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Showing 1 to 7 of 7 entries
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Erratum to: Neuroprotection by Chlorpromazine and Promethazine in Severe Transient and Permanent Ischemic Stroke.

Molecular neurobiology

Geng X, Li F, Yip J, Peng C, Elmadhoun O, Shen J, Ji X, Ding Y.
PMID: 28660483
Mol Neurobiol. 2017 Dec;54(10):8151. doi: 10.1007/s12035-017-0656-6.

No abstract available.

Cite
Geng X, Li F, Yip J, et al. Erratum to: Neuroprotection by Chlorpromazine and Promethazine in Severe Transient and Permanent Ischemic Stroke. Mol Neurobiol. 2017;54(10):8151doi: 10.1007/s12035-017-0656-6.
Geng, X., Li, F., Yip, J., Peng, C., Elmadhoun, O., Shen, J., Ji, X., & Ding, Y. (2017). Erratum to: Neuroprotection by Chlorpromazine and Promethazine in Severe Transient and Permanent Ischemic Stroke. Molecular neurobiology, 54(10), 8151. https://doi.org/10.1007/s12035-017-0656-6
Geng, Xiaokun, et al. "Erratum to: Neuroprotection by Chlorpromazine and Promethazine in Severe Transient and Permanent Ischemic Stroke." Molecular neurobiology vol. 54,10 (2017): 8151. doi: https://doi.org/10.1007/s12035-017-0656-6
Geng X, Li F, Yip J, Peng C, Elmadhoun O, Shen J, Ji X, Ding Y. Erratum to: Neuroprotection by Chlorpromazine and Promethazine in Severe Transient and Permanent Ischemic Stroke. Mol Neurobiol. 2017 Dec;54(10):8151. doi: 10.1007/s12035-017-0656-6. PMID: 28660483.
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The role of vascular endothelial growth factor in angiogenesis and brain circulation after stroke.

Brain circulation

Cosky EEP, Ding Y.
PMID: 30276340
Brain Circ. 2018 Apr-Jun;4(2):73-75. doi: 10.4103/bc.bc_8_18. Epub 2018 Jun 29.

No abstract available.

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Cosky EEP, Ding Y. The role of vascular endothelial growth factor in angiogenesis and brain circulation after stroke. Brain Circ. 2018;4(2):73-75doi: 10.4103/bc.bc_8_18.
Cosky, E. E. P., & Ding, Y. (2018). The role of vascular endothelial growth factor in angiogenesis and brain circulation after stroke. Brain circulation, 4(2), 73-75. https://doi.org/10.4103/bc.bc_8_18
Cosky, Eric Eugene Paul, and Ding, Yuchuan. "The role of vascular endothelial growth factor in angiogenesis and brain circulation after stroke." Brain circulation vol. 4,2 (2018): 73-75. doi: https://doi.org/10.4103/bc.bc_8_18
Cosky EEP, Ding Y. The role of vascular endothelial growth factor in angiogenesis and brain circulation after stroke. Brain Circ. 2018 Apr-Jun;4(2):73-75. doi: 10.4103/bc.bc_8_18. Epub 2018 Jun 29. PMID: 30276340; PMCID: PMC6126238.
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Exacerbation of Brain Injury by Post-Stroke Exercise Is Contingent Upon Exercise Initiation Timing.

Frontiers in cellular neuroscience

Li F, Geng X, Khan H, Pendy JT, Peng C, Li X, Rafols JA, Ding Y.
PMID: 29051728
Front Cell Neurosci. 2017 Oct 05;11:311. doi: 10.3389/fncel.2017.00311. eCollection 2017.

Accumulating evidence has demonstrated that post-stroke physical rehabilitation may reduce morbidity. The effectiveness of post-stroke exercise, however, appears to be contingent upon exercise initiation. This study assessed the hypothesis that very early exercise exacerbates brain injury, induces reactive oxygen...

