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Human Papillomavirus Genotyping and p16(INK4a) Expression in Cervical Lesions: A Combined Test to Avoid Cervical Cancer Progression.

Journal of cancer prevention

Zouheir Y, Fechtali T, Elgnaoui N.
PMID: 27390742
J Cancer Prev. 2016 Jun;21(2):121-5. doi: 10.15430/JCP.2016.21.2.121. Epub 2016 Jun 30.

Cervical cancer is a major public health problem in Morocco. The cervical cancer has a long precancerous period that provides an opportunity for the screening and treatment. Improving screening tests is a priority goal for the early diagnosis of...

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Zouheir Y, Fechtali T, Elgnaoui N. Human Papillomavirus Genotyping and p16(INK4a) Expression in Cervical Lesions: A Combined Test to Avoid Cervical Cancer Progression. J Cancer Prev. 2016;21(2):121-5doi: 10.15430/JCP.2016.21.2.121.
Zouheir, Y., Fechtali, T., & Elgnaoui, N. (2016). Human Papillomavirus Genotyping and p16(INK4a) Expression in Cervical Lesions: A Combined Test to Avoid Cervical Cancer Progression. Journal of cancer prevention, 21(2), 121-5. https://doi.org/10.15430/JCP.2016.21.2.121
Zouheir, Yassine, et al. "Human Papillomavirus Genotyping and p16(INK4a) Expression in Cervical Lesions: A Combined Test to Avoid Cervical Cancer Progression." Journal of cancer prevention vol. 21,2 (2016): 121-5. doi: https://doi.org/10.15430/JCP.2016.21.2.121
Zouheir Y, Fechtali T, Elgnaoui N. Human Papillomavirus Genotyping and p16(INK4a) Expression in Cervical Lesions: A Combined Test to Avoid Cervical Cancer Progression. J Cancer Prev. 2016 Jun;21(2):121-5. doi: 10.15430/JCP.2016.21.2.121. Epub 2016 Jun 30. PMID: 27390742; PMCID: PMC4933437.
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p16Ink4a Prevents the Activation of Aged Quiescent Dentate Gyrus Stem Cells by Physical Exercise.

Frontiers in cellular neuroscience

Micheli L, D'Andrea G, Ceccarelli M, Ferri A, Scardigli R, Tirone F.
PMID: 30792628
Front Cell Neurosci. 2019 Feb 07;13:10. doi: 10.3389/fncel.2019.00010. eCollection 2019.

In the neurogenic niches-the dentate gyrus of the hippocampus and the subventricular zone (SVZ) adjacent to lateral ventricles-stem cells continue to divide during adulthood, generating progenitor cells and new neurons, and to self-renew, thus maintaining the stem cell pool....

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Micheli L, D'Andrea G, Ceccarelli M, et al. p16Ink4a Prevents the Activation of Aged Quiescent Dentate Gyrus Stem Cells by Physical Exercise. Front Cell Neurosci. 2019;13:10doi: 10.3389/fncel.2019.00010.
Micheli, L., D'Andrea, G., Ceccarelli, M., Ferri, A., Scardigli, R., & Tirone, F. (2019). p16Ink4a Prevents the Activation of Aged Quiescent Dentate Gyrus Stem Cells by Physical Exercise. Frontiers in cellular neuroscience, 1310. https://doi.org/10.3389/fncel.2019.00010
Micheli, Laura, et al. "p16Ink4a Prevents the Activation of Aged Quiescent Dentate Gyrus Stem Cells by Physical Exercise." Frontiers in cellular neuroscience vol. 13 (2019): 10. doi: https://doi.org/10.3389/fncel.2019.00010
Micheli L, D'Andrea G, Ceccarelli M, Ferri A, Scardigli R, Tirone F. p16Ink4a Prevents the Activation of Aged Quiescent Dentate Gyrus Stem Cells by Physical Exercise. Front Cell Neurosci. 2019 Feb 07;13:10. doi: 10.3389/fncel.2019.00010. eCollection 2019. PMID: 30792628; PMCID: PMC6374340.
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Expression analysis of p16 and TOP2A protein biomarkers in cervical cancer lesions and their correlation with clinico-histopathological characteristics in a referral hospital, Tanzania.

PloS one

Zuberi Z, Mremi A, Chilongola JO, Semango G, Sauli E.
PMID: 34705880
PLoS One. 2021 Oct 27;16(10):e0259096. doi: 10.1371/journal.pone.0259096. eCollection 2021.

