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Showing 1 to 12 of 19 entries
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Proinflammatory high-density lipoprotein results from oxidized lipid mediators in the pathogenesis of both idiopathic and associated types of pulmonary arterial hypertension.

Pulmonary circulation

Ross DJ, Hough G, Hama S, Aboulhosn J, Belperio JA, Saggar R, Van Lenten BJ, Ardehali A, Eghbali M, Reddy S, Fogelman AM, Navab M.
PMID: 26697171
Pulm Circ. 2015 Dec;5(4):640-8. doi: 10.1086/683695.

Pulmonary arterial hypertension (PAH) is characterized by abnormal elaboration of vasoactive peptides, endothelial cell dysfunction, vascular remodeling, and inflammation, which collectively contribute to its pathogenesis. We investigated the potential for high-density lipoprotein (HDL) dysfunction (i.e., proinflammatory effects) and abnormal...

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Ross DJ, Hough G, Hama S, et al. Proinflammatory high-density lipoprotein results from oxidized lipid mediators in the pathogenesis of both idiopathic and associated types of pulmonary arterial hypertension. Pulm Circ. 2015;5(4):640-8doi: 10.1086/683695.
Ross, D. J., Hough, G., Hama, S., Aboulhosn, J., Belperio, J. A., Saggar, R., Van Lenten, B. J., Ardehali, A., Eghbali, M., Reddy, S., Fogelman, A. M., & Navab, M. (2015). Proinflammatory high-density lipoprotein results from oxidized lipid mediators in the pathogenesis of both idiopathic and associated types of pulmonary arterial hypertension. Pulmonary circulation, 5(4), 640-8. https://doi.org/10.1086/683695
Ross, David J, et al. "Proinflammatory high-density lipoprotein results from oxidized lipid mediators in the pathogenesis of both idiopathic and associated types of pulmonary arterial hypertension." Pulmonary circulation vol. 5,4 (2015): 640-8. doi: https://doi.org/10.1086/683695
Ross DJ, Hough G, Hama S, Aboulhosn J, Belperio JA, Saggar R, Van Lenten BJ, Ardehali A, Eghbali M, Reddy S, Fogelman AM, Navab M. Proinflammatory high-density lipoprotein results from oxidized lipid mediators in the pathogenesis of both idiopathic and associated types of pulmonary arterial hypertension. Pulm Circ. 2015 Dec;5(4):640-8. doi: 10.1086/683695. PMID: 26697171; PMCID: PMC4671738.
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Efficacy of tomato concentrates in mouse models of dyslipidemia and cancer.

Pharmacology research & perspectives

Chattopadhyay A, Grijalva V, Hough G, Su F, Mukherjee P, Farias-Eisner R, Anantharamaiah GM, Faull KF, Hwang LH, Navab M, Fogelman AM, Reddy ST.
PMID: 26171234
Pharmacol Res Perspect. 2015 Aug;3(4):e00154. doi: 10.1002/prp2.154. Epub 2015 Jun 24.

We previously reported that adding freeze-dried tomato powder from transgenic plants expressing the apolipoprotein A-I mimetic peptide 6F at 2.2% by weight to a Western diet (WD) ameliorated dyslipidemia and atherosclerosis in mice. The same dose in a human...

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Chattopadhyay A, Grijalva V, Hough G, et al. Efficacy of tomato concentrates in mouse models of dyslipidemia and cancer. Pharmacol Res Perspect. 2015;3(4):e00154doi: 10.1002/prp2.154.
Chattopadhyay, A., Grijalva, V., Hough, G., Su, F., Mukherjee, P., Farias-Eisner, R., Anantharamaiah, G. M., Faull, K. F., Hwang, L. H., Navab, M., Fogelman, A. M., & Reddy, S. T. (2015). Efficacy of tomato concentrates in mouse models of dyslipidemia and cancer. Pharmacology research & perspectives, 3(4), e00154. https://doi.org/10.1002/prp2.154
Chattopadhyay, Arnab, et al. "Efficacy of tomato concentrates in mouse models of dyslipidemia and cancer." Pharmacology research & perspectives vol. 3,4 (2015): e00154. doi: https://doi.org/10.1002/prp2.154
Chattopadhyay A, Grijalva V, Hough G, Su F, Mukherjee P, Farias-Eisner R, Anantharamaiah GM, Faull KF, Hwang LH, Navab M, Fogelman AM, Reddy ST. Efficacy of tomato concentrates in mouse models of dyslipidemia and cancer. Pharmacol Res Perspect. 2015 Aug;3(4):e00154. doi: 10.1002/prp2.154. Epub 2015 Jun 24. PMID: 26171234; PMCID: PMC4492730.
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The systems genetics resource: a web application to mine global data for complex disease traits.

