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Showing 1 to 12 of 147 entries
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Plant-derived SAC domain of PAR-4 (Prostate Apoptosis Response 4) exhibits growth inhibitory effects in prostate cancer cells.

Frontiers in plant science

Sarkar S, Jain S, Rai V, Sahoo DK, Raha S, Suklabaidya S, Senapati S, Rangnekar VM, Maiti IB, Dey N.
PMID: 26500666
Front Plant Sci. 2015 Oct 07;6:822. doi: 10.3389/fpls.2015.00822. eCollection 2015.

The gene Par-4 (Prostate Apoptosis Response 4) was originally identified in prostate cancer cells undergoing apoptosis and its product Par-4 showed cancer specific pro-apoptotic activity. Particularly, the SAC domain of Par-4 (SAC-Par-4) selectively kills cancer cells leaving normal cells...

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Sarkar S, Jain S, Rai V, et al. Plant-derived SAC domain of PAR-4 (Prostate Apoptosis Response 4) exhibits growth inhibitory effects in prostate cancer cells. Front Plant Sci. 2015;6:822doi: 10.3389/fpls.2015.00822.
Sarkar, S., Jain, S., Rai, V., Sahoo, D. K., Raha, S., Suklabaidya, S., Senapati, S., Rangnekar, V. M., Maiti, I. B., & Dey, N. (2015). Plant-derived SAC domain of PAR-4 (Prostate Apoptosis Response 4) exhibits growth inhibitory effects in prostate cancer cells. Frontiers in plant science, 6822. https://doi.org/10.3389/fpls.2015.00822
Sarkar, Shayan, et al. "Plant-derived SAC domain of PAR-4 (Prostate Apoptosis Response 4) exhibits growth inhibitory effects in prostate cancer cells." Frontiers in plant science vol. 6 (2015): 822. doi: https://doi.org/10.3389/fpls.2015.00822
Sarkar S, Jain S, Rai V, Sahoo DK, Raha S, Suklabaidya S, Senapati S, Rangnekar VM, Maiti IB, Dey N. Plant-derived SAC domain of PAR-4 (Prostate Apoptosis Response 4) exhibits growth inhibitory effects in prostate cancer cells. Front Plant Sci. 2015 Oct 07;6:822. doi: 10.3389/fpls.2015.00822. eCollection 2015. PMID: 26500666; PMCID: PMC4595782.
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Microarray dataset of transient and permanent DNA methylation changes in HeLa cells undergoing inorganic arsenic-mediated epithelial-to-mesenchymal transition.

Data in brief

Eckstein M, Rea M, Fondufe-Mittendorf YN.
PMID: 28589171
Data Brief. 2017 May 10;13:6-9. doi: 10.1016/j.dib.2017.05.002. eCollection 2017 Aug.

The novel dataset presented here represents the results of the changing pattern of DNA methylation profiles in HeLa cells exposed to chronic low dose (0.5 µM) sodium arsenite, resulting in epithelial-to-mesenchymal transition, as well as DNA methylation patterns in...

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Eckstein M, Rea M, Fondufe-Mittendorf YN. Microarray dataset of transient and permanent DNA methylation changes in HeLa cells undergoing inorganic arsenic-mediated epithelial-to-mesenchymal transition. Data Brief. 2017;13:6-9doi: 10.1016/j.dib.2017.05.002.
Eckstein, M., Rea, M., & Fondufe-Mittendorf, Y. N. (2017). Microarray dataset of transient and permanent DNA methylation changes in HeLa cells undergoing inorganic arsenic-mediated epithelial-to-mesenchymal transition. Data in brief, 136-9. https://doi.org/10.1016/j.dib.2017.05.002
Eckstein, Meredith, et al. "Microarray dataset of transient and permanent DNA methylation changes in HeLa cells undergoing inorganic arsenic-mediated epithelial-to-mesenchymal transition." Data in brief vol. 13 (2017): 6-9. doi: https://doi.org/10.1016/j.dib.2017.05.002
Eckstein M, Rea M, Fondufe-Mittendorf YN. Microarray dataset of transient and permanent DNA methylation changes in HeLa cells undergoing inorganic arsenic-mediated epithelial-to-mesenchymal transition. Data Brief. 2017 May 10;13:6-9. doi: 10.1016/j.dib.2017.05.002. eCollection 2017 Aug. PMID: 28589171; PMCID: PMC5443927.
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Strengthening Interactions between Statisticians and Collaborators: Objectives and Sample Sizes.

