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Showing 1 to 12 of 235 entries
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Cannabinoid CB1 Receptors Mediate the Gastroprotective Effect of Neurotensin.

Iranian journal of basic medical sciences

Hassanzadeh P, Arbabi E.
PMID: 23492756
Iran J Basic Med Sci. 2012 May;15(3):803-10.

OBJECTIVES: Several lines of evidence indicate that neuropeptides exhibit protective properties against gastroduodenal ulcers. Neurotensin, a gut-brain neuropeptide, is implicated in a number of physiological processes in the central nervous system and peripheral tissues including gastrointestinal tract. In the...

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Hassanzadeh P, Arbabi E. Cannabinoid CB1 Receptors Mediate the Gastroprotective Effect of Neurotensin. Iran J Basic Med Sci. 2012;15(3):803-10
Hassanzadeh, P., & Arbabi, E. (2012). Cannabinoid CB1 Receptors Mediate the Gastroprotective Effect of Neurotensin. Iranian journal of basic medical sciences, 15(3), 803-10.
Hassanzadeh, Parichehr, and Arbabi, Elham. "Cannabinoid CB1 Receptors Mediate the Gastroprotective Effect of Neurotensin." Iranian journal of basic medical sciences vol. 15,3 (2012): 803-10.
Hassanzadeh P, Arbabi E. Cannabinoid CB1 Receptors Mediate the Gastroprotective Effect of Neurotensin. Iran J Basic Med Sci. 2012 May;15(3):803-10. PMID: 23492756; PMCID: PMC3586888.
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ProNGF derived from rat sciatic nerves downregulates neurite elongation and axon specification in PC12 cells.

Frontiers in cellular neuroscience

Trigos AS, Longart M, García L, Castillo C, Forsyth P, Medina R.
PMID: 26441535
Front Cell Neurosci. 2015 Sep 15;9:364. doi: 10.3389/fncel.2015.00364. eCollection 2015.

Several reports have shown that a sciatic nerve conditioned media (CM) causes neuronal-like differentiation in PC12 cells. This differentiation is featured by neurite outgrowth, which are exclusively dendrites, without axon or sodium current induction. In previous studies, our group...

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Trigos AS, Longart M, García L, et al. ProNGF derived from rat sciatic nerves downregulates neurite elongation and axon specification in PC12 cells. Front Cell Neurosci. 2015;9:364doi: 10.3389/fncel.2015.00364.
Trigos, A. S., Longart, M., García, L., Castillo, C., Forsyth, P., & Medina, R. (2015). ProNGF derived from rat sciatic nerves downregulates neurite elongation and axon specification in PC12 cells. Frontiers in cellular neuroscience, 9364. https://doi.org/10.3389/fncel.2015.00364
Trigos, Anna Sofía, et al. "ProNGF derived from rat sciatic nerves downregulates neurite elongation and axon specification in PC12 cells." Frontiers in cellular neuroscience vol. 9 (2015): 364. doi: https://doi.org/10.3389/fncel.2015.00364
Trigos AS, Longart M, García L, Castillo C, Forsyth P, Medina R. ProNGF derived from rat sciatic nerves downregulates neurite elongation and axon specification in PC12 cells. Front Cell Neurosci. 2015 Sep 15;9:364. doi: 10.3389/fncel.2015.00364. eCollection 2015. PMID: 26441535; PMCID: PMC4569732.
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Interferon beta (IFN-β) treatment exerts potential neuroprotective effects through neurotrophic factors and novel neurotensin/neurotensin high affinity receptor 1 pathway.

Neural regeneration research

Wang Q, Mao-Draayer Y.
PMID: 26889174
Neural Regen Res. 2015 Dec;10(12):1932-3. doi: 10.4103/1673-5374.169636.

No abstract available.

