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Regulatory T-Cell-Mediated Suppression of Conventional T-Cells and Dendritic Cells by Different cAMP Intracellular Pathways.

Frontiers in immunology

Rueda CM, Jackson CM, Chougnet CA.
PMID: 27313580
Front Immunol. 2016 Jun 02;7:216. doi: 10.3389/fimmu.2016.00216. eCollection 2016.

Regulatory T-cells (Tregs) mediate their suppressive action by acting directly on conventional T-cells (Tcons) or dendritic cells (DCs). One mechanism of Treg suppression is the increase of cyclic adenosine 3',5'-monophosphate (cAMP) levels in target cells. Tregs utilize cAMP to...

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Rueda CM, Jackson CM, Chougnet CA. Regulatory T-Cell-Mediated Suppression of Conventional T-Cells and Dendritic Cells by Different cAMP Intracellular Pathways. Front Immunol. 2016;7:216doi: 10.3389/fimmu.2016.00216.
Rueda, C. M., Jackson, C. M., & Chougnet, C. A. (2016). Regulatory T-Cell-Mediated Suppression of Conventional T-Cells and Dendritic Cells by Different cAMP Intracellular Pathways. Frontiers in immunology, 7216. https://doi.org/10.3389/fimmu.2016.00216
Rueda, Cesar M, et al. "Regulatory T-Cell-Mediated Suppression of Conventional T-Cells and Dendritic Cells by Different cAMP Intracellular Pathways." Frontiers in immunology vol. 7 (2016): 216. doi: https://doi.org/10.3389/fimmu.2016.00216
Rueda CM, Jackson CM, Chougnet CA. Regulatory T-Cell-Mediated Suppression of Conventional T-Cells and Dendritic Cells by Different cAMP Intracellular Pathways. Front Immunol. 2016 Jun 02;7:216. doi: 10.3389/fimmu.2016.00216. eCollection 2016. PMID: 27313580; PMCID: PMC4889573.
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PARP1 activation increases expression of modified tumor suppressors and pathways underlying development of aggressive hepatoblastoma.

Communications biology

Valanejad L, Cast A, Wright M, Bissig KD, Karns R, Weirauch MT, Timchenko N.
PMID: 30271949
Commun Biol. 2018 Jun 11;1:67. doi: 10.1038/s42003-018-0077-8. eCollection 2018.

Hepatoblastoma (HBL) is a pediatric liver cancer that affects children under the age of three. Reduction of tumor suppressor proteins (TSPs) is commonly seen in liver cancer. However, in our studies we find that aggressive, chemo-resistant HBLs exhibit an...

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Valanejad L, Cast A, Wright M, et al. PARP1 activation increases expression of modified tumor suppressors and pathways underlying development of aggressive hepatoblastoma. Commun Biol. 2018;1:67doi: 10.1038/s42003-018-0077-8.
Valanejad, L., Cast, A., Wright, M., Bissig, K. D., Karns, R., Weirauch, M. T., & Timchenko, N. (2018). PARP1 activation increases expression of modified tumor suppressors and pathways underlying development of aggressive hepatoblastoma. Communications biology, 167. https://doi.org/10.1038/s42003-018-0077-8
Valanejad, Leila, et al. "PARP1 activation increases expression of modified tumor suppressors and pathways underlying development of aggressive hepatoblastoma." Communications biology vol. 1 (2018): 67. doi: https://doi.org/10.1038/s42003-018-0077-8
Valanejad L, Cast A, Wright M, Bissig KD, Karns R, Weirauch MT, Timchenko N. PARP1 activation increases expression of modified tumor suppressors and pathways underlying development of aggressive hepatoblastoma. Commun Biol. 2018 Jun 11;1:67. doi: 10.1038/s42003-018-0077-8. eCollection 2018. PMID: 30271949; PMCID: PMC6123626.
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Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci.

