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A quantitative system pharmacology computer model for cognitive deficits in schizophrenia.

CPT: pharmacometrics & systems pharmacology

Geerts H, Roberts P, Spiros A.
PMID: 23887686
CPT Pharmacometrics Syst Pharmacol. 2013 Apr 03;2:e36. doi: 10.1038/psp.2013.12.

Although the positive symptoms of schizophrenia are reasonably well-controlled by current antipsychotics, cognitive impairment remains largely unaddressed. The Matrics initiative lays out a regulatory path forward and a number of targets have been tested in the clinic, so far...

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Geerts H, Roberts P, Spiros A. A quantitative system pharmacology computer model for cognitive deficits in schizophrenia. CPT Pharmacometrics Syst Pharmacol. 2013;2:e36doi: 10.1038/psp.2013.12.
Geerts, H., Roberts, P., & Spiros, A. (2013). A quantitative system pharmacology computer model for cognitive deficits in schizophrenia. CPT: pharmacometrics & systems pharmacology, 2e36. https://doi.org/10.1038/psp.2013.12
Geerts, H, et al. "A quantitative system pharmacology computer model for cognitive deficits in schizophrenia." CPT: pharmacometrics & systems pharmacology vol. 2 (2013): e36. doi: https://doi.org/10.1038/psp.2013.12
Geerts H, Roberts P, Spiros A. A quantitative system pharmacology computer model for cognitive deficits in schizophrenia. CPT Pharmacometrics Syst Pharmacol. 2013 Apr 03;2:e36. doi: 10.1038/psp.2013.12. PMID: 23887686; PMCID: PMC3636495.
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The neuropharmacology of implicit learning.

Current neuropharmacology

Uddén J, Folia V, Petersson KM.
PMID: 21629444
Curr Neuropharmacol. 2010 Dec;8(4):367-81. doi: 10.2174/157015910793358178.

Two decades of pharmacologic research on the human capacity to implicitly acquire knowledge as well as cognitive skills and procedures have yielded surprisingly few conclusive insights. We review the empirical literature of the neuropharmacology of implicit learning. We evaluate...

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Uddén J, Folia V, Petersson KM. The neuropharmacology of implicit learning. Curr Neuropharmacol. 2010;8(4):367-81doi: 10.2174/157015910793358178.
Uddén, J., Folia, V., & Petersson, K. M. (2010). The neuropharmacology of implicit learning. Current neuropharmacology, 8(4), 367-81. https://doi.org/10.2174/157015910793358178
Uddén, Julia, et al. "The neuropharmacology of implicit learning." Current neuropharmacology vol. 8,4 (2010): 367-81. doi: https://doi.org/10.2174/157015910793358178
Uddén J, Folia V, Petersson KM. The neuropharmacology of implicit learning. Curr Neuropharmacol. 2010 Dec;8(4):367-81. doi: 10.2174/157015910793358178. PMID: 21629444; PMCID: PMC3080593.
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Ketogenic diet for depression: A potential dietary regimen to maintain euthymia?.

Progress in neuro-psychopharmacology & biological psychiatry

Włodarczyk A, Cubała WJ, Stawicki M.
PMID: 33497756
Prog Neuropsychopharmacol Biol Psychiatry. 2021 Jul 13;109:110257. doi: 10.1016/j.pnpbp.2021.110257. Epub 2021 Jan 23.

Approximately 30% of patients with major depressive disorder (MDD) present resistance to current pharmacological therapies. There is the possibility that an appropriate nutritional regimen can maintain euthymia. Poor dietary pattern and lack of nutritional knowledge are common among today's...

