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A Protective Role for NKG2D-H60a Interaction via Homotypic T Cell Contact in Nonobese Diabetic Autoimmune Diabetes Pathogenesis.

ImmunoHorizons

Trembath AP, Sharma N, Raju S, Polić B, Markiewicz MA.
PMID: 29497709
Immunohorizons. 2017 Nov 01;1(9):198-212. doi: 10.4049/immunohorizons.1700011. Epub 2017 Nov 07.

The NK group 2 member D (NKG2D) immune receptor is implicated in both human and mouse autoimmune diabetes. However, the significance of NKG2D in diabetes pathogenesis has been unclear due to conflicting reports as to the importance of this...

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Trembath AP, Sharma N, Raju S, et al. A Protective Role for NKG2D-H60a Interaction via Homotypic T Cell Contact in Nonobese Diabetic Autoimmune Diabetes Pathogenesis. Immunohorizons. 2017;1(9):198-212doi: 10.4049/immunohorizons.1700011.
Trembath, A. P., Sharma, N., Raju, S., Polić, B., & Markiewicz, M. A. (2017). A Protective Role for NKG2D-H60a Interaction via Homotypic T Cell Contact in Nonobese Diabetic Autoimmune Diabetes Pathogenesis. ImmunoHorizons, 1(9), 198-212. https://doi.org/10.4049/immunohorizons.1700011
Trembath, Andrew P, et al. "A Protective Role for NKG2D-H60a Interaction via Homotypic T Cell Contact in Nonobese Diabetic Autoimmune Diabetes Pathogenesis." ImmunoHorizons vol. 1,9 (2017): 198-212. doi: https://doi.org/10.4049/immunohorizons.1700011
Trembath AP, Sharma N, Raju S, Polić B, Markiewicz MA. A Protective Role for NKG2D-H60a Interaction via Homotypic T Cell Contact in Nonobese Diabetic Autoimmune Diabetes Pathogenesis. Immunohorizons. 2017 Nov 01;1(9):198-212. doi: 10.4049/immunohorizons.1700011. Epub 2017 Nov 07. PMID: 29497709; PMCID: PMC5828234.
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The Role of the Ikaros Family of Transcription Factors in Regulatory T cell Development and Function.

Journal of clinical & cellular immunology

Seng A, Yankee TM.
PMID: 28758047
J Clin Cell Immunol. 2017 Apr;8(2). doi: 10.4172/2155-9899.1000495. Epub 2017 Mar 22.

Regulatory T (Treg) cells are a subset of immune cells that maintain homeostasis by promoting immune tolerance and suppressing the immune response via a variety of mechanisms such as secreting cytokines, killing reactive immune cells, and inducing anergy. Dysfunction...

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Seng A, Yankee TM. The Role of the Ikaros Family of Transcription Factors in Regulatory T cell Development and Function. J Clin Cell Immunol. 2017;8(2)doi: 10.4172/2155-9899.1000495.
Seng, A., & Yankee, T. M. (2017). The Role of the Ikaros Family of Transcription Factors in Regulatory T cell Development and Function. Journal of clinical & cellular immunology, 8(2), . https://doi.org/10.4172/2155-9899.1000495
Seng, Amara, and Yankee, Thomas M. "The Role of the Ikaros Family of Transcription Factors in Regulatory T cell Development and Function." Journal of clinical & cellular immunology vol. 8,2 (2017). doi: https://doi.org/10.4172/2155-9899.1000495
Seng A, Yankee TM. The Role of the Ikaros Family of Transcription Factors in Regulatory T cell Development and Function. J Clin Cell Immunol. 2017 Apr;8(2). doi: 10.4172/2155-9899.1000495. Epub 2017 Mar 22. PMID: 28758047; PMCID: PMC5531196.
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Variations in mRNA and protein levels of Ikaros family members in pediatric T cell acute lymphoblastic leukemia.

Annals of translational medicine

Mitchell JL, Yankee TM.
PMID: 27826566
Ann Transl Med. 2016 Oct;4(19):363. doi: 10.21037/atm.2016.09.29.

BACKGROUND: Pediatric T cell acute lymphoblastic leukemia (T-ALL) is a highly heterogeneous disease in which the cells share phenotypic characteristics with normal human thymocytes. The Ikaros family of transcription factors includes five members that are required for normal T...