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Wang Q, Mao-Draayer Y. Interferon beta (IFN-β) treatment exerts potential neuroprotective effects through neurotrophic factors and novel neurotensin/neurotensin high affinity receptor 1 pathway. Neural Regen Res. 2015;10(12):1932-3doi: 10.4103/1673-5374.169636.
Wang, Q., & Mao-Draayer, Y. (2015). Interferon beta (IFN-β) treatment exerts potential neuroprotective effects through neurotrophic factors and novel neurotensin/neurotensin high affinity receptor 1 pathway. Neural regeneration research, 10(12), 1932-3. https://doi.org/10.4103/1673-5374.169636
Wang, Qin, and Mao-Draayer, Yang. "Interferon beta (IFN-β) treatment exerts potential neuroprotective effects through neurotrophic factors and novel neurotensin/neurotensin high affinity receptor 1 pathway." Neural regeneration research vol. 10,12 (2015): 1932-3. doi: https://doi.org/10.4103/1673-5374.169636
Wang Q, Mao-Draayer Y. Interferon beta (IFN-β) treatment exerts potential neuroprotective effects through neurotrophic factors and novel neurotensin/neurotensin high affinity receptor 1 pathway. Neural Regen Res. 2015 Dec;10(12):1932-3. doi: 10.4103/1673-5374.169636. PMID: 26889174; PMCID: PMC4730810.
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Analysis of spatial and temporal protein expression in the cerebral cortex after ischemia-reperfusion injury.

Journal of clinical neurology (Seoul, Korea)

Chen YH, Chiang YH, Ma HI.
PMID: 24829593
J Clin Neurol. 2014 Apr;10(2):84-93. doi: 10.3988/jcn.2014.10.2.84. Epub 2014 Apr 23.

BACKGROUND AND PURPOSE: Hypoxia, or ischemia, is a common cause of neurological deficits in the elderly. This study elucidated the mechanisms underlying ischemia-induced brain injury that results in neurological sequelae.METHODS: Cerebral ischemia was induced in male Sprague-Dawley rats by...

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Chen YH, Chiang YH, Ma HI. Analysis of spatial and temporal protein expression in the cerebral cortex after ischemia-reperfusion injury. J Clin Neurol. 2014;10(2):84-93doi: 10.3988/jcn.2014.10.2.84.
Chen, Y. H., Chiang, Y. H., & Ma, H. I. (2014). Analysis of spatial and temporal protein expression in the cerebral cortex after ischemia-reperfusion injury. Journal of clinical neurology (Seoul, Korea), 10(2), 84-93. https://doi.org/10.3988/jcn.2014.10.2.84
Chen, Yuan-Hao, et al. "Analysis of spatial and temporal protein expression in the cerebral cortex after ischemia-reperfusion injury." Journal of clinical neurology (Seoul, Korea) vol. 10,2 (2014): 84-93. doi: https://doi.org/10.3988/jcn.2014.10.2.84
Chen YH, Chiang YH, Ma HI. Analysis of spatial and temporal protein expression in the cerebral cortex after ischemia-reperfusion injury. J Clin Neurol. 2014 Apr;10(2):84-93. doi: 10.3988/jcn.2014.10.2.84. Epub 2014 Apr 23. PMID: 24829593; PMCID: PMC4017024.
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Hormonal response to enteral feeding and the possible role of peptide YY in pathogenesis of enteral feeding-related diarrhoea.

Clinical nutrition (Edinburgh, Scotland)

Bowling TE, Silk DB.
PMID: 16844062
Clin Nutr. 1996 Dec;15(6):307-10. doi: 10.1016/s0261-5614(96)80005-7.

Diarrhoea is a common complication of enteral feeding. Previous studies have demonstrated a secretion of water and electrolytes in the ascending colon during intragastric but not intraduodenal enteral feeding. The cause of this secretion is likely to be neurohumoral...

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Bowling TE, Silk DB. Hormonal response to enteral feeding and the possible role of peptide YY in pathogenesis of enteral feeding-related diarrhoea. Clin Nutr. 1996;15(6):307-10doi: 10.1016/s0261-5614(96)80005-7.
Bowling, T. E., & Silk, D. B. (1996). Hormonal response to enteral feeding and the possible role of peptide YY in pathogenesis of enteral feeding-related diarrhoea. Clinical nutrition (Edinburgh, Scotland), 15(6), 307-10. https://doi.org/10.1016/s0261-5614(96)80005-7
Bowling, T E, and Silk, D B. "Hormonal response to enteral feeding and the possible role of peptide YY in pathogenesis of enteral feeding-related diarrhoea." Clinical nutrition (Edinburgh, Scotland) vol. 15,6 (1996): 307-10. doi: https://doi.org/10.1016/s0261-5614(96)80005-7
Bowling TE, Silk DB. Hormonal response to enteral feeding and the possible role of peptide YY in pathogenesis of enteral feeding-related diarrhoea. Clin Nutr. 1996 Dec;15(6):307-10. doi: 10.1016/s0261-5614(96)80005-7. PMID: 16844062.
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Preoptic unit responses to microelectrophoresis of neurotensin and its antiserum in female rats.

