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Showing 1 to 12 of 33 entries
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Glia Maturation Factor and Mitochondrial Uncoupling Proteins 2 and 4 Expression in the Temporal Cortex of Alzheimer's Disease Brain.

Frontiers in aging neuroscience

Thangavel R, Kempuraj D, Zaheer S, Raikwar S, Ahmed ME, Selvakumar GP, Iyer SS, Zaheer A.
PMID: 28572767
Front Aging Neurosci. 2017 May 18;9:150. doi: 10.3389/fnagi.2017.00150. eCollection 2017.

Alzheimer's disease (AD) is characterized by the presence of neuropathological lesions containing amyloid plaques (APs) and neurofibrillary tangles (NFTs). AD is associated with mitochondrial dysfunctions, neuroinflammation and neurodegeneration in the brain. We have previously demonstrated enhanced expression of the...

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Thangavel R, Kempuraj D, Zaheer S, et al. Glia Maturation Factor and Mitochondrial Uncoupling Proteins 2 and 4 Expression in the Temporal Cortex of Alzheimer's Disease Brain. Front Aging Neurosci. 2017;9:150doi: 10.3389/fnagi.2017.00150.
Thangavel, R., Kempuraj, D., Zaheer, S., Raikwar, S., Ahmed, M. E., Selvakumar, G. P., Iyer, S. S., & Zaheer, A. (2017). Glia Maturation Factor and Mitochondrial Uncoupling Proteins 2 and 4 Expression in the Temporal Cortex of Alzheimer's Disease Brain. Frontiers in aging neuroscience, 9150. https://doi.org/10.3389/fnagi.2017.00150
Thangavel, Ramasamy, et al. "Glia Maturation Factor and Mitochondrial Uncoupling Proteins 2 and 4 Expression in the Temporal Cortex of Alzheimer's Disease Brain." Frontiers in aging neuroscience vol. 9 (2017): 150. doi: https://doi.org/10.3389/fnagi.2017.00150
Thangavel R, Kempuraj D, Zaheer S, Raikwar S, Ahmed ME, Selvakumar GP, Iyer SS, Zaheer A. Glia Maturation Factor and Mitochondrial Uncoupling Proteins 2 and 4 Expression in the Temporal Cortex of Alzheimer's Disease Brain. Front Aging Neurosci. 2017 May 18;9:150. doi: 10.3389/fnagi.2017.00150. eCollection 2017. PMID: 28572767; PMCID: PMC5435744.
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Oligosaccharide alterations of rat glioblast membrane-bound glycoproteins during differentiation induced by glia maturation factor.

Neurochemistry international

Ito J, Kato T, Tanaka R.
PMID: 20493026
Neurochem Int. 1986;8(1):31-40. doi: 10.1016/0197-0186(86)90097-5.

Differentiation of normal glioblasts was induced by glia maturation factor (GMF), and the structural change in the oligosaccharide chains of the plasmalemmal glycoproteins was investigated. After the glycopeptides obtained by trypsin treatment of the intact cells had been digested...

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Ito J, Kato T, Tanaka R. Oligosaccharide alterations of rat glioblast membrane-bound glycoproteins during differentiation induced by glia maturation factor. Neurochem Int. 1986;8(1):31-40doi: 10.1016/0197-0186(86)90097-5.
Ito, J., Kato, T., & Tanaka, R. (1986). Oligosaccharide alterations of rat glioblast membrane-bound glycoproteins during differentiation induced by glia maturation factor. Neurochemistry international, 8(1), 31-40. https://doi.org/10.1016/0197-0186(86)90097-5
Ito, J, et al. "Oligosaccharide alterations of rat glioblast membrane-bound glycoproteins during differentiation induced by glia maturation factor." Neurochemistry international vol. 8,1 (1986): 31-40. doi: https://doi.org/10.1016/0197-0186(86)90097-5
Ito J, Kato T, Tanaka R. Oligosaccharide alterations of rat glioblast membrane-bound glycoproteins during differentiation induced by glia maturation factor. Neurochem Int. 1986;8(1):31-40. doi: 10.1016/0197-0186(86)90097-5. PMID: 20493026.
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Absence of Glia Maturation Factor Protects from Axonal Injury and Motor Behavioral Impairments after Traumatic Brain Injury.

