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The role of FADD in pancreatic cancer cell proliferation and drug resistance.

Oncology letters

Zhang R, Liu Y, Hammache K, He L, Zhu B, Cheng W, Hua ZC.
PMID: 28454341
Oncol Lett. 2017 Mar;13(3):1899-1904. doi: 10.3892/ol.2017.5636. Epub 2017 Jan 25.

Pancreatic cancer has one of the poorest patient outcomes and is highly resistant to chemotherapy. Identifying the molecular mechanisms involved in drug resistance is critical in the development of novel strategies to treat pancreatic cancer. The results of the...

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Zhang R, Liu Y, Hammache K, et al. The role of FADD in pancreatic cancer cell proliferation and drug resistance. Oncol Lett. 2017;13(3):1899-1904doi: 10.3892/ol.2017.5636.
Zhang, R., Liu, Y., Hammache, K., He, L., Zhu, B., Cheng, W., & Hua, Z. C. (2017). The role of FADD in pancreatic cancer cell proliferation and drug resistance. Oncology letters, 13(3), 1899-1904. https://doi.org/10.3892/ol.2017.5636
Zhang, Rong, et al. "The role of FADD in pancreatic cancer cell proliferation and drug resistance." Oncology letters vol. 13,3 (2017): 1899-1904. doi: https://doi.org/10.3892/ol.2017.5636
Zhang R, Liu Y, Hammache K, He L, Zhu B, Cheng W, Hua ZC. The role of FADD in pancreatic cancer cell proliferation and drug resistance. Oncol Lett. 2017 Mar;13(3):1899-1904. doi: 10.3892/ol.2017.5636. Epub 2017 Jan 25. PMID: 28454341; PMCID: PMC5403491.
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Engineered FADD Induces Apoptosis via an Artificial Death-Inducing Signaling Complex (DISC).

International journal of biomedical science : IJBS

Suzuki E, Takashina T, Nakayama M.
PMID: 23675143
Int J Biomed Sci. 2009 Sep;5(3):237-45.

An engineered Fas-associated death domain protein (FADD), 2DEDplusE-made previously by fusing the tandem DEDs of FADD to the E protein of lambda phage-greatly enhances apoptosis-inducing activity in adherent cells in vitro. To investigate the mechanism of apoptosis-inducing activity of...

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Suzuki E, Takashina T, Nakayama M. Engineered FADD Induces Apoptosis via an Artificial Death-Inducing Signaling Complex (DISC). Int J Biomed Sci. 2009;5(3):237-45
Suzuki, E., Takashina, T., & Nakayama, M. (2009). Engineered FADD Induces Apoptosis via an Artificial Death-Inducing Signaling Complex (DISC). International journal of biomedical science : IJBS, 5(3), 237-45.
Suzuki, Emiko, et al. "Engineered FADD Induces Apoptosis via an Artificial Death-Inducing Signaling Complex (DISC)." International journal of biomedical science : IJBS vol. 5,3 (2009): 237-45.
Suzuki E, Takashina T, Nakayama M. Engineered FADD Induces Apoptosis via an Artificial Death-Inducing Signaling Complex (DISC). Int J Biomed Sci. 2009 Sep;5(3):237-45. PMID: 23675143; PMCID: PMC3614778.
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Long-chain PUFA ameliorate ETEC-induced intestinal inflammation and cell injury by modulating pyroptosis and necroptosis signaling pathways in IPEC-1 cells.

The British journal of nutrition

Xiao K, Yang Y, Zhang Y, Lv Q, Huang F, Wang D, Zhao J, Liu Y.
PMID: 34915950
Br J Nutr. 2021 Dec 17;1-36. doi: 10.1017/S0007114521005092. Epub 2021 Dec 17.

This study was aimed to investigate whether eicosapentaenoic acid (EPA) and arachidonic acid (ARA), the representative n-3 or n-6 polyunsaturated fatty acids (PUFA), could alleviate enterotoxigenic Escherichia coli (ETEC) K88-induced inflammation and injury of intestinal porcine epithelial cells 1...

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Xiao K, Yang Y, Zhang Y, et al. Long-chain PUFA ameliorate ETEC-induced intestinal inflammation and cell injury by modulating pyroptosis and necroptosis signaling pathways in IPEC-1 cells. Br J Nutr. 2021;1-36doi: 10.1017/S0007114521005092.
Xiao, K., Yang, Y., Zhang, Y., Lv, Q., Huang, F., Wang, D., Zhao, J., & Liu, Y. (2021). Long-chain PUFA ameliorate ETEC-induced intestinal inflammation and cell injury by modulating pyroptosis and necroptosis signaling pathways in IPEC-1 cells. The British journal of nutrition, 1-36. https://doi.org/10.1017/S0007114521005092
Xiao, Kan, et al. "Long-chain PUFA ameliorate ETEC-induced intestinal inflammation and cell injury by modulating pyroptosis and necroptosis signaling pathways in IPEC-1 cells." The British journal of nutrition vol. (2021): 1-36. doi: https://doi.org/10.1017/S0007114521005092
Xiao K, Yang Y, Zhang Y, Lv Q, Huang F, Wang D, Zhao J, Liu Y. Long-chain PUFA ameliorate ETEC-induced intestinal inflammation and cell injury by modulating pyroptosis and necroptosis signaling pathways in IPEC-1 cells. Br J Nutr. 2021 Dec 17;1-36. doi: 10.1017/S0007114521005092. Epub 2021 Dec 17. PMID: 34915950.
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Dasatinib promotes TRAIL-mediated apoptosis by upregulating CHOP-dependent death receptor 5 in gastric cancer.

FEBS open bio

Wang X, Xue Q, Wu L, Wang B, Liang H.
PMID: 29744288
FEBS Open Bio. 2018 Mar 23;8(5):732-742. doi: 10.1002/2211-5463.12404. eCollection 2018 May.

Dasatinib, a tyrosine kinase inhibitor, has been approved for first-line treatment of leukemia and has also been evaluated for use in numerous other cancers. However, its role in gastric cancer (GC) remains unclear. Therefore, the aim of this study...

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Wang X, Xue Q, Wu L, et al. Dasatinib promotes TRAIL-mediated apoptosis by upregulating CHOP-dependent death receptor 5 in gastric cancer. FEBS Open Bio. 2018;8(5):732-742doi: 10.1002/2211-5463.12404.
Wang, X., Xue, Q., Wu, L., Wang, B., & Liang, H. (2018). Dasatinib promotes TRAIL-mediated apoptosis by upregulating CHOP-dependent death receptor 5 in gastric cancer. FEBS open bio, 8(5), 732-742. https://doi.org/10.1002/2211-5463.12404
Wang, Xiaona, et al. "Dasatinib promotes TRAIL-mediated apoptosis by upregulating CHOP-dependent death receptor 5 in gastric cancer." FEBS open bio vol. 8,5 (2018): 732-742. doi: https://doi.org/10.1002/2211-5463.12404
Wang X, Xue Q, Wu L, Wang B, Liang H. Dasatinib promotes TRAIL-mediated apoptosis by upregulating CHOP-dependent death receptor 5 in gastric cancer. FEBS Open Bio. 2018 Mar 23;8(5):732-742. doi: 10.1002/2211-5463.12404. eCollection 2018 May. PMID: 29744288; PMCID: PMC5929929.
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Showing 1 to 4 of 4 entries