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Trends in mortality in schizophrenia and their implications.

Acta psychiatrica Scandinavica

Onwordi E, Howes O.
PMID: 30374964
Acta Psychiatr Scand. 2018 Dec;138(6):489-491. doi: 10.1111/acps.12977.

No abstract available.

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Onwordi E, Howes O. Trends in mortality in schizophrenia and their implications. Acta Psychiatr Scand. 2018;138(6):489-491doi: 10.1111/acps.12977.
Onwordi, E., & Howes, O. (2018). Trends in mortality in schizophrenia and their implications. Acta psychiatrica Scandinavica, 138(6), 489-491. https://doi.org/10.1111/acps.12977
Onwordi, Ellis, and Howes, Oliver. "Trends in mortality in schizophrenia and their implications." Acta psychiatrica Scandinavica vol. 138,6 (2018): 489-491. doi: https://doi.org/10.1111/acps.12977
Onwordi E, Howes O. Trends in mortality in schizophrenia and their implications. Acta Psychiatr Scand. 2018 Dec;138(6):489-491. doi: 10.1111/acps.12977. PMID: 30374964.
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Basic Self-Disturbances Related to Reduced Anterior Cingulate Volume in Subjects at Ultra-High Risk for Psychosis.

Frontiers in psychiatry

Bonoldi I, Allen P, Madeira L, Tognin S, Bossong MG, Azis M, Samson C, Quinn B, Calem M, Valmaggia L, Modinos G, Stone J, Perez J, Howes O, Politi P, Kempton MJ, Fusar-Poli P, McGuire P.
PMID: 31133887
Front Psychiatry. 2019 May 10;10:254. doi: 10.3389/fpsyt.2019.00254. eCollection 2019.

No abstract available.

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Bonoldi I, Allen P, Madeira L, et al. Basic Self-Disturbances Related to Reduced Anterior Cingulate Volume in Subjects at Ultra-High Risk for Psychosis. Front Psychiatry. 2019;10:254doi: 10.3389/fpsyt.2019.00254.
Bonoldi, I., Allen, P., Madeira, L., Tognin, S., Bossong, M. G., Azis, M., Samson, C., Quinn, B., Calem, M., Valmaggia, L., Modinos, G., Stone, J., Perez, J., Howes, O., Politi, P., Kempton, M. J., Fusar-Poli, P., & McGuire, P. (2019). Basic Self-Disturbances Related to Reduced Anterior Cingulate Volume in Subjects at Ultra-High Risk for Psychosis. Frontiers in psychiatry, 10254. https://doi.org/10.3389/fpsyt.2019.00254
Bonoldi, Ilaria, et al. "Basic Self-Disturbances Related to Reduced Anterior Cingulate Volume in Subjects at Ultra-High Risk for Psychosis." Frontiers in psychiatry vol. 10 (2019): 254. doi: https://doi.org/10.3389/fpsyt.2019.00254
Bonoldi I, Allen P, Madeira L, Tognin S, Bossong MG, Azis M, Samson C, Quinn B, Calem M, Valmaggia L, Modinos G, Stone J, Perez J, Howes O, Politi P, Kempton MJ, Fusar-Poli P, McGuire P. Basic Self-Disturbances Related to Reduced Anterior Cingulate Volume in Subjects at Ultra-High Risk for Psychosis. Front Psychiatry. 2019 May 10;10:254. doi: 10.3389/fpsyt.2019.00254. eCollection 2019. PMID: 31133887; PMCID: PMC6526781.
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The neural and molecular basis of working memory function in psychosis: a multimodal PET-fMRI study.

Molecular psychiatry

Borgan F, O'Daly O, Veronese M, Reis Marques T, Laurikainen H, Hietala J, Howes O.
PMID: 31801965
Mol Psychiatry. 2021 Aug;26(8):4464-4474. doi: 10.1038/s41380-019-0619-6. Epub 2019 Dec 04.

Working memory (WM) deficits predict clinical and functional outcomes in schizophrenia but are poorly understood and unaddressed by existing treatments. WM encoding and WM retrieval have not been investigated in schizophrenia without the confounds of illness chronicity or the...

