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Showing 1 to 12 of 13 entries
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Genetics in psychiatry: common variant association studies.

Molecular autism

Buxbaum JD, Baron-Cohen S, Devlin B.
PMID: 20678248
Mol Autism. 2010 Mar 25;1(1):6. doi: 10.1186/2040-2392-1-6.

Many psychiatric conditions and traits are associated with significant heritability. Genetic risk for psychiatric conditions encompass rare variants, identified due to major effect, as well as common variants, the latter analyzed by association analyses. We review guidelines for common...

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Buxbaum JD, Baron-Cohen S, Devlin B. Genetics in psychiatry: common variant association studies. Mol Autism. 2010;1(1):6doi: 10.1186/2040-2392-1-6.
Buxbaum, J. D., Baron-Cohen, S., & Devlin, B. (2010). Genetics in psychiatry: common variant association studies. Molecular autism, 1(1), 6. https://doi.org/10.1186/2040-2392-1-6
Buxbaum, Joseph D, et al. "Genetics in psychiatry: common variant association studies." Molecular autism vol. 1,1 (2010): 6. doi: https://doi.org/10.1186/2040-2392-1-6
Buxbaum JD, Baron-Cohen S, Devlin B. Genetics in psychiatry: common variant association studies. Mol Autism. 2010 Mar 25;1(1):6. doi: 10.1186/2040-2392-1-6. PMID: 20678248; PMCID: PMC2907568.
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DSM-5: the debate continues.

Molecular autism

Buxbaum JD, Baron-Cohen S.
PMID: 23676181
Mol Autism. 2013 May 15;4(1):11. doi: 10.1186/2040-2392-4-11.

We are fortunate to have invited commentaries from the laboratories of Dr Cathy Lord and Dr Fred Volkmar offering their perspectives on the new Diagnostic and Statistical Manual of Mental Disorders (DSM)-5 criteria for the autism spectrum. Both commentaries...

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Buxbaum JD, Baron-Cohen S. DSM-5: the debate continues. Mol Autism. 2013;4(1):11doi: 10.1186/2040-2392-4-11.
Buxbaum, J. D., & Baron-Cohen, S. (2013). DSM-5: the debate continues. Molecular autism, 4(1), 11. https://doi.org/10.1186/2040-2392-4-11
Buxbaum, Joseph D, and Baron-Cohen, Simon. "DSM-5: the debate continues." Molecular autism vol. 4,1 (2013): 11. doi: https://doi.org/10.1186/2040-2392-4-11
Buxbaum JD, Baron-Cohen S. DSM-5: the debate continues. Mol Autism. 2013 May 15;4(1):11. doi: 10.1186/2040-2392-4-11. PMID: 23676181; PMCID: PMC3672020.
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Prevalence of autism in mainland China, Hong Kong and Taiwan: a systematic review and meta-analysis.

Molecular autism

Sun X, Allison C, Matthews FE, Sharp SJ, Auyeung B, Baron-Cohen S, Brayne C.
PMID: 23570419
Mol Autism. 2013 Apr 09;4(1):7. doi: 10.1186/2040-2392-4-7.

BACKGROUND: The prevalence of autism spectrum conditions (ASC) is 1% in developed countries, but little data are available from mainland China, Hong Kong and Taiwan. This study synthesizes evidence relating to the prevalence of ASC in these areas and...

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Sun X, Allison C, Matthews FE, et al. Prevalence of autism in mainland China, Hong Kong and Taiwan: a systematic review and meta-analysis. Mol Autism. 2013;4(1):7doi: 10.1186/2040-2392-4-7.
Sun, X., Allison, C., Matthews, F. E., Sharp, S. J., Auyeung, B., Baron-Cohen, S., & Brayne, C. (2013). Prevalence of autism in mainland China, Hong Kong and Taiwan: a systematic review and meta-analysis. Molecular autism, 4(1), 7. https://doi.org/10.1186/2040-2392-4-7
Sun, Xiang, et al. "Prevalence of autism in mainland China, Hong Kong and Taiwan: a systematic review and meta-analysis." Molecular autism vol. 4,1 (2013): 7. doi: https://doi.org/10.1186/2040-2392-4-7
Sun X, Allison C, Matthews FE, Sharp SJ, Auyeung B, Baron-Cohen S, Brayne C. Prevalence of autism in mainland China, Hong Kong and Taiwan: a systematic review and meta-analysis. Mol Autism. 2013 Apr 09;4(1):7. doi: 10.1186/2040-2392-4-7. PMID: 23570419; PMCID: PMC3643868.
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Do girls with anorexia nervosa have elevated autistic traits?.

