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Showing 1 to 12 of 13 entries
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Flavone from Zhongjiefeng (Herba Sarcandrae Glabrae) inhibits platelet apoptosis in immune-induced bone marrow failure through mitochondrial pathway.

Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan

Jiang Y, Zheng Q, Zhang A, Cui L, Xia L, Luo M.
PMID: 32188225
J Tradit Chin Med. 2017 Oct;37(5):643-649.

OBJECTIVE: To investigate the effect of Flavone from Zhongjiefeng (Herba Sarcandrae Glabrae) on the platelet number in immune-induced bone marrow failure (BMF) and its mechanism of mitochondrial apoptotic pathway.METHODS: Immune-induced BMF model, established in mice, was randomly divided into...

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Jiang Y, Zheng Q, Zhang A, et al. Flavone from Zhongjiefeng (Herba Sarcandrae Glabrae) inhibits platelet apoptosis in immune-induced bone marrow failure through mitochondrial pathway. J Tradit Chin Med. 2017;37(5):643-649
Jiang, Y., Zheng, Q., Zhang, A., Cui, L., Xia, L., & Luo, M. (2017). Flavone from Zhongjiefeng (Herba Sarcandrae Glabrae) inhibits platelet apoptosis in immune-induced bone marrow failure through mitochondrial pathway. Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan, 37(5), 643-649.
Jiang, Yiling, et al. "Flavone from Zhongjiefeng (Herba Sarcandrae Glabrae) inhibits platelet apoptosis in immune-induced bone marrow failure through mitochondrial pathway." Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan vol. 37,5 (2017): 643-649.
Jiang Y, Zheng Q, Zhang A, Cui L, Xia L, Luo M. Flavone from Zhongjiefeng (Herba Sarcandrae Glabrae) inhibits platelet apoptosis in immune-induced bone marrow failure through mitochondrial pathway. J Tradit Chin Med. 2017 Oct;37(5):643-649. PMID: 32188225.
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Effect and Mechanism of .

Diabetes, metabolic syndrome and obesity : targets and therapy

Shaher F, Wang S, Qiu H, Hu Y, Zhang Y, Wang W, Al-Ward H, Abdulghani MAM, Baldi S, Zhou S.
PMID: 33335409
Diabetes Metab Syndr Obes. 2020 Dec 07;13:4809-4822. doi: 10.2147/DMSO.S281527. eCollection 2020.

METHODS: Male SD rats were randomly divided into three groups. Two groups were given STZ (50 mg/kg, i.p.) treatment and when their fasting plasma glucose was above 16.7 mmol/L, among them, one group was given placebo, as diabetic group,...

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Shaher F, Wang S, Qiu H, et al. Effect and Mechanism of . Diabetes Metab Syndr Obes. 2020;13:4809-4822doi: 10.2147/DMSO.S281527.
Shaher, F., Wang, S., Qiu, H., Hu, Y., Zhang, Y., Wang, W., Al-Ward, H., Abdulghani, M. A. M., Baldi, S., & Zhou, S. (2020). Effect and Mechanism of . Diabetes, metabolic syndrome and obesity : targets and therapy, 134809-4822. https://doi.org/10.2147/DMSO.S281527
Shaher, Fahmi, et al. "Effect and Mechanism of ." Diabetes, metabolic syndrome and obesity : targets and therapy vol. 13 (2020): 4809-4822. doi: https://doi.org/10.2147/DMSO.S281527
Shaher F, Wang S, Qiu H, Hu Y, Zhang Y, Wang W, Al-Ward H, Abdulghani MAM, Baldi S, Zhou S. Effect and Mechanism of . Diabetes Metab Syndr Obes. 2020 Dec 07;13:4809-4822. doi: 10.2147/DMSO.S281527. eCollection 2020. PMID: 33335409; PMCID: PMC7736836.
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Caspase-9 and Bax/Bcl-2 regulation in ultraviolet B-induced apoptosis of murine peritoneal macrophages.

