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Showing 1 to 12 of 16 entries
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In Vivo Hypocholesterolemic Effect of MARDI Fermented Red Yeast Rice Water Extract in High Cholesterol Diet Fed Mice.

Evidence-based complementary and alternative medicine : eCAM

Yeap SK, Beh BK, Kong J, Ho WY, Mohd Yusof H, Mohamad NE, Hussin AB, Jaganath IB, Alitheen NB, Jamaluddin A, Long K.
PMID: 25031606
Evid Based Complement Alternat Med. 2014;2014:707829. doi: 10.1155/2014/707829. Epub 2014 Jun 16.

Fermented red yeast rice has been traditionally consumed as medication in Asian cuisine. This study aimed to determine the in vivo hypocholesterolemic and antioxidant effects of fermented red yeast rice water extract produced using Malaysian Agricultural Research and Development...

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Yeap SK, Beh BK, Kong J, et al. In Vivo Hypocholesterolemic Effect of MARDI Fermented Red Yeast Rice Water Extract in High Cholesterol Diet Fed Mice. Evid Based Complement Alternat Med. 2014;2014:707829doi: 10.1155/2014/707829.
Yeap, S. K., Beh, B. K., Kong, J., Ho, W. Y., Mohd Yusof, H., Mohamad, N. E., Hussin, A. B., Jaganath, I. B., Alitheen, N. B., Jamaluddin, A., & Long, K. (2014). In Vivo Hypocholesterolemic Effect of MARDI Fermented Red Yeast Rice Water Extract in High Cholesterol Diet Fed Mice. Evidence-based complementary and alternative medicine : eCAM, 2014707829. https://doi.org/10.1155/2014/707829
Yeap, Swee Keong, et al. "In Vivo Hypocholesterolemic Effect of MARDI Fermented Red Yeast Rice Water Extract in High Cholesterol Diet Fed Mice." Evidence-based complementary and alternative medicine : eCAM vol. 2014 (2014): 707829. doi: https://doi.org/10.1155/2014/707829
Yeap SK, Beh BK, Kong J, Ho WY, Mohd Yusof H, Mohamad NE, Hussin AB, Jaganath IB, Alitheen NB, Jamaluddin A, Long K. In Vivo Hypocholesterolemic Effect of MARDI Fermented Red Yeast Rice Water Extract in High Cholesterol Diet Fed Mice. Evid Based Complement Alternat Med. 2014;2014:707829. doi: 10.1155/2014/707829. Epub 2014 Jun 16. PMID: 25031606; PMCID: PMC4083602.
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The Effects of PPAR Agonists on Atherosclerosis and Nonalcoholic Fatty Liver Disease in ApoE-/-FXR-/- Mice.

Endocrinology and metabolism (Seoul, Korea)

Lee Y, Kim BR, Kang GH, Lee GJ, Park YJ, Kim H, Jang HC, Choi SH.
PMID: 34986301
Endocrinol Metab (Seoul). 2021 Dec;36(6):1243-1253. doi: 10.3803/EnM.2021.1100. Epub 2021 Dec 28.

BACKGROUND: Farnesoid X receptor (FXR), a bile acid-activated nuclear receptor, is a potent regulator of glucose and lipid metabolism as well as of bile acid metabolism. Previous studies have demonstrated that FXR deficiency is associated with metabolic derangements, including...

