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Showing 1 to 12 of 67 entries
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Systemic Inflammation in Cachexia - Is Tumor Cytokine Expression Profile the Culprit?.

Frontiers in immunology

de Matos-Neto EM, Lima JD, de Pereira WO, Figuerêdo RG, Riccardi DM, Radloff K, das Neves RX, Camargo RG, Maximiano LF, Tokeshi F, Otoch JP, Goldszmid R, Câmara NO, Trinchieri G, de Alcântara PS, Seelaender M.
PMID: 26732354
Front Immunol. 2015 Dec 24;6:629. doi: 10.3389/fimmu.2015.00629. eCollection 2015.

Cachexia affects about 80% of gastrointestinal cancer patients. This multifactorial syndrome resulting in involuntary and continuous weight loss is accompanied by systemic inflammation and immune cell infiltration in various tissues. Understanding the interactions among tumor, immune cells, and peripheral...

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de Matos-Neto EM, Lima JD, de Pereira WO, et al. Systemic Inflammation in Cachexia - Is Tumor Cytokine Expression Profile the Culprit?. Front Immunol. 2015;6:629doi: 10.3389/fimmu.2015.00629.
de Matos-Neto, E. M., Lima, J. D., de Pereira, W. O., Figuerêdo, R. G., Riccardi, D. M., Radloff, K., das Neves, R. X., Camargo, R. G., Maximiano, L. F., Tokeshi, F., Otoch, J. P., Goldszmid, R., Câmara, N. O., Trinchieri, G., de Alcântara, P. S., & Seelaender, M. (2015). Systemic Inflammation in Cachexia - Is Tumor Cytokine Expression Profile the Culprit?. Frontiers in immunology, 6629. https://doi.org/10.3389/fimmu.2015.00629
de Matos-Neto, Emidio M, et al. "Systemic Inflammation in Cachexia - Is Tumor Cytokine Expression Profile the Culprit?." Frontiers in immunology vol. 6 (2015): 629. doi: https://doi.org/10.3389/fimmu.2015.00629
de Matos-Neto EM, Lima JD, de Pereira WO, Figuerêdo RG, Riccardi DM, Radloff K, das Neves RX, Camargo RG, Maximiano LF, Tokeshi F, Otoch JP, Goldszmid R, Câmara NO, Trinchieri G, de Alcântara PS, Seelaender M. Systemic Inflammation in Cachexia - Is Tumor Cytokine Expression Profile the Culprit?. Front Immunol. 2015 Dec 24;6:629. doi: 10.3389/fimmu.2015.00629. eCollection 2015. PMID: 26732354; PMCID: PMC4689790.
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Proteases and chemotactic factors in BAL fluid from subjects with subclinical emphysema.

Chest

Nishimura M, Betsuyaku T, Tanino M, Takeyabu K, Miyamoto K, Kawakami Y.
PMID: 10843954
Chest. 2000 May;117(5):282S. doi: 10.1378/chest.117.5_suppl_1.282s.

No abstract available.

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Nishimura M, Betsuyaku T, Tanino M, et al. Proteases and chemotactic factors in BAL fluid from subjects with subclinical emphysema. Chest. 2000;117(5):282Sdoi: 10.1378/chest.117.5_suppl_1.282s.
Nishimura, M., Betsuyaku, T., Tanino, M., Takeyabu, K., Miyamoto, K., & Kawakami, Y. (2000). Proteases and chemotactic factors in BAL fluid from subjects with subclinical emphysema. Chest, 117(5), 282S. https://doi.org/10.1378/chest.117.5_suppl_1.282s
Nishimura, et al. "Proteases and chemotactic factors in BAL fluid from subjects with subclinical emphysema." Chest vol. 117,5 (2000): 282S. doi: https://doi.org/10.1378/chest.117.5_suppl_1.282s
Nishimura M, Betsuyaku T, Tanino M, Takeyabu K, Miyamoto K, Kawakami Y. Proteases and chemotactic factors in BAL fluid from subjects with subclinical emphysema. Chest. 2000 May;117(5):282S. doi: 10.1378/chest.117.5_suppl_1.282s. PMID: 10843954.
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Polymorphonuclear leukocyte chemotactic activity in rabbit serum and Guinea pig serum treated with immune complexes: evidence for c5a as the major chemotactic factor.

