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Showing 1 to 12 of 42 entries
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DASH (Dietary Approaches to Stop Hypertension) Diet and Risk of Subsequent Kidney Disease.

American journal of kidney diseases : the official journal of the National Kidney Foundation

Rebholz CM, Crews DC, Grams ME, Steffen LM, Levey AS, Miller ER, Appel LJ, Coresh J.
PMID: 27519166
Am J Kidney Dis. 2016 Dec;68(6):853-861. doi: 10.1053/j.ajkd.2016.05.019. Epub 2016 Aug 09.

BACKGROUND: There are established guidelines for recommended dietary intake for hypertension treatment and cardiovascular disease prevention. Evidence is lacking for effective dietary patterns for kidney disease prevention.STUDY DESIGN: Prospective cohort study.SETTING & PARTICIPANTS: Atherosclerosis Risk in Communities (ARIC) Study...

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Rebholz CM, Crews DC, Grams ME, et al. DASH (Dietary Approaches to Stop Hypertension) Diet and Risk of Subsequent Kidney Disease. Am J Kidney Dis. 2016;68(6):853-861doi: 10.1053/j.ajkd.2016.05.019.
Rebholz, C. M., Crews, D. C., Grams, M. E., Steffen, L. M., Levey, A. S., Miller, E. R., Appel, L. J., & Coresh, J. (2016). DASH (Dietary Approaches to Stop Hypertension) Diet and Risk of Subsequent Kidney Disease. American journal of kidney diseases : the official journal of the National Kidney Foundation, 68(6), 853-861. https://doi.org/10.1053/j.ajkd.2016.05.019
Rebholz, Casey M, et al. "DASH (Dietary Approaches to Stop Hypertension) Diet and Risk of Subsequent Kidney Disease." American journal of kidney diseases : the official journal of the National Kidney Foundation vol. 68,6 (2016): 853-861. doi: https://doi.org/10.1053/j.ajkd.2016.05.019
Rebholz CM, Crews DC, Grams ME, Steffen LM, Levey AS, Miller ER, Appel LJ, Coresh J. DASH (Dietary Approaches to Stop Hypertension) Diet and Risk of Subsequent Kidney Disease. Am J Kidney Dis. 2016 Dec;68(6):853-861. doi: 10.1053/j.ajkd.2016.05.019. Epub 2016 Aug 09. PMID: 27519166; PMCID: PMC5123940.
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Galectin-3 and Venous Thromboembolism Incidence: the Atherosclerosis Risk in Communities (ARIC) Study.

Research and practice in thrombosis and haemostasis

Fashanu OE, Heckbert SR, Aguilar D, Jensen PN, Ballantyne CM, Basu S, Hoogeveen RC, deFilippi C, Cushman M, Folsom AR.
PMID: 29152608
Res Pract Thromb Haemost. 2017 Oct;1(2):223-230. doi: 10.1002/rth2.12038. Epub 2017 Sep 04.

BACKGROUND: The inflammatory biomarker galectin-3 contributes to pathologic conditions such as heart failure and stimulates murine thrombogenesis. Its association with venous thromboembolism (VTE) has been sparsely studied.OBJECTIVES: To assess the prospective association of plasma galectin-3 and the METHODS: We...

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Fashanu OE, Heckbert SR, Aguilar D, et al. Galectin-3 and Venous Thromboembolism Incidence: the Atherosclerosis Risk in Communities (ARIC) Study. Res Pract Thromb Haemost. 2017;1(2):223-230doi: 10.1002/rth2.12038.
Fashanu, O. E., Heckbert, S. R., Aguilar, D., Jensen, P. N., Ballantyne, C. M., Basu, S., Hoogeveen, R. C., deFilippi, C., Cushman, M., & Folsom, A. R. (2017). Galectin-3 and Venous Thromboembolism Incidence: the Atherosclerosis Risk in Communities (ARIC) Study. Research and practice in thrombosis and haemostasis, 1(2), 223-230. https://doi.org/10.1002/rth2.12038
Fashanu, Oluwaseun E, et al. "Galectin-3 and Venous Thromboembolism Incidence: the Atherosclerosis Risk in Communities (ARIC) Study." Research and practice in thrombosis and haemostasis vol. 1,2 (2017): 223-230. doi: https://doi.org/10.1002/rth2.12038
Fashanu OE, Heckbert SR, Aguilar D, Jensen PN, Ballantyne CM, Basu S, Hoogeveen RC, deFilippi C, Cushman M, Folsom AR. Galectin-3 and Venous Thromboembolism Incidence: the Atherosclerosis Risk in Communities (ARIC) Study. Res Pract Thromb Haemost. 2017 Oct;1(2):223-230. doi: 10.1002/rth2.12038. Epub 2017 Sep 04. PMID: 29152608; PMCID: PMC5685543.
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Mild Cognitive Impairment and Dementia Prevalence: The Atherosclerosis Risk in Communities Neurocognitive Study (ARIC-NCS).

