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Showing 1 to 12 of 96 entries
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Constitutive renal Rel/nuclear factor-κB expression in Lewis polycystic kidney disease rats.

World journal of nephrology

Ta MH, Schwensen KG, Liuwantara D, Huso DL, Watnick T, Rangan GK.
PMID: 27458563
World J Nephrol. 2016 Jul 06;5(4):339-57. doi: 10.5527/wjn.v5.i4.339.

AIM: To determine the temporal expression and pattern of Rel/nuclear factor (NF)-κB proteins in renal tissue in polycystic kidney disease (PKD).METHODS: The renal expression of Rel/NF-κB proteins was determined by immunohistochemistry, immunofluorescence and immunoblot analysis in Lewis polycystic kidney...

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Ta MH, Schwensen KG, Liuwantara D, et al. Constitutive renal Rel/nuclear factor-κB expression in Lewis polycystic kidney disease rats. World J Nephrol. 2016;5(4):339-57doi: 10.5527/wjn.v5.i4.339.
Ta, M. H., Schwensen, K. G., Liuwantara, D., Huso, D. L., Watnick, T., & Rangan, G. K. (2016). Constitutive renal Rel/nuclear factor-κB expression in Lewis polycystic kidney disease rats. World journal of nephrology, 5(4), 339-57. https://doi.org/10.5527/wjn.v5.i4.339
Ta, Michelle H T, et al. "Constitutive renal Rel/nuclear factor-κB expression in Lewis polycystic kidney disease rats." World journal of nephrology vol. 5,4 (2016): 339-57. doi: https://doi.org/10.5527/wjn.v5.i4.339
Ta MH, Schwensen KG, Liuwantara D, Huso DL, Watnick T, Rangan GK. Constitutive renal Rel/nuclear factor-κB expression in Lewis polycystic kidney disease rats. World J Nephrol. 2016 Jul 06;5(4):339-57. doi: 10.5527/wjn.v5.i4.339. PMID: 27458563; PMCID: PMC4936341.
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Systemic inflammation impairs attention and cognitive flexibility but not associative learning in aged rats: possible implications for delirium.

Frontiers in aging neuroscience

Culley DJ, Snayd M, Baxter MG, Xie Z, Lee IH, Rudolph J, Inouye SK, Marcantonio ER, Crosby G.
PMID: 24959140
Front Aging Neurosci. 2014 Jun 10;6:107. doi: 10.3389/fnagi.2014.00107. eCollection 2014.

Delirium is a common and morbid condition in elderly hospitalized patients. Its pathophysiology is poorly understood but inflammation has been implicated based on a clinical association with systemic infection and surgery and preclinical data showing that systemic inflammation adversely...

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Culley DJ, Snayd M, Baxter MG, et al. Systemic inflammation impairs attention and cognitive flexibility but not associative learning in aged rats: possible implications for delirium. Front Aging Neurosci. 2014;6:107doi: 10.3389/fnagi.2014.00107.
Culley, D. J., Snayd, M., Baxter, M. G., Xie, Z., Lee, I. H., Rudolph, J., Inouye, S. K., Marcantonio, E. R., & Crosby, G. (2014). Systemic inflammation impairs attention and cognitive flexibility but not associative learning in aged rats: possible implications for delirium. Frontiers in aging neuroscience, 6107. https://doi.org/10.3389/fnagi.2014.00107
Culley, Deborah J, et al. "Systemic inflammation impairs attention and cognitive flexibility but not associative learning in aged rats: possible implications for delirium." Frontiers in aging neuroscience vol. 6 (2014): 107. doi: https://doi.org/10.3389/fnagi.2014.00107
Culley DJ, Snayd M, Baxter MG, Xie Z, Lee IH, Rudolph J, Inouye SK, Marcantonio ER, Crosby G. Systemic inflammation impairs attention and cognitive flexibility but not associative learning in aged rats: possible implications for delirium. Front Aging Neurosci. 2014 Jun 10;6:107. doi: 10.3389/fnagi.2014.00107. eCollection 2014. PMID: 24959140; PMCID: PMC4050637.
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Multiple Signaling Molecules are Involved in Expression of CCL2 and IL-1β in Response to FSL-1, a Toll-Like Receptor 6 Agonist, in Macrophages.

The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Won K, Kim SM, Lee SA, Rhim BY, Eo SK, Kim K.
PMID: 23271927
Korean J Physiol Pharmacol. 2012 Dec;16(6):447-53. doi: 10.4196/kjpp.2012.16.6.447. Epub 2012 Dec 10.

