Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 12 entries
Sorted by: Best Match Show Resources per page
[Effects of early malnutrition and nutritional rehabilitation in rats].

Jornal de pediatria

Nunes ML, Batista BB, Micheli F, Batistella V.
PMID: 14647810
J Pediatr (Rio J). 2002 Jan-Feb;78(1):39-44.

OBJECTIVE: To verify the effects of malnutrition and nutritional rehabilitation regarding seizure threshold, body weight and brain weight in rats. METHODS: Pregnant Wistar rats and their pups were used. Part of the rat pups were submitted to a malnutrition...

Cite
Nunes ML, Batista BB, Micheli F, et al. Efeitos da desnutrição precoce e reabilitação nutricional em ratos. [Effects of early malnutrition and nutritional rehabilitation in rats]. J Pediatr (Rio J). 2002;78(1):39-44
Nunes, M. L., Batista, B. B., Micheli, F., & Batistella, V. (2002). Efeitos da desnutrição precoce e reabilitação nutricional em ratos. [Effects of early malnutrition and nutritional rehabilitation in rats]. Jornal de pediatria, 78(1), 39-44.
Nunes, Magda Lahorgue, et al. "Efeitos da desnutrição precoce e reabilitação nutricional em ratos." [Effects of early malnutrition and nutritional rehabilitation in rats]. Jornal de pediatria vol. 78,1 (2002): 39-44.
Nunes ML, Batista BB, Micheli F, Batistella V. Efeitos da desnutrição precoce e reabilitação nutricional em ratos. [Effects of early malnutrition and nutritional rehabilitation in rats]. J Pediatr (Rio J). 2002 Jan-Feb;78(1):39-44. Portuguese. PMID: 14647810.
Copy Download .nbib Format: NLM
Zinc/CaMK II Associated-Mitophagy Signaling Contributed to Hippocampal Mossy Fiber Sprouting and Cognitive Deficits Following Neonatal Seizures and Its Regulation by Chronic Leptin Treatment.

Frontiers in neurology

Li LL, Jin MF, Ni H.
PMID: 30319532
Front Neurol. 2018 Sep 26;9:802. doi: 10.3389/fneur.2018.00802. eCollection 2018.

The role of leptin in the pathogenesis of epilepsy is getting more and more attention in clinical and basic research. Although there are data indicating neuroprotective effects of elevated serum/brain leptin levels following acute seizures, no study to date...

Cite
Li LL, Jin MF, Ni H. Zinc/CaMK II Associated-Mitophagy Signaling Contributed to Hippocampal Mossy Fiber Sprouting and Cognitive Deficits Following Neonatal Seizures and Its Regulation by Chronic Leptin Treatment. Front Neurol. 2018;9:802doi: 10.3389/fneur.2018.00802.
Li, L. L., Jin, M. F., & Ni, H. (2018). Zinc/CaMK II Associated-Mitophagy Signaling Contributed to Hippocampal Mossy Fiber Sprouting and Cognitive Deficits Following Neonatal Seizures and Its Regulation by Chronic Leptin Treatment. Frontiers in neurology, 9802. https://doi.org/10.3389/fneur.2018.00802
Li, Li-Li, et al. "Zinc/CaMK II Associated-Mitophagy Signaling Contributed to Hippocampal Mossy Fiber Sprouting and Cognitive Deficits Following Neonatal Seizures and Its Regulation by Chronic Leptin Treatment." Frontiers in neurology vol. 9 (2018): 802. doi: https://doi.org/10.3389/fneur.2018.00802
Li LL, Jin MF, Ni H. Zinc/CaMK II Associated-Mitophagy Signaling Contributed to Hippocampal Mossy Fiber Sprouting and Cognitive Deficits Following Neonatal Seizures and Its Regulation by Chronic Leptin Treatment. Front Neurol. 2018 Sep 26;9:802. doi: 10.3389/fneur.2018.00802. eCollection 2018. PMID: 30319532; PMCID: PMC6168633.
Copy Download .nbib Format: NLM
Dissociation of spontaneous seizures and brainstem seizure thresholds in mice exposed to eight flurothyl-induced generalized seizures.

