Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 12 entries
Sorted by: Best Match Show Resources per page
Common alleles contribute to schizophrenia in CNV carriers.

Molecular psychiatry

Tansey KE, Rees E, Linden DE, Ripke S, Chambert KD, Moran JL, McCarroll SA, Holmans P, Kirov G, Walters J, Owen MJ, O'Donovan MC.
PMID: 26643540
Mol Psychiatry. 2016 Aug;21(8):1153. doi: 10.1038/mp.2015.170. Epub 2015 Dec 08.

No abstract available.

Cite
Tansey KE, Rees E, Linden DE, et al. Common alleles contribute to schizophrenia in CNV carriers. Mol Psychiatry. 2015;21(8):1153doi: 10.1038/mp.2015.170.
Tansey, K. E., Rees, E., Linden, D. E., Ripke, S., Chambert, K. D., Moran, J. L., McCarroll, S. A., Holmans, P., Kirov, G., Walters, J., Owen, M. J., & O'Donovan, M. C. (2016). Common alleles contribute to schizophrenia in CNV carriers. Molecular psychiatry, 21(8), 1153. https://doi.org/10.1038/mp.2015.170
Tansey, K E, et al. "Common alleles contribute to schizophrenia in CNV carriers." Molecular psychiatry vol. 21,8 (2016): 1153. doi: https://doi.org/10.1038/mp.2015.170
Tansey KE, Rees E, Linden DE, Ripke S, Chambert KD, Moran JL, McCarroll SA, Holmans P, Kirov G, Walters J, Owen MJ, O'Donovan MC. Common alleles contribute to schizophrenia in CNV carriers. Mol Psychiatry. 2016 Aug;21(8):1153. doi: 10.1038/mp.2015.170. Epub 2015 Dec 08. PMID: 26643540; PMCID: PMC7609344.
Copy Download .nbib Format: NLM
Examining the independent and joint effects of molecular genetic liability and environmental exposures in schizophrenia: results from the EUGEI study.

World psychiatry : official journal of the World Psychiatric Association (WPA)

Guloksuz S, Pries LK, Delespaul P, Kenis G, Luykx JJ, Lin BD, Richards AL, Akdede B, Binbay T, Altınyazar V, Yalınçetin B, Gümüş-Akay G, Cihan B, Soygür H, Ulaş H, Cankurtaran E, Kaymak SU, Mihaljevic MM, Petrovic SA, Mirjanic T, Bernardo M, Cabrera B, Bobes J, Saiz PA, García-Portilla MP, Sanjuan J, Aguilar EJ, Santos JL, Jiménez-López E, Arrojo M, Carracedo A, López G, González-Peñas J, Parellada M, Maric NP, Atbaşog Lu C, Ucok A, Alptekin K, Saka MC, Arango C, O'Donovan M, Rutten BPF, van Os J.
PMID: 31059627
World Psychiatry. 2019 Jun;18(2):173-182. doi: 10.1002/wps.20629.

Schizophrenia is a heritable complex phenotype associated with a background risk involving multiple common genetic variants of small effect and a multitude of environmental exposures. Early twin and family studies using proxy-genetic liability measures suggest gene-environment interaction in the...

