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Atypical hemolytic uremic syndrome post-kidney transplantation: two case reports and review of the literature.

Frontiers in medicine

Alasfar S, Alachkar N.
PMID: 25593925
Front Med (Lausanne). 2014 Dec 12;1:52. doi: 10.3389/fmed.2014.00052. eCollection 2014.

Atypical hemolytic uremic syndrome (aHUS) is a rare disorder characterized by over-activation and dysregulation of the alternative complement pathway. Its estimated prevalence is 1-2 per million. The disease is characterized by thrombotic microangiopathy, which causes anemia, thrombocytopenia, and acute...

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Alasfar S, Alachkar N. Atypical hemolytic uremic syndrome post-kidney transplantation: two case reports and review of the literature. Front Med (Lausanne). 2014;1:52doi: 10.3389/fmed.2014.00052.
Alasfar, S., & Alachkar, N. (2014). Atypical hemolytic uremic syndrome post-kidney transplantation: two case reports and review of the literature. Frontiers in medicine, 152. https://doi.org/10.3389/fmed.2014.00052
Alasfar, Sami, and Alachkar, Nada. "Atypical hemolytic uremic syndrome post-kidney transplantation: two case reports and review of the literature." Frontiers in medicine vol. 1 (2014): 52. doi: https://doi.org/10.3389/fmed.2014.00052
Alasfar S, Alachkar N. Atypical hemolytic uremic syndrome post-kidney transplantation: two case reports and review of the literature. Front Med (Lausanne). 2014 Dec 12;1:52. doi: 10.3389/fmed.2014.00052. eCollection 2014. PMID: 25593925; PMCID: PMC4292050.
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Autoreactivity to sweat glands and nerves in clinical scabies infection.

North American journal of medical sciences

Abreu-Velez AM, Klein AD, Howard MS.
PMID: 22624112
N Am J Med Sci. 2010 Jan;2(1):42-5.

CONTEXT: Skin changes in pregnancy can be categorized as 1) physiological/hormonal, 2) alterations in pre-existing skin diseases, or 3) represent development of new dermatoses, some of which may be pregnancy specific.CASE REPORT: We describe a 19 years old female...

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Abreu-Velez AM, Klein AD, Howard MS. Autoreactivity to sweat glands and nerves in clinical scabies infection. N Am J Med Sci. 2010;2(1):42-5
Abreu-Velez, A. M., Klein, A. D., & Howard, M. S. (2010). Autoreactivity to sweat glands and nerves in clinical scabies infection. North American journal of medical sciences, 2(1), 42-5.
Abreu-Velez, Ana Maria, et al. "Autoreactivity to sweat glands and nerves in clinical scabies infection." North American journal of medical sciences vol. 2,1 (2010): 42-5.
Abreu-Velez AM, Klein AD, Howard MS. Autoreactivity to sweat glands and nerves in clinical scabies infection. N Am J Med Sci. 2010 Jan;2(1):42-5. PMID: 22624112; PMCID: PMC3354387.
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Complements Spurned: Our Experience with Atypical Hemolytic Uremic Syndrome.

Indian journal of critical care medicine : peer-reviewed, official publication of Indian Society of Critical Care Medicine

Nagar VS, Chaterjee R, Sood A, Sajjan B, Kaushik A, Vyahalkar SV.
PMID: 28250608
Indian J Crit Care Med. 2017 Feb;21(2):102-104. doi: 10.4103/ijccm.IJCCM_341_16.

Atypical hemolytic uremic syndrome (aHUS) is a rare disorder resulting from a dysregulated activation of the alternative pathway of the complement system. It results in significant morbidity and mortality if not diagnosed and treated promptly. It lends itself to...

