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[Actualities in the management of primary myelofibrosis].

Orvosi hetilap

Simon Z, Marton I, Borbényi Z, Illés Á.
PMID: 27667294
Orv Hetil. 2016 Sep;157(39):1547-1556. doi: 10.1556/650.2016.30531.

Primary myelofibrosis is one of the Philadelphia negative chronic myeloproliferative neoplasms. It is a rare disease featured by cytopenias and hepatosplenomegaly. Although the etiology of the disease is still unknown, our knowledge about its pathology and prognosis has been...

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Simon Z, Marton I, Borbényi Z, et al. Aktualitások a primer myelofibrosis ellátásában. [Actualities in the management of primary myelofibrosis]. Orv Hetil. 2016;157(39):1547-1556doi: 10.1556/650.2016.30531.
Simon, Z., Marton, I., Borbényi, Z., & Illés, �. �. (2016). Aktualitások a primer myelofibrosis ellátásában. [Actualities in the management of primary myelofibrosis]. Orvosi hetilap, 157(39), 1547-1556. https://doi.org/10.1556/650.2016.30531
Simon, Zsófia, et al. "Aktualitások a primer myelofibrosis ellátásában." [Actualities in the management of primary myelofibrosis]. Orvosi hetilap vol. 157,39 (2016): 1547-1556. doi: https://doi.org/10.1556/650.2016.30531
Simon Z, Marton I, Borbényi Z, Illés Á. Aktualitások a primer myelofibrosis ellátásában. [Actualities in the management of primary myelofibrosis]. Orv Hetil. 2016 Sep;157(39):1547-1556. doi: 10.1556/650.2016.30531. Hungarian. PMID: 27667294.
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Calreticulin (CALR) mutation in myeloproliferative neoplasms (MPNs).

Stem cell investigation

Luo W, Yu Z.
PMID: 27358884
Stem Cell Investig. 2015 Aug 31;2:16. doi: 10.3978/j.issn.2306-9759.2015.08.01. eCollection 2015.

As a heterogeneous group of disease, myeloproliferative neoplasms (MPNs) have confused hematologists and hematopathologists with their protean clinical presentations and myriads of morphologies. A thought of classifying MPNs based on molecular alterations has gained popularity because there is increasing...

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Luo W, Yu Z. Calreticulin (CALR) mutation in myeloproliferative neoplasms (MPNs). Stem Cell Investig. 2015;2:16doi: 10.3978/j.issn.2306-9759.2015.08.01.
Luo, W., & Yu, Z. (2015). Calreticulin (CALR) mutation in myeloproliferative neoplasms (MPNs). Stem cell investigation, 216. https://doi.org/10.3978/j.issn.2306-9759.2015.08.01
Luo, Wenyi, and Yu, Zhongxin. "Calreticulin (CALR) mutation in myeloproliferative neoplasms (MPNs)." Stem cell investigation vol. 2 (2015): 16. doi: https://doi.org/10.3978/j.issn.2306-9759.2015.08.01
Luo W, Yu Z. Calreticulin (CALR) mutation in myeloproliferative neoplasms (MPNs). Stem Cell Investig. 2015 Aug 31;2:16. doi: 10.3978/j.issn.2306-9759.2015.08.01. eCollection 2015. PMID: 27358884; PMCID: PMC4923638.
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PO-19 - Platelet (PLT) adhesion under flow condition in essential thrombocythemia (ET) and polycythemia vera (PV) is variably influenced according to patient mutational status.

Thrombosis research

Vignoli A, Tessarolo S, Marchetti M, Gamba S, Piras F, Finazzi G, van der Meijden PE, Swieringa F, Ten Cate H, Heemskerk JW, Rambaldi A, Falanga A.
PMID: 27161708
Thromb Res. 2016 Apr;140:S183. doi: 10.1016/S0049-3848(16)30152-9. Epub 2016 Apr 08.

INTRODUCTION: The myeloproliferative neoplasms ET and PV are characterized by a high incidence of both arterial and venous thrombosis, and/or microcirculatory disturbances. Three somatic mutations, i.e. JAK2-V617F, Calreticulin (CalR) and MPL, commonly found in these diseases, correlate with different...