Infection and immunity

Snyderman R, Phillips J, Mergenhagen SE.
PMID: 16557770
Infect Immun. 1970 Jun;1(6):521-5. doi: 10.1128/iai.1.6.521-525.1970.

A number of polymorphonuclear leukocyte (PMN) chemotactic factors derived from complement components have been described. We sought to determine which of these factors accounted for the majority of PMN chemotactic activity in rabbit serum and guinea pig serum treated...

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Snyderman R, Phillips J, Mergenhagen SE. Polymorphonuclear leukocyte chemotactic activity in rabbit serum and Guinea pig serum treated with immune complexes: evidence for c5a as the major chemotactic factor. Infect Immun. 1970;1(6):521-5doi: 10.1128/iai.1.6.521-525.1970.
Snyderman, R., Phillips, J., & Mergenhagen, S. E. (1970). Polymorphonuclear leukocyte chemotactic activity in rabbit serum and Guinea pig serum treated with immune complexes: evidence for c5a as the major chemotactic factor. Infection and immunity, 1(6), 521-5. https://doi.org/10.1128/iai.1.6.521-525.1970
Snyderman, R, et al. "Polymorphonuclear leukocyte chemotactic activity in rabbit serum and Guinea pig serum treated with immune complexes: evidence for c5a as the major chemotactic factor." Infection and immunity vol. 1,6 (1970): 521-5. doi: https://doi.org/10.1128/iai.1.6.521-525.1970
Snyderman R, Phillips J, Mergenhagen SE. Polymorphonuclear leukocyte chemotactic activity in rabbit serum and Guinea pig serum treated with immune complexes: evidence for c5a as the major chemotactic factor. Infect Immun. 1970 Jun;1(6):521-5. doi: 10.1128/iai.1.6.521-525.1970. PMID: 16557770; PMCID: PMC415936.
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Immunoreactants in rheumatoid synovial effusions.

The Journal of experimental medicine

Zvaifler NJ.
PMID: 19867372
J Exp Med. 1971 Sep 01;134(3):276-85.

Measurements were made of the IgG in synovial fluid which is synthesized by the intact rheumatoid synovium. In five rheumatoid subjects 12-24% of the IgG present in their synovial fluid was derived from local production. No evidence for local...

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Zvaifler NJ. Immunoreactants in rheumatoid synovial effusions. J Exp Med. 1971;134(3):276-85
Zvaifler, N. J. (1971). Immunoreactants in rheumatoid synovial effusions. The Journal of experimental medicine, 134(3), 276-85.
Zvaifler, N J. "Immunoreactants in rheumatoid synovial effusions." The Journal of experimental medicine vol. 134,3 (1971): 276-85.
Zvaifler NJ. Immunoreactants in rheumatoid synovial effusions. J Exp Med. 1971 Sep 01;134(3):276-85. PMID: 19867372; PMCID: PMC2139080.
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Ovarian tumor attachment, invasion, and vascularization reflect unique microenvironments in the peritoneum: insights from xenograft and mathematical models.

Frontiers in oncology

Steinkamp MP, Winner KK, Davies S, Muller C, Zhang Y, Hoffman RM, Shirinifard A, Moses M, Jiang Y, Wilson BS.
PMID: 23730620
Front Oncol. 2013 May 17;3:97. doi: 10.3389/fonc.2013.00097. eCollection 2013.

Ovarian cancer relapse is often characterized by metastatic spread throughout the peritoneal cavity with tumors attached to multiple organs. In this study, interaction of ovarian cancer cells with the peritoneal tumor microenvironment was evaluated in a xenograft model based...

