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Metabolism and growth of juveniles of Litopenaeus vannamei: effect of salinity and dietary carbohydrate levels.

Journal of experimental marine biology and ecology

Rosas C, Cuzon G, Gaxiola G, Le Priol Y, Pascual C, Rossignyol J, Contreras F, Sanchez A, Van Wormhoudt A.
PMID: 11325374
J Exp Mar Biol Ecol. 2001 Apr 30;259(1):1-22. doi: 10.1016/s0022-0981(01)00222-2.

The present study was designed to understand how carbohydrate (CBH) and protein metabolism are related in the penaeid shrimp Litopenaeus vannamei. With this information, we obtained a comprehensive schedule of the protein-carbohydrate metabolism including enzymatic, energetic, and functional aspects....

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Rosas C, Cuzon G, Gaxiola G, et al. Metabolism and growth of juveniles of Litopenaeus vannamei: effect of salinity and dietary carbohydrate levels. J Exp Mar Biol Ecol. 2001;259(1):1-22doi: 10.1016/s0022-0981(01)00222-2.
Rosas, C., Cuzon, G., Gaxiola, G., Le Priol, Y., Pascual, C., Rossignyol, J., Contreras, F., Sanchez, A., & Van Wormhoudt, A. (2001). Metabolism and growth of juveniles of Litopenaeus vannamei: effect of salinity and dietary carbohydrate levels. Journal of experimental marine biology and ecology, 259(1), 1-22. https://doi.org/10.1016/s0022-0981(01)00222-2
Rosas, C, et al. "Metabolism and growth of juveniles of Litopenaeus vannamei: effect of salinity and dietary carbohydrate levels." Journal of experimental marine biology and ecology vol. 259,1 (2001): 1-22. doi: https://doi.org/10.1016/s0022-0981(01)00222-2
Rosas C, Cuzon G, Gaxiola G, Le Priol Y, Pascual C, Rossignyol J, Contreras F, Sanchez A, Van Wormhoudt A. Metabolism and growth of juveniles of Litopenaeus vannamei: effect of salinity and dietary carbohydrate levels. J Exp Mar Biol Ecol. 2001 Apr 30;259(1):1-22. doi: 10.1016/s0022-0981(01)00222-2. PMID: 11325374.
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Metabolic flux sampling predicts strain-dependent differences related to aroma production among commercial wine yeasts.

Microbial cell factories

Scott WT, Smid EJ, Block DE, Notebaart RA.
PMID: 34674718
Microb Cell Fact. 2021 Oct 21;20(1):204. doi: 10.1186/s12934-021-01694-0.

BACKGROUND: Metabolomics coupled with genome-scale metabolic modeling approaches have been employed recently to quantitatively analyze the physiological states of various organisms, including Saccharomyces cerevisiae. Although yeast physiology in laboratory strains is well-studied, the metabolic states under industrially relevant scenarios...

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Scott WT, Smid EJ, Block DE, et al. Metabolic flux sampling predicts strain-dependent differences related to aroma production among commercial wine yeasts. Microb Cell Fact. 2021;20(1):204doi: 10.1186/s12934-021-01694-0.
Scott, W. T., Smid, E. J., Block, D. E., & Notebaart, R. A. (2021). Metabolic flux sampling predicts strain-dependent differences related to aroma production among commercial wine yeasts. Microbial cell factories, 20(1), 204. https://doi.org/10.1186/s12934-021-01694-0
Scott, William T, et al. "Metabolic flux sampling predicts strain-dependent differences related to aroma production among commercial wine yeasts." Microbial cell factories vol. 20,1 (2021): 204. doi: https://doi.org/10.1186/s12934-021-01694-0
Scott WT, Smid EJ, Block DE, Notebaart RA. Metabolic flux sampling predicts strain-dependent differences related to aroma production among commercial wine yeasts. Microb Cell Fact. 2021 Oct 21;20(1):204. doi: 10.1186/s12934-021-01694-0. PMID: 34674718; PMCID: PMC8532357.
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Artocarpus communis Forst. root-bark aqueous extract- and streptozotocin-induced ultrastructural and metabolic changes in hepatic tissues of Wistar rats.