Journal of biometrics & biostatistics

Van Meter E, Charnigo R.
PMID: 25932344
J Biom Biostat. 2014;5(1):e127. doi: 10.4172/2155-6180.1000e127.

No abstract available.

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Van Meter E, Charnigo R. Strengthening Interactions between Statisticians and Collaborators: Objectives and Sample Sizes. J Biom Biostat. 2014;5(1):e127doi: 10.4172/2155-6180.1000e127.
Van Meter, E., & Charnigo, R. (2014). Strengthening Interactions between Statisticians and Collaborators: Objectives and Sample Sizes. Journal of biometrics & biostatistics, 5(1), e127. https://doi.org/10.4172/2155-6180.1000e127
Van Meter, Emily, and Charnigo, Richard. "Strengthening Interactions between Statisticians and Collaborators: Objectives and Sample Sizes." Journal of biometrics & biostatistics vol. 5,1 (2014): e127. doi: https://doi.org/10.4172/2155-6180.1000e127
Van Meter E, Charnigo R. Strengthening Interactions between Statisticians and Collaborators: Objectives and Sample Sizes. J Biom Biostat. 2014;5(1):e127. doi: 10.4172/2155-6180.1000e127. PMID: 25932344; PMCID: PMC4412165.
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KDM1 links nuclear GSK3β to epigenetic alterations.

Translational cancer research

Guo W, Zhou BP.
PMID: 30761265
Transl Cancer Res. 2017 Mar;6:S366-S368. doi: 10.21037/tcr.2017.03.26.

No abstract available.

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Guo W, Zhou BP. KDM1 links nuclear GSK3β to epigenetic alterations. Transl Cancer Res. 2017;6:S366-S368doi: 10.21037/tcr.2017.03.26.
Guo, W., & Zhou, B. P. (2017). KDM1 links nuclear GSK3β to epigenetic alterations. Translational cancer research, 6S366-S368. https://doi.org/10.21037/tcr.2017.03.26
Guo, Weijie, and Zhou, Binhua P. "KDM1 links nuclear GSK3β to epigenetic alterations." Translational cancer research vol. 6 (2017): S366-S368. doi: https://doi.org/10.21037/tcr.2017.03.26
Guo W, Zhou BP. KDM1 links nuclear GSK3β to epigenetic alterations. Transl Cancer Res. 2017 Mar;6:S366-S368. doi: 10.21037/tcr.2017.03.26. PMID: 30761265; PMCID: PMC6370299.
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Immunohistochemical Identification of Human Skeletal Muscle Macrophages.

Bio-protocol

Kosmac K, Peck BD, Walton RG, Mula J, Kern PA, Bamman MM, Dennis RA, Jacobs CA, Lattermann C, Johnson DL, Peterson CA.
PMID: 30148186
Bio Protoc. 2018 Jun 20;8(12). doi: 10.21769/BioProtoc.2883.

Macrophages have well-characterized roles in skeletal muscle repair and regeneration. Relatively little is known regarding the role of resident macrophages in skeletal muscle homeostasis, extracellular matrix remodeling, growth, metabolism and adaptation to various stimuli including exercise and training. Despite...

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Kosmac K, Peck BD, Walton RG, et al. Immunohistochemical Identification of Human Skeletal Muscle Macrophages. Bio Protoc. 2018;8(12)doi: 10.21769/BioProtoc.2883.
Kosmac, K., Peck, B. D., Walton, R. G., Mula, J., Kern, P. A., Bamman, M. M., Dennis, R. A., Jacobs, C. A., Lattermann, C., Johnson, D. L., & Peterson, C. A. (2018). Immunohistochemical Identification of Human Skeletal Muscle Macrophages. Bio-protocol, 8(12), . https://doi.org/10.21769/BioProtoc.2883
Kosmac, Kate, et al. "Immunohistochemical Identification of Human Skeletal Muscle Macrophages." Bio-protocol vol. 8,12 (2018). doi: https://doi.org/10.21769/BioProtoc.2883
Kosmac K, Peck BD, Walton RG, Mula J, Kern PA, Bamman MM, Dennis RA, Jacobs CA, Lattermann C, Johnson DL, Peterson CA. Immunohistochemical Identification of Human Skeletal Muscle Macrophages. Bio Protoc. 2018 Jun 20;8(12). doi: 10.21769/BioProtoc.2883. PMID: 30148186; PMCID: PMC6105281.
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The regulation of snail: on the ubiquitin edge.