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Nagar VS, Chaterjee R, Sood A, et al. Complements Spurned: Our Experience with Atypical Hemolytic Uremic Syndrome. Indian J Crit Care Med. 2017;21(2):102-104doi: 10.4103/ijccm.IJCCM_341_16.
Nagar, V. S., Chaterjee, R., Sood, A., Sajjan, B., Kaushik, A., & Vyahalkar, S. V. (2017). Complements Spurned: Our Experience with Atypical Hemolytic Uremic Syndrome. Indian journal of critical care medicine : peer-reviewed, official publication of Indian Society of Critical Care Medicine, 21(2), 102-104. https://doi.org/10.4103/ijccm.IJCCM_341_16
Nagar, Vidya S, et al. "Complements Spurned: Our Experience with Atypical Hemolytic Uremic Syndrome." Indian journal of critical care medicine : peer-reviewed, official publication of Indian Society of Critical Care Medicine vol. 21,2 (2017): 102-104. doi: https://doi.org/10.4103/ijccm.IJCCM_341_16
Nagar VS, Chaterjee R, Sood A, Sajjan B, Kaushik A, Vyahalkar SV. Complements Spurned: Our Experience with Atypical Hemolytic Uremic Syndrome. Indian J Crit Care Med. 2017 Feb;21(2):102-104. doi: 10.4103/ijccm.IJCCM_341_16. PMID: 28250608; PMCID: PMC5330049.
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Complement C5a induces the formation of neutrophil extracellular traps by myeloid-derived suppressor cells to promote metastasis.

Cancer letters

Ortiz-Espinosa S, Morales X, Senent Y, Alignani D, Tavira B, Macaya I, Ruiz B, Moreno H, Remírez A, Sainz C, Rodriguez-Pena A, Oyarbide A, Ariz M, Andueza MP, Valencia K, Teijeira A, Hoehlig K, Vater A, Rolfe B, Woodruff TM, Lopez-Picazo JM, Vicent S, Kochan G, Escors D, Gil-Bazo I, Perez-Gracia JL, Montuenga LM, Lambris JD, Ortiz de Solorzano C, Lecanda F, Ajona D, Pio R.
PMID: 34971753
Cancer Lett. 2021 Dec 28; doi: 10.1016/j.canlet.2021.12.027. Epub 2021 Dec 28.

Myeloid-derived suppressor cells (MDSCs) play a major role in cancer progression. In this study, we investigated the mechanisms by which complement C5a increases the capacity of polymorphonuclear MDSCs (PMN-MDSCs) to promote tumor growth and metastatic spread. Stimulation of PMN-MDSCs...

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Ortiz-Espinosa S, Morales X, Senent Y, et al. Complement C5a induces the formation of neutrophil extracellular traps by myeloid-derived suppressor cells to promote metastasis. Cancer Lett. 2021;doi: 10.1016/j.canlet.2021.12.027.
Ortiz-Espinosa, S., Morales, X., Senent, Y., Alignani, D., Tavira, B., Macaya, I., Ruiz, B., Moreno, H., Remírez, A., Sainz, C., Rodriguez-Pena, A., Oyarbide, A., Ariz, M., Andueza, M. P., Valencia, K., Teijeira, A., Hoehlig, K., Vater, A., Rolfe, B., Woodruff, T. M., Lopez-Picazo, J. M., Vicent, S., Kochan, G., Escors, D., Gil-Bazo, I., Perez-Gracia, J. L., Montuenga, L. M., Lambris, J. D., Ortiz de Solorzano, C., Lecanda, F., Ajona, D., & Pio, R. (2021). Complement C5a induces the formation of neutrophil extracellular traps by myeloid-derived suppressor cells to promote metastasis. Cancer letters, . https://doi.org/10.1016/j.canlet.2021.12.027
Ortiz-Espinosa, Sergio, et al. "Complement C5a induces the formation of neutrophil extracellular traps by myeloid-derived suppressor cells to promote metastasis." Cancer letters vol. (2021). doi: https://doi.org/10.1016/j.canlet.2021.12.027
Ortiz-Espinosa S, Morales X, Senent Y, Alignani D, Tavira B, Macaya I, Ruiz B, Moreno H, Remírez A, Sainz C, Rodriguez-Pena A, Oyarbide A, Ariz M, Andueza MP, Valencia K, Teijeira A, Hoehlig K, Vater A, Rolfe B, Woodruff TM, Lopez-Picazo JM, Vicent S, Kochan G, Escors D, Gil-Bazo I, Perez-Gracia JL, Montuenga LM, Lambris JD, Ortiz de Solorzano C, Lecanda F, Ajona D, Pio R. Complement C5a induces the formation of neutrophil extracellular traps by myeloid-derived suppressor cells to promote metastasis. Cancer Lett. 2021 Dec 28; doi: 10.1016/j.canlet.2021.12.027. Epub 2021 Dec 28. PMID: 34971753.
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Glioblastoma Cell-Derived lncRNA-Containing Exosomes Induce Microglia to Produce Complement C5, Promoting Chemotherapy Resistance.