Alzheimer's & dementia (Amsterdam, Netherlands)

Knopman DS, Gottesman RF, Sharrett AR, Wruck LM, Windham BG, Coker L, Schneider AL, Hengrui S, Alonso A, Coresh J, Albert MS, Mosley TH.
PMID: 26949733
Alzheimers Dement (Amst). 2016;2:1-11. doi: 10.1016/j.dadm.2015.12.002.

INTRODUCTION: We examined prevalence of mild cognitive impairment (MCI) and dementia in the Atherosclerosis Risk in Communities (ARIC) Neurocognitive Study.METHODS: Beginning in June, 2011, we invited all surviving ARIC participants to undergo cognitive, neurological and brain imaging assessments to...

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Knopman DS, Gottesman RF, Sharrett AR, et al. Mild Cognitive Impairment and Dementia Prevalence: The Atherosclerosis Risk in Communities Neurocognitive Study (ARIC-NCS). Alzheimers Dement (Amst). 2016;2:1-11doi: 10.1016/j.dadm.2015.12.002.
Knopman, D. S., Gottesman, R. F., Sharrett, A. R., Wruck, L. M., Windham, B. G., Coker, L., Schneider, A. L., Hengrui, S., Alonso, A., Coresh, J., Albert, M. S., & Mosley, T. H. (2016). Mild Cognitive Impairment and Dementia Prevalence: The Atherosclerosis Risk in Communities Neurocognitive Study (ARIC-NCS). Alzheimer's & dementia (Amsterdam, Netherlands), 21-11. https://doi.org/10.1016/j.dadm.2015.12.002
Knopman, David S, et al. "Mild Cognitive Impairment and Dementia Prevalence: The Atherosclerosis Risk in Communities Neurocognitive Study (ARIC-NCS)." Alzheimer's & dementia (Amsterdam, Netherlands) vol. 2 (2016): 1-11. doi: https://doi.org/10.1016/j.dadm.2015.12.002
Knopman DS, Gottesman RF, Sharrett AR, Wruck LM, Windham BG, Coker L, Schneider AL, Hengrui S, Alonso A, Coresh J, Albert MS, Mosley TH. Mild Cognitive Impairment and Dementia Prevalence: The Atherosclerosis Risk in Communities Neurocognitive Study (ARIC-NCS). Alzheimers Dement (Amst). 2016;2:1-11. doi: 10.1016/j.dadm.2015.12.002. PMID: 26949733; PMCID: PMC4772876.
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Pleiotropy and pathway analyses of genetic variants associated with both type 2 diabetes and prostate cancer.

International journal of molecular epidemiology and genetics

Raynor LA, Pankow JS, Rasmussen-Torvik LJ, Tang W, Prizment A, Couper DJ.
PMID: 23565322
Int J Mol Epidemiol Genet. 2013;4(1):49-60. Epub 2013 Mar 18.

AIMS: Epidemiological evidence shows that diabetes is associated with a reduced risk of prostate cancer. The objective of this study was to identify genes that may contribute to both type 2 diabetes and prostate cancer outcomes and the biological...

