Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 32 entries
Sorted by: Best Match Show Resources per page
Proinflammatory high-density lipoprotein results from oxidized lipid mediators in the pathogenesis of both idiopathic and associated types of pulmonary arterial hypertension.

Pulmonary circulation

Ross DJ, Hough G, Hama S, Aboulhosn J, Belperio JA, Saggar R, Van Lenten BJ, Ardehali A, Eghbali M, Reddy S, Fogelman AM, Navab M.
PMID: 26697171
Pulm Circ. 2015 Dec;5(4):640-8. doi: 10.1086/683695.

Pulmonary arterial hypertension (PAH) is characterized by abnormal elaboration of vasoactive peptides, endothelial cell dysfunction, vascular remodeling, and inflammation, which collectively contribute to its pathogenesis. We investigated the potential for high-density lipoprotein (HDL) dysfunction (i.e., proinflammatory effects) and abnormal...

Cite
Ross DJ, Hough G, Hama S, et al. Proinflammatory high-density lipoprotein results from oxidized lipid mediators in the pathogenesis of both idiopathic and associated types of pulmonary arterial hypertension. Pulm Circ. 2015;5(4):640-8doi: 10.1086/683695.
Ross, D. J., Hough, G., Hama, S., Aboulhosn, J., Belperio, J. A., Saggar, R., Van Lenten, B. J., Ardehali, A., Eghbali, M., Reddy, S., Fogelman, A. M., & Navab, M. (2015). Proinflammatory high-density lipoprotein results from oxidized lipid mediators in the pathogenesis of both idiopathic and associated types of pulmonary arterial hypertension. Pulmonary circulation, 5(4), 640-8. https://doi.org/10.1086/683695
Ross, David J, et al. "Proinflammatory high-density lipoprotein results from oxidized lipid mediators in the pathogenesis of both idiopathic and associated types of pulmonary arterial hypertension." Pulmonary circulation vol. 5,4 (2015): 640-8. doi: https://doi.org/10.1086/683695
Ross DJ, Hough G, Hama S, Aboulhosn J, Belperio JA, Saggar R, Van Lenten BJ, Ardehali A, Eghbali M, Reddy S, Fogelman AM, Navab M. Proinflammatory high-density lipoprotein results from oxidized lipid mediators in the pathogenesis of both idiopathic and associated types of pulmonary arterial hypertension. Pulm Circ. 2015 Dec;5(4):640-8. doi: 10.1086/683695. PMID: 26697171; PMCID: PMC4671738.
Copy Download .nbib Format: NLM
Expression and Function of Eicosanoid-Producing Cytochrome P450 Enzymes in Solid Tumors.

Frontiers in pharmacology

Evangelista EA, Cho CW, Aliwarga T, Totah RA.
PMID: 32581794
Front Pharmacol. 2020 Jun 09;11:828. doi: 10.3389/fphar.2020.00828. eCollection 2020.

Oxylipins derived from the oxidation of polyunsaturated fatty acids (PUFAs) act as important paracrine and autocrine signaling molecules. A subclass of oxylipins, the eicosanoids, have a broad range of physiological outcomes in inflammation, the immune response, cardiovascular homeostasis, and...

Cite
Evangelista EA, Cho CW, Aliwarga T, et al. Expression and Function of Eicosanoid-Producing Cytochrome P450 Enzymes in Solid Tumors. Front Pharmacol. 2020;11:828doi: 10.3389/fphar.2020.00828.
Evangelista, E. A., Cho, C. W., Aliwarga, T., & Totah, R. A. (2020). Expression and Function of Eicosanoid-Producing Cytochrome P450 Enzymes in Solid Tumors. Frontiers in pharmacology, 11828. https://doi.org/10.3389/fphar.2020.00828
Evangelista, Eric A, et al. "Expression and Function of Eicosanoid-Producing Cytochrome P450 Enzymes in Solid Tumors." Frontiers in pharmacology vol. 11 (2020): 828. doi: https://doi.org/10.3389/fphar.2020.00828
Evangelista EA, Cho CW, Aliwarga T, Totah RA. Expression and Function of Eicosanoid-Producing Cytochrome P450 Enzymes in Solid Tumors. Front Pharmacol. 2020 Jun 09;11:828. doi: 10.3389/fphar.2020.00828. eCollection 2020. PMID: 32581794; PMCID: PMC7295938.
Copy Download .nbib Format: NLM
Hydroxyeicosatetraenoic acids in patients with pancreatic cancer: a preliminary report.

