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Showing 1 to 12 of 71 entries
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Menopause and Ischemic Stroke: A Brief Review.

MOJ toxicology

Shekhar S, Travis OK, He X, Roman RJ, Fan F.
PMID: 28936482
MOJ Toxicol. 2017;3(4). doi: 10.15406/mojt.2017.03.00059. Epub 2017 Aug 10.

Ischemic stroke is one of the leading causes of morbidity and mortality worldwide. Females are protected against stroke before the onset of menopause. Menopause results in increased incidence of stroke when compared to men. The mechanisms of these differences...

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Shekhar S, Travis OK, He X, et al. Menopause and Ischemic Stroke: A Brief Review. MOJ Toxicol. 2017;3(4)doi: 10.15406/mojt.2017.03.00059.
Shekhar, S., Travis, O. K., He, X., Roman, R. J., & Fan, F. (2017). Menopause and Ischemic Stroke: A Brief Review. MOJ toxicology, 3(4), . https://doi.org/10.15406/mojt.2017.03.00059
Shekhar, Shashank, et al. "Menopause and Ischemic Stroke: A Brief Review." MOJ toxicology vol. 3,4 (2017). doi: https://doi.org/10.15406/mojt.2017.03.00059
Shekhar S, Travis OK, He X, Roman RJ, Fan F. Menopause and Ischemic Stroke: A Brief Review. MOJ Toxicol. 2017;3(4). doi: 10.15406/mojt.2017.03.00059. Epub 2017 Aug 10. PMID: 28936482; PMCID: PMC5604887.
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Obesity, hypertension, and chronic kidney disease.

International journal of nephrology and renovascular disease

Hall ME, do Carmo JM, da Silva AA, Juncos LA, Wang Z, Hall JE.
PMID: 24600241
Int J Nephrol Renovasc Dis. 2014 Feb 18;7:75-88. doi: 10.2147/IJNRD.S39739. eCollection 2014.

Obesity is a major risk factor for essential hypertension, diabetes, and other comorbid conditions that contribute to development of chronic kidney disease. Obesity raises blood pressure by increasing renal tubular sodium reabsorption, impairing pressure natriuresis, and causing volume expansion...

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Hall ME, do Carmo JM, da Silva AA, et al. Obesity, hypertension, and chronic kidney disease. Int J Nephrol Renovasc Dis. 2014;7:75-88doi: 10.2147/IJNRD.S39739.
Hall, M. E., do Carmo, J. M., da Silva, A. A., Juncos, L. A., Wang, Z., & Hall, J. E. (2014). Obesity, hypertension, and chronic kidney disease. International journal of nephrology and renovascular disease, 775-88. https://doi.org/10.2147/IJNRD.S39739
Hall, Michael E, et al. "Obesity, hypertension, and chronic kidney disease." International journal of nephrology and renovascular disease vol. 7 (2014): 75-88. doi: https://doi.org/10.2147/IJNRD.S39739
Hall ME, do Carmo JM, da Silva AA, Juncos LA, Wang Z, Hall JE. Obesity, hypertension, and chronic kidney disease. Int J Nephrol Renovasc Dis. 2014 Feb 18;7:75-88. doi: 10.2147/IJNRD.S39739. eCollection 2014. PMID: 24600241; PMCID: PMC3933708.
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Coilin association with Box C/D scaRNA suggests a direct role for the Cajal body marker protein in scaRNP biogenesis.

Biology open

Enwerem II, Velma V, Broome HJ, Kuna M, Begum RA, Hebert MD.
PMID: 24659245
Biol Open. 2014 Apr 15;3(4):240-9. doi: 10.1242/bio.20147443.

Spliceosomal small nuclear ribonucleoproteins (snRNPs) are enriched in the Cajal body (CB). Guide RNAs, known as small Cajal body-specific RNAs (scaRNAs), direct modification of the small nuclear RNA (snRNA) component of the snRNP. The protein WRAP53 binds a sequence...

