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The effect of folate supplementation and genotype on cardiovascular and epigenetic measures in schizophrenia subjects.

NPJ schizophrenia

Ellingrod VL, Grove TB, Burghardt KJ, Taylor SF, Dalack G.
PMID: 27336047
NPJ Schizophr. 2015 Nov 11;1:15046. doi: 10.1038/npjschz.2015.46. eCollection 2015.

BACKGROUND: Metabolic syndrome may be related to folate's pharmacogenetically regulated metabolism and atypical antipsychotic (AAP) exposure.AIMS: We examined folate supplementation on metabolic measures, endothelial functioning (Reactive Hyperemia Index (RHI)), and global methylation in AAP-treated schizophrenia subjects meeting NCEP-ATP-III-a metabolic...

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Ellingrod VL, Grove TB, Burghardt KJ, et al. The effect of folate supplementation and genotype on cardiovascular and epigenetic measures in schizophrenia subjects. NPJ Schizophr. 2015;1:15046doi: 10.1038/npjschz.2015.46.
Ellingrod, V. L., Grove, T. B., Burghardt, K. J., Taylor, S. F., & Dalack, G. (2015). The effect of folate supplementation and genotype on cardiovascular and epigenetic measures in schizophrenia subjects. NPJ schizophrenia, 115046. https://doi.org/10.1038/npjschz.2015.46
Ellingrod, Vicki L, et al. "The effect of folate supplementation and genotype on cardiovascular and epigenetic measures in schizophrenia subjects." NPJ schizophrenia vol. 1 (2015): 15046. doi: https://doi.org/10.1038/npjschz.2015.46
Ellingrod VL, Grove TB, Burghardt KJ, Taylor SF, Dalack G. The effect of folate supplementation and genotype on cardiovascular and epigenetic measures in schizophrenia subjects. NPJ Schizophr. 2015 Nov 11;1:15046. doi: 10.1038/npjschz.2015.46. eCollection 2015. PMID: 27336047; PMCID: PMC4849464.
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Association between the COMT 158 G/A polymorphism and lung cancer risk: a meta-analysis.

International journal of clinical and experimental medicine

Peng S, Tong X, Liu S, Feng Y, Fan H.
PMID: 26770364
Int J Clin Exp Med. 2015 Oct 15;8(10):17739-47. eCollection 2015.

Catechol-O-methyltransferase (COMT) 158 G/A gene polymorphism seem to associate with lung cancer, but the results are inconclusive. This meta-analysis aims to investigate the association between COMT 158 G/A gene polymorphism and lung cancer susceptibility. We searched PubMed, Embase, China...

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Peng S, Tong X, Liu S, et al. Association between the COMT 158 G/A polymorphism and lung cancer risk: a meta-analysis. Int J Clin Exp Med. 2015;8(10):17739-47
Peng, S., Tong, X., Liu, S., Feng, Y., & Fan, H. (2015). Association between the COMT 158 G/A polymorphism and lung cancer risk: a meta-analysis. International journal of clinical and experimental medicine, 8(10), 17739-47.
Peng, Shifeng, et al. "Association between the COMT 158 G/A polymorphism and lung cancer risk: a meta-analysis." International journal of clinical and experimental medicine vol. 8,10 (2015): 17739-47.
Peng S, Tong X, Liu S, Feng Y, Fan H. Association between the COMT 158 G/A polymorphism and lung cancer risk: a meta-analysis. Int J Clin Exp Med. 2015 Oct 15;8(10):17739-47. eCollection 2015. PMID: 26770364; PMCID: PMC4694264.
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Dynamic changes of five neurotransmitters and their related enzymes in various rat tissues following β-asarone and levodopa co-administration.

Experimental and therapeutic medicine

Huang L, Deng M, Fang Y, Li L.
PMID: 26622527
Exp Ther Med. 2015 Oct;10(4):1566-1572. doi: 10.3892/etm.2015.2704. Epub 2015 Aug 24.

