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Impulsive delayed reward discounting as a genetically-influenced target for drug abuse prevention: a critical evaluation.

Frontiers in psychology

Gray JC, MacKillop J.
PMID: 26388788
Front Psychol. 2015 Sep 01;6:1104. doi: 10.3389/fpsyg.2015.01104. eCollection 2015.

This review evaluates the viability of delayed reward discounting (DRD), an index of how much an individual devalues a future reward based on its delay in time, for genetically-informed drug abuse prevention. A review of the literature suggests that...

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Gray JC, MacKillop J. Impulsive delayed reward discounting as a genetically-influenced target for drug abuse prevention: a critical evaluation. Front Psychol. 2015;6:1104doi: 10.3389/fpsyg.2015.01104.
Gray, J. C., & MacKillop, J. (2015). Impulsive delayed reward discounting as a genetically-influenced target for drug abuse prevention: a critical evaluation. Frontiers in psychology, 61104. https://doi.org/10.3389/fpsyg.2015.01104
Gray, Joshua C, and MacKillop, James. "Impulsive delayed reward discounting as a genetically-influenced target for drug abuse prevention: a critical evaluation." Frontiers in psychology vol. 6 (2015): 1104. doi: https://doi.org/10.3389/fpsyg.2015.01104
Gray JC, MacKillop J. Impulsive delayed reward discounting as a genetically-influenced target for drug abuse prevention: a critical evaluation. Front Psychol. 2015 Sep 01;6:1104. doi: 10.3389/fpsyg.2015.01104. eCollection 2015. PMID: 26388788; PMCID: PMC4554956.
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Historical Overview of the Effect of β-Adrenergic Agonists on Beef Cattle Production.

Asian-Australasian journal of animal sciences

Johnson BJ, Smith SB, Chung KY.
PMID: 25050012
Asian-Australas J Anim Sci. 2014 May;27(5):757-66. doi: 10.5713/ajas.2012.12524.

Postnatal muscle hypertrophy of beef cattle is the result of enhanced myofibrillar protein synthesis and reduced protein turnover. Skeletal muscle hypertrophy has been studied in cattle fed β-adrenergic agonists (β-AA), which are receptor-mediated enhancers of protein synthesis and inhibitors...

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Johnson BJ, Smith SB, Chung KY. Historical Overview of the Effect of β-Adrenergic Agonists on Beef Cattle Production. Asian-Australas J Anim Sci. 2014;27(5):757-66doi: 10.5713/ajas.2012.12524.
Johnson, B. J., Smith, S. B., & Chung, K. Y. (2014). Historical Overview of the Effect of β-Adrenergic Agonists on Beef Cattle Production. Asian-Australasian journal of animal sciences, 27(5), 757-66. https://doi.org/10.5713/ajas.2012.12524
Johnson, Bradley J, et al. "Historical Overview of the Effect of β-Adrenergic Agonists on Beef Cattle Production." Asian-Australasian journal of animal sciences vol. 27,5 (2014): 757-66. doi: https://doi.org/10.5713/ajas.2012.12524
Johnson BJ, Smith SB, Chung KY. Historical Overview of the Effect of β-Adrenergic Agonists on Beef Cattle Production. Asian-Australas J Anim Sci. 2014 May;27(5):757-66. doi: 10.5713/ajas.2012.12524. PMID: 25050012; PMCID: PMC4093185.
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Ultra-rapid, antagonist-precipitated opiate detoxification under general anaesthesia or sedation.

Addiction biology

Brewer C.
PMID: 26735785
Addict Biol. 1997 Jul;2(3):291-302. doi: 10.1080/13556219772589.

Ultra-rapid opiate withdrawal precipitated by naloxone or naltrexone during general anaesthesia or varying degrees of sedation, usually with the addition of α-2 adrenergic agonists, is an increasingly popular technique. As with anaesthesia or effective analgesia for dentistry or obstetrics,...

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Brewer C. Ultra-rapid, antagonist-precipitated opiate detoxification under general anaesthesia or sedation. Addict Biol. 1997;2(3):291-302doi: 10.1080/13556219772589.
Brewer, C. (1997). Ultra-rapid, antagonist-precipitated opiate detoxification under general anaesthesia or sedation. Addiction biology, 2(3), 291-302. https://doi.org/10.1080/13556219772589
Brewer, C. "Ultra-rapid, antagonist-precipitated opiate detoxification under general anaesthesia or sedation." Addiction biology vol. 2,3 (1997): 291-302. doi: https://doi.org/10.1080/13556219772589
Brewer C. Ultra-rapid, antagonist-precipitated opiate detoxification under general anaesthesia or sedation. Addict Biol. 1997 Jul;2(3):291-302. doi: 10.1080/13556219772589. PMID: 26735785.
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Catecholamines induce metamorphosis in the hydrozoan Halocordyle disticha but not in Hydractinia echinata.

