Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 11 of 11 entries
Sorted by: Best Match Show Resources per page
Colon preneoplastic lesions in animal models.

Journal of toxicologic pathology

Suzui M, Morioka T, Yoshimi N.
PMID: 24526805
J Toxicol Pathol. 2013 Dec;26(4):335-41. doi: 10.1293/tox.2013-0028. Epub 2013 Dec 26.

The animal model is a powerful and fundamental tool in the field of biochemical research including toxicology, carcinogenesis, cancer therapeutics and prevention. In the carcinogenesis animal model system, numerous examples of preneoplastic lesions have been isolated and investigated from...

Cite
Suzui M, Morioka T, Yoshimi N. Colon preneoplastic lesions in animal models. J Toxicol Pathol. 2013;26(4):335-41doi: 10.1293/tox.2013-0028.
Suzui, M., Morioka, T., & Yoshimi, N. (2013). Colon preneoplastic lesions in animal models. Journal of toxicologic pathology, 26(4), 335-41. https://doi.org/10.1293/tox.2013-0028
Suzui, Masumi, et al. "Colon preneoplastic lesions in animal models." Journal of toxicologic pathology vol. 26,4 (2013): 335-41. doi: https://doi.org/10.1293/tox.2013-0028
Suzui M, Morioka T, Yoshimi N. Colon preneoplastic lesions in animal models. J Toxicol Pathol. 2013 Dec;26(4):335-41. doi: 10.1293/tox.2013-0028. Epub 2013 Dec 26. PMID: 24526805; PMCID: PMC3921915.
Copy Download .nbib Format: NLM
Intestinal metaplasia -the effect of Acid on the gastric mucosa and gastric carcinogenesis-.

Journal of toxicologic pathology

Watanabe H.
PMID: 22272022
J Toxicol Pathol. 2010 Sep;23(3):115-23. doi: 10.1293/tox.23.115. Epub 2010 Oct 05.

This review concerns stem cells and their relation to intestinal metaplasia. When gastric regions of mice, Mongolian gerbils or several strains of rats were irradiated with a total dose of 20 Gy of X-rays given in two fractions, intestinal...

Cite
Watanabe H. Intestinal metaplasia -the effect of Acid on the gastric mucosa and gastric carcinogenesis-. J Toxicol Pathol. 2010;23(3):115-23doi: 10.1293/tox.23.115.
Watanabe, H. (2010). Intestinal metaplasia -the effect of Acid on the gastric mucosa and gastric carcinogenesis-. Journal of toxicologic pathology, 23(3), 115-23. https://doi.org/10.1293/tox.23.115
Watanabe, Hiromitsu. "Intestinal metaplasia -the effect of Acid on the gastric mucosa and gastric carcinogenesis-." Journal of toxicologic pathology vol. 23,3 (2010): 115-23. doi: https://doi.org/10.1293/tox.23.115
Watanabe H. Intestinal metaplasia -the effect of Acid on the gastric mucosa and gastric carcinogenesis-. J Toxicol Pathol. 2010 Sep;23(3):115-23. doi: 10.1293/tox.23.115. Epub 2010 Oct 05. PMID: 22272022; PMCID: PMC3234614.
Copy Download .nbib Format: NLM
Enhanced antitumor activity of doxorubicin by naringenin and metformin in breast carcinoma: an experimental study.

Naunyn-Schmiedeberg's archives of pharmacology

Pateliya B, Burade V, Goswami S.
PMID: 34125254
Naunyn Schmiedebergs Arch Pharmacol. 2021 Sep;394(9):1949-1961. doi: 10.1007/s00210-021-02104-3. Epub 2021 Jun 14.

Breast cancer is the most common malignancy in women worldwide. Strategies for cancer chemotherapy commonly require the use of combination therapy for better outcomes of results. The present work is aimed to evaluate the potential of naringenin and metformin...

