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Effects of Putative Neurotransmitters of the Carotid Body on its Own Glomus Cells.

The European journal of neuroscience

Eyzaguirre C, Monti-Bloch L, Woodbury JW.
PMID: 12106105
Eur J Neurosci. 1990 Jan;2(1):77-88. doi: 10.1111/j.1460-9568.1990.tb00383.x.

Carotid body glomus cells produce and release acetylcholine (ACh), catecholamines, and neuropeptides, and there is biochemical evidence that these cells possess receptors for these substances. Thus, we studied the effects of cholinergics [ACh, nicotine (Nic), bethanechol (BN)] and peptides...

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Eyzaguirre C, Monti-Bloch L, Woodbury JW. Effects of Putative Neurotransmitters of the Carotid Body on its Own Glomus Cells. Eur J Neurosci. 1990;2(1):77-88doi: 10.1111/j.1460-9568.1990.tb00383.x.
Eyzaguirre, C., Monti-Bloch, L., & Woodbury, J. W. (1990). Effects of Putative Neurotransmitters of the Carotid Body on its Own Glomus Cells. The European journal of neuroscience, 2(1), 77-88. https://doi.org/10.1111/j.1460-9568.1990.tb00383.x
Eyzaguirre, C., et al. "Effects of Putative Neurotransmitters of the Carotid Body on its Own Glomus Cells." The European journal of neuroscience vol. 2,1 (1990): 77-88. doi: https://doi.org/10.1111/j.1460-9568.1990.tb00383.x
Eyzaguirre C, Monti-Bloch L, Woodbury JW. Effects of Putative Neurotransmitters of the Carotid Body on its Own Glomus Cells. Eur J Neurosci. 1990 Jan;2(1):77-88. doi: 10.1111/j.1460-9568.1990.tb00383.x. PMID: 12106105.
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Deletion of P2X2 and P2X3 receptor subunits does not alter motility of the mouse colon.

Frontiers in neuroscience

Devries MP, Vessalo M, Galligan JJ.
PMID: 20582262
Front Neurosci. 2010 Mar 19;4:22. doi: 10.3389/fnent.2010.00001. eCollection 2010.

Purinergic P2X receptors contribute to neurotransmission in the gut. P2X receptors are ligand-gated cation channels that mediate synaptic excitation in subsets of enteric neurons. The present study evaluated colonic motility in vitro and in vivo in wild type (WT)...

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Devries MP, Vessalo M, Galligan JJ. Deletion of P2X2 and P2X3 receptor subunits does not alter motility of the mouse colon. Front Neurosci. 2010;4:22doi: 10.3389/fnent.2010.00001.
Devries, M. P., Vessalo, M., & Galligan, J. J. (2010). Deletion of P2X2 and P2X3 receptor subunits does not alter motility of the mouse colon. Frontiers in neuroscience, 422. https://doi.org/10.3389/fnent.2010.00001
Devries, Matthew P, et al. "Deletion of P2X2 and P2X3 receptor subunits does not alter motility of the mouse colon." Frontiers in neuroscience vol. 4 (2010): 22. doi: https://doi.org/10.3389/fnent.2010.00001
Devries MP, Vessalo M, Galligan JJ. Deletion of P2X2 and P2X3 receptor subunits does not alter motility of the mouse colon. Front Neurosci. 2010 Mar 19;4:22. doi: 10.3389/fnent.2010.00001. eCollection 2010. PMID: 20582262; PMCID: PMC2858605.
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Sudden Heart Rate Reduction Upon Optogenetic Release of Acetylcholine From Cardiac Parasympathetic Neurons in Perfused Hearts.

Frontiers in physiology

Moreno A, Endicott K, Skancke M, Dwyer MK, Brennan J, Efimov IR, Trachiotis G, Mendelowitz D, Kay MW.
PMID: 30745877
Front Physiol. 2019 Jan 28;10:16. doi: 10.3389/fphys.2019.00016. eCollection 2019.