INTRODUCTION: Biomarkers yield important information for early diagnosis of cervical cancer. However, they are rarely applied for prognosis of cervical cancer in Tanzania, where visual inspection assay with acetic acid or Lugol's iodine and Pap test are being used...

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Zuberi Z, Mremi A, Chilongola JO, et al. Expression analysis of p16 and TOP2A protein biomarkers in cervical cancer lesions and their correlation with clinico-histopathological characteristics in a referral hospital, Tanzania. PLoS One. 2021;16(10):e0259096doi: 10.1371/journal.pone.0259096.
Zuberi, Z., Mremi, A., Chilongola, J. O., Semango, G., & Sauli, E. (2021). Expression analysis of p16 and TOP2A protein biomarkers in cervical cancer lesions and their correlation with clinico-histopathological characteristics in a referral hospital, Tanzania. PloS one, 16(10), e0259096. https://doi.org/10.1371/journal.pone.0259096
Zuberi, Zavuga, et al. "Expression analysis of p16 and TOP2A protein biomarkers in cervical cancer lesions and their correlation with clinico-histopathological characteristics in a referral hospital, Tanzania." PloS one vol. 16,10 (2021): e0259096. doi: https://doi.org/10.1371/journal.pone.0259096
Zuberi Z, Mremi A, Chilongola JO, Semango G, Sauli E. Expression analysis of p16 and TOP2A protein biomarkers in cervical cancer lesions and their correlation with clinico-histopathological characteristics in a referral hospital, Tanzania. PLoS One. 2021 Oct 27;16(10):e0259096. doi: 10.1371/journal.pone.0259096. eCollection 2021. PMID: 34705880; PMCID: PMC8550370.
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A triple-negative breast cancer surrogate subtype classification that correlates with gene expression subtypes.

Breast cancer research and treatment

Yoo TK, Kang J, Lee A, Chae BJ.
PMID: 35018542
Breast Cancer Res Treat. 2022 Jan 12; doi: 10.1007/s10549-021-06437-8. Epub 2022 Jan 12.

BACKGROUND: This study developed a triple-negative breast cancer (TNBC) surrogate subtype classification that represents TNBC subtypes based on the Vanderbilt subtype classification.METHODS: Patients who underwent primary curative surgery for TNBC were included. Representative FFPE blocks were used for gene...

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Yoo TK, Kang J, Lee A, et al. A triple-negative breast cancer surrogate subtype classification that correlates with gene expression subtypes. Breast Cancer Res Treat. 2022;doi: 10.1007/s10549-021-06437-8.
Yoo, T. K., Kang, J., Lee, A., & Chae, B. J. (2022). A triple-negative breast cancer surrogate subtype classification that correlates with gene expression subtypes. Breast cancer research and treatment, . https://doi.org/10.1007/s10549-021-06437-8
Yoo, Tae-Kyung, et al. "A triple-negative breast cancer surrogate subtype classification that correlates with gene expression subtypes." Breast cancer research and treatment vol. (2022). doi: https://doi.org/10.1007/s10549-021-06437-8
Yoo TK, Kang J, Lee A, Chae BJ. A triple-negative breast cancer surrogate subtype classification that correlates with gene expression subtypes. Breast Cancer Res Treat. 2022 Jan 12; doi: 10.1007/s10549-021-06437-8. Epub 2022 Jan 12. PMID: 35018542.
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Promoter and histone methylation and p16(INK4A) gene expression in colon cancer.

Experimental and therapeutic medicine

Yoruker EE, Mert U, Bugra D, Yamaner S, Dalay N.
PMID: 23226740
Exp Ther Med. 2012 Nov;4(5):865-870. doi: 10.3892/etm.2012.683. Epub 2012 Aug 24.

The inactivation of the cyclin-dependent kinase inhibitor p16(INK4A) gene by hypermethylation is observed in numerous types of cancer. New findings indicate that DNA and histone methylation act in concert in gene silencing. In this study, we investigated the methylation...

