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Showing 1 to 12 of 16 entries
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Optimization of focusing through scattering media using the continuous sequential algorithm.

Journal of modern optics

Thompson JV, Hokr BH, Yakovlev VV.
PMID: 27018179
J Mod Opt. 2016;63(1):80-84. doi: 10.1080/09500340.2015.1073804. Epub 2015 Aug 11.

The ability to control the propagation of light through scattering media is essential for atmospheric optics, astronomy, biomedical imaging and remote sensing. The optimization of focusing light through a scattering medium is of particular interest for the case of...

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Thompson JV, Hokr BH, Yakovlev VV. Optimization of focusing through scattering media using the continuous sequential algorithm. J Mod Opt. 2015;63(1):80-84doi: 10.1080/09500340.2015.1073804.
Thompson, J. V., Hokr, B. H., & Yakovlev, V. V. (2016). Optimization of focusing through scattering media using the continuous sequential algorithm. Journal of modern optics, 63(1), 80-84. https://doi.org/10.1080/09500340.2015.1073804
Thompson, J V, et al. "Optimization of focusing through scattering media using the continuous sequential algorithm." Journal of modern optics vol. 63,1 (2016): 80-84. doi: https://doi.org/10.1080/09500340.2015.1073804
Thompson JV, Hokr BH, Yakovlev VV. Optimization of focusing through scattering media using the continuous sequential algorithm. J Mod Opt. 2016;63(1):80-84. doi: 10.1080/09500340.2015.1073804. Epub 2015 Aug 11. PMID: 27018179; PMCID: PMC4803298.
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Pharmaceuticals and personal care products in waters: occurrence, toxicity, and risk.

Environmental chemistry letters

Cizmas L, Sharma VK, Gray CM, McDonald TJ.
PMID: 28592954
Environ Chem Lett. 2015 Dec;13(4):381-394. doi: 10.1007/s10311-015-0524-4. Epub 2015 Aug 26.

Pharmaceuticals and personal care products (PPCP) are compounds with special physical and chemical properties that address the care of animal and human health. PPCP have been detected in surface water and wastewater in the ng/L to µg/L concentration range...

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Cizmas L, Sharma VK, Gray CM, et al. Pharmaceuticals and personal care products in waters: occurrence, toxicity, and risk. Environ Chem Lett. 2015;13(4):381-394doi: 10.1007/s10311-015-0524-4.
Cizmas, L., Sharma, V. K., Gray, C. M., & McDonald, T. J. (2015). Pharmaceuticals and personal care products in waters: occurrence, toxicity, and risk. Environmental chemistry letters, 13(4), 381-394. https://doi.org/10.1007/s10311-015-0524-4
Cizmas, Leslie, et al. "Pharmaceuticals and personal care products in waters: occurrence, toxicity, and risk." Environmental chemistry letters vol. 13,4 (2015): 381-394. doi: https://doi.org/10.1007/s10311-015-0524-4
Cizmas L, Sharma VK, Gray CM, McDonald TJ. Pharmaceuticals and personal care products in waters: occurrence, toxicity, and risk. Environ Chem Lett. 2015 Dec;13(4):381-394. doi: 10.1007/s10311-015-0524-4. Epub 2015 Aug 26. PMID: 28592954; PMCID: PMC5459316.
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The Aryl Hydrocarbon Receptor (AhR) as a Drug Target for Cancer Chemotherapy.

Current opinion in toxicology

Safe S, Cheng Y, Jin UH.
PMID: 28459113
Curr Opin Toxicol. 2017 Feb;2:24-29. doi: 10.1016/j.cotox.2017.01.012. Epub 2017 Feb 01.

The aryl hydrocarbon receptor (AhR) is overexpressed in some patients with different tumor types, and the receptor can be a negative or positive prognostic factor. There is also evidence from both

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Safe S, Cheng Y, Jin UH. The Aryl Hydrocarbon Receptor (AhR) as a Drug Target for Cancer Chemotherapy. Curr Opin Toxicol. 2017;2:24-29doi: 10.1016/j.cotox.2017.01.012.
Safe, S., Cheng, Y., & Jin, U. H. (2017). The Aryl Hydrocarbon Receptor (AhR) as a Drug Target for Cancer Chemotherapy. Current opinion in toxicology, 224-29. https://doi.org/10.1016/j.cotox.2017.01.012
Safe, Stephen, et al. "The Aryl Hydrocarbon Receptor (AhR) as a Drug Target for Cancer Chemotherapy." Current opinion in toxicology vol. 2 (2017): 24-29. doi: https://doi.org/10.1016/j.cotox.2017.01.012
Safe S, Cheng Y, Jin UH. The Aryl Hydrocarbon Receptor (AhR) as a Drug Target for Cancer Chemotherapy. Curr Opin Toxicol. 2017 Feb;2:24-29. doi: 10.1016/j.cotox.2017.01.012. Epub 2017 Feb 01. PMID: 28459113; PMCID: PMC5407490.
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A chemical-biological similarity-based grouping of complex substances as a prototype approach for evaluating chemical alternatives.

