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Showing 1 to 12 of 175 entries
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Anaplastic lymphoma kinase (ALK) gene alteration in signet ring cell carcinoma of the gastrointestinal tract.

Therapeutic advances in medical oncology

Alese OB, El-Rayes BF, Sica G, Zhang G, Alexis D, La Rosa FG, Varella-Garcia M, Chen Z, Rossi MR, Adsay NV, Khuri FR, Owonikoko TK.
PMID: 25755678
Ther Adv Med Oncol. 2015 Mar;7(2):56-62. doi: 10.1177/1758834014567117.

OBJECTIVES: ALK-EML4 translocation is an established driver aberration in non-small cell lung cancer (NSCLC), with reported predilection for cases with signet ring histology. We assessed the presence of anaplastic lymphoma kinase (ALK) gene rearrangements in signet ring cancers arising...

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Alese OB, El-Rayes BF, Sica G, et al. Anaplastic lymphoma kinase (ALK) gene alteration in signet ring cell carcinoma of the gastrointestinal tract. Ther Adv Med Oncol. 2015;7(2):56-62doi: 10.1177/1758834014567117.
Alese, O. B., El-Rayes, B. F., Sica, G., Zhang, G., Alexis, D., La Rosa, F. G., Varella-Garcia, M., Chen, Z., Rossi, M. R., Adsay, N. V., Khuri, F. R., & Owonikoko, T. K. (2015). Anaplastic lymphoma kinase (ALK) gene alteration in signet ring cell carcinoma of the gastrointestinal tract. Therapeutic advances in medical oncology, 7(2), 56-62. https://doi.org/10.1177/1758834014567117
Alese, Olatunji B, et al. "Anaplastic lymphoma kinase (ALK) gene alteration in signet ring cell carcinoma of the gastrointestinal tract." Therapeutic advances in medical oncology vol. 7,2 (2015): 56-62. doi: https://doi.org/10.1177/1758834014567117
Alese OB, El-Rayes BF, Sica G, Zhang G, Alexis D, La Rosa FG, Varella-Garcia M, Chen Z, Rossi MR, Adsay NV, Khuri FR, Owonikoko TK. Anaplastic lymphoma kinase (ALK) gene alteration in signet ring cell carcinoma of the gastrointestinal tract. Ther Adv Med Oncol. 2015 Mar;7(2):56-62. doi: 10.1177/1758834014567117. PMID: 25755678; PMCID: PMC4346214.
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Bias and variance reduction in estimating the proportion of true-null hypotheses.

Biostatistics (Oxford, England)

Cheng Y, Gao D, Tong T.
PMID: 24963010
Biostatistics. 2015 Jan;16(1):189-204. doi: 10.1093/biostatistics/kxu029. Epub 2014 Jun 23.

When testing a large number of hypotheses, estimating the proportion of true nulls, denoted by π(0), becomes increasingly important. This quantity has many applications in practice. For instance, a reliable estimate of π(0) can eliminate the conservative bias of...

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Cheng Y, Gao D, Tong T. Bias and variance reduction in estimating the proportion of true-null hypotheses. Biostatistics. 2014;16(1):189-204doi: 10.1093/biostatistics/kxu029.
Cheng, Y., Gao, D., & Tong, T. (2015). Bias and variance reduction in estimating the proportion of true-null hypotheses. Biostatistics (Oxford, England), 16(1), 189-204. https://doi.org/10.1093/biostatistics/kxu029
Cheng, Yebin, et al. "Bias and variance reduction in estimating the proportion of true-null hypotheses." Biostatistics (Oxford, England) vol. 16,1 (2015): 189-204. doi: https://doi.org/10.1093/biostatistics/kxu029
Cheng Y, Gao D, Tong T. Bias and variance reduction in estimating the proportion of true-null hypotheses. Biostatistics. 2015 Jan;16(1):189-204. doi: 10.1093/biostatistics/kxu029. Epub 2014 Jun 23. PMID: 24963010; PMCID: PMC4263223.
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Depletion of tumor-associated macrophages slows the growth of chemically induced mouse lung adenocarcinomas.

Frontiers in immunology

Fritz JM, Tennis MA, Orlicky DJ, Lin H, Ju C, Redente EF, Choo KS, Staab TA, Bouchard RJ, Merrick DT, Malkinson AM, Dwyer-Nield LD.
PMID: 25505466
Front Immunol. 2014 Nov 25;5:587. doi: 10.3389/fimmu.2014.00587. eCollection 2014.