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Vignoli A, Tessarolo S, Marchetti M, et al. PO-19 - Platelet (PLT) adhesion under flow condition in essential thrombocythemia (ET) and polycythemia vera (PV) is variably influenced according to patient mutational status. Thromb Res. 2016;140:S183doi: 10.1016/S0049-3848(16)30152-9.
Vignoli, A., Tessarolo, S., Marchetti, M., Gamba, S., Piras, F., Finazzi, G., van der Meijden, P. E., Swieringa, F., Ten Cate, H., Heemskerk, J. W., Rambaldi, A., & Falanga, A. (2016). PO-19 - Platelet (PLT) adhesion under flow condition in essential thrombocythemia (ET) and polycythemia vera (PV) is variably influenced according to patient mutational status. Thrombosis research, 140S183. https://doi.org/10.1016/S0049-3848(16)30152-9
Vignoli, A, et al. "PO-19 - Platelet (PLT) adhesion under flow condition in essential thrombocythemia (ET) and polycythemia vera (PV) is variably influenced according to patient mutational status." Thrombosis research vol. 140 (2016): S183. doi: https://doi.org/10.1016/S0049-3848(16)30152-9
Vignoli A, Tessarolo S, Marchetti M, Gamba S, Piras F, Finazzi G, van der Meijden PE, Swieringa F, Ten Cate H, Heemskerk JW, Rambaldi A, Falanga A. PO-19 - Platelet (PLT) adhesion under flow condition in essential thrombocythemia (ET) and polycythemia vera (PV) is variably influenced according to patient mutational status. Thromb Res. 2016 Apr;140:S183. doi: 10.1016/S0049-3848(16)30152-9. Epub 2016 Apr 08. PMID: 27161708.
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Identification of Topping Responsive Proteins in Tobacco Roots.

Frontiers in plant science

Li F, Zhang H, Wang S, Xiao W, Ding C, Liu W, Guo H.
PMID: 27200055
Front Plant Sci. 2016 Apr 28;7:582. doi: 10.3389/fpls.2016.00582. eCollection 2016.

The process of topping elicits many responses in the tobacco plant, including an increase in nicotine biosynthesis, and the secondary growth of roots. Some topping responsive miRNAs and genes have been identified in our previous study, but the mechanism...

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Li F, Zhang H, Wang S, et al. Identification of Topping Responsive Proteins in Tobacco Roots. Front Plant Sci. 2016;7:582doi: 10.3389/fpls.2016.00582.
Li, F., Zhang, H., Wang, S., Xiao, W., Ding, C., Liu, W., & Guo, H. (2016). Identification of Topping Responsive Proteins in Tobacco Roots. Frontiers in plant science, 7582. https://doi.org/10.3389/fpls.2016.00582
Li, Fei, et al. "Identification of Topping Responsive Proteins in Tobacco Roots." Frontiers in plant science vol. 7 (2016): 582. doi: https://doi.org/10.3389/fpls.2016.00582
Li F, Zhang H, Wang S, Xiao W, Ding C, Liu W, Guo H. Identification of Topping Responsive Proteins in Tobacco Roots. Front Plant Sci. 2016 Apr 28;7:582. doi: 10.3389/fpls.2016.00582. eCollection 2016. PMID: 27200055; PMCID: PMC4848317.
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Coexistence of p190 BCR/ABL Transcript and CALR 52-bp Deletion in Chronic Myeloid Leukemia Blast Crisis: A Case Report.

Mediterranean journal of hematology and infectious diseases

Seghatoleslami M, Ketabchi N, Ordo A, Asl JM, Golchin N, Saki N.
PMID: 26740863
Mediterr J Hematol Infect Dis. 2016 Jan 01;8(1):e2016002. doi: 10.4084/MJHID.2016.002. eCollection 2016.

We introduce a 78-year-old woman presented with thrombocytosis and high blast count who had a history of splenectomy. Her cytogenetic analysis revealed aberrant chromosomal rearrangements in different clonal populations harboring 46XX karyotype with t(9;22) (q34;q11). RT-PCR assay detected the...