Frontiers in genetics

van Nas A, Pan C, Ingram-Drake LA, Ghazalpour A, Drake TA, Sobel EM, Papp JC, Lusis AJ.
PMID: 23730305
Front Genet. 2013 May 20;4:84. doi: 10.3389/fgene.2013.00084. eCollection 2013.

The Systems Genetics Resource (SGR) (http://systems.genetics.ucla.edu) is a new open-access web application and database that contains genotypes and clinical and intermediate phenotypes from both human and mouse studies. The mouse data include studies using crosses between specific inbred strains...

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van Nas A, Pan C, Ingram-Drake LA, et al. The systems genetics resource: a web application to mine global data for complex disease traits. Front Genet. 2013;4:84doi: 10.3389/fgene.2013.00084.
van Nas, A., Pan, C., Ingram-Drake, L. A., Ghazalpour, A., Drake, T. A., Sobel, E. M., Papp, J. C., & Lusis, A. J. (2013). The systems genetics resource: a web application to mine global data for complex disease traits. Frontiers in genetics, 484. https://doi.org/10.3389/fgene.2013.00084
van Nas, Atila, et al. "The systems genetics resource: a web application to mine global data for complex disease traits." Frontiers in genetics vol. 4 (2013): 84. doi: https://doi.org/10.3389/fgene.2013.00084
van Nas A, Pan C, Ingram-Drake LA, Ghazalpour A, Drake TA, Sobel EM, Papp JC, Lusis AJ. The systems genetics resource: a web application to mine global data for complex disease traits. Front Genet. 2013 May 20;4:84. doi: 10.3389/fgene.2013.00084. eCollection 2013. PMID: 23730305; PMCID: PMC3657633.
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Maximal information component analysis: a novel non-linear network analysis method.

Frontiers in genetics

Rau CD, Wisniewski N, Orozco LD, Bennett B, Weiss J, Lusis AJ.
PMID: 23487572
Front Genet. 2013 Mar 12;4:28. doi: 10.3389/fgene.2013.00028. eCollection 2013.

BACKGROUND: Network construction and analysis algorithms provide scientists with the ability to sift through high-throughput biological outputs, such as transcription microarrays, for small groups of genes (modules) that are relevant for further research. Most of these algorithms ignore the...

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Rau CD, Wisniewski N, Orozco LD, et al. Maximal information component analysis: a novel non-linear network analysis method. Front Genet. 2013;4:28doi: 10.3389/fgene.2013.00028.
Rau, C. D., Wisniewski, N., Orozco, L. D., Bennett, B., Weiss, J., & Lusis, A. J. (2013). Maximal information component analysis: a novel non-linear network analysis method. Frontiers in genetics, 428. https://doi.org/10.3389/fgene.2013.00028
Rau, Christoph D, et al. "Maximal information component analysis: a novel non-linear network analysis method." Frontiers in genetics vol. 4 (2013): 28. doi: https://doi.org/10.3389/fgene.2013.00028
Rau CD, Wisniewski N, Orozco LD, Bennett B, Weiss J, Lusis AJ. Maximal information component analysis: a novel non-linear network analysis method. Front Genet. 2013 Mar 12;4:28. doi: 10.3389/fgene.2013.00028. eCollection 2013. PMID: 23487572; PMCID: PMC3594742.
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Maternal High-Protein and Low-Protein Diets Perturb Hypothalamus and Liver Transcriptome and Metabolic Homeostasis in Adult Mouse Offspring.

Frontiers in genetics

Martin LJ, Meng Q, Blencowe M, Lagarrigue S, Xiao S, Pan C, Wier J, Temple WC, Devaskar SU, Lusis AJ, Yang X.
PMID: 30619467
Front Genet. 2018 Dec 11;9:642. doi: 10.3389/fgene.2018.00642. eCollection 2018.

Early life nutritional imbalances are risk factors for metabolic dysfunctions in adulthood, but the long term effects of perinatal exposure to high versus low protein diets are not completely understood. We exposed C57BL/6J offspring to a high protein/low carbohydrate...

