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Showing 1 to 12 of 47 entries
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Vitamin A Transport Mechanism of the Multitransmembrane Cell-Surface Receptor STRA6.

Membranes

Kawaguchi R, Zhong M, Kassai M, Ter-Stepanian M, Sun H.
PMID: 26343735
Membranes (Basel). 2015 Aug 28;5(3):425-53. doi: 10.3390/membranes5030425.

Vitamin A has biological functions as diverse as sensing light for vision, regulating stem cell differentiation, maintaining epithelial integrity, promoting immune competency, regulating learning and memory, and acting as a key developmental morphogen. Vitamin A derivatives have also been...

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Kawaguchi R, Zhong M, Kassai M, et al. Vitamin A Transport Mechanism of the Multitransmembrane Cell-Surface Receptor STRA6. Membranes (Basel). 2015;5(3):425-53doi: 10.3390/membranes5030425.
Kawaguchi, R., Zhong, M., Kassai, M., Ter-Stepanian, M., & Sun, H. (2015). Vitamin A Transport Mechanism of the Multitransmembrane Cell-Surface Receptor STRA6. Membranes, 5(3), 425-53. https://doi.org/10.3390/membranes5030425
Kawaguchi, Riki, et al. "Vitamin A Transport Mechanism of the Multitransmembrane Cell-Surface Receptor STRA6." Membranes vol. 5,3 (2015): 425-53. doi: https://doi.org/10.3390/membranes5030425
Kawaguchi R, Zhong M, Kassai M, Ter-Stepanian M, Sun H. Vitamin A Transport Mechanism of the Multitransmembrane Cell-Surface Receptor STRA6. Membranes (Basel). 2015 Aug 28;5(3):425-53. doi: 10.3390/membranes5030425. PMID: 26343735; PMCID: PMC4584289.
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Assessment of anti-inflammatory and anti-arthritic properties of Acmella uliginosa (Sw.) Cass. based on experiments in arthritic rat models and qualitative gas chromatography-mass spectrometry analyses.

Journal of intercultural ethnopharmacology

Paul S, Sarkar S, Dutta T, Bhattacharjee S.
PMID: 27366352
J Intercult Ethnopharmacol. 2016 May 30;5(3):257-62. doi: 10.5455/jice.20160521010145. eCollection 2016.

AIM: The principle objective of the study was to explore the anti-arthritic properties of Acmella uliginosa (AU) (Sw.) Cass. flower in a rat model and to identify potential anti-inflammatory compounds derived from flower extracts. The synergistic role played by...

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Paul S, Sarkar S, Dutta T, et al. Assessment of anti-inflammatory and anti-arthritic properties of Acmella uliginosa (Sw.) Cass. based on experiments in arthritic rat models and qualitative gas chromatography-mass spectrometry analyses. J Intercult Ethnopharmacol. 2016;5(3):257-62doi: 10.5455/jice.20160521010145.
Paul, S., Sarkar, S., Dutta, T., & Bhattacharjee, S. (2016). Assessment of anti-inflammatory and anti-arthritic properties of Acmella uliginosa (Sw.) Cass. based on experiments in arthritic rat models and qualitative gas chromatography-mass spectrometry analyses. Journal of intercultural ethnopharmacology, 5(3), 257-62. https://doi.org/10.5455/jice.20160521010145
Paul, Subhashis, et al. "Assessment of anti-inflammatory and anti-arthritic properties of Acmella uliginosa (Sw.) Cass. based on experiments in arthritic rat models and qualitative gas chromatography-mass spectrometry analyses." Journal of intercultural ethnopharmacology vol. 5,3 (2016): 257-62. doi: https://doi.org/10.5455/jice.20160521010145
Paul S, Sarkar S, Dutta T, Bhattacharjee S. Assessment of anti-inflammatory and anti-arthritic properties of Acmella uliginosa (Sw.) Cass. based on experiments in arthritic rat models and qualitative gas chromatography-mass spectrometry analyses. J Intercult Ethnopharmacol. 2016 May 30;5(3):257-62. doi: 10.5455/jice.20160521010145. eCollection 2016. PMID: 27366352; PMCID: PMC4927131.
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Gateways to clinical trials.

Methods and findings in experimental and clinical pharmacology

Bayés M, Rabasseda X, Prous JR.
PMID: 15148527
Methods Find Exp Clin Pharmacol. 2004 Apr;26(3):211-44.

Gateways to Clinical Trials is a guide to the most recent clinical trials in current literature and congresses. The data in the following tables has been retrieved from the Clinical Studies Knowledge Area of Prous Science Integrity(R), the drug...

