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Erratum: A small compound spindlactone A sensitizes human endometrial cancer cells to TRAIL-induced apoptosis via the inhibition of NAD(P)H dehydrogenase quinone 1 [Corrigendum].

OncoTargets and therapy

[No authors listed]
PMID: 30573981
Onco Targets Ther. 2018 Dec 10;11:8943. doi: 10.2147/OTT.S196977. eCollection 2018.

[This corrects the article on p. 3609 in vol. 11, PMID: 29950865.].

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Erratum: A small compound spindlactone A sensitizes human endometrial cancer cells to TRAIL-induced apoptosis via the inhibition of NAD(P)H dehydrogenase quinone 1 [Corrigendum]. Onco Targets Ther. 2018;11:8943doi: 10.2147/OTT.S196977.
(2018). Erratum: A small compound spindlactone A sensitizes human endometrial cancer cells to TRAIL-induced apoptosis via the inhibition of NAD(P)H dehydrogenase quinone 1 [Corrigendum]. OncoTargets and therapy, 118943. https://doi.org/10.2147/OTT.S196977
"Erratum: A small compound spindlactone A sensitizes human endometrial cancer cells to TRAIL-induced apoptosis via the inhibition of NAD(P)H dehydrogenase quinone 1 [Corrigendum]." OncoTargets and therapy vol. 11 (2018): 8943. doi: https://doi.org/10.2147/OTT.S196977
Erratum: A small compound spindlactone A sensitizes human endometrial cancer cells to TRAIL-induced apoptosis via the inhibition of NAD(P)H dehydrogenase quinone 1 [Corrigendum]. Onco Targets Ther. 2018 Dec 10;11:8943. doi: 10.2147/OTT.S196977. eCollection 2018. PMID: 30573981; PMCID: PMC6292400.
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A Network Pharmacology-Based Study on Antidepressant Effect of Salicornia europaea L. Extract with Experimental Support in Chronic Unpredictable Mild Stress Model Mice.

Chinese journal of integrative medicine

Sun DC, Wang RR, Xu H, Zhu XH, Sun Y, Qiao SQ, Qiao W.
PMID: 35023063
Chin J Integr Med. 2022 Jan 13; doi: 10.1007/s11655-022-2879-2. Epub 2022 Jan 13.

OBJECTIVE: To investigate the pharmacodynamic material basis, mechanism of actions and targeted diseases of Salicornia europaea L. (SE) based on the network pharmacology method, and to verify the antidepressant-like effect of the SE extract by pharmacological experiments.METHODS: Retrieval tools...

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Sun DC, Wang RR, Xu H, et al. A Network Pharmacology-Based Study on Antidepressant Effect of Salicornia europaea L. Extract with Experimental Support in Chronic Unpredictable Mild Stress Model Mice. Chin J Integr Med. 2022;doi: 10.1007/s11655-022-2879-2.
Sun, D. C., Wang, R. R., Xu, H., Zhu, X. H., Sun, Y., Qiao, S. Q., & Qiao, W. (2022). A Network Pharmacology-Based Study on Antidepressant Effect of Salicornia europaea L. Extract with Experimental Support in Chronic Unpredictable Mild Stress Model Mice. Chinese journal of integrative medicine, . https://doi.org/10.1007/s11655-022-2879-2
Sun, Dan-Chen, et al. "A Network Pharmacology-Based Study on Antidepressant Effect of Salicornia europaea L. Extract with Experimental Support in Chronic Unpredictable Mild Stress Model Mice." Chinese journal of integrative medicine vol. (2022). doi: https://doi.org/10.1007/s11655-022-2879-2
Sun DC, Wang RR, Xu H, Zhu XH, Sun Y, Qiao SQ, Qiao W. A Network Pharmacology-Based Study on Antidepressant Effect of Salicornia europaea L. Extract with Experimental Support in Chronic Unpredictable Mild Stress Model Mice. Chin J Integr Med. 2022 Jan 13; doi: 10.1007/s11655-022-2879-2. Epub 2022 Jan 13. PMID: 35023063.
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Baicalein pre-treatment alleviates hepatic ischemia/reperfusion injury in mice by regulating the Nrf2/ARE pathway.