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Li F, Geng X, Khan H, et al. Exacerbation of Brain Injury by Post-Stroke Exercise Is Contingent Upon Exercise Initiation Timing. Front Cell Neurosci. 2017;11:311doi: 10.3389/fncel.2017.00311.
Li, F., Geng, X., Khan, H., Pendy, J. T., Peng, C., Li, X., Rafols, J. A., & Ding, Y. (2017). Exacerbation of Brain Injury by Post-Stroke Exercise Is Contingent Upon Exercise Initiation Timing. Frontiers in cellular neuroscience, 11311. https://doi.org/10.3389/fncel.2017.00311
Li, Fengwu, et al. "Exacerbation of Brain Injury by Post-Stroke Exercise Is Contingent Upon Exercise Initiation Timing." Frontiers in cellular neuroscience vol. 11 (2017): 311. doi: https://doi.org/10.3389/fncel.2017.00311
Li F, Geng X, Khan H, Pendy JT, Peng C, Li X, Rafols JA, Ding Y. Exacerbation of Brain Injury by Post-Stroke Exercise Is Contingent Upon Exercise Initiation Timing. Front Cell Neurosci. 2017 Oct 05;11:311. doi: 10.3389/fncel.2017.00311. eCollection 2017. PMID: 29051728; PMCID: PMC5633611.
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NOX Activation by Subunit Interaction and Underlying Mechanisms in Disease.

Frontiers in cellular neuroscience

Rastogi R, Geng X, Li F, Ding Y.
PMID: 28119569
Front Cell Neurosci. 2017 Jan 10;10:301. doi: 10.3389/fncel.2016.00301. eCollection 2016.

Nicotinamide adenine dinucleotide phosphate (NAPDH) oxidase (NOX) is an enzyme complex with the sole function of producing superoxide anion and reactive oxygen species (ROS) at the expense of NADPH. Vital to the immune system as well as cellular signaling,...

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Rastogi R, Geng X, Li F, et al. NOX Activation by Subunit Interaction and Underlying Mechanisms in Disease. Front Cell Neurosci. 2017;10:301doi: 10.3389/fncel.2016.00301.
Rastogi, R., Geng, X., Li, F., & Ding, Y. (2016). NOX Activation by Subunit Interaction and Underlying Mechanisms in Disease. Frontiers in cellular neuroscience, 10301. https://doi.org/10.3389/fncel.2016.00301
Rastogi, Radhika, et al. "NOX Activation by Subunit Interaction and Underlying Mechanisms in Disease." Frontiers in cellular neuroscience vol. 10 (2016): 301. doi: https://doi.org/10.3389/fncel.2016.00301
Rastogi R, Geng X, Li F, Ding Y. NOX Activation by Subunit Interaction and Underlying Mechanisms in Disease. Front Cell Neurosci. 2017 Jan 10;10:301. doi: 10.3389/fncel.2016.00301. eCollection 2016. PMID: 28119569; PMCID: PMC5222855.
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Cerebral Gluconeogenesis and Diseases.

Frontiers in pharmacology

Yip J, Geng X, Shen J, Ding Y.
PMID: 28101056
Front Pharmacol. 2017 Jan 04;7:521. doi: 10.3389/fphar.2016.00521. eCollection 2016.

The gluconeogenesis pathway, which has been known to normally present in the liver, kidney, intestine, or muscle, has four irreversible steps catalyzed by the enzymes: pyruvate carboxylase, phosphoenolpyruvate carboxykinase, fructose 1,6-bisphosphatase, and glucose 6-phosphatase. Studies have also demonstrated evidence...

