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Showing 1 to 12 of 16 entries
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The Impact of BK Channels on Cellular Excitability Depends on their Subcellular Location.

Frontiers in cellular neuroscience

Bock T, Stuart GJ.
PMID: 27630543
Front Cell Neurosci. 2016 Aug 31;10:206. doi: 10.3389/fncel.2016.00206. eCollection 2016.

Large conductance calcium-activated potassium channels (or BK channels) fulfil a multitude of roles in the central nervous system. At the soma of many neuronal cell types they control the speed of action potential (AP) repolarization and therefore they can...

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Bock T, Stuart GJ. The Impact of BK Channels on Cellular Excitability Depends on their Subcellular Location. Front Cell Neurosci. 2016;10:206doi: 10.3389/fncel.2016.00206.
Bock, T., & Stuart, G. J. (2016). The Impact of BK Channels on Cellular Excitability Depends on their Subcellular Location. Frontiers in cellular neuroscience, 10206. https://doi.org/10.3389/fncel.2016.00206
Bock, Tobias, and Stuart, Greg J. "The Impact of BK Channels on Cellular Excitability Depends on their Subcellular Location." Frontiers in cellular neuroscience vol. 10 (2016): 206. doi: https://doi.org/10.3389/fncel.2016.00206
Bock T, Stuart GJ. The Impact of BK Channels on Cellular Excitability Depends on their Subcellular Location. Front Cell Neurosci. 2016 Aug 31;10:206. doi: 10.3389/fncel.2016.00206. eCollection 2016. PMID: 27630543; PMCID: PMC5006691.
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Large-Conductance Calcium-Activated Potassium Channels in Glomerulus: From Cell Signal Integration to Disease.

Frontiers in physiology

Tao J, Lan Z, Wang Y, Hei H, Tian L, Pan W, Zhang X, Peng W.
PMID: 27445840
Front Physiol. 2016 Jun 21;7:248. doi: 10.3389/fphys.2016.00248. eCollection 2016.

Large-conductance calcium-activated potassium (BK) channels are currently considered as vital players in a variety of renal physiological processes. In podocytes, BK channels become active in response to stimuli that increase local cytosolic Ca(2+), possibly secondary to activation of slit...

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Tao J, Lan Z, Wang Y, et al. Large-Conductance Calcium-Activated Potassium Channels in Glomerulus: From Cell Signal Integration to Disease. Front Physiol. 2016;7:248doi: 10.3389/fphys.2016.00248.
Tao, J., Lan, Z., Wang, Y., Hei, H., Tian, L., Pan, W., Zhang, X., & Peng, W. (2016). Large-Conductance Calcium-Activated Potassium Channels in Glomerulus: From Cell Signal Integration to Disease. Frontiers in physiology, 7248. https://doi.org/10.3389/fphys.2016.00248
Tao, Jie, et al. "Large-Conductance Calcium-Activated Potassium Channels in Glomerulus: From Cell Signal Integration to Disease." Frontiers in physiology vol. 7 (2016): 248. doi: https://doi.org/10.3389/fphys.2016.00248
Tao J, Lan Z, Wang Y, Hei H, Tian L, Pan W, Zhang X, Peng W. Large-Conductance Calcium-Activated Potassium Channels in Glomerulus: From Cell Signal Integration to Disease. Front Physiol. 2016 Jun 21;7:248. doi: 10.3389/fphys.2016.00248. eCollection 2016. PMID: 27445840; PMCID: PMC4915313.
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[Enhanced BK(Ca) single-channel activities in cerebrovascular smooth muscle cells of simulated microgravity rats.].

Sheng li xue bao : [Acta physiologica Sinica]

Xie MJ, Zhang LF, Ma J, Cheng HW.
PMID: 16094490
Sheng Li Xue Bao. 2005 Aug 25;57(4):439-45.

The aim of the present study was to investigate the changes in single-channel currents of large conductance calcium-activated potassium channels (BK(Ca) channels) in cerebral vascular smooth muscle cells (VSMCs) of rats after 1-week simulated microgravity. Sprague-Dawley rats were subjected...