TLR6 forms a heterodimer with TLR2 and TLR4. While proinflammatory roles of TLR2 and TLR4 are well documented, the role of TLR6 in inflammation is poorly understood. In order to understand mechanisms of action of TLR6 in inflammatory responses,...

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Won K, Kim SM, Lee SA, et al. Multiple Signaling Molecules are Involved in Expression of CCL2 and IL-1β in Response to FSL-1, a Toll-Like Receptor 6 Agonist, in Macrophages. Korean J Physiol Pharmacol. 2012;16(6):447-53doi: 10.4196/kjpp.2012.16.6.447.
Won, K., Kim, S. M., Lee, S. A., Rhim, B. Y., Eo, S. K., & Kim, K. (2012). Multiple Signaling Molecules are Involved in Expression of CCL2 and IL-1β in Response to FSL-1, a Toll-Like Receptor 6 Agonist, in Macrophages. The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, 16(6), 447-53. https://doi.org/10.4196/kjpp.2012.16.6.447
Won, Keunsoo, et al. "Multiple Signaling Molecules are Involved in Expression of CCL2 and IL-1β in Response to FSL-1, a Toll-Like Receptor 6 Agonist, in Macrophages." The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology vol. 16,6 (2012): 447-53. doi: https://doi.org/10.4196/kjpp.2012.16.6.447
Won K, Kim SM, Lee SA, Rhim BY, Eo SK, Kim K. Multiple Signaling Molecules are Involved in Expression of CCL2 and IL-1β in Response to FSL-1, a Toll-Like Receptor 6 Agonist, in Macrophages. Korean J Physiol Pharmacol. 2012 Dec;16(6):447-53. doi: 10.4196/kjpp.2012.16.6.447. Epub 2012 Dec 10. PMID: 23271927; PMCID: PMC3526750.
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The Induction of Circulating, ACAID-Inducing Monocytes Requires CCR2/CCL2-Dependent Migration of Circulating F4/80(+) Cells into the Anterior Chamber.

Ophthalmology and eye diseases

Cone RE, Chattopadhyay S, Pais R, Bhowmick S, Sharafieh R, Lemire Y, O'Rourke J.
PMID: 23861614
Ophthalmol Eye Dis. 2010 Nov 11;2:57-68. doi: 10.4137/OED.S6113. Print 2010.

To determine the origin of peripheral blood mononulclear cells (PBMC) that activate regulatory T cells in anterior chamber-associated immune deviation (ACAID), fluorescein-labeled PBMC were intravenously injected into mice before the mice received an intracameral injection of antigen. Six-24 hr...

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Cone RE, Chattopadhyay S, Pais R, et al. The Induction of Circulating, ACAID-Inducing Monocytes Requires CCR2/CCL2-Dependent Migration of Circulating F4/80(+) Cells into the Anterior Chamber. Ophthalmol Eye Dis. 2010;2:57-68doi: 10.4137/OED.S6113.
Cone, R. E., Chattopadhyay, S., Pais, R., Bhowmick, S., Sharafieh, R., Lemire, Y., & O'Rourke, J. (2010). The Induction of Circulating, ACAID-Inducing Monocytes Requires CCR2/CCL2-Dependent Migration of Circulating F4/80(+) Cells into the Anterior Chamber. Ophthalmology and eye diseases, 257-68. https://doi.org/10.4137/OED.S6113
Cone, Robert E, et al. "The Induction of Circulating, ACAID-Inducing Monocytes Requires CCR2/CCL2-Dependent Migration of Circulating F4/80(+) Cells into the Anterior Chamber." Ophthalmology and eye diseases vol. 2 (2010): 57-68. doi: https://doi.org/10.4137/OED.S6113
Cone RE, Chattopadhyay S, Pais R, Bhowmick S, Sharafieh R, Lemire Y, O'Rourke J. The Induction of Circulating, ACAID-Inducing Monocytes Requires CCR2/CCL2-Dependent Migration of Circulating F4/80(+) Cells into the Anterior Chamber. Ophthalmol Eye Dis. 2010 Nov 11;2:57-68. doi: 10.4137/OED.S6113. Print 2010. PMID: 23861614; PMCID: PMC3661475.
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Effect of Persimmon Leaf Extract on Phthalic Anhydride-induced Allergic Response in Mice.

Preventive nutrition and food science

Mok JY, Jeon IH, Cho JK, Park JM, Kim HS, Kang HJ, Kim HS, Jang SI.
PMID: 24471058
Prev Nutr Food Sci. 2012 Mar;17(1):14-21. doi: 10.3746/pnf.2012.17.1.014.