Cite
Pateliya B, Burade V, Goswami S. Enhanced antitumor activity of doxorubicin by naringenin and metformin in breast carcinoma: an experimental study. Naunyn Schmiedebergs Arch Pharmacol. 2021;394(9):1949-1961doi: 10.1007/s00210-021-02104-3.
Pateliya, B., Burade, V., & Goswami, S. (2021). Enhanced antitumor activity of doxorubicin by naringenin and metformin in breast carcinoma: an experimental study. Naunyn-Schmiedeberg's archives of pharmacology, 394(9), 1949-1961. https://doi.org/10.1007/s00210-021-02104-3
Pateliya, Bharat, et al. "Enhanced antitumor activity of doxorubicin by naringenin and metformin in breast carcinoma: an experimental study." Naunyn-Schmiedeberg's archives of pharmacology vol. 394,9 (2021): 1949-1961. doi: https://doi.org/10.1007/s00210-021-02104-3
Pateliya B, Burade V, Goswami S. Enhanced antitumor activity of doxorubicin by naringenin and metformin in breast carcinoma: an experimental study. Naunyn Schmiedebergs Arch Pharmacol. 2021 Sep;394(9):1949-1961. doi: 10.1007/s00210-021-02104-3. Epub 2021 Jun 14. PMID: 34125254.
Copy Download .nbib Format: NLM
Bone marrow mesenchymal stem cells promote the progression of prostate cancer through the SDF-1/CXCR4 axis in vivo and vitro.

Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico

Luo F, Su Y, Zhang Z, Li J.
PMID: 34855138
Clin Transl Oncol. 2021 Dec 02; doi: 10.1007/s12094-021-02740-4. Epub 2021 Dec 02.

PURPOSE: The aim of this study was to investigate the involvement of the SDF-1/CXCR4 axis in the process of BMMSC homing in prostate cancer (PCa) in vivo and in vitro.METHODS: After verification of BMMSCs, we fixed the concentration gradient...

Cite
Luo F, Su Y, Zhang Z, et al. Bone marrow mesenchymal stem cells promote the progression of prostate cancer through the SDF-1/CXCR4 axis in vivo and vitro. Clin Transl Oncol. 2021;doi: 10.1007/s12094-021-02740-4.
Luo, F., Su, Y., Zhang, Z., & Li, J. (2021). Bone marrow mesenchymal stem cells promote the progression of prostate cancer through the SDF-1/CXCR4 axis in vivo and vitro. Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico, . https://doi.org/10.1007/s12094-021-02740-4
Luo, F, et al. "Bone marrow mesenchymal stem cells promote the progression of prostate cancer through the SDF-1/CXCR4 axis in vivo and vitro." Clinical & translational oncology : official publication of the Federation of Spanish Oncology Societies and of the National Cancer Institute of Mexico vol. (2021). doi: https://doi.org/10.1007/s12094-021-02740-4
Luo F, Su Y, Zhang Z, Li J. Bone marrow mesenchymal stem cells promote the progression of prostate cancer through the SDF-1/CXCR4 axis in vivo and vitro. Clin Transl Oncol. 2021 Dec 02; doi: 10.1007/s12094-021-02740-4. Epub 2021 Dec 02. PMID: 34855138.
Copy Download .nbib Format: NLM
Effects of Auraptene on IGF-1 Stimulated Cell Cycle Progression in the Human Breast Cancer Cell Line, MCF-7.

International journal of breast cancer

Krishnan P, Kleiner-Hancock H.
PMID: 23320178
Int J Breast Cancer. 2012;2012:502092. doi: 10.1155/2012/502092. Epub 2012 Dec 18.

Auraptene is being investigated for its chemopreventive effects in many models of cancer including skin, colon, prostate, and breast. Many mechanisms of action including anti-inflammatory, antiproliferative, and antiapoptotic effects are being suggested for the chemopreventive properties of auraptene. We...

Cite
Krishnan P, Kleiner-Hancock H. Effects of Auraptene on IGF-1 Stimulated Cell Cycle Progression in the Human Breast Cancer Cell Line, MCF-7. Int J Breast Cancer. 2012;2012:502092doi: 10.1155/2012/502092.
Krishnan, P., & Kleiner-Hancock, H. (2012). Effects of Auraptene on IGF-1 Stimulated Cell Cycle Progression in the Human Breast Cancer Cell Line, MCF-7. International journal of breast cancer, 2012502092. https://doi.org/10.1155/2012/502092
Krishnan, Prasad, and Kleiner-Hancock, Heather. "Effects of Auraptene on IGF-1 Stimulated Cell Cycle Progression in the Human Breast Cancer Cell Line, MCF-7." International journal of breast cancer vol. 2012 (2012): 502092. doi: https://doi.org/10.1155/2012/502092
Krishnan P, Kleiner-Hancock H. Effects of Auraptene on IGF-1 Stimulated Cell Cycle Progression in the Human Breast Cancer Cell Line, MCF-7. Int J Breast Cancer. 2012;2012:502092. doi: 10.1155/2012/502092. Epub 2012 Dec 18. PMID: 23320178; PMCID: PMC3536038.
Copy Download .nbib Format: NLM
Effect of N-Methyl-N-Nitrosourea on .