Cancer immunology research

Li Z, Meng X, Wu P, Zha C, Han B, Li L, Sun N, Qi T, Qin J, Zhang Y, Tian K, Li S, Yang C, Ren L, Ming J, Wang P, Song Y, Jiang C, Cai J.
PMID: 34667108
Cancer Immunol Res. 2021 Dec;9(12):1383-1399. doi: 10.1158/2326-6066.CIR-21-0258. Epub 2021 Oct 19.

Glioblastoma (GBM), the most common malignant primary brain cancer in adults, nearly always becomes resistant to current treatments, including the chemotherapeutic temozolomide (TMZ). The long noncoding RNA (lncRNA) TMZ-associated lncRNA in GBM recurrence (

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Li Z, Meng X, Wu P, et al. Glioblastoma Cell-Derived lncRNA-Containing Exosomes Induce Microglia to Produce Complement C5, Promoting Chemotherapy Resistance. Cancer Immunol Res. 2021;9(12):1383-1399doi: 10.1158/2326-6066.CIR-21-0258.
Li, Z., Meng, X., Wu, P., Zha, C., Han, B., Li, L., Sun, N., Qi, T., Qin, J., Zhang, Y., Tian, K., Li, S., Yang, C., Ren, L., Ming, J., Wang, P., Song, Y., Jiang, C., & Cai, J. (2021). Glioblastoma Cell-Derived lncRNA-Containing Exosomes Induce Microglia to Produce Complement C5, Promoting Chemotherapy Resistance. Cancer immunology research, 9(12), 1383-1399. https://doi.org/10.1158/2326-6066.CIR-21-0258
Li, Ziwei, et al. "Glioblastoma Cell-Derived lncRNA-Containing Exosomes Induce Microglia to Produce Complement C5, Promoting Chemotherapy Resistance." Cancer immunology research vol. 9,12 (2021): 1383-1399. doi: https://doi.org/10.1158/2326-6066.CIR-21-0258
Li Z, Meng X, Wu P, Zha C, Han B, Li L, Sun N, Qi T, Qin J, Zhang Y, Tian K, Li S, Yang C, Ren L, Ming J, Wang P, Song Y, Jiang C, Cai J. Glioblastoma Cell-Derived lncRNA-Containing Exosomes Induce Microglia to Produce Complement C5, Promoting Chemotherapy Resistance. Cancer Immunol Res. 2021 Dec;9(12):1383-1399. doi: 10.1158/2326-6066.CIR-21-0258. Epub 2021 Oct 19. PMID: 34667108.
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Author Correction: Submolecular probing of the complement C5a receptor-ligand binding reveals a cooperative two-site binding mechanism.

Communications biology

Dumitru AC, Deepak RNVK, Liu H, Koehler M, Zhang C, Fan H, Alsteens D.
PMID: 33980997
Commun Biol. 2021 May 12;4(1):592. doi: 10.1038/s42003-021-02174-2.

No abstract available.

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Dumitru AC, Deepak RNVK, Liu H, et al. Author Correction: Submolecular probing of the complement C5a receptor-ligand binding reveals a cooperative two-site binding mechanism. Commun Biol. 2021;4(1):592doi: 10.1038/s42003-021-02174-2.
Dumitru, A. C., Deepak, R. N. V. K., Liu, H., Koehler, M., Zhang, C., Fan, H., & Alsteens, D. (2021). Author Correction: Submolecular probing of the complement C5a receptor-ligand binding reveals a cooperative two-site binding mechanism. Communications biology, 4(1), 592. https://doi.org/10.1038/s42003-021-02174-2
Dumitru, Andra C, et al. "Author Correction: Submolecular probing of the complement C5a receptor-ligand binding reveals a cooperative two-site binding mechanism." Communications biology vol. 4,1 (2021): 592. doi: https://doi.org/10.1038/s42003-021-02174-2
Dumitru AC, Deepak RNVK, Liu H, Koehler M, Zhang C, Fan H, Alsteens D. Author Correction: Submolecular probing of the complement C5a receptor-ligand binding reveals a cooperative two-site binding mechanism. Commun Biol. 2021 May 12;4(1):592. doi: 10.1038/s42003-021-02174-2. PMID: 33980997; PMCID: PMC8116332.
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Complement C5 inhibition reverses bleomycin-induced thrombotic microangiopathy.