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Raynor LA, Pankow JS, Rasmussen-Torvik LJ, et al. Pleiotropy and pathway analyses of genetic variants associated with both type 2 diabetes and prostate cancer. Int J Mol Epidemiol Genet. 2013;4(1):49-60
Raynor, L. A., Pankow, J. S., Rasmussen-Torvik, L. J., Tang, W., Prizment, A., & Couper, D. J. (2013). Pleiotropy and pathway analyses of genetic variants associated with both type 2 diabetes and prostate cancer. International journal of molecular epidemiology and genetics, 4(1), 49-60.
Raynor, L A, et al. "Pleiotropy and pathway analyses of genetic variants associated with both type 2 diabetes and prostate cancer." International journal of molecular epidemiology and genetics vol. 4,1 (2013): 49-60.
Raynor LA, Pankow JS, Rasmussen-Torvik LJ, Tang W, Prizment A, Couper DJ. Pleiotropy and pathway analyses of genetic variants associated with both type 2 diabetes and prostate cancer. Int J Mol Epidemiol Genet. 2013;4(1):49-60. Epub 2013 Mar 18. PMID: 23565322; PMCID: PMC3612454.
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Galectin-3 and the incidence of abdominal aortic aneurysm: the atherosclerosis risk in communities (ARIC) study.

American journal of cardiovascular disease

Fashanu OE, Folsom AR, Oyenuga A, Ballantyne CM, Lutsey PL, Tang W.
PMID: 29348972
Am J Cardiovasc Dis. 2017 Dec 20;7(6):114-121. eCollection 2017.

Galectin-3, a β-galactosidase binding lectin, known to be involved in inflammatory processes may be associated with abdominal aortic aneurysm (AAA) incidence. We examined the prospective association between plasma galectin-3 and incident AAA in 9,704 participants of the Atherosclerosis Risk...

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Fashanu OE, Folsom AR, Oyenuga A, et al. Galectin-3 and the incidence of abdominal aortic aneurysm: the atherosclerosis risk in communities (ARIC) study. Am J Cardiovasc Dis. 2017;7(6):114-121
Fashanu, O. E., Folsom, A. R., Oyenuga, A., Ballantyne, C. M., Lutsey, P. L., & Tang, W. (2017). Galectin-3 and the incidence of abdominal aortic aneurysm: the atherosclerosis risk in communities (ARIC) study. American journal of cardiovascular disease, 7(6), 114-121.
Fashanu, Oluwaseun E, et al. "Galectin-3 and the incidence of abdominal aortic aneurysm: the atherosclerosis risk in communities (ARIC) study." American journal of cardiovascular disease vol. 7,6 (2017): 114-121.
Fashanu OE, Folsom AR, Oyenuga A, Ballantyne CM, Lutsey PL, Tang W. Galectin-3 and the incidence of abdominal aortic aneurysm: the atherosclerosis risk in communities (ARIC) study. Am J Cardiovasc Dis. 2017 Dec 20;7(6):114-121. eCollection 2017. PMID: 29348972; PMCID: PMC5768869.
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Postprandial lipemia and the risk of coronary heart disease and stroke: the Atherosclerosis Risk in Communities (ARIC) Study.

BMJ open diabetes research & care

Kats D, Sharrett AR, Ginsberg HN, Nambi V, Ballantyne CM, Hoogeveen RC, Heiss G.
PMID: 28191321
BMJ Open Diabetes Res Care. 2017 Feb 01;5(1):e000335. doi: 10.1136/bmjdrc-2016-000335. eCollection 2017.

OBJECTIVE: Excessive levels of triglyceride-rich lipoproteins during postprandial lipemia (PPL) have been reported to be atherogenic. However, it is unclear whether the degree of PPL independently predicts cardiovascular disease (CVD) given the scarcity of longitudinal data with standardised measures...

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Kats D, Sharrett AR, Ginsberg HN, et al. Postprandial lipemia and the risk of coronary heart disease and stroke: the Atherosclerosis Risk in Communities (ARIC) Study. BMJ Open Diabetes Res Care. 2017;5(1):e000335doi: 10.1136/bmjdrc-2016-000335.
Kats, D., Sharrett, A. R., Ginsberg, H. N., Nambi, V., Ballantyne, C. M., Hoogeveen, R. C., & Heiss, G. (2017). Postprandial lipemia and the risk of coronary heart disease and stroke: the Atherosclerosis Risk in Communities (ARIC) Study. BMJ open diabetes research & care, 5(1), e000335. https://doi.org/10.1136/bmjdrc-2016-000335
Kats, Dmitry, et al. "Postprandial lipemia and the risk of coronary heart disease and stroke: the Atherosclerosis Risk in Communities (ARIC) Study." BMJ open diabetes research & care vol. 5,1 (2017): e000335. doi: https://doi.org/10.1136/bmjdrc-2016-000335
Kats D, Sharrett AR, Ginsberg HN, Nambi V, Ballantyne CM, Hoogeveen RC, Heiss G. Postprandial lipemia and the risk of coronary heart disease and stroke: the Atherosclerosis Risk in Communities (ARIC) Study. BMJ Open Diabetes Res Care. 2017 Feb 01;5(1):e000335. doi: 10.1136/bmjdrc-2016-000335. eCollection 2017. PMID: 28191321; PMCID: PMC5293824.
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Double genomic control is not effective to correct for population stratification in meta-analysis for genome-wide association studies.