African journal of traditional, complementary, and alternative medicines : AJTCAM

Adewole SO, Ojewole JA.
PMID: 20161908
Afr J Tradit Complement Altern Med. 2007 Jun 10;4(4):397-410.

Decoctions and infusions of Artocarpus communis (Forst.) (family: Moraceae) root-bark are commonly used traditionally among the Yoruba-speaking people of Western Nigeria as folk remedies for the management, control and/or treatment of an array of human diseases, including type 2,...

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Adewole SO, Ojewole JA. Artocarpus communis Forst. root-bark aqueous extract- and streptozotocin-induced ultrastructural and metabolic changes in hepatic tissues of Wistar rats. Afr J Tradit Complement Altern Med. 2007;4(4):397-410
Adewole, S. O., & Ojewole, J. A. (2007). Artocarpus communis Forst. root-bark aqueous extract- and streptozotocin-induced ultrastructural and metabolic changes in hepatic tissues of Wistar rats. African journal of traditional, complementary, and alternative medicines : AJTCAM, 4(4), 397-410.
Adewole, Stephen O, and Ojewole, John A O. "Artocarpus communis Forst. root-bark aqueous extract- and streptozotocin-induced ultrastructural and metabolic changes in hepatic tissues of Wistar rats." African journal of traditional, complementary, and alternative medicines : AJTCAM vol. 4,4 (2007): 397-410.
Adewole SO, Ojewole JA. Artocarpus communis Forst. root-bark aqueous extract- and streptozotocin-induced ultrastructural and metabolic changes in hepatic tissues of Wistar rats. Afr J Tradit Complement Altern Med. 2007 Jun 10;4(4):397-410. PMID: 20161908; PMCID: PMC2816511.
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Long Non-Coding RNA FEZF1-AS1 Modulates CXCR4 to Promote Cell Proliferation, Warburg Effect and Suppress Cell Apoptosis in Osteosarcoma by Sponging miR-144.

OncoTargets and therapy

Liu J, Feng G, Li Z, Li R, Xia P.
PMID: 32308422
Onco Targets Ther. 2020 Apr 05;13:2899-2910. doi: 10.2147/OTT.S235970. eCollection 2020.

BACKGROUND: Osteosarcoma (OS) is a common bone tumor among children, adolescents, and young adults. Long non-coding RNA (lncRNA) FEZF1 antisense RNA 1 (FEZF1-AS1) has been reported as an oncogene in diverse tumors including colorectal cancer, pancreatic cancer and hepatocellular...

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Liu J, Feng G, Li Z, et al. Long Non-Coding RNA FEZF1-AS1 Modulates CXCR4 to Promote Cell Proliferation, Warburg Effect and Suppress Cell Apoptosis in Osteosarcoma by Sponging miR-144. Onco Targets Ther. 2020;13:2899-2910doi: 10.2147/OTT.S235970.
Liu, J., Feng, G., Li, Z., Li, R., & Xia, P. (2020). Long Non-Coding RNA FEZF1-AS1 Modulates CXCR4 to Promote Cell Proliferation, Warburg Effect and Suppress Cell Apoptosis in Osteosarcoma by Sponging miR-144. OncoTargets and therapy, 132899-2910. https://doi.org/10.2147/OTT.S235970
Liu, Jun, et al. "Long Non-Coding RNA FEZF1-AS1 Modulates CXCR4 to Promote Cell Proliferation, Warburg Effect and Suppress Cell Apoptosis in Osteosarcoma by Sponging miR-144." OncoTargets and therapy vol. 13 (2020): 2899-2910. doi: https://doi.org/10.2147/OTT.S235970
Liu J, Feng G, Li Z, Li R, Xia P. Long Non-Coding RNA FEZF1-AS1 Modulates CXCR4 to Promote Cell Proliferation, Warburg Effect and Suppress Cell Apoptosis in Osteosarcoma by Sponging miR-144. Onco Targets Ther. 2020 Apr 05;13:2899-2910. doi: 10.2147/OTT.S235970. eCollection 2020. PMID: 32308422; PMCID: PMC7147627.
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Expression of Concern: Sirtuin-3 deacetylation of cyclophilin D induces dissociation of hexokinase II from the mitochondria. Nataly Shulga, Robin Wilson-Smith, John G. Pastorino. J Cell Sci doi: 10.1242/jcs.061846.