Genome research

Hong T, Parameswaran S, Donmez OA, Miller D, Forney C, Lape M, Saint Just Ribeiro M, Liang J, Edsall LE, Magnusen AF, Miller W, Chepelev I, Harley JB, Zhao B, Kottyan LC, Weirauch MT.
PMID: 34799401
Genome Res. 2021 Nov 19; doi: 10.1101/gr.264705.120. Epub 2021 Nov 19.

The interplay between environmental and genetic factors plays a key role in the development of many autoimmune diseases. In particular, the Epstein-Barr virus (EBV) is an established contributor to multiple sclerosis, lupus, and other disorders. Previously, we showed that...

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Hong T, Parameswaran S, Donmez OA, et al. Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci. Genome Res. 2021;doi: 10.1101/gr.264705.120.
Hong, T., Parameswaran, S., Donmez, O. A., Miller, D., Forney, C., Lape, M., Saint Just Ribeiro, M., Liang, J., Edsall, L. E., Magnusen, A. F., Miller, W., Chepelev, I., Harley, J. B., Zhao, B., Kottyan, L. C., & Weirauch, M. T. (2021). Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci. Genome research, . https://doi.org/10.1101/gr.264705.120
Hong, Ted, et al. "Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci." Genome research vol. (2021). doi: https://doi.org/10.1101/gr.264705.120
Hong T, Parameswaran S, Donmez OA, Miller D, Forney C, Lape M, Saint Just Ribeiro M, Liang J, Edsall LE, Magnusen AF, Miller W, Chepelev I, Harley JB, Zhao B, Kottyan LC, Weirauch MT. Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci. Genome Res. 2021 Nov 19; doi: 10.1101/gr.264705.120. Epub 2021 Nov 19. PMID: 34799401; PMCID: PMC8647835.
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A Compartmentalized Reduction in Membrane-Proximal Calmodulin Reduces the Immune Surveillance Capabilities of CD8.

Frontiers in pharmacology

Chimote AA, Gawali VS, Newton HS, Wise-Draper TM, Conforti L.
PMID: 32184726
Front Pharmacol. 2020 Feb 28;11:143. doi: 10.3389/fphar.2020.00143. eCollection 2020.

The limited ability of cytotoxic CD8

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Chimote AA, Gawali VS, Newton HS, et al. A Compartmentalized Reduction in Membrane-Proximal Calmodulin Reduces the Immune Surveillance Capabilities of CD8. Front Pharmacol. 2020;11:143doi: 10.3389/fphar.2020.00143.
Chimote, A. A., Gawali, V. S., Newton, H. S., Wise-Draper, T. M., & Conforti, L. (2020). A Compartmentalized Reduction in Membrane-Proximal Calmodulin Reduces the Immune Surveillance Capabilities of CD8. Frontiers in pharmacology, 11143. https://doi.org/10.3389/fphar.2020.00143
Chimote, Ameet A, et al. "A Compartmentalized Reduction in Membrane-Proximal Calmodulin Reduces the Immune Surveillance Capabilities of CD8." Frontiers in pharmacology vol. 11 (2020): 143. doi: https://doi.org/10.3389/fphar.2020.00143
Chimote AA, Gawali VS, Newton HS, Wise-Draper TM, Conforti L. A Compartmentalized Reduction in Membrane-Proximal Calmodulin Reduces the Immune Surveillance Capabilities of CD8. Front Pharmacol. 2020 Feb 28;11:143. doi: 10.3389/fphar.2020.00143. eCollection 2020. PMID: 32184726; PMCID: PMC7059094.
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Discovery of tear biomarkers in children with chronic non-infectious anterior uveitis: a pilot study.

Journal of ophthalmic inflammation and infection

Angeles-Han ST, Yeh S, Patel P, Duong D, Jenkins K, Rouster-Stevens KA, Altaye M, Fall N, Thornton S, Prahalad S, Holland GN.
PMID: 30327966
J Ophthalmic Inflamm Infect. 2018 Oct 16;8(1):17. doi: 10.1186/s12348-018-0156-5.

BACKGROUND: Biomarkers in easily obtained specimens that accurately predict uveitis in children with juvenile idiopathic arthritis (JIA) are needed. Aqueous humor has been studied for biomarkers, but is not routinely available. We evaluated tears from children with chronic anterior...