Experimental and therapeutic medicine

Zhou Y, Tan Z, Huang H, Zeng Y, Chen S, Wei J, Huang G, Qian C, Yuan G, He S.
PMID: 34650628
Exp Ther Med. 2021 Dec;22(6):1380. doi: 10.3892/etm.2021.10816. Epub 2021 Sep 28.

Hepatic ischemia-reperfusion injury (HIRI) is caused by blood flow recovery following ischemia. Baicalein (BAI), a natural antioxidant used in traditional Chinese medicine, eliminates excessive free radicals and protects the structure of the cell membrane. However, its protective mechanism against...

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Zhou Y, Tan Z, Huang H, et al. Baicalein pre-treatment alleviates hepatic ischemia/reperfusion injury in mice by regulating the Nrf2/ARE pathway. Exp Ther Med. 2021;22(6):1380doi: 10.3892/etm.2021.10816.
Zhou, Y., Tan, Z., Huang, H., Zeng, Y., Chen, S., Wei, J., Huang, G., Qian, C., Yuan, G., & He, S. (2021). Baicalein pre-treatment alleviates hepatic ischemia/reperfusion injury in mice by regulating the Nrf2/ARE pathway. Experimental and therapeutic medicine, 22(6), 1380. https://doi.org/10.3892/etm.2021.10816
Zhou, Yi, et al. "Baicalein pre-treatment alleviates hepatic ischemia/reperfusion injury in mice by regulating the Nrf2/ARE pathway." Experimental and therapeutic medicine vol. 22,6 (2021): 1380. doi: https://doi.org/10.3892/etm.2021.10816
Zhou Y, Tan Z, Huang H, Zeng Y, Chen S, Wei J, Huang G, Qian C, Yuan G, He S. Baicalein pre-treatment alleviates hepatic ischemia/reperfusion injury in mice by regulating the Nrf2/ARE pathway. Exp Ther Med. 2021 Dec;22(6):1380. doi: 10.3892/etm.2021.10816. Epub 2021 Sep 28. PMID: 34650628; PMCID: PMC8506949.
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Gamma-tocopherol ameliorates hyperglycemia-induced hepatic inflammation associated with NLRP3 inflammasome in alloxan-induced diabetic mice.

Nutrition research and practice

Lee H, Lim Y.
PMID: 31583056
Nutr Res Pract. 2019 Oct;13(5):377-383. doi: 10.4162/nrp.2019.13.5.377. Epub 2019 Jul 10.

BACKGROUND/OBJECTIVES: Hyperglycemia-induced hepatic damage has been recognized as one of the major cause of complications in diabetes. Hepatic complications are associated with inflammation and oxidative stress in diabetes. In this study, we investigated the hypothesis that gamma-tocopherol (GT) supplementation...

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Lee H, Lim Y. Gamma-tocopherol ameliorates hyperglycemia-induced hepatic inflammation associated with NLRP3 inflammasome in alloxan-induced diabetic mice. Nutr Res Pract. 2019;13(5):377-383doi: 10.4162/nrp.2019.13.5.377.
Lee, H., & Lim, Y. (2019). Gamma-tocopherol ameliorates hyperglycemia-induced hepatic inflammation associated with NLRP3 inflammasome in alloxan-induced diabetic mice. Nutrition research and practice, 13(5), 377-383. https://doi.org/10.4162/nrp.2019.13.5.377
Lee, Heaji, and Lim, Yunsook. "Gamma-tocopherol ameliorates hyperglycemia-induced hepatic inflammation associated with NLRP3 inflammasome in alloxan-induced diabetic mice." Nutrition research and practice vol. 13,5 (2019): 377-383. doi: https://doi.org/10.4162/nrp.2019.13.5.377
Lee H, Lim Y. Gamma-tocopherol ameliorates hyperglycemia-induced hepatic inflammation associated with NLRP3 inflammasome in alloxan-induced diabetic mice. Nutr Res Pract. 2019 Oct;13(5):377-383. doi: 10.4162/nrp.2019.13.5.377. Epub 2019 Jul 10. PMID: 31583056; PMCID: PMC6760984.
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Scavenger Receptor B1 oxidative post-translational modifications are responsible for its loss in Rett syndrome.

