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Health and Cardiometabolic Disease in Transgender Adults in the United States: Behavioral Risk Factor Surveillance System 2015.

Journal of the Endocrine Society

Nokoff NJ, Scarbro S, Juarez-Colunga E, Moreau KL, Kempe A.
PMID: 29577110
J Endocr Soc. 2018 Mar 05;2(4):349-360. doi: 10.1210/js.2017-00465. eCollection 2018 Apr 01.

CONTEXT: Little is known about the health of transgender adults in the United States, a growing population. There have been no large reports examining differences in health status and cardiometabolic disease in subgroups of transgender adults [female-to-male (FTM), male-to-female...

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Nokoff NJ, Scarbro S, Juarez-Colunga E, et al. Health and Cardiometabolic Disease in Transgender Adults in the United States: Behavioral Risk Factor Surveillance System 2015. J Endocr Soc. 2018;2(4):349-360doi: 10.1210/js.2017-00465.
Nokoff, N. J., Scarbro, S., Juarez-Colunga, E., Moreau, K. L., & Kempe, A. (2018). Health and Cardiometabolic Disease in Transgender Adults in the United States: Behavioral Risk Factor Surveillance System 2015. Journal of the Endocrine Society, 2(4), 349-360. https://doi.org/10.1210/js.2017-00465
Nokoff, Natalie J, et al. "Health and Cardiometabolic Disease in Transgender Adults in the United States: Behavioral Risk Factor Surveillance System 2015." Journal of the Endocrine Society vol. 2,4 (2018): 349-360. doi: https://doi.org/10.1210/js.2017-00465
Nokoff NJ, Scarbro S, Juarez-Colunga E, Moreau KL, Kempe A. Health and Cardiometabolic Disease in Transgender Adults in the United States: Behavioral Risk Factor Surveillance System 2015. J Endocr Soc. 2018 Mar 05;2(4):349-360. doi: 10.1210/js.2017-00465. eCollection 2018 Apr 01. PMID: 29577110; PMCID: PMC5863219.
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Validity of Ratings of Perceived Exertion in Patients with Type 2 Diabetes.

Journal of novel physiotherapy and physical rehabilitation

Unick JL, Gaussoin S, Bahnson J, Crow R, Curtis J, Killean T, Regensteiner JG, Stewart KJ, Wing RR, Jakicic JM.
PMID: 25485308
J Nov Physiother Phys Rehabil. 2014 Jul 07;1(1).

PURPOSE: To examine whether a subjective measure of moderate-intensity exercise (12-13 on Borg's ratings of perceived exertion scale; RPE) corresponds to the target heart rate for moderate-intensity exercise (40-59% heart rate reserve; %HRR) and to determine the characteristics of...

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Unick JL, Gaussoin S, Bahnson J, et al. Validity of Ratings of Perceived Exertion in Patients with Type 2 Diabetes. J Nov Physiother Phys Rehabil. 2014;1(1)
Unick, J. L., Gaussoin, S., Bahnson, J., Crow, R., Curtis, J., Killean, T., Regensteiner, J. G., Stewart, K. J., Wing, R. R., Jakicic, J. M. (2014). Validity of Ratings of Perceived Exertion in Patients with Type 2 Diabetes. Journal of novel physiotherapy and physical rehabilitation, 1(1), .
Unick, Jessica L, et al. "Validity of Ratings of Perceived Exertion in Patients with Type 2 Diabetes." Journal of novel physiotherapy and physical rehabilitation vol. 1,1 (2014).
Unick JL, Gaussoin S, Bahnson J, Crow R, Curtis J, Killean T, Regensteiner JG, Stewart KJ, Wing RR, Jakicic JM. Validity of Ratings of Perceived Exertion in Patients with Type 2 Diabetes. J Nov Physiother Phys Rehabil. 2014 Jul 07;1(1). PMID: 25485308; PMCID: PMC4255703.
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Sex differences in obesity development in pair-fed neuronal lipoprotein lipase deficient mice.