Cite
Guloksuz S, Pries LK, Delespaul P, et al. Examining the independent and joint effects of molecular genetic liability and environmental exposures in schizophrenia: results from the EUGEI study. World Psychiatry. 2019;18(2):173-182doi: 10.1002/wps.20629.
Guloksuz, S., Pries, L. K., Delespaul, P., Kenis, G., Luykx, J. J., Lin, B. D., Richards, A. L., Akdede, B., Binbay, T., Altınyazar, V., Yalınçetin, B., Gümüş-Akay, G., Cihan, B., Soygür, H., Ulaş, H., Cankurtaran, E., Kaymak, S. U., Mihaljevic, M. M., Petrovic, S. A., Mirjanic, T., Bernardo, M., Cabrera, B., Bobes, J., Saiz, P. A., García-Portilla, M. P., Sanjuan, J., Aguilar, E. J., Santos, J. L., Jiménez-López, E., Arrojo, M., Carracedo, A., López, G., González-Peñas, J., Parellada, M., Maric, N. P., Atbaşog Lu, C., Ucok, A., Alptekin, K., Saka, M. C., Arango, C., O'Donovan, M., Rutten, B. P. F., & van Os, J. (2019). Examining the independent and joint effects of molecular genetic liability and environmental exposures in schizophrenia: results from the EUGEI study. World psychiatry : official journal of the World Psychiatric Association (WPA), 18(2), 173-182. https://doi.org/10.1002/wps.20629
Guloksuz, Sinan, et al. "Examining the independent and joint effects of molecular genetic liability and environmental exposures in schizophrenia: results from the EUGEI study." World psychiatry : official journal of the World Psychiatric Association (WPA) vol. 18,2 (2019): 173-182. doi: https://doi.org/10.1002/wps.20629
Guloksuz S, Pries LK, Delespaul P, Kenis G, Luykx JJ, Lin BD, Richards AL, Akdede B, Binbay T, Altınyazar V, Yalınçetin B, Gümüş-Akay G, Cihan B, Soygür H, Ulaş H, Cankurtaran E, Kaymak SU, Mihaljevic MM, Petrovic SA, Mirjanic T, Bernardo M, Cabrera B, Bobes J, Saiz PA, García-Portilla MP, Sanjuan J, Aguilar EJ, Santos JL, Jiménez-López E, Arrojo M, Carracedo A, López G, González-Peñas J, Parellada M, Maric NP, Atbaşog Lu C, Ucok A, Alptekin K, Saka MC, Arango C, O'Donovan M, Rutten BPF, van Os J. Examining the independent and joint effects of molecular genetic liability and environmental exposures in schizophrenia: results from the EUGEI study. World Psychiatry. 2019 Jun;18(2):173-182. doi: 10.1002/wps.20629. PMID: 31059627; PMCID: PMC6502485.
Copy Download .nbib Format: NLM
Publisher Correction: Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection.

Nature genetics

Pardiñas AF, Holmans P, Pocklington AJ, Escott-Price V, Ripke S, Carrera N, Legge SE, Bishop S, Cameron D, Hamshere ML, Han J, Hubbard L, Lynham A, Mantripragada K, Rees E, MacCabe JH, McCarroll SA, Baune BT, Breen G, Byrne EM, Dannlowski U, Eley TC, Hayward C, Martin NG, McIntosh AM, Plomin R, Porteous DJ, Wray NR, Caballero A, Geschwind DH, Huckins LM, Ruderfer DM, Santiago E, Sklar P, Stahl EA, Won H, Agerbo E, Als TD, Andreassen OA, Bækvad-Hansen M, Mortensen PB, Pedersen CB, Børglum AD, Bybjerg-Grauholm J, Djurovic S, Durmishi N, Pedersen MG, Golimbet V, Grove J, Hougaard DM, Mattheisen M, Molden E, Mors O, Nordentoft M, Pejovic-Milovancevic M, Sigurdsson E, Silagadze T, Hansen CS, Stefansson K, Stefansson H, Steinberg S, Tosato S, Werge T, Collier DA, Rujescu D, Kirov G, Owen MJ, O'Donovan MC, Walters JTR.
PMID: 31160808
Nat Genet. 2019 Jul;51(7):1193. doi: 10.1038/s41588-019-0450-7.

An amendment to this paper has been published and can be accessed via a link at the top of the paper.