Free radical biology & medicine

Valacchi G, Sticozzi C, Belmonte G, Cervellati F, Pecorelli A, Signorini C, Leoncini S, Ciccoli L, De Felice C, Della Ragione F, Scalabri F, Marracino F, Madonna M, D'Esposito M, Joussef H, Cervellati F, Stefania F.
PMID: 26461280
Free Radic Biol Med. 2014 Oct;75:S10-1. doi: 10.1016/j.freeradbiomed.2014.10.855. Epub 2014 Dec 10.

The modulation of the HDL receptor scavenger receptor B1 (SRB1) was evaluated in skin fibroblasts isolated from Rett syndrome (RTT) patients, a rare neurodevelopmental disorder affecting almost exclusively females associated in up to 95% of cases to de novo...

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Valacchi G, Sticozzi C, Belmonte G, et al. Scavenger Receptor B1 oxidative post-translational modifications are responsible for its loss in Rett syndrome. Free Radic Biol Med. 2014;75:S10-1doi: 10.1016/j.freeradbiomed.2014.10.855.
Valacchi, G., Sticozzi, C., Belmonte, G., Cervellati, F., Pecorelli, A., Signorini, C., Leoncini, S., Ciccoli, L., De Felice, C., Della Ragione, F., Scalabri, F., Marracino, F., Madonna, M., D'Esposito, M., Joussef, H., Cervellati, F., & Stefania, F. (2014). Scavenger Receptor B1 oxidative post-translational modifications are responsible for its loss in Rett syndrome. Free radical biology & medicine, 75S10-1. https://doi.org/10.1016/j.freeradbiomed.2014.10.855
Valacchi, Giuseppe, et al. "Scavenger Receptor B1 oxidative post-translational modifications are responsible for its loss in Rett syndrome." Free radical biology & medicine vol. 75 (2014): S10-1. doi: https://doi.org/10.1016/j.freeradbiomed.2014.10.855
Valacchi G, Sticozzi C, Belmonte G, Cervellati F, Pecorelli A, Signorini C, Leoncini S, Ciccoli L, De Felice C, Della Ragione F, Scalabri F, Marracino F, Madonna M, D'Esposito M, Joussef H, Cervellati F, Stefania F. Scavenger Receptor B1 oxidative post-translational modifications are responsible for its loss in Rett syndrome. Free Radic Biol Med. 2014 Oct;75:S10-1. doi: 10.1016/j.freeradbiomed.2014.10.855. Epub 2014 Dec 10. PMID: 26461280.
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Response of Cytoprotective and Detoxifying Proteins to Vanadate and/or Magnesium in the Rat Liver: The Nrf2-Keap1 System.

Oxidative medicine and cellular longevity

Ścibior A, Wojda I, Wnuk E, Pietrzyk Ł, Plewa Z.
PMID: 34950419
Oxid Med Cell Longev. 2021 Dec 13;2021:8447456. doi: 10.1155/2021/8447456. eCollection 2021.

Oxidative stress (OS) is a mechanism underlying metal-induced toxicity. As a redox-active element, vanadium (V) can act as a strong prooxidant and generate OS at certain levels. It can also attenuate the antioxidant barrier and intensify lipid peroxidation (LPO)....

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Ścibior A, Wojda I, Wnuk E, et al. Response of Cytoprotective and Detoxifying Proteins to Vanadate and/or Magnesium in the Rat Liver: The Nrf2-Keap1 System. Oxid Med Cell Longev. 2021;2021:8447456doi: 10.1155/2021/8447456.
Ścibior, A., Wojda, I., Wnuk, E., Pietrzyk, �. �., & Plewa, Z. (2021). Response of Cytoprotective and Detoxifying Proteins to Vanadate and/or Magnesium in the Rat Liver: The Nrf2-Keap1 System. Oxidative medicine and cellular longevity, 20218447456. https://doi.org/10.1155/2021/8447456
Ścibior, Agnieszka, et al. "Response of Cytoprotective and Detoxifying Proteins to Vanadate and/or Magnesium in the Rat Liver: The Nrf2-Keap1 System." Oxidative medicine and cellular longevity vol. 2021 (2021): 8447456. doi: https://doi.org/10.1155/2021/8447456
Ścibior A, Wojda I, Wnuk E, Pietrzyk Ł, Plewa Z. Response of Cytoprotective and Detoxifying Proteins to Vanadate and/or Magnesium in the Rat Liver: The Nrf2-Keap1 System. Oxid Med Cell Longev. 2021 Dec 13;2021:8447456. doi: 10.1155/2021/8447456. eCollection 2021. PMID: 34950419; PMCID: PMC8689234.
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LMTK2 regulates inflammation in lipopolysaccharide-stimulated BV2 cells.