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Angeles-Han ST, Yeh S, Patel P, et al. Discovery of tear biomarkers in children with chronic non-infectious anterior uveitis: a pilot study. J Ophthalmic Inflamm Infect. 2018;8(1):17doi: 10.1186/s12348-018-0156-5.
Angeles-Han, S. T., Yeh, S., Patel, P., Duong, D., Jenkins, K., Rouster-Stevens, K. A., Altaye, M., Fall, N., Thornton, S., Prahalad, S., & Holland, G. N. (2018). Discovery of tear biomarkers in children with chronic non-infectious anterior uveitis: a pilot study. Journal of ophthalmic inflammation and infection, 8(1), 17. https://doi.org/10.1186/s12348-018-0156-5
Angeles-Han, Sheila T, et al. "Discovery of tear biomarkers in children with chronic non-infectious anterior uveitis: a pilot study." Journal of ophthalmic inflammation and infection vol. 8,1 (2018): 17. doi: https://doi.org/10.1186/s12348-018-0156-5
Angeles-Han ST, Yeh S, Patel P, Duong D, Jenkins K, Rouster-Stevens KA, Altaye M, Fall N, Thornton S, Prahalad S, Holland GN. Discovery of tear biomarkers in children with chronic non-infectious anterior uveitis: a pilot study. J Ophthalmic Inflamm Infect. 2018 Oct 16;8(1):17. doi: 10.1186/s12348-018-0156-5. PMID: 30327966; PMCID: PMC6191408.
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Evaluation of the MC4R gene across eMERGE network identifies many unreported obesity-associated variants.

International journal of obesity (2005)

Namjou B, Stanaway IB, Lingren T, Mentch FD, Benoit B, Dikilitas O, Niu X, Shang N, Shoemaker AH, Carey DJ, Mirshahi T, Singh R, Nestor JG, Hakonarson H, Denny JC, Crosslin DR, Jarvik GP, Kullo IJ, Williams MS, Harley JB.
PMID: 32952152
Int J Obes (Lond). 2021 Jan;45(1):155-169. doi: 10.1038/s41366-020-00675-4. Epub 2020 Sep 20.

BACKGROUND/OBJECTIVES: Melanocortin-4 receptor (MC4R) plays an essential role in food intake and energy homeostasis. More than 170 MC4R variants have been described over the past two decades, with conflicting reports regarding the prevalence and phenotypic effects of these variants...

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Namjou B, Stanaway IB, Lingren T, et al. Evaluation of the MC4R gene across eMERGE network identifies many unreported obesity-associated variants. Int J Obes (Lond). 2020;45(1):155-169doi: 10.1038/s41366-020-00675-4.
Namjou, B., Stanaway, I. B., Lingren, T., Mentch, F. D., Benoit, B., Dikilitas, O., Niu, X., Shang, N., Shoemaker, A. H., Carey, D. J., Mirshahi, T., Singh, R., Nestor, J. G., Hakonarson, H., Denny, J. C., Crosslin, D. R., Jarvik, G. P., Kullo, I. J., Williams, M. S., & Harley, J. B. (2021). Evaluation of the MC4R gene across eMERGE network identifies many unreported obesity-associated variants. International journal of obesity (2005), 45(1), 155-169. https://doi.org/10.1038/s41366-020-00675-4
Namjou, Bahram, et al. "Evaluation of the MC4R gene across eMERGE network identifies many unreported obesity-associated variants." International journal of obesity (2005) vol. 45,1 (2021): 155-169. doi: https://doi.org/10.1038/s41366-020-00675-4
Namjou B, Stanaway IB, Lingren T, Mentch FD, Benoit B, Dikilitas O, Niu X, Shang N, Shoemaker AH, Carey DJ, Mirshahi T, Singh R, Nestor JG, Hakonarson H, Denny JC, Crosslin DR, Jarvik GP, Kullo IJ, Williams MS, Harley JB. Evaluation of the MC4R gene across eMERGE network identifies many unreported obesity-associated variants. Int J Obes (Lond). 2021 Jan;45(1):155-169. doi: 10.1038/s41366-020-00675-4. Epub 2020 Sep 20. PMID: 32952152; PMCID: PMC7752751.
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Targeted knockdown of Kv1.3 channels in T lymphocytes corrects the disease manifestations associated with systemic lupus erythematosus.

