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Showing 1 to 12 of 27 entries
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mir-34b/c and mir-449a/b/c are required for spermatogenesis, but not for the first cleavage division in mice.

Biology open

Yuan S, Tang C, Zhang Y, Wu J, Bao J, Zheng H, Xu C, Yan W.
PMID: 25617420
Biol Open. 2015 Jan 23;4(2):212-23. doi: 10.1242/bio.201410959.

Mammalian sperm are carriers of not only the paternal genome, but also the paternal epigenome in the forms of DNA methylation, retained histones and noncoding RNAs. Although paternal DNA methylation and histone retention sites have been correlated with protein-coding...

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Yuan S, Tang C, Zhang Y, et al. mir-34b/c and mir-449a/b/c are required for spermatogenesis, but not for the first cleavage division in mice. Biol Open. 2015;4(2):212-23doi: 10.1242/bio.201410959.
Yuan, S., Tang, C., Zhang, Y., Wu, J., Bao, J., Zheng, H., Xu, C., & Yan, W. (2015). mir-34b/c and mir-449a/b/c are required for spermatogenesis, but not for the first cleavage division in mice. Biology open, 4(2), 212-23. https://doi.org/10.1242/bio.201410959
Yuan, Shuiqiao, et al. "mir-34b/c and mir-449a/b/c are required for spermatogenesis, but not for the first cleavage division in mice." Biology open vol. 4,2 (2015): 212-23. doi: https://doi.org/10.1242/bio.201410959
Yuan S, Tang C, Zhang Y, Wu J, Bao J, Zheng H, Xu C, Yan W. mir-34b/c and mir-449a/b/c are required for spermatogenesis, but not for the first cleavage division in mice. Biol Open. 2015 Jan 23;4(2):212-23. doi: 10.1242/bio.201410959. PMID: 25617420; PMCID: PMC4365490.
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A Novel Striated Muscle-Specific Myosin-Blocking Drug for the Study of Neuromuscular Physiology.

Frontiers in cellular neuroscience

Heredia DJ, Schubert D, Maligireddy S, Hennig GW, Gould TW.
PMID: 27990107
Front Cell Neurosci. 2016 Dec 01;10:276. doi: 10.3389/fncel.2016.00276. eCollection 2016.

The failure to transmit neural action potentials (APs) into muscle APs is referred to as neuromuscular transmission failure (NTF). Although synaptic dysfunction occurs in a variety of neuromuscular diseases and impaired neurotransmission contributes to muscle fatigue, direct evaluation of...

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Heredia DJ, Schubert D, Maligireddy S, et al. A Novel Striated Muscle-Specific Myosin-Blocking Drug for the Study of Neuromuscular Physiology. Front Cell Neurosci. 2016;10:276doi: 10.3389/fncel.2016.00276.
Heredia, D. J., Schubert, D., Maligireddy, S., Hennig, G. W., & Gould, T. W. (2016). A Novel Striated Muscle-Specific Myosin-Blocking Drug for the Study of Neuromuscular Physiology. Frontiers in cellular neuroscience, 10276. https://doi.org/10.3389/fncel.2016.00276
Heredia, Dante J, et al. "A Novel Striated Muscle-Specific Myosin-Blocking Drug for the Study of Neuromuscular Physiology." Frontiers in cellular neuroscience vol. 10 (2016): 276. doi: https://doi.org/10.3389/fncel.2016.00276
Heredia DJ, Schubert D, Maligireddy S, Hennig GW, Gould TW. A Novel Striated Muscle-Specific Myosin-Blocking Drug for the Study of Neuromuscular Physiology. Front Cell Neurosci. 2016 Dec 01;10:276. doi: 10.3389/fncel.2016.00276. eCollection 2016. PMID: 27990107; PMCID: PMC5130989.
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Expression of the ACE2 Virus Entry Protein in the Nervus Terminalis Reveals the Potential for an Alternative Route to Brain Infection in COVID-19.

Frontiers in cellular neuroscience

Bilinska K, von Bartheld CS, Butowt R.
PMID: 34290590
Front Cell Neurosci. 2021 Jul 05;15:674123. doi: 10.3389/fncel.2021.674123. eCollection 2021.

Previous studies suggested that the SARS-CoV-2 virus may gain access to the brain by using a route along the olfactory nerve. However, there is a general consensus that the obligatory virus entry receptor, angiotensin converting enzyme 2 (ACE2), is...

