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Butyricimonas phoceensis sp. nov., a new anaerobic species isolated from the human gut microbiota of a French morbidly obese patient.

New microbes and new infections

Togo AH, Diop A, Dubourg G, Nguyen TT, Andrieu C, Caputo A, Couderc C, Fournier PE, Maraninchi M, Valero R, Raoult D, Million M.
PMID: 27668083
New Microbes New Infect. 2016 Aug 09;14:38-48. doi: 10.1016/j.nmni.2016.07.010. eCollection 2016 Nov.

Butyricimonas phoceensis strain AT9 (= CSUR 1981 = DSM 100664) was isolated from a stool sample from a morbidly obese French patient living in Marseille using the culturomics approach. The genome of this Gram-negative-staining, anaerobic and non-spore forming rod...

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Togo AH, Diop A, Dubourg G, et al. Butyricimonas phoceensis sp. nov., a new anaerobic species isolated from the human gut microbiota of a French morbidly obese patient. New Microbes New Infect. 2016;14:38-48doi: 10.1016/j.nmni.2016.07.010.
Togo, A. H., Diop, A., Dubourg, G., Nguyen, T. T., Andrieu, C., Caputo, A., Couderc, C., Fournier, P. E., Maraninchi, M., Valero, R., Raoult, D., & Million, M. (2016). Butyricimonas phoceensis sp. nov., a new anaerobic species isolated from the human gut microbiota of a French morbidly obese patient. New microbes and new infections, 1438-48. https://doi.org/10.1016/j.nmni.2016.07.010
Togo, A H, et al. "Butyricimonas phoceensis sp. nov., a new anaerobic species isolated from the human gut microbiota of a French morbidly obese patient." New microbes and new infections vol. 14 (2016): 38-48. doi: https://doi.org/10.1016/j.nmni.2016.07.010
Togo AH, Diop A, Dubourg G, Nguyen TT, Andrieu C, Caputo A, Couderc C, Fournier PE, Maraninchi M, Valero R, Raoult D, Million M. Butyricimonas phoceensis sp. nov., a new anaerobic species isolated from the human gut microbiota of a French morbidly obese patient. New Microbes New Infect. 2016 Aug 09;14:38-48. doi: 10.1016/j.nmni.2016.07.010. eCollection 2016 Nov. PMID: 27668083; PMCID: PMC5024336.
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[News in endocrinology: Management of asymptomatic primary hyperparathyroidism in 2014].

Revue medicale suisse

Köhler BB, Philippe J.
PMID: 25799652
Rev Med Suisse. 2015 Jan 14;11(456):58-61.

Primary hyperparathyroidism, a disorder in wich there is a hypercalcemia caused by autonomous hypersecretion of parathormone, usually by parathyroid adenoma. It is asymptomatic in most cases. After excluding a cause of secondary elevation of parathyroid hormone, a search for...

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Köhler BB, Philippe J. Endocrinologie. L'hyperparathyroïdie primaire asymptomatique en 2014. [News in endocrinology: Management of asymptomatic primary hyperparathyroidism in 2014]. Rev Med Suisse. 2015;11(456):58-61
Köhler, B. B., & Philippe, J. (2015). Endocrinologie. L'hyperparathyroïdie primaire asymptomatique en 2014. [News in endocrinology: Management of asymptomatic primary hyperparathyroidism in 2014]. Revue medicale suisse, 11(456), 58-61.
Köhler, Ballan B, and Philippe, J. "Endocrinologie. L'hyperparathyroïdie primaire asymptomatique en 2014." [News in endocrinology: Management of asymptomatic primary hyperparathyroidism in 2014]. Revue medicale suisse vol. 11,456 (2015): 58-61.
Köhler BB, Philippe J. Endocrinologie. L'hyperparathyroïdie primaire asymptomatique en 2014. [News in endocrinology: Management of asymptomatic primary hyperparathyroidism in 2014]. Rev Med Suisse. 2015 Jan 14;11(456):58-61. French. PMID: 25799652.
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Overview of the genetic determinants of primary aldosteronism.

The application of clinical genetics

Al-Salameh A, Cohen R, Desailloud R.
PMID: 24817817
Appl Clin Genet. 2014 Apr 08;7:67-79. doi: 10.2147/TACG.S45620. eCollection 2014.