Roux's archives of developmental biology : the official organ of the EDBO

Edwards NC, Thomas MB, Long BA, Amyotte SJ.
PMID: 28305639
Rouxs Arch Dev Biol. 1987 Sep;196(6):381-384. doi: 10.1007/BF00375776.

Planula larvae of the marine hydroids Halocordyle disticha and Hydractinia echinata were treated with the catecholamines epinephrine, norepinephrine and dopamine, as well as with certain of their precursors and agonists. Norepinephrine, L-dopa, dopamine and the dopamine agonist ADTN at...

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Edwards NC, Thomas MB, Long BA, et al. Catecholamines induce metamorphosis in the hydrozoan Halocordyle disticha but not in Hydractinia echinata. Rouxs Arch Dev Biol. 1987;196(6):381-384doi: 10.1007/BF00375776.
Edwards, N. C., Thomas, M. B., Long, B. A., & Amyotte, S. J. (1987). Catecholamines induce metamorphosis in the hydrozoan Halocordyle disticha but not in Hydractinia echinata. Roux's archives of developmental biology : the official organ of the EDBO, 196(6), 381-384. https://doi.org/10.1007/BF00375776
Edwards, Nancy Claire, et al. "Catecholamines induce metamorphosis in the hydrozoan Halocordyle disticha but not in Hydractinia echinata." Roux's archives of developmental biology : the official organ of the EDBO vol. 196,6 (1987): 381-384. doi: https://doi.org/10.1007/BF00375776
Edwards NC, Thomas MB, Long BA, Amyotte SJ. Catecholamines induce metamorphosis in the hydrozoan Halocordyle disticha but not in Hydractinia echinata. Rouxs Arch Dev Biol. 1987 Sep;196(6):381-384. doi: 10.1007/BF00375776. PMID: 28305639.
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A Review of Pharmacological Management of Attention-Deficit/Hyperactivity Disorder.

The journal of pediatric pharmacology and therapeutics : JPPT : the official journal of PPAG

Briars L, Todd T.
PMID: 27453697
J Pediatr Pharmacol Ther. 2016 May-Jun;21(3):192-206. doi: 10.5863/1551-6776-21.3.192.

Attention-deficit/hyperactivity disorder (ADHD) is a common psychological diagnosis in children. This disorder impacts children and adolescents in all areas of life, including academic performance, extracurricular activities, and social interactions. ADHD can continue into adulthood where unemployment and substance abuse...

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Briars L, Todd T. A Review of Pharmacological Management of Attention-Deficit/Hyperactivity Disorder. J Pediatr Pharmacol Ther. 2016;21(3):192-206doi: 10.5863/1551-6776-21.3.192.
Briars, L., & Todd, T. (2016). A Review of Pharmacological Management of Attention-Deficit/Hyperactivity Disorder. The journal of pediatric pharmacology and therapeutics : JPPT : the official journal of PPAG, 21(3), 192-206. https://doi.org/10.5863/1551-6776-21.3.192
Briars, Leslie, and Todd, Timothy. "A Review of Pharmacological Management of Attention-Deficit/Hyperactivity Disorder." The journal of pediatric pharmacology and therapeutics : JPPT : the official journal of PPAG vol. 21,3 (2016): 192-206. doi: https://doi.org/10.5863/1551-6776-21.3.192
Briars L, Todd T. A Review of Pharmacological Management of Attention-Deficit/Hyperactivity Disorder. J Pediatr Pharmacol Ther. 2016 May-Jun;21(3):192-206. doi: 10.5863/1551-6776-21.3.192. PMID: 27453697; PMCID: PMC4956327.
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Vasogenic shock physiology.

Open access emergency medicine : OAEM

Gkisioti S, Mentzelopoulos SD.
PMID: 27147845
Open Access Emerg Med. 2011 Jan 06;3:1-6. doi: 10.2147/OAEM.S10388. eCollection 2011.

Shock means inadequate tissue perfusion by oxygen-carrying blood. In vasogenic shock, this circulatory failure results from vasodilation and/or vasoplegia. There is vascular hyporeactivity with reduced vascular smooth muscle contraction in response to α1 adrenergic agonists. Considering vasogenic shock, one...