American journal of cancer research

Bodnarczuk T, Deskur A, Dolegowska K, Dolegowska B, Starzynska T, Blogowski W.
PMID: 30323978
Am J Cancer Res. 2018 Sep 01;8(9):1865-1872. eCollection 2018.

Previous experimental reports have demonstrated that lipoxygenase (LOX) derivatives of arachidonic acid (AA), such as hydroxyeicosatetraenoic acids (HETEs), may be of significance in the pathogenesis of pancreatic cancer. However, these observations have not been confirmed in clinical studies. In...

Cite
Bodnarczuk T, Deskur A, Dolegowska K, et al. Hydroxyeicosatetraenoic acids in patients with pancreatic cancer: a preliminary report. Am J Cancer Res. 2018;8(9):1865-1872
Bodnarczuk, T., Deskur, A., Dolegowska, K., Dolegowska, B., Starzynska, T., & Blogowski, W. (2018). Hydroxyeicosatetraenoic acids in patients with pancreatic cancer: a preliminary report. American journal of cancer research, 8(9), 1865-1872.
Bodnarczuk, Tomasz, et al. "Hydroxyeicosatetraenoic acids in patients with pancreatic cancer: a preliminary report." American journal of cancer research vol. 8,9 (2018): 1865-1872.
Bodnarczuk T, Deskur A, Dolegowska K, Dolegowska B, Starzynska T, Blogowski W. Hydroxyeicosatetraenoic acids in patients with pancreatic cancer: a preliminary report. Am J Cancer Res. 2018 Sep 01;8(9):1865-1872. eCollection 2018. PMID: 30323978; PMCID: PMC6176181.
Copy Download .nbib Format: NLM
Metabolomics reveals a correlation between hydroxyeicosatetraenoic acids and allergic asthma: Evidence from three years' immunotherapy.

Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology

Zheng P, Bian X, Zhai Y, Li C, Li N, Hao C, Huang H, Luo W, Huang Z, Liao C, Xue M, Guo MQ, Sun B, Wu JL.
PMID: 34087025
Pediatr Allergy Immunol. 2021 Nov;32(8):1654-1662. doi: 10.1111/pai.13569. Epub 2021 Jun 18.

BACKGROUND: Subcutaneous immunotherapy (SCIT) is an effective, safe, preventative treatment for allergic asthma; however, potential biomarkers for monitoring SCIT have rarely been reported.OBJECTIVE: Metabolomics was utilized for the discovery of new biomarkers and analyzing disease pathophysiology of allergic asthma,...

Cite
Zheng P, Bian X, Zhai Y, et al. Metabolomics reveals a correlation between hydroxyeicosatetraenoic acids and allergic asthma: Evidence from three years' immunotherapy. Pediatr Allergy Immunol. 2021;32(8):1654-1662doi: 10.1111/pai.13569.
Zheng, P., Bian, X., Zhai, Y., Li, C., Li, N., Hao, C., Huang, H., Luo, W., Huang, Z., Liao, C., Xue, M., Guo, M. Q., Sun, B., & Wu, J. L. (2021). Metabolomics reveals a correlation between hydroxyeicosatetraenoic acids and allergic asthma: Evidence from three years' immunotherapy. Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology, 32(8), 1654-1662. https://doi.org/10.1111/pai.13569
Zheng, Peiyan, et al. "Metabolomics reveals a correlation between hydroxyeicosatetraenoic acids and allergic asthma: Evidence from three years' immunotherapy." Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology vol. 32,8 (2021): 1654-1662. doi: https://doi.org/10.1111/pai.13569
Zheng P, Bian X, Zhai Y, Li C, Li N, Hao C, Huang H, Luo W, Huang Z, Liao C, Xue M, Guo MQ, Sun B, Wu JL. Metabolomics reveals a correlation between hydroxyeicosatetraenoic acids and allergic asthma: Evidence from three years' immunotherapy. Pediatr Allergy Immunol. 2021 Nov;32(8):1654-1662. doi: 10.1111/pai.13569. Epub 2021 Jun 18. PMID: 34087025.
Copy Download .nbib Format: NLM
High-fat diet activates liver iPLA.