Primary aldosteronism is the most common cause of secondary hypertension. The syndrome accounts for 10% of all cases of hypertension and is primarily caused by bilateral adrenal hyperplasia or aldosterone-producing adenoma. Over the last few years, the use of...

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Al-Salameh A, Cohen R, Desailloud R. Overview of the genetic determinants of primary aldosteronism. Appl Clin Genet. 2014;7:67-79doi: 10.2147/TACG.S45620.
Al-Salameh, A., Cohen, R., & Desailloud, R. (2014). Overview of the genetic determinants of primary aldosteronism. The application of clinical genetics, 767-79. https://doi.org/10.2147/TACG.S45620
Al-Salameh, Abdallah, et al. "Overview of the genetic determinants of primary aldosteronism." The application of clinical genetics vol. 7 (2014): 67-79. doi: https://doi.org/10.2147/TACG.S45620
Al-Salameh A, Cohen R, Desailloud R. Overview of the genetic determinants of primary aldosteronism. Appl Clin Genet. 2014 Apr 08;7:67-79. doi: 10.2147/TACG.S45620. eCollection 2014. PMID: 24817817; PMCID: PMC4012345.
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[Growth and regeneration of the eye of the larvae ofAeshna cyanea Müll. (Odonata, Anisoptera)].

Wilhelm Roux' Archiv fur Entwicklungsmechanik der Organismen

Mouze M.
PMID: 28304983
Wilhelm Roux Arch Entwickl Mech Org. 1975 Dec;176(4):267-283. doi: 10.1007/BF00575321.

The growth of the eye inAeshna cyanea has been studied by several experiments involving removal, regeneration and heterotopic grafts.The results show that the ommatidia do not develop from any stemcells that are permanent during the whole larval life. In...

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Mouze M. Croissance et régénération de l'oeil de la larve d'Aeshna cyanea Müll. (Odonate, Anisoptère). [Growth and regeneration of the eye of the larvae ofAeshna cyanea Müll. (Odonata, Anisoptera)]. Wilhelm Roux Arch Entwickl Mech Org. 1975;176(4):267-283doi: 10.1007/BF00575321.
Mouze, M. (1975). Croissance et régénération de l'oeil de la larve d'Aeshna cyanea Müll. (Odonate, Anisoptère). [Growth and regeneration of the eye of the larvae ofAeshna cyanea Müll. (Odonata, Anisoptera)]. Wilhelm Roux' Archiv fur Entwicklungsmechanik der Organismen, 176(4), 267-283. https://doi.org/10.1007/BF00575321
Mouze, M. "Croissance et régénération de l'oeil de la larve d'Aeshna cyanea Müll. (Odonate, Anisoptère)." [Growth and regeneration of the eye of the larvae ofAeshna cyanea Müll. (Odonata, Anisoptera)]. Wilhelm Roux' Archiv fur Entwicklungsmechanik der Organismen vol. 176,4 (1975): 267-283. doi: https://doi.org/10.1007/BF00575321
Mouze M. Croissance et régénération de l'oeil de la larve d'Aeshna cyanea Müll. (Odonate, Anisoptère). [Growth and regeneration of the eye of the larvae ofAeshna cyanea Müll. (Odonata, Anisoptera)]. Wilhelm Roux Arch Entwickl Mech Org. 1975 Dec;176(4):267-283. doi: 10.1007/BF00575321. French. PMID: 28304983.
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In Vivo Effects of Bradykinin B2 Receptor Agonists with Varying Susceptibility to Peptidases.

Frontiers in pharmacology

Jean M, Gera L, Charest-Morin X, Marceau F, Bachelard H.
PMID: 26793104
Front Pharmacol. 2016 Jan 12;6:306. doi: 10.3389/fphar.2015.00306. eCollection 2015.

We reported evidence of bradykinin (BK) regeneration from C-terminal extended BK sequences that behave as peptidase-activated B2 receptor (B2R) agonists. Further to these in vitro studies, we carried out in vivo experiments to verify hemodynamic effects of BK analogs...