Epilepsia open

Kadiyala SB, Ferland RJ.
PMID: 28825051
Epilepsia Open. 2017 Mar;2(1):48-58. doi: 10.1002/epi4.12031. Epub 2016 Dec 19.

OBJECTIVE: C57BL/6J mice exposed to eight flurothyl-induced generalized clonic seizures exhibit a change in seizure phenotype following a 28-day incubation period and subsequent flurothyl rechallenge. Mice now develop a complex seizure semiology originating in the forebrain and propagating into...

Cite
Kadiyala SB, Ferland RJ. Dissociation of spontaneous seizures and brainstem seizure thresholds in mice exposed to eight flurothyl-induced generalized seizures. Epilepsia Open. 2016;2(1):48-58doi: 10.1002/epi4.12031.
Kadiyala, S. B., & Ferland, R. J. (2017). Dissociation of spontaneous seizures and brainstem seizure thresholds in mice exposed to eight flurothyl-induced generalized seizures. Epilepsia open, 2(1), 48-58. https://doi.org/10.1002/epi4.12031
Kadiyala, Sridhar B, and Ferland, Russell J. "Dissociation of spontaneous seizures and brainstem seizure thresholds in mice exposed to eight flurothyl-induced generalized seizures." Epilepsia open vol. 2,1 (2017): 48-58. doi: https://doi.org/10.1002/epi4.12031
Kadiyala SB, Ferland RJ. Dissociation of spontaneous seizures and brainstem seizure thresholds in mice exposed to eight flurothyl-induced generalized seizures. Epilepsia Open. 2017 Mar;2(1):48-58. doi: 10.1002/epi4.12031. Epub 2016 Dec 19. PMID: 28825051; PMCID: PMC5560332.
Copy Download .nbib Format: NLM
The Repeated Flurothyl Seizure Model in Mice.

Bio-protocol

Ferland RJ.
PMID: 28748202
Bio Protoc. 2017 Jun 05;7(11). doi: 10.21769/BioProtoc.2309.

Development of spontaneous seizures is the hallmark of human epilepsy. There is a critical need for new epilepsy models in order to elucidate mechanisms responsible for leading to the development of spontaneous seizures and for testing new anti-epileptic compounds....

Cite
Ferland RJ. The Repeated Flurothyl Seizure Model in Mice. Bio Protoc. 2017;7(11)doi: 10.21769/BioProtoc.2309.
Ferland, R. J. (2017). The Repeated Flurothyl Seizure Model in Mice. Bio-protocol, 7(11), . https://doi.org/10.21769/BioProtoc.2309
Ferland, Russell J. "The Repeated Flurothyl Seizure Model in Mice." Bio-protocol vol. 7,11 (2017). doi: https://doi.org/10.21769/BioProtoc.2309
Ferland RJ. The Repeated Flurothyl Seizure Model in Mice. Bio Protoc. 2017 Jun 05;7(11). doi: 10.21769/BioProtoc.2309. PMID: 28748202; PMCID: PMC5524139.
Copy Download .nbib Format: NLM
IL-33 Provides Neuroprotection through Suppressing Apoptotic, Autophagic and NF-κB-Mediated Inflammatory Pathways in a Rat Model of Recurrent Neonatal Seizure.

Frontiers in molecular neuroscience

Gao Y, Luo CL, Li LL, Ye GH, Gao C, Wang HC, Huang WW, Wang T, Wang ZF, Ni H, Chen XP, Tao LY.
PMID: 29311813
Front Mol Neurosci. 2017 Dec 19;10:423. doi: 10.3389/fnmol.2017.00423. eCollection 2017.

Interleukin-33 (IL-33) is a novel identified chromatin-associated cytokine of IL-1 family cytokines. It signals through a heterodimer comprised of ST2L and IL-1RAcp, and plays a crucial role in many diseases. However, very little is known about the role and...