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Enwerem II, Velma V, Broome HJ, et al. Coilin association with Box C/D scaRNA suggests a direct role for the Cajal body marker protein in scaRNP biogenesis. Biol Open. 2014;3(4):240-9doi: 10.1242/bio.20147443.
Enwerem, I. I., Velma, V., Broome, H. J., Kuna, M., Begum, R. A., & Hebert, M. D. (2014). Coilin association with Box C/D scaRNA suggests a direct role for the Cajal body marker protein in scaRNP biogenesis. Biology open, 3(4), 240-9. https://doi.org/10.1242/bio.20147443
Enwerem, Isioma I, et al. "Coilin association with Box C/D scaRNA suggests a direct role for the Cajal body marker protein in scaRNP biogenesis." Biology open vol. 3,4 (2014): 240-9. doi: https://doi.org/10.1242/bio.20147443
Enwerem II, Velma V, Broome HJ, Kuna M, Begum RA, Hebert MD. Coilin association with Box C/D scaRNA suggests a direct role for the Cajal body marker protein in scaRNP biogenesis. Biol Open. 2014 Apr 15;3(4):240-9. doi: 10.1242/bio.20147443. PMID: 24659245; PMCID: PMC3988793.
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Simulating a virtual population's sensitivity to salt and uninephrectomy.

Interface focus

Clemmer JS, Hester RL, Pruett WA.
PMID: 29285341
Interface Focus. 2018 Feb 06;8(1):20160134. doi: 10.1098/rsfs.2016.0134. Epub 2017 Dec 15.

Salt sensitivity, with or without concomitant hypertension, is associated with increased mortality. Reduced functional renal mass plays an important role in causing salt-sensitive hypertension for many individuals. Factors that are important in the condition of decreased renal mass and...

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Clemmer JS, Hester RL, Pruett WA. Simulating a virtual population's sensitivity to salt and uninephrectomy. Interface Focus. 2017;8(1):20160134doi: 10.1098/rsfs.2016.0134.
Clemmer, J. S., Hester, R. L., & Pruett, W. A. (2018). Simulating a virtual population's sensitivity to salt and uninephrectomy. Interface focus, 8(1), 20160134. https://doi.org/10.1098/rsfs.2016.0134
Clemmer, John S, et al. "Simulating a virtual population's sensitivity to salt and uninephrectomy." Interface focus vol. 8,1 (2018): 20160134. doi: https://doi.org/10.1098/rsfs.2016.0134
Clemmer JS, Hester RL, Pruett WA. Simulating a virtual population's sensitivity to salt and uninephrectomy. Interface Focus. 2018 Feb 06;8(1):20160134. doi: 10.1098/rsfs.2016.0134. Epub 2017 Dec 15. PMID: 29285341; PMCID: PMC5740217.
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Elastin-Like Polypeptide: VEGF-B Fusion Protein for Treatment of Preeclampsia.

Hypertension (Dallas, Tex. : 1979)

Waller JP, Howell JA, Peterson H, George EM, Bidwell GL.
PMID: 34719237
Hypertension. 2021 Dec;78(6):1888-1901. doi: 10.1161/HYPERTENSIONAHA.121.17713. Epub 2021 Nov 01.

[Figure: see text].

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Waller JP, Howell JA, Peterson H, et al. Elastin-Like Polypeptide: VEGF-B Fusion Protein for Treatment of Preeclampsia. Hypertension. 2021;78(6):1888-1901doi: 10.1161/HYPERTENSIONAHA.121.17713.
Waller, J. P., Howell, J. A., Peterson, H., George, E. M., & Bidwell, G. L. (2021). Elastin-Like Polypeptide: VEGF-B Fusion Protein for Treatment of Preeclampsia. Hypertension (Dallas, Tex. : 1979), 78(6), 1888-1901. https://doi.org/10.1161/HYPERTENSIONAHA.121.17713
Waller, Jamarius P, et al. "Elastin-Like Polypeptide: VEGF-B Fusion Protein for Treatment of Preeclampsia." Hypertension (Dallas, Tex. : 1979) vol. 78,6 (2021): 1888-1901. doi: https://doi.org/10.1161/HYPERTENSIONAHA.121.17713
Waller JP, Howell JA, Peterson H, George EM, Bidwell GL. Elastin-Like Polypeptide: VEGF-B Fusion Protein for Treatment of Preeclampsia. Hypertension. 2021 Dec;78(6):1888-1901. doi: 10.1161/HYPERTENSIONAHA.121.17713. Epub 2021 Nov 01. PMID: 34719237; PMCID: PMC8585700.
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The Saltpan Microbiome is Structured by Sediment Depth and Minimally Influenced by Variable Hydration.