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Jean M, Gera L, Charest-Morin X, et al. In Vivo Effects of Bradykinin B2 Receptor Agonists with Varying Susceptibility to Peptidases. Front Pharmacol. 2016;6:306doi: 10.3389/fphar.2015.00306.
Jean, M., Gera, L., Charest-Morin, X., Marceau, F., & Bachelard, H. (2015). In Vivo Effects of Bradykinin B2 Receptor Agonists with Varying Susceptibility to Peptidases. Frontiers in pharmacology, 6306. https://doi.org/10.3389/fphar.2015.00306
Jean, Mélissa, et al. "In Vivo Effects of Bradykinin B2 Receptor Agonists with Varying Susceptibility to Peptidases." Frontiers in pharmacology vol. 6 (2015): 306. doi: https://doi.org/10.3389/fphar.2015.00306
Jean M, Gera L, Charest-Morin X, Marceau F, Bachelard H. In Vivo Effects of Bradykinin B2 Receptor Agonists with Varying Susceptibility to Peptidases. Front Pharmacol. 2016 Jan 12;6:306. doi: 10.3389/fphar.2015.00306. eCollection 2015. PMID: 26793104; PMCID: PMC4709452.
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Activation of a Large-conductance Ca2+-Dependent K+ Channel by Stimulation of Glutamate Phosphoinositide-coupled Receptors in Cultured Cerebellar Granule Cells.

The European journal of neuroscience

Fagni L, Bossu JL, Bockaert J.
PMID: 12106464
Eur J Neurosci. 1991;3(8):778-789. doi: 10.1111/j.1460-9568.1991.tb01674.x.

Trans-1-amino-cyclopentyl-1,3-dicarboxylic acid (trans-ACPD), a specific agonist of the glutamate phosphoinositide-coupled receptor (Qp receptor), increased the amplitude of the outward K+ current recorded in the whole-cell configuration of the patch-clamp technique in mouse cultured cerebellar granule cells. This effect was...

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Fagni L, Bossu JL, Bockaert J. Activation of a Large-conductance Ca2+-Dependent K+ Channel by Stimulation of Glutamate Phosphoinositide-coupled Receptors in Cultured Cerebellar Granule Cells. Eur J Neurosci. 1991;3(8):778-789doi: 10.1111/j.1460-9568.1991.tb01674.x.
Fagni, L., Bossu, J. L., & Bockaert, J. (1991). Activation of a Large-conductance Ca2+-Dependent K+ Channel by Stimulation of Glutamate Phosphoinositide-coupled Receptors in Cultured Cerebellar Granule Cells. The European journal of neuroscience, 3(8), 778-789. https://doi.org/10.1111/j.1460-9568.1991.tb01674.x
Fagni, L., et al. "Activation of a Large-conductance Ca2+-Dependent K+ Channel by Stimulation of Glutamate Phosphoinositide-coupled Receptors in Cultured Cerebellar Granule Cells." The European journal of neuroscience vol. 3,8 (1991): 778-789. doi: https://doi.org/10.1111/j.1460-9568.1991.tb01674.x
Fagni L, Bossu JL, Bockaert J. Activation of a Large-conductance Ca2+-Dependent K+ Channel by Stimulation of Glutamate Phosphoinositide-coupled Receptors in Cultured Cerebellar Granule Cells. Eur J Neurosci. 1991;3(8):778-789. doi: 10.1111/j.1460-9568.1991.tb01674.x. PMID: 12106464.
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[Diagnosis and screening for type 2 diabetes: which test to use?].

Revue medicale suisse

Procopiou M.
PMID: 16025886
Rev Med Suisse. 2005 Jun 01;1(22):1480-4.

The benefits of screening for type 2 diabetes during its preclinical phase are not well established in comparison to starting a treatment after a diagnosis is made when the patient presents with symptoms of hyperglycaemia. Despite this uncertainty, it...

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Procopiou M. Dépistage et diagnostic: du diabète de type 2: quels tests? [Diagnosis and screening for type 2 diabetes: which test to use?]. Rev Med Suisse. 2005;1(22):1480-4
Procopiou, M. (2005). Dépistage et diagnostic: du diabète de type 2: quels tests? [Diagnosis and screening for type 2 diabetes: which test to use?]. Revue medicale suisse, 1(22), 1480-4.
Procopiou, M. "Dépistage et diagnostic: du diabète de type 2: quels tests?" [Diagnosis and screening for type 2 diabetes: which test to use?]. Revue medicale suisse vol. 1,22 (2005): 1480-4.
Procopiou M. Dépistage et diagnostic: du diabète de type 2: quels tests? [Diagnosis and screening for type 2 diabetes: which test to use?]. Rev Med Suisse. 2005 Jun 01;1(22):1480-4. French. PMID: 16025886.
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Genes involved in obesity: Adipocytes, brain and microflora.