Frontiers in genetics

Wang S, Chen W, Chen X, Hu F, Archer KJ, Liu HN, Sun S, Gao G.
PMID: 23269928
Front Genet. 2012 Dec 24;3:300. doi: 10.3389/fgene.2012.00300. eCollection 2012.

Meta-analysis of genome-wide association studies (GWAS) has become a useful tool to identify genetic variants that are associated with complex human diseases. To control spurious associations between genetic variants and disease that are caused by population stratification, double genomic...

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Wang S, Chen W, Chen X, et al. Double genomic control is not effective to correct for population stratification in meta-analysis for genome-wide association studies. Front Genet. 2012;3:300doi: 10.3389/fgene.2012.00300.
Wang, S., Chen, W., Chen, X., Hu, F., Archer, K. J., Liu, H. N., Sun, S., & Gao, G. (2012). Double genomic control is not effective to correct for population stratification in meta-analysis for genome-wide association studies. Frontiers in genetics, 3300. https://doi.org/10.3389/fgene.2012.00300
Wang, Shudong, et al. "Double genomic control is not effective to correct for population stratification in meta-analysis for genome-wide association studies." Frontiers in genetics vol. 3 (2012): 300. doi: https://doi.org/10.3389/fgene.2012.00300
Wang S, Chen W, Chen X, Hu F, Archer KJ, Liu HN, Sun S, Gao G. Double genomic control is not effective to correct for population stratification in meta-analysis for genome-wide association studies. Front Genet. 2012 Dec 24;3:300. doi: 10.3389/fgene.2012.00300. eCollection 2012. PMID: 23269928; PMCID: PMC3529452.
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Whole-exome sequencing in 20,197 persons for rare variants in Alzheimer's disease.

Annals of clinical and translational neurology

Raghavan NS, Brickman AM, Andrews H, Manly JJ, Schupf N, Lantigua R, Wolock CJ, Kamalakaran S, Petrovski S, Tosto G, Vardarajan BN, Goldstein DB, Mayeux R.
PMID: 30009200
Ann Clin Transl Neurol. 2018 May 24;5(7):832-842. doi: 10.1002/acn3.582. eCollection 2018 Jul.

OBJECTIVE: The genetic bases of Alzheimer's disease remain uncertain. An international effort to fully articulate genetic risks and protective factors is underway with the hope of identifying potential therapeutic targets and preventive strategies. The goal here was to identify...

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Raghavan NS, Brickman AM, Andrews H, et al. Whole-exome sequencing in 20,197 persons for rare variants in Alzheimer's disease. Ann Clin Transl Neurol. 2018;5(7):832-842doi: 10.1002/acn3.582.
Raghavan, N. S., Brickman, A. M., Andrews, H., Manly, J. J., Schupf, N., Lantigua, R., Wolock, C. J., Kamalakaran, S., Petrovski, S., Tosto, G., Vardarajan, B. N., Goldstein, D. B., Mayeux, R. (2018). Whole-exome sequencing in 20,197 persons for rare variants in Alzheimer's disease. Annals of clinical and translational neurology, 5(7), 832-842. https://doi.org/10.1002/acn3.582
Raghavan, Neha S, et al. "Whole-exome sequencing in 20,197 persons for rare variants in Alzheimer's disease." Annals of clinical and translational neurology vol. 5,7 (2018): 832-842. doi: https://doi.org/10.1002/acn3.582
Raghavan NS, Brickman AM, Andrews H, Manly JJ, Schupf N, Lantigua R, Wolock CJ, Kamalakaran S, Petrovski S, Tosto G, Vardarajan BN, Goldstein DB, Mayeux R. Whole-exome sequencing in 20,197 persons for rare variants in Alzheimer's disease. Ann Clin Transl Neurol. 2018 May 24;5(7):832-842. doi: 10.1002/acn3.582. eCollection 2018 Jul. PMID: 30009200; PMCID: PMC6043775.
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GxEsum: a novel approach to estimate the phenotypic variance explained by genome-wide GxE interaction based on GWAS summary statistics for biobank-scale data.