Cite
Pardiñas AF, Holmans P, Pocklington AJ, et al. Publisher Correction: Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection. Nat Genet. 2019;51(7):1193doi: 10.1038/s41588-019-0450-7.
Pardiñas, A. F., Holmans, P., Pocklington, A. J., Escott-Price, V., Ripke, S., Carrera, N., Legge, S. E., Bishop, S., Cameron, D., Hamshere, M. L., Han, J., Hubbard, L., Lynham, A., Mantripragada, K., Rees, E., MacCabe, J. H., McCarroll, S. A., Baune, B. T., Breen, G., Byrne, E. M., Dannlowski, U., Eley, T. C., Hayward, C., Martin, N. G., McIntosh, A. M., Plomin, R., Porteous, D. J., Wray, N. R., Caballero, A., Geschwind, D. H., Huckins, L. M., Ruderfer, D. M., Santiago, E., Sklar, P., Stahl, E. A., Won, H., Agerbo, E., Als, T. D., Andreassen, O. A., Bækvad-Hansen, M., Mortensen, P. B., Pedersen, C. B., Børglum, A. D., Bybjerg-Grauholm, J., Djurovic, S., Durmishi, N., Pedersen, M. G., Golimbet, V., Grove, J., Hougaard, D. M., Mattheisen, M., Molden, E., Mors, O., Nordentoft, M., Pejovic-Milovancevic, M., Sigurdsson, E., Silagadze, T., Hansen, C. S., Stefansson, K., Stefansson, H., Steinberg, S., Tosato, S., Werge, T., Collier, D. A., Rujescu, D., Kirov, G., Owen, M. J., O'Donovan, M. C., & Walters, J. T. R. (2019). Publisher Correction: Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection. Nature genetics, 51(7), 1193. https://doi.org/10.1038/s41588-019-0450-7
Pardiñas, Antonio F, et al. "Publisher Correction: Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection." Nature genetics vol. 51,7 (2019): 1193. doi: https://doi.org/10.1038/s41588-019-0450-7
Pardiñas AF, Holmans P, Pocklington AJ, Escott-Price V, Ripke S, Carrera N, Legge SE, Bishop S, Cameron D, Hamshere ML, Han J, Hubbard L, Lynham A, Mantripragada K, Rees E, MacCabe JH, McCarroll SA, Baune BT, Breen G, Byrne EM, Dannlowski U, Eley TC, Hayward C, Martin NG, McIntosh AM, Plomin R, Porteous DJ, Wray NR, Caballero A, Geschwind DH, Huckins LM, Ruderfer DM, Santiago E, Sklar P, Stahl EA, Won H, Agerbo E, Als TD, Andreassen OA, Bækvad-Hansen M, Mortensen PB, Pedersen CB, Børglum AD, Bybjerg-Grauholm J, Djurovic S, Durmishi N, Pedersen MG, Golimbet V, Grove J, Hougaard DM, Mattheisen M, Molden E, Mors O, Nordentoft M, Pejovic-Milovancevic M, Sigurdsson E, Silagadze T, Hansen CS, Stefansson K, Stefansson H, Steinberg S, Tosato S, Werge T, Collier DA, Rujescu D, Kirov G, Owen MJ, O'Donovan MC, Walters JTR. Publisher Correction: Common schizophrenia alleles are enriched in mutation-intolerant genes and in regions under strong background selection. Nat Genet. 2019 Jul;51(7):1193. doi: 10.1038/s41588-019-0450-7. PMID: 31160808.
Copy Download .nbib Format: NLM
Translating insights from neuropsychiatric genetics and genomics for precision psychiatry.

Genome medicine

Rees E, Owen MJ.
PMID: 32349784
Genome Med. 2020 Apr 29;12(1):43. doi: 10.1186/s13073-020-00734-5.

The primary aim of precision medicine is to tailor healthcare more closely to the needs of individual patients. This requires progress in two areas: the development of more precise treatments and the ability to identify patients or groups of...

Cite
Rees E, Owen MJ. Translating insights from neuropsychiatric genetics and genomics for precision psychiatry. Genome Med. 2020;12(1):43doi: 10.1186/s13073-020-00734-5.
Rees, E., & Owen, M. J. (2020). Translating insights from neuropsychiatric genetics and genomics for precision psychiatry. Genome medicine, 12(1), 43. https://doi.org/10.1186/s13073-020-00734-5
Rees, Elliott, and Owen, Michael J. "Translating insights from neuropsychiatric genetics and genomics for precision psychiatry." Genome medicine vol. 12,1 (2020): 43. doi: https://doi.org/10.1186/s13073-020-00734-5
Rees E, Owen MJ. Translating insights from neuropsychiatric genetics and genomics for precision psychiatry. Genome Med. 2020 Apr 29;12(1):43. doi: 10.1186/s13073-020-00734-5. PMID: 32349784; PMCID: PMC7189552.
Copy Download .nbib Format: NLM
Schizophrenia and the neurodevelopmental continuum:evidence from genomics.