The balance of sympathetic and parasympathetic tone provides exquisite control of heart rate and contractility and has also been shown to modulate coronary flow and inflammation. Understanding how autonomic balance is altered by cardiac disease is an active area...

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Moreno A, Endicott K, Skancke M, et al. Sudden Heart Rate Reduction Upon Optogenetic Release of Acetylcholine From Cardiac Parasympathetic Neurons in Perfused Hearts. Front Physiol. 2019;10:16doi: 10.3389/fphys.2019.00016.
Moreno, A., Endicott, K., Skancke, M., Dwyer, M. K., Brennan, J., Efimov, I. R., Trachiotis, G., Mendelowitz, D., & Kay, M. W. (2019). Sudden Heart Rate Reduction Upon Optogenetic Release of Acetylcholine From Cardiac Parasympathetic Neurons in Perfused Hearts. Frontiers in physiology, 1016. https://doi.org/10.3389/fphys.2019.00016
Moreno, Angel, et al. "Sudden Heart Rate Reduction Upon Optogenetic Release of Acetylcholine From Cardiac Parasympathetic Neurons in Perfused Hearts." Frontiers in physiology vol. 10 (2019): 16. doi: https://doi.org/10.3389/fphys.2019.00016
Moreno A, Endicott K, Skancke M, Dwyer MK, Brennan J, Efimov IR, Trachiotis G, Mendelowitz D, Kay MW. Sudden Heart Rate Reduction Upon Optogenetic Release of Acetylcholine From Cardiac Parasympathetic Neurons in Perfused Hearts. Front Physiol. 2019 Jan 28;10:16. doi: 10.3389/fphys.2019.00016. eCollection 2019. PMID: 30745877; PMCID: PMC6360159.
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Bethanechol used to prevent salivary gland dysfunction in patients submitted to radioactive iodine therapy: a double blind, placebo-controlled, randomized study.

Journal of stomatology, oral and maxillofacial surgery

Campanhã D, Pereira EN, Alves FA, Jaguar GC.
PMID: 34954424
J Stomatol Oral Maxillofac Surg. 2021 Dec 23; doi: 10.1016/j.jormas.2021.12.014. Epub 2021 Dec 23.

Symptoms related to salivary gland damage are one of the most frustrating complications after radioactive iodine (

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Campanhã D, Pereira EN, Alves FA, et al. Bethanechol used to prevent salivary gland dysfunction in patients submitted to radioactive iodine therapy: a double blind, placebo-controlled, randomized study. J Stomatol Oral Maxillofac Surg. 2021;doi: 10.1016/j.jormas.2021.12.014.
Campanhã, D., Pereira, E. N., Alves, F. A., & Jaguar, G. C. (2021). Bethanechol used to prevent salivary gland dysfunction in patients submitted to radioactive iodine therapy: a double blind, placebo-controlled, randomized study. Journal of stomatology, oral and maxillofacial surgery, . https://doi.org/10.1016/j.jormas.2021.12.014
Campanhã, Daniel, et al. "Bethanechol used to prevent salivary gland dysfunction in patients submitted to radioactive iodine therapy: a double blind, placebo-controlled, randomized study." Journal of stomatology, oral and maxillofacial surgery vol. (2021). doi: https://doi.org/10.1016/j.jormas.2021.12.014
Campanhã D, Pereira EN, Alves FA, Jaguar GC. Bethanechol used to prevent salivary gland dysfunction in patients submitted to radioactive iodine therapy: a double blind, placebo-controlled, randomized study. J Stomatol Oral Maxillofac Surg. 2021 Dec 23; doi: 10.1016/j.jormas.2021.12.014. Epub 2021 Dec 23. PMID: 34954424.
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Acetylcholine Receptor Agonists Provide Cardioprotection in Doxorubicin-induced Cardiotoxicity via Modulating Muscarinic M.

Translational research : the journal of laboratory and clinical medicine

Prathumsap N, Ongnok B, Khuanjing T, Arinno A, Maneechote C, Apaijai N, Chunchai T, Arunsak B, Shinlapawittayatorn K, Chattipakorn SC, Chattipakorn N.
PMID: 34920165
Transl Res. 2021 Dec 14; doi: 10.1016/j.trsl.2021.12.005. Epub 2021 Dec 14.