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Włodarczyk A, Cubała WJ, Stawicki M. Ketogenic diet for depression: A potential dietary regimen to maintain euthymia?. Prog Neuropsychopharmacol Biol Psychiatry. 2021;109:110257doi: 10.1016/j.pnpbp.2021.110257.
Włodarczyk, A., Cubała, W. J., & Stawicki, M. (2021). Ketogenic diet for depression: A potential dietary regimen to maintain euthymia?. Progress in neuro-psychopharmacology & biological psychiatry, 109110257. https://doi.org/10.1016/j.pnpbp.2021.110257
Włodarczyk, Adam, et al. "Ketogenic diet for depression: A potential dietary regimen to maintain euthymia?." Progress in neuro-psychopharmacology & biological psychiatry vol. 109 (2021): 110257. doi: https://doi.org/10.1016/j.pnpbp.2021.110257
Włodarczyk A, Cubała WJ, Stawicki M. Ketogenic diet for depression: A potential dietary regimen to maintain euthymia?. Prog Neuropsychopharmacol Biol Psychiatry. 2021 Jul 13;109:110257. doi: 10.1016/j.pnpbp.2021.110257. Epub 2021 Jan 23. PMID: 33497756.
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GABAergic but not Antidepressant Medications Increase Risk for .

Open forum infectious diseases

Lalani F, Young EH, Panchal RM, Reveles KR.
PMID: 32939356
Open Forum Infect Dis. 2020 Aug 13;7(9):ofaa353. doi: 10.1093/ofid/ofaa353. eCollection 2020 Sep.

METHODS: This was a retrospective case-control study of patients seen in Veterans Health Administration facilities from October 2002 to September 2014. CDI and non-CDI control patients were propensity score matched 1:1 using a maximum caliper of 0.0001. Antidepressant and...

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Lalani F, Young EH, Panchal RM, et al. GABAergic but not Antidepressant Medications Increase Risk for . Open Forum Infect Dis. 2020;7(9):ofaa353doi: 10.1093/ofid/ofaa353.
Lalani, F., Young, E. H., Panchal, R. M., & Reveles, K. R. (2020). GABAergic but not Antidepressant Medications Increase Risk for . Open forum infectious diseases, 7(9), ofaa353. https://doi.org/10.1093/ofid/ofaa353
Lalani, Falak, et al. "GABAergic but not Antidepressant Medications Increase Risk for ." Open forum infectious diseases vol. 7,9 (2020): ofaa353. doi: https://doi.org/10.1093/ofid/ofaa353
Lalani F, Young EH, Panchal RM, Reveles KR. GABAergic but not Antidepressant Medications Increase Risk for . Open Forum Infect Dis. 2020 Aug 13;7(9):ofaa353. doi: 10.1093/ofid/ofaa353. eCollection 2020 Sep. PMID: 32939356; PMCID: PMC7486948.
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Parkinson's Disease and Neurodegeneration: GABA-Collapse Hypothesis.

Frontiers in neuroscience

Błaszczyk JW.
PMID: 27375426
Front Neurosci. 2016 Jun 09;10:269. doi: 10.3389/fnins.2016.00269. eCollection 2016.

Neurodegenerative diseases constitute a heterogeneous group of age-related disorders that are characterized by a slow but irreversible deterioration of brain functions. Evidence accumulated over more than two decades has implicated calcium-related homeostatic mechanisms, giving rise to the Ca(2+) hypothesis...

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Błaszczyk JW. Parkinson's Disease and Neurodegeneration: GABA-Collapse Hypothesis. Front Neurosci. 2016;10:269doi: 10.3389/fnins.2016.00269.
Błaszczyk, J. W. (2016). Parkinson's Disease and Neurodegeneration: GABA-Collapse Hypothesis. Frontiers in neuroscience, 10269. https://doi.org/10.3389/fnins.2016.00269
Błaszczyk, Janusz W. "Parkinson's Disease and Neurodegeneration: GABA-Collapse Hypothesis." Frontiers in neuroscience vol. 10 (2016): 269. doi: https://doi.org/10.3389/fnins.2016.00269
Błaszczyk JW. Parkinson's Disease and Neurodegeneration: GABA-Collapse Hypothesis. Front Neurosci. 2016 Jun 09;10:269. doi: 10.3389/fnins.2016.00269. eCollection 2016. PMID: 27375426; PMCID: PMC4899466.
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Novel modulatory effects of neurosteroids and benzodiazepines on excitatory and inhibitory neurons excitability: a multi-electrode array recording study.