Molecular autism

Baron-Cohen S, Jaffa T, Davies S, Auyeung B, Allison C, Wheelwright S.
PMID: 23915495
Mol Autism. 2013 Jul 31;4(1):24. doi: 10.1186/2040-2392-4-24.

BACKGROUND: Patients with anorexia may have elevated autistic traits. In this study, we tested test whether patients with anorexia nervosa (anorexia) have an elevated score on a dimensional measure of autistic traits, the Autism Spectrum Quotient (AQ), as well...

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Baron-Cohen S, Jaffa T, Davies S, et al. Do girls with anorexia nervosa have elevated autistic traits?. Mol Autism. 2013;4(1):24doi: 10.1186/2040-2392-4-24.
Baron-Cohen, S., Jaffa, T., Davies, S., Auyeung, B., Allison, C., & Wheelwright, S. (2013). Do girls with anorexia nervosa have elevated autistic traits?. Molecular autism, 4(1), 24. https://doi.org/10.1186/2040-2392-4-24
Baron-Cohen, Simon, et al. "Do girls with anorexia nervosa have elevated autistic traits?." Molecular autism vol. 4,1 (2013): 24. doi: https://doi.org/10.1186/2040-2392-4-24
Baron-Cohen S, Jaffa T, Davies S, Auyeung B, Allison C, Wheelwright S. Do girls with anorexia nervosa have elevated autistic traits?. Mol Autism. 2013 Jul 31;4(1):24. doi: 10.1186/2040-2392-4-24. PMID: 23915495; PMCID: PMC3735388.
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Spatial localisation in autism: evidence for differences in early cortical visual processing.

Molecular autism

Latham K, Chung ST, Allen PM, Tavassoli T, Baron-Cohen S.
PMID: 23422139
Mol Autism. 2013 Feb 19;4(1):4. doi: 10.1186/2040-2392-4-4.

BACKGROUND: Vision in people with autism spectrum conditions (ASC) is reported to be different from people without ASC, but the neural level at which the differences begin to occur is not yet known. Here we examine two variants of...

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Latham K, Chung ST, Allen PM, et al. Spatial localisation in autism: evidence for differences in early cortical visual processing. Mol Autism. 2013;4(1):4doi: 10.1186/2040-2392-4-4.
Latham, K., Chung, S. T., Allen, P. M., Tavassoli, T., & Baron-Cohen, S. (2013). Spatial localisation in autism: evidence for differences in early cortical visual processing. Molecular autism, 4(1), 4. https://doi.org/10.1186/2040-2392-4-4
Latham, Keziah, et al. "Spatial localisation in autism: evidence for differences in early cortical visual processing." Molecular autism vol. 4,1 (2013): 4. doi: https://doi.org/10.1186/2040-2392-4-4
Latham K, Chung ST, Allen PM, Tavassoli T, Baron-Cohen S. Spatial localisation in autism: evidence for differences in early cortical visual processing. Mol Autism. 2013 Feb 19;4(1):4. doi: 10.1186/2040-2392-4-4. PMID: 23422139; PMCID: PMC3599959.
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Failure to deactivate the default mode network indicates a possible endophenotype of autism.

Molecular autism

Spencer MD, Chura LR, Holt RJ, Suckling J, Calder AJ, Bullmore ET, Baron-Cohen S.
PMID: 23206727
Mol Autism. 2012 Dec 03;3(1):15. doi: 10.1186/2040-2392-3-15.

BACKGROUND: Reduced activity during cognitively demanding tasks has been reported in the default mode network in typically developing controls and individuals with autism. However, no study has investigated the default mode network (DMN) in first-degree relatives of those with...

