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Central role of carotid body chemoreceptors in disordered breathing and cardiorenal dysfunction in chronic heart failure.

Frontiers in physiology

Marcus NJ, Del Rio R, Schultz HD.
PMID: 25505417
Front Physiol. 2014 Nov 24;5:438. doi: 10.3389/fphys.2014.00438. eCollection 2014.

Oscillatory breathing (OB) patterns are observed in pre-term infants, patients with cardio-renal impairment, and in otherwise healthy humans exposed to high altitude. Enhanced carotid body (CB) chemoreflex sensitivity is common to all of these populations and is thought to...

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Marcus NJ, Del Rio R, Schultz HD. Central role of carotid body chemoreceptors in disordered breathing and cardiorenal dysfunction in chronic heart failure. Front Physiol. 2014;5:438doi: 10.3389/fphys.2014.00438.
Marcus, N. J., Del Rio, R., & Schultz, H. D. (2014). Central role of carotid body chemoreceptors in disordered breathing and cardiorenal dysfunction in chronic heart failure. Frontiers in physiology, 5438. https://doi.org/10.3389/fphys.2014.00438
Marcus, Noah J, et al. "Central role of carotid body chemoreceptors in disordered breathing and cardiorenal dysfunction in chronic heart failure." Frontiers in physiology vol. 5 (2014): 438. doi: https://doi.org/10.3389/fphys.2014.00438
Marcus NJ, Del Rio R, Schultz HD. Central role of carotid body chemoreceptors in disordered breathing and cardiorenal dysfunction in chronic heart failure. Front Physiol. 2014 Nov 24;5:438. doi: 10.3389/fphys.2014.00438. eCollection 2014. PMID: 25505417; PMCID: PMC4241833.
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Skeletal Muscle Nrf2 Contributes to Exercise-Evoked Systemic Antioxidant Defense Via Extracellular Vesicular Communication.

Exercise and sport sciences reviews

Gao L, Wang HJ, Tian C, Zucker IH.
PMID: 33927165
Exerc Sport Sci Rev. 2021 Jul 01;49(3):213-222. doi: 10.1249/JES.0000000000000257.

This review explores the hypothesis that the repetitive contraction-relaxation that occurs during chronic exercise activates skeletal myocyte nuclear factor erythroid-derived 2-like 2 (Nrf2) to upregulate antioxidant enzymes. These proteins are secreted into the circulation within extracellular vesicles and taken...

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Gao L, Wang HJ, Tian C, et al. Skeletal Muscle Nrf2 Contributes to Exercise-Evoked Systemic Antioxidant Defense Via Extracellular Vesicular Communication. Exerc Sport Sci Rev. 2021;49(3):213-222doi: 10.1249/JES.0000000000000257.
Gao, L., Wang, H. J., Tian, C., & Zucker, I. H. (2021). Skeletal Muscle Nrf2 Contributes to Exercise-Evoked Systemic Antioxidant Defense Via Extracellular Vesicular Communication. Exercise and sport sciences reviews, 49(3), 213-222. https://doi.org/10.1249/JES.0000000000000257
Gao, Lie, et al. "Skeletal Muscle Nrf2 Contributes to Exercise-Evoked Systemic Antioxidant Defense Via Extracellular Vesicular Communication." Exercise and sport sciences reviews vol. 49,3 (2021): 213-222. doi: https://doi.org/10.1249/JES.0000000000000257
Gao L, Wang HJ, Tian C, Zucker IH. Skeletal Muscle Nrf2 Contributes to Exercise-Evoked Systemic Antioxidant Defense Via Extracellular Vesicular Communication. Exerc Sport Sci Rev. 2021 Jul 01;49(3):213-222. doi: 10.1249/JES.0000000000000257. PMID: 33927165; PMCID: PMC8195856.
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BDNF contributes to angiotensin II-mediated reductions in peak voltage-gated K+ current in cultured CATH.a cells.