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Steinkamp MP, Winner KK, Davies S, et al. Ovarian tumor attachment, invasion, and vascularization reflect unique microenvironments in the peritoneum: insights from xenograft and mathematical models. Front Oncol. 2013;3:97doi: 10.3389/fonc.2013.00097.
Steinkamp, M. P., Winner, K. K., Davies, S., Muller, C., Zhang, Y., Hoffman, R. M., Shirinifard, A., Moses, M., Jiang, Y., & Wilson, B. S. (2013). Ovarian tumor attachment, invasion, and vascularization reflect unique microenvironments in the peritoneum: insights from xenograft and mathematical models. Frontiers in oncology, 397. https://doi.org/10.3389/fonc.2013.00097
Steinkamp, Mara P, et al. "Ovarian tumor attachment, invasion, and vascularization reflect unique microenvironments in the peritoneum: insights from xenograft and mathematical models." Frontiers in oncology vol. 3 (2013): 97. doi: https://doi.org/10.3389/fonc.2013.00097
Steinkamp MP, Winner KK, Davies S, Muller C, Zhang Y, Hoffman RM, Shirinifard A, Moses M, Jiang Y, Wilson BS. Ovarian tumor attachment, invasion, and vascularization reflect unique microenvironments in the peritoneum: insights from xenograft and mathematical models. Front Oncol. 2013 May 17;3:97. doi: 10.3389/fonc.2013.00097. eCollection 2013. PMID: 23730620; PMCID: PMC3656359.
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Combined administration of laminin-221 and prostacyclin agonist enhances endogenous cardiac repair in an acute infarct rat heart.

Scientific reports

Sougawa N, Miyagawa S, Kawamura T, Matsuura R, Harada A, Sakai Y, Mochizuki-Oda N, Sato-Nishiuchi R, Sekiguchi K, Sawa Y.
PMID: 34782616
Sci Rep. 2021 Nov 15;11(1):22243. doi: 10.1038/s41598-021-00918-y.

Although endogenous cardiac repair by recruitment of stem cells may serve as a therapeutic approach to healing a damaged heart, how to effectively enhance the migration of stem cells to the damaged heart is unclear. Here, we examined whether...

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Sougawa N, Miyagawa S, Kawamura T, et al. Combined administration of laminin-221 and prostacyclin agonist enhances endogenous cardiac repair in an acute infarct rat heart. Sci Rep. 2021;11(1):22243doi: 10.1038/s41598-021-00918-y.
Sougawa, N., Miyagawa, S., Kawamura, T., Matsuura, R., Harada, A., Sakai, Y., Mochizuki-Oda, N., Sato-Nishiuchi, R., Sekiguchi, K., & Sawa, Y. (2021). Combined administration of laminin-221 and prostacyclin agonist enhances endogenous cardiac repair in an acute infarct rat heart. Scientific reports, 11(1), 22243. https://doi.org/10.1038/s41598-021-00918-y
Sougawa, Nagako, et al. "Combined administration of laminin-221 and prostacyclin agonist enhances endogenous cardiac repair in an acute infarct rat heart." Scientific reports vol. 11,1 (2021): 22243. doi: https://doi.org/10.1038/s41598-021-00918-y
Sougawa N, Miyagawa S, Kawamura T, Matsuura R, Harada A, Sakai Y, Mochizuki-Oda N, Sato-Nishiuchi R, Sekiguchi K, Sawa Y. Combined administration of laminin-221 and prostacyclin agonist enhances endogenous cardiac repair in an acute infarct rat heart. Sci Rep. 2021 Nov 15;11(1):22243. doi: 10.1038/s41598-021-00918-y. PMID: 34782616; PMCID: PMC8593012.
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Role of Innate Inflammation in the Regulation of Tissue Remodeling during Tooth Eruption.

Dentistry journal

Makino Y, Fujikawa K, Matsuki-Fukushima M, Inoue S, Nakamura M.
PMID: 33445432
Dent J (Basel). 2021 Jan 12;9(1). doi: 10.3390/dj9010007.

Tooth eruption is characterized by a coordinated complex cascade of cellular and molecular events that promote tooth movement through the eruptive pathway. During tooth eruption, the stratum intermedium structurally changes to the papillary layer with tooth organ development. We...

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Makino Y, Fujikawa K, Matsuki-Fukushima M, et al. Role of Innate Inflammation in the Regulation of Tissue Remodeling during Tooth Eruption. Dent J (Basel). 2021;9(1)doi: 10.3390/dj9010007.
Makino, Y., Fujikawa, K., Matsuki-Fukushima, M., Inoue, S., & Nakamura, M. (2021). Role of Innate Inflammation in the Regulation of Tissue Remodeling during Tooth Eruption. Dentistry journal, 9(1), . https://doi.org/10.3390/dj9010007
Makino, Yusuke, et al. "Role of Innate Inflammation in the Regulation of Tissue Remodeling during Tooth Eruption." Dentistry journal vol. 9,1 (2021). doi: https://doi.org/10.3390/dj9010007
Makino Y, Fujikawa K, Matsuki-Fukushima M, Inoue S, Nakamura M. Role of Innate Inflammation in the Regulation of Tissue Remodeling during Tooth Eruption. Dent J (Basel). 2021 Jan 12;9(1). doi: 10.3390/dj9010007. PMID: 33445432; PMCID: PMC7827943.
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COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm.