Science advances

Khodoun M, Chimote AA, Ilyas FZ, Duncan HJ, Moncrieffe H, Kant KS, Conforti L.
PMID: 33208373
Sci Adv. 2020 Nov 18;6(47). doi: 10.1126/sciadv.abd1471. Print 2020 Nov.

Lupus nephritis (LN) is an autoimmune disease with substantial morbidity/mortality and limited efficacy of available therapies. Memory T (Tm) lymphocytes infiltrate LN kidneys, contributing to organ damage. Analysis of LN, diabetic nephropathy, and healthy donor kidney biopsies revealed high...

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Khodoun M, Chimote AA, Ilyas FZ, et al. Targeted knockdown of Kv1.3 channels in T lymphocytes corrects the disease manifestations associated with systemic lupus erythematosus. Sci Adv. 2020;6(47)doi: 10.1126/sciadv.abd1471.
Khodoun, M., Chimote, A. A., Ilyas, F. Z., Duncan, H. J., Moncrieffe, H., Kant, K. S., & Conforti, L. (2020). Targeted knockdown of Kv1.3 channels in T lymphocytes corrects the disease manifestations associated with systemic lupus erythematosus. Science advances, 6(47), . https://doi.org/10.1126/sciadv.abd1471
Khodoun, Marat, et al. "Targeted knockdown of Kv1.3 channels in T lymphocytes corrects the disease manifestations associated with systemic lupus erythematosus." Science advances vol. 6,47 (2020). doi: https://doi.org/10.1126/sciadv.abd1471
Khodoun M, Chimote AA, Ilyas FZ, Duncan HJ, Moncrieffe H, Kant KS, Conforti L. Targeted knockdown of Kv1.3 channels in T lymphocytes corrects the disease manifestations associated with systemic lupus erythematosus. Sci Adv. 2020 Nov 18;6(47). doi: 10.1126/sciadv.abd1471. Print 2020 Nov. PMID: 33208373; PMCID: PMC7673800.
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IFN-γ is essential for alveolar macrophage-driven pulmonary inflammation in macrophage activation syndrome.

JCI insight

Gao DK, Salomonis N, Henderlight M, Woods C, Thakkar K, Grom AA, Thornton S, Jordan MB, Wikenheiser-Brokamp KA, Schulert GS.
PMID: 34314387
JCI Insight. 2021 Sep 08;6(17). doi: 10.1172/jci.insight.147593.

Macrophage activation syndrome (MAS) is a life-threatening cytokine storm complicating systemic juvenile idiopathic arthritis (SJIA) driven by IFN-γ. SJIA and MAS are also associated with an unexplained emerging inflammatory lung disease (SJIA-LD), with our recent work supporting pulmonary activation...

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Gao DK, Salomonis N, Henderlight M, et al. IFN-γ is essential for alveolar macrophage-driven pulmonary inflammation in macrophage activation syndrome. JCI Insight. 2021;6(17)doi: 10.1172/jci.insight.147593.
Gao, D. K., Salomonis, N., Henderlight, M., Woods, C., Thakkar, K., Grom, A. A., Thornton, S., Jordan, M. B., Wikenheiser-Brokamp, K. A., & Schulert, G. S. (2021). IFN-γ is essential for alveolar macrophage-driven pulmonary inflammation in macrophage activation syndrome. JCI insight, 6(17), . https://doi.org/10.1172/jci.insight.147593
Gao, Denny K, et al. "IFN-γ is essential for alveolar macrophage-driven pulmonary inflammation in macrophage activation syndrome." JCI insight vol. 6,17 (2021). doi: https://doi.org/10.1172/jci.insight.147593
Gao DK, Salomonis N, Henderlight M, Woods C, Thakkar K, Grom AA, Thornton S, Jordan MB, Wikenheiser-Brokamp KA, Schulert GS. IFN-γ is essential for alveolar macrophage-driven pulmonary inflammation in macrophage activation syndrome. JCI Insight. 2021 Sep 08;6(17). doi: 10.1172/jci.insight.147593. PMID: 34314387; PMCID: PMC8492332.
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Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci.