Journal of neuroendocrinology

Akema T, Kimuraf F.
PMID: 19210376
J Neuroendocrinol. 1990 Apr 01;2(2):141-3. doi: 10.1111/j.1365-2826.1990.tb00843.x.

Abstract We and other investigators have demonstrated the stimulation of luteinizing hormone secretion following the microinjection of neurotensin (NT) into the medial preoptic area in female rats. The present study electrophysiologically examined the action of the peptide in the...

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Akema T, Kimuraf F. Preoptic unit responses to microelectrophoresis of neurotensin and its antiserum in female rats. J Neuroendocrinol. 1990;2(2):141-3doi: 10.1111/j.1365-2826.1990.tb00843.x.
Akema, T., & Kimuraf, F. (1990). Preoptic unit responses to microelectrophoresis of neurotensin and its antiserum in female rats. Journal of neuroendocrinology, 2(2), 141-3. https://doi.org/10.1111/j.1365-2826.1990.tb00843.x
Akema, T, and Kimuraf, F. "Preoptic unit responses to microelectrophoresis of neurotensin and its antiserum in female rats." Journal of neuroendocrinology vol. 2,2 (1990): 141-3. doi: https://doi.org/10.1111/j.1365-2826.1990.tb00843.x
Akema T, Kimuraf F. Preoptic unit responses to microelectrophoresis of neurotensin and its antiserum in female rats. J Neuroendocrinol. 1990 Apr 01;2(2):141-3. doi: 10.1111/j.1365-2826.1990.tb00843.x. PMID: 19210376.
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A search for endogenous modulators of the GABA receptor in different regions of the rat CNS.

Neurochemistry international

Lagos N, Valdés F, Orrego F.
PMID: 20487956
Neurochem Int. 1983;5(3):325-31. doi: 10.1016/0197-0186(83)90035-9.

The possible existence of endogenous substances other than ?-aminobutyric acid (GABA), that can also bind to rat brain GABA receptors, has been investigated in synaptic membranes derived from whole rat brain, or from cerebral cortex; as well as in...

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Lagos N, Valdés F, Orrego F. A search for endogenous modulators of the GABA receptor in different regions of the rat CNS. Neurochem Int. 1983;5(3):325-31doi: 10.1016/0197-0186(83)90035-9.
Lagos, N., Valdés, F., & Orrego, F. (1983). A search for endogenous modulators of the GABA receptor in different regions of the rat CNS. Neurochemistry international, 5(3), 325-31. https://doi.org/10.1016/0197-0186(83)90035-9
Lagos, N, et al. "A search for endogenous modulators of the GABA receptor in different regions of the rat CNS." Neurochemistry international vol. 5,3 (1983): 325-31. doi: https://doi.org/10.1016/0197-0186(83)90035-9
Lagos N, Valdés F, Orrego F. A search for endogenous modulators of the GABA receptor in different regions of the rat CNS. Neurochem Int. 1983;5(3):325-31. doi: 10.1016/0197-0186(83)90035-9. PMID: 20487956.
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Potentiation by thiorphan and bestatin of the naloxone-insensitive analgesic effects of neurotensin and neuromedin n.

Neurochemistry international

Coquerel A, Dubuc I, Kitaegi P, Costentin J.
PMID: 20501239
Neurochem Int. 1988;12(3):361-6. doi: 10.1016/0197-0186(88)90175-1.

Neurotensin induced significant antinociceptive activity as measured in a variety of nociceptive tests 10 and 30 min following intracerebroventricular (i.c.v.) injection in mice. The lowest effective peptide doses were 25 ng in the writhing test, 25-50 ng in the...