Physiological reports

Becker BK, Wang HJ, Tian C, Zucker IH.
PMID: 26537343
Physiol Rep. 2015 Nov;3(11). doi: 10.14814/phy2.12598.

Increased central angiotensin II (Ang II) levels contribute to sympathoexcitation in cardiovascular disease states such as chronic heart failure and hypertension. One mechanism by which Ang II increases neuronal excitability is through a decrease in voltage-gated, rapidly inactivating K(+)...

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Becker BK, Wang HJ, Tian C, et al. BDNF contributes to angiotensin II-mediated reductions in peak voltage-gated K+ current in cultured CATH.a cells. Physiol Rep. 2015;3(11)doi: 10.14814/phy2.12598.
Becker, B. K., Wang, H. J., Tian, C., & Zucker, I. H. (2015). BDNF contributes to angiotensin II-mediated reductions in peak voltage-gated K+ current in cultured CATH.a cells. Physiological reports, 3(11), . https://doi.org/10.14814/phy2.12598
Becker, Bryan K, et al. "BDNF contributes to angiotensin II-mediated reductions in peak voltage-gated K+ current in cultured CATH.a cells." Physiological reports vol. 3,11 (2015). doi: https://doi.org/10.14814/phy2.12598
Becker BK, Wang HJ, Tian C, Zucker IH. BDNF contributes to angiotensin II-mediated reductions in peak voltage-gated K+ current in cultured CATH.a cells. Physiol Rep. 2015 Nov;3(11). doi: 10.14814/phy2.12598. PMID: 26537343; PMCID: PMC4673628.
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Editorial: Carotid body: a new target for rescuing neural control of cardiorespiratory balance in disease.

Frontiers in physiology

Del Rio R, Iturriaga R, Schultz HD.
PMID: 26175689
Front Physiol. 2015 Jun 29;6:181. doi: 10.3389/fphys.2015.00181. eCollection 2015.

No abstract available.

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Del Rio R, Iturriaga R, Schultz HD. Editorial: Carotid body: a new target for rescuing neural control of cardiorespiratory balance in disease. Front Physiol. 2015;6:181doi: 10.3389/fphys.2015.00181.
Del Rio, R., Iturriaga, R., & Schultz, H. D. (2015). Editorial: Carotid body: a new target for rescuing neural control of cardiorespiratory balance in disease. Frontiers in physiology, 6181. https://doi.org/10.3389/fphys.2015.00181
Del Rio, Rodrigo, et al. "Editorial: Carotid body: a new target for rescuing neural control of cardiorespiratory balance in disease." Frontiers in physiology vol. 6 (2015): 181. doi: https://doi.org/10.3389/fphys.2015.00181
Del Rio R, Iturriaga R, Schultz HD. Editorial: Carotid body: a new target for rescuing neural control of cardiorespiratory balance in disease. Front Physiol. 2015 Jun 29;6:181. doi: 10.3389/fphys.2015.00181. eCollection 2015. PMID: 26175689; PMCID: PMC4483515.
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Muscle reflex in heart failure: the role of exercise training.

Frontiers in physiology

Wang HJ, Zucker IH, Wang W.
PMID: 23060821
Front Physiol. 2012 Oct 05;3:398. doi: 10.3389/fphys.2012.00398. eCollection 2012.

Exercise evokes sympathetic activation and increases blood pressure and heart rate (HR). Two neural mechanisms that cause the exercise-induced increase in sympathetic discharge are central command and the exercise pressor reflex (EPR). The former suggests that a volitional signal...

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Wang HJ, Zucker IH, Wang W. Muscle reflex in heart failure: the role of exercise training. Front Physiol. 2012;3:398doi: 10.3389/fphys.2012.00398.
Wang, H. J., Zucker, I. H., & Wang, W. (2012). Muscle reflex in heart failure: the role of exercise training. Frontiers in physiology, 3398. https://doi.org/10.3389/fphys.2012.00398
Wang, Han-Jun, et al. "Muscle reflex in heart failure: the role of exercise training." Frontiers in physiology vol. 3 (2012): 398. doi: https://doi.org/10.3389/fphys.2012.00398
Wang HJ, Zucker IH, Wang W. Muscle reflex in heart failure: the role of exercise training. Front Physiol. 2012 Oct 05;3:398. doi: 10.3389/fphys.2012.00398. eCollection 2012. PMID: 23060821; PMCID: PMC3464681.
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Renal denervation based on experimental rationale.