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Lee Y, Kim BR, Kang GH, et al. The Effects of PPAR Agonists on Atherosclerosis and Nonalcoholic Fatty Liver Disease in ApoE-/-FXR-/- Mice. Endocrinol Metab (Seoul). 2021;36(6):1243-1253doi: 10.3803/EnM.2021.1100.
Lee, Y., Kim, B. R., Kang, G. H., Lee, G. J., Park, Y. J., Kim, H., Jang, H. C., & Choi, S. H. (2021). The Effects of PPAR Agonists on Atherosclerosis and Nonalcoholic Fatty Liver Disease in ApoE-/-FXR-/- Mice. Endocrinology and metabolism (Seoul, Korea), 36(6), 1243-1253. https://doi.org/10.3803/EnM.2021.1100
Lee, Yenna, et al. "The Effects of PPAR Agonists on Atherosclerosis and Nonalcoholic Fatty Liver Disease in ApoE-/-FXR-/- Mice." Endocrinology and metabolism (Seoul, Korea) vol. 36,6 (2021): 1243-1253. doi: https://doi.org/10.3803/EnM.2021.1100
Lee Y, Kim BR, Kang GH, Lee GJ, Park YJ, Kim H, Jang HC, Choi SH. The Effects of PPAR Agonists on Atherosclerosis and Nonalcoholic Fatty Liver Disease in ApoE-/-FXR-/- Mice. Endocrinol Metab (Seoul). 2021 Dec;36(6):1243-1253. doi: 10.3803/EnM.2021.1100. Epub 2021 Dec 28. PMID: 34986301; PMCID: PMC8743579.
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Women's brain aging: Effects of sex-hormone exposure, pregnancies, and genetic risk for Alzheimer's disease.

Human brain mapping

de Lange AG, Barth C, Kaufmann T, Maximov II, van der Meer D, Agartz I, Westlye LT.
PMID: 32856754
Hum Brain Mapp. 2020 Dec 15;41(18):5141-5150. doi: 10.1002/hbm.25180. Epub 2020 Aug 28.

Sex hormones such as estrogen fluctuate across the female lifespan, with high levels during reproductive years and natural decline during the transition to menopause. Women's exposure to estrogen may influence their heightened risk of Alzheimer's disease (AD) relative to...

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de Lange AG, Barth C, Kaufmann T, et al. Women's brain aging: Effects of sex-hormone exposure, pregnancies, and genetic risk for Alzheimer's disease. Hum Brain Mapp. 2020;41(18):5141-5150doi: 10.1002/hbm.25180.
de Lange, A. G., Barth, C., Kaufmann, T., Maximov, I. I., van der Meer, D., Agartz, I., & Westlye, L. T. (2020). Women's brain aging: Effects of sex-hormone exposure, pregnancies, and genetic risk for Alzheimer's disease. Human brain mapping, 41(18), 5141-5150. https://doi.org/10.1002/hbm.25180
de Lange, Ann-Marie G, et al. "Women's brain aging: Effects of sex-hormone exposure, pregnancies, and genetic risk for Alzheimer's disease." Human brain mapping vol. 41,18 (2020): 5141-5150. doi: https://doi.org/10.1002/hbm.25180
de Lange AG, Barth C, Kaufmann T, Maximov II, van der Meer D, Agartz I, Westlye LT. Women's brain aging: Effects of sex-hormone exposure, pregnancies, and genetic risk for Alzheimer's disease. Hum Brain Mapp. 2020 Dec 15;41(18):5141-5150. doi: 10.1002/hbm.25180. Epub 2020 Aug 28. PMID: 32856754; PMCID: PMC7670641.
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Pathobiolgy and Management of Alzheimer's Disease.

Chonnam medical journal

Kim H, Chung JY.
PMID: 34123738
Chonnam Med J. 2021 May;57(2):108-117. doi: 10.4068/cmj.2021.57.2.108. Epub 2021 May 24.

Amyloid and tau protein abnormalities have been identified as the main causes of Alzheimer's disease but exact mechanisms remain to be revealed. Especially, amyloid beta and tau protein coupling and neuroinflammatory and neurovascular contributions to Alzheimer disease are quite...