Molecular metabolism

Wang H, Wang Y, Taussig MD, Eckel RH.
PMID: 27689015
Mol Metab. 2016 May 31;5(10):1025-1032. doi: 10.1016/j.molmet.2016.05.013. eCollection 2016 Oct.

OBJECTIVE: Compared to men, postmenopausal women suffer from a disproportionate burden of many co-morbidities associated with obesity, e.g. cardiovascular disease, cancer, and dementia. The underlying mechanism for this sex difference is not well understood but is believed to relate...

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Wang H, Wang Y, Taussig MD, et al. Sex differences in obesity development in pair-fed neuronal lipoprotein lipase deficient mice. Mol Metab. 2016;5(10):1025-1032doi: 10.1016/j.molmet.2016.05.013.
Wang, H., Wang, Y., Taussig, M. D., & Eckel, R. H. (2016). Sex differences in obesity development in pair-fed neuronal lipoprotein lipase deficient mice. Molecular metabolism, 5(10), 1025-1032. https://doi.org/10.1016/j.molmet.2016.05.013
Wang, Hong, et al. "Sex differences in obesity development in pair-fed neuronal lipoprotein lipase deficient mice." Molecular metabolism vol. 5,10 (2016): 1025-1032. doi: https://doi.org/10.1016/j.molmet.2016.05.013
Wang H, Wang Y, Taussig MD, Eckel RH. Sex differences in obesity development in pair-fed neuronal lipoprotein lipase deficient mice. Mol Metab. 2016 May 31;5(10):1025-1032. doi: 10.1016/j.molmet.2016.05.013. eCollection 2016 Oct. PMID: 27689015; PMCID: PMC5034494.
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Hemoglobin A1c measurement for the diagnosis of Type 2 diabetes in children.

International journal of pediatric endocrinology

Kapadia C, Zeitler P.
PMID: 23256825
Int J Pediatr Endocrinol. 2012 Dec 20;2012(1):31. doi: 10.1186/1687-9856-2012-31.

Laboratory measurements of hemoglobin A1c above 6.5% were approved as an additional diagnostic criteria for diabetes mellitus by the American Diabetes Association in 2010. Several recent pediatric studies have cast HbA1c measurement in children in an unfavorable light in...

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Kapadia C, Zeitler P. Hemoglobin A1c measurement for the diagnosis of Type 2 diabetes in children. Int J Pediatr Endocrinol. 2012;2012(1):31doi: 10.1186/1687-9856-2012-31.
Kapadia, C., Zeitler, P. (2012). Hemoglobin A1c measurement for the diagnosis of Type 2 diabetes in children. International journal of pediatric endocrinology, 2012(1), 31. https://doi.org/10.1186/1687-9856-2012-31
Kapadia, Chirag, et al. "Hemoglobin A1c measurement for the diagnosis of Type 2 diabetes in children." International journal of pediatric endocrinology vol. 2012,1 (2012): 31. doi: https://doi.org/10.1186/1687-9856-2012-31
Kapadia C, Zeitler P. Hemoglobin A1c measurement for the diagnosis of Type 2 diabetes in children. Int J Pediatr Endocrinol. 2012 Dec 20;2012(1):31. doi: 10.1186/1687-9856-2012-31. PMID: 23256825; PMCID: PMC3564771.
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Long-term impact of intensive lifestyle intervention on cognitive function assessed with the National Institutes of Health Toolbox: The Look AHEAD study.

Alzheimer's & dementia (Amsterdam, Netherlands)

Hayden KM, Baker LD, Bray G, Carvajal R, Demos-McDermott K, Hergenroeder AL, Hill JO, Horton E, Jakicic JM, Johnson KC, Neiberg RH, Rapp SR, Wadden TA, Miller ME.
PMID: 29159267
Alzheimers Dement (Amst). 2017 Oct 09;10:41-48. doi: 10.1016/j.dadm.2017.09.002. eCollection 2018.

INTRODUCTION: This study sought to determine whether 10 years of assignment to intensive lifestyle intervention (ILI) relative to diabetes support and education leads to better cognition. We examine intervention effects overall and among clinical subgroups, and report correlations between...