The aim of the present study was to investigate the dynamic changes of five neurotransmitters and their associated enzymes in the rat plasma and brain tissues following the co-administration of β-asarone and levodopa (L-dopa). The rats were divided into...

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Huang L, Deng M, Fang Y, et al. Dynamic changes of five neurotransmitters and their related enzymes in various rat tissues following β-asarone and levodopa co-administration. Exp Ther Med. 2015;10(4):1566-1572doi: 10.3892/etm.2015.2704.
Huang, L., Deng, M., Fang, Y., & Li, L. (2015). Dynamic changes of five neurotransmitters and their related enzymes in various rat tissues following β-asarone and levodopa co-administration. Experimental and therapeutic medicine, 10(4), 1566-1572. https://doi.org/10.3892/etm.2015.2704
Huang, Liping, et al. "Dynamic changes of five neurotransmitters and their related enzymes in various rat tissues following β-asarone and levodopa co-administration." Experimental and therapeutic medicine vol. 10,4 (2015): 1566-1572. doi: https://doi.org/10.3892/etm.2015.2704
Huang L, Deng M, Fang Y, Li L. Dynamic changes of five neurotransmitters and their related enzymes in various rat tissues following β-asarone and levodopa co-administration. Exp Ther Med. 2015 Oct;10(4):1566-1572. doi: 10.3892/etm.2015.2704. Epub 2015 Aug 24. PMID: 26622527; PMCID: PMC4578043.
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The brain-derived neurotrophic factor (BDNF) val66met polymorphism differentially affects performance on subscales of the Wechsler Memory Scale - Third Edition (WMS-III).

Frontiers in psychology

Lamb YN, Thompson CS, McKay NS, Waldie KE, Kirk IJ.
PMID: 26347681
Front Psychol. 2015 Aug 17;6:1212. doi: 10.3389/fpsyg.2015.01212. eCollection 2015.

Single nucleotide polymorphisms in the brain-derived neurotrophic factor (BDNF) gene and the catechol-O-methyltransferase (COMT) gene influence brain structure and function, as well as cognitive abilities. They are most influential in the hippocampus and prefrontal cortex (PFC), respectively. Recall and...

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Lamb YN, Thompson CS, McKay NS, et al. The brain-derived neurotrophic factor (BDNF) val66met polymorphism differentially affects performance on subscales of the Wechsler Memory Scale - Third Edition (WMS-III). Front Psychol. 2015;6:1212doi: 10.3389/fpsyg.2015.01212.
Lamb, Y. N., Thompson, C. S., McKay, N. S., Waldie, K. E., & Kirk, I. J. (2015). The brain-derived neurotrophic factor (BDNF) val66met polymorphism differentially affects performance on subscales of the Wechsler Memory Scale - Third Edition (WMS-III). Frontiers in psychology, 61212. https://doi.org/10.3389/fpsyg.2015.01212
Lamb, Yvette N, et al. "The brain-derived neurotrophic factor (BDNF) val66met polymorphism differentially affects performance on subscales of the Wechsler Memory Scale - Third Edition (WMS-III)." Frontiers in psychology vol. 6 (2015): 1212. doi: https://doi.org/10.3389/fpsyg.2015.01212
Lamb YN, Thompson CS, McKay NS, Waldie KE, Kirk IJ. The brain-derived neurotrophic factor (BDNF) val66met polymorphism differentially affects performance on subscales of the Wechsler Memory Scale - Third Edition (WMS-III). Front Psychol. 2015 Aug 17;6:1212. doi: 10.3389/fpsyg.2015.01212. eCollection 2015. PMID: 26347681; PMCID: PMC4538220.
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Hydroxylation and sulfation of sex steroid hormones in inflammatory liver.

Journal of biomedical research

Lee SR, Lee SY, Kim SY, Ryu SY, Park BK, Hong EJ.
PMID: 28866654
J Biomed Res. 2017 Sep 26;31(5):437-444. doi: 10.7555/JBR.31.20170031.

Sex steroids, also known as gonadal steroids, are oxidized with hydroxylation by cytochrome P450, glucuronidation by UDP-glucuronosyltransferase, sulfation by sulfotransferase, andO-methylation by catechol O-methyltransferase. Thus, it is important to determine the process by which inflammation influences metabolism of gonadal...

