org/10.2108/zsj.17.1075
Nishinokubi, I, and Tomioka, K. "Analysis of the Mechanism underlying the Rhythm Reversal from Diurnal to Nocturnal in the Cricket Gryllus bimaculatus, with Special Reference to the Role of Serotonin." Zoological science vol. 17,8 (2000): 1075-80. doi: https://doi.org/10.2108/zsj.17.1075
Nishinokubi I, Tomioka K. Analysis of the Mechanism underlying the Rhythm Reversal from Diurnal to Nocturnal in the Cricket Gryllus bimaculatus, with Special Reference to the Role of Serotonin. Zoolog Sci. 2000 Nov 01;17(8):1075-80. doi: 10.2108/zsj.17.1075. PMID: 18522461.
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Serotonergic Innervations of the Orbitofrontal and Medial-prefrontal Cortices are Differentially Involved in Visual Discrimination and Reversal Learning in Rats.

Cerebral cortex (New York, N.Y. : 1991)

Alsiö J, Lehmann O, McKenzie C, Theobald DE, Searle L, Xia J, Dalley JW, Robbins TW.
PMID: 33043981
Cereb Cortex. 2021 Jan 05;31(2):1090-1105. doi: 10.1093/cercor/bhaa277.

Cross-species studies have identified an evolutionarily conserved role for serotonin in flexible behavior including reversal learning. The aim of the current study was to investigate the contribution of serotonin within the orbitofrontal cortex (OFC) and medial prefrontal cortex (mPFC)...

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Alsiö J, Lehmann O, McKenzie C, et al. Serotonergic Innervations of the Orbitofrontal and Medial-prefrontal Cortices are Differentially Involved in Visual Discrimination and Reversal Learning in Rats. Cereb Cortex. 2021;31(2):1090-1105doi: 10.1093/cercor/bhaa277.
Alsiö, J., Lehmann, O., McKenzie, C., Theobald, D. E., Searle, L., Xia, J., Dalley, J. W., & Robbins, T. W. (2021). Serotonergic Innervations of the Orbitofrontal and Medial-prefrontal Cortices are Differentially Involved in Visual Discrimination and Reversal Learning in Rats. Cerebral cortex (New York, N.Y. : 1991), 31(2), 1090-1105. https://doi.org/10.1093/cercor/bhaa277
Alsiö, Johan, et al. "Serotonergic Innervations of the Orbitofrontal and Medial-prefrontal Cortices are Differentially Involved in Visual Discrimination and Reversal Learning in Rats." Cerebral cortex (New York, N.Y. : 1991) vol. 31,2 (2021): 1090-1105. doi: https://doi.org/10.1093/cercor/bhaa277
Alsiö J, Lehmann O, McKenzie C, Theobald DE, Searle L, Xia J, Dalley JW, Robbins TW. Serotonergic Innervations of the Orbitofrontal and Medial-prefrontal Cortices are Differentially Involved in Visual Discrimination and Reversal Learning in Rats. Cereb Cortex. 2021 Jan 05;31(2):1090-1105. doi: 10.1093/cercor/bhaa277. PMID: 33043981; PMCID: PMC7906782.
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Effects of ipsapirone, a 5-HT1A agonist, on sleep/wakefulness cycles: probable post-synaptic action.

Journal of sleep research

Tissier MH, Lainey E, Fattaccini CM, Hamon M, Adrien J.
PMID: 10607079
J Sleep Res. 1993 Jun;2(2):103-109. doi: 10.1111/j.1365-2869.1993.tb00070.x.

The effects of ipsapirone, a ligand of the 5-HT1A receptors and a new potential anxiolytic, on sleep/wakefulness regulation were examined in the rat. Injected i.p. at 1, 3 and 5 mg kg-1, this compound induced a dose-dependent reduction of...

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Tissier MH, Lainey E, Fattaccini CM, et al. Effects of ipsapirone, a 5-HT1A agonist, on sleep/wakefulness cycles: probable post-synaptic action. J Sleep Res. 1993;2(2):103-109doi: 10.1111/j.1365-2869.1993.tb00070.x.
Tissier, M. H., Lainey, E., Fattaccini, C. M., Hamon, M., & Adrien, J. (1993). Effects of ipsapirone, a 5-HT1A agonist, on sleep/wakefulness cycles: probable post-synaptic action. Journal of sleep research, 2(2), 103-109. https://doi.org/10.1111/j.1365-2869.1993.tb00070.x
Tissier, et al. "Effects of ipsapirone, a 5-HT1A agonist, on sleep/wakefulness cycles: probable post-synaptic action." Journal of sleep research vol. 2,2 (1993): 103-109. doi: https://doi.org/10.1111/j.1365-2869.1993.tb00070.x
Tissier MH, Lainey E, Fattaccini CM, Hamon M, Adrien J. Effects of ipsapirone, a 5-HT1A agonist, on sleep/wakefulness cycles: probable post-synaptic action. J Sleep Res. 1993 Jun;2(2):103-109. doi: 10.1111/j.1365-2869.1993.tb00070.x. PMID: 10607079.
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Central 5,7-dihydroxytryptamine lesions decrease hippocampal glucocorticoid and mineralocorticoid receptor messenger ribonucleic Acid expression.