World psychiatry : official journal of the World Psychiatric Association (WPA)

Owen MJ, O'Donovan MC.
PMID: 28941101
World Psychiatry. 2017 Oct;16(3):227-235. doi: 10.1002/wps.20440.

The idea that disturbances occurring early in brain development contribute to the pathogenesis of schizophrenia, often referred to as the neurodevelopmental hypothesis, has become widely accepted. Despite this, the disorder is viewed as being distinct nosologically, and by implication...

Cite
Owen MJ, O'Donovan MC. Schizophrenia and the neurodevelopmental continuum:evidence from genomics. World Psychiatry. 2017;16(3):227-235doi: 10.1002/wps.20440.
Owen, M. J., & O'Donovan, M. C. (2017). Schizophrenia and the neurodevelopmental continuum:evidence from genomics. World psychiatry : official journal of the World Psychiatric Association (WPA), 16(3), 227-235. https://doi.org/10.1002/wps.20440
Owen, Michael J, and O'Donovan, Michael C. "Schizophrenia and the neurodevelopmental continuum:evidence from genomics." World psychiatry : official journal of the World Psychiatric Association (WPA) vol. 16,3 (2017): 227-235. doi: https://doi.org/10.1002/wps.20440
Owen MJ, O'Donovan MC. Schizophrenia and the neurodevelopmental continuum:evidence from genomics. World Psychiatry. 2017 Oct;16(3):227-235. doi: 10.1002/wps.20440. PMID: 28941101; PMCID: PMC5608820.
Copy Download .nbib Format: NLM
No Effect of Genome-Wide Significant Schizophrenia Risk Variation at the .

Molecular neuropsychiatry

Toste CC, Duarte RRR, Jeffries AR, Selvackadunco S, Troakes C, O'Donovan MC, Hill MJ, Bray NJ.
PMID: 31768374
Mol Neuropsychiatry. 2019 Oct;5(4):212-217. doi: 10.1159/000501022. Epub 2019 Jul 15.

A genome-wide significant association has been reported between non-coding variants at the dopamine D2 receptor (

Cite
Toste CC, Duarte RRR, Jeffries AR, et al. No Effect of Genome-Wide Significant Schizophrenia Risk Variation at the . Mol Neuropsychiatry. 2019;5(4):212-217doi: 10.1159/000501022.
Toste, C. C., Duarte, R. R. R., Jeffries, A. R., Selvackadunco, S., Troakes, C., O'Donovan, M. C., Hill, M. J., & Bray, N. J. (2019). No Effect of Genome-Wide Significant Schizophrenia Risk Variation at the . Molecular neuropsychiatry, 5(4), 212-217. https://doi.org/10.1159/000501022
Toste, Carolina C, et al. "No Effect of Genome-Wide Significant Schizophrenia Risk Variation at the ." Molecular neuropsychiatry vol. 5,4 (2019): 212-217. doi: https://doi.org/10.1159/000501022
Toste CC, Duarte RRR, Jeffries AR, Selvackadunco S, Troakes C, O'Donovan MC, Hill MJ, Bray NJ. No Effect of Genome-Wide Significant Schizophrenia Risk Variation at the . Mol Neuropsychiatry. 2019 Oct;5(4):212-217. doi: 10.1159/000501022. Epub 2019 Jul 15. PMID: 31768374; PMCID: PMC6873021.
Copy Download .nbib Format: NLM
Analysis of C9orf72 repeat expansions in a large international cohort of dementia with Lewy bodies.