Green chemistry : an international journal and green chemistry resource : GC

Grimm FA, Iwata Y, Sirenko O, Chappell GA, Wright FA, Reif DM, Braisted J, Gerhold DL, Yeakley JM, Shepard P, Seligmann B, Roy T, Boogaard PJ, Ketelslegers HB, Rohde AM, Rusyn I.
PMID: 28035192
Green Chem. 2016 Aug 21;18(16):4407-4419. doi: 10.1039/c6gc01147k. Epub 2016 May 16.

Comparative assessment of potential human health impacts is a critical step in evaluating both chemical alternatives and existing products on the market. Most alternatives assessments are conducted on a chemical-by-chemical basis and it is seldom acknowledged that humans are...

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Grimm FA, Iwata Y, Sirenko O, et al. A chemical-biological similarity-based grouping of complex substances as a prototype approach for evaluating chemical alternatives. Green Chem. 2016;18(16):4407-4419doi: 10.1039/c6gc01147k.
Grimm, F. A., Iwata, Y., Sirenko, O., Chappell, G. A., Wright, F. A., Reif, D. M., Braisted, J., Gerhold, D. L., Yeakley, J. M., Shepard, P., Seligmann, B., Roy, T., Boogaard, P. J., Ketelslegers, H. B., Rohde, A. M., & Rusyn, I. (2016). A chemical-biological similarity-based grouping of complex substances as a prototype approach for evaluating chemical alternatives. Green chemistry : an international journal and green chemistry resource : GC, 18(16), 4407-4419. https://doi.org/10.1039/c6gc01147k
Grimm, Fabian A, et al. "A chemical-biological similarity-based grouping of complex substances as a prototype approach for evaluating chemical alternatives." Green chemistry : an international journal and green chemistry resource : GC vol. 18,16 (2016): 4407-4419. doi: https://doi.org/10.1039/c6gc01147k
Grimm FA, Iwata Y, Sirenko O, Chappell GA, Wright FA, Reif DM, Braisted J, Gerhold DL, Yeakley JM, Shepard P, Seligmann B, Roy T, Boogaard PJ, Ketelslegers HB, Rohde AM, Rusyn I. A chemical-biological similarity-based grouping of complex substances as a prototype approach for evaluating chemical alternatives. Green Chem. 2016 Aug 21;18(16):4407-4419. doi: 10.1039/c6gc01147k. Epub 2016 May 16. PMID: 28035192; PMCID: PMC5179981.
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Chemoprotective epigenetic mechanisms in a colorectal cancer model: Modulation by n-3 PUFA in combination with fermentable fiber.

Current pharmacology reports

Triff K, Kim E, Chapkin RS.
PMID: 25938013
Curr Pharmacol Rep. 2015 Feb;1(1):11-20. doi: 10.1007/s40495-014-0005-7.

Colorectal cancer is the third major cause of cancer-related mortality in both men and women worldwide. The beneficial role of n-3 polyunsaturated fatty acids (PUFA) in preventing colon cancer is substantiated by experimental, epidemiological, and clinical data. From a...

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Triff K, Kim E, Chapkin RS. Chemoprotective epigenetic mechanisms in a colorectal cancer model: Modulation by n-3 PUFA in combination with fermentable fiber. Curr Pharmacol Rep. 2015;1(1):11-20doi: 10.1007/s40495-014-0005-7.
Triff, K., Kim, E., & Chapkin, R. S. (2015). Chemoprotective epigenetic mechanisms in a colorectal cancer model: Modulation by n-3 PUFA in combination with fermentable fiber. Current pharmacology reports, 1(1), 11-20. https://doi.org/10.1007/s40495-014-0005-7
Triff, Karen, et al. "Chemoprotective epigenetic mechanisms in a colorectal cancer model: Modulation by n-3 PUFA in combination with fermentable fiber." Current pharmacology reports vol. 1,1 (2015): 11-20. doi: https://doi.org/10.1007/s40495-014-0005-7
Triff K, Kim E, Chapkin RS. Chemoprotective epigenetic mechanisms in a colorectal cancer model: Modulation by n-3 PUFA in combination with fermentable fiber. Curr Pharmacol Rep. 2015 Feb;1(1):11-20. doi: 10.1007/s40495-014-0005-7. PMID: 25938013; PMCID: PMC4414264.
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The Huntingtin-interacting protein SETD2/HYPB is an actin lysine methyltransferase.

