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High TBX2 expression predicts poor prognosis in non-small cell lung cancer.

Neoplasma

Zhang Z, Guo Y.
PMID: 24712844
Neoplasma. 2014 Apr 09; doi: 10.4149/neo_2014_059. Epub 2014 Apr 09.

Deregulated expression of T-box2 (TBX2) has been implicated in malignancies. However, the expression pattern and the role of TBX2 in non-small cell lung cancer (NSCLC) remain unclear. The aim of this study was to explore the clinical value of...

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Zhang Z, Guo Y. High TBX2 expression predicts poor prognosis in non-small cell lung cancer. Neoplasma. 2014;doi: 10.4149/neo_2014_059.
Zhang, Z., & Guo, Y. (2014). High TBX2 expression predicts poor prognosis in non-small cell lung cancer. Neoplasma, . https://doi.org/10.4149/neo_2014_059
Zhang, Z, and Guo, Y. "High TBX2 expression predicts poor prognosis in non-small cell lung cancer." Neoplasma vol. (2014). doi: https://doi.org/10.4149/neo_2014_059
Zhang Z, Guo Y. High TBX2 expression predicts poor prognosis in non-small cell lung cancer. Neoplasma. 2014 Apr 09; doi: 10.4149/neo_2014_059. Epub 2014 Apr 09. PMID: 24712844.
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Receptor-interacting protein kinase 3 is a predictor of survival and plays a tumor suppressive role in colorectal cancer.

Neoplasma

Feng X, Song Q, Yu A, Tang H, Peng Z, Wang X.
PMID: 25997957
Neoplasma. 2015;62(4):592-601. doi: 10.4149/neo_2015_071.

Receptor-interacting protein kinase 3 (RIP3) is a member of the RIP Ser/Thr kinase family, plays an important role in regulating cell survival, cell apoptosis and cell necrosis. However, the role of RIP3 in the carcinogenesis of colorectal cancer is...

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Feng X, Song Q, Yu A, et al. Receptor-interacting protein kinase 3 is a predictor of survival and plays a tumor suppressive role in colorectal cancer. Neoplasma. 2015;62(4):592-601doi: 10.4149/neo_2015_071.
Feng, X., Song, Q., Yu, A., Tang, H., Peng, Z., & Wang, X. (2015). Receptor-interacting protein kinase 3 is a predictor of survival and plays a tumor suppressive role in colorectal cancer. Neoplasma, 62(4), 592-601. https://doi.org/10.4149/neo_2015_071
Feng, X, et al. "Receptor-interacting protein kinase 3 is a predictor of survival and plays a tumor suppressive role in colorectal cancer." Neoplasma vol. 62,4 (2015): 592-601. doi: https://doi.org/10.4149/neo_2015_071
Feng X, Song Q, Yu A, Tang H, Peng Z, Wang X. Receptor-interacting protein kinase 3 is a predictor of survival and plays a tumor suppressive role in colorectal cancer. Neoplasma. 2015;62(4):592-601. doi: 10.4149/neo_2015_071. PMID: 25997957.
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MR-mammography - impact on disease extent determination and surgical treatment of invasive ductal and lobular breast cancers.

Neoplasma

Lehotska V, Rauova K, Vanovcanova L.
PMID: 25591592
Neoplasma. 2015;62(2):269-77. doi: 10.4149/neo_2015_032.

UNLABELLED: The aim of this study is to assess the impact of multiparametric MR imaging to an exact determination of the extent of invasive ductal and lobular breast cancers in routine clinical practice.180 women aged 27-74 years (median 52.4...