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Martin LJ, Meng Q, Blencowe M, et al. Maternal High-Protein and Low-Protein Diets Perturb Hypothalamus and Liver Transcriptome and Metabolic Homeostasis in Adult Mouse Offspring. Front Genet. 2018;9:642doi: 10.3389/fgene.2018.00642.
Martin, L. J., Meng, Q., Blencowe, M., Lagarrigue, S., Xiao, S., Pan, C., Wier, J., Temple, W. C., Devaskar, S. U., Lusis, A. J., & Yang, X. (2018). Maternal High-Protein and Low-Protein Diets Perturb Hypothalamus and Liver Transcriptome and Metabolic Homeostasis in Adult Mouse Offspring. Frontiers in genetics, 9642. https://doi.org/10.3389/fgene.2018.00642
Martin, Lisa J, et al. "Maternal High-Protein and Low-Protein Diets Perturb Hypothalamus and Liver Transcriptome and Metabolic Homeostasis in Adult Mouse Offspring." Frontiers in genetics vol. 9 (2018): 642. doi: https://doi.org/10.3389/fgene.2018.00642
Martin LJ, Meng Q, Blencowe M, Lagarrigue S, Xiao S, Pan C, Wier J, Temple WC, Devaskar SU, Lusis AJ, Yang X. Maternal High-Protein and Low-Protein Diets Perturb Hypothalamus and Liver Transcriptome and Metabolic Homeostasis in Adult Mouse Offspring. Front Genet. 2018 Dec 11;9:642. doi: 10.3389/fgene.2018.00642. eCollection 2018. PMID: 30619467; PMCID: PMC6297185.
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Options for COVID-19 Entry into Pulmonary Cells.

Biomedical journal of scientific & technical research

Boström KI, Yao Y.
PMID: 33283173
Biomed J Sci Tech Res. 2020 Aug;29(2):22337-22338. doi: 10.26717/bjstr.2020.29.004781. Epub 2020 Aug 05.

No abstract available.

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Boström KI, Yao Y. Options for COVID-19 Entry into Pulmonary Cells. Biomed J Sci Tech Res. 2020;29(2):22337-22338doi: 10.26717/bjstr.2020.29.004781.
Boström, K. I., & Yao, Y. (2020). Options for COVID-19 Entry into Pulmonary Cells. Biomedical journal of scientific & technical research, 29(2), 22337-22338. https://doi.org/10.26717/bjstr.2020.29.004781
Boström, Kristina I, and Yao, Yucheng. "Options for COVID-19 Entry into Pulmonary Cells." Biomedical journal of scientific & technical research vol. 29,2 (2020): 22337-22338. doi: https://doi.org/10.26717/bjstr.2020.29.004781
Boström KI, Yao Y. Options for COVID-19 Entry into Pulmonary Cells. Biomed J Sci Tech Res. 2020 Aug;29(2):22337-22338. doi: 10.26717/bjstr.2020.29.004781. Epub 2020 Aug 05. PMID: 33283173; PMCID: PMC7717074.
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Apolipoprotein mimetics in cancer.

Seminars in cancer biology

Delk SC, Chattopadhyay A, Escola-Gil JC, Fogelman AM, Reddy ST.
PMID: 33188891
Semin Cancer Biol. 2021 Aug;73:158-168. doi: 10.1016/j.semcancer.2020.11.002. Epub 2020 Nov 11.

Peptides have many advantages over traditional therapeutics, including small molecules and other biologics, because of their low toxicity and immunogenicity, while still exhibiting efficacy. This review discusses the benefits and mechanism of action of apolipoprotein mimetic peptides in tumor...

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Delk SC, Chattopadhyay A, Escola-Gil JC, et al. Apolipoprotein mimetics in cancer. Semin Cancer Biol. 2020;73:158-168doi: 10.1016/j.semcancer.2020.11.002.
Delk, S. C., Chattopadhyay, A., Escola-Gil, J. C., Fogelman, A. M., & Reddy, S. T. (2021). Apolipoprotein mimetics in cancer. Seminars in cancer biology, 73158-168. https://doi.org/10.1016/j.semcancer.2020.11.002
Delk, Samuel C, et al. "Apolipoprotein mimetics in cancer." Seminars in cancer biology vol. 73 (2021): 158-168. doi: https://doi.org/10.1016/j.semcancer.2020.11.002
Delk SC, Chattopadhyay A, Escola-Gil JC, Fogelman AM, Reddy ST. Apolipoprotein mimetics in cancer. Semin Cancer Biol. 2021 Aug;73:158-168. doi: 10.1016/j.semcancer.2020.11.002. Epub 2020 Nov 11. PMID: 33188891; PMCID: PMC8110614.
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Peptide Mimetics of Apolipoproteins Improve HDL Function.