Experimental neurobiology

Selvakumar GP, Ahmed ME, Iyer SS, Thangavel R, Kempuraj D, Raikwar SP, Bazley K, Wu K, Khan A, Kukulka K, Bussinger B, Zaheer S, Burton C, James D, Zaheer A.
PMID: 32565489
Exp Neurobiol. 2020 Jun 30;29(3):230-248. doi: 10.5607/en20017.

Traumatic brain injury (TBI) causes disability and death, accelerating the progression towards Alzheimer's disease and Parkinson's disease (PD). TBI causes serious motor and cognitive impairments, as seen in PD that arise during the period of the initial insult. However,...

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Selvakumar GP, Ahmed ME, Iyer SS, et al. Absence of Glia Maturation Factor Protects from Axonal Injury and Motor Behavioral Impairments after Traumatic Brain Injury. Exp Neurobiol. 2020;29(3):230-248doi: 10.5607/en20017.
Selvakumar, G. P., Ahmed, M. E., Iyer, S. S., Thangavel, R., Kempuraj, D., Raikwar, S. P., Bazley, K., Wu, K., Khan, A., Kukulka, K., Bussinger, B., Zaheer, S., Burton, C., James, D., & Zaheer, A. (2020). Absence of Glia Maturation Factor Protects from Axonal Injury and Motor Behavioral Impairments after Traumatic Brain Injury. Experimental neurobiology, 29(3), 230-248. https://doi.org/10.5607/en20017
Selvakumar, Govindhasamy Pushpavathi, et al. "Absence of Glia Maturation Factor Protects from Axonal Injury and Motor Behavioral Impairments after Traumatic Brain Injury." Experimental neurobiology vol. 29,3 (2020): 230-248. doi: https://doi.org/10.5607/en20017
Selvakumar GP, Ahmed ME, Iyer SS, Thangavel R, Kempuraj D, Raikwar SP, Bazley K, Wu K, Khan A, Kukulka K, Bussinger B, Zaheer S, Burton C, James D, Zaheer A. Absence of Glia Maturation Factor Protects from Axonal Injury and Motor Behavioral Impairments after Traumatic Brain Injury. Exp Neurobiol. 2020 Jun 30;29(3):230-248. doi: 10.5607/en20017. PMID: 32565489; PMCID: PMC7344375.
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Proteomics in Schizophrenia: A Gateway to Discover Potential Biomarkers of Psychoneuroimmune Pathways.

Frontiers in psychiatry

Rodrigues-Amorim D, Rivera-Baltanás T, Vallejo-Curto MDC, Rodriguez-Jamardo C, de Las Heras E, Barreiro-Villar C, Blanco-Formoso M, Fernández-Palleiro P, Álvarez-Ariza M, López M, García-Caballero A, Olivares JM, Spuch C.
PMID: 31849731
Front Psychiatry. 2019 Nov 29;10:885. doi: 10.3389/fpsyt.2019.00885. eCollection 2019.

Schizophrenia is a severe and disabling psychiatric disorder with a complex and multifactorial etiology. The lack of consensus regarding the multifaceted dysfunction of this ailment has increased the need to explore new research lines. This research makes use of...

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Rodrigues-Amorim D, Rivera-Baltanás T, Vallejo-Curto MDC, et al. Proteomics in Schizophrenia: A Gateway to Discover Potential Biomarkers of Psychoneuroimmune Pathways. Front Psychiatry. 2019;10:885doi: 10.3389/fpsyt.2019.00885.
Rodrigues-Amorim, D., Rivera-Baltanás, T., Vallejo-Curto, M. D. C., Rodriguez-Jamardo, C., de Las Heras, E., Barreiro-Villar, C., Blanco-Formoso, M., Fernández-Palleiro, P., Álvarez-Ariza, M., López, M., García-Caballero, A., Olivares, J. M., & Spuch, C. (2019). Proteomics in Schizophrenia: A Gateway to Discover Potential Biomarkers of Psychoneuroimmune Pathways. Frontiers in psychiatry, 10885. https://doi.org/10.3389/fpsyt.2019.00885
Rodrigues-Amorim, Daniela, et al. "Proteomics in Schizophrenia: A Gateway to Discover Potential Biomarkers of Psychoneuroimmune Pathways." Frontiers in psychiatry vol. 10 (2019): 885. doi: https://doi.org/10.3389/fpsyt.2019.00885
Rodrigues-Amorim D, Rivera-Baltanás T, Vallejo-Curto MDC, Rodriguez-Jamardo C, de Las Heras E, Barreiro-Villar C, Blanco-Formoso M, Fernández-Palleiro P, Álvarez-Ariza M, López M, García-Caballero A, Olivares JM, Spuch C. Proteomics in Schizophrenia: A Gateway to Discover Potential Biomarkers of Psychoneuroimmune Pathways. Front Psychiatry. 2019 Nov 29;10:885. doi: 10.3389/fpsyt.2019.00885. eCollection 2019. PMID: 31849731; PMCID: PMC6897280.
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Immune Suppression of Glia Maturation Factor Reverses Behavioral Impairment, Attenuates Amyloid Plaque Pathology and Neuroinflammation in an Alzheimer's Disease Mouse Model.

Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology

Ahmed ME, Selvakumar GP, Thangavel R, Kempuraj D, Raikwar SP, Zaheer S, Iyer S, Zaheer A.
PMID: 32504312
J Neuroimmune Pharmacol. 2021 Jun;16(2):363-375. doi: 10.1007/s11481-020-09929-4. Epub 2020 Jun 05.

Alzheimer's disease (AD) is an irreversible progressive neurodegenerative disorder recognized by accumulation of amyloid-plaques (APs) and neurofibrillary tangles (NFTs) and eventually loss of memory. Glia maturation factor (GMF), a neuroinflammatory protein first time isolated and cloned in our laboratory...

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Ahmed ME, Selvakumar GP, Thangavel R, et al. Immune Suppression of Glia Maturation Factor Reverses Behavioral Impairment, Attenuates Amyloid Plaque Pathology and Neuroinflammation in an Alzheimer's Disease Mouse Model. J Neuroimmune Pharmacol. 2020;16(2):363-375doi: 10.1007/s11481-020-09929-4.
Ahmed, M. E., Selvakumar, G. P., Thangavel, R., Kempuraj, D., Raikwar, S. P., Zaheer, S., Iyer, S., & Zaheer, A. (2021). Immune Suppression of Glia Maturation Factor Reverses Behavioral Impairment, Attenuates Amyloid Plaque Pathology and Neuroinflammation in an Alzheimer's Disease Mouse Model. Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology, 16(2), 363-375. https://doi.org/10.1007/s11481-020-09929-4
Ahmed, Mohammad Ejaz, et al. "Immune Suppression of Glia Maturation Factor Reverses Behavioral Impairment, Attenuates Amyloid Plaque Pathology and Neuroinflammation in an Alzheimer's Disease Mouse Model." Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology vol. 16,2 (2021): 363-375. doi: https://doi.org/10.1007/s11481-020-09929-4
Ahmed ME, Selvakumar GP, Thangavel R, Kempuraj D, Raikwar SP, Zaheer S, Iyer S, Zaheer A. Immune Suppression of Glia Maturation Factor Reverses Behavioral Impairment, Attenuates Amyloid Plaque Pathology and Neuroinflammation in an Alzheimer's Disease Mouse Model. J Neuroimmune Pharmacol. 2021 Jun;16(2):363-375. doi: 10.1007/s11481-020-09929-4. Epub 2020 Jun 05. PMID: 32504312; PMCID: PMC8240471.
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The Actin-Disassembly Protein Glia Maturation Factor γ Enhances Actin Remodeling and B Cell Antigen Receptor Signaling at the Immune Synapse.

Frontiers in cell and developmental biology

Deretic N, Bolger-Munro M, Choi K, Abraham L, Gold MR.
PMID: 34336818
Front Cell Dev Biol. 2021 Jul 09;9:647063. doi: 10.3389/fcell.2021.647063. eCollection 2021.

Signaling by the B cell antigen receptor (BCR) initiates actin remodeling. The assembly of branched actin networks that are nucleated by the Arp2/3 complex exert outward force on the plasma membrane, allowing B cells to form membrane protrusions that...