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Borgan F, O'Daly O, Veronese M, et al. The neural and molecular basis of working memory function in psychosis: a multimodal PET-fMRI study. Mol Psychiatry. 2019;26(8):4464-4474doi: 10.1038/s41380-019-0619-6.
Borgan, F., O'Daly, O., Veronese, M., Reis Marques, T., Laurikainen, H., Hietala, J., & Howes, O. (2021). The neural and molecular basis of working memory function in psychosis: a multimodal PET-fMRI study. Molecular psychiatry, 26(8), 4464-4474. https://doi.org/10.1038/s41380-019-0619-6
Borgan, Faith, et al. "The neural and molecular basis of working memory function in psychosis: a multimodal PET-fMRI study." Molecular psychiatry vol. 26,8 (2021): 4464-4474. doi: https://doi.org/10.1038/s41380-019-0619-6
Borgan F, O'Daly O, Veronese M, Reis Marques T, Laurikainen H, Hietala J, Howes O. The neural and molecular basis of working memory function in psychosis: a multimodal PET-fMRI study. Mol Psychiatry. 2021 Aug;26(8):4464-4474. doi: 10.1038/s41380-019-0619-6. Epub 2019 Dec 04. PMID: 31801965; PMCID: PMC8550949.
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Editorial: Reporting guidelines for psychopharmacology.

Psychopharmacology

Steckler T, Curran HV, de Wit H, Howes O, Hoyer D, Lucki I, Miczek KA, Morrow AL, Price LH, Robbins TW.
PMID: 26915651
Psychopharmacology (Berl). 2016 Apr;233(7):1131-4. doi: 10.1007/s00213-016-4252-7.

No abstract available.

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Steckler T, Curran HV, de Wit H, et al. Editorial: Reporting guidelines for psychopharmacology. Psychopharmacology (Berl). 2016;233(7):1131-4doi: 10.1007/s00213-016-4252-7.
Steckler, T., Curran, H. V., de Wit, H., Howes, O., Hoyer, D., Lucki, I., Miczek, K. A., Morrow, A. L., Price, L. H., & Robbins, T. W. (2016). Editorial: Reporting guidelines for psychopharmacology. Psychopharmacology, 233(7), 1131-4. https://doi.org/10.1007/s00213-016-4252-7
Steckler, Thomas, et al. "Editorial: Reporting guidelines for psychopharmacology." Psychopharmacology vol. 233,7 (2016): 1131-4. doi: https://doi.org/10.1007/s00213-016-4252-7
Steckler T, Curran HV, de Wit H, Howes O, Hoyer D, Lucki I, Miczek KA, Morrow AL, Price LH, Robbins TW. Editorial: Reporting guidelines for psychopharmacology. Psychopharmacology (Berl). 2016 Apr;233(7):1131-4. doi: 10.1007/s00213-016-4252-7. PMID: 26915651.
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Correction: Regulation of dopaminergic function: An [.

Translational psychiatry

Jauhar S, Veronese M, Rogdaki M, Bloomfield M, Natesan S, Turkheimer F, Kapur S, Howes OD.
PMID: 31186401
Transl Psychiatry. 2019 Jun 11;9(1):168. doi: 10.1038/s41398-019-0501-y.

An amendment to this paper has been published and can be accessed via a link at the top of the paper.

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Jauhar S, Veronese M, Rogdaki M, et al. Correction: Regulation of dopaminergic function: An [. Transl Psychiatry. 2019;9(1):168doi: 10.1038/s41398-019-0501-y.
Jauhar, S., Veronese, M., Rogdaki, M., Bloomfield, M., Natesan, S., Turkheimer, F., Kapur, S., & Howes, O. D. (2019). Correction: Regulation of dopaminergic function: An [. Translational psychiatry, 9(1), 168. https://doi.org/10.1038/s41398-019-0501-y
Jauhar, S, et al. "Correction: Regulation of dopaminergic function: An [." Translational psychiatry vol. 9,1 (2019): 168. doi: https://doi.org/10.1038/s41398-019-0501-y
Jauhar S, Veronese M, Rogdaki M, Bloomfield M, Natesan S, Turkheimer F, Kapur S, Howes OD. Correction: Regulation of dopaminergic function: An [. Transl Psychiatry. 2019 Jun 11;9(1):168. doi: 10.1038/s41398-019-0501-y. PMID: 31186401; PMCID: PMC6559949.
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Dopamine and glutamate in schizophrenia: biology, symptoms and treatment.

World psychiatry : official journal of the World Psychiatric Association (WPA)

McCutcheon RA, Krystal JH, Howes OD.
PMID: 31922684
World Psychiatry. 2020 Feb;19(1):15-33. doi: 10.1002/wps.20693.

Glutamate and dopamine systems play distinct roles in terms of neuronal signalling, yet both have been proposed to contribute significantly to the pathophysiology of schizophrenia. In this paper we assess research that has implicated both systems in the aetiology...