Journal of cancer prevention

Lee JY, Kim N, Choi YJ, Nam RH, Choi YJ, Lee S, Choi D, Lee HS, Kim JW, Lee DH.
PMID: 27722144
J Cancer Prev. 2016 Sep;21(3):182-186. doi: 10.15430/JCP.2016.21.3.182. Epub 2016 Sep 30.

BACKGROUND: The aim of this study was to investigate the effect of N-methyl-N-nitrosourea (MNU) treatment followed by chronic METHODS: A total of 69 C57BL/6 mice at 4 weeks of age were divided into 6 groups according to MNU treatment...

Cite
Lee JY, Kim N, Choi YJ, et al. Effect of N-Methyl-N-Nitrosourea on . J Cancer Prev. 2016;21(3):182-186doi: 10.15430/JCP.2016.21.3.182.
Lee, J. Y., Kim, N., Choi, Y. J., Nam, R. H., Choi, Y. J., Lee, S., Choi, D., Lee, H. S., Kim, J. W., & Lee, D. H. (2016). Effect of N-Methyl-N-Nitrosourea on . Journal of cancer prevention, 21(3), 182-186. https://doi.org/10.15430/JCP.2016.21.3.182
Lee, Ju Yup, et al. "Effect of N-Methyl-N-Nitrosourea on ." Journal of cancer prevention vol. 21,3 (2016): 182-186. doi: https://doi.org/10.15430/JCP.2016.21.3.182
Lee JY, Kim N, Choi YJ, Nam RH, Choi YJ, Lee S, Choi D, Lee HS, Kim JW, Lee DH. Effect of N-Methyl-N-Nitrosourea on . J Cancer Prev. 2016 Sep;21(3):182-186. doi: 10.15430/JCP.2016.21.3.182. Epub 2016 Sep 30. PMID: 27722144; PMCID: PMC5051592.
Copy Download .nbib Format: NLM
Pharmacometabolomic Pathway Response of Effective Anticancer Agents on Different Diets in Rats with Induced Mammary Tumors.

Metabolites

Cao Z, Miller MS, Lubet RA, Grubbs CJ, Beger RD.
PMID: 31336604
Metabolites. 2019 Jul 22;9(7). doi: 10.3390/metabo9070149.

Metabolomics is an effective approach to characterize the metabotype which can reflect the influence of genetics, physiological status, and environmental factors such as drug intakes, diet. Diet may change the chemopreventive efficacy of given agents due to the altered...

Cite
Cao Z, Miller MS, Lubet RA, et al. Pharmacometabolomic Pathway Response of Effective Anticancer Agents on Different Diets in Rats with Induced Mammary Tumors. Metabolites. 2019;9(7)doi: 10.3390/metabo9070149.
Cao, Z., Miller, M. S., Lubet, R. A., Grubbs, C. J., & Beger, R. D. (2019). Pharmacometabolomic Pathway Response of Effective Anticancer Agents on Different Diets in Rats with Induced Mammary Tumors. Metabolites, 9(7), . https://doi.org/10.3390/metabo9070149
Cao, Zhijun, et al. "Pharmacometabolomic Pathway Response of Effective Anticancer Agents on Different Diets in Rats with Induced Mammary Tumors." Metabolites vol. 9,7 (2019). doi: https://doi.org/10.3390/metabo9070149
Cao Z, Miller MS, Lubet RA, Grubbs CJ, Beger RD. Pharmacometabolomic Pathway Response of Effective Anticancer Agents on Different Diets in Rats with Induced Mammary Tumors. Metabolites. 2019 Jul 22;9(7). doi: 10.3390/metabo9070149. PMID: 31336604; PMCID: PMC6680681.
Copy Download .nbib Format: NLM
Effect of multiple doses of N-methyl-N-nitrosourea, an end product of methylguanidine (found in processed food), on the fertility of female Swiss albino mice.

