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Publisher Correction: Guided self-organization and cortical plate formation in human brain organoids.

Nature biotechnology

Lancaster MA, Corsini NS, Wolfinger S, Gustafson EH, Phillips AW, Burkard TR, Otani T, Livesey FJ, Knoblich JA.
PMID: 30307926
Nat Biotechnol. 2018 Oct 11;36(10):1016. doi: 10.1038/nbt1018-1016a.

This corrects the article DOI: 10.1038/nbt.3906.

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Lancaster MA, Corsini NS, Wolfinger S, et al. Publisher Correction: Guided self-organization and cortical plate formation in human brain organoids. Nat Biotechnol. 2018;36(10):1016doi: 10.1038/nbt1018-1016a.
Lancaster, M. A., Corsini, N. S., Wolfinger, S., Gustafson, E. H., Phillips, A. W., Burkard, T. R., Otani, T., Livesey, F. J., & Knoblich, J. A. (2018). Publisher Correction: Guided self-organization and cortical plate formation in human brain organoids. Nature biotechnology, 36(10), 1016. https://doi.org/10.1038/nbt1018-1016a
Lancaster, Madeline A, et al. "Publisher Correction: Guided self-organization and cortical plate formation in human brain organoids." Nature biotechnology vol. 36,10 (2018): 1016. doi: https://doi.org/10.1038/nbt1018-1016a
Lancaster MA, Corsini NS, Wolfinger S, Gustafson EH, Phillips AW, Burkard TR, Otani T, Livesey FJ, Knoblich JA. Publisher Correction: Guided self-organization and cortical plate formation in human brain organoids. Nat Biotechnol. 2018 Oct 11;36(10):1016. doi: 10.1038/nbt1018-1016a. PMID: 30307926.
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Biomagnetic biomarkers for dementia: A pilot multicentre study with a recommended methodological framework for magnetoencephalography.

Alzheimer's & dementia (Amsterdam, Netherlands)

Hughes LE, Henson RN, Pereda E, Bruña R, López-Sanz D, Quinn AJ, Woolrich MW, Nobre AC, Rowe JB, Maestú F.
PMID: 31431918
Alzheimers Dement (Amst). 2019 Jun 14;11:450-462. doi: 10.1016/j.dadm.2019.04.009. eCollection 2019 Dec.

INTRODUCTION: An increasing number of studies are using magnetoencephalography (MEG) to study dementia. Here we define a common methodological framework for MEG resting-state acquisition and analysis to facilitate the pooling of data from different sites.METHODS: Two groups of patients...

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Hughes LE, Henson RN, Pereda E, et al. Biomagnetic biomarkers for dementia: A pilot multicentre study with a recommended methodological framework for magnetoencephalography. Alzheimers Dement (Amst). 2019;11:450-462doi: 10.1016/j.dadm.2019.04.009.
Hughes, L. E., Henson, R. N., Pereda, E., Bruña, R., López-Sanz, D., Quinn, A. J., Woolrich, M. W., Nobre, A. C., Rowe, J. B., Maestú, F. (2019). Biomagnetic biomarkers for dementia: A pilot multicentre study with a recommended methodological framework for magnetoencephalography. Alzheimer's & dementia (Amsterdam, Netherlands), 11450-462. https://doi.org/10.1016/j.dadm.2019.04.009
Hughes, Laura E, et al. "Biomagnetic biomarkers for dementia: A pilot multicentre study with a recommended methodological framework for magnetoencephalography." Alzheimer's & dementia (Amsterdam, Netherlands) vol. 11 (2019): 450-462. doi: https://doi.org/10.1016/j.dadm.2019.04.009
Hughes LE, Henson RN, Pereda E, Bruña R, López-Sanz D, Quinn AJ, Woolrich MW, Nobre AC, Rowe JB, Maestú F. Biomagnetic biomarkers for dementia: A pilot multicentre study with a recommended methodological framework for magnetoencephalography. Alzheimers Dement (Amst). 2019 Jun 14;11:450-462. doi: 10.1016/j.dadm.2019.04.009. eCollection 2019 Dec. PMID: 31431918; PMCID: PMC6579903.
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Clinical presentations of Wilson disease.