Microorganisms

Weingarten EA, Lawson LA, Jackson CR.
PMID: 32276533
Microorganisms. 2020 Apr 08;8(4). doi: 10.3390/microorganisms8040538.

Saltpans are a class of ephemeral wetland characterized by alternating periods of inundation, rising salinity, and desiccation. We obtained soil cores from a saltpan on the Mississippi Gulf coast in both the inundated and desiccated state. The microbiomes of...

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Weingarten EA, Lawson LA, Jackson CR. The Saltpan Microbiome is Structured by Sediment Depth and Minimally Influenced by Variable Hydration. Microorganisms. 2020;8(4)doi: 10.3390/microorganisms8040538.
Weingarten, E. A., Lawson, L. A., & Jackson, C. R. (2020). The Saltpan Microbiome is Structured by Sediment Depth and Minimally Influenced by Variable Hydration. Microorganisms, 8(4), . https://doi.org/10.3390/microorganisms8040538
Weingarten, Eric A, et al. "The Saltpan Microbiome is Structured by Sediment Depth and Minimally Influenced by Variable Hydration." Microorganisms vol. 8,4 (2020). doi: https://doi.org/10.3390/microorganisms8040538
Weingarten EA, Lawson LA, Jackson CR. The Saltpan Microbiome is Structured by Sediment Depth and Minimally Influenced by Variable Hydration. Microorganisms. 2020 Apr 08;8(4). doi: 10.3390/microorganisms8040538. PMID: 32276533; PMCID: PMC7232383.
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Development and Validation of a Prognostic Nomogram to Predict Cancer-Specific Survival in Adult Patients With Pineoblastoma.

Frontiers in oncology

Jing Y, Deng W, Zhang H, Jiang Y, Dong Z, Fan F, Sun P.
PMID: 32793463
Front Oncol. 2020 Jul 24;10:1021. doi: 10.3389/fonc.2020.01021. eCollection 2020.

Pineoblastoma (PB) is a rare neoplasm of the central nervous system. This analysis aimed to identify factors and establish a predictive model for the prognosis of adult patients with PB. Data for 213 adult patients with PB (Surveillance, Epidemiology,...

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Jing Y, Deng W, Zhang H, et al. Development and Validation of a Prognostic Nomogram to Predict Cancer-Specific Survival in Adult Patients With Pineoblastoma. Front Oncol. 2020;10:1021doi: 10.3389/fonc.2020.01021.
Jing, Y., Deng, W., Zhang, H., Jiang, Y., Dong, Z., Fan, F., & Sun, P. (2020). Development and Validation of a Prognostic Nomogram to Predict Cancer-Specific Survival in Adult Patients With Pineoblastoma. Frontiers in oncology, 101021. https://doi.org/10.3389/fonc.2020.01021
Jing, Yajun, et al. "Development and Validation of a Prognostic Nomogram to Predict Cancer-Specific Survival in Adult Patients With Pineoblastoma." Frontiers in oncology vol. 10 (2020): 1021. doi: https://doi.org/10.3389/fonc.2020.01021
Jing Y, Deng W, Zhang H, Jiang Y, Dong Z, Fan F, Sun P. Development and Validation of a Prognostic Nomogram to Predict Cancer-Specific Survival in Adult Patients With Pineoblastoma. Front Oncol. 2020 Jul 24;10:1021. doi: 10.3389/fonc.2020.01021. eCollection 2020. PMID: 32793463; PMCID: PMC7393244.
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Targeting Heme Oxygenase-1 in Cardiovascular and Kidney Disease.

Antioxidants (Basel, Switzerland)

Drummond HA, Mitchell ZL, Abraham NG, Stec DE.
PMID: 31216709
Antioxidants (Basel). 2019 Jun 18;8(6). doi: 10.3390/antiox8060181.

Heme oxygenase (HO) plays an important role in the cardiovascular system. It is involved in many physiological and pathophysiological processes in all organs of the cardiovascular system. From the regulation of blood pressure and blood flow to the adaptive...