The balance between cardiac sympathetic and parasympathetic activities has been intricately linked to mitochondrial function, cellular oxidative status, and immunomodulation in healthy and diseased myocardium. Cardiac autonomic neuropathy, along with the associated mitochondrial and cellular dysfunction, is an important...

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Prathumsap N, Ongnok B, Khuanjing T, et al. Acetylcholine Receptor Agonists Provide Cardioprotection in Doxorubicin-induced Cardiotoxicity via Modulating Muscarinic M. Transl Res. 2021;doi: 10.1016/j.trsl.2021.12.005.
Prathumsap, N., Ongnok, B., Khuanjing, T., Arinno, A., Maneechote, C., Apaijai, N., Chunchai, T., Arunsak, B., Shinlapawittayatorn, K., Chattipakorn, S. C., & Chattipakorn, N. (2021). Acetylcholine Receptor Agonists Provide Cardioprotection in Doxorubicin-induced Cardiotoxicity via Modulating Muscarinic M. Translational research : the journal of laboratory and clinical medicine, . https://doi.org/10.1016/j.trsl.2021.12.005
Prathumsap, Nanthip, et al. "Acetylcholine Receptor Agonists Provide Cardioprotection in Doxorubicin-induced Cardiotoxicity via Modulating Muscarinic M." Translational research : the journal of laboratory and clinical medicine vol. (2021). doi: https://doi.org/10.1016/j.trsl.2021.12.005
Prathumsap N, Ongnok B, Khuanjing T, Arinno A, Maneechote C, Apaijai N, Chunchai T, Arunsak B, Shinlapawittayatorn K, Chattipakorn SC, Chattipakorn N. Acetylcholine Receptor Agonists Provide Cardioprotection in Doxorubicin-induced Cardiotoxicity via Modulating Muscarinic M. Transl Res. 2021 Dec 14; doi: 10.1016/j.trsl.2021.12.005. Epub 2021 Dec 14. PMID: 34920165.
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Gastric motor dysfunction coincides with the onset of obesity in rats fed with high-fat diet.

Clinical and experimental pharmacology & physiology

Kaya SD, Sinen O, Bülbül M.
PMID: 33352619
Clin Exp Pharmacol Physiol. 2021 Apr;48(4):553-562. doi: 10.1111/1440-1681.13448. Epub 2020 Dec 22.

Exposure to a high-fat diet (HFD) has been reported to impair central autonomic and enteric neurocircuitries, however, the relevant mechanisms and their time course are inadequately clarified. This study aimed to investigate the effects of HFD consumption through the...

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Kaya SD, Sinen O, Bülbül M. Gastric motor dysfunction coincides with the onset of obesity in rats fed with high-fat diet. Clin Exp Pharmacol Physiol. 2020;48(4):553-562doi: 10.1111/1440-1681.13448.
Kaya, S. D., Sinen, O., & Bülbül, M. (2021). Gastric motor dysfunction coincides with the onset of obesity in rats fed with high-fat diet. Clinical and experimental pharmacology & physiology, 48(4), 553-562. https://doi.org/10.1111/1440-1681.13448
Kaya, Sabriye Defne, et al. "Gastric motor dysfunction coincides with the onset of obesity in rats fed with high-fat diet." Clinical and experimental pharmacology & physiology vol. 48,4 (2021): 553-562. doi: https://doi.org/10.1111/1440-1681.13448
Kaya SD, Sinen O, Bülbül M. Gastric motor dysfunction coincides with the onset of obesity in rats fed with high-fat diet. Clin Exp Pharmacol Physiol. 2021 Apr;48(4):553-562. doi: 10.1111/1440-1681.13448. Epub 2020 Dec 22. PMID: 33352619.
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Enhanced Contractive Tension and Upregulated Muscarinic Receptor 2/3 in Colorectum Contribute to Constipation in 6-Hydroxydopamine-Induced Parkinson's Disease Rats.