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Kim H, Chung JY. Pathobiolgy and Management of Alzheimer's Disease. Chonnam Med J. 2021;57(2):108-117doi: 10.4068/cmj.2021.57.2.108.
Kim, H., & Chung, J. Y. (2021). Pathobiolgy and Management of Alzheimer's Disease. Chonnam medical journal, 57(2), 108-117. https://doi.org/10.4068/cmj.2021.57.2.108
Kim, Hoowon, and Chung, Ji Yeon. "Pathobiolgy and Management of Alzheimer's Disease." Chonnam medical journal vol. 57,2 (2021): 108-117. doi: https://doi.org/10.4068/cmj.2021.57.2.108
Kim H, Chung JY. Pathobiolgy and Management of Alzheimer's Disease. Chonnam Med J. 2021 May;57(2):108-117. doi: 10.4068/cmj.2021.57.2.108. Epub 2021 May 24. PMID: 34123738; PMCID: PMC8167446.
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Effect of physical exercise on cognitive function in older adults' carriers versus noncarriers of apolipoprotein E4: systematic review and meta-analysis.

Journal of exercise rehabilitation

Cancela-Carral JM, López-Rodríguez A, Mollinedo-Cardalda I.
PMID: 34012932
J Exerc Rehabil. 2021 Apr 27;17(2):69-80. doi: 10.12965/jer.2142130.065. eCollection 2021 Apr.

The presence of apolipoprotein (Apo) E4 is a genetic risk factor in cognitive impairment. Physical exercise contributes to slowing cognitive impairment in older adults, but little is known about the influence of exercise on ApoE4 carriers and noncarriers. The...

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Cancela-Carral JM, López-Rodríguez A, Mollinedo-Cardalda I. Effect of physical exercise on cognitive function in older adults' carriers versus noncarriers of apolipoprotein E4: systematic review and meta-analysis. J Exerc Rehabil. 2021;17(2):69-80doi: 10.12965/jer.2142130.065.
Cancela-Carral, J. M., López-Rodríguez, A., & Mollinedo-Cardalda, I. (2021). Effect of physical exercise on cognitive function in older adults' carriers versus noncarriers of apolipoprotein E4: systematic review and meta-analysis. Journal of exercise rehabilitation, 17(2), 69-80. https://doi.org/10.12965/jer.2142130.065
Cancela-Carral, José María, et al. "Effect of physical exercise on cognitive function in older adults' carriers versus noncarriers of apolipoprotein E4: systematic review and meta-analysis." Journal of exercise rehabilitation vol. 17,2 (2021): 69-80. doi: https://doi.org/10.12965/jer.2142130.065
Cancela-Carral JM, López-Rodríguez A, Mollinedo-Cardalda I. Effect of physical exercise on cognitive function in older adults' carriers versus noncarriers of apolipoprotein E4: systematic review and meta-analysis. J Exerc Rehabil. 2021 Apr 27;17(2):69-80. doi: 10.12965/jer.2142130.065. eCollection 2021 Apr. PMID: 34012932; PMCID: PMC8103191.
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Association of apolipoproteins C-I, C-II, C-III and E with coagulation markers and venous thromboembolism risk.

Clinical epidemiology

Orsi FA, Lijfering WM, Van der Laarse A, Ruhaak LR, Rosendaal FR, Cannegieter SC, Cobbaert C.
PMID: 31413640
Clin Epidemiol. 2019 Jul 22;11:625-633. doi: 10.2147/CLEP.S196266. eCollection 2019.

PURPOSE: Apolipoproteins C-I, C-II, C-III and E have been associated with risk of arterial thrombotic diseases. We investigated whether these apolipoproteins have prothrombotic properties and are associated with risk of venous thromboembolism (VTE).PATIENTS AND METHODS: A total of 127...