Toxicology mechanisms and methods

Sodhi A, Sethi G.
PMID: 20021098
Toxicol Mech Methods. 2004;14(6):323-9. doi: 10.1080/15376520490434656.

This study was performed to determine the effect of UVB radiation on the activation of apoptosis regulatory proteins using murine peritoneal macrophages, which are terminally, differentiated nondividing cells. UVB (100 mJ/cm2) irradiation induced apoptosis in murine peritoneal macrophages concurrent...

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Sodhi A, Sethi G. Caspase-9 and Bax/Bcl-2 regulation in ultraviolet B-induced apoptosis of murine peritoneal macrophages. Toxicol Mech Methods. 2004;14(6):323-9doi: 10.1080/15376520490434656.
Sodhi, A., & Sethi, G. (2004). Caspase-9 and Bax/Bcl-2 regulation in ultraviolet B-induced apoptosis of murine peritoneal macrophages. Toxicology mechanisms and methods, 14(6), 323-9. https://doi.org/10.1080/15376520490434656
Sodhi, Ajit, and Sethi, Gautam. "Caspase-9 and Bax/Bcl-2 regulation in ultraviolet B-induced apoptosis of murine peritoneal macrophages." Toxicology mechanisms and methods vol. 14,6 (2004): 323-9. doi: https://doi.org/10.1080/15376520490434656
Sodhi A, Sethi G. Caspase-9 and Bax/Bcl-2 regulation in ultraviolet B-induced apoptosis of murine peritoneal macrophages. Toxicol Mech Methods. 2004;14(6):323-9. doi: 10.1080/15376520490434656. PMID: 20021098.
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The influence of sleep deprivation on expression of apoptosis regulatory proteins p53, bcl-2 and bax following rat tongue carcinogenesis induced by 4-nitroquinoline 1-oxide.

Dental research journal

Noguti J, Alvarenga TA, Andersen ML, Tufik S, Oshima CT, Ribeiro DA.
PMID: 23946744
Dent Res J (Isfahan). 2013 Mar;10(2):247-53. doi: 10.4103/1735-3327.113360.

BACKGROUND: The aim of this study was to evaluate whether paradoxical sleep deprivation could affects the mechanisms and pathways essentials for cancer cells in tongue cancer induced by 4-nitroquinole 1-oxide in Wistar rats.MATERIALS AND METHODS: For this purpose, the...

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Noguti J, Alvarenga TA, Andersen ML, et al. The influence of sleep deprivation on expression of apoptosis regulatory proteins p53, bcl-2 and bax following rat tongue carcinogenesis induced by 4-nitroquinoline 1-oxide. Dent Res J (Isfahan). 2013;10(2):247-53doi: 10.4103/1735-3327.113360.
Noguti, J., Alvarenga, T. A., Andersen, M. L., Tufik, S., Oshima, C. T., & Ribeiro, D. A. (2013). The influence of sleep deprivation on expression of apoptosis regulatory proteins p53, bcl-2 and bax following rat tongue carcinogenesis induced by 4-nitroquinoline 1-oxide. Dental research journal, 10(2), 247-53. https://doi.org/10.4103/1735-3327.113360
Noguti, Juliana, et al. "The influence of sleep deprivation on expression of apoptosis regulatory proteins p53, bcl-2 and bax following rat tongue carcinogenesis induced by 4-nitroquinoline 1-oxide." Dental research journal vol. 10,2 (2013): 247-53. doi: https://doi.org/10.4103/1735-3327.113360
Noguti J, Alvarenga TA, Andersen ML, Tufik S, Oshima CT, Ribeiro DA. The influence of sleep deprivation on expression of apoptosis regulatory proteins p53, bcl-2 and bax following rat tongue carcinogenesis induced by 4-nitroquinoline 1-oxide. Dent Res J (Isfahan). 2013 Mar;10(2):247-53. doi: 10.4103/1735-3327.113360. PMID: 23946744; PMCID: PMC3731968.
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Anti-Cancer Effect of 3-(4-dimethylamino phenyl)-N-hydroxy-2-propenamide in MCF-7 Human Breast Cancer.