Journal of cell science

Way M.
PMID: 26905965
J Cell Sci. 2016 Feb 15;129(4):868. doi: 10.1242/jcs.186999.

No abstract available.

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Way M. Expression of Concern: Sirtuin-3 deacetylation of cyclophilin D induces dissociation of hexokinase II from the mitochondria. Nataly Shulga, Robin Wilson-Smith, John G. Pastorino. J Cell Sci doi: 10.1242/jcs.061846. J Cell Sci. 2016;129(4):868doi: 10.1242/jcs.186999.
Way, M. (2016). Expression of Concern: Sirtuin-3 deacetylation of cyclophilin D induces dissociation of hexokinase II from the mitochondria. Nataly Shulga, Robin Wilson-Smith, John G. Pastorino. J Cell Sci doi: 10.1242/jcs.061846. Journal of cell science, 129(4), 868. https://doi.org/10.1242/jcs.186999
Way, Michael. "Expression of Concern: Sirtuin-3 deacetylation of cyclophilin D induces dissociation of hexokinase II from the mitochondria. Nataly Shulga, Robin Wilson-Smith, John G. Pastorino. J Cell Sci doi: 10.1242/jcs.061846." Journal of cell science vol. 129,4 (2016): 868. doi: https://doi.org/10.1242/jcs.186999
Way M. Expression of Concern: Sirtuin-3 deacetylation of cyclophilin D induces dissociation of hexokinase II from the mitochondria. Nataly Shulga, Robin Wilson-Smith, John G. Pastorino. J Cell Sci doi: 10.1242/jcs.061846. J Cell Sci. 2016 Feb 15;129(4):868. doi: 10.1242/jcs.186999. PMID: 26905965; PMCID: PMC4916905.
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FDG-PET is a good biomarker of both early response and acquired resistance in BRAFV600 mutant melanomas treated with vemurafenib and the MEK inhibitor GDC-0973.

EJNMMI research

Baudy AR, Dogan T, Flores-Mercado JE, Hoeflich KP, Su F, van Bruggen N, Williams SP.
PMID: 22651703
EJNMMI Res. 2012 May 31;2(1):22. doi: 10.1186/2191-219X-2-22.

BACKGROUND: The BRAF inhibitor, vemurafenib, has recently been approved for the treatment of metastatic melanoma in patients harboring BRAFV600 mutations. Currently, dual BRAF and MEK inhibition are ongoing in clinical trials with the goal of overcoming the acquired resistance...

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Baudy AR, Dogan T, Flores-Mercado JE, et al. FDG-PET is a good biomarker of both early response and acquired resistance in BRAFV600 mutant melanomas treated with vemurafenib and the MEK inhibitor GDC-0973. EJNMMI Res. 2012;2(1):22doi: 10.1186/2191-219X-2-22.
Baudy, A. R., Dogan, T., Flores-Mercado, J. E., Hoeflich, K. P., Su, F., van Bruggen, N., & Williams, S. P. (2012). FDG-PET is a good biomarker of both early response and acquired resistance in BRAFV600 mutant melanomas treated with vemurafenib and the MEK inhibitor GDC-0973. EJNMMI research, 2(1), 22. https://doi.org/10.1186/2191-219X-2-22
Baudy, Andreas R, et al. "FDG-PET is a good biomarker of both early response and acquired resistance in BRAFV600 mutant melanomas treated with vemurafenib and the MEK inhibitor GDC-0973." EJNMMI research vol. 2,1 (2012): 22. doi: https://doi.org/10.1186/2191-219X-2-22
Baudy AR, Dogan T, Flores-Mercado JE, Hoeflich KP, Su F, van Bruggen N, Williams SP. FDG-PET is a good biomarker of both early response and acquired resistance in BRAFV600 mutant melanomas treated with vemurafenib and the MEK inhibitor GDC-0973. EJNMMI Res. 2012 May 31;2(1):22. doi: 10.1186/2191-219X-2-22. PMID: 22651703; PMCID: PMC3405466.
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Reversal of insulin resistance by Ficus benghalensis bark in fructose-induced insulin-resistant rats.