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McCutcheon RA, Krystal JH, Howes OD. Dopamine and glutamate in schizophrenia: biology, symptoms and treatment. World Psychiatry. 2020;19(1):15-33doi: 10.1002/wps.20693.
McCutcheon, R. A., Krystal, J. H., & Howes, O. D. (2020). Dopamine and glutamate in schizophrenia: biology, symptoms and treatment. World psychiatry : official journal of the World Psychiatric Association (WPA), 19(1), 15-33. https://doi.org/10.1002/wps.20693
McCutcheon, Robert A, et al. "Dopamine and glutamate in schizophrenia: biology, symptoms and treatment." World psychiatry : official journal of the World Psychiatric Association (WPA) vol. 19,1 (2020): 15-33. doi: https://doi.org/10.1002/wps.20693
McCutcheon RA, Krystal JH, Howes OD. Dopamine and glutamate in schizophrenia: biology, symptoms and treatment. World Psychiatry. 2020 Feb;19(1):15-33. doi: 10.1002/wps.20693. PMID: 31922684; PMCID: PMC6953551.
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The neurobiology of treatment-resistant schizophrenia: paths to antipsychotic resistance and a roadmap for future research.

NPJ schizophrenia

Potkin SG, Kane JM, Correll CU, Lindenmayer JP, Agid O, Marder SR, Olfson M, Howes OD.
PMID: 31911624
NPJ Schizophr. 2020 Jan 07;6(1):1. doi: 10.1038/s41537-019-0090-z.

Treatment-resistant schizophrenia (TRS), the persistence of positive symptoms despite ≥2 trials of adequate dose and duration of antipsychotic medication with documented adherence, is a serious clinical problem with heterogeneous presentations. TRS can vary in its onset (at the first...

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Potkin SG, Kane JM, Correll CU, et al. The neurobiology of treatment-resistant schizophrenia: paths to antipsychotic resistance and a roadmap for future research. NPJ Schizophr. 2020;6(1):1doi: 10.1038/s41537-019-0090-z.
Potkin, S. G., Kane, J. M., Correll, C. U., Lindenmayer, J. P., Agid, O., Marder, S. R., Olfson, M., & Howes, O. D. (2020). The neurobiology of treatment-resistant schizophrenia: paths to antipsychotic resistance and a roadmap for future research. NPJ schizophrenia, 6(1), 1. https://doi.org/10.1038/s41537-019-0090-z
Potkin, Steven G, et al. "The neurobiology of treatment-resistant schizophrenia: paths to antipsychotic resistance and a roadmap for future research." NPJ schizophrenia vol. 6,1 (2020): 1. doi: https://doi.org/10.1038/s41537-019-0090-z
Potkin SG, Kane JM, Correll CU, Lindenmayer JP, Agid O, Marder SR, Olfson M, Howes OD. The neurobiology of treatment-resistant schizophrenia: paths to antipsychotic resistance and a roadmap for future research. NPJ Schizophr. 2020 Jan 07;6(1):1. doi: 10.1038/s41537-019-0090-z. PMID: 31911624; PMCID: PMC6946650.
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Variability in Action Selection Relates to Striatal Dopamine 2/3 Receptor Availability in Humans: A PET Neuroimaging Study Using Reinforcement Learning and Active Inference Models.

Cerebral cortex (New York, N.Y. : 1991)

Adams RA, Moutoussis M, Nour MM, Dahoun T, Lewis D, Illingworth B, Veronese M, Mathys C, de Boer L, Guitart-Masip M, Friston KJ, Howes OD, Roiser JP.
PMID: 32083297
Cereb Cortex. 2020 May 18;30(6):3573-3589. doi: 10.1093/cercor/bhz327.

Choosing actions that result in advantageous outcomes is a fundamental function of nervous systems. All computational decision-making models contain a mechanism that controls the variability of (or confidence in) action selection, but its neural implementation is unclear-especially in humans....