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Lehotska V, Rauova K, Vanovcanova L. MR-mammography - impact on disease extent determination and surgical treatment of invasive ductal and lobular breast cancers. Neoplasma. 2015;62(2):269-77doi: 10.4149/neo_2015_032.
Lehotska, V., Rauova, K., & Vanovcanova, L. (2015). MR-mammography - impact on disease extent determination and surgical treatment of invasive ductal and lobular breast cancers. Neoplasma, 62(2), 269-77. https://doi.org/10.4149/neo_2015_032
Lehotska, V, et al. "MR-mammography - impact on disease extent determination and surgical treatment of invasive ductal and lobular breast cancers." Neoplasma vol. 62,2 (2015): 269-77. doi: https://doi.org/10.4149/neo_2015_032
Lehotska V, Rauova K, Vanovcanova L. MR-mammography - impact on disease extent determination and surgical treatment of invasive ductal and lobular breast cancers. Neoplasma. 2015;62(2):269-77. doi: 10.4149/neo_2015_032. PMID: 25591592.
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Pre- and post-operative evaluation: percentages of circulating myeloid-derived suppressor cells in rectal cancer patients.

Neoplasma

Yuan L, Xu B, Fan H, Yuan P, Zhao P, Suo Z.
PMID: 25591589
Neoplasma. 2015;62(2):239-49. doi: 10.4149/neo_2015_029.

UNLABELLED: This study aims to explore whether pre-operative or post-operative percentages of circulating myeloid-derived suppressor cells (MDSC) have any correlations with clinicopathological parameters in patients with rectal cancer and have any predictive values for local recurrence and disease free...

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Yuan L, Xu B, Fan H, et al. Pre- and post-operative evaluation: percentages of circulating myeloid-derived suppressor cells in rectal cancer patients. Neoplasma. 2015;62(2):239-49doi: 10.4149/neo_2015_029.
Yuan, L., Xu, B., Fan, H., Yuan, P., Zhao, P., & Suo, Z. (2015). Pre- and post-operative evaluation: percentages of circulating myeloid-derived suppressor cells in rectal cancer patients. Neoplasma, 62(2), 239-49. https://doi.org/10.4149/neo_2015_029
Yuan, L, et al. "Pre- and post-operative evaluation: percentages of circulating myeloid-derived suppressor cells in rectal cancer patients." Neoplasma vol. 62,2 (2015): 239-49. doi: https://doi.org/10.4149/neo_2015_029
Yuan L, Xu B, Fan H, Yuan P, Zhao P, Suo Z. Pre- and post-operative evaluation: percentages of circulating myeloid-derived suppressor cells in rectal cancer patients. Neoplasma. 2015;62(2):239-49. doi: 10.4149/neo_2015_029. PMID: 25591589.
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Sorafenib-irinotecan sequential therapy augmented the anti-tumor efficacy of monotherapy in hepatocellular carcinoma cells HepG2.

Neoplasma

Wang Z, Zhao Z, Wu T, Song L, Zhang Y.
PMID: 25738310
Neoplasma. 2015;62(2):172-9. doi: 10.4149/neo_2015_022.

UNLABELLED: The current study aimed to evaluate the efficacy of sorafenib-based combined therapy against hepatocellular carcinoma (HCC). HepG2 cells were exposed to sorafenib, irinotecan, and oxaliplatin and then subjected to MTT assay to determine chemosensitivity. Flow cytometry was used...

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Wang Z, Zhao Z, Wu T, et al. Sorafenib-irinotecan sequential therapy augmented the anti-tumor efficacy of monotherapy in hepatocellular carcinoma cells HepG2. Neoplasma. 2015;62(2):172-9doi: 10.4149/neo_2015_022.
Wang, Z., Zhao, Z., Wu, T., Song, L., & Zhang, Y. (2015). Sorafenib-irinotecan sequential therapy augmented the anti-tumor efficacy of monotherapy in hepatocellular carcinoma cells HepG2. Neoplasma, 62(2), 172-9. https://doi.org/10.4149/neo_2015_022
Wang, Z, et al. "Sorafenib-irinotecan sequential therapy augmented the anti-tumor efficacy of monotherapy in hepatocellular carcinoma cells HepG2." Neoplasma vol. 62,2 (2015): 172-9. doi: https://doi.org/10.4149/neo_2015_022
Wang Z, Zhao Z, Wu T, Song L, Zhang Y. Sorafenib-irinotecan sequential therapy augmented the anti-tumor efficacy of monotherapy in hepatocellular carcinoma cells HepG2. Neoplasma. 2015;62(2):172-9. doi: 10.4149/neo_2015_022. PMID: 25738310.
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Changes in gene expression of CXCR4, CCR7 and BCL2 after treatment of breast cancer cells with saponin extract from Tribulus terrestris.