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Spencer MD, Chura LR, Holt RJ, et al. Failure to deactivate the default mode network indicates a possible endophenotype of autism. Mol Autism. 2012;3(1):15doi: 10.1186/2040-2392-3-15.
Spencer, M. D., Chura, L. R., Holt, R. J., Suckling, J., Calder, A. J., Bullmore, E. T., & Baron-Cohen, S. (2012). Failure to deactivate the default mode network indicates a possible endophenotype of autism. Molecular autism, 3(1), 15. https://doi.org/10.1186/2040-2392-3-15
Spencer, Michael D, et al. "Failure to deactivate the default mode network indicates a possible endophenotype of autism." Molecular autism vol. 3,1 (2012): 15. doi: https://doi.org/10.1186/2040-2392-3-15
Spencer MD, Chura LR, Holt RJ, Suckling J, Calder AJ, Bullmore ET, Baron-Cohen S. Failure to deactivate the default mode network indicates a possible endophenotype of autism. Mol Autism. 2012 Dec 03;3(1):15. doi: 10.1186/2040-2392-3-15. PMID: 23206727; PMCID: PMC3539860.
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Prenatal versus postnatal sex steroid hormone effects on autistic traits in children at 18 to 24 months of age.

Molecular autism

Auyeung B, Ahluwalia J, Thomson L, Taylor K, Hackett G, O'Donnell KJ, Baron-Cohen S.
PMID: 23231861
Mol Autism. 2012 Dec 11;3(1):17. doi: 10.1186/2040-2392-3-17.

BACKGROUND: Studies of prenatal exposure to sex steroid hormones predict autistic traits in children at 18 to 24 and at 96 months of age. However, it is not known whether postnatal exposure to these hormones has a similar effect....

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Auyeung B, Ahluwalia J, Thomson L, et al. Prenatal versus postnatal sex steroid hormone effects on autistic traits in children at 18 to 24 months of age. Mol Autism. 2012;3(1):17doi: 10.1186/2040-2392-3-17.
Auyeung, B., Ahluwalia, J., Thomson, L., Taylor, K., Hackett, G., O'Donnell, K. J., & Baron-Cohen, S. (2012). Prenatal versus postnatal sex steroid hormone effects on autistic traits in children at 18 to 24 months of age. Molecular autism, 3(1), 17. https://doi.org/10.1186/2040-2392-3-17
Auyeung, Bonnie, et al. "Prenatal versus postnatal sex steroid hormone effects on autistic traits in children at 18 to 24 months of age." Molecular autism vol. 3,1 (2012): 17. doi: https://doi.org/10.1186/2040-2392-3-17
Auyeung B, Ahluwalia J, Thomson L, Taylor K, Hackett G, O'Donnell KJ, Baron-Cohen S. Prenatal versus postnatal sex steroid hormone effects on autistic traits in children at 18 to 24 months of age. Mol Autism. 2012 Dec 11;3(1):17. doi: 10.1186/2040-2392-3-17. PMID: 23231861; PMCID: PMC3554559.
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Variation in the autism candidate gene GABRB3 modulates tactile sensitivity in typically developing children.

Molecular autism

Tavassoli T, Auyeung B, Murphy LC, Baron-Cohen S, Chakrabarti B.
PMID: 22769427
Mol Autism. 2012 Jul 06;3(1):6. doi: 10.1186/2040-2392-3-6.

BACKGROUND: Autism spectrum conditions have a strong genetic component. Atypical sensory sensitivities are one of the core but neglected features of autism spectrum conditions. GABRB3 is a well-characterised candidate gene for autism spectrum conditions. In mice, heterozygous Gabrb3 deletion...