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Seghatoleslami M, Ketabchi N, Ordo A, et al. Coexistence of p190 BCR/ABL Transcript and CALR 52-bp Deletion in Chronic Myeloid Leukemia Blast Crisis: A Case Report. Mediterr J Hematol Infect Dis. 2016;8(1):e2016002doi: 10.4084/MJHID.2016.002.
Seghatoleslami, M., Ketabchi, N., Ordo, A., Asl, J. M., Golchin, N., & Saki, N. (2016). Coexistence of p190 BCR/ABL Transcript and CALR 52-bp Deletion in Chronic Myeloid Leukemia Blast Crisis: A Case Report. Mediterranean journal of hematology and infectious diseases, 8(1), e2016002. https://doi.org/10.4084/MJHID.2016.002
Seghatoleslami, Mohammad, et al. "Coexistence of p190 BCR/ABL Transcript and CALR 52-bp Deletion in Chronic Myeloid Leukemia Blast Crisis: A Case Report." Mediterranean journal of hematology and infectious diseases vol. 8,1 (2016): e2016002. doi: https://doi.org/10.4084/MJHID.2016.002
Seghatoleslami M, Ketabchi N, Ordo A, Asl JM, Golchin N, Saki N. Coexistence of p190 BCR/ABL Transcript and CALR 52-bp Deletion in Chronic Myeloid Leukemia Blast Crisis: A Case Report. Mediterr J Hematol Infect Dis. 2016 Jan 01;8(1):e2016002. doi: 10.4084/MJHID.2016.002. eCollection 2016. PMID: 26740863; PMCID: PMC4696471.
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Development of paroxysmal nocturnal hemoglobinuria in CALR-positive myeloproliferative neoplasm.

Journal of blood medicine

Fraiman YS, Cuka N, Batista D, Vuica-Ross M, Moliterno AR.
PMID: 27313483
J Blood Med. 2016 May 30;7:107-10. doi: 10.2147/JBM.S103473. eCollection 2016.

Paroxysmal nocturnal hemoglobinuria (PNH), a disease characterized by intravascular hemolysis, thrombosis, and bone marrow failure, is associated with mutations in the PIG-A gene, resulting in a deficiency of glycosylphosphatidylinositol-anchored proteins. Many hypotheses have been posed as to whether PNH...

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Fraiman YS, Cuka N, Batista D, et al. Development of paroxysmal nocturnal hemoglobinuria in CALR-positive myeloproliferative neoplasm. J Blood Med. 2016;7:107-10doi: 10.2147/JBM.S103473.
Fraiman, Y. S., Cuka, N., Batista, D., Vuica-Ross, M., & Moliterno, A. R. (2016). Development of paroxysmal nocturnal hemoglobinuria in CALR-positive myeloproliferative neoplasm. Journal of blood medicine, 7107-10. https://doi.org/10.2147/JBM.S103473
Fraiman, Yarden S, et al. "Development of paroxysmal nocturnal hemoglobinuria in CALR-positive myeloproliferative neoplasm." Journal of blood medicine vol. 7 (2016): 107-10. doi: https://doi.org/10.2147/JBM.S103473
Fraiman YS, Cuka N, Batista D, Vuica-Ross M, Moliterno AR. Development of paroxysmal nocturnal hemoglobinuria in CALR-positive myeloproliferative neoplasm. J Blood Med. 2016 May 30;7:107-10. doi: 10.2147/JBM.S103473. eCollection 2016. PMID: 27313483; PMCID: PMC4892839.
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Dendritic cell activation and maturation induced by recombinant calreticulin fragment 39-272.

International journal of clinical and experimental medicine

Li Y, Zeng X, He L, Yuan H.
PMID: 26221268
Int J Clin Exp Med. 2015 May 15;8(5):7288-96. eCollection 2015.

Dendritic cells (DC) are the most potent antigen-presenting cells for initiating immune responses. DC maturation can be induced by exposing of immature DC to pathogen products or pro-inflammatory factor, which dramatically enhances the ability of DC to activate Ag-specific...

