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Association of HLA antigens and BCR-ABL transcripts in leukemia patients with the Philadelphia chromosome.

Revista brasileira de hematologia e hemoterapia

de Carvalho DL, Barbosa CD, de Carvalho AL, Beck ST.
PMID: 23049441
Rev Bras Hematol Hemoter. 2012;34(4):280-4. doi: 10.5581/1516-8484.20120072.

OBJECTIVE: This study aimed to verify the association between human leukocyte antigens and the bcr-abl fusion protein resulting from t(9;22)(q34;q11) in chronic leukemia myeloid and acute lymphoblastic leukemia patients.METHODS: Forty-seven bcr-abl positive individuals were evaluated. Typing was performed bymicrolymphocytotoxicity...

Cite
de Carvalho DL, Barbosa CD, de Carvalho AL, et al. Association of HLA antigens and BCR-ABL transcripts in leukemia patients with the Philadelphia chromosome. Rev Bras Hematol Hemoter. 2012;34(4):280-4doi: 10.5581/1516-8484.20120072.
de Carvalho, D. L., Barbosa, C. D., de Carvalho, A. L., & Beck, S. T. (2012). Association of HLA antigens and BCR-ABL transcripts in leukemia patients with the Philadelphia chromosome. Revista brasileira de hematologia e hemoterapia, 34(4), 280-4. https://doi.org/10.5581/1516-8484.20120072
de Carvalho, Daiana Landenberger, et al. "Association of HLA antigens and BCR-ABL transcripts in leukemia patients with the Philadelphia chromosome." Revista brasileira de hematologia e hemoterapia vol. 34,4 (2012): 280-4. doi: https://doi.org/10.5581/1516-8484.20120072
de Carvalho DL, Barbosa CD, de Carvalho AL, Beck ST. Association of HLA antigens and BCR-ABL transcripts in leukemia patients with the Philadelphia chromosome. Rev Bras Hematol Hemoter. 2012;34(4):280-4. doi: 10.5581/1516-8484.20120072. PMID: 23049441; PMCID: PMC3460407.
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New alternative splicing BCR/ABL-OOF shows an oncogenic role by lack of inhibition of BCR GTPase activity and an increased of persistence of Rac activation in chronic myeloid leukemia.

Oncoscience

Panuzzo C, Volpe G, Cibrario Rocchietti E, Casnici C, Crotta K, Crivellaro S, Carrà G, Lorenzatti R, Peracino B, Torti D, Morotti A, Camacho-Leal MP, Defilippi P, Marelli O, Saglio G.
PMID: 26682280
Oncoscience. 2015 Nov 11;2(10):880-91. doi: 10.18632/oncoscience.260. eCollection 2015.

In Chronic Myeloid Leukemia 80% of patients present alternative splice variants involving BCR exons 1, 13 or 14 and ABL exon 4, with a consequent impairment in the reading frame of the ABL gene. Therefore BCR/ABL fusion proteins (BCR/ABL-OOF)...

Cite
Panuzzo C, Volpe G, Cibrario Rocchietti E, et al. New alternative splicing BCR/ABL-OOF shows an oncogenic role by lack of inhibition of BCR GTPase activity and an increased of persistence of Rac activation in chronic myeloid leukemia. Oncoscience. 2015;2(10):880-91doi: 10.18632/oncoscience.260.
Panuzzo, C., Volpe, G., Cibrario Rocchietti, E., Casnici, C., Crotta, K., Crivellaro, S., Carrà, G., Lorenzatti, R., Peracino, B., Torti, D., Morotti, A., Camacho-Leal, M. P., Defilippi, P., Marelli, O., & Saglio, G. (2015). New alternative splicing BCR/ABL-OOF shows an oncogenic role by lack of inhibition of BCR GTPase activity and an increased of persistence of Rac activation in chronic myeloid leukemia. Oncoscience, 2(10), 880-91. https://doi.org/10.18632/oncoscience.260
Panuzzo, Cristina, et al. "New alternative splicing BCR/ABL-OOF shows an oncogenic role by lack of inhibition of BCR GTPase activity and an increased of persistence of Rac activation in chronic myeloid leukemia." Oncoscience vol. 2,10 (2015): 880-91. doi: https://doi.org/10.18632/oncoscience.260
Panuzzo C, Volpe G, Cibrario Rocchietti E, Casnici C, Crotta K, Crivellaro S, Carrà G, Lorenzatti R, Peracino B, Torti D, Morotti A, Camacho-Leal MP, Defilippi P, Marelli O, Saglio G. New alternative splicing BCR/ABL-OOF shows an oncogenic role by lack of inhibition of BCR GTPase activity and an increased of persistence of Rac activation in chronic myeloid leukemia. Oncoscience. 2015 Nov 11;2(10):880-91. doi: 10.18632/oncoscience.260. eCollection 2015. PMID: 26682280; PMCID: PMC4671955.
Copy Download .nbib Format: NLM
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