Heliyon

Kandimalla R, Das M, Bhattacharjee S, Choudhury P, Devi R, Talukdar NC, Samanta SK.
PMID: 33912714
Heliyon. 2021 Apr 12;7(4):e06738. doi: 10.1016/j.heliyon.2021.e06738. eCollection 2021 Apr.

Methylguanidine, an originator of carcinogenic methylnitrosourea, has been found in many animal meats and processed stored food often in high concentration. The present study was designed to understand the multiple dose effect of N-methyl-N-nitrosourea (MNU), an end product of...

Cite
Kandimalla R, Das M, Bhattacharjee S, et al. Effect of multiple doses of N-methyl-N-nitrosourea, an end product of methylguanidine (found in processed food), on the fertility of female Swiss albino mice. Heliyon. 2021;7(4):e06738doi: 10.1016/j.heliyon.2021.e06738.
Kandimalla, R., Das, M., Bhattacharjee, S., Choudhury, P., Devi, R., Talukdar, N. C., & Samanta, S. K. (2021). Effect of multiple doses of N-methyl-N-nitrosourea, an end product of methylguanidine (found in processed food), on the fertility of female Swiss albino mice. Heliyon, 7(4), e06738. https://doi.org/10.1016/j.heliyon.2021.e06738
Kandimalla, Raghuram, et al. "Effect of multiple doses of N-methyl-N-nitrosourea, an end product of methylguanidine (found in processed food), on the fertility of female Swiss albino mice." Heliyon vol. 7,4 (2021): e06738. doi: https://doi.org/10.1016/j.heliyon.2021.e06738
Kandimalla R, Das M, Bhattacharjee S, Choudhury P, Devi R, Talukdar NC, Samanta SK. Effect of multiple doses of N-methyl-N-nitrosourea, an end product of methylguanidine (found in processed food), on the fertility of female Swiss albino mice. Heliyon. 2021 Apr 12;7(4):e06738. doi: 10.1016/j.heliyon.2021.e06738. eCollection 2021 Apr. PMID: 33912714; PMCID: PMC8065291.
Copy Download .nbib Format: NLM
Upstream molecular signaling pathways of p27(Kip1) expression: effects of 4-hydroxytamoxifen, dexamethasone, and retinoic acids.

Cancer cell international

Eto I.
PMID: 20170512
Cancer Cell Int. 2010 Feb 19;10:3. doi: 10.1186/1475-2867-10-3.

BACKGROUND: p27(Kip1) is a cyclin-dependent kinase inhibitor that inhibits G1-to-S phase transition of the cell cycle. It is known that a relatively large number of nutritional and chemopreventive anti-cancer agents specifically up-regulate expression of p27 without directly affecting the...

Cite
Eto I. Upstream molecular signaling pathways of p27(Kip1) expression: effects of 4-hydroxytamoxifen, dexamethasone, and retinoic acids. Cancer Cell Int. 2010;10:3doi: 10.1186/1475-2867-10-3.
Eto, I. (2010). Upstream molecular signaling pathways of p27(Kip1) expression: effects of 4-hydroxytamoxifen, dexamethasone, and retinoic acids. Cancer cell international, 103. https://doi.org/10.1186/1475-2867-10-3
Eto, Isao. "Upstream molecular signaling pathways of p27(Kip1) expression: effects of 4-hydroxytamoxifen, dexamethasone, and retinoic acids." Cancer cell international vol. 10 (2010): 3. doi: https://doi.org/10.1186/1475-2867-10-3
Eto I. Upstream molecular signaling pathways of p27(Kip1) expression: effects of 4-hydroxytamoxifen, dexamethasone, and retinoic acids. Cancer Cell Int. 2010 Feb 19;10:3. doi: 10.1186/1475-2867-10-3. PMID: 20170512; PMCID: PMC2841156.
Copy Download .nbib Format: NLM
DNA base adducts in urine andwhite blood cells of cancer patients receiving combination chemotherapies which include N-methyl-N-nitrosourea.

Biomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals

Prevost V, Likhachev AJ, Loktionova NA, Bartsch H, Wild CP, Kazanova OI, Arkhipov AI, Gershanovich ML, Shuker DE.
PMID: 23888991
Biomarkers. 1996;1(4):244-51. doi: 10.3109/13547509609079364.