International journal of dermatology

Ionescu MA.
PMID: 34213786
Int J Dermatol. 2022 Jan;61(1):3-6. doi: 10.1111/ijd.15749. Epub 2021 Jul 02.

BACKGROUND: Several skin manifestations have been reported since the start of the COVID-19 pandemic: chilblains-like, livedoid lesions, urticaria-like, pseudo-Kawasaki disease, and others. Histopathologic images of these lesions most often show aspects of endothelitis, images similar to autoimmune vasculitis. Cutaneous...

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Ionescu MA. COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. Int J Dermatol. 2021;61(1):3-6doi: 10.1111/ijd.15749.
Ionescu, M. A. (2022). COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. International journal of dermatology, 61(1), 3-6. https://doi.org/10.1111/ijd.15749
Ionescu, Marius-Anton. "COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm." International journal of dermatology vol. 61,1 (2022): 3-6. doi: https://doi.org/10.1111/ijd.15749
Ionescu MA. COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. Int J Dermatol. 2022 Jan;61(1):3-6. doi: 10.1111/ijd.15749. Epub 2021 Jul 02. PMID: 34213786; PMCID: PMC8444652.
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COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm.

International journal of dermatology

Ionescu MA.
PMID: 34213786
Int J Dermatol. 2022 Jan;61(1):3-6. doi: 10.1111/ijd.15749. Epub 2021 Jul 02.

BACKGROUND: Several skin manifestations have been reported since the start of the COVID-19 pandemic: chilblains-like, livedoid lesions, urticaria-like, pseudo-Kawasaki disease, and others. Histopathologic images of these lesions most often show aspects of endothelitis, images similar to autoimmune vasculitis. Cutaneous...

Cite
Ionescu MA. COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. Int J Dermatol. 2021;61(1):3-6doi: 10.1111/ijd.15749.
Ionescu, M. A. (2022). COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. International journal of dermatology, 61(1), 3-6. https://doi.org/10.1111/ijd.15749
Ionescu, Marius-Anton. "COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm." International journal of dermatology vol. 61,1 (2022): 3-6. doi: https://doi.org/10.1111/ijd.15749
Ionescu MA. COVID-19 skin lesions are rarely positive at RT-PCR test: the macrophage activation with vascular impact and SARS-CoV-2-induced cytokine storm. Int J Dermatol. 2022 Jan;61(1):3-6. doi: 10.1111/ijd.15749. Epub 2021 Jul 02. PMID: 34213786; PMCID: PMC8444652.
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Steering Mast Cells or Their Mediators as a Prospective Novel Therapeutic Approach for the Treatment of Hematological Malignancies.

Frontiers in oncology

Mehtani D, Puri N.
PMID: 34631562
Front Oncol. 2021 Sep 24;11:731323. doi: 10.3389/fonc.2021.731323. eCollection 2021.

Tumor cells require signaling and close interaction with their microenvironment for their survival and proliferation. In the recent years, Mast cells have earned a greater importance for their presence and role in cancers. It is known that mast cells...

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Mehtani D, Puri N. Steering Mast Cells or Their Mediators as a Prospective Novel Therapeutic Approach for the Treatment of Hematological Malignancies. Front Oncol. 2021;11:731323doi: 10.3389/fonc.2021.731323.
Mehtani, D., & Puri, N. (2021). Steering Mast Cells or Their Mediators as a Prospective Novel Therapeutic Approach for the Treatment of Hematological Malignancies. Frontiers in oncology, 11731323. https://doi.org/10.3389/fonc.2021.731323
Mehtani, Deeksha, and Puri, Niti. "Steering Mast Cells or Their Mediators as a Prospective Novel Therapeutic Approach for the Treatment of Hematological Malignancies." Frontiers in oncology vol. 11 (2021): 731323. doi: https://doi.org/10.3389/fonc.2021.731323
Mehtani D, Puri N. Steering Mast Cells or Their Mediators as a Prospective Novel Therapeutic Approach for the Treatment of Hematological Malignancies. Front Oncol. 2021 Sep 24;11:731323. doi: 10.3389/fonc.2021.731323. eCollection 2021. PMID: 34631562; PMCID: PMC8497976.
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Effects of Prednisolone Derivative and Panaxydol: Biosurfactants on Cell Wall Integrity of Acne-Causing Resistant Bacteria.