Experimental and therapeutic medicine

Rui Q, Cao S, Wang X, Duan X, Iao X, Dong W, Fang Q, Zhang X, Xue Q.
PMID: 33603828
Exp Ther Med. 2021 Mar;21(3):219. doi: 10.3892/etm.2021.9621. Epub 2021 Jan 18.

Microglia activation plays vital roles in neuroinflammatory pathologys. Lemurs tyrosine kinase 2 (LMTK2) was reported to regulate NF-κB signals. In the present study, the roles of LMTK2 were investigated in lipopolysaccharide (LPS)-treated BV-2 cells. Reverse transcription-quantitative (RT-q)PCR and western...

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Rui Q, Cao S, Wang X, et al. LMTK2 regulates inflammation in lipopolysaccharide-stimulated BV2 cells. Exp Ther Med. 2021;21(3):219doi: 10.3892/etm.2021.9621.
Rui, Q., Cao, S., Wang, X., Duan, X., Iao, X., Dong, W., Fang, Q., Zhang, X., & Xue, Q. (2021). LMTK2 regulates inflammation in lipopolysaccharide-stimulated BV2 cells. Experimental and therapeutic medicine, 21(3), 219. https://doi.org/10.3892/etm.2021.9621
Rui, Qianyun, et al. "LMTK2 regulates inflammation in lipopolysaccharide-stimulated BV2 cells." Experimental and therapeutic medicine vol. 21,3 (2021): 219. doi: https://doi.org/10.3892/etm.2021.9621
Rui Q, Cao S, Wang X, Duan X, Iao X, Dong W, Fang Q, Zhang X, Xue Q. LMTK2 regulates inflammation in lipopolysaccharide-stimulated BV2 cells. Exp Ther Med. 2021 Mar;21(3):219. doi: 10.3892/etm.2021.9621. Epub 2021 Jan 18. PMID: 33603828; PMCID: PMC7851617.
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Indoxyl Sulfate Affects Glial Function Increasing Oxidative Stress and Neuroinflammation in Chronic Kidney Disease: Interaction between Astrocytes and Microglia.

Frontiers in pharmacology

Adesso S, Magnus T, Cuzzocrea S, Campolo M, Rissiek B, Paciello O, Autore G, Pinto A, Marzocco S.
PMID: 28659803
Front Pharmacol. 2017 Jun 12;8:370. doi: 10.3389/fphar.2017.00370. eCollection 2017.

Indoxyl sulfate (IS) is a protein-bound uremic toxin resulting from the metabolism of dietary tryptophan which accumulates in patients with impaired renal function, such as chronic kidney disease (CKD). IS is a well-known nephrovascular toxin but little is known...

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Adesso S, Magnus T, Cuzzocrea S, et al. Indoxyl Sulfate Affects Glial Function Increasing Oxidative Stress and Neuroinflammation in Chronic Kidney Disease: Interaction between Astrocytes and Microglia. Front Pharmacol. 2017;8:370doi: 10.3389/fphar.2017.00370.
Adesso, S., Magnus, T., Cuzzocrea, S., Campolo, M., Rissiek, B., Paciello, O., Autore, G., Pinto, A., & Marzocco, S. (2017). Indoxyl Sulfate Affects Glial Function Increasing Oxidative Stress and Neuroinflammation in Chronic Kidney Disease: Interaction between Astrocytes and Microglia. Frontiers in pharmacology, 8370. https://doi.org/10.3389/fphar.2017.00370
Adesso, Simona, et al. "Indoxyl Sulfate Affects Glial Function Increasing Oxidative Stress and Neuroinflammation in Chronic Kidney Disease: Interaction between Astrocytes and Microglia." Frontiers in pharmacology vol. 8 (2017): 370. doi: https://doi.org/10.3389/fphar.2017.00370
Adesso S, Magnus T, Cuzzocrea S, Campolo M, Rissiek B, Paciello O, Autore G, Pinto A, Marzocco S. Indoxyl Sulfate Affects Glial Function Increasing Oxidative Stress and Neuroinflammation in Chronic Kidney Disease: Interaction between Astrocytes and Microglia. Front Pharmacol. 2017 Jun 12;8:370. doi: 10.3389/fphar.2017.00370. eCollection 2017. PMID: 28659803; PMCID: PMC5466960.
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A small compound spindlactone A sensitizes human endometrial cancer cells to TRAIL-induced apoptosis via the inhibition of NAD(P)H dehydrogenase quinone 1.