Science advances

Seervai RNH, Jangid RK, Karki M, Tripathi DN, Jung SY, Kearns SE, Verhey KJ, Cianfrocco MA, Millis BA, Tyska MJ, Mason FM, Rathmell WK, Park IY, Dere R, Walker CL.
PMID: 33008892
Sci Adv. 2020 Oct 02;6(40). doi: 10.1126/sciadv.abb7854. Print 2020 Oct.

The methyltransferase SET domain-containing 2 (SETD2) was originally identified as Huntingtin (HTT) yeast partner B. However, a SETD2 function associated with the HTT scaffolding protein has not been elucidated, and no linkage between HTT and methylation has yet been...

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Seervai RNH, Jangid RK, Karki M, et al. The Huntingtin-interacting protein SETD2/HYPB is an actin lysine methyltransferase. Sci Adv. 2020;6(40)doi: 10.1126/sciadv.abb7854.
Seervai, R. N. H., Jangid, R. K., Karki, M., Tripathi, D. N., Jung, S. Y., Kearns, S. E., Verhey, K. J., Cianfrocco, M. A., Millis, B. A., Tyska, M. J., Mason, F. M., Rathmell, W. K., Park, I. Y., Dere, R., & Walker, C. L. (2020). The Huntingtin-interacting protein SETD2/HYPB is an actin lysine methyltransferase. Science advances, 6(40), . https://doi.org/10.1126/sciadv.abb7854
Seervai, Riyad N H, et al. "The Huntingtin-interacting protein SETD2/HYPB is an actin lysine methyltransferase." Science advances vol. 6,40 (2020). doi: https://doi.org/10.1126/sciadv.abb7854
Seervai RNH, Jangid RK, Karki M, Tripathi DN, Jung SY, Kearns SE, Verhey KJ, Cianfrocco MA, Millis BA, Tyska MJ, Mason FM, Rathmell WK, Park IY, Dere R, Walker CL. The Huntingtin-interacting protein SETD2/HYPB is an actin lysine methyltransferase. Sci Adv. 2020 Oct 02;6(40). doi: 10.1126/sciadv.abb7854. Print 2020 Oct. PMID: 33008892; PMCID: PMC7852384.
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MicroRNA-Specificity Protein (Sp) Transcription Factor Interactions and Significance in Carcinogenesis.

Current pharmacology reports

Safe S.
PMID: 26457240
Curr Pharmacol Rep. 2015 Apr;1(2):73-78. doi: 10.1007/s40495-014-0012-8. Epub 2015 Jan 11.

Specificity protein (Sp) transcription factors (TFs) such as Sp1, Sp3 and Sp4 are overexpressed in tumors and Sp1 is a negative prognostic factor for multiple tumor types. Sp TFs regulate expression of pro-oncogenic factors important for cell proliferation, survival,...

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Safe S. MicroRNA-Specificity Protein (Sp) Transcription Factor Interactions and Significance in Carcinogenesis. Curr Pharmacol Rep. 2015;1(2):73-78doi: 10.1007/s40495-014-0012-8.
Safe, S. (2015). MicroRNA-Specificity Protein (Sp) Transcription Factor Interactions and Significance in Carcinogenesis. Current pharmacology reports, 1(2), 73-78. https://doi.org/10.1007/s40495-014-0012-8
Safe, Stephen. "MicroRNA-Specificity Protein (Sp) Transcription Factor Interactions and Significance in Carcinogenesis." Current pharmacology reports vol. 1,2 (2015): 73-78. doi: https://doi.org/10.1007/s40495-014-0012-8
Safe S. MicroRNA-Specificity Protein (Sp) Transcription Factor Interactions and Significance in Carcinogenesis. Curr Pharmacol Rep. 2015 Apr;1(2):73-78. doi: 10.1007/s40495-014-0012-8. Epub 2015 Jan 11. PMID: 26457240; PMCID: PMC4596546.
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Rapid isolation of blood plasma using a cascaded inertial microfluidic device.

Biomicrofluidics

Robinson M, Marks H, Hinsdale T, Maitland K, Coté G.
PMID: 28405258
Biomicrofluidics. 2017 Mar 24;11(2):024109. doi: 10.1063/1.4979198. eCollection 2017 Mar.