Journal of ethnopharmacology

Khanal P, Patil BM.
PMID: 34678414
J Ethnopharmacol. 2022 Feb 10;284:114761. doi: 10.1016/j.jep.2021.114761. Epub 2021 Oct 20.

ETHNOPHARMACOLOGICAL RELEVANCE: Bark of Ficus benghalensis L. (family: Moraceae), commonly known as Banyan is recorded as Nyagrodha in Ayurvedic Pharmacopeia of India to manage burning sensation, obesity, diabetes, bleeding disorders, thirst, skin diseases, wounds, and dysmenorrhoea. However, the effect...

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Khanal P, Patil BM. Reversal of insulin resistance by Ficus benghalensis bark in fructose-induced insulin-resistant rats. J Ethnopharmacol. 2021;284:114761doi: 10.1016/j.jep.2021.114761.
Khanal, P., & Patil, B. M. (2022). Reversal of insulin resistance by Ficus benghalensis bark in fructose-induced insulin-resistant rats. Journal of ethnopharmacology, 284114761. https://doi.org/10.1016/j.jep.2021.114761
Khanal, Pukar, and Patil, B M. "Reversal of insulin resistance by Ficus benghalensis bark in fructose-induced insulin-resistant rats." Journal of ethnopharmacology vol. 284 (2022): 114761. doi: https://doi.org/10.1016/j.jep.2021.114761
Khanal P, Patil BM. Reversal of insulin resistance by Ficus benghalensis bark in fructose-induced insulin-resistant rats. J Ethnopharmacol. 2022 Feb 10;284:114761. doi: 10.1016/j.jep.2021.114761. Epub 2021 Oct 20. PMID: 34678414.
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[Enzyme activities and substrate levels of carbohydrate metabolism in proliferating and suberin synthesizing potato tuber cells].

Planta

Lange H, Kahl G, Rosenstock G.
PMID: 24499978
Planta. 1970 Mar;91(1):18-31. doi: 10.1007/BF00390162.

Isolation of tissue fragments from the potato tuber can initiate either periderm formation including suberin synthesis or cell proliferation without cicatrization effects. TCA-cycle activity has been shown to develop only in causal correlation with suberin synthesis (Lange, 1970). Biochemical...

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Lange H, Kahl G, Rosenstock G. Enzymaktivitäten und Intermediatspiegel des Glucosekatabolismus bei proliferierenden und Suberin synthetisierenden Speicherparenchymzellen von Solanum tuberosum L. [Enzyme activities and substrate levels of carbohydrate metabolism in proliferating and suberin synthesizing potato tuber cells]. Planta. 1970;91(1):18-31doi: 10.1007/BF00390162.
Lange, H., Kahl, G., & Rosenstock, G. (1970). Enzymaktivitäten und Intermediatspiegel des Glucosekatabolismus bei proliferierenden und Suberin synthetisierenden Speicherparenchymzellen von Solanum tuberosum L. [Enzyme activities and substrate levels of carbohydrate metabolism in proliferating and suberin synthesizing potato tuber cells]. Planta, 91(1), 18-31. https://doi.org/10.1007/BF00390162
Lange, H, et al. "Enzymaktivitäten und Intermediatspiegel des Glucosekatabolismus bei proliferierenden und Suberin synthetisierenden Speicherparenchymzellen von Solanum tuberosum L." [Enzyme activities and substrate levels of carbohydrate metabolism in proliferating and suberin synthesizing potato tuber cells]. Planta vol. 91,1 (1970): 18-31. doi: https://doi.org/10.1007/BF00390162
Lange H, Kahl G, Rosenstock G. Enzymaktivitäten und Intermediatspiegel des Glucosekatabolismus bei proliferierenden und Suberin synthetisierenden Speicherparenchymzellen von Solanum tuberosum L. [Enzyme activities and substrate levels of carbohydrate metabolism in proliferating and suberin synthesizing potato tuber cells]. Planta. 1970 Mar;91(1):18-31. doi: 10.1007/BF00390162. German. PMID: 24499978.
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1,25-Dihydroxyvitamin D3 modulates calcium transport in goat mammary epithelial cells in a dose- and energy-dependent manner.