Neurobiology of aging

Kun-Rodrigues C, Ross OA, Orme T, Shepherd C, Parkkinen L, Darwent L, Hernandez D, Ansorge O, Clark LN, Honig LS, Marder K, Lemstra A, Scheltens P, van der Flier W, Louwersheimer E, Holstege H, Rogaeva E, St George-Hyslop P, Londos E, Zetterberg H, Barber I, Braae A, Brown K, Morgan K, Maetzler W, Berg D, Troakes C, Al-Sarraj S, Lashley T, Holton J, Compta Y, Van Deerlin V, Trojanowski JQ, Serrano GE, Beach TG, Clarimon J, Lleó A, Morenas-Rodríguez E, Lesage S, Galasko D, Masliah E, Santana I, Diez M, Pastor P, Tienari PJ, Myllykangas L, Oinas M, Revesz T, Lees A, Boeve BF, Petersen RC, Ferman TJ, Escott-Price V, Graff-Radford N, Cairns NJ, Morris JC, Stone DJ, Pickering-Brown S, Mann D, Dickson DW, Halliday GM, Singleton A, Guerreiro R, Bras J.
PMID: 27666590
Neurobiol Aging. 2017 Jan;49:214.e13-214.e15. doi: 10.1016/j.neurobiolaging.2016.08.023. Epub 2016 Sep 02.

C9orf72 repeat expansions are a common cause of amyotrophic lateral sclerosis and frontotemporal dementia. To date, no large-scale study of dementia with Lewy bodies (DLB) has been undertaken to assess the role of C9orf72 repeat expansions in the disease....

Cite
Kun-Rodrigues C, Ross OA, Orme T, et al. Analysis of C9orf72 repeat expansions in a large international cohort of dementia with Lewy bodies. Neurobiol Aging. 2016;49:214.e13-214.e15doi: 10.1016/j.neurobiolaging.2016.08.023.
Kun-Rodrigues, C., Ross, O. A., Orme, T., Shepherd, C., Parkkinen, L., Darwent, L., Hernandez, D., Ansorge, O., Clark, L. N., Honig, L. S., Marder, K., Lemstra, A., Scheltens, P., van der Flier, W., Louwersheimer, E., Holstege, H., Rogaeva, E., St George-Hyslop, P., Londos, E., Zetterberg, H., Barber, I., Braae, A., Brown, K., Morgan, K., Maetzler, W., Berg, D., Troakes, C., Al-Sarraj, S., Lashley, T., Holton, J., Compta, Y., Van Deerlin, V., Trojanowski, J. Q., Serrano, G. E., Beach, T. G., Clarimon, J., Lleó, A., Morenas-Rodríguez, E., Lesage, S., Galasko, D., Masliah, E., Santana, I., Diez, M., Pastor, P., Tienari, P. J., Myllykangas, L., Oinas, M., Revesz, T., Lees, A., Boeve, B. F., Petersen, R. C., Ferman, T. J., Escott-Price, V., Graff-Radford, N., Cairns, N. J., Morris, J. C., Stone, D. J., Pickering-Brown, S., Mann, D., Dickson, D. W., Halliday, G. M., Singleton, A., Guerreiro, R., & Bras, J. (2017). Analysis of C9orf72 repeat expansions in a large international cohort of dementia with Lewy bodies. Neurobiology of aging, 49214.e13-214.e15. https://doi.org/10.1016/j.neurobiolaging.2016.08.023
Kun-Rodrigues, Celia, et al. "Analysis of C9orf72 repeat expansions in a large international cohort of dementia with Lewy bodies." Neurobiology of aging vol. 49 (2017): 214.e13-214.e15. doi: https://doi.org/10.1016/j.neurobiolaging.2016.08.023
Kun-Rodrigues C, Ross OA, Orme T, Shepherd C, Parkkinen L, Darwent L, Hernandez D, Ansorge O, Clark LN, Honig LS, Marder K, Lemstra A, Scheltens P, van der Flier W, Louwersheimer E, Holstege H, Rogaeva E, St George-Hyslop P, Londos E, Zetterberg H, Barber I, Braae A, Brown K, Morgan K, Maetzler W, Berg D, Troakes C, Al-Sarraj S, Lashley T, Holton J, Compta Y, Van Deerlin V, Trojanowski JQ, Serrano GE, Beach TG, Clarimon J, Lleó A, Morenas-Rodríguez E, Lesage S, Galasko D, Masliah E, Santana I, Diez M, Pastor P, Tienari PJ, Myllykangas L, Oinas M, Revesz T, Lees A, Boeve BF, Petersen RC, Ferman TJ, Escott-Price V, Graff-Radford N, Cairns NJ, Morris JC, Stone DJ, Pickering-Brown S, Mann D, Dickson DW, Halliday GM, Singleton A, Guerreiro R, Bras J. Analysis of C9orf72 repeat expansions in a large international cohort of dementia with Lewy bodies. Neurobiol Aging. 2017 Jan;49:214.e13-214.e15. doi: 10.1016/j.neurobiolaging.2016.08.023. Epub 2016 Sep 02. PMID: 27666590; PMCID: PMC5154872.
Copy Download .nbib Format: NLM
Genetic association of FMRP targets with psychiatric disorders.