Cite
Gao Y, Luo CL, Li LL, et al. IL-33 Provides Neuroprotection through Suppressing Apoptotic, Autophagic and NF-κB-Mediated Inflammatory Pathways in a Rat Model of Recurrent Neonatal Seizure. Front Mol Neurosci. 2017;10:423doi: 10.3389/fnmol.2017.00423.
Gao, Y., Luo, C. L., Li, L. L., Ye, G. H., Gao, C., Wang, H. C., Huang, W. W., Wang, T., Wang, Z. F., Ni, H., Chen, X. P., & Tao, L. Y. (2017). IL-33 Provides Neuroprotection through Suppressing Apoptotic, Autophagic and NF-κB-Mediated Inflammatory Pathways in a Rat Model of Recurrent Neonatal Seizure. Frontiers in molecular neuroscience, 10423. https://doi.org/10.3389/fnmol.2017.00423
Gao, Yuan, et al. "IL-33 Provides Neuroprotection through Suppressing Apoptotic, Autophagic and NF-κB-Mediated Inflammatory Pathways in a Rat Model of Recurrent Neonatal Seizure." Frontiers in molecular neuroscience vol. 10 (2017): 423. doi: https://doi.org/10.3389/fnmol.2017.00423
Gao Y, Luo CL, Li LL, Ye GH, Gao C, Wang HC, Huang WW, Wang T, Wang ZF, Ni H, Chen XP, Tao LY. IL-33 Provides Neuroprotection through Suppressing Apoptotic, Autophagic and NF-κB-Mediated Inflammatory Pathways in a Rat Model of Recurrent Neonatal Seizure. Front Mol Neurosci. 2017 Dec 19;10:423. doi: 10.3389/fnmol.2017.00423. eCollection 2017. PMID: 29311813; PMCID: PMC5742123.
Copy Download .nbib Format: NLM
Rapamycin, but not minocycline, significantly alters ultrasonic vocalization behavior in C57BL/6J pups in a flurothyl seizure model.

Behavioural brain research

Hodges SL, Womble PD, Kwok EM, Darner AM, Senger SS, Binder MS, Faust AM, Condon SM, Nolan SO, Quintero SI, Lugo JN.
PMID: 33910029
Behav Brain Res. 2021 Jul 23;410:113317. doi: 10.1016/j.bbr.2021.113317. Epub 2021 Apr 25.

Epilepsy is one of the most common neurological disorders, with individuals having an increased susceptibility of seizures in the first few years of life, making children at risk of developing a multitude of cognitive and behavioral comorbidities throughout development....

Cite
Hodges SL, Womble PD, Kwok EM, et al. Rapamycin, but not minocycline, significantly alters ultrasonic vocalization behavior in C57BL/6J pups in a flurothyl seizure model. Behav Brain Res. 2021;410:113317doi: 10.1016/j.bbr.2021.113317.
Hodges, S. L., Womble, P. D., Kwok, E. M., Darner, A. M., Senger, S. S., Binder, M. S., Faust, A. M., Condon, S. M., Nolan, S. O., Quintero, S. I., & Lugo, J. N. (2021). Rapamycin, but not minocycline, significantly alters ultrasonic vocalization behavior in C57BL/6J pups in a flurothyl seizure model. Behavioural brain research, 410113317. https://doi.org/10.1016/j.bbr.2021.113317
Hodges, Samantha L, et al. "Rapamycin, but not minocycline, significantly alters ultrasonic vocalization behavior in C57BL/6J pups in a flurothyl seizure model." Behavioural brain research vol. 410 (2021): 113317. doi: https://doi.org/10.1016/j.bbr.2021.113317
Hodges SL, Womble PD, Kwok EM, Darner AM, Senger SS, Binder MS, Faust AM, Condon SM, Nolan SO, Quintero SI, Lugo JN. Rapamycin, but not minocycline, significantly alters ultrasonic vocalization behavior in C57BL/6J pups in a flurothyl seizure model. Behav Brain Res. 2021 Jul 23;410:113317. doi: 10.1016/j.bbr.2021.113317. Epub 2021 Apr 25. PMID: 33910029; PMCID: PMC8628310.
Copy Download .nbib Format: NLM
Effects of methoxyflurane and flurothyl in mice trained to discriminate pentylenetetrazol from saline.