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Coquerel A, Dubuc I, Kitaegi P, et al. Potentiation by thiorphan and bestatin of the naloxone-insensitive analgesic effects of neurotensin and neuromedin n. Neurochem Int. 1988;12(3):361-6doi: 10.1016/0197-0186(88)90175-1.
Coquerel, A., Dubuc, I., Kitaegi, P., & Costentin, J. (1988). Potentiation by thiorphan and bestatin of the naloxone-insensitive analgesic effects of neurotensin and neuromedin n. Neurochemistry international, 12(3), 361-6. https://doi.org/10.1016/0197-0186(88)90175-1
Coquerel, A, et al. "Potentiation by thiorphan and bestatin of the naloxone-insensitive analgesic effects of neurotensin and neuromedin n." Neurochemistry international vol. 12,3 (1988): 361-6. doi: https://doi.org/10.1016/0197-0186(88)90175-1
Coquerel A, Dubuc I, Kitaegi P, Costentin J. Potentiation by thiorphan and bestatin of the naloxone-insensitive analgesic effects of neurotensin and neuromedin n. Neurochem Int. 1988;12(3):361-6. doi: 10.1016/0197-0186(88)90175-1. PMID: 20501239.
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Neurotensin and its receptors in the control of glucose homeostasis.

Frontiers in endocrinology

Mazella J, Béraud-Dufour S, Devader C, Massa F, Coppola T.
PMID: 23230428
Front Endocrinol (Lausanne). 2012 Nov 26;3:143. doi: 10.3389/fendo.2012.00143. eCollection 2012.

The pharmacological roles of the neuropeptide neurotensin through its three known receptors are various and complex. Neurotensin is involved in several important biological functions including analgesia and hypothermia in the central nervous system and also food intake and glucose...

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Mazella J, Béraud-Dufour S, Devader C, et al. Neurotensin and its receptors in the control of glucose homeostasis. Front Endocrinol (Lausanne). 2012;3:143doi: 10.3389/fendo.2012.00143.
Mazella, J., Béraud-Dufour, S., Devader, C., Massa, F., & Coppola, T. (2012). Neurotensin and its receptors in the control of glucose homeostasis. Frontiers in endocrinology, 3143. https://doi.org/10.3389/fendo.2012.00143
Mazella, Jean, et al. "Neurotensin and its receptors in the control of glucose homeostasis." Frontiers in endocrinology vol. 3 (2012): 143. doi: https://doi.org/10.3389/fendo.2012.00143
Mazella J, Béraud-Dufour S, Devader C, Massa F, Coppola T. Neurotensin and its receptors in the control of glucose homeostasis. Front Endocrinol (Lausanne). 2012 Nov 26;3:143. doi: 10.3389/fendo.2012.00143. eCollection 2012. PMID: 23230428; PMCID: PMC3515879.
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Chemometric data analysis for deconvolution of overlapped ion mobility profiles.

Journal of the American Society for Mass Spectrometry

Zekavat B, Solouki T.
PMID: 22948903
J Am Soc Mass Spectrom. 2012 Nov;23(11):1873-84. doi: 10.1007/s13361-012-0471-2. Epub 2012 Sep 05.

We present the details of a data analysis approach for deconvolution of the ion mobility (IM) overlapped or unresolved species. This approach takes advantage of the ion fragmentation variations as a function of the IM arrival time. The data...

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Zekavat B, Solouki T. Chemometric data analysis for deconvolution of overlapped ion mobility profiles. J Am Soc Mass Spectrom. 2012;23(11):1873-84doi: 10.1007/s13361-012-0471-2.
Zekavat, B., & Solouki, T. (2012). Chemometric data analysis for deconvolution of overlapped ion mobility profiles. Journal of the American Society for Mass Spectrometry, 23(11), 1873-84. https://doi.org/10.1007/s13361-012-0471-2
Zekavat, Behrooz, and Solouki, Touradj. "Chemometric data analysis for deconvolution of overlapped ion mobility profiles." Journal of the American Society for Mass Spectrometry vol. 23,11 (2012): 1873-84. doi: https://doi.org/10.1007/s13361-012-0471-2
Zekavat B, Solouki T. Chemometric data analysis for deconvolution of overlapped ion mobility profiles. J Am Soc Mass Spectrom. 2012 Nov;23(11):1873-84. doi: 10.1007/s13361-012-0471-2. Epub 2012 Sep 05. PMID: 22948903.
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Dependence of Relative Expression of NTR1 and EGFR on Cell Density and Extracellular pH in Human Pancreatic Cancer Cell Lines.