Environmental health and toxicology

Min KN, Joung KE, Kim DK, Sheen YY.
PMID: 22639737
Environ Health Toxicol. 2012;27:e2012010. doi: 10.5620/eht.2012.27.e2012010. Epub 2012 Apr 25.

OBJECTIVES: In recent years, a number of structurally diverse Histone deacetylase (HDAC) inhibitors have been identified and these HDAC inhibitors induce growth arrest, differentiation and/or apoptosis of cancer cells in vitro and in vivo. This study aimed at investigating...

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Min KN, Joung KE, Kim DK, et al. Anti-Cancer Effect of 3-(4-dimethylamino phenyl)-N-hydroxy-2-propenamide in MCF-7 Human Breast Cancer. Environ Health Toxicol. 2012;27:e2012010doi: 10.5620/eht.2012.27.e2012010.
Min, K. N., Joung, K. E., Kim, D. K., & Sheen, Y. Y. (2012). Anti-Cancer Effect of 3-(4-dimethylamino phenyl)-N-hydroxy-2-propenamide in MCF-7 Human Breast Cancer. Environmental health and toxicology, 27e2012010. https://doi.org/10.5620/eht.2012.27.e2012010
Min, Kyung Nan, et al. "Anti-Cancer Effect of 3-(4-dimethylamino phenyl)-N-hydroxy-2-propenamide in MCF-7 Human Breast Cancer." Environmental health and toxicology vol. 27 (2012): e2012010. doi: https://doi.org/10.5620/eht.2012.27.e2012010
Min KN, Joung KE, Kim DK, Sheen YY. Anti-Cancer Effect of 3-(4-dimethylamino phenyl)-N-hydroxy-2-propenamide in MCF-7 Human Breast Cancer. Environ Health Toxicol. 2012;27:e2012010. doi: 10.5620/eht.2012.27.e2012010. Epub 2012 Apr 25. PMID: 22639737; PMCID: PMC3355273.
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Sodium Danshensu promotes the healing of stage 2 pressure injury wounds in ischemia/reperfusion injury rat models: possible regulation of apoptosis and inflammatory response.

Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan

Zhang QZ, Fu TT, Dai JN, Zhou ZN, Shen CZ.
PMID: 34392650
J Tradit Chin Med. 2021 Aug;41(4):571-580. doi: 10.19852/j.cnki.jtcm.2021.03.009.

OBJECTIVE: To investigate the protective effect and possible mechanism of sodium Danshensu (SDSS) against pressure injury caused by ischemia/reperfusion (I/R) injury.METHODS: Sprague-Dawley rats were randomly divided into five groups of eight rats each: control group, model group, 10 mg/kg...

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Zhang QZ, Fu TT, Dai JN, et al. Sodium Danshensu promotes the healing of stage 2 pressure injury wounds in ischemia/reperfusion injury rat models: possible regulation of apoptosis and inflammatory response. J Tradit Chin Med. 2021;41(4):571-580doi: 10.19852/j.cnki.jtcm.2021.03.009.
Zhang, Q. Z., Fu, T. T., Dai, J. N., Zhou, Z. N., & Shen, C. Z. (2021). Sodium Danshensu promotes the healing of stage 2 pressure injury wounds in ischemia/reperfusion injury rat models: possible regulation of apoptosis and inflammatory response. Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan, 41(4), 571-580. https://doi.org/10.19852/j.cnki.jtcm.2021.03.009
Zhang, Qiong-Zhi, et al. "Sodium Danshensu promotes the healing of stage 2 pressure injury wounds in ischemia/reperfusion injury rat models: possible regulation of apoptosis and inflammatory response." Journal of traditional Chinese medicine = Chung i tsa chih ying wen pan vol. 41,4 (2021): 571-580. doi: https://doi.org/10.19852/j.cnki.jtcm.2021.03.009
Zhang QZ, Fu TT, Dai JN, Zhou ZN, Shen CZ. Sodium Danshensu promotes the healing of stage 2 pressure injury wounds in ischemia/reperfusion injury rat models: possible regulation of apoptosis and inflammatory response. J Tradit Chin Med. 2021 Aug;41(4):571-580. doi: 10.19852/j.cnki.jtcm.2021.03.009. PMID: 34392650.
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TNF-alpha downregulates E-cadherin and sensitizes response to γ-irradiation in Caco-2 cells.