Journal of lipid research

Moon SH, Dilthey BG, Liu X, Guan S, Sims HF, Gross RW.
PMID: 33636162
J Lipid Res. 2021;62:100052. doi: 10.1016/j.jlr.2021.100052. Epub 2021 Feb 24.

High-fat (HF) diet-induced obesity precipitates multiple metabolic disorders including insulin resistance, glucose intolerance, oxidative stress, and inflammation, resulting in the initiation of cell death programs. Previously, we demonstrated murine germline knockout of calcium-independent phospholipase A

Cite
Moon SH, Dilthey BG, Liu X, et al. High-fat diet activates liver iPLA. J Lipid Res. 2021;62:100052doi: 10.1016/j.jlr.2021.100052.
Moon, S. H., Dilthey, B. G., Liu, X., Guan, S., Sims, H. F., & Gross, R. W. (2021). High-fat diet activates liver iPLA. Journal of lipid research, 62100052. https://doi.org/10.1016/j.jlr.2021.100052
Moon, Sung Ho, et al. "High-fat diet activates liver iPLA." Journal of lipid research vol. 62 (2021): 100052. doi: https://doi.org/10.1016/j.jlr.2021.100052
Moon SH, Dilthey BG, Liu X, Guan S, Sims HF, Gross RW. High-fat diet activates liver iPLA. J Lipid Res. 2021;62:100052. doi: 10.1016/j.jlr.2021.100052. Epub 2021 Feb 24. PMID: 33636162; PMCID: PMC8010217.
Copy Download .nbib Format: NLM
Sex differences in eicosanoid formation and metabolism: A possible mediator of sex discrepancies in cardiovascular diseases.

Pharmacology & therapeutics

Gerges SH, El-Kadi AOS.
PMID: 34808133
Pharmacol Ther. 2021 Nov 19;108046. doi: 10.1016/j.pharmthera.2021.108046. Epub 2021 Nov 19.

Arachidonic acid is metabolized by cyclooxygenase, lipoxygenase, and cytochrome P450 enzymes to produce prostaglandins, leukotrienes, epoxyeicosatrienoic acids (EETs), and hydroxyeicosatetraenoic acids (HETEs), along with other eicosanoids. Eicosanoids have important physiological and pathological roles in the body, including the cardiovascular...

Cite
Gerges SH, El-Kadi AOS. Sex differences in eicosanoid formation and metabolism: A possible mediator of sex discrepancies in cardiovascular diseases. Pharmacol Ther. 2021;108046doi: 10.1016/j.pharmthera.2021.108046.
Gerges, S. H., & El-Kadi, A. O. S. (2021). Sex differences in eicosanoid formation and metabolism: A possible mediator of sex discrepancies in cardiovascular diseases. Pharmacology & therapeutics, 108046. https://doi.org/10.1016/j.pharmthera.2021.108046
Gerges, Samar H, and El-Kadi, Ayman O S. "Sex differences in eicosanoid formation and metabolism: A possible mediator of sex discrepancies in cardiovascular diseases." Pharmacology & therapeutics vol. (2021): 108046. doi: https://doi.org/10.1016/j.pharmthera.2021.108046
Gerges SH, El-Kadi AOS. Sex differences in eicosanoid formation and metabolism: A possible mediator of sex discrepancies in cardiovascular diseases. Pharmacol Ther. 2021 Nov 19;108046. doi: 10.1016/j.pharmthera.2021.108046. Epub 2021 Nov 19. PMID: 34808133.
Copy Download .nbib Format: NLM
Effect of Glutamate and Ionomycin on the Release of Arachidonic Acid, Prostaglandins and HETEs from Cultured Neurons and Astrocytes.

The European journal of neuroscience

Oomagari K, Buisson B, Dumuis A, Bockaert J, Pin JP.
PMID: 12106250
Eur J Neurosci. 1991;3(10):928-939. doi: 10.1111/j.1460-9568.1991.tb00028.x.

The release of arachidonic acid (ArA) metabolites from mouse neurons and astrocytes in primary culture has been studied in response to ionomycin or glutamate stimulation. Cells were preincubated with [3H]ArA for 24 h and the radioactivity released was examined...