Journal of neuroendocrinology

Seckl JR, Dickson KL, Fink G.
PMID: 19215437
J Neuroendocrinol. 1990 Dec 01;2(6):911-6. doi: 10.1111/j.1365-2826.1990.tb00659.x.

Abstract Corticosteroids exert effects on the hippocampus by binding to intracellular glucocorticoid and/or mineralocorticoid receptors, but the relative importance of each receptor type in mediating corticosteroid effects is poorly understood. There is an extensive serotoninergic (5-HT) innervation of the...

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Seckl JR, Dickson KL, Fink G. Central 5,7-dihydroxytryptamine lesions decrease hippocampal glucocorticoid and mineralocorticoid receptor messenger ribonucleic Acid expression. J Neuroendocrinol. 1990;2(6):911-6doi: 10.1111/j.1365-2826.1990.tb00659.x.
Seckl, J. R., Dickson, K. L., & Fink, G. (1990). Central 5,7-dihydroxytryptamine lesions decrease hippocampal glucocorticoid and mineralocorticoid receptor messenger ribonucleic Acid expression. Journal of neuroendocrinology, 2(6), 911-6. https://doi.org/10.1111/j.1365-2826.1990.tb00659.x
Seckl, J R, et al. "Central 5,7-dihydroxytryptamine lesions decrease hippocampal glucocorticoid and mineralocorticoid receptor messenger ribonucleic Acid expression." Journal of neuroendocrinology vol. 2,6 (1990): 911-6. doi: https://doi.org/10.1111/j.1365-2826.1990.tb00659.x
Seckl JR, Dickson KL, Fink G. Central 5,7-dihydroxytryptamine lesions decrease hippocampal glucocorticoid and mineralocorticoid receptor messenger ribonucleic Acid expression. J Neuroendocrinol. 1990 Dec 01;2(6):911-6. doi: 10.1111/j.1365-2826.1990.tb00659.x. PMID: 19215437.
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Are 5-HT receptors involved in the sprouting of serotoninergic terminals following neonatal 5,7-dihydroxytryptamine treatment in the rat?.

Neurochemistry international

Hamon M, Nelson DL, Mallat M, Bourgoin S.
PMID: 20487810
Neurochem Int. 1981 Mar;3(1):69-79. doi: 10.1016/0197-0186(81)90051-6.

The neonatal administration of 5,7-dihydroxytryptamine to rats (100 mg kg(?1) s.c. on the 1st and 2nd day after birth) resulted in marked reductions in serotoninergic presynaptic markers ([(3)H]-5-HT synaptosomal uptake, tryptophan hydroxylase activity and endogenous 5-HT content) in various...

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Hamon M, Nelson DL, Mallat M, et al. Are 5-HT receptors involved in the sprouting of serotoninergic terminals following neonatal 5,7-dihydroxytryptamine treatment in the rat?. Neurochem Int. 1981;3(1):69-79doi: 10.1016/0197-0186(81)90051-6.
Hamon, M., Nelson, D. L., Mallat, M., & Bourgoin, S. (1981). Are 5-HT receptors involved in the sprouting of serotoninergic terminals following neonatal 5,7-dihydroxytryptamine treatment in the rat?. Neurochemistry international, 3(1), 69-79. https://doi.org/10.1016/0197-0186(81)90051-6
Hamon, M, et al. "Are 5-HT receptors involved in the sprouting of serotoninergic terminals following neonatal 5,7-dihydroxytryptamine treatment in the rat?." Neurochemistry international vol. 3,1 (1981): 69-79. doi: https://doi.org/10.1016/0197-0186(81)90051-6
Hamon M, Nelson DL, Mallat M, Bourgoin S. Are 5-HT receptors involved in the sprouting of serotoninergic terminals following neonatal 5,7-dihydroxytryptamine treatment in the rat?. Neurochem Int. 1981 Mar;3(1):69-79. doi: 10.1016/0197-0186(81)90051-6. PMID: 20487810.
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