Journal of animal science and biotechnology

Sun F, Cao Y, Yu C, Wei X, Yao J.
PMID: 27471592
J Anim Sci Biotechnol. 2016 Jul 28;7:41. doi: 10.1186/s40104-016-0101-0. eCollection 2016.

BACKGROUND: Calcium is a vital mineral and an indispensable component of milk for ruminants. The regulation of transcellular calcium transport by 1,25-dihydroxyvitamin D3 (1,25-(OH)2D3, the active form of vitamin D) has been confirmed in humans and rodents, and regulators,...

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Sun F, Cao Y, Yu C, et al. 1,25-Dihydroxyvitamin D3 modulates calcium transport in goat mammary epithelial cells in a dose- and energy-dependent manner. J Anim Sci Biotechnol. 2016;7:41doi: 10.1186/s40104-016-0101-0.
Sun, F., Cao, Y., Yu, C., Wei, X., & Yao, J. (2016). 1,25-Dihydroxyvitamin D3 modulates calcium transport in goat mammary epithelial cells in a dose- and energy-dependent manner. Journal of animal science and biotechnology, 741. https://doi.org/10.1186/s40104-016-0101-0
Sun, Feifei, et al. "1,25-Dihydroxyvitamin D3 modulates calcium transport in goat mammary epithelial cells in a dose- and energy-dependent manner." Journal of animal science and biotechnology vol. 7 (2016): 41. doi: https://doi.org/10.1186/s40104-016-0101-0
Sun F, Cao Y, Yu C, Wei X, Yao J. 1,25-Dihydroxyvitamin D3 modulates calcium transport in goat mammary epithelial cells in a dose- and energy-dependent manner. J Anim Sci Biotechnol. 2016 Jul 28;7:41. doi: 10.1186/s40104-016-0101-0. eCollection 2016. PMID: 27471592; PMCID: PMC4964070.
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Protein synthesis inhibitors stimulate MondoA transcriptional activity by driving an accumulation of glucose 6-phosphate.

Cancer & metabolism

Wilde BR, Kaadige MR, Guillen KP, Butterfield A, Welm BE, Ayer DE.
PMID: 33292639
Cancer Metab. 2020 Dec 04;8(1):27. doi: 10.1186/s40170-020-00233-6.

BACKGROUND: Protein synthesis is regulated by the availability of amino acids, the engagement of growth factor signaling pathways, and adenosine triphosphate (ATP) levels sufficient to support translation. Crosstalk between these inputs is extensive, yet other regulatory mechanisms remain to...

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Wilde BR, Kaadige MR, Guillen KP, et al. Protein synthesis inhibitors stimulate MondoA transcriptional activity by driving an accumulation of glucose 6-phosphate. Cancer Metab. 2020;8(1):27doi: 10.1186/s40170-020-00233-6.
Wilde, B. R., Kaadige, M. R., Guillen, K. P., Butterfield, A., Welm, B. E., & Ayer, D. E. (2020). Protein synthesis inhibitors stimulate MondoA transcriptional activity by driving an accumulation of glucose 6-phosphate. Cancer & metabolism, 8(1), 27. https://doi.org/10.1186/s40170-020-00233-6
Wilde, Blake R, et al. "Protein synthesis inhibitors stimulate MondoA transcriptional activity by driving an accumulation of glucose 6-phosphate." Cancer & metabolism vol. 8,1 (2020): 27. doi: https://doi.org/10.1186/s40170-020-00233-6
Wilde BR, Kaadige MR, Guillen KP, Butterfield A, Welm BE, Ayer DE. Protein synthesis inhibitors stimulate MondoA transcriptional activity by driving an accumulation of glucose 6-phosphate. Cancer Metab. 2020 Dec 04;8(1):27. doi: 10.1186/s40170-020-00233-6. PMID: 33292639; PMCID: PMC7718662.
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MitomiRs: their roles in mitochondria and importance in cancer cell metabolism.