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Bilinska K, von Bartheld CS, Butowt R. Expression of the ACE2 Virus Entry Protein in the Nervus Terminalis Reveals the Potential for an Alternative Route to Brain Infection in COVID-19. Front Cell Neurosci. 2021;15:674123doi: 10.3389/fncel.2021.674123.
Bilinska, K., von Bartheld, C. S., & Butowt, R. (2021). Expression of the ACE2 Virus Entry Protein in the Nervus Terminalis Reveals the Potential for an Alternative Route to Brain Infection in COVID-19. Frontiers in cellular neuroscience, 15674123. https://doi.org/10.3389/fncel.2021.674123
Bilinska, Katarzyna, et al. "Expression of the ACE2 Virus Entry Protein in the Nervus Terminalis Reveals the Potential for an Alternative Route to Brain Infection in COVID-19." Frontiers in cellular neuroscience vol. 15 (2021): 674123. doi: https://doi.org/10.3389/fncel.2021.674123
Bilinska K, von Bartheld CS, Butowt R. Expression of the ACE2 Virus Entry Protein in the Nervus Terminalis Reveals the Potential for an Alternative Route to Brain Infection in COVID-19. Front Cell Neurosci. 2021 Jul 05;15:674123. doi: 10.3389/fncel.2021.674123. eCollection 2021. PMID: 34290590; PMCID: PMC8287262.
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The Possible Role of Nonbilayer Structures in Regulating ATP Synthase Activity in Mitochondrial Membranes.

Biophysics

Gasanov SE, Kim AA, Dagda RK.
PMID: 28065984
Biophysics (Oxf). 2016 Jul;61(4):596-600. doi: 10.1134/S0006350916040084. Epub 2016 Oct 19.

The effects of temperature and of the membrane-active protein CTII on the formation of nonbilayer structures in mitochondrial membranes were studied by

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Gasanov SE, Kim AA, Dagda RK. The Possible Role of Nonbilayer Structures in Regulating ATP Synthase Activity in Mitochondrial Membranes. Biophysics (Oxf). 2016;61(4):596-600doi: 10.1134/S0006350916040084.
Gasanov, S. E., Kim, A. A., & Dagda, R. K. (2016). The Possible Role of Nonbilayer Structures in Regulating ATP Synthase Activity in Mitochondrial Membranes. Biophysics, 61(4), 596-600. https://doi.org/10.1134/S0006350916040084
Gasanov, S E, et al. "The Possible Role of Nonbilayer Structures in Regulating ATP Synthase Activity in Mitochondrial Membranes." Biophysics vol. 61,4 (2016): 596-600. doi: https://doi.org/10.1134/S0006350916040084
Gasanov SE, Kim AA, Dagda RK. The Possible Role of Nonbilayer Structures in Regulating ATP Synthase Activity in Mitochondrial Membranes. Biophysics (Oxf). 2016 Jul;61(4):596-600. doi: 10.1134/S0006350916040084. Epub 2016 Oct 19. PMID: 28065984; PMCID: PMC5215001.
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Antioxidants Protect against Arsenic Induced Mitochondrial Cardio-Toxicity.

Toxics

Pace C, Dagda R, Angermann J.
PMID: 29206204
Toxics. 2017 Dec 05;5(4). doi: 10.3390/toxics5040038.

Arsenic is a potent cardiovascular toxicant associated with numerous biomarkers of cardiovascular diseases in exposed human populations. Arsenic is also a carcinogen, yet arsenic trioxide is used as a therapeutic agent in the treatment of acute promyelotic leukemia (APL)....

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Pace C, Dagda R, Angermann J. Antioxidants Protect against Arsenic Induced Mitochondrial Cardio-Toxicity. Toxics. 2017;5(4)doi: 10.3390/toxics5040038.
Pace, C., Dagda, R., & Angermann, J. (2017). Antioxidants Protect against Arsenic Induced Mitochondrial Cardio-Toxicity. Toxics, 5(4), . https://doi.org/10.3390/toxics5040038
Pace, Clare, et al. "Antioxidants Protect against Arsenic Induced Mitochondrial Cardio-Toxicity." Toxics vol. 5,4 (2017). doi: https://doi.org/10.3390/toxics5040038
Pace C, Dagda R, Angermann J. Antioxidants Protect against Arsenic Induced Mitochondrial Cardio-Toxicity. Toxics. 2017 Dec 05;5(4). doi: 10.3390/toxics5040038. PMID: 29206204; PMCID: PMC5750566.
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Protocols for assessing mitophagy in neuronal cell lines and primary neurons.