Molecular psychiatry

Clifton NE, Rees E, Holmans PA, Pardiñas AF, Harwood JC, Di Florio A, Kirov G, Walters JTR, O'Donovan MC, Owen MJ, Hall J, Pocklington AJ.
PMID: 33077856
Mol Psychiatry. 2021 Jul;26(7):2977-2990. doi: 10.1038/s41380-020-00912-2. Epub 2020 Oct 19.

Genes encoding the mRNA targets of fragile X mental retardation protein (FMRP) are enriched for genetic association with psychiatric disorders. However, many FMRP targets possess functions that are themselves genetically associated with psychiatric disorders, including synaptic transmission and plasticity,...

Cite
Clifton NE, Rees E, Holmans PA, et al. Genetic association of FMRP targets with psychiatric disorders. Mol Psychiatry. 2020;26(7):2977-2990doi: 10.1038/s41380-020-00912-2.
Clifton, N. E., Rees, E., Holmans, P. A., Pardiñas, A. F., Harwood, J. C., Di Florio, A., Kirov, G., Walters, J. T. R., O'Donovan, M. C., Owen, M. J., Hall, J., & Pocklington, A. J. (2021). Genetic association of FMRP targets with psychiatric disorders. Molecular psychiatry, 26(7), 2977-2990. https://doi.org/10.1038/s41380-020-00912-2
Clifton, Nicholas E, et al. "Genetic association of FMRP targets with psychiatric disorders." Molecular psychiatry vol. 26,7 (2021): 2977-2990. doi: https://doi.org/10.1038/s41380-020-00912-2
Clifton NE, Rees E, Holmans PA, Pardiñas AF, Harwood JC, Di Florio A, Kirov G, Walters JTR, O'Donovan MC, Owen MJ, Hall J, Pocklington AJ. Genetic association of FMRP targets with psychiatric disorders. Mol Psychiatry. 2021 Jul;26(7):2977-2990. doi: 10.1038/s41380-020-00912-2. Epub 2020 Oct 19. PMID: 33077856; PMCID: PMC8505260.
Copy Download .nbib Format: NLM
Machine learning for genetic prediction of psychiatric disorders: a systematic review.

Molecular psychiatry

Bracher-Smith M, Crawford K, Escott-Price V.
PMID: 32591634
Mol Psychiatry. 2021 Jan;26(1):70-79. doi: 10.1038/s41380-020-0825-2. Epub 2020 Jun 26.

Machine learning methods have been employed to make predictions in psychiatry from genotypes, with the potential to bring improved prediction of outcomes in psychiatric genetics; however, their current performance is unclear. We aim to systematically review machine learning methods...