Clinical kidney journal

Salhi S, Ribes D, Faguer S.
PMID: 33841872
Clin Kidney J. 2020 Jul 09;14(4):1275-1276. doi: 10.1093/ckj/sfaa101. eCollection 2021 Apr.

Whether C5 blocking may improve the outcomes of patients developing chemotherapy-induced thrombotic microangiopathy (TMA) remains elusive. Lung fibrosis is a well-known complication of bleomycin, whereas TMAs are very rare (

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Salhi S, Ribes D, Faguer S. Complement C5 inhibition reverses bleomycin-induced thrombotic microangiopathy. Clin Kidney J. 2020;14(4):1275-1276doi: 10.1093/ckj/sfaa101.
Salhi, S., Ribes, D., & Faguer, S. (2021). Complement C5 inhibition reverses bleomycin-induced thrombotic microangiopathy. Clinical kidney journal, 14(4), 1275-1276. https://doi.org/10.1093/ckj/sfaa101
Salhi, Sofiane, et al. "Complement C5 inhibition reverses bleomycin-induced thrombotic microangiopathy." Clinical kidney journal vol. 14,4 (2021): 1275-1276. doi: https://doi.org/10.1093/ckj/sfaa101
Salhi S, Ribes D, Faguer S. Complement C5 inhibition reverses bleomycin-induced thrombotic microangiopathy. Clin Kidney J. 2020 Jul 09;14(4):1275-1276. doi: 10.1093/ckj/sfaa101. eCollection 2021 Apr. PMID: 33841872; PMCID: PMC8023184.
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Molecular Studies and an .

Frontiers in medicine

Piras R, Iatropoulos P, Bresin E, Todeschini M, Gastoldi S, Valoti E, Alberti M, Mele C, Galbusera M, Cuccarolo P, Benigni A, Remuzzi G, Noris M.
PMID: 33224962
Front Med (Lausanne). 2020 Nov 03;7:579418. doi: 10.3389/fmed.2020.579418. eCollection 2020.

Atypical hemolytic uremic syndrome (aHUS) is an ultra-rare disease characterized by microangiopathic hemolysis, thrombocytopenia, and renal impairment and is associated with dysregulation of the alternative complement pathway on the microvascular endothelium. Outcomes have improved greatly with pharmacologic complement C5...

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Piras R, Iatropoulos P, Bresin E, et al. Molecular Studies and an . Front Med (Lausanne). 2020;7:579418doi: 10.3389/fmed.2020.579418.
Piras, R., Iatropoulos, P., Bresin, E., Todeschini, M., Gastoldi, S., Valoti, E., Alberti, M., Mele, C., Galbusera, M., Cuccarolo, P., Benigni, A., Remuzzi, G., & Noris, M. (2020). Molecular Studies and an . Frontiers in medicine, 7579418. https://doi.org/10.3389/fmed.2020.579418
Piras, Rossella, et al. "Molecular Studies and an ." Frontiers in medicine vol. 7 (2020): 579418. doi: https://doi.org/10.3389/fmed.2020.579418
Piras R, Iatropoulos P, Bresin E, Todeschini M, Gastoldi S, Valoti E, Alberti M, Mele C, Galbusera M, Cuccarolo P, Benigni A, Remuzzi G, Noris M. Molecular Studies and an . Front Med (Lausanne). 2020 Nov 03;7:579418. doi: 10.3389/fmed.2020.579418. eCollection 2020. PMID: 33224962; PMCID: PMC7670076.
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A Rare Case of Paroxysmal Nocturnal Hemoglobinuria With Bilateral Renal Vein Thrombosis.

Cureus

Lodhi OUH, Sohail S, Hassan D.
PMID: 32724752
Cureus. 2020 Jun 24;12(6):e8806. doi: 10.7759/cureus.8806.

Paroxysmal nocturnal hemoglobinuria (PNH) is an acquired hematopoietic stem cell (HSC) disorder characterized by a partial or complete deficiency of glycosyl-phosphatidylinositol (GPI)-linked membrane proteins, which leads to intravascular hemolysis. The loss of CD55 and CD59, two GPI-anchored proteins on...