Neuromethods

Dagda RK, Rice M.
PMID: 28603343
Neuromethods. 2017;123:249-277. doi: 10.1007/978-1-4939-6890-9_13. Epub 2017 Mar 18.

Mitochondria are organelles that regulate essential eukaryotic functions including generating energy, sequestering excess calcium, and modulating cell survival. In order for neurons to thrive, mitochondria have to be continuously replenished by maintaining autophagic-lysosomal mediated degradation of mitochondria (mitophagy) and...

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Dagda RK, Rice M. Protocols for assessing mitophagy in neuronal cell lines and primary neurons. Neuromethods. 2017;123:249-277doi: 10.1007/978-1-4939-6890-9_13.
Dagda, R. K., & Rice, M. (2017). Protocols for assessing mitophagy in neuronal cell lines and primary neurons. Neuromethods, 123249-277. https://doi.org/10.1007/978-1-4939-6890-9_13
Dagda, Ruben K, and Rice, Monica. "Protocols for assessing mitophagy in neuronal cell lines and primary neurons." Neuromethods vol. 123 (2017): 249-277. doi: https://doi.org/10.1007/978-1-4939-6890-9_13
Dagda RK, Rice M. Protocols for assessing mitophagy in neuronal cell lines and primary neurons. Neuromethods. 2017;123:249-277. doi: 10.1007/978-1-4939-6890-9_13. Epub 2017 Mar 18. PMID: 28603343; PMCID: PMC5461875.
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Correction: The WAGR syndrome gene PRRG4 is a functional homologue of the commissureless axon guidance gene.

PLoS genetics

Justice ED, Barnum SJ, Kidd T.
PMID: 29059187
PLoS Genet. 2017 Oct 23;13(10):e1007061. doi: 10.1371/journal.pgen.1007061. eCollection 2017 Oct.

[This corrects the article DOI: 10.1371/journal.pgen.1006865.].

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Justice ED, Barnum SJ, Kidd T. Correction: The WAGR syndrome gene PRRG4 is a functional homologue of the commissureless axon guidance gene. PLoS Genet. 2017;13(10):e1007061doi: 10.1371/journal.pgen.1007061.
Justice, E. D., Barnum, S. J., & Kidd, T. (2017). Correction: The WAGR syndrome gene PRRG4 is a functional homologue of the commissureless axon guidance gene. PLoS genetics, 13(10), e1007061. https://doi.org/10.1371/journal.pgen.1007061
Justice, Elizabeth D, et al. "Correction: The WAGR syndrome gene PRRG4 is a functional homologue of the commissureless axon guidance gene." PLoS genetics vol. 13,10 (2017): e1007061. doi: https://doi.org/10.1371/journal.pgen.1007061
Justice ED, Barnum SJ, Kidd T. Correction: The WAGR syndrome gene PRRG4 is a functional homologue of the commissureless axon guidance gene. PLoS Genet. 2017 Oct 23;13(10):e1007061. doi: 10.1371/journal.pgen.1007061. eCollection 2017 Oct. PMID: 29059187; PMCID: PMC5653178.
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Prediction of junior faculty success in biomedical research: comparison of metrics and effects of mentoring programs.

PeerJ

von Bartheld CS, Houmanfar R, Candido A.
PMID: 26421238
PeerJ. 2015 Sep 24;3:e1262. doi: 10.7717/peerj.1262. eCollection 2015.

Measuring and predicting the success of junior faculty is of considerable interest to faculty, academic institutions, funding agencies and faculty development and mentoring programs. Various metrics have been proposed to evaluate and predict research success and impact, such as...