Frontiers in aging neuroscience

Zhang XL, Zhang XH, Yu X, Zheng LF, Feng XY, Liu CZ, Quan ZS, Zhang Y, Zhu JX.
PMID: 35002677
Front Aging Neurosci. 2021 Dec 23;13:770841. doi: 10.3389/fnagi.2021.770841. eCollection 2021.

Constipation and defecatory dysfunctions are frequent symptoms in patients with Parkinson's disease (PD). The pathology of Lewy bodies in colonic and rectal cholinergic neurons suggests that cholinergic pathways are involved in colorectal dysmotility in PD. However, the underlying mechanism...

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Zhang XL, Zhang XH, Yu X, et al. Enhanced Contractive Tension and Upregulated Muscarinic Receptor 2/3 in Colorectum Contribute to Constipation in 6-Hydroxydopamine-Induced Parkinson's Disease Rats. Front Aging Neurosci. 2021;13:770841doi: 10.3389/fnagi.2021.770841.
Zhang, X. L., Zhang, X. H., Yu, X., Zheng, L. F., Feng, X. Y., Liu, C. Z., Quan, Z. S., Zhang, Y., & Zhu, J. X. (2021). Enhanced Contractive Tension and Upregulated Muscarinic Receptor 2/3 in Colorectum Contribute to Constipation in 6-Hydroxydopamine-Induced Parkinson's Disease Rats. Frontiers in aging neuroscience, 13770841. https://doi.org/10.3389/fnagi.2021.770841
Zhang, Xiao-Li, et al. "Enhanced Contractive Tension and Upregulated Muscarinic Receptor 2/3 in Colorectum Contribute to Constipation in 6-Hydroxydopamine-Induced Parkinson's Disease Rats." Frontiers in aging neuroscience vol. 13 (2021): 770841. doi: https://doi.org/10.3389/fnagi.2021.770841
Zhang XL, Zhang XH, Yu X, Zheng LF, Feng XY, Liu CZ, Quan ZS, Zhang Y, Zhu JX. Enhanced Contractive Tension and Upregulated Muscarinic Receptor 2/3 in Colorectum Contribute to Constipation in 6-Hydroxydopamine-Induced Parkinson's Disease Rats. Front Aging Neurosci. 2021 Dec 23;13:770841. doi: 10.3389/fnagi.2021.770841. eCollection 2021. PMID: 35002677; PMCID: PMC8733788.
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The effectiveness of parasympathomimetics for treating underactive bladder: A systematic review and meta-analysis.

Neurourology and urodynamics

Moro C, Phelps C, Veer V, Clark J, Glasziou P, Tikkinen KAO, Scott AM.
PMID: 34816481
Neurourol Urodyn. 2022 Jan;41(1):127-139. doi: 10.1002/nau.24839. Epub 2021 Nov 24.

AIMS: Biological rationale suggests that parasympathomimetics (cholinergic receptor stimulating agents) could be beneficial for patients with underactive bladder. However, no systematic review with meta-analysis addressing potential benefits or adverse effects exists. The aim of this review was to assess...

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Moro C, Phelps C, Veer V, et al. The effectiveness of parasympathomimetics for treating underactive bladder: A systematic review and meta-analysis. Neurourol Urodyn. 2021;41(1):127-139doi: 10.1002/nau.24839.
Moro, C., Phelps, C., Veer, V., Clark, J., Glasziou, P., Tikkinen, K. A. O., & Scott, A. M. (2022). The effectiveness of parasympathomimetics for treating underactive bladder: A systematic review and meta-analysis. Neurourology and urodynamics, 41(1), 127-139. https://doi.org/10.1002/nau.24839
Moro, Christian, et al. "The effectiveness of parasympathomimetics for treating underactive bladder: A systematic review and meta-analysis." Neurourology and urodynamics vol. 41,1 (2022): 127-139. doi: https://doi.org/10.1002/nau.24839
Moro C, Phelps C, Veer V, Clark J, Glasziou P, Tikkinen KAO, Scott AM. The effectiveness of parasympathomimetics for treating underactive bladder: A systematic review and meta-analysis. Neurourol Urodyn. 2022 Jan;41(1):127-139. doi: 10.1002/nau.24839. Epub 2021 Nov 24. PMID: 34816481.
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Muscarinic pharmacology of the inhibition of adenylate cyclase in N18TG2 neuroblastoma cells.