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Orsi FA, Lijfering WM, Van der Laarse A, et al. Association of apolipoproteins C-I, C-II, C-III and E with coagulation markers and venous thromboembolism risk. Clin Epidemiol. 2019;11:625-633doi: 10.2147/CLEP.S196266.
Orsi, F. A., Lijfering, W. M., Van der Laarse, A., Ruhaak, L. R., Rosendaal, F. R., Cannegieter, S. C., & Cobbaert, C. (2019). Association of apolipoproteins C-I, C-II, C-III and E with coagulation markers and venous thromboembolism risk. Clinical epidemiology, 11625-633. https://doi.org/10.2147/CLEP.S196266
Orsi, Fernanda A, et al. "Association of apolipoproteins C-I, C-II, C-III and E with coagulation markers and venous thromboembolism risk." Clinical epidemiology vol. 11 (2019): 625-633. doi: https://doi.org/10.2147/CLEP.S196266
Orsi FA, Lijfering WM, Van der Laarse A, Ruhaak LR, Rosendaal FR, Cannegieter SC, Cobbaert C. Association of apolipoproteins C-I, C-II, C-III and E with coagulation markers and venous thromboembolism risk. Clin Epidemiol. 2019 Jul 22;11:625-633. doi: 10.2147/CLEP.S196266. eCollection 2019. PMID: 31413640; PMCID: PMC6659780.
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APOE E2/E2 Is Associated with Slower Rate of Cognitive Decline with Age.

Journal of Alzheimer's disease : JAD

Sweigart B, Andersen SL, Gurinovich A, Cosentino S, Schupf N, Perls TT, Sebastiani P.
PMID: 34366332
J Alzheimers Dis. 2021;83(2):853-860. doi: 10.3233/JAD-201205.

BACKGROUND: The E4 allele of the APOE gene is known to be associated with cognitive impairment. However, a limited number of studies have examined the association between the E2 allele and longitudinal changes of cognitive function.OBJECTIVE: To determine whether...

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Sweigart B, Andersen SL, Gurinovich A, et al. APOE E2/E2 Is Associated with Slower Rate of Cognitive Decline with Age. J Alzheimers Dis. 2021;83(2):853-860doi: 10.3233/JAD-201205.
Sweigart, B., Andersen, S. L., Gurinovich, A., Cosentino, S., Schupf, N., Perls, T. T., & Sebastiani, P. (2021). APOE E2/E2 Is Associated with Slower Rate of Cognitive Decline with Age. Journal of Alzheimer's disease : JAD, 83(2), 853-860. https://doi.org/10.3233/JAD-201205
Sweigart, Benjamin, et al. "APOE E2/E2 Is Associated with Slower Rate of Cognitive Decline with Age." Journal of Alzheimer's disease : JAD vol. 83,2 (2021): 853-860. doi: https://doi.org/10.3233/JAD-201205
Sweigart B, Andersen SL, Gurinovich A, Cosentino S, Schupf N, Perls TT, Sebastiani P. APOE E2/E2 Is Associated with Slower Rate of Cognitive Decline with Age. J Alzheimers Dis. 2021;83(2):853-860. doi: 10.3233/JAD-201205. PMID: 34366332.
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Effect of .

Alzheimer's & dementia (Amsterdam, Netherlands)

Ben Khedher MR, Haddad M, Laurin D, Ramassamy C.
PMID: 34541286
Alzheimers Dement (Amst). 2021 Sep 14;13(1):e12231. doi: 10.1002/dad2.12231. eCollection 2021.

INTRODUCTION: The substantial link between apolipoprotein E (METHODS: Apolipoproteins E, J, and D and antioxidant response markers were determined in cEVs and plasma using immunoblotting, electrochemical examination, and spectrofluorimetry.RESULTS: Total antioxidant capacity and apolipoprotein D levels in cEVs, as...