The purpose of this study was to investigate the anti-allergy activities of persimmon leaf extract (PLE) on a phthalic anhydride (PA)-induced allergic mouse model. A human leukemic mast cell line (HMC-1) was used to examine the inhibitory activity of...

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Mok JY, Jeon IH, Cho JK, et al. Effect of Persimmon Leaf Extract on Phthalic Anhydride-induced Allergic Response in Mice. Prev Nutr Food Sci. 2012;17(1):14-21doi: 10.3746/pnf.2012.17.1.014.
Mok, J. Y., Jeon, I. H., Cho, J. K., Park, J. M., Kim, H. S., Kang, H. J., Kim, H. S., & Jang, S. I. (2012). Effect of Persimmon Leaf Extract on Phthalic Anhydride-induced Allergic Response in Mice. Preventive nutrition and food science, 17(1), 14-21. https://doi.org/10.3746/pnf.2012.17.1.014
Mok, Ji Ye, et al. "Effect of Persimmon Leaf Extract on Phthalic Anhydride-induced Allergic Response in Mice." Preventive nutrition and food science vol. 17,1 (2012): 14-21. doi: https://doi.org/10.3746/pnf.2012.17.1.014
Mok JY, Jeon IH, Cho JK, Park JM, Kim HS, Kang HJ, Kim HS, Jang SI. Effect of Persimmon Leaf Extract on Phthalic Anhydride-induced Allergic Response in Mice. Prev Nutr Food Sci. 2012 Mar;17(1):14-21. doi: 10.3746/pnf.2012.17.1.014. PMID: 24471058; PMCID: PMC3866758.
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The Inhibitory Effect of Premature Citrus unshiu Extract on Atopic Dermatitis In Vitro and In Vivo.

Toxicological research

Kang GJ, Han SC, Yi EJ, Kang HK, Yoo ES.
PMID: 24278569
Toxicol Res. 2011 Sep;27(3):173-80. doi: 10.5487/TR.2011.27.3.173.

Atopic dermatitis (AD) is a chronic, recurrent inflammatory skin disease that is associated with Th2 cellmediated allergy. The process that leads to infiltration of inflammatory cells into an AD lesion is remarkably dependent on various chemokines, especially TARC (thymus...

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Kang GJ, Han SC, Yi EJ, et al. The Inhibitory Effect of Premature Citrus unshiu Extract on Atopic Dermatitis In Vitro and In Vivo. Toxicol Res. 2011;27(3):173-80doi: 10.5487/TR.2011.27.3.173.
Kang, G. J., Han, S. C., Yi, E. J., Kang, H. K., & Yoo, E. S. (2011). The Inhibitory Effect of Premature Citrus unshiu Extract on Atopic Dermatitis In Vitro and In Vivo. Toxicological research, 27(3), 173-80. https://doi.org/10.5487/TR.2011.27.3.173
Kang, Gyeoung-Jin, et al. "The Inhibitory Effect of Premature Citrus unshiu Extract on Atopic Dermatitis In Vitro and In Vivo." Toxicological research vol. 27,3 (2011): 173-80. doi: https://doi.org/10.5487/TR.2011.27.3.173
Kang GJ, Han SC, Yi EJ, Kang HK, Yoo ES. The Inhibitory Effect of Premature Citrus unshiu Extract on Atopic Dermatitis In Vitro and In Vivo. Toxicol Res. 2011 Sep;27(3):173-80. doi: 10.5487/TR.2011.27.3.173. PMID: 24278569; PMCID: PMC3834378.
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Resolution of central nervous system astrocytic and endothelial sources of CCL2 gene expression during evolving neuroinflammation.

Fluids and barriers of the CNS

Shrestha B, Ge S, Pachter JS.
PMID: 24589378
Fluids Barriers CNS. 2014 Mar 04;11(1):6. doi: 10.1186/2045-8118-11-6.

BACKGROUND: The chemokine CCL2 is a critical mediator of neuroinflammation in diseases such as multiple sclerosis (MS) and its animal model, experimental autoimmune encephalomyelitis (EAE). CCL2 drives mononuclear cell infiltration into the central nervous system (CNS), alters expression and...