Chronic inflammation is a risk factor for lung cancer, and low-dose aspirin intake reduces lung cancer risk. However, the roles that specific inflammatory cells and their products play in lung carcinogenesis have yet to be fully elucidated. In mice,...

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Fritz JM, Tennis MA, Orlicky DJ, et al. Depletion of tumor-associated macrophages slows the growth of chemically induced mouse lung adenocarcinomas. Front Immunol. 2014;5:587doi: 10.3389/fimmu.2014.00587.
Fritz, J. M., Tennis, M. A., Orlicky, D. J., Lin, H., Ju, C., Redente, E. F., Choo, K. S., Staab, T. A., Bouchard, R. J., Merrick, D. T., Malkinson, A. M., & Dwyer-Nield, L. D. (2014). Depletion of tumor-associated macrophages slows the growth of chemically induced mouse lung adenocarcinomas. Frontiers in immunology, 5587. https://doi.org/10.3389/fimmu.2014.00587
Fritz, Jason M, et al. "Depletion of tumor-associated macrophages slows the growth of chemically induced mouse lung adenocarcinomas." Frontiers in immunology vol. 5 (2014): 587. doi: https://doi.org/10.3389/fimmu.2014.00587
Fritz JM, Tennis MA, Orlicky DJ, Lin H, Ju C, Redente EF, Choo KS, Staab TA, Bouchard RJ, Merrick DT, Malkinson AM, Dwyer-Nield LD. Depletion of tumor-associated macrophages slows the growth of chemically induced mouse lung adenocarcinomas. Front Immunol. 2014 Nov 25;5:587. doi: 10.3389/fimmu.2014.00587. eCollection 2014. PMID: 25505466; PMCID: PMC4243558.
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Non-invasive imaging to monitor lupus nephritis and neuropsychiatric systemic lupus erythematosus.

F1000Research

Thurman JM, Serkova NJ.
PMID: 26309728
F1000Res. 2015 Jun 16;4:153. doi: 10.12688/f1000research.6587.2. eCollection 2015.

Systemic lupus erythematosus (SLE) is an autoimmune disease that can affect multiple different organs, including the kidneys and central nervous system (CNS). Conventional radiological examinations in SLE patients include volumetric/ anatomical computed tomography (CT), magnetic resonance imaging (MRI) and...

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Thurman JM, Serkova NJ. Non-invasive imaging to monitor lupus nephritis and neuropsychiatric systemic lupus erythematosus. F1000Res. 2015;4:153doi: 10.12688/f1000research.6587.2.
Thurman, J. M., & Serkova, N. J. (2015). Non-invasive imaging to monitor lupus nephritis and neuropsychiatric systemic lupus erythematosus. F1000Research, 4153. https://doi.org/10.12688/f1000research.6587.2
Thurman, Joshua M, and Serkova, Natalie J. "Non-invasive imaging to monitor lupus nephritis and neuropsychiatric systemic lupus erythematosus." F1000Research vol. 4 (2015): 153. doi: https://doi.org/10.12688/f1000research.6587.2
Thurman JM, Serkova NJ. Non-invasive imaging to monitor lupus nephritis and neuropsychiatric systemic lupus erythematosus. F1000Res. 2015 Jun 16;4:153. doi: 10.12688/f1000research.6587.2. eCollection 2015. PMID: 26309728; PMCID: PMC4536614.
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Expected Power for the False Discovery Rate with Independence.

Communications in statistics: theory and methods

Glueck DH, Muller KE, Karimpour-Fard A, Hunter L.
PMID: 20975846
Commun Stat Theory Methods. 2008 Jan;37(12):1855-1866. doi: 10.1080/03610920801893731.

The Benjamini-Hochberg procedure is widely used in multiple comparisons. Previous power results for this procedure have been based on simulations. This article produces theoretical expressions for expected power. To derive them, we make assumptions about the number of hypotheses...

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Glueck DH, Muller KE, Karimpour-Fard A, et al. Expected Power for the False Discovery Rate with Independence. Commun Stat Theory Methods. 2008;37(12):1855-1866doi: 10.1080/03610920801893731.
Glueck, D. H., Muller, K. E., Karimpour-Fard, A., & Hunter, L. (2008). Expected Power for the False Discovery Rate with Independence. Communications in statistics: theory and methods, 37(12), 1855-1866. https://doi.org/10.1080/03610920801893731
Glueck, D H, et al. "Expected Power for the False Discovery Rate with Independence." Communications in statistics: theory and methods vol. 37,12 (2008): 1855-1866. doi: https://doi.org/10.1080/03610920801893731
Glueck DH, Muller KE, Karimpour-Fard A, Hunter L. Expected Power for the False Discovery Rate with Independence. Commun Stat Theory Methods. 2008 Jan;37(12):1855-1866. doi: 10.1080/03610920801893731. PMID: 20975846; PMCID: PMC2962418.
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Live multicellular tumor spheroid models for high-content imaging and screening in cancer drug discovery.