Journal of clinical lipidology

Navab M, Anantharamaiah GM, Reddy ST, Van Lenten BJ, Buga GM, Fogelman AM.
PMID: 18449337
J Clin Lipidol. 2007 May;1(2):142-7. doi: 10.1016/j.jacl.2007.03.002.

Over the past decade evidence has accumulated that suggests that the anti-inflammatory properties of HDL may be at least as important as the levels of HDL-cholesterol. The recent failure of the torcetrapib clinical trails has highlighted the potential differences...

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Navab M, Anantharamaiah GM, Reddy ST, et al. Peptide Mimetics of Apolipoproteins Improve HDL Function. J Clin Lipidol. 2007;1(2):142-7doi: 10.1016/j.jacl.2007.03.002.
Navab, M., Anantharamaiah, G. M., Reddy, S. T., Van Lenten, B. J., Buga, G. M., & Fogelman, A. M. (2007). Peptide Mimetics of Apolipoproteins Improve HDL Function. Journal of clinical lipidology, 1(2), 142-7. https://doi.org/10.1016/j.jacl.2007.03.002
Navab, Mohamad, et al. "Peptide Mimetics of Apolipoproteins Improve HDL Function." Journal of clinical lipidology vol. 1,2 (2007): 142-7. doi: https://doi.org/10.1016/j.jacl.2007.03.002
Navab M, Anantharamaiah GM, Reddy ST, Van Lenten BJ, Buga GM, Fogelman AM. Peptide Mimetics of Apolipoproteins Improve HDL Function. J Clin Lipidol. 2007 May;1(2):142-7. doi: 10.1016/j.jacl.2007.03.002. PMID: 18449337; PMCID: PMC2130772.
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SOX Transcription Factors in Endothelial Differentiation and Endothelial-Mesenchymal Transitions.

Frontiers in cardiovascular medicine

Yao Y, Yao J, Boström KI.
PMID: 30984768
Front Cardiovasc Med. 2019 Mar 28;6:30. doi: 10.3389/fcvm.2019.00030. eCollection 2019.

The SRY (Sex Determining Region Y)-related HMG box of DNA binding proteins, referred to as SOX transcription factors, were first identified as critical regulators of male sex determination but are now known to play an important role in vascular...

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Yao Y, Yao J, Boström KI. SOX Transcription Factors in Endothelial Differentiation and Endothelial-Mesenchymal Transitions. Front Cardiovasc Med. 2019;6:30doi: 10.3389/fcvm.2019.00030.
Yao, Y., Yao, J., & Boström, K. I. (2019). SOX Transcription Factors in Endothelial Differentiation and Endothelial-Mesenchymal Transitions. Frontiers in cardiovascular medicine, 630. https://doi.org/10.3389/fcvm.2019.00030
Yao, Yucheng, et al. "SOX Transcription Factors in Endothelial Differentiation and Endothelial-Mesenchymal Transitions." Frontiers in cardiovascular medicine vol. 6 (2019): 30. doi: https://doi.org/10.3389/fcvm.2019.00030
Yao Y, Yao J, Boström KI. SOX Transcription Factors in Endothelial Differentiation and Endothelial-Mesenchymal Transitions. Front Cardiovasc Med. 2019 Mar 28;6:30. doi: 10.3389/fcvm.2019.00030. eCollection 2019. PMID: 30984768; PMCID: PMC6447608.
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Atherosclerosis: Association between the gut microbiome and atherosclerosis.

Nature reviews. Cardiology

Barrington WT, Lusis AJ.
PMID: 29099096
Nat Rev Cardiol. 2017 Dec;14(12):699-700. doi: 10.1038/nrcardio.2017.169. Epub 2017 Nov 03.

No abstract available.