Cancer cell & microenvironment

Yu Q, Zhou BP, Wu Y.
PMID: 29147673
Cancer Cell Microenviron. 2017;4(2). Epub 2017 Jul 03.

Metastasis accounts for a majority of cancer death. One key feature during metastasis is epithelial-mesenchymal transition (EMT), which is regulated by transcription factors such as Snail and Twist. In non-malignant cells, Snail has a short half-life and is degraded...

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Yu Q, Zhou BP, Wu Y. The regulation of snail: on the ubiquitin edge. Cancer Cell Microenviron. 2017;4(2)
Yu, Q., Zhou, B. P., & Wu, Y. (2017). The regulation of snail: on the ubiquitin edge. Cancer cell & microenvironment, 4(2), .
Yu, Qian, et al. "The regulation of snail: on the ubiquitin edge." Cancer cell & microenvironment vol. 4,2 (2017).
Yu Q, Zhou BP, Wu Y. The regulation of snail: on the ubiquitin edge. Cancer Cell Microenviron. 2017;4(2). Epub 2017 Jul 03. PMID: 29147673; PMCID: PMC5685547.
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ASO Author Reflections: How Can We Improve Quality of Cancer Treatment at Community Hospitals?.

Annals of surgical oncology

Tucker TC, Mullett TW.
PMID: 32761332
Ann Surg Oncol. 2020 Dec;27:871-872. doi: 10.1245/s10434-020-08965-x. Epub 2020 Aug 05.

No abstract available.

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Tucker TC, Mullett TW. ASO Author Reflections: How Can We Improve Quality of Cancer Treatment at Community Hospitals?. Ann Surg Oncol. 2020;27:871-872doi: 10.1245/s10434-020-08965-x.
Tucker, T. C., & Mullett, T. W. (2020). ASO Author Reflections: How Can We Improve Quality of Cancer Treatment at Community Hospitals?. Annals of surgical oncology, 27871-872. https://doi.org/10.1245/s10434-020-08965-x
Tucker, Thomas C, and Mullett, Timothy W. "ASO Author Reflections: How Can We Improve Quality of Cancer Treatment at Community Hospitals?." Annals of surgical oncology vol. 27 (2020): 871-872. doi: https://doi.org/10.1245/s10434-020-08965-x
Tucker TC, Mullett TW. ASO Author Reflections: How Can We Improve Quality of Cancer Treatment at Community Hospitals?. Ann Surg Oncol. 2020 Dec;27:871-872. doi: 10.1245/s10434-020-08965-x. Epub 2020 Aug 05. PMID: 32761332; PMCID: PMC7680272.
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Overview of the BioCreative VI Precision Medicine Track: mining protein interactions and mutations for precision medicine.

Database : the journal of biological databases and curation

Islamaj Dogan R, Kim S, Chatr-Aryamontri A, Wei CH, Comeau DC, Antunes R, Matos S, Chen Q, Elangovan A, Panyam NC, Verspoor K, Liu H, Wang Y, Liu Z, Altinel B, Hüsünbeyi ZM, Özgür A, Fergadis A, Wang CK, Dai HJ, Tran T, Kavuluru R, Luo L, Steppi A, Zhang J, Qu J, Lu Z.
PMID: 30689846
Database (Oxford). 2019 Jan 01;2019. doi: 10.1093/database/bay147.

The Precision Medicine Initiative is a multicenter effort aiming at formulating personalized treatments leveraging on individual patient data (clinical, genome sequence and functional genomic data) together with the information in large knowledge bases (KBs) that integrate genome annotation, disease...