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Deretic N, Bolger-Munro M, Choi K, et al. The Actin-Disassembly Protein Glia Maturation Factor γ Enhances Actin Remodeling and B Cell Antigen Receptor Signaling at the Immune Synapse. Front Cell Dev Biol. 2021;9:647063doi: 10.3389/fcell.2021.647063.
Deretic, N., Bolger-Munro, M., Choi, K., Abraham, L., & Gold, M. R. (2021). The Actin-Disassembly Protein Glia Maturation Factor γ Enhances Actin Remodeling and B Cell Antigen Receptor Signaling at the Immune Synapse. Frontiers in cell and developmental biology, 9647063. https://doi.org/10.3389/fcell.2021.647063
Deretic, Nikola, et al. "The Actin-Disassembly Protein Glia Maturation Factor γ Enhances Actin Remodeling and B Cell Antigen Receptor Signaling at the Immune Synapse." Frontiers in cell and developmental biology vol. 9 (2021): 647063. doi: https://doi.org/10.3389/fcell.2021.647063
Deretic N, Bolger-Munro M, Choi K, Abraham L, Gold MR. The Actin-Disassembly Protein Glia Maturation Factor γ Enhances Actin Remodeling and B Cell Antigen Receptor Signaling at the Immune Synapse. Front Cell Dev Biol. 2021 Jul 09;9:647063. doi: 10.3389/fcell.2021.647063. eCollection 2021. PMID: 34336818; PMCID: PMC8318000.
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Brain and Peripheral Atypical Inflammatory Mediators Potentiate Neuroinflammation and Neurodegeneration.

Frontiers in cellular neuroscience

Kempuraj D, Thangavel R, Selvakumar GP, Zaheer S, Ahmed ME, Raikwar SP, Zahoor H, Saeed D, Natteru PA, Iyer S, Zaheer A.
PMID: 28790893
Front Cell Neurosci. 2017 Jul 24;11:216. doi: 10.3389/fncel.2017.00216. eCollection 2017.

Neuroinflammatory response is primarily a protective mechanism in the brain. However, excessive and chronic inflammatory responses can lead to deleterious effects involving immune cells, brain cells and signaling molecules. Neuroinflammation induces and accelerates pathogenesis of Parkinson's disease (PD), Alzheimer's...

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Kempuraj D, Thangavel R, Selvakumar GP, et al. Brain and Peripheral Atypical Inflammatory Mediators Potentiate Neuroinflammation and Neurodegeneration. Front Cell Neurosci. 2017;11:216doi: 10.3389/fncel.2017.00216.
Kempuraj, D., Thangavel, R., Selvakumar, G. P., Zaheer, S., Ahmed, M. E., Raikwar, S. P., Zahoor, H., Saeed, D., Natteru, P. A., Iyer, S., & Zaheer, A. (2017). Brain and Peripheral Atypical Inflammatory Mediators Potentiate Neuroinflammation and Neurodegeneration. Frontiers in cellular neuroscience, 11216. https://doi.org/10.3389/fncel.2017.00216
Kempuraj, Duraisamy, et al. "Brain and Peripheral Atypical Inflammatory Mediators Potentiate Neuroinflammation and Neurodegeneration." Frontiers in cellular neuroscience vol. 11 (2017): 216. doi: https://doi.org/10.3389/fncel.2017.00216
Kempuraj D, Thangavel R, Selvakumar GP, Zaheer S, Ahmed ME, Raikwar SP, Zahoor H, Saeed D, Natteru PA, Iyer S, Zaheer A. Brain and Peripheral Atypical Inflammatory Mediators Potentiate Neuroinflammation and Neurodegeneration. Front Cell Neurosci. 2017 Jul 24;11:216. doi: 10.3389/fncel.2017.00216. eCollection 2017. PMID: 28790893; PMCID: PMC5522882.
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Inhibition by diphenylhydantoin of growth and morphological differentiation of glial cells: Implication of calcium.

Neurochemistry international

Okumura-Noji K, Kato T, Ito J, Tanaka R, Wakabayashi S, Wada Y.
PMID: 20504437
Neurochem Int. 1989;14(3):353-9. doi: 10.1016/0197-0186(89)90062-4.

The effects of diphenylhydantoin (DPH) and other anticonvulsants on the growth of glial cells as well as neuroblastoma cell lines was studied. The DPH inhibition of the DNA synthesis was most marked in rat fetal glioblasts induced by glia...