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Adams RA, Moutoussis M, Nour MM, et al. Variability in Action Selection Relates to Striatal Dopamine 2/3 Receptor Availability in Humans: A PET Neuroimaging Study Using Reinforcement Learning and Active Inference Models. Cereb Cortex. 2020;30(6):3573-3589doi: 10.1093/cercor/bhz327.
Adams, R. A., Moutoussis, M., Nour, M. M., Dahoun, T., Lewis, D., Illingworth, B., Veronese, M., Mathys, C., de Boer, L., Guitart-Masip, M., Friston, K. J., Howes, O. D., & Roiser, J. P. (2020). Variability in Action Selection Relates to Striatal Dopamine 2/3 Receptor Availability in Humans: A PET Neuroimaging Study Using Reinforcement Learning and Active Inference Models. Cerebral cortex (New York, N.Y. : 1991), 30(6), 3573-3589. https://doi.org/10.1093/cercor/bhz327
Adams, Rick A, et al. "Variability in Action Selection Relates to Striatal Dopamine 2/3 Receptor Availability in Humans: A PET Neuroimaging Study Using Reinforcement Learning and Active Inference Models." Cerebral cortex (New York, N.Y. : 1991) vol. 30,6 (2020): 3573-3589. doi: https://doi.org/10.1093/cercor/bhz327
Adams RA, Moutoussis M, Nour MM, Dahoun T, Lewis D, Illingworth B, Veronese M, Mathys C, de Boer L, Guitart-Masip M, Friston KJ, Howes OD, Roiser JP. Variability in Action Selection Relates to Striatal Dopamine 2/3 Receptor Availability in Humans: A PET Neuroimaging Study Using Reinforcement Learning and Active Inference Models. Cereb Cortex. 2020 May 18;30(6):3573-3589. doi: 10.1093/cercor/bhz327. PMID: 32083297; PMCID: PMC7233027.
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The effects of voice content on stress reactivity: A simulation paradigm of auditory verbal hallucinations.

Schizophrenia research

Baumeister D, Peters E, Pruessner J, Howes O, Chadwick P.
PMID: 31377050
Schizophr Res. 2019 Aug 01; doi: 10.1016/j.schres.2019.07.019. Epub 2019 Aug 01.

OBJECTIVES: Psychosis is associated with increased subjective and altered endocrine and autonomic nervous system stress-reactivity. Psychosis patients often experience auditory verbal hallucinations, with negative voice content being particularly associated with distress. The present study developed a voice-simulation paradigm and...

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Baumeister D, Peters E, Pruessner J, et al. The effects of voice content on stress reactivity: A simulation paradigm of auditory verbal hallucinations. Schizophr Res. 2019;doi: 10.1016/j.schres.2019.07.019.
Baumeister, D., Peters, E., Pruessner, J., Howes, O., & Chadwick, P. (2019). The effects of voice content on stress reactivity: A simulation paradigm of auditory verbal hallucinations. Schizophrenia research, . https://doi.org/10.1016/j.schres.2019.07.019
Baumeister, David, et al. "The effects of voice content on stress reactivity: A simulation paradigm of auditory verbal hallucinations." Schizophrenia research vol. (2019). doi: https://doi.org/10.1016/j.schres.2019.07.019
Baumeister D, Peters E, Pruessner J, Howes O, Chadwick P. The effects of voice content on stress reactivity: A simulation paradigm of auditory verbal hallucinations. Schizophr Res. 2019 Aug 01; doi: 10.1016/j.schres.2019.07.019. Epub 2019 Aug 01. PMID: 31377050.
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Prevalence of serum N-methyl-d-aspartate receptor autoantibodies in refractory psychosis - ADDENDUM.

The British journal of psychiatry : the journal of mental science

Beck K, Lally J, Shergill SS, Bloomfield MAP, MacCabe JH, Gaughran F, Howes OD.
PMID: 31180301
Br J Psychiatry. 2019 Aug;215(2):507. doi: 10.1192/bjp.2019.141. Epub 2019 Jun 10.

No abstract available.

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Beck K, Lally J, Shergill SS, et al. Prevalence of serum N-methyl-d-aspartate receptor autoantibodies in refractory psychosis - ADDENDUM. Br J Psychiatry. 2019;215(2):507doi: 10.1192/bjp.2019.141.
Beck, K., Lally, J., Shergill, S. S., Bloomfield, M. A. P., MacCabe, J. H., Gaughran, F., & Howes, O. D. (2019). Prevalence of serum N-methyl-d-aspartate receptor autoantibodies in refractory psychosis - ADDENDUM. The British journal of psychiatry : the journal of mental science, 215(2), 507. https://doi.org/10.1192/bjp.2019.141
Beck, Katherine, et al. "Prevalence of serum N-methyl-d-aspartate receptor autoantibodies in refractory psychosis - ADDENDUM." The British journal of psychiatry : the journal of mental science vol. 215,2 (2019): 507. doi: https://doi.org/10.1192/bjp.2019.141
Beck K, Lally J, Shergill SS, Bloomfield MAP, MacCabe JH, Gaughran F, Howes OD. Prevalence of serum N-methyl-d-aspartate receptor autoantibodies in refractory psychosis - ADDENDUM. Br J Psychiatry. 2019 Aug;215(2):507. doi: 10.1192/bjp.2019.141. Epub 2019 Jun 10. PMID: 31180301; PMCID: PMC8055578.
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What causes psychosis? An umbrella review of risk and protective factors.