Cite
Oomagari K, Buisson B, Dumuis A, et al. Effect of Glutamate and Ionomycin on the Release of Arachidonic Acid, Prostaglandins and HETEs from Cultured Neurons and Astrocytes. Eur J Neurosci. 1991;3(10):928-939doi: 10.1111/j.1460-9568.1991.tb00028.x.
Oomagari, K., Buisson, B., Dumuis, A., Bockaert, J., & Pin, J. P. (1991). Effect of Glutamate and Ionomycin on the Release of Arachidonic Acid, Prostaglandins and HETEs from Cultured Neurons and Astrocytes. The European journal of neuroscience, 3(10), 928-939. https://doi.org/10.1111/j.1460-9568.1991.tb00028.x
Oomagari, Kiyoshi, et al. "Effect of Glutamate and Ionomycin on the Release of Arachidonic Acid, Prostaglandins and HETEs from Cultured Neurons and Astrocytes." The European journal of neuroscience vol. 3,10 (1991): 928-939. doi: https://doi.org/10.1111/j.1460-9568.1991.tb00028.x
Oomagari K, Buisson B, Dumuis A, Bockaert J, Pin JP. Effect of Glutamate and Ionomycin on the Release of Arachidonic Acid, Prostaglandins and HETEs from Cultured Neurons and Astrocytes. Eur J Neurosci. 1991;3(10):928-939. doi: 10.1111/j.1460-9568.1991.tb00028.x. PMID: 12106250.
Copy Download .nbib Format: NLM
Altered Protein Expression of Cardiac CYP2J and Hepatic CYP2C, CYP4A, and CYP4F in a Mouse Model of Type II Diabetes-A Link in the Onset and Development of Cardiovascular Disease?.

Pharmaceutics

Drolet B, Pilote S, Gélinas C, Kamaliza AD, Blais-Boilard A, Virgili J, Patoine D, Simard C.
PMID: 29023376
Pharmaceutics. 2017 Oct 12;9(4). doi: 10.3390/pharmaceutics9040044.

Arachidonic acid can be metabolized by cytochrome P450 (CYP450) enzymes in a tissue- and cell-specific manner to generate vasoactive products such as epoxyeicosatrienoic acids (EETs-cardioprotective) and hydroxyeicosatetraenoic acids (HETEs-cardiotoxic). Type II diabetes is a well-recognized risk factor for developing...

Cite
Drolet B, Pilote S, Gélinas C, et al. Altered Protein Expression of Cardiac CYP2J and Hepatic CYP2C, CYP4A, and CYP4F in a Mouse Model of Type II Diabetes-A Link in the Onset and Development of Cardiovascular Disease?. Pharmaceutics. 2017;9(4)doi: 10.3390/pharmaceutics9040044.
Drolet, B., Pilote, S., Gélinas, C., Kamaliza, A. D., Blais-Boilard, A., Virgili, J., Patoine, D., & Simard, C. (2017). Altered Protein Expression of Cardiac CYP2J and Hepatic CYP2C, CYP4A, and CYP4F in a Mouse Model of Type II Diabetes-A Link in the Onset and Development of Cardiovascular Disease?. Pharmaceutics, 9(4), . https://doi.org/10.3390/pharmaceutics9040044
Drolet, Benoit, et al. "Altered Protein Expression of Cardiac CYP2J and Hepatic CYP2C, CYP4A, and CYP4F in a Mouse Model of Type II Diabetes-A Link in the Onset and Development of Cardiovascular Disease?." Pharmaceutics vol. 9,4 (2017). doi: https://doi.org/10.3390/pharmaceutics9040044
Drolet B, Pilote S, Gélinas C, Kamaliza AD, Blais-Boilard A, Virgili J, Patoine D, Simard C. Altered Protein Expression of Cardiac CYP2J and Hepatic CYP2C, CYP4A, and CYP4F in a Mouse Model of Type II Diabetes-A Link in the Onset and Development of Cardiovascular Disease?. Pharmaceutics. 2017 Oct 12;9(4). doi: 10.3390/pharmaceutics9040044. PMID: 29023376; PMCID: PMC5750650.
Copy Download .nbib Format: NLM
New insights into eicosanoid biosynthetic pathways: implications for arthritis.