Annals of translational medicine

Shribman S, Warner TT, Dooley JS.
PMID: 31179297
Ann Transl Med. 2019 Apr;7:S60. doi: 10.21037/atm.2019.04.27.

The main presenting features of Wilson disease, many of which mimic common hepatic and neurologic disorders, are discussed. There is a need for specialists in these and related fields to be aware of hints from within and, more importantly,...

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Shribman S, Warner TT, Dooley JS. Clinical presentations of Wilson disease. Ann Transl Med. 2019;7:S60doi: 10.21037/atm.2019.04.27.
Shribman, S., Warner, T. T., & Dooley, J. S. (2019). Clinical presentations of Wilson disease. Annals of translational medicine, 7S60. https://doi.org/10.21037/atm.2019.04.27
Shribman, Samuel, et al. "Clinical presentations of Wilson disease." Annals of translational medicine vol. 7 (2019): S60. doi: https://doi.org/10.21037/atm.2019.04.27
Shribman S, Warner TT, Dooley JS. Clinical presentations of Wilson disease. Ann Transl Med. 2019 Apr;7:S60. doi: 10.21037/atm.2019.04.27. PMID: 31179297; PMCID: PMC6531646.
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A single systemic inflammatory insult causes acute motor deficits and accelerates disease progression in a mouse model of human tauopathy.

Alzheimer's & dementia (New York, N. Y.)

Torvell M, Hampton DW, Connick P, MacLullich AMJ, Cunningham C, Chandran S.
PMID: 31650014
Alzheimers Dement (N Y). 2019 Oct 09;5:579-591. doi: 10.1016/j.trci.2019.09.001. eCollection 2019.

INTRODUCTION: Neuroinflammation, which contributes to neurodegeneration, is a consistent hallmark of dementia. Emerging evidence suggests that systemic inflammation also contributes to disease progression.METHODS: The ability of systemically administered lipopolysaccharide (LPS - 500 μg/kg) to effect acute and chronic behavioural...

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Torvell M, Hampton DW, Connick P, et al. A single systemic inflammatory insult causes acute motor deficits and accelerates disease progression in a mouse model of human tauopathy. Alzheimers Dement (N Y). 2019;5:579-591doi: 10.1016/j.trci.2019.09.001.
Torvell, M., Hampton, D. W., Connick, P., MacLullich, A. M. J., Cunningham, C., & Chandran, S. (2019). A single systemic inflammatory insult causes acute motor deficits and accelerates disease progression in a mouse model of human tauopathy. Alzheimer's & dementia (New York, N. Y.), 5579-591. https://doi.org/10.1016/j.trci.2019.09.001
Torvell, Megan, et al. "A single systemic inflammatory insult causes acute motor deficits and accelerates disease progression in a mouse model of human tauopathy." Alzheimer's & dementia (New York, N. Y.) vol. 5 (2019): 579-591. doi: https://doi.org/10.1016/j.trci.2019.09.001
Torvell M, Hampton DW, Connick P, MacLullich AMJ, Cunningham C, Chandran S. A single systemic inflammatory insult causes acute motor deficits and accelerates disease progression in a mouse model of human tauopathy. Alzheimers Dement (N Y). 2019 Oct 09;5:579-591. doi: 10.1016/j.trci.2019.09.001. eCollection 2019. PMID: 31650014; PMCID: PMC6804509.
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Fourth European stroke science workshop.

European stroke journal

Debette S, Strbian D, Wardlaw JM, van der Worp HB, Rinkel G, Caso V, Dichgans M.
PMID: 31009021
Eur Stroke J. 2018 Sep;3(3):206-219. doi: 10.1177/2396987318774443. Epub 2018 May 24.

Lake Eibsee, Garmisch-Partenkirchen, 16 to 18 November, 2017: The European Stroke Organisation convened >120 stroke experts from 21 countries to discuss latest results and hot topics in clinical, translational and basic stroke research. Since its inception in 2011, the...