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Barrington WT, Lusis AJ. Atherosclerosis: Association between the gut microbiome and atherosclerosis. Nat Rev Cardiol. 2017;14(12):699-700doi: 10.1038/nrcardio.2017.169.
Barrington, W. T., & Lusis, A. J. (2017). Atherosclerosis: Association between the gut microbiome and atherosclerosis. Nature reviews. Cardiology, 14(12), 699-700. https://doi.org/10.1038/nrcardio.2017.169
Barrington, William T, and Lusis, Aldons J. "Atherosclerosis: Association between the gut microbiome and atherosclerosis." Nature reviews. Cardiology vol. 14,12 (2017): 699-700. doi: https://doi.org/10.1038/nrcardio.2017.169
Barrington WT, Lusis AJ. Atherosclerosis: Association between the gut microbiome and atherosclerosis. Nat Rev Cardiol. 2017 Dec;14(12):699-700. doi: 10.1038/nrcardio.2017.169. Epub 2017 Nov 03. PMID: 29099096; PMCID: PMC5815826.
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Dedifferentiated fat cells: A cell source for regenerative medicine.

World journal of stem cells

Jumabay M, Boström KI.
PMID: 26640620
World J Stem Cells. 2015 Nov 26;7(10):1202-14. doi: 10.4252/wjsc.v7.i10.1202.

The identification of an ideal cell source for tissue regeneration remains a challenge in the stem cell field. The ability of progeny cells to differentiate into other cell types is important for the processes of tissue reconstruction and tissue...

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Jumabay M, Boström KI. Dedifferentiated fat cells: A cell source for regenerative medicine. World J Stem Cells. 2015;7(10):1202-14doi: 10.4252/wjsc.v7.i10.1202.
Jumabay, M., & Boström, K. I. (2015). Dedifferentiated fat cells: A cell source for regenerative medicine. World journal of stem cells, 7(10), 1202-14. https://doi.org/10.4252/wjsc.v7.i10.1202
Jumabay, Medet, and Boström, Kristina I. "Dedifferentiated fat cells: A cell source for regenerative medicine." World journal of stem cells vol. 7,10 (2015): 1202-14. doi: https://doi.org/10.4252/wjsc.v7.i10.1202
Jumabay M, Boström KI. Dedifferentiated fat cells: A cell source for regenerative medicine. World J Stem Cells. 2015 Nov 26;7(10):1202-14. doi: 10.4252/wjsc.v7.i10.1202. PMID: 26640620; PMCID: PMC4663373.
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ABCC6 deficiency is associated with activation of BMP signaling in liver and kidney.

FEBS open bio

Blazquez-Medela AM, Guihard PJ, Yao J, Jumabay M, Lusis AJ, Boström KI, Yao Y.
PMID: 25893161
FEBS Open Bio. 2015 Mar 23;5:257-63. doi: 10.1016/j.fob.2015.03.009. eCollection 2015.

Mutations in ABCC6 (ATP-binding cassette, subfamily C, member 6), an orphan transporter expressed in the liver, are the cause of pseudoxanthoma elasticum. Since ABCC6 was reported to affect matrix Gla protein (MGP), an inhibitor of bone morphogenetic proteins (BMPs),...

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Blazquez-Medela AM, Guihard PJ, Yao J, et al. ABCC6 deficiency is associated with activation of BMP signaling in liver and kidney. FEBS Open Bio. 2015;5:257-63doi: 10.1016/j.fob.2015.03.009.
Blazquez-Medela, A. M., Guihard, P. J., Yao, J., Jumabay, M., Lusis, A. J., Boström, K. I., & Yao, Y. (2015). ABCC6 deficiency is associated with activation of BMP signaling in liver and kidney. FEBS open bio, 5257-63. https://doi.org/10.1016/j.fob.2015.03.009
Blazquez-Medela, Ana M, et al. "ABCC6 deficiency is associated with activation of BMP signaling in liver and kidney." FEBS open bio vol. 5 (2015): 257-63. doi: https://doi.org/10.1016/j.fob.2015.03.009
Blazquez-Medela AM, Guihard PJ, Yao J, Jumabay M, Lusis AJ, Boström KI, Yao Y. ABCC6 deficiency is associated with activation of BMP signaling in liver and kidney. FEBS Open Bio. 2015 Mar 23;5:257-63. doi: 10.1016/j.fob.2015.03.009. eCollection 2015. PMID: 25893161; PMCID: PMC4398664.
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Showing 1 to 12 of 19 entries