Hypertension research : official journal of the Japanese Society of Hypertension

Katsurada K, Ogoyama Y, Imai Y, Patel KP, Kario K.
PMID: 34518650
Hypertens Res. 2021 Nov;44(11):1385-1394. doi: 10.1038/s41440-021-00746-7. Epub 2021 Sep 13.

Excessive activation of the sympathetic nervous system is one of the pathophysiological hallmarks of hypertension and heart failure. Within the central nervous system, the paraventricular nucleus (PVN) of the hypothalamus and the rostral ventrolateral medulla in the brain stem...

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Katsurada K, Ogoyama Y, Imai Y, et al. Renal denervation based on experimental rationale. Hypertens Res. 2021;44(11):1385-1394doi: 10.1038/s41440-021-00746-7.
Katsurada, K., Ogoyama, Y., Imai, Y., Patel, K. P., & Kario, K. (2021). Renal denervation based on experimental rationale. Hypertension research : official journal of the Japanese Society of Hypertension, 44(11), 1385-1394. https://doi.org/10.1038/s41440-021-00746-7
Katsurada, Kenichi, et al. "Renal denervation based on experimental rationale." Hypertension research : official journal of the Japanese Society of Hypertension vol. 44,11 (2021): 1385-1394. doi: https://doi.org/10.1038/s41440-021-00746-7
Katsurada K, Ogoyama Y, Imai Y, Patel KP, Kario K. Renal denervation based on experimental rationale. Hypertens Res. 2021 Nov;44(11):1385-1394. doi: 10.1038/s41440-021-00746-7. Epub 2021 Sep 13. PMID: 34518650.
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The renal nerves in chronic heart failure: efferent and afferent mechanisms.

Frontiers in physiology

Schiller AM, Pellegrino PR, Zucker IH.
PMID: 26300788
Front Physiol. 2015 Aug 07;6:224. doi: 10.3389/fphys.2015.00224. eCollection 2015.

The function of the renal nerves has been an area of scientific and medical interest for many years. The recent advent of a minimally invasive catheter-based method of renal denervation has renewed excitement in understanding the afferent and efferent...

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Schiller AM, Pellegrino PR, Zucker IH. The renal nerves in chronic heart failure: efferent and afferent mechanisms. Front Physiol. 2015;6:224doi: 10.3389/fphys.2015.00224.
Schiller, A. M., Pellegrino, P. R., & Zucker, I. H. (2015). The renal nerves in chronic heart failure: efferent and afferent mechanisms. Frontiers in physiology, 6224. https://doi.org/10.3389/fphys.2015.00224
Schiller, Alicia M, et al. "The renal nerves in chronic heart failure: efferent and afferent mechanisms." Frontiers in physiology vol. 6 (2015): 224. doi: https://doi.org/10.3389/fphys.2015.00224
Schiller AM, Pellegrino PR, Zucker IH. The renal nerves in chronic heart failure: efferent and afferent mechanisms. Front Physiol. 2015 Aug 07;6:224. doi: 10.3389/fphys.2015.00224. eCollection 2015. PMID: 26300788; PMCID: PMC4528173.
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Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves.

Circulation. Heart failure

Katsurada K, Nandi SS, Sharma NM, Patel KP.
PMID: 34789005
Circ Heart Fail. 2021 Dec;14(12):e008365. doi: 10.1161/CIRCHEARTFAILURE.121.008365. Epub 2021 Nov 18.