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Xie MJ, Zhang LF, Ma J, et al. [Enhanced BK(Ca) single-channel activities in cerebrovascular smooth muscle cells of simulated microgravity rats.]. Sheng Li Xue Bao. 2005;57(4):439-45
Xie, M. J., Zhang, L. F., Ma, J., & Cheng, H. W. (2005). [Enhanced BK(Ca) single-channel activities in cerebrovascular smooth muscle cells of simulated microgravity rats.]. Sheng li xue bao : [Acta physiologica Sinica], 57(4), 439-45.
Xie, Man-Jiang, et al. "[Enhanced BK(Ca) single-channel activities in cerebrovascular smooth muscle cells of simulated microgravity rats.]." Sheng li xue bao : [Acta physiologica Sinica] vol. 57,4 (2005): 439-45.
Xie MJ, Zhang LF, Ma J, Cheng HW. [Enhanced BK(Ca) single-channel activities in cerebrovascular smooth muscle cells of simulated microgravity rats.]. Sheng Li Xue Bao. 2005 Aug 25;57(4):439-45. Chinese. PMID: 16094490.
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[Research progress on the impact of renin-angiotensin-aldosterone system on large-conductance calcium-activated potassium channels].

Zhonghua xin xue guan bing za zhi

Zhang ZY, Qian LL, Zheng J.
PMID: 29562435
Zhonghua Xin Xue Guan Bing Za Zhi. 2018 Mar 24;46(3):239-242. doi: 10.3760/cma.j.issn.0253-3758.2018.03.018.

No abstract available.

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Zhang ZY, Qian LL, Zheng J. [Research progress on the impact of renin-angiotensin-aldosterone system on large-conductance calcium-activated potassium channels]. Zhonghua Xin Xue Guan Bing Za Zhi. 2018;46(3):239-242doi: 10.3760/cma.j.issn.0253-3758.2018.03.018.
Zhang, Z. Y., Qian, L. L., & Zheng, J. (2018). [Research progress on the impact of renin-angiotensin-aldosterone system on large-conductance calcium-activated potassium channels]. Zhonghua xin xue guan bing za zhi, 46(3), 239-242. https://doi.org/10.3760/cma.j.issn.0253-3758.2018.03.018
Zhang, Z Y, et al. "[Research progress on the impact of renin-angiotensin-aldosterone system on large-conductance calcium-activated potassium channels]." Zhonghua xin xue guan bing za zhi vol. 46,3 (2018): 239-242. doi: https://doi.org/10.3760/cma.j.issn.0253-3758.2018.03.018
Zhang ZY, Qian LL, Zheng J. [Research progress on the impact of renin-angiotensin-aldosterone system on large-conductance calcium-activated potassium channels]. Zhonghua Xin Xue Guan Bing Za Zhi. 2018 Mar 24;46(3):239-242. doi: 10.3760/cma.j.issn.0253-3758.2018.03.018. Chinese. PMID: 29562435.
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Large-Conductance Calcium-Activated Potassium Channels and Voltage-Dependent Sodium Channels in Human Cementoblasts.

Frontiers in physiology

Kamata S, Kimura M, Ohyama S, Yamashita S, Shibukawa Y.
PMID: 33959036
Front Physiol. 2021 Apr 20;12:634846. doi: 10.3389/fphys.2021.634846. eCollection 2021.

Cementum, which is excreted by cementoblasts, provides an attachment site for collagen fibers that connect to the alveolar bone and fix the teeth into the alveolar sockets. Transmembrane ionic signaling, associated with ionic transporters, regulate various physiological processes in...

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Kamata S, Kimura M, Ohyama S, et al. Large-Conductance Calcium-Activated Potassium Channels and Voltage-Dependent Sodium Channels in Human Cementoblasts. Front Physiol. 2021;12:634846doi: 10.3389/fphys.2021.634846.
Kamata, S., Kimura, M., Ohyama, S., Yamashita, S., & Shibukawa, Y. (2021). Large-Conductance Calcium-Activated Potassium Channels and Voltage-Dependent Sodium Channels in Human Cementoblasts. Frontiers in physiology, 12634846. https://doi.org/10.3389/fphys.2021.634846
Kamata, Satomi, et al. "Large-Conductance Calcium-Activated Potassium Channels and Voltage-Dependent Sodium Channels in Human Cementoblasts." Frontiers in physiology vol. 12 (2021): 634846. doi: https://doi.org/10.3389/fphys.2021.634846
Kamata S, Kimura M, Ohyama S, Yamashita S, Shibukawa Y. Large-Conductance Calcium-Activated Potassium Channels and Voltage-Dependent Sodium Channels in Human Cementoblasts. Front Physiol. 2021 Apr 20;12:634846. doi: 10.3389/fphys.2021.634846. eCollection 2021. PMID: 33959036; PMCID: PMC8093401.
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Carbon monoxide activates large-conductance calcium-activated potassium channels of human cardiac fibroblasts through various mechanisms.