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Tavassoli T, Auyeung B, Murphy LC, et al. Variation in the autism candidate gene GABRB3 modulates tactile sensitivity in typically developing children. Mol Autism. 2012;3(1):6doi: 10.1186/2040-2392-3-6.
Tavassoli, T., Auyeung, B., Murphy, L. C., Baron-Cohen, S., & Chakrabarti, B. (2012). Variation in the autism candidate gene GABRB3 modulates tactile sensitivity in typically developing children. Molecular autism, 3(1), 6. https://doi.org/10.1186/2040-2392-3-6
Tavassoli, Teresa, et al. "Variation in the autism candidate gene GABRB3 modulates tactile sensitivity in typically developing children." Molecular autism vol. 3,1 (2012): 6. doi: https://doi.org/10.1186/2040-2392-3-6
Tavassoli T, Auyeung B, Murphy LC, Baron-Cohen S, Chakrabarti B. Variation in the autism candidate gene GABRB3 modulates tactile sensitivity in typically developing children. Mol Autism. 2012 Jul 06;3(1):6. doi: 10.1186/2040-2392-3-6. PMID: 22769427; PMCID: PMC3434022.
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Clinical heterogeneity among people with high functioning autism spectrum conditions: evidence favouring a continuous severity gradient.

Behavioral and brain functions : BBF

Ring H, Woodbury-Smith M, Watson P, Wheelwright S, Baron-Cohen S.
PMID: 18289376
Behav Brain Funct. 2008 Feb 20;4:11. doi: 10.1186/1744-9081-4-11.

BACKGROUND: Autism Spectrum Conditions (ASCs) are characterized by a high degree of clinical heterogeneity, but the extent to which this variation represents a severity gradient versus discrete phenotypes is unclear. This issue has complicated genetic studies seeking to investigate...

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Ring H, Woodbury-Smith M, Watson P, et al. Clinical heterogeneity among people with high functioning autism spectrum conditions: evidence favouring a continuous severity gradient. Behav Brain Funct. 2008;4:11doi: 10.1186/1744-9081-4-11.
Ring, H., Woodbury-Smith, M., Watson, P., Wheelwright, S., & Baron-Cohen, S. (2008). Clinical heterogeneity among people with high functioning autism spectrum conditions: evidence favouring a continuous severity gradient. Behavioral and brain functions : BBF, 411. https://doi.org/10.1186/1744-9081-4-11
Ring, Howard, et al. "Clinical heterogeneity among people with high functioning autism spectrum conditions: evidence favouring a continuous severity gradient." Behavioral and brain functions : BBF vol. 4 (2008): 11. doi: https://doi.org/10.1186/1744-9081-4-11
Ring H, Woodbury-Smith M, Watson P, Wheelwright S, Baron-Cohen S. Clinical heterogeneity among people with high functioning autism spectrum conditions: evidence favouring a continuous severity gradient. Behav Brain Funct. 2008 Feb 20;4:11. doi: 10.1186/1744-9081-4-11. PMID: 18289376; PMCID: PMC2265729.
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Default Mode Hypoconnectivity Underlies a Sex-Related Autism Spectrum.

Biological psychiatry. Cognitive neuroscience and neuroimaging

Ypma RJ, Moseley RL, Holt RJ, Rughooputh N, Floris DL, Chura LR, Spencer MD, Baron-Cohen S, Suckling J, Bullmore ET, Rubinov M.
PMID: 27430030
Biol Psychiatry Cogn Neurosci Neuroimaging. 2016 Jul;1(4):364-371. doi: 10.1016/j.bpsc.2016.04.006.

BACKGROUND: Females and males differ significantly in the prevalence and presentation of autism spectrum conditions. One theory of this effect postulates that autistic traits lie on a sex-related continuum in the general population, and autism represents the extreme male...