Expert review of clinical immunology

Kojima F, Kapoor M, Kawai S, Crofford LJ.
PMID: 20477078
Expert Rev Clin Immunol. 2006 Mar;2(2):277-91. doi: 10.1586/1744666X.2.2.277.

Eicosanoid is a collective term for the family of lipid mediators derived from arachidonic acid (AA) metabolism. This complex family of lipids is comprised of prostaglandins, thromboxanes, leukotrienes, hydroxyeicosatetraenoic acids (HETEs) and lipoxins (LXs). The most studied enzymatic pathway...

Cite
Kojima F, Kapoor M, Kawai S, et al. New insights into eicosanoid biosynthetic pathways: implications for arthritis. Expert Rev Clin Immunol. 2006;2(2):277-91doi: 10.1586/1744666X.2.2.277.
Kojima, F., Kapoor, M., Kawai, S., & Crofford, L. J. (2006). New insights into eicosanoid biosynthetic pathways: implications for arthritis. Expert review of clinical immunology, 2(2), 277-91. https://doi.org/10.1586/1744666X.2.2.277
Kojima, Fumiaki, et al. "New insights into eicosanoid biosynthetic pathways: implications for arthritis." Expert review of clinical immunology vol. 2,2 (2006): 277-91. doi: https://doi.org/10.1586/1744666X.2.2.277
Kojima F, Kapoor M, Kawai S, Crofford LJ. New insights into eicosanoid biosynthetic pathways: implications for arthritis. Expert Rev Clin Immunol. 2006 Mar;2(2):277-91. doi: 10.1586/1744666X.2.2.277. PMID: 20477078.
Copy Download .nbib Format: NLM
Effect of Different Omega-6/Omega-3 Polyunsaturated Fatty Acid Ratios on the Formation of Monohydroxylated Fatty Acids in THP-1 Derived Macrophages.

Biology

Keeren K, Huang D, Smyl C, Fischer A, Rothe M, Weylandt KH.
PMID: 25860776
Biology (Basel). 2015 Apr 09;4(2):314-26. doi: 10.3390/biology4020314.

Omega-6 and omega-3 polyunsaturated fatty acids (n-6 and n-3 PUFA) can modulate inflammatory processes. In western diets, the content of n-6 PUFA is much higher than that of n-3 PUFA, which has been suggested to promote a pro-inflammatory phenotype....

Cite
Keeren K, Huang D, Smyl C, et al. Effect of Different Omega-6/Omega-3 Polyunsaturated Fatty Acid Ratios on the Formation of Monohydroxylated Fatty Acids in THP-1 Derived Macrophages. Biology (Basel). 2015;4(2):314-26doi: 10.3390/biology4020314.
Keeren, K., Huang, D., Smyl, C., Fischer, A., Rothe, M., & Weylandt, K. H. (2015). Effect of Different Omega-6/Omega-3 Polyunsaturated Fatty Acid Ratios on the Formation of Monohydroxylated Fatty Acids in THP-1 Derived Macrophages. Biology, 4(2), 314-26. https://doi.org/10.3390/biology4020314
Keeren, Kathrin, et al. "Effect of Different Omega-6/Omega-3 Polyunsaturated Fatty Acid Ratios on the Formation of Monohydroxylated Fatty Acids in THP-1 Derived Macrophages." Biology vol. 4,2 (2015): 314-26. doi: https://doi.org/10.3390/biology4020314
Keeren K, Huang D, Smyl C, Fischer A, Rothe M, Weylandt KH. Effect of Different Omega-6/Omega-3 Polyunsaturated Fatty Acid Ratios on the Formation of Monohydroxylated Fatty Acids in THP-1 Derived Macrophages. Biology (Basel). 2015 Apr 09;4(2):314-26. doi: 10.3390/biology4020314. PMID: 25860776; PMCID: PMC4498302.
Copy Download .nbib Format: NLM
miR-18a promotes glioblastoma development by down-regulating ALOXE3-mediated ferroptotic and anti-migration activities.