The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Bae H, Kim T, Lim I.
PMID: 33859063
Korean J Physiol Pharmacol. 2021 May 01;25(3):227-237. doi: 10.4196/kjpp.2021.25.3.227.

Carbon monoxide (CO) is a cardioprotectant and potential cardiovascular therapeutic agent. Human cardiac fibroblasts (HCFs) are important determinants of myocardial structure and function. Large-conductance Ca

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Bae H, Kim T, Lim I. Carbon monoxide activates large-conductance calcium-activated potassium channels of human cardiac fibroblasts through various mechanisms. Korean J Physiol Pharmacol. 2021;25(3):227-237doi: 10.4196/kjpp.2021.25.3.227.
Bae, H., Kim, T., & Lim, I. (2021). Carbon monoxide activates large-conductance calcium-activated potassium channels of human cardiac fibroblasts through various mechanisms. The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology, 25(3), 227-237. https://doi.org/10.4196/kjpp.2021.25.3.227
Bae, Hyemi, et al. "Carbon monoxide activates large-conductance calcium-activated potassium channels of human cardiac fibroblasts through various mechanisms." The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology vol. 25,3 (2021): 227-237. doi: https://doi.org/10.4196/kjpp.2021.25.3.227
Bae H, Kim T, Lim I. Carbon monoxide activates large-conductance calcium-activated potassium channels of human cardiac fibroblasts through various mechanisms. Korean J Physiol Pharmacol. 2021 May 01;25(3):227-237. doi: 10.4196/kjpp.2021.25.3.227. PMID: 33859063; PMCID: PMC8050612.
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Reversal of behavioural phenotype by the cannabinoid-like compound VSN16R in fragile X syndrome mice.

Brain : a journal of neurology

Hurley MJ, Deacon RMJ, Chan AWE, Baker D, Selwood DL, Cogram P.
PMID: 34196695
Brain. 2021 Jul 01; doi: 10.1093/brain/awab246. Epub 2021 Jul 01.

Fragile X Syndrome is the most common inherited intellectual disability and mono-genetic cause of autism spectrum disorder. It is a neurodevelopmental condition occurring due to a CGG trinucleotide expansion in the FMR1 gene. Polymorphisms and variants in large-conductance calcium-activated...

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Hurley MJ, Deacon RMJ, Chan AWE, et al. Reversal of behavioural phenotype by the cannabinoid-like compound VSN16R in fragile X syndrome mice. Brain. 2021;doi: 10.1093/brain/awab246.
Hurley, M. J., Deacon, R. M. J., Chan, A. W. E., Baker, D., Selwood, D. L., & Cogram, P. (2021). Reversal of behavioural phenotype by the cannabinoid-like compound VSN16R in fragile X syndrome mice. Brain : a journal of neurology, . https://doi.org/10.1093/brain/awab246
Hurley, Michael J, et al. "Reversal of behavioural phenotype by the cannabinoid-like compound VSN16R in fragile X syndrome mice." Brain : a journal of neurology vol. (2021). doi: https://doi.org/10.1093/brain/awab246
Hurley MJ, Deacon RMJ, Chan AWE, Baker D, Selwood DL, Cogram P. Reversal of behavioural phenotype by the cannabinoid-like compound VSN16R in fragile X syndrome mice. Brain. 2021 Jul 01; doi: 10.1093/brain/awab246. Epub 2021 Jul 01. PMID: 34196695.
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Molecular Mechanisms Underlying Renin-Angiotensin-Aldosterone System Mediated Regulation of BK Channels.

Frontiers in physiology

Zhang ZY, Qian LL, Wang RX.
PMID: 28955251
Front Physiol. 2017 Sep 13;8:698. doi: 10.3389/fphys.2017.00698. eCollection 2017.