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Bayés M, Rabasseda X, Prous JR. Gateways to clinical trials. Methods Find Exp Clin Pharmacol. 2004;26(3):211-44
Bayés, M., Rabasseda, X., & Prous, J. R. (2004). Gateways to clinical trials. Methods and findings in experimental and clinical pharmacology, 26(3), 211-44.
Bayés, M, et al. "Gateways to clinical trials." Methods and findings in experimental and clinical pharmacology vol. 26,3 (2004): 211-44.
Bayés M, Rabasseda X, Prous JR. Gateways to clinical trials. Methods Find Exp Clin Pharmacol. 2004 Apr;26(3):211-44. PMID: 15148527.
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Gateways to clinical trials.

Methods and findings in experimental and clinical pharmacology

Bayes M, Rabasseda X, Prous JR.
PMID: 16845450
Methods Find Exp Clin Pharmacol. 2006 Jun;28(5):323-43.

Gateways to Clinical Trials are a guide to the most recent clinical trials in current literature and congresses. The data in the following tables have been retrieved from the Clinical Trials Knowledge Area of Prous Science Integrity, the drug...

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Bayes M, Rabasseda X, Prous JR. Gateways to clinical trials. Methods Find Exp Clin Pharmacol. 2006;28(5):323-43
Bayes, M., Rabasseda, X., & Prous, J. R. (2006). Gateways to clinical trials. Methods and findings in experimental and clinical pharmacology, 28(5), 323-43.
Bayes, M, et al. "Gateways to clinical trials." Methods and findings in experimental and clinical pharmacology vol. 28,5 (2006): 323-43.
Bayes M, Rabasseda X, Prous JR. Gateways to clinical trials. Methods Find Exp Clin Pharmacol. 2006 Jun;28(5):323-43. PMID: 16845450.
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Role for PKC δ in Fenretinide-Mediated Apoptosis in Lymphoid Leukemia Cells.

Journal of signal transduction

Ruvolo VR, Karanjeet KB, Schuster TF, Brown R, Deng Y, Hinchcliffe E, Ruvolo PP.
PMID: 20844597
J Signal Transduct. 2010 Jan 01;2010:584657. doi: 10.1155/2010/584657.

The synthetic Vitamin A analog fenretinide is a promising chemotherapeutic agent. In the current paper, the role of PKC δ was examined in fenretinide-induced apoptosis in lymphoid leukemia cells. Levels of proapoptotic cleaved PKC δ positively correlated with drug...

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Ruvolo VR, Karanjeet KB, Schuster TF, et al. Role for PKC δ in Fenretinide-Mediated Apoptosis in Lymphoid Leukemia Cells. J Signal Transduct. 2010;2010:584657doi: 10.1155/2010/584657.
Ruvolo, V. R., Karanjeet, K. B., Schuster, T. F., Brown, R., Deng, Y., Hinchcliffe, E., & Ruvolo, P. P. (2010). Role for PKC δ in Fenretinide-Mediated Apoptosis in Lymphoid Leukemia Cells. Journal of signal transduction, 2010584657. https://doi.org/10.1155/2010/584657
Ruvolo, Vivian R, et al. "Role for PKC δ in Fenretinide-Mediated Apoptosis in Lymphoid Leukemia Cells." Journal of signal transduction vol. 2010 (2010): 584657. doi: https://doi.org/10.1155/2010/584657
Ruvolo VR, Karanjeet KB, Schuster TF, Brown R, Deng Y, Hinchcliffe E, Ruvolo PP. Role for PKC δ in Fenretinide-Mediated Apoptosis in Lymphoid Leukemia Cells. J Signal Transduct. 2010 Jan 01;2010:584657. doi: 10.1155/2010/584657. PMID: 20844597; PMCID: PMC2938797.
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American Society of Clinical Oncology Clinical Practice Guideline Update on the Use of Pharmacologic Interventions Including Tamoxifen, Raloxifene, and Aromatase Inhibition for Breast Cancer Risk Reduction.

Journal of oncology practice

[No authors listed]
PMID: 29452019
J Oncol Pract. 2009 Jul;5(4):196-199. doi: 10.1200/JOP.0948502.

ASCO's update to its Clinical Practice Guideline on the use of pharmacologic interventions to reduce the risk of breast cancer addresses the roles of tamoxifen, raloxifene, aromatase inhibitors, and fenretinide in reducing risk for the disease.