Genome biology

Shin J, Lee SH.
PMID: 34154633
Genome Biol. 2021 Jun 21;22(1):183. doi: 10.1186/s13059-021-02403-1.

Genetic variation in response to the environment, that is, genotype-by-environment interaction (GxE), is fundamental in the biology of complex traits and diseases. However, existing methods are computationally demanding and infeasible to handle biobank-scale data. Here, we introduce GxEsum, a...

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Shin J, Lee SH. GxEsum: a novel approach to estimate the phenotypic variance explained by genome-wide GxE interaction based on GWAS summary statistics for biobank-scale data. Genome Biol. 2021;22(1):183doi: 10.1186/s13059-021-02403-1.
Shin, J., & Lee, S. H. (2021). GxEsum: a novel approach to estimate the phenotypic variance explained by genome-wide GxE interaction based on GWAS summary statistics for biobank-scale data. Genome biology, 22(1), 183. https://doi.org/10.1186/s13059-021-02403-1
Shin, Jisu, and Lee, Sang Hong. "GxEsum: a novel approach to estimate the phenotypic variance explained by genome-wide GxE interaction based on GWAS summary statistics for biobank-scale data." Genome biology vol. 22,1 (2021): 183. doi: https://doi.org/10.1186/s13059-021-02403-1
Shin J, Lee SH. GxEsum: a novel approach to estimate the phenotypic variance explained by genome-wide GxE interaction based on GWAS summary statistics for biobank-scale data. Genome Biol. 2021 Jun 21;22(1):183. doi: 10.1186/s13059-021-02403-1. PMID: 34154633; PMCID: PMC8218431.
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Genetic risk for major depressive disorder and loneliness in sex-specific associations with coronary artery disease.

Molecular psychiatry

Dennis J, Sealock J, Levinson RT, Farber-Eger E, Franco J, Fong S, Straub P, Hucks D, Song WL, Linton MF, Fontanillas P, Elson SL, Ruderfer D, Abdellaoui A, Sanchez-Roige S, Palmer AA, Boomsma DI, Cox NJ, Chen G, Mosley JD, Wells QS, Davis LK.
PMID: 31796895
Mol Psychiatry. 2021 Aug;26(8):4254-4264. doi: 10.1038/s41380-019-0614-y. Epub 2019 Dec 03.

Major depressive disorder (MDD) and loneliness are phenotypically and genetically correlated with coronary artery disease (CAD), but whether these associations are explained by pleiotropic genetic variants or shared comorbidities is unclear. To tease apart these scenarios, we first assessed...

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Dennis J, Sealock J, Levinson RT, et al. Genetic risk for major depressive disorder and loneliness in sex-specific associations with coronary artery disease. Mol Psychiatry. 2019;26(8):4254-4264doi: 10.1038/s41380-019-0614-y.
Dennis, J., Sealock, J., Levinson, R. T., Farber-Eger, E., Franco, J., Fong, S., Straub, P., Hucks, D., Song, W. L., Linton, M. F., Fontanillas, P., Elson, S. L., Ruderfer, D., Abdellaoui, A., Sanchez-Roige, S., Palmer, A. A., Boomsma, D. I., Cox, N. J., Chen, G., Mosley, J. D., Wells, Q. S., & Davis, L. K. (2021). Genetic risk for major depressive disorder and loneliness in sex-specific associations with coronary artery disease. Molecular psychiatry, 26(8), 4254-4264. https://doi.org/10.1038/s41380-019-0614-y
Dennis, Jessica, et al. "Genetic risk for major depressive disorder and loneliness in sex-specific associations with coronary artery disease." Molecular psychiatry vol. 26,8 (2021): 4254-4264. doi: https://doi.org/10.1038/s41380-019-0614-y
Dennis J, Sealock J, Levinson RT, Farber-Eger E, Franco J, Fong S, Straub P, Hucks D, Song WL, Linton MF, Fontanillas P, Elson SL, Ruderfer D, Abdellaoui A, Sanchez-Roige S, Palmer AA, Boomsma DI, Cox NJ, Chen G, Mosley JD, Wells QS, Davis LK. Genetic risk for major depressive disorder and loneliness in sex-specific associations with coronary artery disease. Mol Psychiatry. 2021 Aug;26(8):4254-4264. doi: 10.1038/s41380-019-0614-y. Epub 2019 Dec 03. PMID: 31796895; PMCID: PMC7266730.
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Association of SERPINA9 gene variants with carotid artery atherosclerosis: the Atherosclerosis Risk in Communities (ARIC) Carotid MRI Study.