BACKGROUND: Recent clinical studies demonstrate that SGLT2 (sodium-glucose cotransporter 2) inhibitors ameliorate heart failure (HF). The present study was conducted to assess the expression and function of renal SGLT2 and the influence of enhanced renal sympathetic tone in HF.METHODS:...

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Katsurada K, Nandi SS, Sharma NM, et al. Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves. Circ Heart Fail. 2021;14(12):e008365doi: 10.1161/CIRCHEARTFAILURE.121.008365.
Katsurada, K., Nandi, S. S., Sharma, N. M., & Patel, K. P. (2021). Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves. Circulation. Heart failure, 14(12), e008365. https://doi.org/10.1161/CIRCHEARTFAILURE.121.008365
Katsurada, Kenichi, et al. "Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves." Circulation. Heart failure vol. 14,12 (2021): e008365. doi: https://doi.org/10.1161/CIRCHEARTFAILURE.121.008365
Katsurada K, Nandi SS, Sharma NM, Patel KP. Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves. Circ Heart Fail. 2021 Dec;14(12):e008365. doi: 10.1161/CIRCHEARTFAILURE.121.008365. Epub 2021 Nov 18. PMID: 34789005; PMCID: PMC8692398.
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Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves.

Circulation. Heart failure

Katsurada K, Nandi SS, Sharma NM, Patel KP.
PMID: 34789005
Circ Heart Fail. 2021 Dec;14(12):e008365. doi: 10.1161/CIRCHEARTFAILURE.121.008365. Epub 2021 Nov 18.

BACKGROUND: Recent clinical studies demonstrate that SGLT2 (sodium-glucose cotransporter 2) inhibitors ameliorate heart failure (HF). The present study was conducted to assess the expression and function of renal SGLT2 and the influence of enhanced renal sympathetic tone in HF.METHODS:...

Cite
Katsurada K, Nandi SS, Sharma NM, et al. Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves. Circ Heart Fail. 2021;14(12):e008365doi: 10.1161/CIRCHEARTFAILURE.121.008365.
Katsurada, K., Nandi, S. S., Sharma, N. M., & Patel, K. P. (2021). Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves. Circulation. Heart failure, 14(12), e008365. https://doi.org/10.1161/CIRCHEARTFAILURE.121.008365
Katsurada, Kenichi, et al. "Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves." Circulation. Heart failure vol. 14,12 (2021): e008365. doi: https://doi.org/10.1161/CIRCHEARTFAILURE.121.008365
Katsurada K, Nandi SS, Sharma NM, Patel KP. Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves. Circ Heart Fail. 2021 Dec;14(12):e008365. doi: 10.1161/CIRCHEARTFAILURE.121.008365. Epub 2021 Nov 18. PMID: 34789005.
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Disruption of cardiovascular circadian rhythms in mice post myocardial infarction: relationship with central angiotensin II receptor expression.

Physiological reports

Mousa TM, Schiller AM, Zucker IH.
PMID: 25413327
Physiol Rep. 2014 Nov 20;2(11). doi: 10.14814/phy2.12210. Print 2014 Nov 01.

Angiotensin II (Ang II) is well known to participate in the abnormal autonomic cardiovascular control that occurs during the development of chronic heart failure (CHF). Disrupted cardiovascular circadian rhythm in CHF is also well accepted; however, the mechanisms underlying...

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Mousa TM, Schiller AM, Zucker IH. Disruption of cardiovascular circadian rhythms in mice post myocardial infarction: relationship with central angiotensin II receptor expression. Physiol Rep. 2014;2(11)doi: 10.14814/phy2.12210.
Mousa, T. M., Schiller, A. M., & Zucker, I. H. (2014). Disruption of cardiovascular circadian rhythms in mice post myocardial infarction: relationship with central angiotensin II receptor expression. Physiological reports, 2(11), . https://doi.org/10.14814/phy2.12210
Mousa, Tarek M, et al. "Disruption of cardiovascular circadian rhythms in mice post myocardial infarction: relationship with central angiotensin II receptor expression." Physiological reports vol. 2,11 (2014). doi: https://doi.org/10.14814/phy2.12210
Mousa TM, Schiller AM, Zucker IH. Disruption of cardiovascular circadian rhythms in mice post myocardial infarction: relationship with central angiotensin II receptor expression. Physiol Rep. 2014 Nov 20;2(11). doi: 10.14814/phy2.12210. Print 2014 Nov 01. PMID: 25413327; PMCID: PMC4255816.
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Diabetic Cardiomyopathy: An Immunometabolic Perspective.