Large-conductance calcium-activated potassium channels (BK channels) belong to a family of Ca

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Zhang ZY, Qian LL, Wang RX. Molecular Mechanisms Underlying Renin-Angiotensin-Aldosterone System Mediated Regulation of BK Channels. Front Physiol. 2017;8:698doi: 10.3389/fphys.2017.00698.
Zhang, Z. Y., Qian, L. L., & Wang, R. X. (2017). Molecular Mechanisms Underlying Renin-Angiotensin-Aldosterone System Mediated Regulation of BK Channels. Frontiers in physiology, 8698. https://doi.org/10.3389/fphys.2017.00698
Zhang, Zhen-Ye, et al. "Molecular Mechanisms Underlying Renin-Angiotensin-Aldosterone System Mediated Regulation of BK Channels." Frontiers in physiology vol. 8 (2017): 698. doi: https://doi.org/10.3389/fphys.2017.00698
Zhang ZY, Qian LL, Wang RX. Molecular Mechanisms Underlying Renin-Angiotensin-Aldosterone System Mediated Regulation of BK Channels. Front Physiol. 2017 Sep 13;8:698. doi: 10.3389/fphys.2017.00698. eCollection 2017. PMID: 28955251; PMCID: PMC5601423.
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Protective Mechanisms of Quercetin Against Myocardial Ischemia Reperfusion Injury.

Frontiers in physiology

Zhang YM, Zhang ZY, Wang RX.
PMID: 32848878
Front Physiol. 2020 Jul 31;11:956. doi: 10.3389/fphys.2020.00956. eCollection 2020.

Quercetin has attracted more attention in recent years due to its protective role against ischemia/reperfusion injury. Quercetin can alleviate oxidative stress injury through the inhibition of NADPH oxidase and xanthine oxidase, blockage of the Fenton reaction, and scavenging of...

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Zhang YM, Zhang ZY, Wang RX. Protective Mechanisms of Quercetin Against Myocardial Ischemia Reperfusion Injury. Front Physiol. 2020;11:956doi: 10.3389/fphys.2020.00956.
Zhang, Y. M., Zhang, Z. Y., & Wang, R. X. (2020). Protective Mechanisms of Quercetin Against Myocardial Ischemia Reperfusion Injury. Frontiers in physiology, 11956. https://doi.org/10.3389/fphys.2020.00956
Zhang, Yu-Min, et al. "Protective Mechanisms of Quercetin Against Myocardial Ischemia Reperfusion Injury." Frontiers in physiology vol. 11 (2020): 956. doi: https://doi.org/10.3389/fphys.2020.00956
Zhang YM, Zhang ZY, Wang RX. Protective Mechanisms of Quercetin Against Myocardial Ischemia Reperfusion Injury. Front Physiol. 2020 Jul 31;11:956. doi: 10.3389/fphys.2020.00956. eCollection 2020. PMID: 32848878; PMCID: PMC7412593.
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BK Channel Dysfunction in Diabetic Coronary Artery: Role of the E3 Ubiquitin Ligases.

Frontiers in physiology

Qian LL, Liu XY, Yu ZM, Wang RX.
PMID: 32547406
Front Physiol. 2020 May 29;11:453. doi: 10.3389/fphys.2020.00453. eCollection 2020.

Diabetic coronary arterial disease is a leading cause of morbidity and mortality in diabetic patients. The impaired function of large-conductance calcium-activated potassium channels (BK channels) is involved in diabetic coronary arterial disease. Many studies have indicated that the reduced...

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Qian LL, Liu XY, Yu ZM, et al. BK Channel Dysfunction in Diabetic Coronary Artery: Role of the E3 Ubiquitin Ligases. Front Physiol. 2020;11:453doi: 10.3389/fphys.2020.00453.
Qian, L. L., Liu, X. Y., Yu, Z. M., & Wang, R. X. (2020). BK Channel Dysfunction in Diabetic Coronary Artery: Role of the E3 Ubiquitin Ligases. Frontiers in physiology, 11453. https://doi.org/10.3389/fphys.2020.00453
Qian, Ling-Ling, et al. "BK Channel Dysfunction in Diabetic Coronary Artery: Role of the E3 Ubiquitin Ligases." Frontiers in physiology vol. 11 (2020): 453. doi: https://doi.org/10.3389/fphys.2020.00453
Qian LL, Liu XY, Yu ZM, Wang RX. BK Channel Dysfunction in Diabetic Coronary Artery: Role of the E3 Ubiquitin Ligases. Front Physiol. 2020 May 29;11:453. doi: 10.3389/fphys.2020.00453. eCollection 2020. PMID: 32547406; PMCID: PMC7274077.
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The Functional Availability of Arterial Kv7 Channels Is Suppressed Considerably by Large-Conductance Calcium-Activated Potassium Channels in 2- to 3-Month Old but Not in 10- to 15-Day Old Rats.