OncoTargets and therapy

Zhao XZ, Wu XH.
PMID: 29950865
Onco Targets Ther. 2018 Jun 21;11:3609-3617. doi: 10.2147/OTT.S165723. eCollection 2018.

INTRODUCTION: Spindlactone A (SPL-A) is a novel small molecule inhibitor of TACC3 that selectively inhibits the nucleation of centrosome microtubules and induces mitotic arrest in ovarian cancer cells. SPL-A is derived from dicoumarol which inhibits the activity of NAD(P)H...

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Zhao XZ, Wu XH. A small compound spindlactone A sensitizes human endometrial cancer cells to TRAIL-induced apoptosis via the inhibition of NAD(P)H dehydrogenase quinone 1. Onco Targets Ther. 2018;11:3609-3617doi: 10.2147/OTT.S165723.
Zhao, X. Z., & Wu, X. H. (2018). A small compound spindlactone A sensitizes human endometrial cancer cells to TRAIL-induced apoptosis via the inhibition of NAD(P)H dehydrogenase quinone 1. OncoTargets and therapy, 113609-3617. https://doi.org/10.2147/OTT.S165723
Zhao, Xiang-Zhai, and Wu, Xiao-Hua. "A small compound spindlactone A sensitizes human endometrial cancer cells to TRAIL-induced apoptosis via the inhibition of NAD(P)H dehydrogenase quinone 1." OncoTargets and therapy vol. 11 (2018): 3609-3617. doi: https://doi.org/10.2147/OTT.S165723
Zhao XZ, Wu XH. A small compound spindlactone A sensitizes human endometrial cancer cells to TRAIL-induced apoptosis via the inhibition of NAD(P)H dehydrogenase quinone 1. Onco Targets Ther. 2018 Jun 21;11:3609-3617. doi: 10.2147/OTT.S165723. eCollection 2018. PMID: 29950865; PMCID: PMC6016593.
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Mechanism of circadian regulation of the NRF2/ARE pathway in renal ischemia-reperfusion.

Experimental and therapeutic medicine

Sun Q, Zeng C, Du L, Dong C.
PMID: 33488799
Exp Ther Med. 2021 Mar;21(3):190. doi: 10.3892/etm.2021.9622. Epub 2021 Jan 07.

The nuclear erythroid 2-related factor 2 (NRF2)/antioxidant response element (ARE) pathway has been shown to provide strong protection against oxidative stress injury induced by renal ischemia-reperfusion (IR). However, the endogenous regulatory mechanism of the NRF2/ARE pathway in renal IR...

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Sun Q, Zeng C, Du L, et al. Mechanism of circadian regulation of the NRF2/ARE pathway in renal ischemia-reperfusion. Exp Ther Med. 2021;21(3):190doi: 10.3892/etm.2021.9622.
Sun, Q., Zeng, C., Du, L., & Dong, C. (2021). Mechanism of circadian regulation of the NRF2/ARE pathway in renal ischemia-reperfusion. Experimental and therapeutic medicine, 21(3), 190. https://doi.org/10.3892/etm.2021.9622
Sun, Qian, et al. "Mechanism of circadian regulation of the NRF2/ARE pathway in renal ischemia-reperfusion." Experimental and therapeutic medicine vol. 21,3 (2021): 190. doi: https://doi.org/10.3892/etm.2021.9622
Sun Q, Zeng C, Du L, Dong C. Mechanism of circadian regulation of the NRF2/ARE pathway in renal ischemia-reperfusion. Exp Ther Med. 2021 Mar;21(3):190. doi: 10.3892/etm.2021.9622. Epub 2021 Jan 07. PMID: 33488799; PMCID: PMC7812573.
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CYP1B1 deiciency ameliorates learning and memory deficits caused by high fat diet in mice.