World psychiatry : official journal of the World Psychiatric Association (WPA)

Radua J, Ramella-Cravaro V, Ioannidis JPA, Reichenberg A, Phiphopthatsanee N, Amir T, Yenn Thoo H, Oliver D, Davies C, Morgan C, McGuire P, Murray RM, Fusar-Poli P.
PMID: 29352556
World Psychiatry. 2018 Feb;17(1):49-66. doi: 10.1002/wps.20490.

Psychosis is a heterogeneous psychiatric condition for which a multitude of risk and protective factors have been suggested. This umbrella review aimed to classify the strength of evidence for the associations between each factor and psychotic disorders whilst controlling...

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Radua J, Ramella-Cravaro V, Ioannidis JPA, et al. What causes psychosis? An umbrella review of risk and protective factors. World Psychiatry. 2018;17(1):49-66doi: 10.1002/wps.20490.
Radua, J., Ramella-Cravaro, V., Ioannidis, J. P. A., Reichenberg, A., Phiphopthatsanee, N., Amir, T., Yenn Thoo, H., Oliver, D., Davies, C., Morgan, C., McGuire, P., Murray, R. M., & Fusar-Poli, P. (2018). What causes psychosis? An umbrella review of risk and protective factors. World psychiatry : official journal of the World Psychiatric Association (WPA), 17(1), 49-66. https://doi.org/10.1002/wps.20490
Radua, Joaquim, et al. "What causes psychosis? An umbrella review of risk and protective factors." World psychiatry : official journal of the World Psychiatric Association (WPA) vol. 17,1 (2018): 49-66. doi: https://doi.org/10.1002/wps.20490
Radua J, Ramella-Cravaro V, Ioannidis JPA, Reichenberg A, Phiphopthatsanee N, Amir T, Yenn Thoo H, Oliver D, Davies C, Morgan C, McGuire P, Murray RM, Fusar-Poli P. What causes psychosis? An umbrella review of risk and protective factors. World Psychiatry. 2018 Feb;17(1):49-66. doi: 10.1002/wps.20490. PMID: 29352556; PMCID: PMC5775150.
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Enhanced Dopamine in Prodromal Schizophrenia (EDiPS): a new animal model of relevance to schizophrenia.

NPJ schizophrenia

Petty A, Cui X, Tesiram Y, Kirik D, Howes O, Eyles D.
PMID: 30926827
NPJ Schizophr. 2019 Mar 29;5(1):6. doi: 10.1038/s41537-019-0074-z.

One of the most robust neurochemical abnormalities reported in patients living with schizophrenia is an increase in dopamine (DA) synthesis and release in the dorsal striatum (DS). Importantly, it appears that this increase progresses as a patient transitions from...

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Petty A, Cui X, Tesiram Y, et al. Enhanced Dopamine in Prodromal Schizophrenia (EDiPS): a new animal model of relevance to schizophrenia. NPJ Schizophr. 2019;5(1):6doi: 10.1038/s41537-019-0074-z.
Petty, A., Cui, X., Tesiram, Y., Kirik, D., Howes, O., & Eyles, D. (2019). Enhanced Dopamine in Prodromal Schizophrenia (EDiPS): a new animal model of relevance to schizophrenia. NPJ schizophrenia, 5(1), 6. https://doi.org/10.1038/s41537-019-0074-z
Petty, Alice, et al. "Enhanced Dopamine in Prodromal Schizophrenia (EDiPS): a new animal model of relevance to schizophrenia." NPJ schizophrenia vol. 5,1 (2019): 6. doi: https://doi.org/10.1038/s41537-019-0074-z
Petty A, Cui X, Tesiram Y, Kirik D, Howes O, Eyles D. Enhanced Dopamine in Prodromal Schizophrenia (EDiPS): a new animal model of relevance to schizophrenia. NPJ Schizophr. 2019 Mar 29;5(1):6. doi: 10.1038/s41537-019-0074-z. PMID: 30926827; PMCID: PMC6441087.
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