Frontiers in endocrinology

Mishra PK, Ying W, Nandi SS, Bandyopadhyay GK, Patel KK, Mahata SK.
PMID: 28439258
Front Endocrinol (Lausanne). 2017 Apr 07;8:72. doi: 10.3389/fendo.2017.00072. eCollection 2017.

The heart possesses a remarkable inherent capability to adapt itself to a wide array of genetic and extrinsic factors to maintain contractile function. Failure to sustain its compensatory responses results in cardiac dysfunction, leading to cardiomyopathy. Diabetic cardiomyopathy (DCM)...

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Mishra PK, Ying W, Nandi SS, et al. Diabetic Cardiomyopathy: An Immunometabolic Perspective. Front Endocrinol (Lausanne). 2017;8:72doi: 10.3389/fendo.2017.00072.
Mishra, P. K., Ying, W., Nandi, S. S., Bandyopadhyay, G. K., Patel, K. K., & Mahata, S. K. (2017). Diabetic Cardiomyopathy: An Immunometabolic Perspective. Frontiers in endocrinology, 872. https://doi.org/10.3389/fendo.2017.00072
Mishra, Paras K, et al. "Diabetic Cardiomyopathy: An Immunometabolic Perspective." Frontiers in endocrinology vol. 8 (2017): 72. doi: https://doi.org/10.3389/fendo.2017.00072
Mishra PK, Ying W, Nandi SS, Bandyopadhyay GK, Patel KK, Mahata SK. Diabetic Cardiomyopathy: An Immunometabolic Perspective. Front Endocrinol (Lausanne). 2017 Apr 07;8:72. doi: 10.3389/fendo.2017.00072. eCollection 2017. PMID: 28439258; PMCID: PMC5384479.
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Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves.

Circulation. Heart failure

Katsurada K, Nandi SS, Sharma NM, Patel KP.
PMID: 34789005
Circ Heart Fail. 2021 Dec;14(12):e008365. doi: 10.1161/CIRCHEARTFAILURE.121.008365. Epub 2021 Nov 18.

BACKGROUND: Recent clinical studies demonstrate that SGLT2 (sodium-glucose cotransporter 2) inhibitors ameliorate heart failure (HF). The present study was conducted to assess the expression and function of renal SGLT2 and the influence of enhanced renal sympathetic tone in HF.METHODS:...

Cite
Katsurada K, Nandi SS, Sharma NM, et al. Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves. Circ Heart Fail. 2021;14(12):e008365doi: 10.1161/CIRCHEARTFAILURE.121.008365.
Katsurada, K., Nandi, S. S., Sharma, N. M., & Patel, K. P. (2021). Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves. Circulation. Heart failure, 14(12), e008365. https://doi.org/10.1161/CIRCHEARTFAILURE.121.008365
Katsurada, Kenichi, et al. "Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves." Circulation. Heart failure vol. 14,12 (2021): e008365. doi: https://doi.org/10.1161/CIRCHEARTFAILURE.121.008365
Katsurada K, Nandi SS, Sharma NM, Patel KP. Enhanced Expression and Function of Renal SGLT2 (Sodium-Glucose Cotransporter 2) in Heart Failure: Role of Renal Nerves. Circ Heart Fail. 2021 Dec;14(12):e008365. doi: 10.1161/CIRCHEARTFAILURE.121.008365. Epub 2021 Nov 18. PMID: 34789005; PMCID: PMC8692398.
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