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Li Y, Zeng X, He L, et al. Dendritic cell activation and maturation induced by recombinant calreticulin fragment 39-272. Int J Clin Exp Med. 2015;8(5):7288-96
Li, Y., Zeng, X., He, L., & Yuan, H. (2015). Dendritic cell activation and maturation induced by recombinant calreticulin fragment 39-272. International journal of clinical and experimental medicine, 8(5), 7288-96.
Li, Yue, et al. "Dendritic cell activation and maturation induced by recombinant calreticulin fragment 39-272." International journal of clinical and experimental medicine vol. 8,5 (2015): 7288-96.
Li Y, Zeng X, He L, Yuan H. Dendritic cell activation and maturation induced by recombinant calreticulin fragment 39-272. Int J Clin Exp Med. 2015 May 15;8(5):7288-96. eCollection 2015. PMID: 26221268; PMCID: PMC4509213.
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Capsaicin induces immunogenic cell death in human osteosarcoma cells.

Experimental and therapeutic medicine

Jin T, Wu H, Wang Y, Peng H.
PMID: 27446273
Exp Ther Med. 2016 Aug;12(2):765-770. doi: 10.3892/etm.2016.3368. Epub 2016 May 19.

Immunogenic cell death (ICD) is characterized by the early surface exposure of calreticulin (CRT). As a specific signaling molecule, CRT on the surface of apoptotic tumor cells mediates the recognition and phagocytosis of tumor cells by antigen presenting cells....

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Jin T, Wu H, Wang Y, et al. Capsaicin induces immunogenic cell death in human osteosarcoma cells. Exp Ther Med. 2016;12(2):765-770doi: 10.3892/etm.2016.3368.
Jin, T., Wu, H., Wang, Y., & Peng, H. (2016). Capsaicin induces immunogenic cell death in human osteosarcoma cells. Experimental and therapeutic medicine, 12(2), 765-770. https://doi.org/10.3892/etm.2016.3368
Jin, Tao, et al. "Capsaicin induces immunogenic cell death in human osteosarcoma cells." Experimental and therapeutic medicine vol. 12,2 (2016): 765-770. doi: https://doi.org/10.3892/etm.2016.3368
Jin T, Wu H, Wang Y, Peng H. Capsaicin induces immunogenic cell death in human osteosarcoma cells. Exp Ther Med. 2016 Aug;12(2):765-770. doi: 10.3892/etm.2016.3368. Epub 2016 May 19. PMID: 27446273; PMCID: PMC4950522.
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Meloidogyne incognita - rice (Oryza sativa) interaction: a new model system to study plant-root-knot nematode interactions in monocotyledons.

Rice (New York, N.Y.)

Nguyễn PV, Bellafiore S, Petitot AS, Haidar R, Bak A, Abed A, Gantet P, Mezzalira I, de Almeida Engler J, Fernandez D.
PMID: 26224554
Rice (N Y). 2014 Dec;7(1):23. doi: 10.1186/s12284-014-0023-4. Epub 2014 Sep 22.

BACKGROUND: Plant-parasitic nematodes developed strategies to invade and colonize their host plants, including expression of immune suppressors to overcome host defenses. Meloidogyne graminicola and M. incognita are root-knot nematode (RKN) species reported to damage rice (Oryza sativa L.) cultivated...

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Nguyễn PV, Bellafiore S, Petitot AS, et al. Meloidogyne incognita - rice (Oryza sativa) interaction: a new model system to study plant-root-knot nematode interactions in monocotyledons. Rice (N Y). 2014;7(1):23doi: 10.1186/s12284-014-0023-4.
Nguyễn, P. V., Bellafiore, S., Petitot, A. S., Haidar, R., Bak, A., Abed, A., Gantet, P., Mezzalira, I., de Almeida Engler, J., & Fernandez, D. (2014). Meloidogyne incognita - rice (Oryza sativa) interaction: a new model system to study plant-root-knot nematode interactions in monocotyledons. Rice (New York, N.Y.), 7(1), 23. https://doi.org/10.1186/s12284-014-0023-4
Nguyễn, Phong Vũ, et al. "Meloidogyne incognita - rice (Oryza sativa) interaction: a new model system to study plant-root-knot nematode interactions in monocotyledons." Rice (New York, N.Y.) vol. 7,1 (2014): 23. doi: https://doi.org/10.1186/s12284-014-0023-4
Nguyễn PV, Bellafiore S, Petitot AS, Haidar R, Bak A, Abed A, Gantet P, Mezzalira I, de Almeida Engler J, Fernandez D. Meloidogyne incognita - rice (Oryza sativa) interaction: a new model system to study plant-root-knot nematode interactions in monocotyledons. Rice (N Y). 2014 Dec;7(1):23. doi: 10.1186/s12284-014-0023-4. Epub 2014 Sep 22. PMID: 26224554; PMCID: PMC4884005.
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Coexisting JAK2V617F and CALR Exon 9 Mutation in Essential Thrombocythemia.