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Ypma RJ, Moseley RL, Holt RJ, et al. Default Mode Hypoconnectivity Underlies a Sex-Related Autism Spectrum. Biol Psychiatry Cogn Neurosci Neuroimaging. 2016;1(4):364-371doi: 10.1016/j.bpsc.2016.04.006.
Ypma, R. J., Moseley, R. L., Holt, R. J., Rughooputh, N., Floris, D. L., Chura, L. R., Spencer, M. D., Baron-Cohen, S., Suckling, J., Bullmore, E. T., & Rubinov, M. (2016). Default Mode Hypoconnectivity Underlies a Sex-Related Autism Spectrum. Biological psychiatry. Cognitive neuroscience and neuroimaging, 1(4), 364-371. https://doi.org/10.1016/j.bpsc.2016.04.006
Ypma, Rolf J F, et al. "Default Mode Hypoconnectivity Underlies a Sex-Related Autism Spectrum." Biological psychiatry. Cognitive neuroscience and neuroimaging vol. 1,4 (2016): 364-371. doi: https://doi.org/10.1016/j.bpsc.2016.04.006
Ypma RJ, Moseley RL, Holt RJ, Rughooputh N, Floris DL, Chura LR, Spencer MD, Baron-Cohen S, Suckling J, Bullmore ET, Rubinov M. Default Mode Hypoconnectivity Underlies a Sex-Related Autism Spectrum. Biol Psychiatry Cogn Neurosci Neuroimaging. 2016 Jul;1(4):364-371. doi: 10.1016/j.bpsc.2016.04.006. PMID: 27430030; PMCID: PMC4936761.
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Defining the broader, medium and narrow autism phenotype among parents using the Autism Spectrum Quotient (AQ).

Molecular autism

Wheelwright S, Auyeung B, Allison C, Baron-Cohen S.
PMID: 20678260
Mol Autism. 2010 Jun 17;1(1):10. doi: 10.1186/2040-2392-1-10.

BACKGROUND: The Autism Spectrum Quotient (AQ) is a self-report questionnaire for quantifying autistic traits. This study tests whether the AQ can differentiate between parents of children with an autism spectrum condition (ASC) and control parents. In this paper, the...

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Wheelwright S, Auyeung B, Allison C, et al. Defining the broader, medium and narrow autism phenotype among parents using the Autism Spectrum Quotient (AQ). Mol Autism. 2010;1(1):10doi: 10.1186/2040-2392-1-10.
Wheelwright, S., Auyeung, B., Allison, C., & Baron-Cohen, S. (2010). Defining the broader, medium and narrow autism phenotype among parents using the Autism Spectrum Quotient (AQ). Molecular autism, 1(1), 10. https://doi.org/10.1186/2040-2392-1-10
Wheelwright, Sally, et al. "Defining the broader, medium and narrow autism phenotype among parents using the Autism Spectrum Quotient (AQ)." Molecular autism vol. 1,1 (2010): 10. doi: https://doi.org/10.1186/2040-2392-1-10
Wheelwright S, Auyeung B, Allison C, Baron-Cohen S. Defining the broader, medium and narrow autism phenotype among parents using the Autism Spectrum Quotient (AQ). Mol Autism. 2010 Jun 17;1(1):10. doi: 10.1186/2040-2392-1-10. PMID: 20678260; PMCID: PMC2913943.
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Foetal testosterone and autistic traits in 18 to 24-month-old children.

Molecular autism

Auyeung B, Taylor K, Hackett G, Baron-Cohen S.
PMID: 20678186
Mol Autism. 2010 Jul 12;1(1):11. doi: 10.1186/2040-2392-1-11.

BACKGROUND: Autism spectrum conditions have been characterised as an extreme presentation of certain male-typical psychological traits. In addition, several studies have established a link between prenatal exposure to testosterone and cognitive sex differences in later life, and one study...

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Auyeung B, Taylor K, Hackett G, et al. Foetal testosterone and autistic traits in 18 to 24-month-old children. Mol Autism. 2010;1(1):11doi: 10.1186/2040-2392-1-11.
Auyeung, B., Taylor, K., Hackett, G., & Baron-Cohen, S. (2010). Foetal testosterone and autistic traits in 18 to 24-month-old children. Molecular autism, 1(1), 11. https://doi.org/10.1186/2040-2392-1-11
Auyeung, Bonnie, et al. "Foetal testosterone and autistic traits in 18 to 24-month-old children." Molecular autism vol. 1,1 (2010): 11. doi: https://doi.org/10.1186/2040-2392-1-11
Auyeung B, Taylor K, Hackett G, Baron-Cohen S. Foetal testosterone and autistic traits in 18 to 24-month-old children. Mol Autism. 2010 Jul 12;1(1):11. doi: 10.1186/2040-2392-1-11. PMID: 20678186; PMCID: PMC2916006.
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