Cancer research and treatment

Yi JY, Jung YJ, Choi SS, Chung E.
PMID: 19809566
Cancer Res Treat. 2009 Sep;41(3):164-70. doi: 10.4143/crt.2009.41.3.164. Epub 2009 Sep 30.

PURPOSE: The purpose of the present study was to assess the biological effects of TNF-alpha in Caco-2 well-differentiated colon adenocarcinoma cells and to determine radiation sensitivity in order to develop TNF-alpha into a cancer therapeutic agent.MATERIALS AND METHODS: A...

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Yi JY, Jung YJ, Choi SS, et al. TNF-alpha downregulates E-cadherin and sensitizes response to γ-irradiation in Caco-2 cells. Cancer Res Treat. 2009;41(3):164-70doi: 10.4143/crt.2009.41.3.164.
Yi, J. Y., Jung, Y. J., Choi, S. S., & Chung, E. (2009). TNF-alpha downregulates E-cadherin and sensitizes response to γ-irradiation in Caco-2 cells. Cancer research and treatment, 41(3), 164-70. https://doi.org/10.4143/crt.2009.41.3.164
Yi, Jae Youn, et al. "TNF-alpha downregulates E-cadherin and sensitizes response to γ-irradiation in Caco-2 cells." Cancer research and treatment vol. 41,3 (2009): 164-70. doi: https://doi.org/10.4143/crt.2009.41.3.164
Yi JY, Jung YJ, Choi SS, Chung E. TNF-alpha downregulates E-cadherin and sensitizes response to γ-irradiation in Caco-2 cells. Cancer Res Treat. 2009 Sep;41(3):164-70. doi: 10.4143/crt.2009.41.3.164. Epub 2009 Sep 30. PMID: 19809566; PMCID: PMC2757662.
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Aqueous Extract of Shi-Liu-Wei-Liu-Qi-Yin Induces G2/M Phase Arrest and Apoptosis in Human Bladder Carcinoma Cells via Fas and Mitochondrial Pathway.

Evidence-based complementary and alternative medicine : eCAM

Ou TT, Wang CJ, Hung GU, Wu CH, Lee HJ.
PMID: 19383839
Evid Based Complement Alternat Med. 2011;2011:nep016. doi: 10.1093/ecam/nep016. Epub 2011 Jan 03.

Shi-Liu-Wei-Liu-Qi-Yin (SLWLQY) was traditionally used to treat cancers. However, scientific evidence of the anticancer effects still remains undefined. In this study, we aimed to clarify the possible mechanisms of SLWLQY in treating cancer. We evaluated the effects of SLWLQY...

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Ou TT, Wang CJ, Hung GU, et al. Aqueous Extract of Shi-Liu-Wei-Liu-Qi-Yin Induces G2/M Phase Arrest and Apoptosis in Human Bladder Carcinoma Cells via Fas and Mitochondrial Pathway. Evid Based Complement Alternat Med. 2011;2011:nep016doi: 10.1093/ecam/nep016.
Ou, T. T., Wang, C. J., Hung, G. U., Wu, C. H., & Lee, H. J. (2011). Aqueous Extract of Shi-Liu-Wei-Liu-Qi-Yin Induces G2/M Phase Arrest and Apoptosis in Human Bladder Carcinoma Cells via Fas and Mitochondrial Pathway. Evidence-based complementary and alternative medicine : eCAM, 2011nep016. https://doi.org/10.1093/ecam/nep016
Ou, Ting-Tsz, et al. "Aqueous Extract of Shi-Liu-Wei-Liu-Qi-Yin Induces G2/M Phase Arrest and Apoptosis in Human Bladder Carcinoma Cells via Fas and Mitochondrial Pathway." Evidence-based complementary and alternative medicine : eCAM vol. 2011 (2011): nep016. doi: https://doi.org/10.1093/ecam/nep016
Ou TT, Wang CJ, Hung GU, Wu CH, Lee HJ. Aqueous Extract of Shi-Liu-Wei-Liu-Qi-Yin Induces G2/M Phase Arrest and Apoptosis in Human Bladder Carcinoma Cells via Fas and Mitochondrial Pathway. Evid Based Complement Alternat Med. 2011;2011:nep016. doi: 10.1093/ecam/nep016. Epub 2011 Jan 03. PMID: 19383839; PMCID: PMC3135273.
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Immunotoxicological Effects of Aripiprazole: In vivo and In vitro Studies.