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Shrestha B, Ge S, Pachter JS. Resolution of central nervous system astrocytic and endothelial sources of CCL2 gene expression during evolving neuroinflammation. Fluids Barriers CNS. 2014;11(1):6doi: 10.1186/2045-8118-11-6.
Shrestha, B., Ge, S., & Pachter, J. S. (2014). Resolution of central nervous system astrocytic and endothelial sources of CCL2 gene expression during evolving neuroinflammation. Fluids and barriers of the CNS, 11(1), 6. https://doi.org/10.1186/2045-8118-11-6
Shrestha, Bandana, et al. "Resolution of central nervous system astrocytic and endothelial sources of CCL2 gene expression during evolving neuroinflammation." Fluids and barriers of the CNS vol. 11,1 (2014): 6. doi: https://doi.org/10.1186/2045-8118-11-6
Shrestha B, Ge S, Pachter JS. Resolution of central nervous system astrocytic and endothelial sources of CCL2 gene expression during evolving neuroinflammation. Fluids Barriers CNS. 2014 Mar 04;11(1):6. doi: 10.1186/2045-8118-11-6. PMID: 24589378; PMCID: PMC3944978.
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Chemokine receptor expression by inflammatory T cells in EAE.

Frontiers in cellular neuroscience

Mony JT, Khorooshi R, Owens T.
PMID: 25071447
Front Cell Neurosci. 2014 Jul 04;8:187. doi: 10.3389/fncel.2014.00187. eCollection 2014.

Chemokines direct cellular infiltration to tissues, and their receptors and signaling pathways represent targets for therapy in diseases such as multiple sclerosis (MS). The chemokine CCL20 is expressed in choroid plexus, a site of entry of T cells to...

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Mony JT, Khorooshi R, Owens T. Chemokine receptor expression by inflammatory T cells in EAE. Front Cell Neurosci. 2014;8:187doi: 10.3389/fncel.2014.00187.
Mony, J. T., Khorooshi, R., & Owens, T. (2014). Chemokine receptor expression by inflammatory T cells in EAE. Frontiers in cellular neuroscience, 8187. https://doi.org/10.3389/fncel.2014.00187
Mony, Jyothi Thyagabhavan, et al. "Chemokine receptor expression by inflammatory T cells in EAE." Frontiers in cellular neuroscience vol. 8 (2014): 187. doi: https://doi.org/10.3389/fncel.2014.00187
Mony JT, Khorooshi R, Owens T. Chemokine receptor expression by inflammatory T cells in EAE. Front Cell Neurosci. 2014 Jul 04;8:187. doi: 10.3389/fncel.2014.00187. eCollection 2014. PMID: 25071447; PMCID: PMC4081975.
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A role for CCL2 in both tumor progression and immunosurveillance.

Oncoimmunology

Li M, Knight DA, A Snyder L, Smyth MJ, Stewart TJ.
PMID: 24073384
Oncoimmunology. 2013 Jul 01;2(7):e25474. doi: 10.4161/onci.25474.

The chemokine CCL2, which is best known for its chemotactic functions, is expressed not only by immune cells, but also by several types of malignant and stromal cells. CCL2 has been shown to exert both pro- and anti-tumor effects....

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Li M, Knight DA, A Snyder L, et al. A role for CCL2 in both tumor progression and immunosurveillance. Oncoimmunology. 2013;2(7):e25474doi: 10.4161/onci.25474.
Li, M., Knight, D. A., A Snyder, L., Smyth, M. J., & Stewart, T. J. (2013). A role for CCL2 in both tumor progression and immunosurveillance. Oncoimmunology, 2(7), e25474. https://doi.org/10.4161/onci.25474
Li, Ming, et al. "A role for CCL2 in both tumor progression and immunosurveillance." Oncoimmunology vol. 2,7 (2013): e25474. doi: https://doi.org/10.4161/onci.25474
Li M, Knight DA, A Snyder L, Smyth MJ, Stewart TJ. A role for CCL2 in both tumor progression and immunosurveillance. Oncoimmunology. 2013 Jul 01;2(7):e25474. doi: 10.4161/onci.25474. PMID: 24073384; PMCID: PMC3782157.
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Display of human and rabbit monocyte chemoattractant protein-1 on human embryonic kidney 293T cell surface.

Research in pharmaceutical sciences

Boshtam M, Asgary S, Rahimmanesh I, Kouhpayeh S, Naderi J, Hejazi Z, Mohammad-Dezashibi H, Pieper IL, Khanahmad H.
PMID: 30271445
Res Pharm Sci. 2018 Oct;13(5):430-439. doi: 10.4103/1735-5362.236836.