Neurochemistry international

Bidaut-Russell M, Howlett AC.
PMID: 20501173
Neurochem Int. 1987;11(3):287-92. doi: 10.1016/0197-0186(87)90048-9.

This study characterizes the muscarinic cholinergic receptors associated with the inhibition of adenylate cyclase on N18TG2 neuroblastoma cell membranes. Agonists could be divided into two classes: oxotremorine, acetylcholine, carbachol and arecoline exerted the most efficacious and potent inhibition, while...

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Bidaut-Russell M, Howlett AC. Muscarinic pharmacology of the inhibition of adenylate cyclase in N18TG2 neuroblastoma cells. Neurochem Int. 1987;11(3):287-92doi: 10.1016/0197-0186(87)90048-9.
Bidaut-Russell, M., & Howlett, A. C. (1987). Muscarinic pharmacology of the inhibition of adenylate cyclase in N18TG2 neuroblastoma cells. Neurochemistry international, 11(3), 287-92. https://doi.org/10.1016/0197-0186(87)90048-9
Bidaut-Russell, M, and Howlett, A C. "Muscarinic pharmacology of the inhibition of adenylate cyclase in N18TG2 neuroblastoma cells." Neurochemistry international vol. 11,3 (1987): 287-92. doi: https://doi.org/10.1016/0197-0186(87)90048-9
Bidaut-Russell M, Howlett AC. Muscarinic pharmacology of the inhibition of adenylate cyclase in N18TG2 neuroblastoma cells. Neurochem Int. 1987;11(3):287-92. doi: 10.1016/0197-0186(87)90048-9. PMID: 20501173.
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Bibliometric profile of neurogenic bladder in the literature: a 20-year bibliometric analysis.

Neural regeneration research

Gao Y, Qu B, Shen Y, Su XJ, Dong XY, Chen XM, Zhou YH, Pi HY.
PMID: 26109957
Neural Regen Res. 2015 May;10(5):797-803. doi: 10.4103/1673-5374.156985.

Neurogenic bladder is a dysfunction of the lower urinary tract caused by nervous system disorder. We investigated the trends in publication of articles under the topic "neurogenic bladder" using bibliometric analysis. Articles on neurogenic bladder, published between 1995 and...

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Gao Y, Qu B, Shen Y, et al. Bibliometric profile of neurogenic bladder in the literature: a 20-year bibliometric analysis. Neural Regen Res. 2015;10(5):797-803doi: 10.4103/1673-5374.156985.
Gao, Y., Qu, B., Shen, Y., Su, X. J., Dong, X. Y., Chen, X. M., Zhou, Y. H., & Pi, H. Y. (2015). Bibliometric profile of neurogenic bladder in the literature: a 20-year bibliometric analysis. Neural regeneration research, 10(5), 797-803. https://doi.org/10.4103/1673-5374.156985
Gao, Yuan, et al. "Bibliometric profile of neurogenic bladder in the literature: a 20-year bibliometric analysis." Neural regeneration research vol. 10,5 (2015): 797-803. doi: https://doi.org/10.4103/1673-5374.156985
Gao Y, Qu B, Shen Y, Su XJ, Dong XY, Chen XM, Zhou YH, Pi HY. Bibliometric profile of neurogenic bladder in the literature: a 20-year bibliometric analysis. Neural Regen Res. 2015 May;10(5):797-803. doi: 10.4103/1673-5374.156985. PMID: 26109957; PMCID: PMC4468774.
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Effects of (r,r)- and (r,r/s,s)-formoterol on airway relaxation and contraction in an experimental rat model.

Current therapeutic research, clinical and experimental

Mhanna MJ, Koester JF, Cohn RC.
PMID: 24683215
Curr Ther Res Clin Exp. 2007 Jul;68(4):249-61. doi: 10.1016/j.curtheres.2007.08.005.