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American Society of Clinical Oncology Clinical Practice Guideline Update on the Use of Pharmacologic Interventions Including Tamoxifen, Raloxifene, and Aromatase Inhibition for Breast Cancer Risk Reduction. J Oncol Pract. 2009;5(4):196-199doi: 10.1200/JOP.0948502.
(2009). American Society of Clinical Oncology Clinical Practice Guideline Update on the Use of Pharmacologic Interventions Including Tamoxifen, Raloxifene, and Aromatase Inhibition for Breast Cancer Risk Reduction. Journal of oncology practice, 5(4), 196-199. https://doi.org/10.1200/JOP.0948502
"American Society of Clinical Oncology Clinical Practice Guideline Update on the Use of Pharmacologic Interventions Including Tamoxifen, Raloxifene, and Aromatase Inhibition for Breast Cancer Risk Reduction." Journal of oncology practice vol. 5,4 (2009): 196-199. doi: https://doi.org/10.1200/JOP.0948502
American Society of Clinical Oncology Clinical Practice Guideline Update on the Use of Pharmacologic Interventions Including Tamoxifen, Raloxifene, and Aromatase Inhibition for Breast Cancer Risk Reduction. J Oncol Pract. 2009 Jul;5(4):196-199. doi: 10.1200/JOP.0948502. PMID: 29452019; PMCID: PMC2795413.
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High-fat diet exacerbates cognitive and metabolic abnormalities in neuronal BACE1 knock-in mice - partial prevention by Fenretinide.

Nutritional neuroscience

Plucińska K, Mody N, Dekeryte R, Shearer K, Mcilroy GD, Delibegovic M, Platt B.
PMID: 32862802
Nutr Neurosci. 2020 Aug 29;1-18. doi: 10.1080/1028415X.2020.1806190. Epub 2020 Aug 29.

No abstract available.

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Plucińska K, Mody N, Dekeryte R, et al. High-fat diet exacerbates cognitive and metabolic abnormalities in neuronal BACE1 knock-in mice - partial prevention by Fenretinide. Nutr Neurosci. 2020;1-18doi: 10.1080/1028415X.2020.1806190.
Plucińska, K., Mody, N., Dekeryte, R., Shearer, K., Mcilroy, G. D., Delibegovic, M., & Platt, B. (2020). High-fat diet exacerbates cognitive and metabolic abnormalities in neuronal BACE1 knock-in mice - partial prevention by Fenretinide. Nutritional neuroscience, 1-18. https://doi.org/10.1080/1028415X.2020.1806190
Plucińska, Kaja, et al. "High-fat diet exacerbates cognitive and metabolic abnormalities in neuronal BACE1 knock-in mice - partial prevention by Fenretinide." Nutritional neuroscience vol. (2020): 1-18. doi: https://doi.org/10.1080/1028415X.2020.1806190
Plucińska K, Mody N, Dekeryte R, Shearer K, Mcilroy GD, Delibegovic M, Platt B. High-fat diet exacerbates cognitive and metabolic abnormalities in neuronal BACE1 knock-in mice - partial prevention by Fenretinide. Nutr Neurosci. 2020 Aug 29;1-18. doi: 10.1080/1028415X.2020.1806190. Epub 2020 Aug 29. PMID: 32862802.
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Retinal toxicities of systemic anticancer drugs.

Survey of ophthalmology

Arora S, Surakiatchanukul T, Arora T, Errera MH, Agrawal H, Lupidi M, Chhablani J.
PMID: 34048859
Surv Ophthalmol. 2022 Jan-Feb;67(1):97-148. doi: 10.1016/j.survophthal.2021.05.007. Epub 2021 May 26.

Newer anticancer drugs have revolutionized cancer treatment in the last decade, but conventional chemotherapy still occupies a central position in many cancers, with combination therapy and newer methods of delivery increasing their efficacy while minimizing toxicities. We discuss the...

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Arora S, Surakiatchanukul T, Arora T, et al. Retinal toxicities of systemic anticancer drugs. Surv Ophthalmol. 2021;67(1):97-148doi: 10.1016/j.survophthal.2021.05.007.
Arora, S., Surakiatchanukul, T., Arora, T., Errera, M. H., Agrawal, H., Lupidi, M., & Chhablani, J. (2022). Retinal toxicities of systemic anticancer drugs. Survey of ophthalmology, 67(1), 97-148. https://doi.org/10.1016/j.survophthal.2021.05.007
Arora, Supriya, et al. "Retinal toxicities of systemic anticancer drugs." Survey of ophthalmology vol. 67,1 (2022): 97-148. doi: https://doi.org/10.1016/j.survophthal.2021.05.007
Arora S, Surakiatchanukul T, Arora T, Errera MH, Agrawal H, Lupidi M, Chhablani J. Retinal toxicities of systemic anticancer drugs. Surv Ophthalmol. 2022 Jan-Feb;67(1):97-148. doi: 10.1016/j.survophthal.2021.05.007. Epub 2021 May 26. PMID: 34048859.
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Fenretinide-induced Apoptosis of Acute Myeloid Leukemia Cells via NR4A1 Translocation into Mitochondria and Bcl-2 Transformation.