The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Baek KS, Ahn S, Lee J, Kim JH, Kim HG, Kim E, Kim JH, Sung NY, Yang S, Kim MS, Hong S, Kim JH, Cho JY.
PMID: 26170741
Korean J Physiol Pharmacol. 2015 Jul;19(4):365-72. doi: 10.4196/kjpp.2015.19.4.365. Epub 2015 Jun 30.

Aripiprazole (ARI) is a commonly prescribed medication used to treat schizophrenia and bipolar disorder. To date, there have been no studies regarding the molecular pathological and immunotoxicological profiling of aripiprazole. Thus, in the present study, we prepared two different...

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Baek KS, Ahn S, Lee J, et al. Immunotoxicological Effects of Aripiprazole: In vivo and In vitro Studies. Korean J Physiol Pharmacol. 2015;19(4):365-72doi: 10.4196/kjpp.2015.19.4.365.
Baek, K. S., Ahn, S., Lee, J., Kim, J. H., Kim, H. G., Kim, E., Kim, J. H., Sung, N. Y., Yang, S., Kim, M. S., Hong, S., Kim, J. H., & Cho, J. Y. (2015). Immunotoxicological Effects of Aripiprazole: In vivo and In vitro Studies. The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, 19(4), 365-72. https://doi.org/10.4196/kjpp.2015.19.4.365
Baek, Kwang-Soo, et al. "Immunotoxicological Effects of Aripiprazole: In vivo and In vitro Studies." The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology vol. 19,4 (2015): 365-72. doi: https://doi.org/10.4196/kjpp.2015.19.4.365
Baek KS, Ahn S, Lee J, Kim JH, Kim HG, Kim E, Kim JH, Sung NY, Yang S, Kim MS, Hong S, Kim JH, Cho JY. Immunotoxicological Effects of Aripiprazole: In vivo and In vitro Studies. Korean J Physiol Pharmacol. 2015 Jul;19(4):365-72. doi: 10.4196/kjpp.2015.19.4.365. Epub 2015 Jun 30. PMID: 26170741; PMCID: PMC4499649.
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Anti-Cancer Effect of IN-2001 in T47D Human Breast Cancer.

Biomolecules & therapeutics

Joung KE, Min KN, Kim DK, Sheen YY.
PMID: 24116279
Biomol Ther (Seoul). 2012 Jan;20(1):81-8. doi: 10.4062/biomolther.2012.20.1.081.

Histone deacetylases (HDACs) are enzymes involved in the remodelling of chromatin, and have a key role in the epigenetic regulation of gene expression. Histone deacetylase (HDAC) inhibitors are emerging as an exciting new class of potential anti-cancer agents. In...

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Joung KE, Min KN, Kim DK, et al. Anti-Cancer Effect of IN-2001 in T47D Human Breast Cancer. Biomol Ther (Seoul). 2012;20(1):81-8doi: 10.4062/biomolther.2012.20.1.081.
Joung, K. E., Min, K. N., Kim, D. K., & Sheen, Y. Y. (2012). Anti-Cancer Effect of IN-2001 in T47D Human Breast Cancer. Biomolecules & therapeutics, 20(1), 81-8. https://doi.org/10.4062/biomolther.2012.20.1.081
Joung, Ki Eun, et al. "Anti-Cancer Effect of IN-2001 in T47D Human Breast Cancer." Biomolecules & therapeutics vol. 20,1 (2012): 81-8. doi: https://doi.org/10.4062/biomolther.2012.20.1.081
Joung KE, Min KN, Kim DK, Sheen YY. Anti-Cancer Effect of IN-2001 in T47D Human Breast Cancer. Biomol Ther (Seoul). 2012 Jan;20(1):81-8. doi: 10.4062/biomolther.2012.20.1.081. PMID: 24116279; PMCID: PMC3792206.
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Time point expression of apoptosis regulatory proteins in a photochemically-induced focal cerebral ischemic rat brain.