Genes & nutrition

Macia L, Viltart O, Verwaerde C, Delacre M, Delanoye A, Grangette C, Wolowczuk I.
PMID: 18850214
Genes Nutr. 2006 Sep;1(3):189-212. doi: 10.1007/BF02829968.

The incidence of obesity and related metabolic disorders such as cardiovascular diseases and type 2 diabetes, are reaching worldwide epidemic proportions. It results from an imbalance between caloric intake and energy expenditure leading to excess energy storage, mostly due...

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Macia L, Viltart O, Verwaerde C, et al. Genes involved in obesity: Adipocytes, brain and microflora. Genes Nutr. 2006;1(3):189-212doi: 10.1007/BF02829968.
Macia, L., Viltart, O., Verwaerde, C., Delacre, M., Delanoye, A., Grangette, C., & Wolowczuk, I. (2006). Genes involved in obesity: Adipocytes, brain and microflora. Genes & nutrition, 1(3), 189-212. https://doi.org/10.1007/BF02829968
Macia, L, et al. "Genes involved in obesity: Adipocytes, brain and microflora." Genes & nutrition vol. 1,3 (2006): 189-212. doi: https://doi.org/10.1007/BF02829968
Macia L, Viltart O, Verwaerde C, Delacre M, Delanoye A, Grangette C, Wolowczuk I. Genes involved in obesity: Adipocytes, brain and microflora. Genes Nutr. 2006 Sep;1(3):189-212. doi: 10.1007/BF02829968. PMID: 18850214; PMCID: PMC3454837.
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Functional neurochemistry of the basal ganglia.

Handbook of clinical neurology

Samadi P, Rouillard C, Bédard PJ, Di Paolo T.
PMID: 18808909
Handb Clin Neurol. 2007;83:19-66. doi: 10.1016/S0072-9752(07)83002-8.

No abstract available.

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Samadi P, Rouillard C, Bédard PJ, et al. Functional neurochemistry of the basal ganglia. Handb Clin Neurol. 2007;83:19-66doi: 10.1016/S0072-9752(07)83002-8.
Samadi, P., Rouillard, C., Bédard, P. J., & Di Paolo, T. (2007). Functional neurochemistry of the basal ganglia. Handbook of clinical neurology, 8319-66. https://doi.org/10.1016/S0072-9752(07)83002-8
Samadi, Pershia, et al. "Functional neurochemistry of the basal ganglia." Handbook of clinical neurology vol. 83 (2007): 19-66. doi: https://doi.org/10.1016/S0072-9752(07)83002-8
Samadi P, Rouillard C, Bédard PJ, Di Paolo T. Functional neurochemistry of the basal ganglia. Handb Clin Neurol. 2007;83:19-66. doi: 10.1016/S0072-9752(07)83002-8. PMID: 18808909.
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[Therapeutic patient education--method of optimizing treatment in chronic diseases].

Revista medico-chirurgicala a Societatii de Medici si Naturalisti din Iasi

Vulpoi C, Ungureanu G, Stoica O.
PMID: 17595841
Rev Med Chir Soc Med Nat Iasi. 2007 Jan-Mar;111(1):13-8.

The technological revolution of the 20th century has changed not only the life style but also the human interrelations, including the physician-patient relationship. The old, primarily patriarchal, system (in which the patient trusted completely the physician and followed religiously...