Genome research

Hong T, Parameswaran S, Donmez OA, Miller D, Forney C, Lape M, Saint Just Ribeiro M, Liang J, Edsall LE, Magnusen AF, Miller W, Chepelev I, Harley JB, Zhao B, Kottyan LC, Weirauch MT.
PMID: 34799401
Genome Res. 2021 Nov 19; doi: 10.1101/gr.264705.120. Epub 2021 Nov 19.

The interplay between environmental and genetic factors plays a key role in the development of many autoimmune diseases. In particular, the Epstein-Barr virus (EBV) is an established contributor to multiple sclerosis, lupus, and other disorders. Previously, we showed that...

Cite
Hong T, Parameswaran S, Donmez OA, et al. Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci. Genome Res. 2021;doi: 10.1101/gr.264705.120.
Hong, T., Parameswaran, S., Donmez, O. A., Miller, D., Forney, C., Lape, M., Saint Just Ribeiro, M., Liang, J., Edsall, L. E., Magnusen, A. F., Miller, W., Chepelev, I., Harley, J. B., Zhao, B., Kottyan, L. C., & Weirauch, M. T. (2021). Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci. Genome research, . https://doi.org/10.1101/gr.264705.120
Hong, Ted, et al. "Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci." Genome research vol. (2021). doi: https://doi.org/10.1101/gr.264705.120
Hong T, Parameswaran S, Donmez OA, Miller D, Forney C, Lape M, Saint Just Ribeiro M, Liang J, Edsall LE, Magnusen AF, Miller W, Chepelev I, Harley JB, Zhao B, Kottyan LC, Weirauch MT. Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci. Genome Res. 2021 Nov 19; doi: 10.1101/gr.264705.120. Epub 2021 Nov 19. PMID: 34799401; PMCID: PMC8647835.
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Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci.

Genome research

Hong T, Parameswaran S, Donmez OA, Miller D, Forney C, Lape M, Saint Just Ribeiro M, Liang J, Edsall LE, Magnusen AF, Miller W, Chepelev I, Harley JB, Zhao B, Kottyan LC, Weirauch MT.
PMID: 34799401
Genome Res. 2021 Nov 19; doi: 10.1101/gr.264705.120. Epub 2021 Nov 19.

The interplay between environmental and genetic factors plays a key role in the development of many autoimmune diseases. In particular, the Epstein-Barr virus (EBV) is an established contributor to multiple sclerosis, lupus, and other disorders. Previously, we showed that...

Cite
Hong T, Parameswaran S, Donmez OA, et al. Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci. Genome Res. 2021;doi: 10.1101/gr.264705.120.
Hong, T., Parameswaran, S., Donmez, O. A., Miller, D., Forney, C., Lape, M., Saint Just Ribeiro, M., Liang, J., Edsall, L. E., Magnusen, A. F., Miller, W., Chepelev, I., Harley, J. B., Zhao, B., Kottyan, L. C., & Weirauch, M. T. (2021). Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci. Genome research, . https://doi.org/10.1101/gr.264705.120
Hong, Ted, et al. "Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci." Genome research vol. (2021). doi: https://doi.org/10.1101/gr.264705.120
Hong T, Parameswaran S, Donmez OA, Miller D, Forney C, Lape M, Saint Just Ribeiro M, Liang J, Edsall LE, Magnusen AF, Miller W, Chepelev I, Harley JB, Zhao B, Kottyan LC, Weirauch MT. Epstein-Barr virus nuclear antigen 2 extensively rewires the human chromatin landscape at autoimmune risk loci. Genome Res. 2021 Nov 19; doi: 10.1101/gr.264705.120. Epub 2021 Nov 19. PMID: 34799401; PMCID: PMC8647835.
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Successful Urine Multiplex Bead Assay to Measure Lupus Nephritis Activity.