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Hayden KM, Baker LD, Bray G, et al. Long-term impact of intensive lifestyle intervention on cognitive function assessed with the National Institutes of Health Toolbox: The Look AHEAD study. Alzheimers Dement (Amst). 2017;10:41-48doi: 10.1016/j.dadm.2017.09.002.
Hayden, K. M., Baker, L. D., Bray, G., Carvajal, R., Demos-McDermott, K., Hergenroeder, A. L., Hill, J. O., Horton, E., Jakicic, J. M., Johnson, K. C., Neiberg, R. H., Rapp, S. R., Wadden, T. A., & Miller, M. E. (2018). Long-term impact of intensive lifestyle intervention on cognitive function assessed with the National Institutes of Health Toolbox: The Look AHEAD study. Alzheimer's & dementia (Amsterdam, Netherlands), 1041-48. https://doi.org/10.1016/j.dadm.2017.09.002
Hayden, Kathleen M, et al. "Long-term impact of intensive lifestyle intervention on cognitive function assessed with the National Institutes of Health Toolbox: The Look AHEAD study." Alzheimer's & dementia (Amsterdam, Netherlands) vol. 10 (2018): 41-48. doi: https://doi.org/10.1016/j.dadm.2017.09.002
Hayden KM, Baker LD, Bray G, Carvajal R, Demos-McDermott K, Hergenroeder AL, Hill JO, Horton E, Jakicic JM, Johnson KC, Neiberg RH, Rapp SR, Wadden TA, Miller ME. Long-term impact of intensive lifestyle intervention on cognitive function assessed with the National Institutes of Health Toolbox: The Look AHEAD study. Alzheimers Dement (Amst). 2017 Oct 09;10:41-48. doi: 10.1016/j.dadm.2017.09.002. eCollection 2018. PMID: 29159267; PMCID: PMC5675717.
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Pyrroloquinoline quinone prevents developmental programming of microbial dysbiosis and macrophage polarization to attenuate liver fibrosis in offspring of obese mice.

Hepatology communications

Friedman JE, Dobrinskikh E, Alfonso-Garcia A, Fast A, Janssen RC, Soderborg TK, Anderson AL, Reisz JA, D'Alessandro A, Frank DN, Robertson CE, de la Houssaye BA, Johnson LK, Orlicky DJ, Wang XX, Levi M, Potma EO, El Kasmi KC, Jonscher KR.
PMID: 29507905
Hepatol Commun. 2018 Jan 22;2(3):313-328. doi: 10.1002/hep4.1139. eCollection 2018 Mar.

Increasingly, evidence suggests that exposure to maternal obesity creates an inflammatory environment

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Friedman JE, Dobrinskikh E, Alfonso-Garcia A, et al. Pyrroloquinoline quinone prevents developmental programming of microbial dysbiosis and macrophage polarization to attenuate liver fibrosis in offspring of obese mice. Hepatol Commun. 2018;2(3):313-328doi: 10.1002/hep4.1139.
Friedman, J. E., Dobrinskikh, E., Alfonso-Garcia, A., Fast, A., Janssen, R. C., Soderborg, T. K., Anderson, A. L., Reisz, J. A., D'Alessandro, A., Frank, D. N., Robertson, C. E., de la Houssaye, B. A., Johnson, L. K., Orlicky, D. J., Wang, X. X., Levi, M., Potma, E. O., El Kasmi, K. C., & Jonscher, K. R. (2018). Pyrroloquinoline quinone prevents developmental programming of microbial dysbiosis and macrophage polarization to attenuate liver fibrosis in offspring of obese mice. Hepatology communications, 2(3), 313-328. https://doi.org/10.1002/hep4.1139
Friedman, Jacob E, et al. "Pyrroloquinoline quinone prevents developmental programming of microbial dysbiosis and macrophage polarization to attenuate liver fibrosis in offspring of obese mice." Hepatology communications vol. 2,3 (2018): 313-328. doi: https://doi.org/10.1002/hep4.1139
Friedman JE, Dobrinskikh E, Alfonso-Garcia A, Fast A, Janssen RC, Soderborg TK, Anderson AL, Reisz JA, D'Alessandro A, Frank DN, Robertson CE, de la Houssaye BA, Johnson LK, Orlicky DJ, Wang XX, Levi M, Potma EO, El Kasmi KC, Jonscher KR. Pyrroloquinoline quinone prevents developmental programming of microbial dysbiosis and macrophage polarization to attenuate liver fibrosis in offspring of obese mice. Hepatol Commun. 2018 Jan 22;2(3):313-328. doi: 10.1002/hep4.1139. eCollection 2018 Mar. PMID: 29507905; PMCID: PMC5831029.
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Lipoprotein Lipase Is a Feature of Alternatively-Activated Microglia and May Facilitate Lipid Uptake in the CNS During Demyelination.