Current chemical genomics and translational medicine

Reid BG, Jerjian T, Patel P, Zhou Q, Yoo BH, Kabos P, Sartorius CA, Labarbera DV.
PMID: 24596682
Curr Chem Genom Transl Med. 2014 Feb 07;8:27-35. doi: 10.2174/2213988501408010027. eCollection 2014.

The multi cellular tumor spheroid (MCTS) model has been used for decades with proven superiority over monolayer cell culture models at recapitulating in vivo tumor growth. Yet its use in high-throughput drug discovery has been limited, particularly with image...

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Reid BG, Jerjian T, Patel P, et al. Live multicellular tumor spheroid models for high-content imaging and screening in cancer drug discovery. Curr Chem Genom Transl Med. 2014;8:27-35doi: 10.2174/2213988501408010027.
Reid, B. G., Jerjian, T., Patel, P., Zhou, Q., Yoo, B. H., Kabos, P., Sartorius, C. A., & Labarbera, D. V. (2014). Live multicellular tumor spheroid models for high-content imaging and screening in cancer drug discovery. Current chemical genomics and translational medicine, 827-35. https://doi.org/10.2174/2213988501408010027
Reid, Brian G, et al. "Live multicellular tumor spheroid models for high-content imaging and screening in cancer drug discovery." Current chemical genomics and translational medicine vol. 8 (2014): 27-35. doi: https://doi.org/10.2174/2213988501408010027
Reid BG, Jerjian T, Patel P, Zhou Q, Yoo BH, Kabos P, Sartorius CA, Labarbera DV. Live multicellular tumor spheroid models for high-content imaging and screening in cancer drug discovery. Curr Chem Genom Transl Med. 2014 Feb 07;8:27-35. doi: 10.2174/2213988501408010027. eCollection 2014. PMID: 24596682; PMCID: PMC3941083.
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Immunotherapeutic implications of the immunophenotype of pediatric brain tumors.

Oncoimmunology

Griesinger AM, Donson AM, Foreman NK.
PMID: 24575386
Oncoimmunology. 2014 Jan 01;3(1):e27256. doi: 10.4161/onci.27256.

The cytofluorometric analysis of dissociated tumor samples identified distinct immunophenotypes among the most common variants of pediatric brain tumor. These findings suggest that immunotherapeutic regimens against pediatric brain malignancies should be tailored to individual tumor types.

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Griesinger AM, Donson AM, Foreman NK. Immunotherapeutic implications of the immunophenotype of pediatric brain tumors. Oncoimmunology. 2014;3(1):e27256doi: 10.4161/onci.27256.
Griesinger, A. M., Donson, A. M., & Foreman, N. K. (2014). Immunotherapeutic implications of the immunophenotype of pediatric brain tumors. Oncoimmunology, 3(1), e27256. https://doi.org/10.4161/onci.27256
Griesinger, Andrea M, et al. "Immunotherapeutic implications of the immunophenotype of pediatric brain tumors." Oncoimmunology vol. 3,1 (2014): e27256. doi: https://doi.org/10.4161/onci.27256
Griesinger AM, Donson AM, Foreman NK. Immunotherapeutic implications of the immunophenotype of pediatric brain tumors. Oncoimmunology. 2014 Jan 01;3(1):e27256. doi: 10.4161/onci.27256. PMID: 24575386; PMCID: PMC3929359.
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PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells.

PPAR research

Wood WM, Sharma V, Bauerle KT, Pike LA, Zhou Q, Fretwell DL, Schweppe RE, Haugen BR.
PMID: 22194735
PPAR Res. 2011;2011:171765. doi: 10.1155/2011/171765. Epub 2011 Dec 12.

Undifferentiated (anaplastic) thyroid cancer (ATC) is one of the most aggressive human malignancies and no effective therapy is currently available. We show here that PPARγ levels are elevated in cells derived from ATC. Depletion of PPARγ in HTh74 ATC...