Frontiers in physiology

Ma D, Gaynullina D, Schmidt N, Mladenov M, Schubert R.
PMID: 33384611
Front Physiol. 2020 Dec 15;11:597395. doi: 10.3389/fphys.2020.597395. eCollection 2020.

BACKGROUND: Voltage-gated potassium (Kv) channels, especially Kv7 channels, are major potassium channels identified in vascular smooth muscle cells with a great, albeit differential functional impact in various vessels. Vascular smooth muscle Kv7 channels always coexist with other K channels,...

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Ma D, Gaynullina D, Schmidt N, et al. The Functional Availability of Arterial Kv7 Channels Is Suppressed Considerably by Large-Conductance Calcium-Activated Potassium Channels in 2- to 3-Month Old but Not in 10- to 15-Day Old Rats. Front Physiol. 2020;11:597395doi: 10.3389/fphys.2020.597395.
Ma, D., Gaynullina, D., Schmidt, N., Mladenov, M., & Schubert, R. (2020). The Functional Availability of Arterial Kv7 Channels Is Suppressed Considerably by Large-Conductance Calcium-Activated Potassium Channels in 2- to 3-Month Old but Not in 10- to 15-Day Old Rats. Frontiers in physiology, 11597395. https://doi.org/10.3389/fphys.2020.597395
Ma, Dongyu, et al. "The Functional Availability of Arterial Kv7 Channels Is Suppressed Considerably by Large-Conductance Calcium-Activated Potassium Channels in 2- to 3-Month Old but Not in 10- to 15-Day Old Rats." Frontiers in physiology vol. 11 (2020): 597395. doi: https://doi.org/10.3389/fphys.2020.597395
Ma D, Gaynullina D, Schmidt N, Mladenov M, Schubert R. The Functional Availability of Arterial Kv7 Channels Is Suppressed Considerably by Large-Conductance Calcium-Activated Potassium Channels in 2- to 3-Month Old but Not in 10- to 15-Day Old Rats. Front Physiol. 2020 Dec 15;11:597395. doi: 10.3389/fphys.2020.597395. eCollection 2020. PMID: 33384611; PMCID: PMC7770149.
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Kynurenine Relaxes Arteries of Normotensive Women and Those With Preeclampsia.

Circulation research

Worton SA, Pritchard HAT, Greenwood SL, Alakrawi M, Heazell AEP, Wareing M, Greenstein A, Myers JE.
PMID: 33656370
Circ Res. 2021 May 28;128(11):1679-1693. doi: 10.1161/CIRCRESAHA.120.317612. Epub 2021 Mar 03.

[Figure: see text].

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Worton SA, Pritchard HAT, Greenwood SL, et al. Kynurenine Relaxes Arteries of Normotensive Women and Those With Preeclampsia. Circ Res. 2021;128(11):1679-1693doi: 10.1161/CIRCRESAHA.120.317612.
Worton, S. A., Pritchard, H. A. T., Greenwood, S. L., Alakrawi, M., Heazell, A. E. P., Wareing, M., Greenstein, A., & Myers, J. E. (2021). Kynurenine Relaxes Arteries of Normotensive Women and Those With Preeclampsia. Circulation research, 128(11), 1679-1693. https://doi.org/10.1161/CIRCRESAHA.120.317612
Worton, Stephanie A, et al. "Kynurenine Relaxes Arteries of Normotensive Women and Those With Preeclampsia." Circulation research vol. 128,11 (2021): 1679-1693. doi: https://doi.org/10.1161/CIRCRESAHA.120.317612
Worton SA, Pritchard HAT, Greenwood SL, Alakrawi M, Heazell AEP, Wareing M, Greenstein A, Myers JE. Kynurenine Relaxes Arteries of Normotensive Women and Those With Preeclampsia. Circ Res. 2021 May 28;128(11):1679-1693. doi: 10.1161/CIRCRESAHA.120.317612. Epub 2021 Mar 03. PMID: 33656370; PMCID: PMC8154175.
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Showing 1 to 12 of 16 entries