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Showing 1 to 12 of 26 entries
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Termination of ventricular tachycardia after methoxamine.

Critical care and resuscitation : journal of the Australasian Academy of Critical Care Medicine

Brown J.
PMID: 16573515
Crit Care Resusc. 2001 Dec;3(4):259-61.

A case of ventricular tachycardia which reverted with intravenous methoxamine is presented. The methoxamine was used to increase blood pressure during anaesthesia for direct current cardioversion. While alpha adrenergic agonists have been used previously to increase vagal tone via...

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Brown J. Termination of ventricular tachycardia after methoxamine. Crit Care Resusc. 2001;3(4):259-61
Brown, J. (2001). Termination of ventricular tachycardia after methoxamine. Critical care and resuscitation : journal of the Australasian Academy of Critical Care Medicine, 3(4), 259-61.
Brown, J. "Termination of ventricular tachycardia after methoxamine." Critical care and resuscitation : journal of the Australasian Academy of Critical Care Medicine vol. 3,4 (2001): 259-61.
Brown J. Termination of ventricular tachycardia after methoxamine. Crit Care Resusc. 2001 Dec;3(4):259-61. PMID: 16573515.
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Determination of phenylpropanolamine and methoxamine using flow-injection with fluorimetric detection.

Talanta

Viñas P, López-Erroz C, Cerdán FJ, Hernández-Córdoba M.
PMID: 18967346
Talanta. 1998 Oct;47(2):455-62. doi: 10.1016/s0039-9140(98)00154-4.

A new flow-injection procedure for the determination of phenylpropanolamine and methoxamine is proposed. The method is based on the derivatization reaction of the primary amine group with o-phthalaldehyde in the presence of 2-mercaptoethanol using fluorimetric detection. The calibration graphs...

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Viñas P, López-Erroz C, Cerdán FJ, et al. Determination of phenylpropanolamine and methoxamine using flow-injection with fluorimetric detection. Talanta. 1998;47(2):455-62doi: 10.1016/s0039-9140(98)00154-4.
Viñas, P., López-Erroz, C., Cerdán, F. J., & Hernández-Córdoba, M. (1998). Determination of phenylpropanolamine and methoxamine using flow-injection with fluorimetric detection. Talanta, 47(2), 455-62. https://doi.org/10.1016/s0039-9140(98)00154-4
Viñas, P, et al. "Determination of phenylpropanolamine and methoxamine using flow-injection with fluorimetric detection." Talanta vol. 47,2 (1998): 455-62. doi: https://doi.org/10.1016/s0039-9140(98)00154-4
Viñas P, López-Erroz C, Cerdán FJ, Hernández-Córdoba M. Determination of phenylpropanolamine and methoxamine using flow-injection with fluorimetric detection. Talanta. 1998 Oct;47(2):455-62. doi: 10.1016/s0039-9140(98)00154-4. PMID: 18967346.
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Stimulation of Alpha-Adrenoceptors Facilitates the Release of Growth Hormone-Releasing Hormone from Rat Hypothalamus in vitro.

Journal of neuroendocrinology

Tsagarakis S, Ge F, Rees LH, Besser GM, Grossman A.
PMID: 19210470
J Neuroendocrinol. 1989 Apr 01;1(2):129-33. doi: 10.1111/j.1365-2826.1989.tb00091.x.

Abstract While extensive evidence suggests that adrenoceptors play an important role in the control of growth hormone in the rat, there are few studies involving the direct measurement of growth hormone-releasing hormone (GHRH). We have therefore developed a radioimmunoassay...