Monocyte chemoattractant protein-1 (MCP-1/CCL2) is a protein that is secreted immediately upon endothelial injury, and thereby it plays a key role in inflammation via recruitment of leucocytes to the site of inflammation at the beginning and throughout the inflammatory...

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Boshtam M, Asgary S, Rahimmanesh I, et al. Display of human and rabbit monocyte chemoattractant protein-1 on human embryonic kidney 293T cell surface. Res Pharm Sci. 2018;13(5):430-439doi: 10.4103/1735-5362.236836.
Boshtam, M., Asgary, S., Rahimmanesh, I., Kouhpayeh, S., Naderi, J., Hejazi, Z., Mohammad-Dezashibi, H., Pieper, I. L., & Khanahmad, H. (2018). Display of human and rabbit monocyte chemoattractant protein-1 on human embryonic kidney 293T cell surface. Research in pharmaceutical sciences, 13(5), 430-439. https://doi.org/10.4103/1735-5362.236836
Boshtam, Maryam, et al. "Display of human and rabbit monocyte chemoattractant protein-1 on human embryonic kidney 293T cell surface." Research in pharmaceutical sciences vol. 13,5 (2018): 430-439. doi: https://doi.org/10.4103/1735-5362.236836
Boshtam M, Asgary S, Rahimmanesh I, Kouhpayeh S, Naderi J, Hejazi Z, Mohammad-Dezashibi H, Pieper IL, Khanahmad H. Display of human and rabbit monocyte chemoattractant protein-1 on human embryonic kidney 293T cell surface. Res Pharm Sci. 2018 Oct;13(5):430-439. doi: 10.4103/1735-5362.236836. PMID: 30271445; PMCID: PMC6082034.
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[No title available]

Journal of cancer metastasis and treatment

Khazal KF, Hill DL.
PMID: 28090590
J Cancer Metastasis Treat. 2015 Jul;1(2):94-100. doi: 10.4103/2394-4722.157601. Epub 2015 Jul 15.

AIM: The aim was to examine the anti-proliferative effect of a METHODS: WS root extract was used to treat tumor cells at concentrations up to 100 µg and for nude mouse experiments, the mice received daily WS at 300...

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Khazal KF, Hill DL. . J Cancer Metastasis Treat. 2015;1(2):94-100doi: 10.4103/2394-4722.157601.
Khazal, K. F., & Hill, D. L. (2015). . Journal of cancer metastasis and treatment, 1(2), 94-100. https://doi.org/10.4103/2394-4722.157601
Khazal, Kamel F, and Hill, Donald L. "." Journal of cancer metastasis and treatment vol. 1,2 (2015): 94-100. doi: https://doi.org/10.4103/2394-4722.157601
Khazal KF, Hill DL. . J Cancer Metastasis Treat. 2015 Jul;1(2):94-100. doi: 10.4103/2394-4722.157601. Epub 2015 Jul 15. PMID: 28090590; PMCID: PMC5228601.
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Fbxw7 suppresses cancer metastasis by inhibiting niche formation.

Oncoimmunology

Yumimoto K, Nakayama KI.
PMID: 26405580
Oncoimmunology. 2015 Jun 03;4(8):e1022308. doi: 10.1080/2162402X.2015.1022308. eCollection 2015 Aug.

Fbxw7 has been identified as an oncosuppressor protein in many types of cancer. We have recently shown that loss of Fbxw7 in bone marrow-derived stromal cells (BMSCs) promotes cancer metastasis by increasing production of the chemokine CCL2, which attracts...

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Yumimoto K, Nakayama KI. Fbxw7 suppresses cancer metastasis by inhibiting niche formation. Oncoimmunology. 2015;4(8):e1022308doi: 10.1080/2162402X.2015.1022308.
Yumimoto, K., & Nakayama, K. I. (2015). Fbxw7 suppresses cancer metastasis by inhibiting niche formation. Oncoimmunology, 4(8), e1022308. https://doi.org/10.1080/2162402X.2015.1022308
Yumimoto, Kanae, and Nakayama, Keiichi I. "Fbxw7 suppresses cancer metastasis by inhibiting niche formation." Oncoimmunology vol. 4,8 (2015): e1022308. doi: https://doi.org/10.1080/2162402X.2015.1022308
Yumimoto K, Nakayama KI. Fbxw7 suppresses cancer metastasis by inhibiting niche formation. Oncoimmunology. 2015 Jun 03;4(8):e1022308. doi: 10.1080/2162402X.2015.1022308. eCollection 2015 Aug. PMID: 26405580; PMCID: PMC4570111.
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Showing 1 to 12 of 96 entries