Cancers

Olszewski-Hamilton U, Hamilton G.
PMID: 24212612
Cancers (Basel). 2011 Jan 04;3(1):182-97. doi: 10.3390/cancers3010182.

Pancreatic adenocarcinoma is a devastating disease characterized by early dissemination and poor prognosis. These solid tumors express receptors for neuropeptides like neurotensin (NT) or epidermal growth factor (EGF) and exhibit acidic regions when grown beyond a certain size. We...

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Olszewski-Hamilton U, Hamilton G. Dependence of Relative Expression of NTR1 and EGFR on Cell Density and Extracellular pH in Human Pancreatic Cancer Cell Lines. Cancers (Basel). 2011;3(1):182-97doi: 10.3390/cancers3010182.
Olszewski-Hamilton, U., & Hamilton, G. (2011). Dependence of Relative Expression of NTR1 and EGFR on Cell Density and Extracellular pH in Human Pancreatic Cancer Cell Lines. Cancers, 3(1), 182-97. https://doi.org/10.3390/cancers3010182
Olszewski-Hamilton, Ulrike, and Hamilton, Gerhard. "Dependence of Relative Expression of NTR1 and EGFR on Cell Density and Extracellular pH in Human Pancreatic Cancer Cell Lines." Cancers vol. 3,1 (2011): 182-97. doi: https://doi.org/10.3390/cancers3010182
Olszewski-Hamilton U, Hamilton G. Dependence of Relative Expression of NTR1 and EGFR on Cell Density and Extracellular pH in Human Pancreatic Cancer Cell Lines. Cancers (Basel). 2011 Jan 04;3(1):182-97. doi: 10.3390/cancers3010182. PMID: 24212612; PMCID: PMC3756355.
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Neurotensin as a source of cyclic AMP and co-mitogen in fibrolamellar hepatocellular carcinoma.

Oncotarget

Riehle KJ, Kenerson HL, Riggle KM, Turnham R, Sullivan K, Bauer R, Scott JD, Yeung RS.
PMID: 31489118
Oncotarget. 2019 Aug 20;10(49):5092-5102. doi: 10.18632/oncotarget.27149. eCollection 2019 Aug 20.

Fibrolamellar hepatocellular carcinomas (FL-HCCs) possess a unique mutation that encodes a chimeric form of protein kinase A (DNAJ-PKAc), which includes a chaperonin binding domain. DNAJ-PKAc retains most of the biochemical properties of the native enzyme, however, and activity remains...

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Riehle KJ, Kenerson HL, Riggle KM, et al. Neurotensin as a source of cyclic AMP and co-mitogen in fibrolamellar hepatocellular carcinoma. Oncotarget. 2019;10(49):5092-5102doi: 10.18632/oncotarget.27149.
Riehle, K. J., Kenerson, H. L., Riggle, K. M., Turnham, R., Sullivan, K., Bauer, R., Scott, J. D., & Yeung, R. S. (2019). Neurotensin as a source of cyclic AMP and co-mitogen in fibrolamellar hepatocellular carcinoma. Oncotarget, 10(49), 5092-5102. https://doi.org/10.18632/oncotarget.27149
Riehle, Kimberly J, et al. "Neurotensin as a source of cyclic AMP and co-mitogen in fibrolamellar hepatocellular carcinoma." Oncotarget vol. 10,49 (2019): 5092-5102. doi: https://doi.org/10.18632/oncotarget.27149
Riehle KJ, Kenerson HL, Riggle KM, Turnham R, Sullivan K, Bauer R, Scott JD, Yeung RS. Neurotensin as a source of cyclic AMP and co-mitogen in fibrolamellar hepatocellular carcinoma. Oncotarget. 2019 Aug 20;10(49):5092-5102. doi: 10.18632/oncotarget.27149. eCollection 2019 Aug 20. PMID: 31489118; PMCID: PMC6707953.
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Showing 1 to 12 of 235 entries