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Gkisioti S, Mentzelopoulos SD. Vasogenic shock physiology. Open Access Emerg Med. 2011;3:1-6doi: 10.2147/OAEM.S10388.
Gkisioti, S., & Mentzelopoulos, S. D. (2011). Vasogenic shock physiology. Open access emergency medicine : OAEM, 31-6. https://doi.org/10.2147/OAEM.S10388
Gkisioti, Sotiria, and Mentzelopoulos, Spyros D. "Vasogenic shock physiology." Open access emergency medicine : OAEM vol. 3 (2011): 1-6. doi: https://doi.org/10.2147/OAEM.S10388
Gkisioti S, Mentzelopoulos SD. Vasogenic shock physiology. Open Access Emerg Med. 2011 Jan 06;3:1-6. doi: 10.2147/OAEM.S10388. eCollection 2011. PMID: 27147845; PMCID: PMC4753960.
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Rationale for the short term use of intravenous milrinone under hemodynamic guidance in patients with severe systolic heart failure.

Congestive heart failure (Greenwich, Conn.)

Quigg RJ.
PMID: 12147954
Congest Heart Fail. 2000 Jul-Aug;6(4):202-214. doi: 10.1111/j.1527-5299.2000.80160.x.

Patients with severe systolic heart failure have a decrease in both number and function of cardiac beta receptors, which may result in a poor inotropic response to I.V. beta-adrenergic agonists such as dobutamine. The I.V. use of the phosphodiesterase...

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Quigg RJ. Rationale for the short term use of intravenous milrinone under hemodynamic guidance in patients with severe systolic heart failure. Congest Heart Fail. 2000;6(4):202-214doi: 10.1111/j.1527-5299.2000.80160.x.
Quigg, R. J. (2000). Rationale for the short term use of intravenous milrinone under hemodynamic guidance in patients with severe systolic heart failure. Congestive heart failure (Greenwich, Conn.), 6(4), 202-214. https://doi.org/10.1111/j.1527-5299.2000.80160.x
Quigg, R J. "Rationale for the short term use of intravenous milrinone under hemodynamic guidance in patients with severe systolic heart failure." Congestive heart failure (Greenwich, Conn.) vol. 6,4 (2000): 202-214. doi: https://doi.org/10.1111/j.1527-5299.2000.80160.x
Quigg RJ. Rationale for the short term use of intravenous milrinone under hemodynamic guidance in patients with severe systolic heart failure. Congest Heart Fail. 2000 Jul-Aug;6(4):202-214. doi: 10.1111/j.1527-5299.2000.80160.x. PMID: 12147954.
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Physiological Mechanisms of Pheromonostatic Responses: effects of Adrenergic Agonists and Antagonists on Moth (Helicoverpa armigera) Pheromone Biosynthesis.

Journal of insect physiology

CAO M, YONGLIANG F, GILEADI C, RAFAELI A.
PMID: 12769910
J Insect Physiol. 1997 Mar;43(3):261-269. doi: 10.1016/s0022-1910(96)00088-1.

The adrenergic agonists octopamine, tyramine and clonidine inhibited the normal pheromonotropic action due to PBAN (pheromone biosynthesis activating neuropeptide) in incubations of intersegmental tissues that are situated between the 8th and 9th abdominal segments of the moth ovipositor tip....

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CAO M, YONGLIANG F, GILEADI C, et al. Physiological Mechanisms of Pheromonostatic Responses: effects of Adrenergic Agonists and Antagonists on Moth (Helicoverpa armigera) Pheromone Biosynthesis. J Insect Physiol. 1997;43(3):261-269doi: 10.1016/s0022-1910(96)00088-1.
CAO, M., YONGLIANG, F., GILEADI, C., & RAFAELI, A. (1997). Physiological Mechanisms of Pheromonostatic Responses: effects of Adrenergic Agonists and Antagonists on Moth (Helicoverpa armigera) Pheromone Biosynthesis. Journal of insect physiology, 43(3), 261-269. https://doi.org/10.1016/s0022-1910(96)00088-1
CAO, M, et al. "Physiological Mechanisms of Pheromonostatic Responses: effects of Adrenergic Agonists and Antagonists on Moth (Helicoverpa armigera) Pheromone Biosynthesis." Journal of insect physiology vol. 43,3 (1997): 261-269. doi: https://doi.org/10.1016/s0022-1910(96)00088-1
CAO M, YONGLIANG F, GILEADI C, RAFAELI A. Physiological Mechanisms of Pheromonostatic Responses: effects of Adrenergic Agonists and Antagonists on Moth (Helicoverpa armigera) Pheromone Biosynthesis. J Insect Physiol. 1997 Mar;43(3):261-269. doi: 10.1016/s0022-1910(96)00088-1. PMID: 12769910.
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Termination of ventricular tachycardia after methoxamine.

Critical care and resuscitation : journal of the Australasian Academy of Critical Care Medicine

Brown J.
PMID: 16573515
Crit Care Resusc. 2001 Dec;3(4):259-61.

A case of ventricular tachycardia which reverted with intravenous methoxamine is presented. The methoxamine was used to increase blood pressure during anaesthesia for direct current cardioversion. While alpha adrenergic agonists have been used previously to increase vagal tone via...