Journal of Cancer

Xiong J, Kuang X, Lu T, Liu X, Cheng B, Wang W, Wei D, Li X, Zhang Z, Fang Q, Wu D, Wang J.
PMID: 31839811
J Cancer. 2019 Nov 01;10(27):6767-6778. doi: 10.7150/jca.32167. eCollection 2019.

No abstract available.

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Xiong J, Kuang X, Lu T, et al. Fenretinide-induced Apoptosis of Acute Myeloid Leukemia Cells via NR4A1 Translocation into Mitochondria and Bcl-2 Transformation. J Cancer. 2019;10(27):6767-6778doi: 10.7150/jca.32167.
Xiong, J., Kuang, X., Lu, T., Liu, X., Cheng, B., Wang, W., Wei, D., Li, X., Zhang, Z., Fang, Q., Wu, D., & Wang, J. (2019). Fenretinide-induced Apoptosis of Acute Myeloid Leukemia Cells via NR4A1 Translocation into Mitochondria and Bcl-2 Transformation. Journal of Cancer, 10(27), 6767-6778. https://doi.org/10.7150/jca.32167
Xiong, Jie, et al. "Fenretinide-induced Apoptosis of Acute Myeloid Leukemia Cells via NR4A1 Translocation into Mitochondria and Bcl-2 Transformation." Journal of Cancer vol. 10,27 (2019): 6767-6778. doi: https://doi.org/10.7150/jca.32167
Xiong J, Kuang X, Lu T, Liu X, Cheng B, Wang W, Wei D, Li X, Zhang Z, Fang Q, Wu D, Wang J. Fenretinide-induced Apoptosis of Acute Myeloid Leukemia Cells via NR4A1 Translocation into Mitochondria and Bcl-2 Transformation. J Cancer. 2019 Nov 01;10(27):6767-6778. doi: 10.7150/jca.32167. eCollection 2019. PMID: 31839811; PMCID: PMC6909957.
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mRNA-Sequencing Identifies Liver as a Potential Target Organ for Triphenyl Phosphate in Embryonic Zebrafish.

Toxicological sciences : an official journal of the Society of Toxicology

Reddam A, Mitchell CA, Dasgupta S, Kirkwood JS, Vollaro A, Hur M, Volz DC.
PMID: 31368501
Toxicol Sci. 2019 Jul 31; doi: 10.1093/toxsci/kfz169. Epub 2019 Jul 31.

Triphenyl phosphate (TPHP) is a commonly used organophosphate flame retardant and plasticizer in the United States. Using zebrafish as a model, the overall objective of this study was to identify potential organs that might be targeted by TPHP during...

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Reddam A, Mitchell CA, Dasgupta S, et al. mRNA-Sequencing Identifies Liver as a Potential Target Organ for Triphenyl Phosphate in Embryonic Zebrafish. Toxicol Sci. 2019;doi: 10.1093/toxsci/kfz169.
Reddam, A., Mitchell, C. A., Dasgupta, S., Kirkwood, J. S., Vollaro, A., Hur, M., & Volz, D. C. (2019). mRNA-Sequencing Identifies Liver as a Potential Target Organ for Triphenyl Phosphate in Embryonic Zebrafish. Toxicological sciences : an official journal of the Society of Toxicology, . https://doi.org/10.1093/toxsci/kfz169
Reddam, Aalekhya, et al. "mRNA-Sequencing Identifies Liver as a Potential Target Organ for Triphenyl Phosphate in Embryonic Zebrafish." Toxicological sciences : an official journal of the Society of Toxicology vol. (2019). doi: https://doi.org/10.1093/toxsci/kfz169
Reddam A, Mitchell CA, Dasgupta S, Kirkwood JS, Vollaro A, Hur M, Volz DC. mRNA-Sequencing Identifies Liver as a Potential Target Organ for Triphenyl Phosphate in Embryonic Zebrafish. Toxicol Sci. 2019 Jul 31; doi: 10.1093/toxsci/kfz169. Epub 2019 Jul 31. PMID: 31368501; PMCID: PMC6813745.
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Fenretinide binding to the lysosomal protein saposin D alters ceramide solubilization and hydrolysis.