Neoplasma

Goranova TE, Bozhanov SS, Lozanov VS, Mitev VI, Kaneva RP, Georgieva EI.
PMID: 25563364
Neoplasma. 2015;62(1):27-33. doi: 10.4149/neo_2015_004.

UNLABELLED: Saponins are natural substances produced by a large number of plants, one of which is Tribulus terrestris L. (TT). They have been reported to possess an antitumor activity exerted by regulating various signaling pathways in the cell. Although...

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Goranova TE, Bozhanov SS, Lozanov VS, et al. Changes in gene expression of CXCR4, CCR7 and BCL2 after treatment of breast cancer cells with saponin extract from Tribulus terrestris. Neoplasma. 2015;62(1):27-33doi: 10.4149/neo_2015_004.
Goranova, T. E., Bozhanov, S. S., Lozanov, V. S., Mitev, V. I., Kaneva, R. P., & Georgieva, E. I. (2015). Changes in gene expression of CXCR4, CCR7 and BCL2 after treatment of breast cancer cells with saponin extract from Tribulus terrestris. Neoplasma, 62(1), 27-33. https://doi.org/10.4149/neo_2015_004
Goranova, T E, et al. "Changes in gene expression of CXCR4, CCR7 and BCL2 after treatment of breast cancer cells with saponin extract from Tribulus terrestris." Neoplasma vol. 62,1 (2015): 27-33. doi: https://doi.org/10.4149/neo_2015_004
Goranova TE, Bozhanov SS, Lozanov VS, Mitev VI, Kaneva RP, Georgieva EI. Changes in gene expression of CXCR4, CCR7 and BCL2 after treatment of breast cancer cells with saponin extract from Tribulus terrestris. Neoplasma. 2015;62(1):27-33. doi: 10.4149/neo_2015_004. PMID: 25563364.
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Correlation of carbonic anhydrase IX expression with clinico-morphological parameters, hormonal receptor status and HER-2 expression in breast cancer.

Neoplasma

Furjelova M, Kovalska M, Jurkova K, Chylikova J, Mestanova V, Adamkov M.
PMID: 25563371
Neoplasma. 2015;62(1):88-97. doi: 10.4149/neo_2015_011.

UNLABELLED: The hypoxia-inducible protein carbonic anhydrase IX is widely expressed in most human cancers, including breast carcinomas. CA IX attracts significant interest due to its strong association with neoplasms and its absence from corresponding normal tissues, suggesting its potential...

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Furjelova M, Kovalska M, Jurkova K, et al. Correlation of carbonic anhydrase IX expression with clinico-morphological parameters, hormonal receptor status and HER-2 expression in breast cancer. Neoplasma. 2015;62(1):88-97doi: 10.4149/neo_2015_011.
Furjelova, M., Kovalska, M., Jurkova, K., Chylikova, J., Mestanova, V., & Adamkov, M. (2015). Correlation of carbonic anhydrase IX expression with clinico-morphological parameters, hormonal receptor status and HER-2 expression in breast cancer. Neoplasma, 62(1), 88-97. https://doi.org/10.4149/neo_2015_011
Furjelova, M, et al. "Correlation of carbonic anhydrase IX expression with clinico-morphological parameters, hormonal receptor status and HER-2 expression in breast cancer." Neoplasma vol. 62,1 (2015): 88-97. doi: https://doi.org/10.4149/neo_2015_011
Furjelova M, Kovalska M, Jurkova K, Chylikova J, Mestanova V, Adamkov M. Correlation of carbonic anhydrase IX expression with clinico-morphological parameters, hormonal receptor status and HER-2 expression in breast cancer. Neoplasma. 2015;62(1):88-97. doi: 10.4149/neo_2015_011. PMID: 25563371.
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Overexpression of Survivin mutant Thr34Ala induces apoptosis and inhibits gastric cancer growth.

Neoplasma

Dang SC, Feng S, Wang PJ, Cui L, Qu JG, Zhang JX.
PMID: 25563370
Neoplasma. 2015;62(1):81-7. doi: 10.4149/neo_2015_010.