American journal of translational research

Yang Z, Li H, Tang Y, Liu X, Liao Q, Fan C, Wang S.
PMID: 31105828
Am J Transl Res. 2019 Apr 15;11(4):2194-2206. eCollection 2019.

Our previous study showed that Cyp1b1 deficiency could protect male mice from high fat diet (HFD) obesity. In this paper, we aim to explore the role of Cyp1b1 in HFD induced learning and memory deficits in female C57BL/6J mice.METHODS:...

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Yang Z, Li H, Tang Y, et al. CYP1B1 deiciency ameliorates learning and memory deficits caused by high fat diet in mice. Am J Transl Res. 2019;11(4):2194-2206
Yang, Z., Li, H., Tang, Y., Liu, X., Liao, Q., Fan, C., & Wang, S. (2019). CYP1B1 deiciency ameliorates learning and memory deficits caused by high fat diet in mice. American journal of translational research, 11(4), 2194-2206.
Yang, Zhuanhong, et al. "CYP1B1 deiciency ameliorates learning and memory deficits caused by high fat diet in mice." American journal of translational research vol. 11,4 (2019): 2194-2206.
Yang Z, Li H, Tang Y, Liu X, Liao Q, Fan C, Wang S. CYP1B1 deiciency ameliorates learning and memory deficits caused by high fat diet in mice. Am J Transl Res. 2019 Apr 15;11(4):2194-2206. eCollection 2019. PMID: 31105828; PMCID: PMC6511759.
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Gene Expression Profile of the Hypothalamus in DNP-KLH Immunized Mice Following Electroacupuncture Stimulation.

Evidence-based complementary and alternative medicine : eCAM

Kim SK, Kim J, Ko E, Kim H, Hwang DS, Lee S, Baek Y, Min BI, Nam S, Bae H.
PMID: 21799680
Evid Based Complement Alternat Med. 2011;2011:508689. doi: 10.1093/ecam/nep222. Epub 2011 Jun 05.

Clinical evidence indicates that electroacupuncture (EA) is effective for allergic disorder. Recent animal studies have shown that EA treatment reduces levels of IgE and Th2 cytokines in BALB/c mice immunized with 2,4-dinitrophenylated keyhole limpet protein (DNP-KLH). The hypothalamus, a...

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Kim SK, Kim J, Ko E, et al. Gene Expression Profile of the Hypothalamus in DNP-KLH Immunized Mice Following Electroacupuncture Stimulation. Evid Based Complement Alternat Med. 2011;2011:508689doi: 10.1093/ecam/nep222.
Kim, S. K., Kim, J., Ko, E., Kim, H., Hwang, D. S., Lee, S., Baek, Y., Min, B. I., Nam, S., & Bae, H. (2011). Gene Expression Profile of the Hypothalamus in DNP-KLH Immunized Mice Following Electroacupuncture Stimulation. Evidence-based complementary and alternative medicine : eCAM, 2011508689. https://doi.org/10.1093/ecam/nep222
Kim, Sun Kwang, et al. "Gene Expression Profile of the Hypothalamus in DNP-KLH Immunized Mice Following Electroacupuncture Stimulation." Evidence-based complementary and alternative medicine : eCAM vol. 2011 (2011): 508689. doi: https://doi.org/10.1093/ecam/nep222
Kim SK, Kim J, Ko E, Kim H, Hwang DS, Lee S, Baek Y, Min BI, Nam S, Bae H. Gene Expression Profile of the Hypothalamus in DNP-KLH Immunized Mice Following Electroacupuncture Stimulation. Evid Based Complement Alternat Med. 2011;2011:508689. doi: 10.1093/ecam/nep222. Epub 2011 Jun 05. PMID: 21799680; PMCID: PMC3136536.
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