Blood, saliva, mucus, sweat, sputum, and other biological fluids are often hindered in their ability to be used in point-of-care (POC) diagnostics because their assays require some form of off-site sample pre-preparation to effectively separate biomarkers from larger components...

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Robinson M, Marks H, Hinsdale T, et al. Rapid isolation of blood plasma using a cascaded inertial microfluidic device. Biomicrofluidics. 2017;11(2):024109doi: 10.1063/1.4979198.
Robinson, M., Marks, H., Hinsdale, T., Maitland, K., & Coté, G. (2017). Rapid isolation of blood plasma using a cascaded inertial microfluidic device. Biomicrofluidics, 11(2), 024109. https://doi.org/10.1063/1.4979198
Robinson, M, et al. "Rapid isolation of blood plasma using a cascaded inertial microfluidic device." Biomicrofluidics vol. 11,2 (2017): 024109. doi: https://doi.org/10.1063/1.4979198
Robinson M, Marks H, Hinsdale T, Maitland K, Coté G. Rapid isolation of blood plasma using a cascaded inertial microfluidic device. Biomicrofluidics. 2017 Mar 24;11(2):024109. doi: 10.1063/1.4979198. eCollection 2017 Mar. PMID: 28405258; PMCID: PMC5367146.
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Nrf2 status affects tumor growth, HDAC3 gene promoter associations, and the response to sulforaphane in the colon.

Clinical epigenetics

Rajendran P, Dashwood WM, Li L, Kang Y, Kim E, Johnson G, Fischer KA, Löhr CV, Williams DE, Ho E, Yamamoto M, Lieberman DA, Dashwood RH.
PMID: 26388957
Clin Epigenetics. 2015 Sep 18;7:102. doi: 10.1186/s13148-015-0132-y. eCollection 2015.

BACKGROUND: The dietary agent sulforaphane (SFN) has been reported to induce nuclear factor erythroid 2 (NF-E2)-related factor 2 (Nrf2)-dependent pathways as well as inhibiting histone deacetylase (HDAC) activity. The current investigation sought to examine the relationships between Nrf2 status...

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Rajendran P, Dashwood WM, Li L, et al. Nrf2 status affects tumor growth, HDAC3 gene promoter associations, and the response to sulforaphane in the colon. Clin Epigenetics. 2015;7:102doi: 10.1186/s13148-015-0132-y.
Rajendran, P., Dashwood, W. M., Li, L., Kang, Y., Kim, E., Johnson, G., Fischer, K. A., Löhr, C. V., Williams, D. E., Ho, E., Yamamoto, M., Lieberman, D. A., & Dashwood, R. H. (2015). Nrf2 status affects tumor growth, HDAC3 gene promoter associations, and the response to sulforaphane in the colon. Clinical epigenetics, 7102. https://doi.org/10.1186/s13148-015-0132-y
Rajendran, Praveen, et al. "Nrf2 status affects tumor growth, HDAC3 gene promoter associations, and the response to sulforaphane in the colon." Clinical epigenetics vol. 7 (2015): 102. doi: https://doi.org/10.1186/s13148-015-0132-y
Rajendran P, Dashwood WM, Li L, Kang Y, Kim E, Johnson G, Fischer KA, Löhr CV, Williams DE, Ho E, Yamamoto M, Lieberman DA, Dashwood RH. Nrf2 status affects tumor growth, HDAC3 gene promoter associations, and the response to sulforaphane in the colon. Clin Epigenetics. 2015 Sep 18;7:102. doi: 10.1186/s13148-015-0132-y. eCollection 2015. PMID: 26388957; PMCID: PMC4575421.
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Gestational exposure to particulate air pollution exacerbates the growth phenotypes induced by preconception paternal alcohol use: a multiplex model of exposure.

Environmental epigenetics

Mustapha TA, Chang RC, Garcia-Rhodes D, Pendleton D, Johnson NM, Golding MC.
PMID: 33214907
Environ Epigenet. 2020 Sep 27;6(1):dvaa011. doi: 10.1093/eep/dvaa011. eCollection 2020.

It is now clear that parental histories of drug use, toxicant exposure, and social stress all have a significant influence on the health and development of the next generation. However, the ability of epigenetic parental life memories to interact...