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Tsagarakis S, Ge F, Rees LH, et al. Stimulation of Alpha-Adrenoceptors Facilitates the Release of Growth Hormone-Releasing Hormone from Rat Hypothalamus in vitro. J Neuroendocrinol. 1989;1(2):129-33doi: 10.1111/j.1365-2826.1989.tb00091.x.
Tsagarakis, S., Ge, F., Rees, L. H., Besser, G. M., & Grossman, A. (1989). Stimulation of Alpha-Adrenoceptors Facilitates the Release of Growth Hormone-Releasing Hormone from Rat Hypothalamus in vitro. Journal of neuroendocrinology, 1(2), 129-33. https://doi.org/10.1111/j.1365-2826.1989.tb00091.x
Tsagarakis, S, et al. "Stimulation of Alpha-Adrenoceptors Facilitates the Release of Growth Hormone-Releasing Hormone from Rat Hypothalamus in vitro." Journal of neuroendocrinology vol. 1,2 (1989): 129-33. doi: https://doi.org/10.1111/j.1365-2826.1989.tb00091.x
Tsagarakis S, Ge F, Rees LH, Besser GM, Grossman A. Stimulation of Alpha-Adrenoceptors Facilitates the Release of Growth Hormone-Releasing Hormone from Rat Hypothalamus in vitro. J Neuroendocrinol. 1989 Apr 01;1(2):129-33. doi: 10.1111/j.1365-2826.1989.tb00091.x. PMID: 19210470.
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Inhibition of human prostate stromal cell growth and smooth muscle contraction by thalidomide: A novel remedy in LUTS?.

The Prostate

Tamalunas A, Sauckel C, Ciotkowska A, Rutz B, Wang R, Huang R, Li B, Stief CG, Gratzke C, Hennenberg M.
PMID: 33687083
Prostate. 2021 May;81(7):377-389. doi: 10.1002/pros.24114. Epub 2021 Mar 09.

BACKGROUND: Medical treatment in benign prostatic hyperplasia targets prostate size to prevent disease progression, complications, and surgery, and prostate smooth muscle tone for rapid relief of lower urinary tract symptoms. Combination therapies are still required to target both at...

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Tamalunas A, Sauckel C, Ciotkowska A, et al. Inhibition of human prostate stromal cell growth and smooth muscle contraction by thalidomide: A novel remedy in LUTS?. Prostate. 2021;81(7):377-389doi: 10.1002/pros.24114.
Tamalunas, A., Sauckel, C., Ciotkowska, A., Rutz, B., Wang, R., Huang, R., Li, B., Stief, C. G., Gratzke, C., & Hennenberg, M. (2021). Inhibition of human prostate stromal cell growth and smooth muscle contraction by thalidomide: A novel remedy in LUTS?. The Prostate, 81(7), 377-389. https://doi.org/10.1002/pros.24114
Tamalunas, Alexander, et al. "Inhibition of human prostate stromal cell growth and smooth muscle contraction by thalidomide: A novel remedy in LUTS?." The Prostate vol. 81,7 (2021): 377-389. doi: https://doi.org/10.1002/pros.24114
Tamalunas A, Sauckel C, Ciotkowska A, Rutz B, Wang R, Huang R, Li B, Stief CG, Gratzke C, Hennenberg M. Inhibition of human prostate stromal cell growth and smooth muscle contraction by thalidomide: A novel remedy in LUTS?. Prostate. 2021 May;81(7):377-389. doi: 10.1002/pros.24114. Epub 2021 Mar 09. PMID: 33687083.
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Liquid chromatography-mass spectrometry behavior of Girard's reagent T derivatives of oxosteroid intact phase II metabolites for doping control purposes.

Drug testing and analysis

Kiousi P, Fragkaki AG, Kioukia-Fougia N, Angelis YS.
PMID: 33942526
Drug Test Anal. 2021 May 03; doi: 10.1002/dta.3056. Epub 2021 May 03.

Intact phase II steroid metabolites have poor product ion mass spectra under collision-induced dissociation (CID) conditions. Therefore, we present herein the liquid chromatography-electrospray ionization-tandem mass spectrometry (LC-ESI-MS/(MS)) behavior of intact phase II metabolites of oxosteroids after derivatization. Based on...