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Wood WM, Sharma V, Bauerle KT, et al. PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells. PPAR Res. 2011;2011:171765doi: 10.1155/2011/171765.
Wood, W. M., Sharma, V., Bauerle, K. T., Pike, L. A., Zhou, Q., Fretwell, D. L., Schweppe, R. E., & Haugen, B. R. (2011). PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells. PPAR research, 2011171765. https://doi.org/10.1155/2011/171765
Wood, William M, et al. "PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells." PPAR research vol. 2011 (2011): 171765. doi: https://doi.org/10.1155/2011/171765
Wood WM, Sharma V, Bauerle KT, Pike LA, Zhou Q, Fretwell DL, Schweppe RE, Haugen BR. PPARγ Promotes Growth and Invasion of Thyroid Cancer Cells. PPAR Res. 2011;2011:171765. doi: 10.1155/2011/171765. Epub 2011 Dec 12. PMID: 22194735; PMCID: PMC3236353.
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Global Decrease of Histone H3K27 Acetylation in ZEB1-Induced Epithelial to Mesenchymal Transition in Lung Cancer Cells.

Cancers

Roche J, Nasarre P, Gemmill R, Baldys A, Pontis J, Korch C, Guilhot J, Ait-Si-Ali S, Drabkin H.
PMID: 24216980
Cancers (Basel). 2013 Apr 03;5(2):334-56. doi: 10.3390/cancers5020334.

The epithelial to mesenchymal transition (EMT) enables epithelial cells with a migratory mesenchymal phenotype. It is activated in cancer cells and is involved in invasion, metastasis and stem-like properties. ZEB1, an E-box binding transcription factor, is a major suppressor...

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Roche J, Nasarre P, Gemmill R, et al. Global Decrease of Histone H3K27 Acetylation in ZEB1-Induced Epithelial to Mesenchymal Transition in Lung Cancer Cells. Cancers (Basel). 2013;5(2):334-56doi: 10.3390/cancers5020334.
Roche, J., Nasarre, P., Gemmill, R., Baldys, A., Pontis, J., Korch, C., Guilhot, J., Ait-Si-Ali, S., & Drabkin, H. (2013). Global Decrease of Histone H3K27 Acetylation in ZEB1-Induced Epithelial to Mesenchymal Transition in Lung Cancer Cells. Cancers, 5(2), 334-56. https://doi.org/10.3390/cancers5020334
Roche, Joëlle, et al. "Global Decrease of Histone H3K27 Acetylation in ZEB1-Induced Epithelial to Mesenchymal Transition in Lung Cancer Cells." Cancers vol. 5,2 (2013): 334-56. doi: https://doi.org/10.3390/cancers5020334
Roche J, Nasarre P, Gemmill R, Baldys A, Pontis J, Korch C, Guilhot J, Ait-Si-Ali S, Drabkin H. Global Decrease of Histone H3K27 Acetylation in ZEB1-Induced Epithelial to Mesenchymal Transition in Lung Cancer Cells. Cancers (Basel). 2013 Apr 03;5(2):334-56. doi: 10.3390/cancers5020334. PMID: 24216980; PMCID: PMC3730320.
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Inhibition of MerTK increases chemosensitivity and decreases oncogenic potential in T-cell acute lymphoblastic leukemia.

Blood cancer journal

Brandao LN, Winges A, Christoph S, Sather S, Migdall-Wilson J, Schlegel J, McGranahan A, Gao D, Liang X, Deryckere D, Graham DK.
PMID: 23353780
Blood Cancer J. 2013 Jan 25;3:e101. doi: 10.1038/bcj.2012.46.

Pediatric leukemia survival rates have improved dramatically over the past decades. However, current treatment protocols are still largely ineffective in cases of relapsed leukemia and are associated with a significant rate of chronic health conditions. Thus, there is a...

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Brandao LN, Winges A, Christoph S, et al. Inhibition of MerTK increases chemosensitivity and decreases oncogenic potential in T-cell acute lymphoblastic leukemia. Blood Cancer J. 2013;3:e101doi: 10.1038/bcj.2012.46.
Brandao, L. N., Winges, A., Christoph, S., Sather, S., Migdall-Wilson, J., Schlegel, J., McGranahan, A., Gao, D., Liang, X., Deryckere, D., & Graham, D. K. (2013). Inhibition of MerTK increases chemosensitivity and decreases oncogenic potential in T-cell acute lymphoblastic leukemia. Blood cancer journal, 3e101. https://doi.org/10.1038/bcj.2012.46
Brandao, L N, et al. "Inhibition of MerTK increases chemosensitivity and decreases oncogenic potential in T-cell acute lymphoblastic leukemia." Blood cancer journal vol. 3 (2013): e101. doi: https://doi.org/10.1038/bcj.2012.46
Brandao LN, Winges A, Christoph S, Sather S, Migdall-Wilson J, Schlegel J, McGranahan A, Gao D, Liang X, Deryckere D, Graham DK. Inhibition of MerTK increases chemosensitivity and decreases oncogenic potential in T-cell acute lymphoblastic leukemia. Blood Cancer J. 2013 Jan 25;3:e101. doi: 10.1038/bcj.2012.46. PMID: 23353780; PMCID: PMC3556576.
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Protein Kinase C α Modulates Estrogen-Receptor-Dependent Transcription and Proliferation in Endometrial Cancer Cells.