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Okumura-Noji K, Kato T, Ito J, et al. Inhibition by diphenylhydantoin of growth and morphological differentiation of glial cells: Implication of calcium. Neurochem Int. 1989;14(3):353-9doi: 10.1016/0197-0186(89)90062-4.
Okumura-Noji, K., Kato, T., Ito, J., Tanaka, R., Wakabayashi, S., & Wada, Y. (1989). Inhibition by diphenylhydantoin of growth and morphological differentiation of glial cells: Implication of calcium. Neurochemistry international, 14(3), 353-9. https://doi.org/10.1016/0197-0186(89)90062-4
Okumura-Noji, K, et al. "Inhibition by diphenylhydantoin of growth and morphological differentiation of glial cells: Implication of calcium." Neurochemistry international vol. 14,3 (1989): 353-9. doi: https://doi.org/10.1016/0197-0186(89)90062-4
Okumura-Noji K, Kato T, Ito J, Tanaka R, Wakabayashi S, Wada Y. Inhibition by diphenylhydantoin of growth and morphological differentiation of glial cells: Implication of calcium. Neurochem Int. 1989;14(3):353-9. doi: 10.1016/0197-0186(89)90062-4. PMID: 20504437.
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Cytoskeletal regulation of normal rat glioblasts differentiated by glia maturation factor.

Neurochemistry international

Ito J, Kato T, Tanaka R.
PMID: 20504550
Neurochem Int. 1990;16(2):133-40. doi: 10.1016/0197-0186(90)90080-d.

The morphological differentiation of normal rat glioblasts induced by glia maturation factor (GMF) was investigated with regard to the reorganization of the cytoskeletal structures by the use of indirect immunofluorescence staining methods. The microfilaments were reorganized from a stress...

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Ito J, Kato T, Tanaka R. Cytoskeletal regulation of normal rat glioblasts differentiated by glia maturation factor. Neurochem Int. 1990;16(2):133-40doi: 10.1016/0197-0186(90)90080-d.
Ito, J., Kato, T., & Tanaka, R. (1990). Cytoskeletal regulation of normal rat glioblasts differentiated by glia maturation factor. Neurochemistry international, 16(2), 133-40. https://doi.org/10.1016/0197-0186(90)90080-d
Ito, J, et al. "Cytoskeletal regulation of normal rat glioblasts differentiated by glia maturation factor." Neurochemistry international vol. 16,2 (1990): 133-40. doi: https://doi.org/10.1016/0197-0186(90)90080-d
Ito J, Kato T, Tanaka R. Cytoskeletal regulation of normal rat glioblasts differentiated by glia maturation factor. Neurochem Int. 1990;16(2):133-40. doi: 10.1016/0197-0186(90)90080-d. PMID: 20504550.
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Glia Maturation Factor in the Pathogenesis of Alzheimer's disease.

Open access journal of neurology & neurosurgery

Ramaswamy SB, Bhagavan SM, Kaur H, Giler GE, Kempuraj D, Thangavel R, Ahmed ME, Selvakumar GP, Raikwar SP, Zaheer S, Iyer SS, Govindarajan R, Zaheer A.
PMID: 32775957
Open Access J Neurol Neurosurg. 2019;12(3):79-82. Epub 2019 Dec 17.

Alzheimer's disease (AD) is a neurodegenerative and neuroinflammatory disease characterized by the presence of extracellular amyloid plaques (APs) and intracellular neurofibrillary tangles (NFTs) in the brain. There is no disease modifying therapeutic options currently available for this disease. Hippocampus,...

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Ramaswamy SB, Bhagavan SM, Kaur H, et al. Glia Maturation Factor in the Pathogenesis of Alzheimer's disease. Open Access J Neurol Neurosurg. 2019;12(3):79-82
Ramaswamy, S. B., Bhagavan, S. M., Kaur, H., Giler, G. E., Kempuraj, D., Thangavel, R., Ahmed, M. E., Selvakumar, G. P., Raikwar, S. P., Zaheer, S., Iyer, S. S., Govindarajan, R., & Zaheer, A. (2019). Glia Maturation Factor in the Pathogenesis of Alzheimer's disease. Open access journal of neurology & neurosurgery, 12(3), 79-82.
Ramaswamy, Swathi Beladakere, et al. "Glia Maturation Factor in the Pathogenesis of Alzheimer's disease." Open access journal of neurology & neurosurgery vol. 12,3 (2019): 79-82.
Ramaswamy SB, Bhagavan SM, Kaur H, Giler GE, Kempuraj D, Thangavel R, Ahmed ME, Selvakumar GP, Raikwar SP, Zaheer S, Iyer SS, Govindarajan R, Zaheer A. Glia Maturation Factor in the Pathogenesis of Alzheimer's disease. Open Access J Neurol Neurosurg. 2019;12(3):79-82. Epub 2019 Dec 17. PMID: 32775957; PMCID: PMC7413177.
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Glia Maturation Factor Beta as a Novel Biomarker and Therapeutic Target for Hepatocellular Carcinoma.