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Debette S, Strbian D, Wardlaw JM, et al. Fourth European stroke science workshop. Eur Stroke J. 2018;3(3):206-219doi: 10.1177/2396987318774443.
Debette, S., Strbian, D., Wardlaw, J. M., van der Worp, H. B., Rinkel, G., Caso, V., Dichgans, M. (2018). Fourth European stroke science workshop. European stroke journal, 3(3), 206-219. https://doi.org/10.1177/2396987318774443
Debette, S, et al. "Fourth European stroke science workshop." European stroke journal vol. 3,3 (2018): 206-219. doi: https://doi.org/10.1177/2396987318774443
Debette S, Strbian D, Wardlaw JM, van der Worp HB, Rinkel G, Caso V, Dichgans M. Fourth European stroke science workshop. Eur Stroke J. 2018 Sep;3(3):206-219. doi: 10.1177/2396987318774443. Epub 2018 May 24. PMID: 31009021; PMCID: PMC6453207.
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Tumour necrosis factor induces increased production of extracellular amyloid-β- and α-synuclein-containing aggregates by human Alzheimer's disease neurons.

Brain communications

Whiten DR, Brownjohn PW, Moore S, De S, Strano A, Zuo Y, Haneklaus M, Klenerman D, Livesey FJ.
PMID: 33543132
Brain Commun. 2020 Sep 15;2(2):fcaa146. doi: 10.1093/braincomms/fcaa146. eCollection 2020.

In addition to increased aberrant protein aggregation, inflammation has been proposed as a key element in the pathogenesis and progression of Alzheimer's disease. How inflammation interacts with other disease pathways and how protein aggregation increases during disease are not...

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Whiten DR, Brownjohn PW, Moore S, et al. Tumour necrosis factor induces increased production of extracellular amyloid-β- and α-synuclein-containing aggregates by human Alzheimer's disease neurons. Brain Commun. 2020;2(2):fcaa146doi: 10.1093/braincomms/fcaa146.
Whiten, D. R., Brownjohn, P. W., Moore, S., De, S., Strano, A., Zuo, Y., Haneklaus, M., Klenerman, D., & Livesey, F. J. (2020). Tumour necrosis factor induces increased production of extracellular amyloid-β- and α-synuclein-containing aggregates by human Alzheimer's disease neurons. Brain communications, 2(2), fcaa146. https://doi.org/10.1093/braincomms/fcaa146
Whiten, Daniel R, et al. "Tumour necrosis factor induces increased production of extracellular amyloid-β- and α-synuclein-containing aggregates by human Alzheimer's disease neurons." Brain communications vol. 2,2 (2020): fcaa146. doi: https://doi.org/10.1093/braincomms/fcaa146
Whiten DR, Brownjohn PW, Moore S, De S, Strano A, Zuo Y, Haneklaus M, Klenerman D, Livesey FJ. Tumour necrosis factor induces increased production of extracellular amyloid-β- and α-synuclein-containing aggregates by human Alzheimer's disease neurons. Brain Commun. 2020 Sep 15;2(2):fcaa146. doi: 10.1093/braincomms/fcaa146. eCollection 2020. PMID: 33543132; PMCID: PMC7850285.
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Efficient Reuse of Natural Language Processing Models for Phenotype-Mention Identification in Free-text Electronic Medical Records: A Phenotype Embedding Approach.

JMIR medical informatics

Wu H, Hodgson K, Dyson S, Morley KI, Ibrahim ZM, Iqbal E, Stewart R, Dobson RJ, Sudlow C.
PMID: 31845899
JMIR Med Inform. 2019 Dec 17;7(4):e14782. doi: 10.2196/14782.

BACKGROUND: Much effort has been put into the use of automated approaches, such as natural language processing (NLP), to mine or extract data from free-text medical records in order to construct comprehensive patient profiles for delivering better health care....