International journal of molecular epidemiology and genetics

Tang W, Morrison A, Wasserman BA, Folsom AR, Sun W, Campbell S, Kao WH, Boerwinkle E.
PMID: 24319541
Int J Mol Epidemiol Genet. 2013 Nov 28;4(4):258-67. eCollection 2013.

The SNP rs11628722 in the SERPINA9 gene was previously associated with incident ischemic stroke in the Atherosclerosis Risk in Communities (ARIC) study. Centerin, the protein encoded by SERPINA9, is involved in maturation and maintenance of naïve B cells, which...

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Tang W, Morrison A, Wasserman BA, et al. Association of SERPINA9 gene variants with carotid artery atherosclerosis: the Atherosclerosis Risk in Communities (ARIC) Carotid MRI Study. Int J Mol Epidemiol Genet. 2013;4(4):258-67
Tang, W., Morrison, A., Wasserman, B. A., Folsom, A. R., Sun, W., Campbell, S., Kao, W. H., & Boerwinkle, E. (2013). Association of SERPINA9 gene variants with carotid artery atherosclerosis: the Atherosclerosis Risk in Communities (ARIC) Carotid MRI Study. International journal of molecular epidemiology and genetics, 4(4), 258-67.
Tang, Weihong, et al. "Association of SERPINA9 gene variants with carotid artery atherosclerosis: the Atherosclerosis Risk in Communities (ARIC) Carotid MRI Study." International journal of molecular epidemiology and genetics vol. 4,4 (2013): 258-67.
Tang W, Morrison A, Wasserman BA, Folsom AR, Sun W, Campbell S, Kao WH, Boerwinkle E. Association of SERPINA9 gene variants with carotid artery atherosclerosis: the Atherosclerosis Risk in Communities (ARIC) Carotid MRI Study. Int J Mol Epidemiol Genet. 2013 Nov 28;4(4):258-67. eCollection 2013. PMID: 24319541; PMCID: PMC3852645.
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A causal network analysis in an observational study identifies metabolomics pathways influencing plasma triglyceride levels.

Metabolomics : Official journal of the Metabolomic Society

Yazdani A, Yazdani A, Saniei A, Boerwinkle E.
PMID: 27330524
Metabolomics. 2016;12:104. doi: 10.1007/s11306-016-1045-2. Epub 2016 May 11.

INTRODUCTION: Plasma triglyceride levels are a risk factor for coronary heart disease. Triglyceride metabolism is well characterized, but challenges remain to identify novel paths to lower levels. A metabolomics analysis may help identify such novel pathways and, therefore, provide...

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Yazdani A, Yazdani A, Saniei A, et al. A causal network analysis in an observational study identifies metabolomics pathways influencing plasma triglyceride levels. Metabolomics. 2016;12:104doi: 10.1007/s11306-016-1045-2.
Yazdani, A., Yazdani, A., Saniei, A., & Boerwinkle, E. (2016). A causal network analysis in an observational study identifies metabolomics pathways influencing plasma triglyceride levels. Metabolomics : Official journal of the Metabolomic Society, 12104. https://doi.org/10.1007/s11306-016-1045-2
Yazdani, Azam, et al. "A causal network analysis in an observational study identifies metabolomics pathways influencing plasma triglyceride levels." Metabolomics : Official journal of the Metabolomic Society vol. 12 (2016): 104. doi: https://doi.org/10.1007/s11306-016-1045-2
Yazdani A, Yazdani A, Saniei A, Boerwinkle E. A causal network analysis in an observational study identifies metabolomics pathways influencing plasma triglyceride levels. Metabolomics. 2016;12:104. doi: 10.1007/s11306-016-1045-2. Epub 2016 May 11. PMID: 27330524; PMCID: PMC4869741.
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