Frontiers in molecular neuroscience

Bruce KD, Gorkhali S, Given K, Coates AM, Boyle KE, Macklin WB, Eckel RH.
PMID: 29599706
Front Mol Neurosci. 2018 Mar 15;11:57. doi: 10.3389/fnmol.2018.00057. eCollection 2018.

Severe demyelinating disorders of the central nervous system (CNS) such as multiple sclerosis (MS), can be devastating for many young lives. To date, the factors resulting in poor remyelination and repair are not well understood, and reparative therapies that...

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Bruce KD, Gorkhali S, Given K, et al. Lipoprotein Lipase Is a Feature of Alternatively-Activated Microglia and May Facilitate Lipid Uptake in the CNS During Demyelination. Front Mol Neurosci. 2018;11:57doi: 10.3389/fnmol.2018.00057.
Bruce, K. D., Gorkhali, S., Given, K., Coates, A. M., Boyle, K. E., Macklin, W. B., & Eckel, R. H. (2018). Lipoprotein Lipase Is a Feature of Alternatively-Activated Microglia and May Facilitate Lipid Uptake in the CNS During Demyelination. Frontiers in molecular neuroscience, 1157. https://doi.org/10.3389/fnmol.2018.00057
Bruce, Kimberley D, et al. "Lipoprotein Lipase Is a Feature of Alternatively-Activated Microglia and May Facilitate Lipid Uptake in the CNS During Demyelination." Frontiers in molecular neuroscience vol. 11 (2018): 57. doi: https://doi.org/10.3389/fnmol.2018.00057
Bruce KD, Gorkhali S, Given K, Coates AM, Boyle KE, Macklin WB, Eckel RH. Lipoprotein Lipase Is a Feature of Alternatively-Activated Microglia and May Facilitate Lipid Uptake in the CNS During Demyelination. Front Mol Neurosci. 2018 Mar 15;11:57. doi: 10.3389/fnmol.2018.00057. eCollection 2018. PMID: 29599706; PMCID: PMC5862862.
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Lipid catabolism inhibition sensitizes prostate cancer cells to antiandrogen blockade.

Oncotarget

Flaig TW, Salzmann-Sullivan M, Su LJ, Zhang Z, Joshi M, Gijón MA, Kim J, Arcaroli JJ, Van Bokhoven A, Lucia MS, La Rosa FG, Schlaepfer IR.
PMID: 28915573
Oncotarget. 2017 Apr 21;8(34):56051-56065. doi: 10.18632/oncotarget.17359. eCollection 2017 Aug 22.

Prostate cancer (PCa) is the most common malignancy among Western men and the second leading-cause of cancer related deaths. For men who develop metastatic castration resistant PCa (mCRPC), survival is limited, making the identification of novel therapies for mCRPC...