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Lee SR, Lee SY, Kim SY, et al. Hydroxylation and sulfation of sex steroid hormones in inflammatory liver. J Biomed Res. 2017;31(5):437-444doi: 10.7555/JBR.31.20170031.
Lee, S. R., Lee, S. Y., Kim, S. Y., Ryu, S. Y., Park, B. K., & Hong, E. J. (2017). Hydroxylation and sulfation of sex steroid hormones in inflammatory liver. Journal of biomedical research, 31(5), 437-444. https://doi.org/10.7555/JBR.31.20170031
Lee, Sang R, et al. "Hydroxylation and sulfation of sex steroid hormones in inflammatory liver." Journal of biomedical research vol. 31,5 (2017): 437-444. doi: https://doi.org/10.7555/JBR.31.20170031
Lee SR, Lee SY, Kim SY, Ryu SY, Park BK, Hong EJ. Hydroxylation and sulfation of sex steroid hormones in inflammatory liver. J Biomed Res. 2017 Sep 26;31(5):437-444. doi: 10.7555/JBR.31.20170031. PMID: 28866654; PMCID: PMC5706436.
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[Duration of therapeutic remission alcohol dependence: a role of dopamine system genes polymorphism and family history density].

Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova

Kibitov AO, Chuprova NA, Brodyansky VM, Voskoboeva EY.
PMID: 26288303
Zh Nevrol Psikhiatr Im S S Korsakova. 2015;115(4):51-58. doi: 10.17116/jnevro20151154251-58.

AIM: A quantitative assessment of the impact of genetic factors (density of family history of alcohol dependence and dopamine system genes polymorphisms) on the average time to relapse (ATR) after alcohol dependence treatment (duration of therapeutic remission from alcohol...

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Kibitov AO, Chuprova NA, Brodyansky VM, et al. Dlitel'nost' terapevticheskoĭ remissii u bol'nykh s alkogol'noĭ zavisimost'iu: rol' polimorfizma genov dofaminovoĭ sistemy i stepeni semeĭnoĭ otiagoshchennosti. [Duration of therapeutic remission alcohol dependence: a role of dopamine system genes polymorphism and family history density]. Zh Nevrol Psikhiatr Im S S Korsakova. 2015;115(4):51-58doi: 10.17116/jnevro20151154251-58.
Kibitov, A. O., Chuprova, N. A., Brodyansky, V. M., & Voskoboeva, E. Y. (2015). Dlitel'nost' terapevticheskoĭ remissii u bol'nykh s alkogol'noĭ zavisimost'iu: rol' polimorfizma genov dofaminovoĭ sistemy i stepeni semeĭnoĭ otiagoshchennosti. [Duration of therapeutic remission alcohol dependence: a role of dopamine system genes polymorphism and family history density]. Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova, 115(4), 51-58. https://doi.org/10.17116/jnevro20151154251-58
Kibitov, A O, et al. "Dlitel'nost' terapevticheskoĭ remissii u bol'nykh s alkogol'noĭ zavisimost'iu: rol' polimorfizma genov dofaminovoĭ sistemy i stepeni semeĭnoĭ otiagoshchennosti." [Duration of therapeutic remission alcohol dependence: a role of dopamine system genes polymorphism and family history density]. Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova vol. 115,4 (2015): 51-58. doi: https://doi.org/10.17116/jnevro20151154251-58
Kibitov AO, Chuprova NA, Brodyansky VM, Voskoboeva EY. Dlitel'nost' terapevticheskoĭ remissii u bol'nykh s alkogol'noĭ zavisimost'iu: rol' polimorfizma genov dofaminovoĭ sistemy i stepeni semeĭnoĭ otiagoshchennosti. [Duration of therapeutic remission alcohol dependence: a role of dopamine system genes polymorphism and family history density]. Zh Nevrol Psikhiatr Im S S Korsakova. 2015;115(4):51-58. doi: 10.17116/jnevro20151154251-58. Russian. PMID: 26288303.
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Influence of Equatorial CH⋅⋅⋅O Interactions on Secondary Kinetic Isotope Effects for Methyl Transfer.