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Islamaj Dogan R, Kim S, Chatr-Aryamontri A, et al. Overview of the BioCreative VI Precision Medicine Track: mining protein interactions and mutations for precision medicine. Database (Oxford). 2019;2019doi: 10.1093/database/bay147.
Islamaj Dogan, R., Kim, S., Chatr-Aryamontri, A., Wei, C. H., Comeau, D. C., Antunes, R., Matos, S., Chen, Q., Elangovan, A., Panyam, N. C., Verspoor, K., Liu, H., Wang, Y., Liu, Z., Altinel, B., Hüsünbeyi, Z. M., Özgür, A., Fergadis, A., Wang, C. K., Dai, H. J., Tran, T., Kavuluru, R., Luo, L., Steppi, A., Zhang, J., Qu, J., & Lu, Z. (2019). Overview of the BioCreative VI Precision Medicine Track: mining protein interactions and mutations for precision medicine. Database : the journal of biological databases and curation, 2019. https://doi.org/10.1093/database/bay147
Islamaj Dogan, Rezarta, et al. "Overview of the BioCreative VI Precision Medicine Track: mining protein interactions and mutations for precision medicine." Database : the journal of biological databases and curation vol. 2019 (2019). doi: https://doi.org/10.1093/database/bay147
Islamaj Dogan R, Kim S, Chatr-Aryamontri A, Wei CH, Comeau DC, Antunes R, Matos S, Chen Q, Elangovan A, Panyam NC, Verspoor K, Liu H, Wang Y, Liu Z, Altinel B, Hüsünbeyi ZM, Özgür A, Fergadis A, Wang CK, Dai HJ, Tran T, Kavuluru R, Luo L, Steppi A, Zhang J, Qu J, Lu Z. Overview of the BioCreative VI Precision Medicine Track: mining protein interactions and mutations for precision medicine. Database (Oxford). 2019 Jan 01;2019. doi: 10.1093/database/bay147. PMID: 30689846; PMCID: PMC6348314.
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Twitter discourse on nicotine as potential prophylactic or therapeutic for COVID-19.

The International journal on drug policy

Kavuluru R, Noh J, Rose SW.
PMID: 34607223
Int J Drug Policy. 2022 Jan;99:103470. doi: 10.1016/j.drugpo.2021.103470. Epub 2021 Sep 20.

BACKGROUND: An unproven "nicotine hypothesis" that indicates nicotine's therapeutic potential for COVID-19 has been proposed in recent literature. This study is about Twitter posts that misinterpret this hypothesis to make baseless claims about benefits of smoking and vaping in...

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Kavuluru R, Noh J, Rose SW. Twitter discourse on nicotine as potential prophylactic or therapeutic for COVID-19. Int J Drug Policy. 2021;99:103470doi: 10.1016/j.drugpo.2021.103470.
Kavuluru, R., Noh, J., & Rose, S. W. (2022). Twitter discourse on nicotine as potential prophylactic or therapeutic for COVID-19. The International journal on drug policy, 99103470. https://doi.org/10.1016/j.drugpo.2021.103470
Kavuluru, Ramakanth, et al. "Twitter discourse on nicotine as potential prophylactic or therapeutic for COVID-19." The International journal on drug policy vol. 99 (2022): 103470. doi: https://doi.org/10.1016/j.drugpo.2021.103470
Kavuluru R, Noh J, Rose SW. Twitter discourse on nicotine as potential prophylactic or therapeutic for COVID-19. Int J Drug Policy. 2022 Jan;99:103470. doi: 10.1016/j.drugpo.2021.103470. Epub 2021 Sep 20. PMID: 34607223; PMCID: PMC8450069.
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Pharmacologic manipulation of skin pigmentation.

Pigment cell & melanoma research

Kindl GH, D'Orazio JA.
PMID: 33666358
Pigment Cell Melanoma Res. 2021 Jul;34(4):777-785. doi: 10.1111/pcmr.12969. Epub 2021 Mar 12.

Skin complexion is among the most recognizable phenotypes between individuals and is mainly determined by the amount and type of melanin pigment deposited in the epidermis. Persons with dark skin complexion have more of a brown/black pigment known as...