Indian journal of hematology & blood transfusion : an official journal of Indian Society of Hematology and Blood Transfusion

Rashid M, Ahmed RZ, Ahmed S, Nadeem M, Ahmed N, Shamsi TS.
PMID: 27408370
Indian J Hematol Blood Transfus. 2016 Jun;32:112-116. doi: 10.1007/s12288-016-0658-y. Epub 2016 Feb 09.

Classic "BCR-ABL1-negative" MPN is an operational sub-category of MPN that includes polycythemia vera (PV), essential thrombocythemia (ET), and primary myelofibrosis (PMF) harboring JAK2V617F as the most common mutation. JAK2V617F can be detected in about 95 % of patients with...

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Rashid M, Ahmed RZ, Ahmed S, et al. Coexisting JAK2V617F and CALR Exon 9 Mutation in Essential Thrombocythemia. Indian J Hematol Blood Transfus. 2016;32:112-116doi: 10.1007/s12288-016-0658-y.
Rashid, M., Ahmed, R. Z., Ahmed, S., Nadeem, M., Ahmed, N., & Shamsi, T. S. (2016). Coexisting JAK2V617F and CALR Exon 9 Mutation in Essential Thrombocythemia. Indian journal of hematology & blood transfusion : an official journal of Indian Society of Hematology and Blood Transfusion, 32112-116. https://doi.org/10.1007/s12288-016-0658-y
Rashid, Munazza, et al. "Coexisting JAK2V617F and CALR Exon 9 Mutation in Essential Thrombocythemia." Indian journal of hematology & blood transfusion : an official journal of Indian Society of Hematology and Blood Transfusion vol. 32 (2016): 112-116. doi: https://doi.org/10.1007/s12288-016-0658-y
Rashid M, Ahmed RZ, Ahmed S, Nadeem M, Ahmed N, Shamsi TS. Coexisting JAK2V617F and CALR Exon 9 Mutation in Essential Thrombocythemia. Indian J Hematol Blood Transfus. 2016 Jun;32:112-116. doi: 10.1007/s12288-016-0658-y. Epub 2016 Feb 09. PMID: 27408370; PMCID: PMC4925557.
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Myeloproliferative neoplasms working group consensus recommendations for diagnosis and management of primary myelofibrosis, polycythemia vera, and essential thrombocythemia.

Indian journal of medical and paediatric oncology : official journal of Indian Society of Medical & Paediatric Oncology

Agarwal MB, Malhotra H, Chakrabarti P, Varma N, Mathews V, Bhattacharyya J, Seth T, Gayathri K, Menon H, Subramanian PG, Sharma A, Bhattacharyya M, Mehta J, Vaid AK, Shah S, Aggarwal S, Gogoi PK, Nair R, Agarwal U, Varma S, Prasad SV, Manipadam MT.
PMID: 25810569
Indian J Med Paediatr Oncol. 2015 Jan-Mar;36(1):3-16. doi: 10.4103/0971-5851.151770.

According to the 2008 revision of the World Health Organization (WHO) classification of myeloid malignancies, philadelphia chromosome (Ph)-negative myeloproliferative neoplasms (MPNs) include clonal, hematologic disorders such as polycythemia vera, primary myelofibrosis, and essential thrombocythemia.Recent years have witnessed major advances...