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Ben Khedher MR, Haddad M, Laurin D, et al. Effect of . Alzheimers Dement (Amst). 2021;13(1):e12231doi: 10.1002/dad2.12231.
Ben Khedher, M. R., Haddad, M., Laurin, D., & Ramassamy, C. (2021). Effect of . Alzheimer's & dementia (Amsterdam, Netherlands), 13(1), e12231. https://doi.org/10.1002/dad2.12231
Ben Khedher, Mohamed Raâfet, et al. "Effect of ." Alzheimer's & dementia (Amsterdam, Netherlands) vol. 13,1 (2021): e12231. doi: https://doi.org/10.1002/dad2.12231
Ben Khedher MR, Haddad M, Laurin D, Ramassamy C. Effect of . Alzheimers Dement (Amst). 2021 Sep 14;13(1):e12231. doi: 10.1002/dad2.12231. eCollection 2021. PMID: 34541286; PMCID: PMC8438681.
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Implications of GABAergic Neurotransmission in Alzheimer's Disease.

Frontiers in aging neuroscience

Li Y, Sun H, Chen Z, Xu H, Bu G, Zheng H.
PMID: 26941642
Front Aging Neurosci. 2016 Feb 23;8:31. doi: 10.3389/fnagi.2016.00031. eCollection 2016.

Alzheimer's disease (AD) is characterized pathologically by the deposition of β-amyloid peptides (Aβ) and the accumulation of neurofibrillary tangles (NFTs) composed of hyper-phosphorylated tau. Regardless of the pathological hallmarks, synaptic dysfunction is widely accepted as a causal event in...

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Li Y, Sun H, Chen Z, et al. Implications of GABAergic Neurotransmission in Alzheimer's Disease. Front Aging Neurosci. 2016;8:31doi: 10.3389/fnagi.2016.00031.
Li, Y., Sun, H., Chen, Z., Xu, H., Bu, G., & Zheng, H. (2016). Implications of GABAergic Neurotransmission in Alzheimer's Disease. Frontiers in aging neuroscience, 831. https://doi.org/10.3389/fnagi.2016.00031
Li, Yanfang, et al. "Implications of GABAergic Neurotransmission in Alzheimer's Disease." Frontiers in aging neuroscience vol. 8 (2016): 31. doi: https://doi.org/10.3389/fnagi.2016.00031
Li Y, Sun H, Chen Z, Xu H, Bu G, Zheng H. Implications of GABAergic Neurotransmission in Alzheimer's Disease. Front Aging Neurosci. 2016 Feb 23;8:31. doi: 10.3389/fnagi.2016.00031. eCollection 2016. PMID: 26941642; PMCID: PMC4763334.
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Cognitive decline is related to high blood glucose levels in older Chinese adults with the ApoE ε3/ε3 genotype.

Translational neurodegeneration

Qiu Q, Lin X, Sun L, Zhu MJ, Wang T, Wang JH, Li GJ, Xiao SF, Li X.
PMID: 30984391
Transl Neurodegener. 2019 Apr 03;8:12. doi: 10.1186/s40035-019-0151-2. eCollection 2019.

BACKGROUND: Few studies have investigated the effects of blood glucose (BG) on cognitive function in community-dwelling elderly individuals carrying the apolipoprotein E (APOE) ε3 allele.OBJECTIVE: To explore the effect of high BG levels on cognitive function in APOE ε3-carrying,...

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Qiu Q, Lin X, Sun L, et al. Cognitive decline is related to high blood glucose levels in older Chinese adults with the ApoE ε3/ε3 genotype. Transl Neurodegener. 2019;8:12doi: 10.1186/s40035-019-0151-2.
Qiu, Q., Lin, X., Sun, L., Zhu, M. J., Wang, T., Wang, J. H., Li, G. J., Xiao, S. F., & Li, X. (2019). Cognitive decline is related to high blood glucose levels in older Chinese adults with the ApoE ε3/ε3 genotype. Translational neurodegeneration, 812. https://doi.org/10.1186/s40035-019-0151-2
Qiu, Qi, et al. "Cognitive decline is related to high blood glucose levels in older Chinese adults with the ApoE ε3/ε3 genotype." Translational neurodegeneration vol. 8 (2019): 12. doi: https://doi.org/10.1186/s40035-019-0151-2
Qiu Q, Lin X, Sun L, Zhu MJ, Wang T, Wang JH, Li GJ, Xiao SF, Li X. Cognitive decline is related to high blood glucose levels in older Chinese adults with the ApoE ε3/ε3 genotype. Transl Neurodegener. 2019 Apr 03;8:12. doi: 10.1186/s40035-019-0151-2. eCollection 2019. PMID: 30984391; PMCID: PMC6446313.
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Apolipoprotein E (APOE) Polymorphisms and Susceptibility to Breast Cancer: A Meta-Analysis.