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Wu H, Hodgson K, Dyson S, et al. Efficient Reuse of Natural Language Processing Models for Phenotype-Mention Identification in Free-text Electronic Medical Records: A Phenotype Embedding Approach. JMIR Med Inform. 2019;7(4):e14782doi: 10.2196/14782.
Wu, H., Hodgson, K., Dyson, S., Morley, K. I., Ibrahim, Z. M., Iqbal, E., Stewart, R., Dobson, R. J., & Sudlow, C. (2019). Efficient Reuse of Natural Language Processing Models for Phenotype-Mention Identification in Free-text Electronic Medical Records: A Phenotype Embedding Approach. JMIR medical informatics, 7(4), e14782. https://doi.org/10.2196/14782
Wu, Honghan, et al. "Efficient Reuse of Natural Language Processing Models for Phenotype-Mention Identification in Free-text Electronic Medical Records: A Phenotype Embedding Approach." JMIR medical informatics vol. 7,4 (2019): e14782. doi: https://doi.org/10.2196/14782
Wu H, Hodgson K, Dyson S, Morley KI, Ibrahim ZM, Iqbal E, Stewart R, Dobson RJ, Sudlow C. Efficient Reuse of Natural Language Processing Models for Phenotype-Mention Identification in Free-text Electronic Medical Records: A Phenotype Embedding Approach. JMIR Med Inform. 2019 Dec 17;7(4):e14782. doi: 10.2196/14782. PMID: 31845899; PMCID: PMC6938594.
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Mirtazapine's effect on the QT interval in medically hospitalized patients.

The mental health clinician

Allen ND, Leung JG, Palmer BA.
PMID: 31942276
Ment Health Clin. 2020 Jan 09;10(1):30-33. doi: 10.9740/mhc.2020.01.030. eCollection 2020 Jan.

INTRODUCTION: Mirtazapine is generally well tolerated in medically ill patients with and without formal psychiatric comorbidity to target sleep, appetite, nausea, and pain. However, there is little data regarding mirtazapine's potential to prolong the corrected QT interval (QTc) in...

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Allen ND, Leung JG, Palmer BA. Mirtazapine's effect on the QT interval in medically hospitalized patients. Ment Health Clin. 2020;10(1):30-33doi: 10.9740/mhc.2020.01.030.
Allen, N. D., Leung, J. G., & Palmer, B. A. (2020). Mirtazapine's effect on the QT interval in medically hospitalized patients. The mental health clinician, 10(1), 30-33. https://doi.org/10.9740/mhc.2020.01.030
Allen, Nicholas D, et al. "Mirtazapine's effect on the QT interval in medically hospitalized patients." The mental health clinician vol. 10,1 (2020): 30-33. doi: https://doi.org/10.9740/mhc.2020.01.030
Allen ND, Leung JG, Palmer BA. Mirtazapine's effect on the QT interval in medically hospitalized patients. Ment Health Clin. 2020 Jan 09;10(1):30-33. doi: 10.9740/mhc.2020.01.030. eCollection 2020 Jan. PMID: 31942276; PMCID: PMC6956977.
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Depression and memory function - evidence from cross-lagged panel models with unit fixed effects in ELSA and HRS.

Psychological medicine

Jindra C, Li C, Tsang RSM, Bauermeister S, Gallacher J.
PMID: 32914740
Psychol Med. 2020 Sep 11;1-9. doi: 10.1017/S0033291720003037. Epub 2020 Sep 11.

BACKGROUND: Individuals with depression are often found to perform worse on cognitive tests and to have an increased risk of dementia. The causes and the direction of these associations are however not well understood. We looked at two specific...