Behavioural pharmacology

Evans EB, Balster RL.
PMID: 11224149
Behav Pharmacol. 1992 Oct;3(5):465-473.

A procedure is described for training mice to discriminate 30mg/kg pentylenetetrazol (PTZ) from saline when lever pressing was maintained under a fixed-ratio 20 schedule of milk presentation. To define the pharmacological profile of the PTZ stimulus in mice, generalization...

Cite
Evans EB, Balster RL. Effects of methoxyflurane and flurothyl in mice trained to discriminate pentylenetetrazol from saline. Behav Pharmacol. 1992;3(5):465-473
Evans, E. B., & Balster, R. L. (1992). Effects of methoxyflurane and flurothyl in mice trained to discriminate pentylenetetrazol from saline. Behavioural pharmacology, 3(5), 465-473.
Evans, E.B., and Balster, R.L.. "Effects of methoxyflurane and flurothyl in mice trained to discriminate pentylenetetrazol from saline." Behavioural pharmacology vol. 3,5 (1992): 465-473.
Evans EB, Balster RL. Effects of methoxyflurane and flurothyl in mice trained to discriminate pentylenetetrazol from saline. Behav Pharmacol. 1992 Oct;3(5):465-473. PMID: 11224149.
Copy Download .nbib Format: NLM
Relationships between Pregnancy and Vitamin B-6 Nutriture on Brain 3-Hydroxykynurenine Concentrations in Mice.

Nutritional neuroscience

van de Kamp JL, Smolen A.
PMID: 27416370
Nutr Neurosci. 2000;3(2):131-8. doi: 10.1080/1028415X.2000.11747309.

The kynurenine (KYN) pathway of tryptophan metabolism produces several neuroactive metabolites, including 3-hydroxykynurenine (3HK). The pathway is subject to regulation by a number of effectors including pregnancy and availability of vitamin B-6. Vitamin B-6 depleted humans and animals excrete...

Cite
van de Kamp JL, Smolen A. Relationships between Pregnancy and Vitamin B-6 Nutriture on Brain 3-Hydroxykynurenine Concentrations in Mice. Nutr Neurosci. 2000;3(2):131-8doi: 10.1080/1028415X.2000.11747309.
van de Kamp, J. L., & Smolen, A. (2000). Relationships between Pregnancy and Vitamin B-6 Nutriture on Brain 3-Hydroxykynurenine Concentrations in Mice. Nutritional neuroscience, 3(2), 131-8. https://doi.org/10.1080/1028415X.2000.11747309
van de Kamp, J L, and Smolen, A. "Relationships between Pregnancy and Vitamin B-6 Nutriture on Brain 3-Hydroxykynurenine Concentrations in Mice." Nutritional neuroscience vol. 3,2 (2000): 131-8. doi: https://doi.org/10.1080/1028415X.2000.11747309
van de Kamp JL, Smolen A. Relationships between Pregnancy and Vitamin B-6 Nutriture on Brain 3-Hydroxykynurenine Concentrations in Mice. Nutr Neurosci. 2000;3(2):131-8. doi: 10.1080/1028415X.2000.11747309. PMID: 27416370.
Copy Download .nbib Format: NLM
Relationship between changes in mitochondrial function and hippocampal neuronal apoptosis after recurrent convulsion during developmental stage.

Experimental and therapeutic medicine

Liu Y, Chen J, Jin M, Li Z, Tian T, Li L, Ni H.
PMID: 29896232
Exp Ther Med. 2018 Jul;16(1):127-132. doi: 10.3892/etm.2018.6147. Epub 2018 May 10.

The aim of the present study was to establish a recurrent convulsion model during the developmental stage using inhalation of flurothyl, and to observe the relationship between the changes in mitochondrial function in hippocampal neurons and hippocampal neuronal apoptosis...