Abstract Urinary 3-methyladenine (3-MeAde) excretion andlymphocyte DNA adduct formation was studied in 15 patients receiving methylnitrosourea (MNU) at several dose levels (250 mg, 300 mg and600 mg total dose, 143-385 mg m(-2)) as part of various combination chemotherapies for...

Cite
Prevost V, Likhachev AJ, Loktionova NA, et al. DNA base adducts in urine andwhite blood cells of cancer patients receiving combination chemotherapies which include N-methyl-N-nitrosourea. Biomarkers. 1996;1(4):244-51doi: 10.3109/13547509609079364.
Prevost, V., Likhachev, A. J., Loktionova, N. A., Bartsch, H., Wild, C. P., Kazanova, O. I., Arkhipov, A. I., Gershanovich, M. L., & Shuker, D. E. (1996). DNA base adducts in urine andwhite blood cells of cancer patients receiving combination chemotherapies which include N-methyl-N-nitrosourea. Biomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals, 1(4), 244-51. https://doi.org/10.3109/13547509609079364
Prevost, V, et al. "DNA base adducts in urine andwhite blood cells of cancer patients receiving combination chemotherapies which include N-methyl-N-nitrosourea." Biomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals vol. 1,4 (1996): 244-51. doi: https://doi.org/10.3109/13547509609079364
Prevost V, Likhachev AJ, Loktionova NA, Bartsch H, Wild CP, Kazanova OI, Arkhipov AI, Gershanovich ML, Shuker DE. DNA base adducts in urine andwhite blood cells of cancer patients receiving combination chemotherapies which include N-methyl-N-nitrosourea. Biomarkers. 1996;1(4):244-51. doi: 10.3109/13547509609079364. PMID: 23888991.
Copy Download .nbib Format: NLM
Quercetin Confers Tumoricidal Activity Through Multipathway Mechanisms in A N-Methylnitrosourea Rat Model of Colon Cancer.

Asian Pacific journal of cancer prevention : APJCP

Ahmed HH, Aglan HA, Zaazaa AM, Shalby AB, El Toumy SA.
PMID: 28032729
Asian Pac J Cancer Prev. 2016 Nov 01;17(11):4991-4998. doi: 10.22034/APJCP.2016.17.11.4991.

Objective: This research was conducted to explore mechanisms behind the potency of quercetin in inhibiting colon cancer induced in an experimental model. Materials and Methods: Forty adult male rats of Wistar strain were distributed into 4 groups; a negative...

Cite
Ahmed HH, Aglan HA, Zaazaa AM, et al. Quercetin Confers Tumoricidal Activity Through Multipathway Mechanisms in A N-Methylnitrosourea Rat Model of Colon Cancer. Asian Pac J Cancer Prev. 2016;17(11):4991-4998doi: 10.22034/APJCP.2016.17.11.4991.
Ahmed, H. H., Aglan, H. A., Zaazaa, A. M., Shalby, A. B., & El Toumy, S. A. (2016). Quercetin Confers Tumoricidal Activity Through Multipathway Mechanisms in A N-Methylnitrosourea Rat Model of Colon Cancer. Asian Pacific journal of cancer prevention : APJCP, 17(11), 4991-4998. https://doi.org/10.22034/APJCP.2016.17.11.4991
Ahmed, Hanaa H, et al. "Quercetin Confers Tumoricidal Activity Through Multipathway Mechanisms in A N-Methylnitrosourea Rat Model of Colon Cancer." Asian Pacific journal of cancer prevention : APJCP vol. 17,11 (2016): 4991-4998. doi: https://doi.org/10.22034/APJCP.2016.17.11.4991
Ahmed HH, Aglan HA, Zaazaa AM, Shalby AB, El Toumy SA. Quercetin Confers Tumoricidal Activity Through Multipathway Mechanisms in A N-Methylnitrosourea Rat Model of Colon Cancer. Asian Pac J Cancer Prev. 2016 Nov 01;17(11):4991-4998. doi: 10.22034/APJCP.2016.17.11.4991. PMID: 28032729; PMCID: PMC5454709.
Copy Download .nbib Format: NLM
Showing 1 to 11 of 11 entries