Cite
Bracher-Smith M, Crawford K, Escott-Price V. Machine learning for genetic prediction of psychiatric disorders: a systematic review. Mol Psychiatry. 2020;26(1):70-79doi: 10.1038/s41380-020-0825-2.
Bracher-Smith, M., Crawford, K., & Escott-Price, V. (2021). Machine learning for genetic prediction of psychiatric disorders: a systematic review. Molecular psychiatry, 26(1), 70-79. https://doi.org/10.1038/s41380-020-0825-2
Bracher-Smith, Matthew, et al. "Machine learning for genetic prediction of psychiatric disorders: a systematic review." Molecular psychiatry vol. 26,1 (2021): 70-79. doi: https://doi.org/10.1038/s41380-020-0825-2
Bracher-Smith M, Crawford K, Escott-Price V. Machine learning for genetic prediction of psychiatric disorders: a systematic review. Mol Psychiatry. 2021 Jan;26(1):70-79. doi: 10.1038/s41380-020-0825-2. Epub 2020 Jun 26. PMID: 32591634; PMCID: PMC7610853.
Copy Download .nbib Format: NLM
Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders.

Cell

[No authors listed]
PMID: 31835028
Cell. 2019 Dec 12;179(7):1469-1482.e11. doi: 10.1016/j.cell.2019.11.020.

Genetic influences on psychiatric disorders transcend diagnostic boundaries, suggesting substantial pleiotropy of contributing loci. However, the nature and mechanisms of these pleiotropic effects remain unclear. We performed analyses of 232,964 cases and 494,162 controls from genome-wide studies of anorexia...

Cite
Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell. 2019;179(7):1469-1482.e11doi: 10.1016/j.cell.2019.11.020.
(2019). Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell, 179(7), 1469-1482.e11. https://doi.org/10.1016/j.cell.2019.11.020
"Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders." Cell vol. 179,7 (2019): 1469-1482.e11. doi: https://doi.org/10.1016/j.cell.2019.11.020
Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell. 2019 Dec 12;179(7):1469-1482.e11. doi: 10.1016/j.cell.2019.11.020. PMID: 31835028; PMCID: PMC7077032.
Copy Download .nbib Format: NLM
Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders.

Cell

[No authors listed]
PMID: 31835028
Cell. 2019 Dec 12;179(7):1469-1482.e11. doi: 10.1016/j.cell.2019.11.020.

Genetic influences on psychiatric disorders transcend diagnostic boundaries, suggesting substantial pleiotropy of contributing loci. However, the nature and mechanisms of these pleiotropic effects remain unclear. We performed analyses of 232,964 cases and 494,162 controls from genome-wide studies of anorexia...

Cite
Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell. 2019;179(7):1469-1482.e11doi: 10.1016/j.cell.2019.11.020.
(2019). Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell, 179(7), 1469-1482.e11. https://doi.org/10.1016/j.cell.2019.11.020
"Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders." Cell vol. 179,7 (2019): 1469-1482.e11. doi: https://doi.org/10.1016/j.cell.2019.11.020
Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders. Cell. 2019 Dec 12;179(7):1469-1482.e11. doi: 10.1016/j.cell.2019.11.020. PMID: 31835028; PMCID: PMC7077032.
Copy Download .nbib Format: NLM
The nature of schizophrenia: As broad as it is long.

Schizophrenia research

Owen MJ, Legge SE.
PMID: 34756599
Schizophr Res. 2021 Oct 28; doi: 10.1016/j.schres.2021.10.012. Epub 2021 Oct 28.

No abstract available.

Cite
Owen MJ, Legge SE. The nature of schizophrenia: As broad as it is long. Schizophr Res. 2021;doi: 10.1016/j.schres.2021.10.012.
Owen, M. J., & Legge, S. E. (2021). The nature of schizophrenia: As broad as it is long. Schizophrenia research, . https://doi.org/10.1016/j.schres.2021.10.012
Owen, Michael J, and Legge, Sophie E. "The nature of schizophrenia: As broad as it is long." Schizophrenia research vol. (2021). doi: https://doi.org/10.1016/j.schres.2021.10.012
Owen MJ, Legge SE. The nature of schizophrenia: As broad as it is long. Schizophr Res. 2021 Oct 28; doi: 10.1016/j.schres.2021.10.012. Epub 2021 Oct 28. PMID: 34756599.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 12 entries