UNLABELLED: Survivin is highly expressed in human gastric cancer and correlated with chemoresistance and poor prognosis. In this study, we explored the effect of adeno-associated virus (AAV)-mediated survivin dominant mutant Thr34Ala (rAAV-Sur-Mut(T34A)) on gastric cancer growth. AAV-Sur-Mut (T34A) virus...

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Dang SC, Feng S, Wang PJ, et al. Overexpression of Survivin mutant Thr34Ala induces apoptosis and inhibits gastric cancer growth. Neoplasma. 2015;62(1):81-7doi: 10.4149/neo_2015_010.
Dang, S. C., Feng, S., Wang, P. J., Cui, L., Qu, J. G., & Zhang, J. X. (2015). Overexpression of Survivin mutant Thr34Ala induces apoptosis and inhibits gastric cancer growth. Neoplasma, 62(1), 81-7. https://doi.org/10.4149/neo_2015_010
Dang, S C, et al. "Overexpression of Survivin mutant Thr34Ala induces apoptosis and inhibits gastric cancer growth." Neoplasma vol. 62,1 (2015): 81-7. doi: https://doi.org/10.4149/neo_2015_010
Dang SC, Feng S, Wang PJ, Cui L, Qu JG, Zhang JX. Overexpression of Survivin mutant Thr34Ala induces apoptosis and inhibits gastric cancer growth. Neoplasma. 2015;62(1):81-7. doi: 10.4149/neo_2015_010. PMID: 25563370.
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Antioxidant defense system in prostate adenocarcinoma and benign prostate hyperplasia of elderly patients.

Neoplasma

Szewczyk-Golec K, Tyloch J, Czuczejko J.
PMID: 25563375
Neoplasma. 2015;62(1):119-23. doi: 10.4149/neo_2015_015.

UNLABELLED: Prostate adenocarcinoma (PC) and benign prostate hyperplasia (BPH) are age-related diseases. The augmented oxidative stress is suggested to play an important role in the pathogenesis of both mentioned prostate disorders. In the presented study the antioxidant defense system...

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Szewczyk-Golec K, Tyloch J, Czuczejko J. Antioxidant defense system in prostate adenocarcinoma and benign prostate hyperplasia of elderly patients. Neoplasma. 2015;62(1):119-23doi: 10.4149/neo_2015_015.
Szewczyk-Golec, K., Tyloch, J., & Czuczejko, J. (2015). Antioxidant defense system in prostate adenocarcinoma and benign prostate hyperplasia of elderly patients. Neoplasma, 62(1), 119-23. https://doi.org/10.4149/neo_2015_015
Szewczyk-Golec, K, et al. "Antioxidant defense system in prostate adenocarcinoma and benign prostate hyperplasia of elderly patients." Neoplasma vol. 62,1 (2015): 119-23. doi: https://doi.org/10.4149/neo_2015_015
Szewczyk-Golec K, Tyloch J, Czuczejko J. Antioxidant defense system in prostate adenocarcinoma and benign prostate hyperplasia of elderly patients. Neoplasma. 2015;62(1):119-23. doi: 10.4149/neo_2015_015. PMID: 25563375.
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Allogeneic stem cell transplantation can improve outcome of AML patients without complete cytogenetic response after induction and consolidation treatment.

Neoplasma

Dvorak P, Lysak D, Vokurka S, Karas M, Subrt I.
PMID: 25563378
Neoplasma. 2015;62(1):140-5. doi: 10.4149/neo_2015_018.

UNLABELLED: Our retrospective analysis was performed on 376 consecutive patients diagnosed with AML. A total of 256 (68%) were treated with standard "7+3" induction and high-dose cytarabine and mitoxantrone containing "4+3" consolidation/intensification regimens. Our study focused on patients with...