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Brown J. Termination of ventricular tachycardia after methoxamine. Crit Care Resusc. 2001;3(4):259-61
Brown, J. (2001). Termination of ventricular tachycardia after methoxamine. Critical care and resuscitation : journal of the Australasian Academy of Critical Care Medicine, 3(4), 259-61.
Brown, J. "Termination of ventricular tachycardia after methoxamine." Critical care and resuscitation : journal of the Australasian Academy of Critical Care Medicine vol. 3,4 (2001): 259-61.
Brown J. Termination of ventricular tachycardia after methoxamine. Crit Care Resusc. 2001 Dec;3(4):259-61. PMID: 16573515.
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Immunological manipulation of body composition.

Expert opinion on investigational drugs

Beattie J, Flint DJ.
PMID: 15992055
Expert Opin Investig Drugs. 1999 Jan;8(1):19-27. doi: 10.1517/13543784.8.1.19.

Hormonal growth promoters (e.g., growth hormone [GH], beta-adrenergic agonists, steroids), which improve growth rate and/or lean:fat ratios in the carcass, have received considerable adverse publicity and are either banned or have no licence for their use in the EC....

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Beattie J, Flint DJ. Immunological manipulation of body composition. Expert Opin Investig Drugs. 1999;8(1):19-27doi: 10.1517/13543784.8.1.19.
Beattie, J., & Flint, D. J. (1999). Immunological manipulation of body composition. Expert opinion on investigational drugs, 8(1), 19-27. https://doi.org/10.1517/13543784.8.1.19
Beattie, J, and Flint, D J. "Immunological manipulation of body composition." Expert opinion on investigational drugs vol. 8,1 (1999): 19-27. doi: https://doi.org/10.1517/13543784.8.1.19
Beattie J, Flint DJ. Immunological manipulation of body composition. Expert Opin Investig Drugs. 1999 Jan;8(1):19-27. doi: 10.1517/13543784.8.1.19. PMID: 15992055.
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Side Effects of alpha-Adrenergic Agonists.

Journal of glaucoma

Morrison JC.
PMID: 19920646
J Glaucoma. 1995 Feb;4:S36. doi: 10.1097/00061198-199502001-00007.

No abstract available.

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Morrison JC. Side Effects of alpha-Adrenergic Agonists. J Glaucoma. 1995;4:S36doi: 10.1097/00061198-199502001-00007.
Morrison, J. C. (1995). Side Effects of alpha-Adrenergic Agonists. Journal of glaucoma, 4S36. https://doi.org/10.1097/00061198-199502001-00007
Morrison, J C. "Side Effects of alpha-Adrenergic Agonists." Journal of glaucoma vol. 4 (1995): S36. doi: https://doi.org/10.1097/00061198-199502001-00007
Morrison JC. Side Effects of alpha-Adrenergic Agonists. J Glaucoma. 1995 Feb;4:S36. doi: 10.1097/00061198-199502001-00007. PMID: 19920646.
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[Dexmedetomidine in anesthesiology.].

Revista brasileira de anestesiologia

Villela NR, Nascimento Júnior Pd.
PMID: 19475263
Rev Bras Anestesiol. 2003 Feb;53(1):97-113. doi: 10.1590/s0034-70942003000100013.

BACKGROUND AND OBJECTIVES: Dexmedetomidine is a new alpha2-adrenergic agonist with a selectivity ratio of 1600:1 (alpha2:alpha1). It has important sedative and analgesic effects, good hemodynamic control at stress situations and may, by itself, induce anesthesia. This drug has been...

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Villela NR, Nascimento Júnior Pd. Uso de dexmedetomidina em anestesiologia. [Dexmedetomidine in anesthesiology.]. Rev Bras Anestesiol. 2003;53(1):97-113doi: 10.1590/s0034-70942003000100013.
Villela, N. R., & Nascimento Júnior, P. d. (2003). Uso de dexmedetomidina em anestesiologia. [Dexmedetomidine in anesthesiology.]. Revista brasileira de anestesiologia, 53(1), 97-113. https://doi.org/10.1590/s0034-70942003000100013
Villela, Nivaldo Ribeiro, and Nascimento Júnior, Paulo do. "Uso de dexmedetomidina em anestesiologia." [Dexmedetomidine in anesthesiology.]. Revista brasileira de anestesiologia vol. 53,1 (2003): 97-113. doi: https://doi.org/10.1590/s0034-70942003000100013
Villela NR, Nascimento Júnior Pd. Uso de dexmedetomidina em anestesiologia. [Dexmedetomidine in anesthesiology.]. Rev Bras Anestesiol. 2003 Feb;53(1):97-113. doi: 10.1590/s0034-70942003000100013. Portuguese. PMID: 19475263.
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Showing 1 to 12 of 173 entries