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Yoruker EE, Mert U, Bugra D, et al. Promoter and histone methylation and p16(INK4A) gene expression in colon cancer. Exp Ther Med. 2012;4(5):865-870doi: 10.3892/etm.2012.683.
Yoruker, E. E., Mert, U., Bugra, D., Yamaner, S., & Dalay, N. (2012). Promoter and histone methylation and p16(INK4A) gene expression in colon cancer. Experimental and therapeutic medicine, 4(5), 865-870. https://doi.org/10.3892/etm.2012.683
Yoruker, Ebru Esin, et al. "Promoter and histone methylation and p16(INK4A) gene expression in colon cancer." Experimental and therapeutic medicine vol. 4,5 (2012): 865-870. doi: https://doi.org/10.3892/etm.2012.683
Yoruker EE, Mert U, Bugra D, Yamaner S, Dalay N. Promoter and histone methylation and p16(INK4A) gene expression in colon cancer. Exp Ther Med. 2012 Nov;4(5):865-870. doi: 10.3892/etm.2012.683. Epub 2012 Aug 24. PMID: 23226740; PMCID: PMC3493785.
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Distribution of Human Papillomavirus 52 and 58 Genotypes, and Their Expression of p16 and p53 in Cervical Neoplasia.

Korean journal of pathology

Kim TE, Kim HW, Lee KE.
PMID: 24627691
Korean J Pathol. 2014 Feb;48(1):24-9. doi: 10.4132/KoreanJPathol.2014.48.1.24. Epub 2014 Feb 25.

BACKGROUND: This study investigates the prevalence of human papillomavirus (HPV) 52 and 58 genotypes among women residing in Busan, and the expression of p16 and p53 proteins in cervical neoplasia with HPV 52 and 58 infections.METHODS: A total of...

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Kim TE, Kim HW, Lee KE. Distribution of Human Papillomavirus 52 and 58 Genotypes, and Their Expression of p16 and p53 in Cervical Neoplasia. Korean J Pathol. 2014;48(1):24-9doi: 10.4132/KoreanJPathol.2014.48.1.24.
Kim, T. E., Kim, H. W., & Lee, K. E. (2014). Distribution of Human Papillomavirus 52 and 58 Genotypes, and Their Expression of p16 and p53 in Cervical Neoplasia. Korean journal of pathology, 48(1), 24-9. https://doi.org/10.4132/KoreanJPathol.2014.48.1.24
Kim, Tae Eun, et al. "Distribution of Human Papillomavirus 52 and 58 Genotypes, and Their Expression of p16 and p53 in Cervical Neoplasia." Korean journal of pathology vol. 48,1 (2014): 24-9. doi: https://doi.org/10.4132/KoreanJPathol.2014.48.1.24
Kim TE, Kim HW, Lee KE. Distribution of Human Papillomavirus 52 and 58 Genotypes, and Their Expression of p16 and p53 in Cervical Neoplasia. Korean J Pathol. 2014 Feb;48(1):24-9. doi: 10.4132/KoreanJPathol.2014.48.1.24. Epub 2014 Feb 25. PMID: 24627691; PMCID: PMC3950231.
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Cyclin D1 overexpression, p16 loss, and pRb inactivation play a key role in pulmonary carcinogenesis and have a prognostic implication for the long-term survival in non-small cell lung carcinoma patients.

Cancer research and treatment

Myong NH.
PMID: 19688048
Cancer Res Treat. 2008 Jun;40(2):45-52. doi: 10.4143/crt.2008.40.2.45. Epub 2008 Jun 30.

PURPOSE: We investigated the immunoexpressions of cyclin D1, cyclin-dependent kinase inhibitor p16 and phosphorylated retinoblastoma (p-pRb) proteins in non-small cell lung carcinoma (NSCLC) to demonstrate their key roles in tumorigenesis, their relationship with the clinicopathologic factors, and their prognostic...

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Myong NH. Cyclin D1 overexpression, p16 loss, and pRb inactivation play a key role in pulmonary carcinogenesis and have a prognostic implication for the long-term survival in non-small cell lung carcinoma patients. Cancer Res Treat. 2008;40(2):45-52doi: 10.4143/crt.2008.40.2.45.
Myong, N. H. (2008). Cyclin D1 overexpression, p16 loss, and pRb inactivation play a key role in pulmonary carcinogenesis and have a prognostic implication for the long-term survival in non-small cell lung carcinoma patients. Cancer research and treatment, 40(2), 45-52. https://doi.org/10.4143/crt.2008.40.2.45
Myong, Na-Hye. "Cyclin D1 overexpression, p16 loss, and pRb inactivation play a key role in pulmonary carcinogenesis and have a prognostic implication for the long-term survival in non-small cell lung carcinoma patients." Cancer research and treatment vol. 40,2 (2008): 45-52. doi: https://doi.org/10.4143/crt.2008.40.2.45
Myong NH. Cyclin D1 overexpression, p16 loss, and pRb inactivation play a key role in pulmonary carcinogenesis and have a prognostic implication for the long-term survival in non-small cell lung carcinoma patients. Cancer Res Treat. 2008 Jun;40(2):45-52. doi: 10.4143/crt.2008.40.2.45. Epub 2008 Jun 30. PMID: 19688048; PMCID: PMC2697490.
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Human papillomavirus prevalence and cell cycle related protein expression in tonsillar squamous cell carcinomas of korean patients with clinicopathologic analysis.