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Lodhi OUH, Sohail S, Hassan D. A Rare Case of Paroxysmal Nocturnal Hemoglobinuria With Bilateral Renal Vein Thrombosis. Cureus. 2020;12(6):e8806doi: 10.7759/cureus.8806.
Lodhi, O. U. H., Sohail, S., & Hassan, D. (2020). A Rare Case of Paroxysmal Nocturnal Hemoglobinuria With Bilateral Renal Vein Thrombosis. Cureus, 12(6), e8806. https://doi.org/10.7759/cureus.8806
Lodhi, Omair Ul Haq, et al. "A Rare Case of Paroxysmal Nocturnal Hemoglobinuria With Bilateral Renal Vein Thrombosis." Cureus vol. 12,6 (2020): e8806. doi: https://doi.org/10.7759/cureus.8806
Lodhi OUH, Sohail S, Hassan D. A Rare Case of Paroxysmal Nocturnal Hemoglobinuria With Bilateral Renal Vein Thrombosis. Cureus. 2020 Jun 24;12(6):e8806. doi: 10.7759/cureus.8806. PMID: 32724752; PMCID: PMC7381877.
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Quantitative proteomic analysis comparing grades ICRS1 and ICRS3 in patients with osteoarthritis.

Experimental and therapeutic medicine

Chae DS, Kim ME, Kang KY, Lee NY, Lee WS, Lee JS.
PMID: 34737810
Exp Ther Med. 2021 Dec;22(6):1470. doi: 10.3892/etm.2021.10905. Epub 2021 Oct 21.

Osteoarthritis (OA), which is caused by joint damage, is the most common form of arthritis, affecting millions of people worldwide. This damage can accumulate over time, which is why aging is one of the main contributors to joint damage...

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Chae DS, Kim ME, Kang KY, et al. Quantitative proteomic analysis comparing grades ICRS1 and ICRS3 in patients with osteoarthritis. Exp Ther Med. 2021;22(6):1470doi: 10.3892/etm.2021.10905.
Chae, D. S., Kim, M. E., Kang, K. Y., Lee, N. Y., Lee, W. S., & Lee, J. S. (2021). Quantitative proteomic analysis comparing grades ICRS1 and ICRS3 in patients with osteoarthritis. Experimental and therapeutic medicine, 22(6), 1470. https://doi.org/10.3892/etm.2021.10905
Chae, Dong-Sik, et al. "Quantitative proteomic analysis comparing grades ICRS1 and ICRS3 in patients with osteoarthritis." Experimental and therapeutic medicine vol. 22,6 (2021): 1470. doi: https://doi.org/10.3892/etm.2021.10905
Chae DS, Kim ME, Kang KY, Lee NY, Lee WS, Lee JS. Quantitative proteomic analysis comparing grades ICRS1 and ICRS3 in patients with osteoarthritis. Exp Ther Med. 2021 Dec;22(6):1470. doi: 10.3892/etm.2021.10905. Epub 2021 Oct 21. PMID: 34737810; PMCID: PMC8561757.
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Complement C5a induces the formation of neutrophil extracellular traps by myeloid-derived suppressor cells to promote metastasis.

Cancer letters

Ortiz-Espinosa S, Morales X, Senent Y, Alignani D, Tavira B, Macaya I, Ruiz B, Moreno H, Remírez A, Sainz C, Rodriguez-Pena A, Oyarbide A, Ariz M, Andueza MP, Valencia K, Teijeira A, Hoehlig K, Vater A, Rolfe B, Woodruff TM, Lopez-Picazo JM, Vicent S, Kochan G, Escors D, Gil-Bazo I, Perez-Gracia JL, Montuenga LM, Lambris JD, Ortiz de Solorzano C, Lecanda F, Ajona D, Pio R.
PMID: 34971753
Cancer Lett. 2021 Dec 28;529:70-84. doi: 10.1016/j.canlet.2021.12.027. Epub 2021 Dec 28.

Myeloid-derived suppressor cells (MDSCs) play a major role in cancer progression. In this study, we investigated the mechanisms by which complement C5a increases the capacity of polymorphonuclear MDSCs (PMN-MDSCs) to promote tumor growth and metastatic spread. Stimulation of PMN-MDSCs...