Angewandte Chemie (International ed. in English)

Wilson PB, Williams IH.
PMID: 26823274
Angew Chem Int Ed Engl. 2016 Feb 24;55(9):3192-5. doi: 10.1002/anie.201511708. Epub 2016 Jan 28.

DFT calculations for methyl cation complexed within a constrained cage of water molecules permit the controlled manipulation of the "axial" donor/acceptor distance and the "equatorial" distance to hydrogen-bond acceptors. The kinetic isotope effect k(CH3)/k(CT3) for methyl transfer within a...

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Wilson PB, Williams IH. Influence of Equatorial CH⋅⋅⋅O Interactions on Secondary Kinetic Isotope Effects for Methyl Transfer. Angew Chem Int Ed Engl. 2016;55(9):3192-5doi: 10.1002/anie.201511708.
Wilson, P. B., & Williams, I. H. (2016). Influence of Equatorial CH⋅⋅⋅O Interactions on Secondary Kinetic Isotope Effects for Methyl Transfer. Angewandte Chemie (International ed. in English), 55(9), 3192-5. https://doi.org/10.1002/anie.201511708
Wilson, Philippe B, and Williams, Ian H. "Influence of Equatorial CH⋅⋅⋅O Interactions on Secondary Kinetic Isotope Effects for Methyl Transfer." Angewandte Chemie (International ed. in English) vol. 55,9 (2016): 3192-5. doi: https://doi.org/10.1002/anie.201511708
Wilson PB, Williams IH. Influence of Equatorial CH⋅⋅⋅O Interactions on Secondary Kinetic Isotope Effects for Methyl Transfer. Angew Chem Int Ed Engl. 2016 Feb 24;55(9):3192-5. doi: 10.1002/anie.201511708. Epub 2016 Jan 28. PMID: 26823274; PMCID: PMC4770435.
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Mercury promotes catecholamines which potentiate mercurial autoimmunity and vasodilation: implications for inositol 1,4,5-triphosphate 3-kinase C susceptibility in kawasaki syndrome.

Korean circulation journal

Yeter D, Deth R, Kuo HC.
PMID: 24174958
Korean Circ J. 2013 Sep;43(9):581-91. doi: 10.4070/kcj.2013.43.9.581.

Previously, we reviewed biological evidence that mercury could induce autoimmunity and coronary arterial wall relaxation as observed in Kawasaki syndrome (KS) through its effects on calcium signaling, and that inositol 1,4,5-triphosphate 3-kinase C (ITPKC) susceptibility in KS would predispose...

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Yeter D, Deth R, Kuo HC. Mercury promotes catecholamines which potentiate mercurial autoimmunity and vasodilation: implications for inositol 1,4,5-triphosphate 3-kinase C susceptibility in kawasaki syndrome. Korean Circ J. 2013;43(9):581-91doi: 10.4070/kcj.2013.43.9.581.
Yeter, D., Deth, R., & Kuo, H. C. (2013). Mercury promotes catecholamines which potentiate mercurial autoimmunity and vasodilation: implications for inositol 1,4,5-triphosphate 3-kinase C susceptibility in kawasaki syndrome. Korean circulation journal, 43(9), 581-91. https://doi.org/10.4070/kcj.2013.43.9.581
Yeter, Deniz, et al. "Mercury promotes catecholamines which potentiate mercurial autoimmunity and vasodilation: implications for inositol 1,4,5-triphosphate 3-kinase C susceptibility in kawasaki syndrome." Korean circulation journal vol. 43,9 (2013): 581-91. doi: https://doi.org/10.4070/kcj.2013.43.9.581
Yeter D, Deth R, Kuo HC. Mercury promotes catecholamines which potentiate mercurial autoimmunity and vasodilation: implications for inositol 1,4,5-triphosphate 3-kinase C susceptibility in kawasaki syndrome. Korean Circ J. 2013 Sep;43(9):581-91. doi: 10.4070/kcj.2013.43.9.581. PMID: 24174958; PMCID: PMC3808853.
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Lack of association between COMT gene and deficit/nondeficit schizophrenia.