Cite
Liu Y, Chen J, Jin M, et al. Relationship between changes in mitochondrial function and hippocampal neuronal apoptosis after recurrent convulsion during developmental stage. Exp Ther Med. 2018;16(1):127-132doi: 10.3892/etm.2018.6147.
Liu, Y., Chen, J., Jin, M., Li, Z., Tian, T., Li, L., & Ni, H. (2018). Relationship between changes in mitochondrial function and hippocampal neuronal apoptosis after recurrent convulsion during developmental stage. Experimental and therapeutic medicine, 16(1), 127-132. https://doi.org/10.3892/etm.2018.6147
Liu, Yueying, et al. "Relationship between changes in mitochondrial function and hippocampal neuronal apoptosis after recurrent convulsion during developmental stage." Experimental and therapeutic medicine vol. 16,1 (2018): 127-132. doi: https://doi.org/10.3892/etm.2018.6147
Liu Y, Chen J, Jin M, Li Z, Tian T, Li L, Ni H. Relationship between changes in mitochondrial function and hippocampal neuronal apoptosis after recurrent convulsion during developmental stage. Exp Ther Med. 2018 Jul;16(1):127-132. doi: 10.3892/etm.2018.6147. Epub 2018 May 10. PMID: 29896232; PMCID: PMC5995061.
Copy Download .nbib Format: NLM
Seizure Susceptibility Correlates with Brain Injury in Male Mice Treated with Hypothermia after Neonatal Hypoxia-Ischemia.

Developmental neuroscience

McNally MA, Chavez-Valdez R, Felling RJ, Flock DL, Northington FJ, Stafstrom CE.
PMID: 30820019
Dev Neurosci. 2019 Feb 28;1-10. doi: 10.1159/000496468. Epub 2019 Feb 28.

Hypoxic-ischemic encephalopathy is a common neonatal brain injury associated with significant morbidity and mortality despite the administration of therapeutic hypothermia (TH). Neonatal seizures and subsequent chronic epilepsy are frequent in this patient population and current treatments are partially effective....

Cite
McNally MA, Chavez-Valdez R, Felling RJ, et al. Seizure Susceptibility Correlates with Brain Injury in Male Mice Treated with Hypothermia after Neonatal Hypoxia-Ischemia. Dev Neurosci. 2019;1-10doi: 10.1159/000496468.
McNally, M. A., Chavez-Valdez, R., Felling, R. J., Flock, D. L., Northington, F. J., & Stafstrom, C. E. (2019). Seizure Susceptibility Correlates with Brain Injury in Male Mice Treated with Hypothermia after Neonatal Hypoxia-Ischemia. Developmental neuroscience, 1-10. https://doi.org/10.1159/000496468
McNally, Melanie A, et al. "Seizure Susceptibility Correlates with Brain Injury in Male Mice Treated with Hypothermia after Neonatal Hypoxia-Ischemia." Developmental neuroscience vol. (2019): 1-10. doi: https://doi.org/10.1159/000496468
McNally MA, Chavez-Valdez R, Felling RJ, Flock DL, Northington FJ, Stafstrom CE. Seizure Susceptibility Correlates with Brain Injury in Male Mice Treated with Hypothermia after Neonatal Hypoxia-Ischemia. Dev Neurosci. 2019 Feb 28;1-10. doi: 10.1159/000496468. Epub 2019 Feb 28. PMID: 30820019.
Copy Download .nbib Format: NLM
Effects of progesterone on glutamate transporter 2 and gamma-aminobutyric acid transporter 1 expression in the developing rat brain after recurrent seizures.

Neural regeneration research

Liu L, Mao D, Liu L, Huang Y, Bo T.
PMID: 25624835
Neural Regen Res. 2012 Sep 15;7(26):2036-42. doi: 10.3969/j.issn.1673-5374.2012.26.005.