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Drummond HA, Mitchell ZL, Abraham NG, et al. Targeting Heme Oxygenase-1 in Cardiovascular and Kidney Disease. Antioxidants (Basel). 2019;8(6)doi: 10.3390/antiox8060181.
Drummond, H. A., Mitchell, Z. L., Abraham, N. G., & Stec, D. E. (2019). Targeting Heme Oxygenase-1 in Cardiovascular and Kidney Disease. Antioxidants (Basel, Switzerland), 8(6), . https://doi.org/10.3390/antiox8060181
Drummond, Heather A, et al. "Targeting Heme Oxygenase-1 in Cardiovascular and Kidney Disease." Antioxidants (Basel, Switzerland) vol. 8,6 (2019). doi: https://doi.org/10.3390/antiox8060181
Drummond HA, Mitchell ZL, Abraham NG, Stec DE. Targeting Heme Oxygenase-1 in Cardiovascular and Kidney Disease. Antioxidants (Basel). 2019 Jun 18;8(6). doi: 10.3390/antiox8060181. PMID: 31216709; PMCID: PMC6617021.
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Capillary Stalling: A Mechanism of Decreased Cerebral Blood Flow in AD/ADRD.

Journal of experimental neurology

Crumpler R, Roman RJ, Fan F.
PMID: 35028643
J Exp Neurol. 2021;2(4):149-153. doi: 10.33696/neurol.2.048.

Alzheimer's Disease (AD) and Alzheimer's Disease-Related Dementias (ADRD) are debilitating conditions that are highly associated with aging populations, especially those with comorbidities such as diabetes and hypertension. In addition to the classical pathological findings of AD, such as beta-amyloid...

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Crumpler R, Roman RJ, Fan F. Capillary Stalling: A Mechanism of Decreased Cerebral Blood Flow in AD/ADRD. J Exp Neurol. 2021;2(4):149-153doi: 10.33696/neurol.2.048.
Crumpler, R., Roman, R. J., & Fan, F. (2021). Capillary Stalling: A Mechanism of Decreased Cerebral Blood Flow in AD/ADRD. Journal of experimental neurology, 2(4), 149-153. https://doi.org/10.33696/neurol.2.048
Crumpler, Reece, et al. "Capillary Stalling: A Mechanism of Decreased Cerebral Blood Flow in AD/ADRD." Journal of experimental neurology vol. 2,4 (2021): 149-153. doi: https://doi.org/10.33696/neurol.2.048
Crumpler R, Roman RJ, Fan F. Capillary Stalling: A Mechanism of Decreased Cerebral Blood Flow in AD/ADRD. J Exp Neurol. 2021;2(4):149-153. doi: 10.33696/neurol.2.048. PMID: 35028643; PMCID: PMC8754422.
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Magnesium Sulfate Prevents Placental Ischemia-Induced Increases in Brain Water Content and Cerebrospinal Fluid Cytokines in Pregnant Rats.

Frontiers in neuroscience

Zhang LW, Warrington JP.
PMID: 28008305
Front Neurosci. 2016 Dec 08;10:561. doi: 10.3389/fnins.2016.00561. eCollection 2016.

Magnesium sulfate (MgSO

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Zhang LW, Warrington JP. Magnesium Sulfate Prevents Placental Ischemia-Induced Increases in Brain Water Content and Cerebrospinal Fluid Cytokines in Pregnant Rats. Front Neurosci. 2016;10:561doi: 10.3389/fnins.2016.00561.
Zhang, L. W., & Warrington, J. P. (2016). Magnesium Sulfate Prevents Placental Ischemia-Induced Increases in Brain Water Content and Cerebrospinal Fluid Cytokines in Pregnant Rats. Frontiers in neuroscience, 10561. https://doi.org/10.3389/fnins.2016.00561
Zhang, Linda W, and Warrington, Junie P. "Magnesium Sulfate Prevents Placental Ischemia-Induced Increases in Brain Water Content and Cerebrospinal Fluid Cytokines in Pregnant Rats." Frontiers in neuroscience vol. 10 (2016): 561. doi: https://doi.org/10.3389/fnins.2016.00561
Zhang LW, Warrington JP. Magnesium Sulfate Prevents Placental Ischemia-Induced Increases in Brain Water Content and Cerebrospinal Fluid Cytokines in Pregnant Rats. Front Neurosci. 2016 Dec 08;10:561. doi: 10.3389/fnins.2016.00561. eCollection 2016. PMID: 28008305; PMCID: PMC5143678.
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Transient Receptor Potential Cation Channel 6 (TRPC6) Contributes to Kidney Injury Induced by Diabetes and Hypertension.