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Mustapha TA, Chang RC, Garcia-Rhodes D, et al. Gestational exposure to particulate air pollution exacerbates the growth phenotypes induced by preconception paternal alcohol use: a multiplex model of exposure. Environ Epigenet. 2020;6(1):dvaa011doi: 10.1093/eep/dvaa011.
Mustapha, T. A., Chang, R. C., Garcia-Rhodes, D., Pendleton, D., Johnson, N. M., & Golding, M. C. (2020). Gestational exposure to particulate air pollution exacerbates the growth phenotypes induced by preconception paternal alcohol use: a multiplex model of exposure. Environmental epigenetics, 6(1), dvaa011. https://doi.org/10.1093/eep/dvaa011
Mustapha, Toriq A, et al. "Gestational exposure to particulate air pollution exacerbates the growth phenotypes induced by preconception paternal alcohol use: a multiplex model of exposure." Environmental epigenetics vol. 6,1 (2020): dvaa011. doi: https://doi.org/10.1093/eep/dvaa011
Mustapha TA, Chang RC, Garcia-Rhodes D, Pendleton D, Johnson NM, Golding MC. Gestational exposure to particulate air pollution exacerbates the growth phenotypes induced by preconception paternal alcohol use: a multiplex model of exposure. Environ Epigenet. 2020 Sep 27;6(1):dvaa011. doi: 10.1093/eep/dvaa011. eCollection 2020. PMID: 33214907; PMCID: PMC7660119.
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Mechanisms by Which Pleiotropic Amphiphilic .

Current colorectal cancer reports

Chapkin RS, DeClercq V, Kim E, Fuentes NR, Fan YY.
PMID: 25400530
Curr Colorectal Cancer Rep. 2014 Dec 01;10(4):442-452. doi: 10.1007/s11888-014-0241-6.

Colorectal cancer is one of the major causes of cancer-related mortality in both men and women worldwide. Genetic susceptibility and diet are primary determinants of cancer risk and tumor behavior. Experimental, epidemiological, and clinical data substantiate the beneficial role...

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Chapkin RS, DeClercq V, Kim E, et al. Mechanisms by Which Pleiotropic Amphiphilic . Curr Colorectal Cancer Rep. 2014;10(4):442-452doi: 10.1007/s11888-014-0241-6.
Chapkin, R. S., DeClercq, V., Kim, E., Fuentes, N. R., & Fan, Y. Y. (2014). Mechanisms by Which Pleiotropic Amphiphilic . Current colorectal cancer reports, 10(4), 442-452. https://doi.org/10.1007/s11888-014-0241-6
Chapkin, Robert S, et al. "Mechanisms by Which Pleiotropic Amphiphilic ." Current colorectal cancer reports vol. 10,4 (2014): 442-452. doi: https://doi.org/10.1007/s11888-014-0241-6
Chapkin RS, DeClercq V, Kim E, Fuentes NR, Fan YY. Mechanisms by Which Pleiotropic Amphiphilic . Curr Colorectal Cancer Rep. 2014 Dec 01;10(4):442-452. doi: 10.1007/s11888-014-0241-6. PMID: 25400530; PMCID: PMC4228477.
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Adenosine promotes vascular barrier function in hyperoxic lung injury.

Physiological reports

Davies J, Karmouty-Quintana H, Le TT, Chen NY, Weng T, Luo F, Molina J, Moorthy B, Blackburn MR.
PMID: 25263205
Physiol Rep. 2014 Sep 28;2(9). doi: 10.14814/phy2.12155. Print 2014 Sep 01.

Hyperoxic lung injury is characterized by cellular damage from high oxygen concentrations that lead to an inflammatory response in the lung with cellular infiltration and pulmonary edema. Adenosine is a signaling molecule that is generated extracellularly by CD73 in...

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Davies J, Karmouty-Quintana H, Le TT, et al. Adenosine promotes vascular barrier function in hyperoxic lung injury. Physiol Rep. 2014;2(9)doi: 10.14814/phy2.12155.
Davies, J., Karmouty-Quintana, H., Le, T. T., Chen, N. Y., Weng, T., Luo, F., Molina, J., Moorthy, B., & Blackburn, M. R. (2014). Adenosine promotes vascular barrier function in hyperoxic lung injury. Physiological reports, 2(9), . https://doi.org/10.14814/phy2.12155
Davies, Jonathan, et al. "Adenosine promotes vascular barrier function in hyperoxic lung injury." Physiological reports vol. 2,9 (2014). doi: https://doi.org/10.14814/phy2.12155
Davies J, Karmouty-Quintana H, Le TT, Chen NY, Weng T, Luo F, Molina J, Moorthy B, Blackburn MR. Adenosine promotes vascular barrier function in hyperoxic lung injury. Physiol Rep. 2014 Sep 28;2(9). doi: 10.14814/phy2.12155. Print 2014 Sep 01. PMID: 25263205; PMCID: PMC4270235.
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Showing 1 to 12 of 16 entries