Korean journal of pathology

Lee M, Kim SB, Lee SW, Roh JL, Choi SH, Nam SY, Kim SY, Cho KJ.
PMID: 23667374
Korean J Pathol. 2013 Apr;47(2):148-57. doi: 10.4132/KoreanJPathol.2013.47.2.148. Epub 2013 Apr 24.

BACKGROUND: Human papillomavirus (HPV)-related tonsillar squamous cell carcinoma (TSCC) has recently been characterized as a distinct subset with a favorable prognosis. The prevalence and clinicopathologic significance of HPV-related TSCC in Koreans are not well known.METHODS: HPV in situ hybridization...

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Lee M, Kim SB, Lee SW, et al. Human papillomavirus prevalence and cell cycle related protein expression in tonsillar squamous cell carcinomas of korean patients with clinicopathologic analysis. Korean J Pathol. 2013;47(2):148-57doi: 10.4132/KoreanJPathol.2013.47.2.148.
Lee, M., Kim, S. B., Lee, S. W., Roh, J. L., Choi, S. H., Nam, S. Y., Kim, S. Y., & Cho, K. J. (2013). Human papillomavirus prevalence and cell cycle related protein expression in tonsillar squamous cell carcinomas of korean patients with clinicopathologic analysis. Korean journal of pathology, 47(2), 148-57. https://doi.org/10.4132/KoreanJPathol.2013.47.2.148
Lee, Miji, et al. "Human papillomavirus prevalence and cell cycle related protein expression in tonsillar squamous cell carcinomas of korean patients with clinicopathologic analysis." Korean journal of pathology vol. 47,2 (2013): 148-57. doi: https://doi.org/10.4132/KoreanJPathol.2013.47.2.148
Lee M, Kim SB, Lee SW, Roh JL, Choi SH, Nam SY, Kim SY, Cho KJ. Human papillomavirus prevalence and cell cycle related protein expression in tonsillar squamous cell carcinomas of korean patients with clinicopathologic analysis. Korean J Pathol. 2013 Apr;47(2):148-57. doi: 10.4132/KoreanJPathol.2013.47.2.148. Epub 2013 Apr 24. PMID: 23667374; PMCID: PMC3647127.
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p16 promotes proliferation in cervical carcinoma cells through CDK6-HuR-IL1A axis.

Journal of Cancer

Li M, Yang J, Liu K, Yang J, Zhan X, Wang L, Shen X, Chen J, Mao Z.
PMID: 32047552
J Cancer. 2020 Jan 14;11(6):1457-1467. doi: 10.7150/jca.35479. eCollection 2020.

The Cyclin-Dependent Kinase Inhibitor p16 (p16) acts as a tumor suppressor in most cells, but for HPV transformed cervical cancer, in which oncoprotein E7 expressed by human papillomavirus (HPV) mediates the degradation of retinoblastoma protein (Rb), p16 exhibits oncogenic...

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Li M, Yang J, Liu K, et al. p16 promotes proliferation in cervical carcinoma cells through CDK6-HuR-IL1A axis. J Cancer. 2020;11(6):1457-1467doi: 10.7150/jca.35479.
Li, M., Yang, J., Liu, K., Yang, J., Zhan, X., Wang, L., Shen, X., Chen, J., & Mao, Z. (2020). p16 promotes proliferation in cervical carcinoma cells through CDK6-HuR-IL1A axis. Journal of Cancer, 11(6), 1457-1467. https://doi.org/10.7150/jca.35479
Li, Mingzhe, et al. "p16 promotes proliferation in cervical carcinoma cells through CDK6-HuR-IL1A axis." Journal of Cancer vol. 11,6 (2020): 1457-1467. doi: https://doi.org/10.7150/jca.35479
Li M, Yang J, Liu K, Yang J, Zhan X, Wang L, Shen X, Chen J, Mao Z. p16 promotes proliferation in cervical carcinoma cells through CDK6-HuR-IL1A axis. J Cancer. 2020 Jan 14;11(6):1457-1467. doi: 10.7150/jca.35479. eCollection 2020. PMID: 32047552; PMCID: PMC6995400.
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Showing 1 to 9 of 9 entries