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Flaig TW, Salzmann-Sullivan M, Su LJ, et al. Lipid catabolism inhibition sensitizes prostate cancer cells to antiandrogen blockade. Oncotarget. 2017;8(34):56051-56065doi: 10.18632/oncotarget.17359.
Flaig, T. W., Salzmann-Sullivan, M., Su, L. J., Zhang, Z., Joshi, M., Gijón, M. A., Kim, J., Arcaroli, J. J., Van Bokhoven, A., Lucia, M. S., La Rosa, F. G., & Schlaepfer, I. R. (2017). Lipid catabolism inhibition sensitizes prostate cancer cells to antiandrogen blockade. Oncotarget, 8(34), 56051-56065. https://doi.org/10.18632/oncotarget.17359
Flaig, Thomas W, et al. "Lipid catabolism inhibition sensitizes prostate cancer cells to antiandrogen blockade." Oncotarget vol. 8,34 (2017): 56051-56065. doi: https://doi.org/10.18632/oncotarget.17359
Flaig TW, Salzmann-Sullivan M, Su LJ, Zhang Z, Joshi M, Gijón MA, Kim J, Arcaroli JJ, Van Bokhoven A, Lucia MS, La Rosa FG, Schlaepfer IR. Lipid catabolism inhibition sensitizes prostate cancer cells to antiandrogen blockade. Oncotarget. 2017 Apr 21;8(34):56051-56065. doi: 10.18632/oncotarget.17359. eCollection 2017 Aug 22. PMID: 28915573; PMCID: PMC5593544.
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Local environmental quality positively predicts breastfeeding in the UK's Millennium Cohort Study.

Evolution, medicine, and public health

Brown LJ, Sear R.
PMID: 29354262
Evol Med Public Health. 2017 Aug 21;2017(1):120-135. doi: 10.1093/emph/eox011. eCollection 2017.

No abstract available.

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Brown LJ, Sear R. Local environmental quality positively predicts breastfeeding in the UK's Millennium Cohort Study. Evol Med Public Health. 2017;2017(1):120-135doi: 10.1093/emph/eox011.
Brown, L. J., & Sear, R. (2017). Local environmental quality positively predicts breastfeeding in the UK's Millennium Cohort Study. Evolution, medicine, and public health, 2017(1), 120-135. https://doi.org/10.1093/emph/eox011
Brown, Laura J, and Sear, Rebecca. "Local environmental quality positively predicts breastfeeding in the UK's Millennium Cohort Study." Evolution, medicine, and public health vol. 2017,1 (2017): 120-135. doi: https://doi.org/10.1093/emph/eox011
Brown LJ, Sear R. Local environmental quality positively predicts breastfeeding in the UK's Millennium Cohort Study. Evol Med Public Health. 2017 Aug 21;2017(1):120-135. doi: 10.1093/emph/eox011. eCollection 2017. PMID: 29354262; PMCID: PMC5766197.
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Propensity to obesity impacts the neuronal response to energy imbalance.

Frontiers in behavioral neuroscience

Cornier MA, McFadden KL, Thomas EA, Bechtell JL, Bessesen DH, Tregellas JR.
PMID: 25767441
Front Behav Neurosci. 2015 Feb 26;9:52. doi: 10.3389/fnbeh.2015.00052. eCollection 2015.

The mechanisms responsible for the propensity to gain weight or remain normal weight are poorly understood. The objective of this study was to study the neuronal response to visual food cues during short-term energy imbalance in healthy adults recruited...

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Cornier MA, McFadden KL, Thomas EA, et al. Propensity to obesity impacts the neuronal response to energy imbalance. Front Behav Neurosci. 2015;9:52doi: 10.3389/fnbeh.2015.00052.
Cornier, M. A., McFadden, K. L., Thomas, E. A., Bechtell, J. L., Bessesen, D. H., & Tregellas, J. R. (2015). Propensity to obesity impacts the neuronal response to energy imbalance. Frontiers in behavioral neuroscience, 952. https://doi.org/10.3389/fnbeh.2015.00052
Cornier, Marc-Andre, et al. "Propensity to obesity impacts the neuronal response to energy imbalance." Frontiers in behavioral neuroscience vol. 9 (2015): 52. doi: https://doi.org/10.3389/fnbeh.2015.00052
Cornier MA, McFadden KL, Thomas EA, Bechtell JL, Bessesen DH, Tregellas JR. Propensity to obesity impacts the neuronal response to energy imbalance. Front Behav Neurosci. 2015 Feb 26;9:52. doi: 10.3389/fnbeh.2015.00052. eCollection 2015. PMID: 25767441; PMCID: PMC4341570.
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Meal Microstructure Characterization from Sensor-Based Food Intake Detection.