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Vulpoi C, Ungureanu G, Stoica O. Educaţia terapeutică a pacientului--metodă de optimizare a tratamentului bolilor cronice. [Therapeutic patient education--method of optimizing treatment in chronic diseases]. Rev Med Chir Soc Med Nat Iasi. 2007;111(1):13-8
Vulpoi, C., Ungureanu, G., & Stoica, O. (2007). Educaţia terapeutică a pacientului--metodă de optimizare a tratamentului bolilor cronice. [Therapeutic patient education--method of optimizing treatment in chronic diseases]. Revista medico-chirurgicala a Societatii de Medici si Naturalisti din Iasi, 111(1), 13-8.
Vulpoi, Carmen, et al. "Educaţia terapeutică a pacientului--metodă de optimizare a tratamentului bolilor cronice." [Therapeutic patient education--method of optimizing treatment in chronic diseases]. Revista medico-chirurgicala a Societatii de Medici si Naturalisti din Iasi vol. 111,1 (2007): 13-8.
Vulpoi C, Ungureanu G, Stoica O. Educaţia terapeutică a pacientului--metodă de optimizare a tratamentului bolilor cronice. [Therapeutic patient education--method of optimizing treatment in chronic diseases]. Rev Med Chir Soc Med Nat Iasi. 2007 Jan-Mar;111(1):13-8. Romanian. PMID: 17595841.
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?(1)- and ?(2)-adrenergic receptors in rat corpus striatum.

Neurochemistry international

Sebben-Perez M, Ebersolt C, Blanc G, Bockaert J.
PMID: 20488027
Neurochem Int. 1984;6(1):103-8. doi: 10.1016/0197-0186(84)90033-0.

Rat corpus striatum contained ?(2)-adrenergic receptor which were labelled with [(3)H]clonidine (95 +/- 6 fmol/mg protein). The affinity of these receptors (K(d) = 1.3 to 3.6 nM) was similar to that found in cerebral cortex. Five days after kainic...

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Sebben-Perez M, Ebersolt C, Blanc G, et al. ?(1)- and ?(2)-adrenergic receptors in rat corpus striatum. Neurochem Int. 1984;6(1):103-8doi: 10.1016/0197-0186(84)90033-0.
Sebben-Perez, M., Ebersolt, C., Blanc, G., & Bockaert, J. (1984). ?(1)- and ?(2)-adrenergic receptors in rat corpus striatum. Neurochemistry international, 6(1), 103-8. https://doi.org/10.1016/0197-0186(84)90033-0
Sebben-Perez, M, et al. "?(1)- and ?(2)-adrenergic receptors in rat corpus striatum." Neurochemistry international vol. 6,1 (1984): 103-8. doi: https://doi.org/10.1016/0197-0186(84)90033-0
Sebben-Perez M, Ebersolt C, Blanc G, Bockaert J. ?(1)- and ?(2)-adrenergic receptors in rat corpus striatum. Neurochem Int. 1984;6(1):103-8. doi: 10.1016/0197-0186(84)90033-0. PMID: 20488027.
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Cross-tissue comparisons of leptin and adiponectin: DNA methylation profiles.

Adipocyte

Houde AA, Légaré C, Hould FS, Lebel S, Marceau P, Tchernof A, Vohl MC, Hivert MF, Bouchard L.
PMID: 24719787
Adipocyte. 2014 Apr 01;3(2):132-40. doi: 10.4161/adip.28308. Epub 2014 Feb 25.

DNA methylation has been mostly studied in circulating blood cells. Although being readily accessible, metabolically active tissues such as adipose tissue would be more informative for the study of metabolic disorders. However, whether or not the blood DNA methylation...

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Houde AA, Légaré C, Hould FS, et al. Cross-tissue comparisons of leptin and adiponectin: DNA methylation profiles. Adipocyte. 2014;3(2):132-40doi: 10.4161/adip.28308.
Houde, A. A., Légaré, C., Hould, F. S., Lebel, S., Marceau, P., Tchernof, A., Vohl, M. C., Hivert, M. F., & Bouchard, L. (2014). Cross-tissue comparisons of leptin and adiponectin: DNA methylation profiles. Adipocyte, 3(2), 132-40. https://doi.org/10.4161/adip.28308
Houde, Andrée-Anne, et al. "Cross-tissue comparisons of leptin and adiponectin: DNA methylation profiles." Adipocyte vol. 3,2 (2014): 132-40. doi: https://doi.org/10.4161/adip.28308
Houde AA, Légaré C, Hould FS, Lebel S, Marceau P, Tchernof A, Vohl MC, Hivert MF, Bouchard L. Cross-tissue comparisons of leptin and adiponectin: DNA methylation profiles. Adipocyte. 2014 Apr 01;3(2):132-40. doi: 10.4161/adip.28308. Epub 2014 Feb 25. PMID: 24719787; PMCID: PMC3979878.
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Showing 1 to 12 of 196 entries