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Jindra C, Li C, Tsang RSM, et al. Depression and memory function - evidence from cross-lagged panel models with unit fixed effects in ELSA and HRS. Psychol Med. 2020;1-9doi: 10.1017/S0033291720003037.
Jindra, C., Li, C., Tsang, R. S. M., Bauermeister, S., & Gallacher, J. (2020). Depression and memory function - evidence from cross-lagged panel models with unit fixed effects in ELSA and HRS. Psychological medicine, 1-9. https://doi.org/10.1017/S0033291720003037
Jindra, Christoph, et al. "Depression and memory function - evidence from cross-lagged panel models with unit fixed effects in ELSA and HRS." Psychological medicine vol. (2020): 1-9. doi: https://doi.org/10.1017/S0033291720003037
Jindra C, Li C, Tsang RSM, Bauermeister S, Gallacher J. Depression and memory function - evidence from cross-lagged panel models with unit fixed effects in ELSA and HRS. Psychol Med. 2020 Sep 11;1-9. doi: 10.1017/S0033291720003037. Epub 2020 Sep 11. PMID: 32914740.
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Cardiovascular .

Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology

Andrews JPM, MacNaught G, Moss AJ, Doris MK, Pawade T, Adamson PD, van Beek EJR, Lucatelli C, Lassen ML, Robson PM, Fayad ZA, Kwiecinski J, Slomka PJ, Berman DS, Newby DE, Dweck MR.
PMID: 31792913
J Nucl Cardiol. 2021 Oct;28(5):1-12. doi: 10.1007/s12350-019-01962-y. Epub 2019 Dec 02.

METHODS AND RESULTS: 18 patients with aortic stenosis or recent myocardial infarction underwent CONCLUSION: Cardiovascular PET/MR demonstrates good visual and quantitative agreement with PET/CT. However, PET/MR is hampered by stent-related artifacts currently limiting clinical application.

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Andrews JPM, MacNaught G, Moss AJ, et al. Cardiovascular . J Nucl Cardiol. 2019;28(5):1-12doi: 10.1007/s12350-019-01962-y.
Andrews, J. P. M., MacNaught, G., Moss, A. J., Doris, M. K., Pawade, T., Adamson, P. D., van Beek, E. J. R., Lucatelli, C., Lassen, M. L., Robson, P. M., Fayad, Z. A., Kwiecinski, J., Slomka, P. J., Berman, D. S., Newby, D. E., & Dweck, M. R. (2021). Cardiovascular . Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology, 28(5), 1-12. https://doi.org/10.1007/s12350-019-01962-y
Andrews, Jack P M, et al. "Cardiovascular ." Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology vol. 28,5 (2021): 1-12. doi: https://doi.org/10.1007/s12350-019-01962-y
Andrews JPM, MacNaught G, Moss AJ, Doris MK, Pawade T, Adamson PD, van Beek EJR, Lucatelli C, Lassen ML, Robson PM, Fayad ZA, Kwiecinski J, Slomka PJ, Berman DS, Newby DE, Dweck MR. Cardiovascular . J Nucl Cardiol. 2021 Oct;28(5):1-12. doi: 10.1007/s12350-019-01962-y. Epub 2019 Dec 02. PMID: 31792913; PMCID: PMC8616877.
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Plasma phospho-tau181 in presymptomatic and symptomatic familial Alzheimer's disease: a longitudinal cohort study.

Molecular psychiatry

O'Connor A, Karikari TK, Poole T, Ashton NJ, Lantero Rodriguez J, Khatun A, Swift I, Heslegrave AJ, Abel E, Chung E, Weston PSJ, Pavisic IM, Ryan NS, Barker S, Rossor MN, Polke JM, Frost C, Mead S, Blennow K, Zetterberg H, Fox NC.
PMID: 32665603
Mol Psychiatry. 2021 Oct;26(10):5967-5976. doi: 10.1038/s41380-020-0838-x. Epub 2020 Jul 14.

Blood biomarkers have great potential to advance clinical care and accelerate trials in Alzheimer's disease (AD). Plasma phospho-tau181 (p-tau181) is a promising blood biomarker however, it is unknown if levels increase in presymptomatic AD. Therefore, we investigated the timing...