Frontiers in nutrition

Doulah A, Farooq M, Yang X, Parton J, McCrory MA, Higgins JA, Sazonov E.
PMID: 28770206
Front Nutr. 2017 Jul 17;4:31. doi: 10.3389/fnut.2017.00031. eCollection 2017.

To avoid the pitfalls of self-reported dietary intake, wearable sensors can be used. Many food ingestion sensors offer the ability to automatically detect food intake using time resolutions that range from 23 ms to 8 min. There is no...

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Doulah A, Farooq M, Yang X, et al. Meal Microstructure Characterization from Sensor-Based Food Intake Detection. Front Nutr. 2017;4:31doi: 10.3389/fnut.2017.00031.
Doulah, A., Farooq, M., Yang, X., Parton, J., McCrory, M. A., Higgins, J. A., & Sazonov, E. (2017). Meal Microstructure Characterization from Sensor-Based Food Intake Detection. Frontiers in nutrition, 431. https://doi.org/10.3389/fnut.2017.00031
Doulah, Abul, et al. "Meal Microstructure Characterization from Sensor-Based Food Intake Detection." Frontiers in nutrition vol. 4 (2017): 31. doi: https://doi.org/10.3389/fnut.2017.00031
Doulah A, Farooq M, Yang X, Parton J, McCrory MA, Higgins JA, Sazonov E. Meal Microstructure Characterization from Sensor-Based Food Intake Detection. Front Nutr. 2017 Jul 17;4:31. doi: 10.3389/fnut.2017.00031. eCollection 2017. PMID: 28770206; PMCID: PMC5512009.
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Harnessing centred identity transformation to reduce executive function burden for maintenance of health behaviour change: the Maintain IT model.

Health psychology review

Caldwell AE, Masters KS, Peters JC, Bryan AD, Grigsby J, Hooker SA, Wyatt HR, Hill JO.
PMID: 29402182
Health Psychol Rev. 2018 Sep;12(3):231-253. doi: 10.1080/17437199.2018.1437551. Epub 2018 Feb 19.

The inability to produce sustainable lifestyle modifications (e.g., physical activity, healthy diet) remains a major barrier to reducing morbidity and mortality from prevalent, preventable conditions. The objective of this paper is to present a model that builds on and...

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Caldwell AE, Masters KS, Peters JC, et al. Harnessing centred identity transformation to reduce executive function burden for maintenance of health behaviour change: the Maintain IT model. Health Psychol Rev. 2018;12(3):231-253doi: 10.1080/17437199.2018.1437551.
Caldwell, A. E., Masters, K. S., Peters, J. C., Bryan, A. D., Grigsby, J., Hooker, S. A., Wyatt, H. R., & Hill, J. O. (2018). Harnessing centred identity transformation to reduce executive function burden for maintenance of health behaviour change: the Maintain IT model. Health psychology review, 12(3), 231-253. https://doi.org/10.1080/17437199.2018.1437551
Caldwell, Ann E, et al. "Harnessing centred identity transformation to reduce executive function burden for maintenance of health behaviour change: the Maintain IT model." Health psychology review vol. 12,3 (2018): 231-253. doi: https://doi.org/10.1080/17437199.2018.1437551
Caldwell AE, Masters KS, Peters JC, Bryan AD, Grigsby J, Hooker SA, Wyatt HR, Hill JO. Harnessing centred identity transformation to reduce executive function burden for maintenance of health behaviour change: the Maintain IT model. Health Psychol Rev. 2018 Sep;12(3):231-253. doi: 10.1080/17437199.2018.1437551. Epub 2018 Feb 19. PMID: 29402182; PMCID: PMC6124500.
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