Oncogenesis

Yang X, Liu J, Wang C, Cheng KK, Xu H, Li Q, Hua T, Jiang X, Sheng L, Mao J, Liu Z.
PMID: 33579899
Oncogenesis. 2021 Feb 12;10(2):15. doi: 10.1038/s41389-021-00304-3.

The development of glioblastoma (GBM) is typically accompanied by marked changes in lipid metabolism. Oxylipins and their catalyzed enzymes lipoxygenases (LOXs) have been shown to participate in the development of cancers via multiple pathways, while the understanding of LOXs...

Cite
Yang X, Liu J, Wang C, et al. miR-18a promotes glioblastoma development by down-regulating ALOXE3-mediated ferroptotic and anti-migration activities. Oncogenesis. 2021;10(2):15doi: 10.1038/s41389-021-00304-3.
Yang, X., Liu, J., Wang, C., Cheng, K. K., Xu, H., Li, Q., Hua, T., Jiang, X., Sheng, L., Mao, J., & Liu, Z. (2021). miR-18a promotes glioblastoma development by down-regulating ALOXE3-mediated ferroptotic and anti-migration activities. Oncogenesis, 10(2), 15. https://doi.org/10.1038/s41389-021-00304-3
Yang, Xinzhi, et al. "miR-18a promotes glioblastoma development by down-regulating ALOXE3-mediated ferroptotic and anti-migration activities." Oncogenesis vol. 10,2 (2021): 15. doi: https://doi.org/10.1038/s41389-021-00304-3
Yang X, Liu J, Wang C, Cheng KK, Xu H, Li Q, Hua T, Jiang X, Sheng L, Mao J, Liu Z. miR-18a promotes glioblastoma development by down-regulating ALOXE3-mediated ferroptotic and anti-migration activities. Oncogenesis. 2021 Feb 12;10(2):15. doi: 10.1038/s41389-021-00304-3. PMID: 33579899; PMCID: PMC7881152.
Copy Download .nbib Format: NLM
Metabolomics reveals a correlation between hydroxyeicosatetraenoic acids and allergic asthma: Evidence from three years' immunotherapy.

Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology

Zheng P, Bian X, Zhai Y, Li C, Li N, Hao C, Huang H, Luo W, Huang Z, Liao C, Xue M, Guo MQ, Sun B, Wu JL.
PMID: 34087025
Pediatr Allergy Immunol. 2021 Nov;32(8):1654-1662. doi: 10.1111/pai.13569. Epub 2021 Jun 18.

BACKGROUND: Subcutaneous immunotherapy (SCIT) is an effective, safe, preventative treatment for allergic asthma; however, potential biomarkers for monitoring SCIT have rarely been reported.OBJECTIVE: Metabolomics was utilized for the discovery of new biomarkers and analyzing disease pathophysiology of allergic asthma,...

Cite
Zheng P, Bian X, Zhai Y, et al. Metabolomics reveals a correlation between hydroxyeicosatetraenoic acids and allergic asthma: Evidence from three years' immunotherapy. Pediatr Allergy Immunol. 2021;32(8):1654-1662doi: 10.1111/pai.13569.
Zheng, P., Bian, X., Zhai, Y., Li, C., Li, N., Hao, C., Huang, H., Luo, W., Huang, Z., Liao, C., Xue, M., Guo, M. Q., Sun, B., & Wu, J. L. (2021). Metabolomics reveals a correlation between hydroxyeicosatetraenoic acids and allergic asthma: Evidence from three years' immunotherapy. Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology, 32(8), 1654-1662. https://doi.org/10.1111/pai.13569
Zheng, Peiyan, et al. "Metabolomics reveals a correlation between hydroxyeicosatetraenoic acids and allergic asthma: Evidence from three years' immunotherapy." Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology vol. 32,8 (2021): 1654-1662. doi: https://doi.org/10.1111/pai.13569
Zheng P, Bian X, Zhai Y, Li C, Li N, Hao C, Huang H, Luo W, Huang Z, Liao C, Xue M, Guo MQ, Sun B, Wu JL. Metabolomics reveals a correlation between hydroxyeicosatetraenoic acids and allergic asthma: Evidence from three years' immunotherapy. Pediatr Allergy Immunol. 2021 Nov;32(8):1654-1662. doi: 10.1111/pai.13569. Epub 2021 Jun 18. PMID: 34087025.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 32 entries