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Kiousi P, Fragkaki AG, Kioukia-Fougia N, et al. Liquid chromatography-mass spectrometry behavior of Girard's reagent T derivatives of oxosteroid intact phase II metabolites for doping control purposes. Drug Test Anal. 2021;doi: 10.1002/dta.3056.
Kiousi, P., Fragkaki, A. G., Kioukia-Fougia, N., & Angelis, Y. S. (2021). Liquid chromatography-mass spectrometry behavior of Girard's reagent T derivatives of oxosteroid intact phase II metabolites for doping control purposes. Drug testing and analysis, . https://doi.org/10.1002/dta.3056
Kiousi, Polyxeni, et al. "Liquid chromatography-mass spectrometry behavior of Girard's reagent T derivatives of oxosteroid intact phase II metabolites for doping control purposes." Drug testing and analysis vol. (2021). doi: https://doi.org/10.1002/dta.3056
Kiousi P, Fragkaki AG, Kioukia-Fougia N, Angelis YS. Liquid chromatography-mass spectrometry behavior of Girard's reagent T derivatives of oxosteroid intact phase II metabolites for doping control purposes. Drug Test Anal. 2021 May 03; doi: 10.1002/dta.3056. Epub 2021 May 03. PMID: 33942526.
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Role of lipoxygenase products in the effects of angiotensin II in the isolated aorta and perfused heart of the rat.

Mediators of inflammation

Dam JP, Worm E, Vleeming W, Post MJ, Porsius AJ, Wemer J.
PMID: 18475674
Mediators Inflamm. 1995;4(6):417-25. doi: 10.1155/S0962935195000676.

The objective of this study was to determine whether arachidonate metabolites are involved in the vasoconstrictive effects of angiotensin II in rats. In the isolated perfused heart, dexamethasone (4 mg/kg) significantly suppressed the maximal decreases in coronary flow induced...

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Dam JP, Worm E, Vleeming W, et al. Role of lipoxygenase products in the effects of angiotensin II in the isolated aorta and perfused heart of the rat. Mediators Inflamm. 1995;4(6):417-25doi: 10.1155/S0962935195000676.
Dam, J. P., Worm, E., Vleeming, W., Post, M. J., Porsius, A. J., & Wemer, J. (1995). Role of lipoxygenase products in the effects of angiotensin II in the isolated aorta and perfused heart of the rat. Mediators of inflammation, 4(6), 417-25. https://doi.org/10.1155/S0962935195000676
Dam, J P, et al. "Role of lipoxygenase products in the effects of angiotensin II in the isolated aorta and perfused heart of the rat." Mediators of inflammation vol. 4,6 (1995): 417-25. doi: https://doi.org/10.1155/S0962935195000676
Dam JP, Worm E, Vleeming W, Post MJ, Porsius AJ, Wemer J. Role of lipoxygenase products in the effects of angiotensin II in the isolated aorta and perfused heart of the rat. Mediators Inflamm. 1995;4(6):417-25. doi: 10.1155/S0962935195000676. PMID: 18475674; PMCID: PMC2365669.
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Oxyntomodulin increases intrinsic heart rate through the glucagon receptor.

Physiological reports

Mukharji A, Drucker DJ, Charron MJ, Swoap SJ.
PMID: 24303183
Physiol Rep. 2013 Oct;1(5):e00112. doi: 10.1002/phy2.112. Epub 2013 Oct 20.

Two hormones from the gastrointestinal tract, glucagon and oxyntomodulin (OXM), vigorously elevate the intrinsic heart rate (IHR) of mice. We have previously shown that OXM influences murine heart rate (HR) independent of the glucagon-like peptide 1 (GLP-1) receptor. Here,...