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Kindl GH, D'Orazio JA. Pharmacologic manipulation of skin pigmentation. Pigment Cell Melanoma Res. 2021;34(4):777-785doi: 10.1111/pcmr.12969.
Kindl, G. H., & D'Orazio, J. A. (2021). Pharmacologic manipulation of skin pigmentation. Pigment cell & melanoma research, 34(4), 777-785. https://doi.org/10.1111/pcmr.12969
Kindl, Gabriel H, and D'Orazio, John A. "Pharmacologic manipulation of skin pigmentation." Pigment cell & melanoma research vol. 34,4 (2021): 777-785. doi: https://doi.org/10.1111/pcmr.12969
Kindl GH, D'Orazio JA. Pharmacologic manipulation of skin pigmentation. Pigment Cell Melanoma Res. 2021 Jul;34(4):777-785. doi: 10.1111/pcmr.12969. Epub 2021 Mar 12. PMID: 33666358; PMCID: PMC8277713.
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Hematological adverse events in the management of glioblastoma.

Journal of neuro-oncology

Garcia CR, Myint ZW, Jayswal R, Wang C, Morgan RM, Butts AR, Weiss HL, Villano JL.
PMID: 34820776
J Neurooncol. 2021 Nov 24; doi: 10.1007/s11060-021-03891-8. Epub 2021 Nov 24.

BACKGROUND: Hematological adverse events (HAEs) are common during treatment for glioblastoma (GBM), usually associated with temozolomide (TMZ). Their clinical value is uncertain, as few investigations have focused on outcomes for HAEs during GBM treatment.METHODS: We combined data from two...

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Garcia CR, Myint ZW, Jayswal R, et al. Hematological adverse events in the management of glioblastoma. J Neurooncol. 2021;doi: 10.1007/s11060-021-03891-8.
Garcia, C. R., Myint, Z. W., Jayswal, R., Wang, C., Morgan, R. M., Butts, A. R., Weiss, H. L., & Villano, J. L. (2021). Hematological adverse events in the management of glioblastoma. Journal of neuro-oncology, . https://doi.org/10.1007/s11060-021-03891-8
Garcia, Catherine R, et al. "Hematological adverse events in the management of glioblastoma." Journal of neuro-oncology vol. (2021). doi: https://doi.org/10.1007/s11060-021-03891-8
Garcia CR, Myint ZW, Jayswal R, Wang C, Morgan RM, Butts AR, Weiss HL, Villano JL. Hematological adverse events in the management of glioblastoma. J Neurooncol. 2021 Nov 24; doi: 10.1007/s11060-021-03891-8. Epub 2021 Nov 24. PMID: 34820776.
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Blockade of 6-phosphogluconate dehydrogenase generates CD8.

Cell reports

Daneshmandi S, Cassel T, Lin P, Higashi RM, Wulf GM, Boussiotis VA, Fan TW, Seth P.
PMID: 33691103
Cell Rep. 2021 Mar 09;34(10):108831. doi: 10.1016/j.celrep.2021.108831.

Although T cell expansion depends on glycolysis, T effector cell differentiation requires signaling via the production of reactive oxygen species (ROS). Because the pentose phosphate pathway (PPP) regulates ROS by generating nicotinamide adenine dinucleotide phosphate (NADPH), we examined how...

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Daneshmandi S, Cassel T, Lin P, et al. Blockade of 6-phosphogluconate dehydrogenase generates CD8. Cell Rep. 2021;34(10):108831doi: 10.1016/j.celrep.2021.108831.
Daneshmandi, S., Cassel, T., Lin, P., Higashi, R. M., Wulf, G. M., Boussiotis, V. A., Fan, T. W., & Seth, P. (2021). Blockade of 6-phosphogluconate dehydrogenase generates CD8. Cell reports, 34(10), 108831. https://doi.org/10.1016/j.celrep.2021.108831
Daneshmandi, Saeed, et al. "Blockade of 6-phosphogluconate dehydrogenase generates CD8." Cell reports vol. 34,10 (2021): 108831. doi: https://doi.org/10.1016/j.celrep.2021.108831
Daneshmandi S, Cassel T, Lin P, Higashi RM, Wulf GM, Boussiotis VA, Fan TW, Seth P. Blockade of 6-phosphogluconate dehydrogenase generates CD8. Cell Rep. 2021 Mar 09;34(10):108831. doi: 10.1016/j.celrep.2021.108831. PMID: 33691103; PMCID: PMC8051863.
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Showing 1 to 12 of 147 entries