Behavioral and brain functions : BBF

Wonodi I, Mitchell BD, Stine OC, Hong LE, Elliott A, Kirkpatrick B, Carpenter WT, Thaker GK, Buchanan RW.
PMID: 17173666
Behav Brain Funct. 2006 Dec 15;2:42. doi: 10.1186/1744-9081-2-42.

BACKGROUND: The dopamine dysregulation hypothesis of schizophrenia posits that positive, negative and cognitive symptoms correlate with cortical/subcortical imbalances in dopaminergic transmission. A functional polymorphism (Val158Met) in the catechol-O-methyltransferase (COMT) gene is implicated in the pathophysiology of schizophrenia by its...

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Wonodi I, Mitchell BD, Stine OC, et al. Lack of association between COMT gene and deficit/nondeficit schizophrenia. Behav Brain Funct. 2006;2:42doi: 10.1186/1744-9081-2-42.
Wonodi, I., Mitchell, B. D., Stine, O. C., Hong, L. E., Elliott, A., Kirkpatrick, B., Carpenter, W. T., Thaker, G. K., & Buchanan, R. W. (2006). Lack of association between COMT gene and deficit/nondeficit schizophrenia. Behavioral and brain functions : BBF, 242. https://doi.org/10.1186/1744-9081-2-42
Wonodi, Ikwunga, et al. "Lack of association between COMT gene and deficit/nondeficit schizophrenia." Behavioral and brain functions : BBF vol. 2 (2006): 42. doi: https://doi.org/10.1186/1744-9081-2-42
Wonodi I, Mitchell BD, Stine OC, Hong LE, Elliott A, Kirkpatrick B, Carpenter WT, Thaker GK, Buchanan RW. Lack of association between COMT gene and deficit/nondeficit schizophrenia. Behav Brain Funct. 2006 Dec 15;2:42. doi: 10.1186/1744-9081-2-42. PMID: 17173666; PMCID: PMC1716164.
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Genetic contributions to age-related decline in executive function: a 10-year longitudinal study of COMT and BDNF polymorphisms.

Frontiers in human neuroscience

Erickson KI, Kim JS, Suever BL, Voss MW, Francis BM, Kramer AF.
PMID: 18958211
Front Hum Neurosci. 2008 Sep 23;2:11. doi: 10.3389/neuro.09.011.2008. eCollection 2008.

Genetic variability in the dopaminergic and neurotrophic systems could contribute to age-related impairments in executive control and memory function. In this study we examined whether genetic polymorphisms for catechol-O-methyltransferase (COMT) and brain-derived neurotrophic factor (BDNF) were related to the...

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Erickson KI, Kim JS, Suever BL, et al. Genetic contributions to age-related decline in executive function: a 10-year longitudinal study of COMT and BDNF polymorphisms. Front Hum Neurosci. 2008;2:11doi: 10.3389/neuro.09.011.2008.
Erickson, K. I., Kim, J. S., Suever, B. L., Voss, M. W., Francis, B. M., & Kramer, A. F. (2008). Genetic contributions to age-related decline in executive function: a 10-year longitudinal study of COMT and BDNF polymorphisms. Frontiers in human neuroscience, 211. https://doi.org/10.3389/neuro.09.011.2008
Erickson, Kirk I, et al. "Genetic contributions to age-related decline in executive function: a 10-year longitudinal study of COMT and BDNF polymorphisms." Frontiers in human neuroscience vol. 2 (2008): 11. doi: https://doi.org/10.3389/neuro.09.011.2008
Erickson KI, Kim JS, Suever BL, Voss MW, Francis BM, Kramer AF. Genetic contributions to age-related decline in executive function: a 10-year longitudinal study of COMT and BDNF polymorphisms. Front Hum Neurosci. 2008 Sep 23;2:11. doi: 10.3389/neuro.09.011.2008. eCollection 2008. PMID: 18958211; PMCID: PMC2572207.
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COMT Val158Met Polymorphism Modulates Cognitive Effects of Dietary Intervention.