Frontiers in neural circuits

Puia G, Gullo F, Dossi E, Lecchi M, Wanke E.
PMID: 23233835
Front Neural Circuits. 2012 Nov 27;6:94. doi: 10.3389/fncir.2012.00094. eCollection 2012.

The balance between glutamate- and GABA-mediated neurotransmission in the brain is fundamental in the nervous system, but it is regulated by the "tonic" release of a variety of endogenous factors. One such important group of molecules are the neurosteroids...

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Puia G, Gullo F, Dossi E, et al. Novel modulatory effects of neurosteroids and benzodiazepines on excitatory and inhibitory neurons excitability: a multi-electrode array recording study. Front Neural Circuits. 2012;6:94doi: 10.3389/fncir.2012.00094.
Puia, G., Gullo, F., Dossi, E., Lecchi, M., & Wanke, E. (2012). Novel modulatory effects of neurosteroids and benzodiazepines on excitatory and inhibitory neurons excitability: a multi-electrode array recording study. Frontiers in neural circuits, 694. https://doi.org/10.3389/fncir.2012.00094
Puia, Giulia, et al. "Novel modulatory effects of neurosteroids and benzodiazepines on excitatory and inhibitory neurons excitability: a multi-electrode array recording study." Frontiers in neural circuits vol. 6 (2012): 94. doi: https://doi.org/10.3389/fncir.2012.00094
Puia G, Gullo F, Dossi E, Lecchi M, Wanke E. Novel modulatory effects of neurosteroids and benzodiazepines on excitatory and inhibitory neurons excitability: a multi-electrode array recording study. Front Neural Circuits. 2012 Nov 27;6:94. doi: 10.3389/fncir.2012.00094. eCollection 2012. PMID: 23233835; PMCID: PMC3516127.
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GABA-BZD Receptor Modulating Mechanism of Panax quinquefolius against 72-h Sleep Deprivation Induced Anxiety like Behavior: Possible Roles of Oxidative Stress, Mitochondrial Dysfunction and Neuroinflammation.

Frontiers in neuroscience

Chanana P, Kumar A.
PMID: 27013946
Front Neurosci. 2016 Mar 07;10:84. doi: 10.3389/fnins.2016.00084. eCollection 2016.

RATIONALE: Panax quinquefolius (American Ginseng) is known for its therapeutic potential against various neurological disorders, but its plausible mechanism of action still remains undeciphered. GABA (Gamma Amino Butyric Acid) plays an important role in sleep wake cycle homeostasis. Thus,...

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Chanana P, Kumar A. GABA-BZD Receptor Modulating Mechanism of Panax quinquefolius against 72-h Sleep Deprivation Induced Anxiety like Behavior: Possible Roles of Oxidative Stress, Mitochondrial Dysfunction and Neuroinflammation. Front Neurosci. 2016;10:84doi: 10.3389/fnins.2016.00084.
Chanana, P., & Kumar, A. (2016). GABA-BZD Receptor Modulating Mechanism of Panax quinquefolius against 72-h Sleep Deprivation Induced Anxiety like Behavior: Possible Roles of Oxidative Stress, Mitochondrial Dysfunction and Neuroinflammation. Frontiers in neuroscience, 1084. https://doi.org/10.3389/fnins.2016.00084
Chanana, Priyanka, and Kumar, Anil. "GABA-BZD Receptor Modulating Mechanism of Panax quinquefolius against 72-h Sleep Deprivation Induced Anxiety like Behavior: Possible Roles of Oxidative Stress, Mitochondrial Dysfunction and Neuroinflammation." Frontiers in neuroscience vol. 10 (2016): 84. doi: https://doi.org/10.3389/fnins.2016.00084
Chanana P, Kumar A. GABA-BZD Receptor Modulating Mechanism of Panax quinquefolius against 72-h Sleep Deprivation Induced Anxiety like Behavior: Possible Roles of Oxidative Stress, Mitochondrial Dysfunction and Neuroinflammation. Front Neurosci. 2016 Mar 07;10:84. doi: 10.3389/fnins.2016.00084. eCollection 2016. PMID: 27013946; PMCID: PMC4779932.
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Showing 1 to 7 of 7 entries