Chonnam medical journal

Kim HS, Park MS, Lee JK, Kim HJ, Park JT, Lee MC.
PMID: 22247913
Chonnam Med J. 2011 Dec;47(3):144-9. doi: 10.4068/cmj.2011.47.3.144. Epub 2011 Dec 26.

Apoptosis after global or focal cerebral ischemia plays a crucial role in mediating cell death. In this study, we observed the time point expression of physiologic events involving apoptosis regulatory proteins after photochemically-induced focal cerebral ischemia in Sprague-Dawley rats....

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Kim HS, Park MS, Lee JK, et al. Time point expression of apoptosis regulatory proteins in a photochemically-induced focal cerebral ischemic rat brain. Chonnam Med J. 2011;47(3):144-9doi: 10.4068/cmj.2011.47.3.144.
Kim, H. S., Park, M. S., Lee, J. K., Kim, H. J., Park, J. T., & Lee, M. C. (2011). Time point expression of apoptosis regulatory proteins in a photochemically-induced focal cerebral ischemic rat brain. Chonnam medical journal, 47(3), 144-9. https://doi.org/10.4068/cmj.2011.47.3.144
Kim, Hyung-Seok, et al. "Time point expression of apoptosis regulatory proteins in a photochemically-induced focal cerebral ischemic rat brain." Chonnam medical journal vol. 47,3 (2011): 144-9. doi: https://doi.org/10.4068/cmj.2011.47.3.144
Kim HS, Park MS, Lee JK, Kim HJ, Park JT, Lee MC. Time point expression of apoptosis regulatory proteins in a photochemically-induced focal cerebral ischemic rat brain. Chonnam Med J. 2011 Dec;47(3):144-9. doi: 10.4068/cmj.2011.47.3.144. Epub 2011 Dec 26. PMID: 22247913; PMCID: PMC3252501.
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Bim and VDAC1 are hierarchically essential for mitochondrial ATF2 mediated cell death.

Cancer cell international

Liu Z, Luo Q, Guo C.
PMID: 25852302
Cancer Cell Int. 2015 Mar 30;15:34. doi: 10.1186/s12935-015-0188-y. eCollection 2015.

BACKGROUND: ATF2 mediated cytochrome c release is the formation of a channel with some unknown factors larger than that of the individual proteins. BHS-only proteins (BH3s), such as Bim, could induce BAX and VDAC, forming a new channel. According...

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Liu Z, Luo Q, Guo C. Bim and VDAC1 are hierarchically essential for mitochondrial ATF2 mediated cell death. Cancer Cell Int. 2015;15:34doi: 10.1186/s12935-015-0188-y.
Liu, Z., Luo, Q., & Guo, C. (2015). Bim and VDAC1 are hierarchically essential for mitochondrial ATF2 mediated cell death. Cancer cell international, 1534. https://doi.org/10.1186/s12935-015-0188-y
Liu, Zhaoyun, et al. "Bim and VDAC1 are hierarchically essential for mitochondrial ATF2 mediated cell death." Cancer cell international vol. 15 (2015): 34. doi: https://doi.org/10.1186/s12935-015-0188-y
Liu Z, Luo Q, Guo C. Bim and VDAC1 are hierarchically essential for mitochondrial ATF2 mediated cell death. Cancer Cell Int. 2015 Mar 30;15:34. doi: 10.1186/s12935-015-0188-y. eCollection 2015. PMID: 25852302; PMCID: PMC4387661.
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