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Agarwal MB, Malhotra H, Chakrabarti P, et al. Myeloproliferative neoplasms working group consensus recommendations for diagnosis and management of primary myelofibrosis, polycythemia vera, and essential thrombocythemia. Indian J Med Paediatr Oncol. 2015;36(1):3-16doi: 10.4103/0971-5851.151770.
Agarwal, M. B., Malhotra, H., Chakrabarti, P., Varma, N., Mathews, V., Bhattacharyya, J., Seth, T., Gayathri, K., Menon, H., Subramanian, P. G., Sharma, A., Bhattacharyya, M., Mehta, J., Vaid, A. K., Shah, S., Aggarwal, S., Gogoi, P. K., Nair, R., Agarwal, U., Varma, S., Prasad, S. V., & Manipadam, M. T. (2015). Myeloproliferative neoplasms working group consensus recommendations for diagnosis and management of primary myelofibrosis, polycythemia vera, and essential thrombocythemia. Indian journal of medical and paediatric oncology : official journal of Indian Society of Medical & Paediatric Oncology, 36(1), 3-16. https://doi.org/10.4103/0971-5851.151770
Agarwal, M B, et al. "Myeloproliferative neoplasms working group consensus recommendations for diagnosis and management of primary myelofibrosis, polycythemia vera, and essential thrombocythemia." Indian journal of medical and paediatric oncology : official journal of Indian Society of Medical & Paediatric Oncology vol. 36,1 (2015): 3-16. doi: https://doi.org/10.4103/0971-5851.151770
Agarwal MB, Malhotra H, Chakrabarti P, Varma N, Mathews V, Bhattacharyya J, Seth T, Gayathri K, Menon H, Subramanian PG, Sharma A, Bhattacharyya M, Mehta J, Vaid AK, Shah S, Aggarwal S, Gogoi PK, Nair R, Agarwal U, Varma S, Prasad SV, Manipadam MT. Myeloproliferative neoplasms working group consensus recommendations for diagnosis and management of primary myelofibrosis, polycythemia vera, and essential thrombocythemia. Indian J Med Paediatr Oncol. 2015 Jan-Mar;36(1):3-16. doi: 10.4103/0971-5851.151770. PMID: 25810569; PMCID: PMC4363847.
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Zinc supplementation is required for the cytotoxic and immunogenic effects of chemotherapy in chemoresistant p53-functionally deficient cells.

Oncoimmunology

Cirone M, Garufi A, Di Renzo L, Granato M, Faggioni A, D'Orazi G.
PMID: 24228232
Oncoimmunology. 2013 Sep 01;2(9):e26198. doi: 10.4161/onci.26198. Epub 2013 Sep 12.

Optimal tumor eradication often results from the death of malignant cells, as induced by chemotherapeutic agents, coupled to the induction of antitumor immune responses. However, cancer cells frequently become resistant to the cytotoxic activity of chemotherapy. The aim of...

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Cirone M, Garufi A, Di Renzo L, et al. Zinc supplementation is required for the cytotoxic and immunogenic effects of chemotherapy in chemoresistant p53-functionally deficient cells. Oncoimmunology. 2013;2(9):e26198doi: 10.4161/onci.26198.
Cirone, M., Garufi, A., Di Renzo, L., Granato, M., Faggioni, A., & D'Orazi, G. (2013). Zinc supplementation is required for the cytotoxic and immunogenic effects of chemotherapy in chemoresistant p53-functionally deficient cells. Oncoimmunology, 2(9), e26198. https://doi.org/10.4161/onci.26198
Cirone, Mara, et al. "Zinc supplementation is required for the cytotoxic and immunogenic effects of chemotherapy in chemoresistant p53-functionally deficient cells." Oncoimmunology vol. 2,9 (2013): e26198. doi: https://doi.org/10.4161/onci.26198
Cirone M, Garufi A, Di Renzo L, Granato M, Faggioni A, D'Orazi G. Zinc supplementation is required for the cytotoxic and immunogenic effects of chemotherapy in chemoresistant p53-functionally deficient cells. Oncoimmunology. 2013 Sep 01;2(9):e26198. doi: 10.4161/onci.26198. Epub 2013 Sep 12. PMID: 24228232; PMCID: PMC3820813.
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