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Mukharji A, Drucker DJ, Charron MJ, et al. Oxyntomodulin increases intrinsic heart rate through the glucagon receptor. Physiol Rep. 2013;1(5):e00112doi: 10.1002/phy2.112.
Mukharji, A., Drucker, D. J., Charron, M. J., & Swoap, S. J. (2013). Oxyntomodulin increases intrinsic heart rate through the glucagon receptor. Physiological reports, 1(5), e00112. https://doi.org/10.1002/phy2.112
Mukharji, Auyon, et al. "Oxyntomodulin increases intrinsic heart rate through the glucagon receptor." Physiological reports vol. 1,5 (2013): e00112. doi: https://doi.org/10.1002/phy2.112
Mukharji A, Drucker DJ, Charron MJ, Swoap SJ. Oxyntomodulin increases intrinsic heart rate through the glucagon receptor. Physiol Rep. 2013 Oct;1(5):e00112. doi: 10.1002/phy2.112. Epub 2013 Oct 20. PMID: 24303183; PMCID: PMC3841047.
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Inhibition of human prostate smooth muscle contraction by the inhibitors of protein kinase C, GF109203X, and Go6983.

The Prostate

Huang R, Liu Y, Li B, Wang R, Tamalunas A, Waidelich R, Strittmatter F, Stief CG, Hennenberg M.
PMID: 34633103
Prostate. 2022 Jan;82(1):59-77. doi: 10.1002/pros.24248. Epub 2021 Oct 11.

INTRODUCTION: Prostate smooth muscle contraction is promoted by receptor-induced activation of intracellular signaling pathways. The presumed involvement in etiology and medical treatment of lower urinary tract symptoms (LUTS) suggestive of benign prostatic hyperplasia (BPH) imparts a high clinical relevance...

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Huang R, Liu Y, Li B, et al. Inhibition of human prostate smooth muscle contraction by the inhibitors of protein kinase C, GF109203X, and Go6983. Prostate. 2021;82(1):59-77doi: 10.1002/pros.24248.
Huang, R., Liu, Y., Li, B., Wang, R., Tamalunas, A., Waidelich, R., Strittmatter, F., Stief, C. G., & Hennenberg, M. (2022). Inhibition of human prostate smooth muscle contraction by the inhibitors of protein kinase C, GF109203X, and Go6983. The Prostate, 82(1), 59-77. https://doi.org/10.1002/pros.24248
Huang, Ru, et al. "Inhibition of human prostate smooth muscle contraction by the inhibitors of protein kinase C, GF109203X, and Go6983." The Prostate vol. 82,1 (2022): 59-77. doi: https://doi.org/10.1002/pros.24248
Huang R, Liu Y, Li B, Wang R, Tamalunas A, Waidelich R, Strittmatter F, Stief CG, Hennenberg M. Inhibition of human prostate smooth muscle contraction by the inhibitors of protein kinase C, GF109203X, and Go6983. Prostate. 2022 Jan;82(1):59-77. doi: 10.1002/pros.24248. Epub 2021 Oct 11. PMID: 34633103.
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The Functional Availability of Arterial Kv7 Channels Is Suppressed Considerably by Large-Conductance Calcium-Activated Potassium Channels in 2- to 3-Month Old but Not in 10- to 15-Day Old Rats.

Frontiers in physiology

Ma D, Gaynullina D, Schmidt N, Mladenov M, Schubert R.
PMID: 33384611
Front Physiol. 2020 Dec 15;11:597395. doi: 10.3389/fphys.2020.597395. eCollection 2020.

BACKGROUND: Voltage-gated potassium (Kv) channels, especially Kv7 channels, are major potassium channels identified in vascular smooth muscle cells with a great, albeit differential functional impact in various vessels. Vascular smooth muscle Kv7 channels always coexist with other K channels,...