Kidney international reports

Cody EM, Bennett MR, Gulati G, Ma Q, Altaye M, Devarajan P, Brunner HI.
PMID: 34307989
Kidney Int Rep. 2021 Apr 28;6(7):1949-1960. doi: 10.1016/j.ekir.2021.04.016. eCollection 2021 Jul.

INTRODUCTION: Lupus nephritis (LN) confers a poor prognosis, mainly from lack of effective laboratory tests to diagnose and to evaluate therapies. We have previously shown that a set of 6 urinary biomarkers (NGAL, KIM-1, MCP-1, adiponectin, hemopexin, and ceruloplasmin)...

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Cody EM, Bennett MR, Gulati G, et al. Successful Urine Multiplex Bead Assay to Measure Lupus Nephritis Activity. Kidney Int Rep. 2021;6(7):1949-1960doi: 10.1016/j.ekir.2021.04.016.
Cody, E. M., Bennett, M. R., Gulati, G., Ma, Q., Altaye, M., Devarajan, P., & Brunner, H. I. (2021). Successful Urine Multiplex Bead Assay to Measure Lupus Nephritis Activity. Kidney international reports, 6(7), 1949-1960. https://doi.org/10.1016/j.ekir.2021.04.016
Cody, Ellen M, et al. "Successful Urine Multiplex Bead Assay to Measure Lupus Nephritis Activity." Kidney international reports vol. 6,7 (2021): 1949-1960. doi: https://doi.org/10.1016/j.ekir.2021.04.016
Cody EM, Bennett MR, Gulati G, Ma Q, Altaye M, Devarajan P, Brunner HI. Successful Urine Multiplex Bead Assay to Measure Lupus Nephritis Activity. Kidney Int Rep. 2021 Apr 28;6(7):1949-1960. doi: 10.1016/j.ekir.2021.04.016. eCollection 2021 Jul. PMID: 34307989; PMCID: PMC8258454.
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Diesel exhaust and house dust mite allergen lead to common changes in the airway methylome and hydroxymethylome.

Environmental epigenetics

Zhang X, Chen X, Weirauch MT, Zhang X, Burleson JD, Brandt EB, Ji H.
PMID: 30090644
Environ Epigenet. 2018 Jul 27;4(3):dvy020. doi: 10.1093/eep/dvy020. eCollection 2018 Jul.

Exposures to diesel exhaust particles (DEP) from traffic and house dust mite (HDM) allergens significantly increase risks of airway diseases, including asthma. This negative impact of DEP and HDM may in part be mediated by epigenetic mechanisms. Beyond functioning...

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Zhang X, Chen X, Weirauch MT, et al. Diesel exhaust and house dust mite allergen lead to common changes in the airway methylome and hydroxymethylome. Environ Epigenet. 2018;4(3):dvy020doi: 10.1093/eep/dvy020.
Zhang, X., Chen, X., Weirauch, M. T., Zhang, X., Burleson, J. D., Brandt, E. B., & Ji, H. (2018). Diesel exhaust and house dust mite allergen lead to common changes in the airway methylome and hydroxymethylome. Environmental epigenetics, 4(3), dvy020. https://doi.org/10.1093/eep/dvy020
Zhang, Xue, et al. "Diesel exhaust and house dust mite allergen lead to common changes in the airway methylome and hydroxymethylome." Environmental epigenetics vol. 4,3 (2018): dvy020. doi: https://doi.org/10.1093/eep/dvy020
Zhang X, Chen X, Weirauch MT, Zhang X, Burleson JD, Brandt EB, Ji H. Diesel exhaust and house dust mite allergen lead to common changes in the airway methylome and hydroxymethylome. Environ Epigenet. 2018 Jul 27;4(3):dvy020. doi: 10.1093/eep/dvy020. eCollection 2018 Jul. PMID: 30090644; PMCID: PMC6063278.
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