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Dvorak P, Lysak D, Vokurka S, et al. Allogeneic stem cell transplantation can improve outcome of AML patients without complete cytogenetic response after induction and consolidation treatment. Neoplasma. 2015;62(1):140-5doi: 10.4149/neo_2015_018.
Dvorak, P., Lysak, D., Vokurka, S., Karas, M., & Subrt, I. (2015). Allogeneic stem cell transplantation can improve outcome of AML patients without complete cytogenetic response after induction and consolidation treatment. Neoplasma, 62(1), 140-5. https://doi.org/10.4149/neo_2015_018
Dvorak, P, et al. "Allogeneic stem cell transplantation can improve outcome of AML patients without complete cytogenetic response after induction and consolidation treatment." Neoplasma vol. 62,1 (2015): 140-5. doi: https://doi.org/10.4149/neo_2015_018
Dvorak P, Lysak D, Vokurka S, Karas M, Subrt I. Allogeneic stem cell transplantation can improve outcome of AML patients without complete cytogenetic response after induction and consolidation treatment. Neoplasma. 2015;62(1):140-5. doi: 10.4149/neo_2015_018. PMID: 25563378.
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The impact of Caspase-8 on non-small cell lung cancer brain metastasis in II/III stage patient.

Neoplasma

Liao Y, Yang F, Li X, Chen K, Zhou L, Wang Y, Wang J.
PMID: 25866216
Neoplasma. 2015 Apr 13; doi: 10.4149/neo_2015_043. Epub 2015 Apr 13.

The aim of this study is to evaluate the role of Caspase-8 in predicting non-small cell lung cancer (NSCLC) metastasis to brain. The expression of Caspase-8 was analyzed by immunohistochemistry in 203 surgical resected NSCLC patients, who were followed...

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Liao Y, Yang F, Li X, et al. The impact of Caspase-8 on non-small cell lung cancer brain metastasis in II/III stage patient. Neoplasma. 2015;doi: 10.4149/neo_2015_043.
Liao, Y., Yang, F., Li, X., Chen, K., Zhou, L., Wang, Y., & Wang, J. (2015). The impact of Caspase-8 on non-small cell lung cancer brain metastasis in II/III stage patient. Neoplasma, . https://doi.org/10.4149/neo_2015_043
Liao, Y, et al. "The impact of Caspase-8 on non-small cell lung cancer brain metastasis in II/III stage patient." Neoplasma vol. (2015). doi: https://doi.org/10.4149/neo_2015_043
Liao Y, Yang F, Li X, Chen K, Zhou L, Wang Y, Wang J. The impact of Caspase-8 on non-small cell lung cancer brain metastasis in II/III stage patient. Neoplasma. 2015 Apr 13; doi: 10.4149/neo_2015_043. Epub 2015 Apr 13. PMID: 25866216.
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17-AAG enhances the cytotoxicity of flavopiridol in mantle cell lymphoma via autophagy suppression.

Neoplasma

Xiao Y, Guan J.
PMID: 25866220
Neoplasma. 2015;62(3):391-7. doi: 10.4149/neo_2015_047.

Flavopiridol, a cyclin-dependent kinase inhibitor (CDKI), shows promising anti-tumor activity in hematologic malignancies. However, Flavopiridol-induced protective autophagy may lead to drug resistance. Here we found that Hsp90 inhibitor 17-AAG can sensitize mantle cell lymphoma (MCL) cells to flavopiridol by...

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Xiao Y, Guan J. 17-AAG enhances the cytotoxicity of flavopiridol in mantle cell lymphoma via autophagy suppression. Neoplasma. 2015;62(3):391-7doi: 10.4149/neo_2015_047.
Xiao, Y., & Guan, J. (2015). 17-AAG enhances the cytotoxicity of flavopiridol in mantle cell lymphoma via autophagy suppression. Neoplasma, 62(3), 391-7. https://doi.org/10.4149/neo_2015_047
Xiao, Y, and Guan, J. "17-AAG enhances the cytotoxicity of flavopiridol in mantle cell lymphoma via autophagy suppression." Neoplasma vol. 62,3 (2015): 391-7. doi: https://doi.org/10.4149/neo_2015_047
Xiao Y, Guan J. 17-AAG enhances the cytotoxicity of flavopiridol in mantle cell lymphoma via autophagy suppression. Neoplasma. 2015;62(3):391-7. doi: 10.4149/neo_2015_047. PMID: 25866220.
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Showing 1 to 12 of 272 entries