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Adolescent Alcohol Exposure Persistently Impacts Adult Neurobiology and Behavior.

Pharmacological reviews

Crews FT, Vetreno RP, Broadwater MA, Robinson DL.
PMID: 27677720
Pharmacol Rev. 2016 Oct;68(4):1074-1109. doi: 10.1124/pr.115.012138.

Adolescence is a developmental period when physical and cognitive abilities are optimized, when social skills are consolidated, and when sexuality, adolescent behaviors, and frontal cortical functions mature to adult levels. Adolescents also have unique responses to alcohol compared with...

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Crews FT, Vetreno RP, Broadwater MA, et al. Adolescent Alcohol Exposure Persistently Impacts Adult Neurobiology and Behavior. Pharmacol Rev. 2016;68(4):1074-1109doi: 10.1124/pr.115.012138.
Crews, F. T., Vetreno, R. P., Broadwater, M. A., & Robinson, D. L. (2016). Adolescent Alcohol Exposure Persistently Impacts Adult Neurobiology and Behavior. Pharmacological reviews, 68(4), 1074-1109. https://doi.org/10.1124/pr.115.012138
Crews, Fulton T, et al. "Adolescent Alcohol Exposure Persistently Impacts Adult Neurobiology and Behavior." Pharmacological reviews vol. 68,4 (2016): 1074-1109. doi: https://doi.org/10.1124/pr.115.012138
Crews FT, Vetreno RP, Broadwater MA, Robinson DL. Adolescent Alcohol Exposure Persistently Impacts Adult Neurobiology and Behavior. Pharmacol Rev. 2016 Oct;68(4):1074-1109. doi: 10.1124/pr.115.012138. PMID: 27677720; PMCID: PMC5050442.
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Addiction, adolescence, and innate immune gene induction.

Frontiers in psychiatry

Crews FT, Vetreno RP.
PMID: 21629837
Front Psychiatry. 2011 Apr 27;2:19. doi: 10.3389/fpsyt.2011.00019. eCollection 2011.

Repeated drug use/abuse amplifies psychopathology, progressively reducing frontal lobe behavioral control, and cognitive flexibility while simultaneously increasing limbic temporal lobe negative emotionality. The period of adolescence is a neurodevelopmental stage characterized by poor behavioral control as well as strong...

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Crews FT, Vetreno RP. Addiction, adolescence, and innate immune gene induction. Front Psychiatry. 2011;2:19doi: 10.3389/fpsyt.2011.00019.
Crews, F. T., & Vetreno, R. P. (2011). Addiction, adolescence, and innate immune gene induction. Frontiers in psychiatry, 219. https://doi.org/10.3389/fpsyt.2011.00019
Crews, Fulton T, and Vetreno, Ryan Peter. "Addiction, adolescence, and innate immune gene induction." Frontiers in psychiatry vol. 2 (2011): 19. doi: https://doi.org/10.3389/fpsyt.2011.00019
Crews FT, Vetreno RP. Addiction, adolescence, and innate immune gene induction. Front Psychiatry. 2011 Apr 27;2:19. doi: 10.3389/fpsyt.2011.00019. eCollection 2011. PMID: 21629837; PMCID: PMC3098669.
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Binge ethanol exposure during adolescence leads to a persistent loss of neurogenesis in the dorsal and ventral hippocampus that is associated with impaired adult cognitive functioning.

Frontiers in neuroscience

Vetreno RP, Crews FT.
PMID: 25729346
Front Neurosci. 2015 Feb 12;9:35. doi: 10.3389/fnins.2015.00035. eCollection 2015.

Adolescence is a developmental period that coincides with the maturation of adult cognitive faculties. Binge drinking is common during adolescence and can impact brain maturation. Using a rodent model of adolescent intermittent ethanol (AIE; 5.0 g/kg, i.g., 20% EtOH...

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Vetreno RP, Crews FT. Binge ethanol exposure during adolescence leads to a persistent loss of neurogenesis in the dorsal and ventral hippocampus that is associated with impaired adult cognitive functioning. Front Neurosci. 2015;9:35doi: 10.3389/fnins.2015.00035.
Vetreno, R. P., & Crews, F. T. (2015). Binge ethanol exposure during adolescence leads to a persistent loss of neurogenesis in the dorsal and ventral hippocampus that is associated with impaired adult cognitive functioning. Frontiers in neuroscience, 935. https://doi.org/10.3389/fnins.2015.00035
Vetreno, Ryan P, and Crews, Fulton T. "Binge ethanol exposure during adolescence leads to a persistent loss of neurogenesis in the dorsal and ventral hippocampus that is associated with impaired adult cognitive functioning." Frontiers in neuroscience vol. 9 (2015): 35. doi: https://doi.org/10.3389/fnins.2015.00035
Vetreno RP, Crews FT. Binge ethanol exposure during adolescence leads to a persistent loss of neurogenesis in the dorsal and ventral hippocampus that is associated with impaired adult cognitive functioning. Front Neurosci. 2015 Feb 12;9:35. doi: 10.3389/fnins.2015.00035. eCollection 2015. PMID: 25729346; PMCID: PMC4325907.
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Loss of Basal Forebrain Cholinergic Neurons Following Adolescent Binge Ethanol Exposure: Recovery With the Cholinesterase Inhibitor Galantamine.

Frontiers in behavioral neuroscience

Crews FT, Fisher R, Deason C, Vetreno RP.
PMID: 33716687
Front Behav Neurosci. 2021 Feb 26;15:652494. doi: 10.3389/fnbeh.2021.652494. eCollection 2021.

Binge drinking and alcohol abuse are common during adolescence and cause both cognitive deficits and lasting cholinergic pathology in the adult basal forebrain. Acetylcholine is anti-inflammatory and studies using the preclinical adolescent intermittent ethanol (AIE; 5.0 g/kg, i.g., 2...

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Crews FT, Fisher R, Deason C, et al. Loss of Basal Forebrain Cholinergic Neurons Following Adolescent Binge Ethanol Exposure: Recovery With the Cholinesterase Inhibitor Galantamine. Front Behav Neurosci. 2021;15:652494doi: 10.3389/fnbeh.2021.652494.
Crews, F. T., Fisher, R., Deason, C., & Vetreno, R. P. (2021). Loss of Basal Forebrain Cholinergic Neurons Following Adolescent Binge Ethanol Exposure: Recovery With the Cholinesterase Inhibitor Galantamine. Frontiers in behavioral neuroscience, 15652494. https://doi.org/10.3389/fnbeh.2021.652494
Crews, Fulton T, et al. "Loss of Basal Forebrain Cholinergic Neurons Following Adolescent Binge Ethanol Exposure: Recovery With the Cholinesterase Inhibitor Galantamine." Frontiers in behavioral neuroscience vol. 15 (2021): 652494. doi: https://doi.org/10.3389/fnbeh.2021.652494
Crews FT, Fisher R, Deason C, Vetreno RP. Loss of Basal Forebrain Cholinergic Neurons Following Adolescent Binge Ethanol Exposure: Recovery With the Cholinesterase Inhibitor Galantamine. Front Behav Neurosci. 2021 Feb 26;15:652494. doi: 10.3389/fnbeh.2021.652494. eCollection 2021. PMID: 33716687; PMCID: PMC7953159.
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Showing 1 to 4 of 4 entries