Radiology and oncology

Rencelj A, Gvozdenovic N, Cemazar M.
PMID: 34821131
Radiol Oncol. 2021 Nov 19;55(4):379-392. doi: 10.2478/raon-2021-0042.

BACKGROUND: MicroRNAs (miRNAs) are short non-coding RNAs that play important roles in almost all biological pathways. They regulate post-transcriptional gene expression by binding to the 3'untranslated region (3'UTR) of messenger RNAs (mRNAs). MitomiRs are miRNAs of nuclear or mitochondrial...

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Rencelj A, Gvozdenovic N, Cemazar M. MitomiRs: their roles in mitochondria and importance in cancer cell metabolism. Radiol Oncol. 2021;55(4):379-392doi: 10.2478/raon-2021-0042.
Rencelj, A., Gvozdenovic, N., & Cemazar, M. (2021). MitomiRs: their roles in mitochondria and importance in cancer cell metabolism. Radiology and oncology, 55(4), 379-392. https://doi.org/10.2478/raon-2021-0042
Rencelj, Andrej, et al. "MitomiRs: their roles in mitochondria and importance in cancer cell metabolism." Radiology and oncology vol. 55,4 (2021): 379-392. doi: https://doi.org/10.2478/raon-2021-0042
Rencelj A, Gvozdenovic N, Cemazar M. MitomiRs: their roles in mitochondria and importance in cancer cell metabolism. Radiol Oncol. 2021 Nov 19;55(4):379-392. doi: 10.2478/raon-2021-0042. PMID: 34821131.
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Early glycolytic reprogramming controls microglial inflammatory activation.

Journal of neuroinflammation

Cheng J, Zhang R, Xu Z, Ke Y, Sun R, Yang H, Zhang X, Zhen X, Zheng LT.
PMID: 34107997
J Neuroinflammation. 2021 Jun 09;18(1):129. doi: 10.1186/s12974-021-02187-y.

BACKGROUND: Microglial activation-mediated neuroinflammation plays an important role in the progression of neurodegenerative diseases. Inflammatory activation of microglial cells is often accompanied by a metabolic switch from oxidative phosphorylation to aerobic glycolysis. However, the roles and molecular mechanisms of...

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Cheng J, Zhang R, Xu Z, et al. Early glycolytic reprogramming controls microglial inflammatory activation. J Neuroinflammation. 2021;18(1):129doi: 10.1186/s12974-021-02187-y.
Cheng, J., Zhang, R., Xu, Z., Ke, Y., Sun, R., Yang, H., Zhang, X., Zhen, X., & Zheng, L. T. (2021). Early glycolytic reprogramming controls microglial inflammatory activation. Journal of neuroinflammation, 18(1), 129. https://doi.org/10.1186/s12974-021-02187-y
Cheng, Junjie, et al. "Early glycolytic reprogramming controls microglial inflammatory activation." Journal of neuroinflammation vol. 18,1 (2021): 129. doi: https://doi.org/10.1186/s12974-021-02187-y
Cheng J, Zhang R, Xu Z, Ke Y, Sun R, Yang H, Zhang X, Zhen X, Zheng LT. Early glycolytic reprogramming controls microglial inflammatory activation. J Neuroinflammation. 2021 Jun 09;18(1):129. doi: 10.1186/s12974-021-02187-y. PMID: 34107997; PMCID: PMC8191212.
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