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Ma D, Gaynullina D, Schmidt N, et al. The Functional Availability of Arterial Kv7 Channels Is Suppressed Considerably by Large-Conductance Calcium-Activated Potassium Channels in 2- to 3-Month Old but Not in 10- to 15-Day Old Rats. Front Physiol. 2020;11:597395doi: 10.3389/fphys.2020.597395.
Ma, D., Gaynullina, D., Schmidt, N., Mladenov, M., & Schubert, R. (2020). The Functional Availability of Arterial Kv7 Channels Is Suppressed Considerably by Large-Conductance Calcium-Activated Potassium Channels in 2- to 3-Month Old but Not in 10- to 15-Day Old Rats. Frontiers in physiology, 11597395. https://doi.org/10.3389/fphys.2020.597395
Ma, Dongyu, et al. "The Functional Availability of Arterial Kv7 Channels Is Suppressed Considerably by Large-Conductance Calcium-Activated Potassium Channels in 2- to 3-Month Old but Not in 10- to 15-Day Old Rats." Frontiers in physiology vol. 11 (2020): 597395. doi: https://doi.org/10.3389/fphys.2020.597395
Ma D, Gaynullina D, Schmidt N, Mladenov M, Schubert R. The Functional Availability of Arterial Kv7 Channels Is Suppressed Considerably by Large-Conductance Calcium-Activated Potassium Channels in 2- to 3-Month Old but Not in 10- to 15-Day Old Rats. Front Physiol. 2020 Dec 15;11:597395. doi: 10.3389/fphys.2020.597395. eCollection 2020. PMID: 33384611; PMCID: PMC7770149.
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TdP Incidence in Methoxamine-Sensitized Rabbit Model Is Reduced With Age but Not Influenced by Hypercholesterolemia.

Frontiers in physiology

Nalos L, Jarkovská D, Švíglerová J, Süß A, Záleský J, Rajdl D, Krejčová M, Kuncová J, Rosenberg J, Štengl M.
PMID: 34234694
Front Physiol. 2021 Jun 21;12:692921. doi: 10.3389/fphys.2021.692921. eCollection 2021.

Metabolic syndrome is associated with hypercholesterolemia, cardiac remodeling, and increased susceptibility to ventricular arrhythmias. Effects of diet-induced hypercholesterolemia on susceptibility to torsades de pointes arrhythmias (TdP) together with potential indicators of arrhythmic risk were investigated in three experimental groups...

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Nalos L, Jarkovská D, Švíglerová J, et al. TdP Incidence in Methoxamine-Sensitized Rabbit Model Is Reduced With Age but Not Influenced by Hypercholesterolemia. Front Physiol. 2021;12:692921doi: 10.3389/fphys.2021.692921.
Nalos, L., Jarkovská, D., Švíglerová, J., Süß, A., Záleský, J., Rajdl, D., Krejčová, M., Kuncová, J., Rosenberg, J., & Štengl, M. (2021). TdP Incidence in Methoxamine-Sensitized Rabbit Model Is Reduced With Age but Not Influenced by Hypercholesterolemia. Frontiers in physiology, 12692921. https://doi.org/10.3389/fphys.2021.692921
Nalos, Lukáš, et al. "TdP Incidence in Methoxamine-Sensitized Rabbit Model Is Reduced With Age but Not Influenced by Hypercholesterolemia." Frontiers in physiology vol. 12 (2021): 692921. doi: https://doi.org/10.3389/fphys.2021.692921
Nalos L, Jarkovská D, Švíglerová J, Süß A, Záleský J, Rajdl D, Krejčová M, Kuncová J, Rosenberg J, Štengl M. TdP Incidence in Methoxamine-Sensitized Rabbit Model Is Reduced With Age but Not Influenced by Hypercholesterolemia. Front Physiol. 2021 Jun 21;12:692921. doi: 10.3389/fphys.2021.692921. eCollection 2021. PMID: 34234694; PMCID: PMC8255784.
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Noradrenergic modulation of intrinsic and synaptic properties of lumbar motoneurons in the neonatal rat spinal cord.

Frontiers in neural circuits

Tartas M, Morin F, Barrière G, Goillandeau M, Lacaille JC, Cazalets JR, Bertrand SS.
PMID: 20300468
Front Neural Circuits. 2010 Mar 08;4:4. doi: 10.3389/neuro.04.004.2010. eCollection 2010.