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von Bartheld CS, Houmanfar R, Candido A. Prediction of junior faculty success in biomedical research: comparison of metrics and effects of mentoring programs. PeerJ. 2015;3:e1262doi: 10.7717/peerj.1262.
von Bartheld, C. S., Houmanfar, R., & Candido, A. (2015). Prediction of junior faculty success in biomedical research: comparison of metrics and effects of mentoring programs. PeerJ, 3e1262. https://doi.org/10.7717/peerj.1262
von Bartheld, Christopher S, et al. "Prediction of junior faculty success in biomedical research: comparison of metrics and effects of mentoring programs." PeerJ vol. 3 (2015): e1262. doi: https://doi.org/10.7717/peerj.1262
von Bartheld CS, Houmanfar R, Candido A. Prediction of junior faculty success in biomedical research: comparison of metrics and effects of mentoring programs. PeerJ. 2015 Sep 24;3:e1262. doi: 10.7717/peerj.1262. eCollection 2015. PMID: 26421238; PMCID: PMC4586804.
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Using crickets to introduce neurophysiology to early undergraduate students.

Journal of undergraduate neuroscience education : JUNE : a publication of FUN, Faculty for Undergraduate Neuroscience

Dagda RK, Thalhauser RM, Dagda R, Marzullo TC, Gage GJ.
PMID: 24319394
J Undergrad Neurosci Educ. 2013 Oct 15;12(1):A66-74. eCollection 2013.

Anatomy and physiology instructors often face the daunting task of teaching the principles of neurophysiology as part of a laboratory course with very limited resources. Teaching neurophysiology can be a difficult undertaking as sophisticated electrophysiology and data acquisition equipment...

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Dagda RK, Thalhauser RM, Dagda R, et al. Using crickets to introduce neurophysiology to early undergraduate students. J Undergrad Neurosci Educ. 2013;12(1):A66-74
Dagda, R. K., Thalhauser, R. M., Dagda, R., Marzullo, T. C., & Gage, G. J. (2013). Using crickets to introduce neurophysiology to early undergraduate students. Journal of undergraduate neuroscience education : JUNE : a publication of FUN, Faculty for Undergraduate Neuroscience, 12(1), A66-74.
Dagda, Ruben K, et al. "Using crickets to introduce neurophysiology to early undergraduate students." Journal of undergraduate neuroscience education : JUNE : a publication of FUN, Faculty for Undergraduate Neuroscience vol. 12,1 (2013): A66-74.
Dagda RK, Thalhauser RM, Dagda R, Marzullo TC, Gage GJ. Using crickets to introduce neurophysiology to early undergraduate students. J Undergrad Neurosci Educ. 2013 Oct 15;12(1):A66-74. eCollection 2013. PMID: 24319394; PMCID: PMC3852874.
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Use of Genetically Encoded Calcium Indicators (GECIs) Combined with Advanced Motion Tracking Techniques to Examine the Behavior of Neurons and Glia in the Enteric Nervous System of the Intact Murine Colon.

Frontiers in cellular neuroscience

Hennig GW, Gould TW, Koh SD, Corrigan RD, Heredia DJ, Shonnard MC, Smith TK.
PMID: 26617487
Front Cell Neurosci. 2015 Nov 10;9:436. doi: 10.3389/fncel.2015.00436. eCollection 2015.

Genetically encoded Ca(2+) indicators (GECIs) have been used extensively in many body systems to detect Ca(2+) transients associated with neuronal activity. Their adoption in enteric neurobiology has been slower, although they offer many advantages in terms of selectivity, signal-to-noise...

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Hennig GW, Gould TW, Koh SD, et al. Use of Genetically Encoded Calcium Indicators (GECIs) Combined with Advanced Motion Tracking Techniques to Examine the Behavior of Neurons and Glia in the Enteric Nervous System of the Intact Murine Colon. Front Cell Neurosci. 2015;9:436doi: 10.3389/fncel.2015.00436.
Hennig, G. W., Gould, T. W., Koh, S. D., Corrigan, R. D., Heredia, D. J., Shonnard, M. C., & Smith, T. K. (2015). Use of Genetically Encoded Calcium Indicators (GECIs) Combined with Advanced Motion Tracking Techniques to Examine the Behavior of Neurons and Glia in the Enteric Nervous System of the Intact Murine Colon. Frontiers in cellular neuroscience, 9436. https://doi.org/10.3389/fncel.2015.00436
Hennig, Grant W, et al. "Use of Genetically Encoded Calcium Indicators (GECIs) Combined with Advanced Motion Tracking Techniques to Examine the Behavior of Neurons and Glia in the Enteric Nervous System of the Intact Murine Colon." Frontiers in cellular neuroscience vol. 9 (2015): 436. doi: https://doi.org/10.3389/fncel.2015.00436
Hennig GW, Gould TW, Koh SD, Corrigan RD, Heredia DJ, Shonnard MC, Smith TK. Use of Genetically Encoded Calcium Indicators (GECIs) Combined with Advanced Motion Tracking Techniques to Examine the Behavior of Neurons and Glia in the Enteric Nervous System of the Intact Murine Colon. Front Cell Neurosci. 2015 Nov 10;9:436. doi: 10.3389/fncel.2015.00436. eCollection 2015. PMID: 26617487; PMCID: PMC4639702.
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Expression of the ACE2 virus entry protein in the nervus terminalis reveals the potential for an alternative route to brain infection in COVID-19.

