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Crk-like adapter protein is overexpressed in cervical carcinoma, facilitates proliferation, invasion and chemoresistance, and regulates Src and Akt signaling.

Oncology letters

Ji H, Li B, Zhang S, He Z, Zhou Y, Ouyang L.
PMID: 27895735
Oncol Lett. 2016 Nov;12(5):3811-3817. doi: 10.3892/ol.2016.5160. Epub 2016 Sep 21.

Overexpression of Crk-like (CrkL) adapter protein has been implicated in a number of types of human cancer. However, its involvement in human cervical carcinoma remains unclear. The present study aimed to explore the clinical significance and biological characteristics of...

Cite
Ji H, Li B, Zhang S, et al. Crk-like adapter protein is overexpressed in cervical carcinoma, facilitates proliferation, invasion and chemoresistance, and regulates Src and Akt signaling. Oncol Lett. 2016;12(5):3811-3817doi: 10.3892/ol.2016.5160.
Ji, H., Li, B., Zhang, S., He, Z., Zhou, Y., & Ouyang, L. (2016). Crk-like adapter protein is overexpressed in cervical carcinoma, facilitates proliferation, invasion and chemoresistance, and regulates Src and Akt signaling. Oncology letters, 12(5), 3811-3817. https://doi.org/10.3892/ol.2016.5160
Ji, Hong, et al. "Crk-like adapter protein is overexpressed in cervical carcinoma, facilitates proliferation, invasion and chemoresistance, and regulates Src and Akt signaling." Oncology letters vol. 12,5 (2016): 3811-3817. doi: https://doi.org/10.3892/ol.2016.5160
Ji H, Li B, Zhang S, He Z, Zhou Y, Ouyang L. Crk-like adapter protein is overexpressed in cervical carcinoma, facilitates proliferation, invasion and chemoresistance, and regulates Src and Akt signaling. Oncol Lett. 2016 Nov;12(5):3811-3817. doi: 10.3892/ol.2016.5160. Epub 2016 Sep 21. PMID: 27895735; PMCID: PMC5104173.
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CRKL overexpression promotes cell proliferation and inhibits apoptosis in endometrial carcinoma.

Oncology letters

Cai L, Wang H, Yang Q.
PMID: 28123521
Oncol Lett. 2017 Jan;13(1):51-56. doi: 10.3892/ol.2016.5394. Epub 2016 Nov 17.

The v-Crk avian sarcoma virus CT10 oncogene homolog-like (CRKL) protein is important in cancer progression. However, its expression pattern and biological roles in human endometrial carcinoma remain unexplored. The potential mechanism of CRKL-induced cancer progression is still unclear. The...

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Cai L, Wang H, Yang Q. CRKL overexpression promotes cell proliferation and inhibits apoptosis in endometrial carcinoma. Oncol Lett. 2016;13(1):51-56doi: 10.3892/ol.2016.5394.
Cai, L., Wang, H., & Yang, Q. (2017). CRKL overexpression promotes cell proliferation and inhibits apoptosis in endometrial carcinoma. Oncology letters, 13(1), 51-56. https://doi.org/10.3892/ol.2016.5394
Cai, Le, et al. "CRKL overexpression promotes cell proliferation and inhibits apoptosis in endometrial carcinoma." Oncology letters vol. 13,1 (2017): 51-56. doi: https://doi.org/10.3892/ol.2016.5394
Cai L, Wang H, Yang Q. CRKL overexpression promotes cell proliferation and inhibits apoptosis in endometrial carcinoma. Oncol Lett. 2017 Jan;13(1):51-56. doi: 10.3892/ol.2016.5394. Epub 2016 Nov 17. PMID: 28123521; PMCID: PMC5245097.
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