RSC medicinal chemistry

Milliken BT, Melegari L, Smith GL, Grohn K, Wolfe AJ, Moody K, Bou-Abdallah F, Doyle RP.
PMID: 33479697
RSC Med Chem. 2020 Jul 14;11(9):1048-1052. doi: 10.1039/d0md00182a. eCollection 2020 Sep 01.

Fenretinide is a synthetic retinoid pharmaceutical linked to ceramide build-up

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Milliken BT, Melegari L, Smith GL, et al. Fenretinide binding to the lysosomal protein saposin D alters ceramide solubilization and hydrolysis. RSC Med Chem. 2020;11(9):1048-1052doi: 10.1039/d0md00182a.
Milliken, B. T., Melegari, L., Smith, G. L., Grohn, K., Wolfe, A. J., Moody, K., Bou-Abdallah, F., & Doyle, R. P. (2020). Fenretinide binding to the lysosomal protein saposin D alters ceramide solubilization and hydrolysis. RSC medicinal chemistry, 11(9), 1048-1052. https://doi.org/10.1039/d0md00182a
Milliken, Brandon T, et al. "Fenretinide binding to the lysosomal protein saposin D alters ceramide solubilization and hydrolysis." RSC medicinal chemistry vol. 11,9 (2020): 1048-1052. doi: https://doi.org/10.1039/d0md00182a
Milliken BT, Melegari L, Smith GL, Grohn K, Wolfe AJ, Moody K, Bou-Abdallah F, Doyle RP. Fenretinide binding to the lysosomal protein saposin D alters ceramide solubilization and hydrolysis. RSC Med Chem. 2020 Jul 14;11(9):1048-1052. doi: 10.1039/d0md00182a. eCollection 2020 Sep 01. PMID: 33479697; PMCID: PMC7513591.
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CFTR Correctors and Antioxidants Partially Normalize Lipid Imbalance but not Abnormal Basal Inflammatory Cytokine Profile in CF Bronchial Epithelial Cells.

Frontiers in physiology

Veltman M, De Sanctis JB, Stolarczyk M, Klymiuk N, Bähr A, Brouwer RW, Oole E, Shah J, Ozdian T, Liao J, Martini C, Radzioch D, Hanrahan JW, Scholte BJ.
PMID: 33613309
Front Physiol. 2021 Feb 04;12:619442. doi: 10.3389/fphys.2021.619442. eCollection 2021.

A deficiency in cystic fibrosis transmembrane conductance regulator (CFTR) function in CF leads to chronic lung disease. CF is associated with abnormalities in fatty acids, ceramides, and cholesterol, their relationship with CF lung pathology is not completely understood. Therefore,...

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Veltman M, De Sanctis JB, Stolarczyk M, et al. CFTR Correctors and Antioxidants Partially Normalize Lipid Imbalance but not Abnormal Basal Inflammatory Cytokine Profile in CF Bronchial Epithelial Cells. Front Physiol. 2021;12:619442doi: 10.3389/fphys.2021.619442.
Veltman, M., De Sanctis, J. B., Stolarczyk, M., Klymiuk, N., Bähr, A., Brouwer, R. W., Oole, E., Shah, J., Ozdian, T., Liao, J., Martini, C., Radzioch, D., Hanrahan, J. W., & Scholte, B. J. (2021). CFTR Correctors and Antioxidants Partially Normalize Lipid Imbalance but not Abnormal Basal Inflammatory Cytokine Profile in CF Bronchial Epithelial Cells. Frontiers in physiology, 12619442. https://doi.org/10.3389/fphys.2021.619442
Veltman, Mieke, et al. "CFTR Correctors and Antioxidants Partially Normalize Lipid Imbalance but not Abnormal Basal Inflammatory Cytokine Profile in CF Bronchial Epithelial Cells." Frontiers in physiology vol. 12 (2021): 619442. doi: https://doi.org/10.3389/fphys.2021.619442
Veltman M, De Sanctis JB, Stolarczyk M, Klymiuk N, Bähr A, Brouwer RW, Oole E, Shah J, Ozdian T, Liao J, Martini C, Radzioch D, Hanrahan JW, Scholte BJ. CFTR Correctors and Antioxidants Partially Normalize Lipid Imbalance but not Abnormal Basal Inflammatory Cytokine Profile in CF Bronchial Epithelial Cells. Front Physiol. 2021 Feb 04;12:619442. doi: 10.3389/fphys.2021.619442. eCollection 2021. PMID: 33613309; PMCID: PMC7891400.
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Showing 1 to 12 of 47 entries