Frontiers in aging neuroscience

Witte AV, Jansen S, Schirmacher A, Young P, Flöel A.
PMID: 21119769
Front Aging Neurosci. 2010 Nov 05;2:146. doi: 10.3389/fnagi.2010.00146. eCollection 2010.

A common single nucleotide polymorphism (SNP) in the gene encoding catechol-O-methyltransferase (COMT), Val158Met, is thought to influence cognitive performance due to differences in prefrontal dopaminergic neurotransmission. Previous studies lend support for the hypothesis that the "at risk" genotype comprising...

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Witte AV, Jansen S, Schirmacher A, et al. COMT Val158Met Polymorphism Modulates Cognitive Effects of Dietary Intervention. Front Aging Neurosci. 2010;2:146doi: 10.3389/fnagi.2010.00146.
Witte, A. V., Jansen, S., Schirmacher, A., Young, P., & Flöel, A. (2010). COMT Val158Met Polymorphism Modulates Cognitive Effects of Dietary Intervention. Frontiers in aging neuroscience, 2146. https://doi.org/10.3389/fnagi.2010.00146
Witte, Anja Veronica, et al. "COMT Val158Met Polymorphism Modulates Cognitive Effects of Dietary Intervention." Frontiers in aging neuroscience vol. 2 (2010): 146. doi: https://doi.org/10.3389/fnagi.2010.00146
Witte AV, Jansen S, Schirmacher A, Young P, Flöel A. COMT Val158Met Polymorphism Modulates Cognitive Effects of Dietary Intervention. Front Aging Neurosci. 2010 Nov 05;2:146. doi: 10.3389/fnagi.2010.00146. eCollection 2010. PMID: 21119769; PMCID: PMC2990456.
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Polymorphism of the catechol-O-methyltransferase gene in Han Chinese patients with psoriasis vulgaris.

Genetics and molecular biology

Gao L, Li H, Li K, Shen Z, Liu L, Li C, Zhang Z, Liu Y.
PMID: 21637643
Genet Mol Biol. 2009 Jan;32(1):32-6. doi: 10.1590/S1415-47572009005000002. Epub 2009 Jan 10.

PSORIASIS VULGARIS IS DEFINED BY A SERIES OF LINKED CELLULAR CHANGES IN THE SKIN: hyperplasia of epidermal keratinocytes, vascular hyperplasia and ectasia, and infiltration of T lymphocytes, neutrophils and other types of leukocytes in the affected skin. Catechol-O-methyltransferase (COMT)...

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Gao L, Li H, Li K, et al. Polymorphism of the catechol-O-methyltransferase gene in Han Chinese patients with psoriasis vulgaris. Genet Mol Biol. 2009;32(1):32-6doi: 10.1590/S1415-47572009005000002.
Gao, L., Li, H., Li, K., Shen, Z., Liu, L., Li, C., Zhang, Z., & Liu, Y. (2009). Polymorphism of the catechol-O-methyltransferase gene in Han Chinese patients with psoriasis vulgaris. Genetics and molecular biology, 32(1), 32-6. https://doi.org/10.1590/S1415-47572009005000002
Gao, Lin, et al. "Polymorphism of the catechol-O-methyltransferase gene in Han Chinese patients with psoriasis vulgaris." Genetics and molecular biology vol. 32,1 (2009): 32-6. doi: https://doi.org/10.1590/S1415-47572009005000002
Gao L, Li H, Li K, Shen Z, Liu L, Li C, Zhang Z, Liu Y. Polymorphism of the catechol-O-methyltransferase gene in Han Chinese patients with psoriasis vulgaris. Genet Mol Biol. 2009 Jan;32(1):32-6. doi: 10.1590/S1415-47572009005000002. Epub 2009 Jan 10. PMID: 21637643; PMCID: PMC3032959.
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Showing 1 to 12 of 258 entries