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O'Connor A, Karikari TK, Poole T, et al. Plasma phospho-tau181 in presymptomatic and symptomatic familial Alzheimer's disease: a longitudinal cohort study. Mol Psychiatry. 2020;26(10):5967-5976doi: 10.1038/s41380-020-0838-x.
O'Connor, A., Karikari, T. K., Poole, T., Ashton, N. J., Lantero Rodriguez, J., Khatun, A., Swift, I., Heslegrave, A. J., Abel, E., Chung, E., Weston, P. S. J., Pavisic, I. M., Ryan, N. S., Barker, S., Rossor, M. N., Polke, J. M., Frost, C., Mead, S., Blennow, K., Zetterberg, H., & Fox, N. C. (2021). Plasma phospho-tau181 in presymptomatic and symptomatic familial Alzheimer's disease: a longitudinal cohort study. Molecular psychiatry, 26(10), 5967-5976. https://doi.org/10.1038/s41380-020-0838-x
O'Connor, Antoinette, et al. "Plasma phospho-tau181 in presymptomatic and symptomatic familial Alzheimer's disease: a longitudinal cohort study." Molecular psychiatry vol. 26,10 (2021): 5967-5976. doi: https://doi.org/10.1038/s41380-020-0838-x
O'Connor A, Karikari TK, Poole T, Ashton NJ, Lantero Rodriguez J, Khatun A, Swift I, Heslegrave AJ, Abel E, Chung E, Weston PSJ, Pavisic IM, Ryan NS, Barker S, Rossor MN, Polke JM, Frost C, Mead S, Blennow K, Zetterberg H, Fox NC. Plasma phospho-tau181 in presymptomatic and symptomatic familial Alzheimer's disease: a longitudinal cohort study. Mol Psychiatry. 2021 Oct;26(10):5967-5976. doi: 10.1038/s41380-020-0838-x. Epub 2020 Jul 14. PMID: 32665603.
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Investigating the association between cancer and the risk of dementia: Results from the Memento cohort.

Alzheimer's & dementia : the journal of the Alzheimer's Association

Chamberlain JD, Rouanet A, Dubois B, Pasquier F, Hanon O, Gabelle A, Ceccaldi M, Krolak-Salmon P, Béjot Y, Godefroy O, Wallon D, Gentric A, Chêne G, Dufouil C.
PMID: 33656287
Alzheimers Dement. 2021 Sep;17(9):1415-1421. doi: 10.1002/alz.12308. Epub 2021 Mar 03.

INTRODUCTION: Studies on the association of cancer and risk of dementia are inconclusive due to result heterogeneity and concerns of survivor bias and unmeasured confounding.METHODS: This study uses data from the Memento cohort, a French multicenter cohort following persons...

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Chamberlain JD, Rouanet A, Dubois B, et al. Investigating the association between cancer and the risk of dementia: Results from the Memento cohort. Alzheimers Dement. 2021;17(9):1415-1421doi: 10.1002/alz.12308.
Chamberlain, J. D., Rouanet, A., Dubois, B., Pasquier, F., Hanon, O., Gabelle, A., Ceccaldi, M., Krolak-Salmon, P., Béjot, Y., Godefroy, O., Wallon, D., Gentric, A., Chêne, G., Dufouil, C. (2021). Investigating the association between cancer and the risk of dementia: Results from the Memento cohort. Alzheimer's & dementia : the journal of the Alzheimer's Association, 17(9), 1415-1421. https://doi.org/10.1002/alz.12308
Chamberlain, Jonviea D, et al. "Investigating the association between cancer and the risk of dementia: Results from the Memento cohort." Alzheimer's & dementia : the journal of the Alzheimer's Association vol. 17,9 (2021): 1415-1421. doi: https://doi.org/10.1002/alz.12308
Chamberlain JD, Rouanet A, Dubois B, Pasquier F, Hanon O, Gabelle A, Ceccaldi M, Krolak-Salmon P, Béjot Y, Godefroy O, Wallon D, Gentric A, Chêne G, Dufouil C. Investigating the association between cancer and the risk of dementia: Results from the Memento cohort. Alzheimers Dement. 2021 Sep;17(9):1415-1421. doi: 10.1002/alz.12308. Epub 2021 Mar 03. PMID: 33656287; PMCID: PMC8518910.
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