Cell biochemistry and biophysics

Vartika, Chaudhary M, Bhagyawant SS, Srivastava N.
PMID: 34709575
Cell Biochem Biophys. 2021 Oct 28; doi: 10.1007/s12013-021-01038-0. Epub 2021 Oct 28.

Acne is one of the most common dermatological skin problem caused due to inflammation of the skin, leading to unfavorable growth of Propionibacterium acnes. It is a slow growing anaerobic, gram-positive bacterium that releases chemotactic factors and leads to...

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Vartika, Chaudhary M, Bhagyawant SS, et al. Effects of Prednisolone Derivative and Panaxydol: Biosurfactants on Cell Wall Integrity of Acne-Causing Resistant Bacteria. Cell Biochem Biophys. 2021;doi: 10.1007/s12013-021-01038-0.
Vartika, Chaudhary, M., Bhagyawant, S. S., & Srivastava, N. (2021). Effects of Prednisolone Derivative and Panaxydol: Biosurfactants on Cell Wall Integrity of Acne-Causing Resistant Bacteria. Cell biochemistry and biophysics, . https://doi.org/10.1007/s12013-021-01038-0
Vartika, et al. "Effects of Prednisolone Derivative and Panaxydol: Biosurfactants on Cell Wall Integrity of Acne-Causing Resistant Bacteria." Cell biochemistry and biophysics vol. (2021). doi: https://doi.org/10.1007/s12013-021-01038-0
Vartika, Chaudhary M, Bhagyawant SS, Srivastava N. Effects of Prednisolone Derivative and Panaxydol: Biosurfactants on Cell Wall Integrity of Acne-Causing Resistant Bacteria. Cell Biochem Biophys. 2021 Oct 28; doi: 10.1007/s12013-021-01038-0. Epub 2021 Oct 28. PMID: 34709575.
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Role of Inflammatory Mediators, Macrophages, and Neutrophils in Glioma Maintenance and Progression: Mechanistic Understanding and Potential Therapeutic Applications.

Cancers

Basheer AS, Abas F, Othman I, Naidu R.
PMID: 34439380
Cancers (Basel). 2021 Aug 23;13(16). doi: 10.3390/cancers13164226.

Gliomas are the most common, highly malignant, and deadliest forms of brain tumors. These intra-cranial solid tumors are comprised of both cancerous and non-cancerous cells, which contribute to tumor development, progression, and resistance to the therapeutic regimen. A variety...

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Basheer AS, Abas F, Othman I, et al. Role of Inflammatory Mediators, Macrophages, and Neutrophils in Glioma Maintenance and Progression: Mechanistic Understanding and Potential Therapeutic Applications. Cancers (Basel). 2021;13(16)doi: 10.3390/cancers13164226.
Basheer, A. S., Abas, F., Othman, I., & Naidu, R. (2021). Role of Inflammatory Mediators, Macrophages, and Neutrophils in Glioma Maintenance and Progression: Mechanistic Understanding and Potential Therapeutic Applications. Cancers, 13(16), . https://doi.org/10.3390/cancers13164226
Basheer, Abdul Samad, et al. "Role of Inflammatory Mediators, Macrophages, and Neutrophils in Glioma Maintenance and Progression: Mechanistic Understanding and Potential Therapeutic Applications." Cancers vol. 13,16 (2021). doi: https://doi.org/10.3390/cancers13164226
Basheer AS, Abas F, Othman I, Naidu R. Role of Inflammatory Mediators, Macrophages, and Neutrophils in Glioma Maintenance and Progression: Mechanistic Understanding and Potential Therapeutic Applications. Cancers (Basel). 2021 Aug 23;13(16). doi: 10.3390/cancers13164226. PMID: 34439380; PMCID: PMC8393628.
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Showing 1 to 12 of 67 entries