Frontiers in oncology

Sun W, Hu C, Wang T, Wang J, Zhang J, Gao F, Ou Q, Tian H, Jin C, Xu J, Zhang J, Xu GT, Lu L.
PMID: 34796110
Front Oncol. 2021 Nov 02;11:744331. doi: 10.3389/fonc.2021.744331. eCollection 2021.

Hepatocellular carcinoma (HCC) is one of the most common types of cancer. The novel sensitive biomarkers and therapeutic targets are urgently needed for the early diagnosis of HCC and improvement of clinical outcomes. Glia maturation factor-β (GMFB) is a...

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Sun W, Hu C, Wang T, et al. Glia Maturation Factor Beta as a Novel Biomarker and Therapeutic Target for Hepatocellular Carcinoma. Front Oncol. 2021;11:744331doi: 10.3389/fonc.2021.744331.
Sun, W., Hu, C., Wang, T., Wang, J., Zhang, J., Gao, F., Ou, Q., Tian, H., Jin, C., Xu, J., Zhang, J., Xu, G. T., & Lu, L. (2021). Glia Maturation Factor Beta as a Novel Biomarker and Therapeutic Target for Hepatocellular Carcinoma. Frontiers in oncology, 11744331. https://doi.org/10.3389/fonc.2021.744331
Sun, Wan, et al. "Glia Maturation Factor Beta as a Novel Biomarker and Therapeutic Target for Hepatocellular Carcinoma." Frontiers in oncology vol. 11 (2021): 744331. doi: https://doi.org/10.3389/fonc.2021.744331
Sun W, Hu C, Wang T, Wang J, Zhang J, Gao F, Ou Q, Tian H, Jin C, Xu J, Zhang J, Xu GT, Lu L. Glia Maturation Factor Beta as a Novel Biomarker and Therapeutic Target for Hepatocellular Carcinoma. Front Oncol. 2021 Nov 02;11:744331. doi: 10.3389/fonc.2021.744331. eCollection 2021. PMID: 34796110; PMCID: PMC8593204.
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Deletion of gmfA induces keratocyte-like migration in Dictyostelium.

FEBS open bio

Fujimoto K, Nakano K, Kuwayama H, Yumura S.
PMID: 34855306
FEBS Open Bio. 2021 Dec 02; doi: 10.1002/2211-5463.13339. Epub 2021 Dec 02.

Glia maturation factor (GMF) has been established as an inactivating factor of the actin-related protein 2/3 (Arp2/3) complex, which regulates actin assembly. Regulation of actin assembly and reorganization is crucial for various cellular events, such as cell migration, cell...

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Fujimoto K, Nakano K, Kuwayama H, et al. Deletion of gmfA induces keratocyte-like migration in Dictyostelium. FEBS Open Bio. 2021;doi: 10.1002/2211-5463.13339.
Fujimoto, K., Nakano, K., Kuwayama, H., & Yumura, S. (2021). Deletion of gmfA induces keratocyte-like migration in Dictyostelium. FEBS open bio, . https://doi.org/10.1002/2211-5463.13339
Fujimoto, Koushiro, et al. "Deletion of gmfA induces keratocyte-like migration in Dictyostelium." FEBS open bio vol. (2021). doi: https://doi.org/10.1002/2211-5463.13339
Fujimoto K, Nakano K, Kuwayama H, Yumura S. Deletion of gmfA induces keratocyte-like migration in Dictyostelium. FEBS Open Bio. 2021 Dec 02; doi: 10.1002/2211-5463.13339. Epub 2021 Dec 02. PMID: 34855306.
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