Seizures were induced by flurothyl inhalation. Rats were intramuscularly treated with progesterone after each seizure. Results demonstrated that glutamate transporter 2 and γ-aminobutyric acid transporter 1 expression levels were significantly increased in the cerebral cortex and hippocampus of the...

Cite
Liu L, Mao D, Liu L, et al. Effects of progesterone on glutamate transporter 2 and gamma-aminobutyric acid transporter 1 expression in the developing rat brain after recurrent seizures. Neural Regen Res. 2012;7(26):2036-42doi: 10.3969/j.issn.1673-5374.2012.26.005.
Liu, L., Mao, D., Liu, L., Huang, Y., & Bo, T. (2012). Effects of progesterone on glutamate transporter 2 and gamma-aminobutyric acid transporter 1 expression in the developing rat brain after recurrent seizures. Neural regeneration research, 7(26), 2036-42. https://doi.org/10.3969/j.issn.1673-5374.2012.26.005
Liu, Lingjuan, et al. "Effects of progesterone on glutamate transporter 2 and gamma-aminobutyric acid transporter 1 expression in the developing rat brain after recurrent seizures." Neural regeneration research vol. 7,26 (2012): 2036-42. doi: https://doi.org/10.3969/j.issn.1673-5374.2012.26.005
Liu L, Mao D, Liu L, Huang Y, Bo T. Effects of progesterone on glutamate transporter 2 and gamma-aminobutyric acid transporter 1 expression in the developing rat brain after recurrent seizures. Neural Regen Res. 2012 Sep 15;7(26):2036-42. doi: 10.3969/j.issn.1673-5374.2012.26.005. PMID: 25624835; PMCID: PMC4296423.
Copy Download .nbib Format: NLM
Sex specific correlation between GABAergic disruption in the dorsal hippocampus and flurothyl seizure susceptibility after neonatal hypoxic-ischemic brain injury.

Neurobiology of disease

Lechner CR, McNally MA, St Pierre M, Felling RJ, Northington FJ, Stafstrom CE, Chavez-Valdez R.
PMID: 33309937
Neurobiol Dis. 2021 Jan;148:105222. doi: 10.1016/j.nbd.2020.105222. Epub 2020 Dec 09.

Since neonatal hypoxia-ischemia (HI) disrupts the hippocampal (Hp) GABAergic network in the mouse and Hp injury in this model correlates with flurothyl seizure susceptibility only in male mice, we hypothesized that GABAergic disruption correlates with flurothyl seizure susceptibility in...

Cite
Lechner CR, McNally MA, St Pierre M, et al. Sex specific correlation between GABAergic disruption in the dorsal hippocampus and flurothyl seizure susceptibility after neonatal hypoxic-ischemic brain injury. Neurobiol Dis. 2020;148:105222doi: 10.1016/j.nbd.2020.105222.
Lechner, C. R., McNally, M. A., St Pierre, M., Felling, R. J., Northington, F. J., Stafstrom, C. E., & Chavez-Valdez, R. (2021). Sex specific correlation between GABAergic disruption in the dorsal hippocampus and flurothyl seizure susceptibility after neonatal hypoxic-ischemic brain injury. Neurobiology of disease, 148105222. https://doi.org/10.1016/j.nbd.2020.105222
Lechner, Charles R, et al. "Sex specific correlation between GABAergic disruption in the dorsal hippocampus and flurothyl seizure susceptibility after neonatal hypoxic-ischemic brain injury." Neurobiology of disease vol. 148 (2021): 105222. doi: https://doi.org/10.1016/j.nbd.2020.105222
Lechner CR, McNally MA, St Pierre M, Felling RJ, Northington FJ, Stafstrom CE, Chavez-Valdez R. Sex specific correlation between GABAergic disruption in the dorsal hippocampus and flurothyl seizure susceptibility after neonatal hypoxic-ischemic brain injury. Neurobiol Dis. 2021 Jan;148:105222. doi: 10.1016/j.nbd.2020.105222. Epub 2020 Dec 09. PMID: 33309937; PMCID: PMC7864119.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 12 entries