Cancer research and treatment

Saadat M.
PMID: 22802750
Cancer Res Treat. 2012 Jun;44(2):121-6. doi: 10.4143/crt.2012.44.2.121. Epub 2012 Jun 30.

PURPOSE: APOLIPOPROTEIN E (APOE, MIM: 107741) has three functionally distinct isoforms of the protein (E2, E3, and E4), encoded by corresponding alleles ε2, ε3, and ε4, which have been well described. Findings from previous studies investigating association between APOE...

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Saadat M. Apolipoprotein E (APOE) Polymorphisms and Susceptibility to Breast Cancer: A Meta-Analysis. Cancer Res Treat. 2012;44(2):121-6doi: 10.4143/crt.2012.44.2.121.
Saadat, M. (2012). Apolipoprotein E (APOE) Polymorphisms and Susceptibility to Breast Cancer: A Meta-Analysis. Cancer research and treatment, 44(2), 121-6. https://doi.org/10.4143/crt.2012.44.2.121
Saadat, Mostafa. "Apolipoprotein E (APOE) Polymorphisms and Susceptibility to Breast Cancer: A Meta-Analysis." Cancer research and treatment vol. 44,2 (2012): 121-6. doi: https://doi.org/10.4143/crt.2012.44.2.121
Saadat M. Apolipoprotein E (APOE) Polymorphisms and Susceptibility to Breast Cancer: A Meta-Analysis. Cancer Res Treat. 2012 Jun;44(2):121-6. doi: 10.4143/crt.2012.44.2.121. Epub 2012 Jun 30. PMID: 22802750; PMCID: PMC3394861.
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A Review of APOE Genotype-Dependent Autophagic Flux Regulation in Alzheimer's Disease.

Journal of Alzheimer's disease : JAD

Chen H, Chen F, Zhang M, Chen Y, Cui L, Liang C.
PMID: 34569952
J Alzheimers Dis. 2021;84(2):535-555. doi: 10.3233/JAD-210602.

Autophagy is a basic physiological process maintaining cell renewal, the degradation of dysfunctional organelles, and the clearance of abnormal proteins and has recently been identified as a main mechanism underlying the onset and progression of Alzheimer's disease (AD). The...

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Chen H, Chen F, Zhang M, et al. A Review of APOE Genotype-Dependent Autophagic Flux Regulation in Alzheimer's Disease. J Alzheimers Dis. 2021;84(2):535-555doi: 10.3233/JAD-210602.
Chen, H., Chen, F., Zhang, M., Chen, Y., Cui, L., & Liang, C. (2021). A Review of APOE Genotype-Dependent Autophagic Flux Regulation in Alzheimer's Disease. Journal of Alzheimer's disease : JAD, 84(2), 535-555. https://doi.org/10.3233/JAD-210602
Chen, Huiyi, et al. "A Review of APOE Genotype-Dependent Autophagic Flux Regulation in Alzheimer's Disease." Journal of Alzheimer's disease : JAD vol. 84,2 (2021): 535-555. doi: https://doi.org/10.3233/JAD-210602
Chen H, Chen F, Zhang M, Chen Y, Cui L, Liang C. A Review of APOE Genotype-Dependent Autophagic Flux Regulation in Alzheimer's Disease. J Alzheimers Dis. 2021;84(2):535-555. doi: 10.3233/JAD-210602. PMID: 34569952.
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Showing 1 to 12 of 16 entries