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Ortiz-Espinosa S, Morales X, Senent Y, et al. Complement C5a induces the formation of neutrophil extracellular traps by myeloid-derived suppressor cells to promote metastasis. Cancer Lett. 2021;529:70-84doi: 10.1016/j.canlet.2021.12.027.
Ortiz-Espinosa, S., Morales, X., Senent, Y., Alignani, D., Tavira, B., Macaya, I., Ruiz, B., Moreno, H., Remírez, A., Sainz, C., Rodriguez-Pena, A., Oyarbide, A., Ariz, M., Andueza, M. P., Valencia, K., Teijeira, A., Hoehlig, K., Vater, A., Rolfe, B., Woodruff, T. M., Lopez-Picazo, J. M., Vicent, S., Kochan, G., Escors, D., Gil-Bazo, I., Perez-Gracia, J. L., Montuenga, L. M., Lambris, J. D., Ortiz de Solorzano, C., Lecanda, F., Ajona, D., & Pio, R. (2021). Complement C5a induces the formation of neutrophil extracellular traps by myeloid-derived suppressor cells to promote metastasis. Cancer letters, 52970-84. https://doi.org/10.1016/j.canlet.2021.12.027
Ortiz-Espinosa, Sergio, et al. "Complement C5a induces the formation of neutrophil extracellular traps by myeloid-derived suppressor cells to promote metastasis." Cancer letters vol. 529 (2021): 70-84. doi: https://doi.org/10.1016/j.canlet.2021.12.027
Ortiz-Espinosa S, Morales X, Senent Y, Alignani D, Tavira B, Macaya I, Ruiz B, Moreno H, Remírez A, Sainz C, Rodriguez-Pena A, Oyarbide A, Ariz M, Andueza MP, Valencia K, Teijeira A, Hoehlig K, Vater A, Rolfe B, Woodruff TM, Lopez-Picazo JM, Vicent S, Kochan G, Escors D, Gil-Bazo I, Perez-Gracia JL, Montuenga LM, Lambris JD, Ortiz de Solorzano C, Lecanda F, Ajona D, Pio R. Complement C5a induces the formation of neutrophil extracellular traps by myeloid-derived suppressor cells to promote metastasis. Cancer Lett. 2021 Dec 28;529:70-84. doi: 10.1016/j.canlet.2021.12.027. Epub 2021 Dec 28. PMID: 34971753.
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Treatment of a patient with classical paroxysmal nocturnal hemoglobinuria and Budd-Chiari syndrome, with complement inhibitor eculizumab: Case Report.

Hippokratia

Mandala E, Lafaras C, Goulis I, Tsioni K, Georgopoulou V, Ilonidis G.
PMID: 23935352
Hippokratia. 2013 Jan;17(1):81-4.

Background. Paroxysmal nocturnal haemoglobinuria (PNH) is a rare acquired clonal disorder of hematopoietic stem cells involving all blood cells. Erythrocytes have increased susceptibility to complement-mediated haemolysis. Thrombosis is the leading cause of mortality and follows episodes of acute hemolysis....

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Mandala E, Lafaras C, Goulis I, et al. Treatment of a patient with classical paroxysmal nocturnal hemoglobinuria and Budd-Chiari syndrome, with complement inhibitor eculizumab: Case Report. Hippokratia. 2013;17(1):81-4
Mandala, E., Lafaras, C., Goulis, I., Tsioni, K., Georgopoulou, V., & Ilonidis, G. (2013). Treatment of a patient with classical paroxysmal nocturnal hemoglobinuria and Budd-Chiari syndrome, with complement inhibitor eculizumab: Case Report. Hippokratia, 17(1), 81-4.
Mandala, E, et al. "Treatment of a patient with classical paroxysmal nocturnal hemoglobinuria and Budd-Chiari syndrome, with complement inhibitor eculizumab: Case Report." Hippokratia vol. 17,1 (2013): 81-4.
Mandala E, Lafaras C, Goulis I, Tsioni K, Georgopoulou V, Ilonidis G. Treatment of a patient with classical paroxysmal nocturnal hemoglobinuria and Budd-Chiari syndrome, with complement inhibitor eculizumab: Case Report. Hippokratia. 2013 Jan;17(1):81-4. PMID: 23935352; PMCID: PMC3738287.
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