BACKGROUND: Racemic (R,R/S,S)-formoterol is a long-acting β-agonist composed of a 50:50 mixture of (R,R)- and (S,S)-enantiomers.OBJECTIVE: The aim of this study was to determine whether (R,R)-formoterol and (R,R/S,S)-formoterol have differing effects on airway contraction and relaxation in vitro.METHODS: Cylindrical...

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Mhanna MJ, Koester JF, Cohn RC. Effects of (r,r)- and (r,r/s,s)-formoterol on airway relaxation and contraction in an experimental rat model. Curr Ther Res Clin Exp. 2007;68(4):249-61doi: 10.1016/j.curtheres.2007.08.005.
Mhanna, M. J., Koester, J. F., & Cohn, R. C. (2007). Effects of (r,r)- and (r,r/s,s)-formoterol on airway relaxation and contraction in an experimental rat model. Current therapeutic research, clinical and experimental, 68(4), 249-61. https://doi.org/10.1016/j.curtheres.2007.08.005
Mhanna, Maroun J, et al. "Effects of (r,r)- and (r,r/s,s)-formoterol on airway relaxation and contraction in an experimental rat model." Current therapeutic research, clinical and experimental vol. 68,4 (2007): 249-61. doi: https://doi.org/10.1016/j.curtheres.2007.08.005
Mhanna MJ, Koester JF, Cohn RC. Effects of (r,r)- and (r,r/s,s)-formoterol on airway relaxation and contraction in an experimental rat model. Curr Ther Res Clin Exp. 2007 Jul;68(4):249-61. doi: 10.1016/j.curtheres.2007.08.005. PMID: 24683215; PMCID: PMC3967278.
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A dual function-enabled novel zwitterion to stabilize a Pb-I framework and passivate defects for highly efficient inverted planar perovskite solar cells.

Chemical communications (Cambridge, England)

Wang D, Huang L, Chen Q, Hu L, Zeng F, Zhou X, Zhang L, Liu C, Wang X, Yan L, Xu B.
PMID: 32462166
Chem Commun (Camb). 2020 Jun 25;56(51):6929-6932. doi: 10.1039/d0cc02613a.

A novel zwitterion named bethanechol chloride (BTCC) was introduced to simultaneously stabilize a Pb-I framework and passivate defects for efficient inverted perovskite solar cells. The BTCC-assisted device yielded an elevated power conversion efficiency of 20.45% with an open-circuit voltage...

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Wang D, Huang L, Chen Q, et al. A dual function-enabled novel zwitterion to stabilize a Pb-I framework and passivate defects for highly efficient inverted planar perovskite solar cells. Chem Commun (Camb). 2020;56(51):6929-6932doi: 10.1039/d0cc02613a.
Wang, D., Huang, L., Chen, Q., Hu, L., Zeng, F., Zhou, X., Zhang, L., Liu, C., Wang, X., Yan, L., & Xu, B. (2020). A dual function-enabled novel zwitterion to stabilize a Pb-I framework and passivate defects for highly efficient inverted planar perovskite solar cells. Chemical communications (Cambridge, England), 56(51), 6929-6932. https://doi.org/10.1039/d0cc02613a
Wang, Deng, et al. "A dual function-enabled novel zwitterion to stabilize a Pb-I framework and passivate defects for highly efficient inverted planar perovskite solar cells." Chemical communications (Cambridge, England) vol. 56,51 (2020): 6929-6932. doi: https://doi.org/10.1039/d0cc02613a
Wang D, Huang L, Chen Q, Hu L, Zeng F, Zhou X, Zhang L, Liu C, Wang X, Yan L, Xu B. A dual function-enabled novel zwitterion to stabilize a Pb-I framework and passivate defects for highly efficient inverted planar perovskite solar cells. Chem Commun (Camb). 2020 Jun 25;56(51):6929-6932. doi: 10.1039/d0cc02613a. PMID: 32462166.
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Showing 1 to 12 of 32 entries