American journal of physiology. Renal physiology

Wang Z, Fu Y, do Carmo JM, da Silva AA, Li X, Mouton AJ, Omoto ACM, Sears J, Hall JE.
PMID: 34866402
Am J Physiol Renal Physiol. 2021 Dec 06; doi: 10.1152/ajprenal.00296.2021. Epub 2021 Dec 06.

Diabetes (DM) and hypertension (HTN) are major risk factors for chronic kidney injury, together accounting for >70% of end-stage renal disease. In this study, we assessed whether DM and HTN interact synergistically to promote kidney dysfunction and if Transient...

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Wang Z, Fu Y, do Carmo JM, et al. Transient Receptor Potential Cation Channel 6 (TRPC6) Contributes to Kidney Injury Induced by Diabetes and Hypertension. Am J Physiol Renal Physiol. 2021;doi: 10.1152/ajprenal.00296.2021.
Wang, Z., Fu, Y., do Carmo, J. M., da Silva, A. A., Li, X., Mouton, A. J., Omoto, A. C. M., Sears, J., & Hall, J. E. (2021). Transient Receptor Potential Cation Channel 6 (TRPC6) Contributes to Kidney Injury Induced by Diabetes and Hypertension. American journal of physiology. Renal physiology, . https://doi.org/10.1152/ajprenal.00296.2021
Wang, Zhen, et al. "Transient Receptor Potential Cation Channel 6 (TRPC6) Contributes to Kidney Injury Induced by Diabetes and Hypertension." American journal of physiology. Renal physiology vol. (2021). doi: https://doi.org/10.1152/ajprenal.00296.2021
Wang Z, Fu Y, do Carmo JM, da Silva AA, Li X, Mouton AJ, Omoto ACM, Sears J, Hall JE. Transient Receptor Potential Cation Channel 6 (TRPC6) Contributes to Kidney Injury Induced by Diabetes and Hypertension. Am J Physiol Renal Physiol. 2021 Dec 06; doi: 10.1152/ajprenal.00296.2021. Epub 2021 Dec 06. PMID: 34866402.
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Duration and magnitude of bidirectional fluctuation in blood pressure: the link between cerebrovascular dysfunction and cognitive impairment following spinal cord injury.

Journal of neurobiology and physiology

Wang S, Roman RJ, Fan F.
PMID: 33336208
J Neurobiol Physiol. 2020;2(1):15-18. doi: 10.46439/neurobiology.2.008.

Individuals with spinal cord injury (SCI) have a significantly increased risk for cognitive impairment that is associated with cerebrovascular remodeling and endothelial dysfunction. The sub-acute stage following high thoracic SCI is characterized by increased fibrosis and stiffness of cerebral...

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Wang S, Roman RJ, Fan F. Duration and magnitude of bidirectional fluctuation in blood pressure: the link between cerebrovascular dysfunction and cognitive impairment following spinal cord injury. J Neurobiol Physiol. 2020;2(1):15-18doi: 10.46439/neurobiology.2.008.
Wang, S., Roman, R. J., & Fan, F. (2020). Duration and magnitude of bidirectional fluctuation in blood pressure: the link between cerebrovascular dysfunction and cognitive impairment following spinal cord injury. Journal of neurobiology and physiology, 2(1), 15-18. https://doi.org/10.46439/neurobiology.2.008
Wang, Shaoxun, et al. "Duration and magnitude of bidirectional fluctuation in blood pressure: the link between cerebrovascular dysfunction and cognitive impairment following spinal cord injury." Journal of neurobiology and physiology vol. 2,1 (2020): 15-18. doi: https://doi.org/10.46439/neurobiology.2.008
Wang S, Roman RJ, Fan F. Duration and magnitude of bidirectional fluctuation in blood pressure: the link between cerebrovascular dysfunction and cognitive impairment following spinal cord injury. J Neurobiol Physiol. 2020;2(1):15-18. doi: 10.46439/neurobiology.2.008. PMID: 33336208; PMCID: PMC7739907.
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Showing 1 to 12 of 71 entries