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Yip J, Geng X, Shen J, et al. Cerebral Gluconeogenesis and Diseases. Front Pharmacol. 2017;7:521doi: 10.3389/fphar.2016.00521.
Yip, J., Geng, X., Shen, J., & Ding, Y. (2016). Cerebral Gluconeogenesis and Diseases. Frontiers in pharmacology, 7521. https://doi.org/10.3389/fphar.2016.00521
Yip, James, et al. "Cerebral Gluconeogenesis and Diseases." Frontiers in pharmacology vol. 7 (2016): 521. doi: https://doi.org/10.3389/fphar.2016.00521
Yip J, Geng X, Shen J, Ding Y. Cerebral Gluconeogenesis and Diseases. Front Pharmacol. 2017 Jan 04;7:521. doi: 10.3389/fphar.2016.00521. eCollection 2016. PMID: 28101056; PMCID: PMC5209353.
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Nicotinamide adenine dinucleotide phosphate oxidase activation and neuronal death after ischemic stroke.

Neural regeneration research

Shen J, Rastogi R, Geng X, Ding Y.
PMID: 30761998
Neural Regen Res. 2019 Jun;14(6):948-953. doi: 10.4103/1673-5374.250568.

Nicotinamide adenine dinucleotide phosphate oxidase (NOX) is a multisubunit enzyme complex that utilizes nicotinamide adenine dinucleotide phosphate to produce superoxide anions and other reactive oxygen species. Under normal circumstances, reactive oxygen species mediate a number of important cellular functions,...

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Shen J, Rastogi R, Geng X, et al. Nicotinamide adenine dinucleotide phosphate oxidase activation and neuronal death after ischemic stroke. Neural Regen Res. 2019;14(6):948-953doi: 10.4103/1673-5374.250568.
Shen, J., Rastogi, R., Geng, X., & Ding, Y. (2019). Nicotinamide adenine dinucleotide phosphate oxidase activation and neuronal death after ischemic stroke. Neural regeneration research, 14(6), 948-953. https://doi.org/10.4103/1673-5374.250568
Shen, Jiamei, et al. "Nicotinamide adenine dinucleotide phosphate oxidase activation and neuronal death after ischemic stroke." Neural regeneration research vol. 14,6 (2019): 948-953. doi: https://doi.org/10.4103/1673-5374.250568
Shen J, Rastogi R, Geng X, Ding Y. Nicotinamide adenine dinucleotide phosphate oxidase activation and neuronal death after ischemic stroke. Neural Regen Res. 2019 Jun;14(6):948-953. doi: 10.4103/1673-5374.250568. PMID: 30761998; PMCID: PMC6404502.
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Hypoxia-inducible factor-1 α and RIP3 triggers NLRP3 inflammasome in ischemic stroke.

Brain circulation

Jiang Q, Stone CR, Geng X, Ding Y.
PMID: 30693347
Brain Circ. 2018 Oct-Dec;4(4):191-192. doi: 10.4103/bc.bc_35_18. Epub 2018 Dec 31.

No abstract available.

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Jiang Q, Stone CR, Geng X, et al. Hypoxia-inducible factor-1 α and RIP3 triggers NLRP3 inflammasome in ischemic stroke. Brain Circ. 2018;4(4):191-192doi: 10.4103/bc.bc_35_18.
Jiang, Q., Stone, C. R., Geng, X., & Ding, Y. (2018). Hypoxia-inducible factor-1 α and RIP3 triggers NLRP3 inflammasome in ischemic stroke. Brain circulation, 4(4), 191-192. https://doi.org/10.4103/bc.bc_35_18
Jiang, Qian, et al. "Hypoxia-inducible factor-1 α and RIP3 triggers NLRP3 inflammasome in ischemic stroke." Brain circulation vol. 4,4 (2018): 191-192. doi: https://doi.org/10.4103/bc.bc_35_18
Jiang Q, Stone CR, Geng X, Ding Y. Hypoxia-inducible factor-1 α and RIP3 triggers NLRP3 inflammasome in ischemic stroke. Brain Circ. 2018 Oct-Dec;4(4):191-192. doi: 10.4103/bc.bc_35_18. Epub 2018 Dec 31. PMID: 30693347; PMCID: PMC6329214.
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Showing 1 to 7 of 7 entries