bioRxiv : the preprint server for biology

Bilinska K, von Bartheld CS, Butowt R.
PMID: 33880469
bioRxiv. 2021 Jun 07; doi: 10.1101/2021.04.11.439398.

Previous studies suggested that the SARS-CoV-2 virus may gain access to the brain by using a route along the olfactory nerve. However, there is a general consensus that the obligatory virus entry receptor, angiotensin converting enzyme 2 (ACE2), is...

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Bilinska K, von Bartheld CS, Butowt R. Expression of the ACE2 virus entry protein in the nervus terminalis reveals the potential for an alternative route to brain infection in COVID-19. bioRxiv. 2021;doi: 10.1101/2021.04.11.439398.
Bilinska, K., von Bartheld, C. S., & Butowt, R. (2021). Expression of the ACE2 virus entry protein in the nervus terminalis reveals the potential for an alternative route to brain infection in COVID-19. bioRxiv : the preprint server for biology, . https://doi.org/10.1101/2021.04.11.439398
Bilinska, Katarzyna, et al. "Expression of the ACE2 virus entry protein in the nervus terminalis reveals the potential for an alternative route to brain infection in COVID-19." bioRxiv : the preprint server for biology vol. (2021). doi: https://doi.org/10.1101/2021.04.11.439398
Bilinska K, von Bartheld CS, Butowt R. Expression of the ACE2 virus entry protein in the nervus terminalis reveals the potential for an alternative route to brain infection in COVID-19. bioRxiv. 2021 Jun 07; doi: 10.1101/2021.04.11.439398. PMID: 33880469; PMCID: PMC8057234.
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Mitochondrial PKA Is Neuroprotective in a Cell Culture Model of Alzheimer's Disease.

Molecular neurobiology

Banerjee TD, Reihl K, Swain M, Torres M, Dagda RK.
PMID: 33624140
Mol Neurobiol. 2021 Jul;58(7):3071-3083. doi: 10.1007/s12035-021-02333-w. Epub 2021 Feb 23.

Alzheimer's disease (AD) is a neurodegenerative disease characterized by progressive memory loss and cognitive decline. In hippocampal neurons, the pathological features of AD include the accumulation of extracellular amyloid-beta peptide (Aβ) accompanied by oxidative stress, mitochondrial dysfunction, and neuron...

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Banerjee TD, Reihl K, Swain M, et al. Mitochondrial PKA Is Neuroprotective in a Cell Culture Model of Alzheimer's Disease. Mol Neurobiol. 2021;58(7):3071-3083doi: 10.1007/s12035-021-02333-w.
Banerjee, T. D., Reihl, K., Swain, M., Torres, M., & Dagda, R. K. (2021). Mitochondrial PKA Is Neuroprotective in a Cell Culture Model of Alzheimer's Disease. Molecular neurobiology, 58(7), 3071-3083. https://doi.org/10.1007/s12035-021-02333-w
Banerjee, Tania Das, et al. "Mitochondrial PKA Is Neuroprotective in a Cell Culture Model of Alzheimer's Disease." Molecular neurobiology vol. 58,7 (2021): 3071-3083. doi: https://doi.org/10.1007/s12035-021-02333-w
Banerjee TD, Reihl K, Swain M, Torres M, Dagda RK. Mitochondrial PKA Is Neuroprotective in a Cell Culture Model of Alzheimer's Disease. Mol Neurobiol. 2021 Jul;58(7):3071-3083. doi: 10.1007/s12035-021-02333-w. Epub 2021 Feb 23. PMID: 33624140; PMCID: PMC8260456.
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Showing 1 to 12 of 27 entries