Although it is known that noradrenaline (NA) powerfully controls spinal motor networks, few data are available regarding the noradrenergic (NAergic) modulation of intrinsic and synaptic properties of neurons in motor networks. Our work explores the cellular basis of NAergic...

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Tartas M, Morin F, Barrière G, et al. Noradrenergic modulation of intrinsic and synaptic properties of lumbar motoneurons in the neonatal rat spinal cord. Front Neural Circuits. 2010;4:4doi: 10.3389/neuro.04.004.2010.
Tartas, M., Morin, F., Barrière, G., Goillandeau, M., Lacaille, J. C., Cazalets, J. R., & Bertrand, S. S. (2010). Noradrenergic modulation of intrinsic and synaptic properties of lumbar motoneurons in the neonatal rat spinal cord. Frontiers in neural circuits, 44. https://doi.org/10.3389/neuro.04.004.2010
Tartas, Maylis, et al. "Noradrenergic modulation of intrinsic and synaptic properties of lumbar motoneurons in the neonatal rat spinal cord." Frontiers in neural circuits vol. 4 (2010): 4. doi: https://doi.org/10.3389/neuro.04.004.2010
Tartas M, Morin F, Barrière G, Goillandeau M, Lacaille JC, Cazalets JR, Bertrand SS. Noradrenergic modulation of intrinsic and synaptic properties of lumbar motoneurons in the neonatal rat spinal cord. Front Neural Circuits. 2010 Mar 08;4:4. doi: 10.3389/neuro.04.004.2010. eCollection 2010. PMID: 20300468; PMCID: PMC2839852.
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Increased neuronal nitric oxide synthase interaction with soluble guanylate cyclase contributes to the splanchnic arterial vasodilation in portal hypertensive rats.

Hepatology research : the official journal of the Japan Society of Hepatology

Kwon SY, Groszmann RJ, Iwakiri Y.
PMID: 17300699
Hepatol Res. 2007 Jan;37(1):58-67. doi: 10.1111/j.1872-034X.2007.00005.x.

Splanchnic arterial vasodilation represents the pathophysiological hallmark of the hemodynamic dysfunction observed in portal hypertensive states. The role of neuronal nitric oxide synthase (nNOS) in the splanchnic arterial vasodilation remains to be elucidated. We therefore investigated: (i) if nNOS...

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Kwon SY, Groszmann RJ, Iwakiri Y. Increased neuronal nitric oxide synthase interaction with soluble guanylate cyclase contributes to the splanchnic arterial vasodilation in portal hypertensive rats. Hepatol Res. 2007;37(1):58-67doi: 10.1111/j.1872-034X.2007.00005.x.
Kwon, S. Y., Groszmann, R. J., & Iwakiri, Y. (2007). Increased neuronal nitric oxide synthase interaction with soluble guanylate cyclase contributes to the splanchnic arterial vasodilation in portal hypertensive rats. Hepatology research : the official journal of the Japan Society of Hepatology, 37(1), 58-67. https://doi.org/10.1111/j.1872-034X.2007.00005.x
Kwon, So Young, et al. "Increased neuronal nitric oxide synthase interaction with soluble guanylate cyclase contributes to the splanchnic arterial vasodilation in portal hypertensive rats." Hepatology research : the official journal of the Japan Society of Hepatology vol. 37,1 (2007): 58-67. doi: https://doi.org/10.1111/j.1872-034X.2007.00005.x
Kwon SY, Groszmann RJ, Iwakiri Y. Increased neuronal nitric oxide synthase interaction with soluble guanylate cyclase contributes to the splanchnic arterial vasodilation in portal hypertensive rats. Hepatol Res. 2007 Jan;37(1):58-67. doi: 10.1111/j.1872-034X.2007.00005.x. PMID: 17300699.
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Showing 1 to 12 of 26 entries