Obstetrics and gynecology international

Thorne AM, Jackson TA, Willis VC, Bradford AP.
PMID: 23843797
Obstet Gynecol Int. 2013;2013:537479. doi: 10.1155/2013/537479. Epub 2013 Jun 17.

Endometrial cancer is the most common invasive gynecologic malignancy in developed countries. The most prevalent endometrioid tumors are linked to excessive estrogen exposure and hyperplasia. However, molecular mechanisms and signaling pathways underlying their etiology and pathophysiology remain poorly understood....

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Thorne AM, Jackson TA, Willis VC, et al. Protein Kinase C α Modulates Estrogen-Receptor-Dependent Transcription and Proliferation in Endometrial Cancer Cells. Obstet Gynecol Int. 2013;2013:537479doi: 10.1155/2013/537479.
Thorne, A. M., Jackson, T. A., Willis, V. C., & Bradford, A. P. (2013). Protein Kinase C α Modulates Estrogen-Receptor-Dependent Transcription and Proliferation in Endometrial Cancer Cells. Obstetrics and gynecology international, 2013537479. https://doi.org/10.1155/2013/537479
Thorne, Alicia M, et al. "Protein Kinase C α Modulates Estrogen-Receptor-Dependent Transcription and Proliferation in Endometrial Cancer Cells." Obstetrics and gynecology international vol. 2013 (2013): 537479. doi: https://doi.org/10.1155/2013/537479
Thorne AM, Jackson TA, Willis VC, Bradford AP. Protein Kinase C α Modulates Estrogen-Receptor-Dependent Transcription and Proliferation in Endometrial Cancer Cells. Obstet Gynecol Int. 2013;2013:537479. doi: 10.1155/2013/537479. Epub 2013 Jun 17. PMID: 23843797; PMCID: PMC3703424.
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Renal epithelioid angiomyolipoma with a negative premelanosome marker immunoprofile: a case report and review of the literature.

Journal of medical case reports

Hohensee SE, La Rosa FG, Homer P, Suby-Long T, Wilson S, Lucia SM, Iczkowski KA.
PMID: 23628229
J Med Case Rep. 2013 Apr 29;7:118. doi: 10.1186/1752-1947-7-118.

INTRODUCTION: The rare variant of renal epithelioid/pleomorphic angiomyolipoma has been reported in approximately 120 cases. One of the most important characteristics to differentiate these tumors from other renal cell neoplasms is their typical reactivity to premelanosome antigens. If such...

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Hohensee SE, La Rosa FG, Homer P, et al. Renal epithelioid angiomyolipoma with a negative premelanosome marker immunoprofile: a case report and review of the literature. J Med Case Rep. 2013;7:118doi: 10.1186/1752-1947-7-118.
Hohensee, S. E., La Rosa, F. G., Homer, P., Suby-Long, T., Wilson, S., Lucia, S. M., & Iczkowski, K. A. (2013). Renal epithelioid angiomyolipoma with a negative premelanosome marker immunoprofile: a case report and review of the literature. Journal of medical case reports, 7118. https://doi.org/10.1186/1752-1947-7-118
Hohensee, Samantha E, et al. "Renal epithelioid angiomyolipoma with a negative premelanosome marker immunoprofile: a case report and review of the literature." Journal of medical case reports vol. 7 (2013): 118. doi: https://doi.org/10.1186/1752-1947-7-118
Hohensee SE, La Rosa FG, Homer P, Suby-Long T, Wilson S, Lucia SM, Iczkowski KA. Renal epithelioid angiomyolipoma with a negative premelanosome marker immunoprofile: a case report and review of the literature. J Med Case Rep. 2013 Apr 29;7:118. doi: 10.1186/1752-1947-7-118. PMID: 23628229; PMCID: PMC3667146.
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Showing 1 to 12 of 175 entries