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Mitchell JL, Yankee TM. Variations in mRNA and protein levels of Ikaros family members in pediatric T cell acute lymphoblastic leukemia. Ann Transl Med. 2016;4(19):363doi: 10.21037/atm.2016.09.29.
Mitchell, J. L., & Yankee, T. M. (2016). Variations in mRNA and protein levels of Ikaros family members in pediatric T cell acute lymphoblastic leukemia. Annals of translational medicine, 4(19), 363. https://doi.org/10.21037/atm.2016.09.29
Mitchell, Julie L, and Yankee, Thomas M. "Variations in mRNA and protein levels of Ikaros family members in pediatric T cell acute lymphoblastic leukemia." Annals of translational medicine vol. 4,19 (2016): 363. doi: https://doi.org/10.21037/atm.2016.09.29
Mitchell JL, Yankee TM. Variations in mRNA and protein levels of Ikaros family members in pediatric T cell acute lymphoblastic leukemia. Ann Transl Med. 2016 Oct;4(19):363. doi: 10.21037/atm.2016.09.29. PMID: 27826566; PMCID: PMC5075847.
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Aurora A kinase regulates non-homologous end-joining and poly(ADP-ribose) polymerase function in ovarian carcinoma cells.

Oncotarget

Do TV, Hirst J, Hyter S, Roby KF, Godwin AK.
PMID: 28881569
Oncotarget. 2017 Jul 05;8(31):50376-50392. doi: 10.18632/oncotarget.18970. eCollection 2017 Aug 01.

Ovarian cancer is usually diagnosed at late stages when cancer has spread beyond the ovary and patients ultimately succumb to the development of drug-resistant disease. There is an urgent and unmet need to develop therapeutic strategies that effectively treat...

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Do TV, Hirst J, Hyter S, et al. Aurora A kinase regulates non-homologous end-joining and poly(ADP-ribose) polymerase function in ovarian carcinoma cells. Oncotarget. 2017;8(31):50376-50392doi: 10.18632/oncotarget.18970.
Do, T. V., Hirst, J., Hyter, S., Roby, K. F., & Godwin, A. K. (2017). Aurora A kinase regulates non-homologous end-joining and poly(ADP-ribose) polymerase function in ovarian carcinoma cells. Oncotarget, 8(31), 50376-50392. https://doi.org/10.18632/oncotarget.18970
Do, Thuy-Vy, et al. "Aurora A kinase regulates non-homologous end-joining and poly(ADP-ribose) polymerase function in ovarian carcinoma cells." Oncotarget vol. 8,31 (2017): 50376-50392. doi: https://doi.org/10.18632/oncotarget.18970
Do TV, Hirst J, Hyter S, Roby KF, Godwin AK. Aurora A kinase regulates non-homologous end-joining and poly(ADP-ribose) polymerase function in ovarian carcinoma cells. Oncotarget. 2017 Jul 05;8(31):50376-50392. doi: 10.18632/oncotarget.18970. eCollection 2017 Aug 01. PMID: 28881569; PMCID: PMC5584138.
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3-Substituted Biquinolinium Inhibitors of AraC Family Transcriptional Activator VirF from .

RSC advances

Jain P, Li J, Porubsky P, Neuenswander B, Egan SM, Aubé J, Rogers S.
PMID: 25258678
RSC Adv. 2014 Jan 01;4(75):39809-39816. doi: 10.1039/C4RA08384A.

During a structure-activity relationship optimization campaign to develop an inhibitor of AraC family transcriptional activators, we discovered an unexpected transformation of a previously reported inhibitor that occurs under the assay conditions. Once placed in the assay media, the 3,...

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Jain P, Li J, Porubsky P, et al. 3-Substituted Biquinolinium Inhibitors of AraC Family Transcriptional Activator VirF from . RSC Adv. 2014;4(75):39809-39816doi: 10.1039/C4RA08384A.
Jain, P., Li, J., Porubsky, P., Neuenswander, B., Egan, S. M., Aubé, J., & Rogers, S. (2014). 3-Substituted Biquinolinium Inhibitors of AraC Family Transcriptional Activator VirF from . RSC advances, 4(75), 39809-39816. https://doi.org/10.1039/C4RA08384A
Jain, Prashi, et al. "3-Substituted Biquinolinium Inhibitors of AraC Family Transcriptional Activator VirF from ." RSC advances vol. 4,75 (2014): 39809-39816. doi: https://doi.org/10.1039/C4RA08384A
Jain P, Li J, Porubsky P, Neuenswander B, Egan SM, Aubé J, Rogers S. 3-Substituted Biquinolinium Inhibitors of AraC Family Transcriptional Activator VirF from . RSC Adv. 2014 Jan 01;4(75):39809-39816. doi: 10.1039/C4RA08384A. PMID: 25258678; PMCID: PMC4170738.
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Progesterone promotes immunomodulation and tumor development in the murine mammary gland.

Journal for immunotherapy of cancer

Werner LR, Gibson KA, Goodman ML, Helm DE, Walter KR, Holloran SM, Trinca GM, Hastings RC, Yang HH, Hu Y, Wei J, Lei G, Yang XY, Madan R, Molinolo AA, Markiewicz MA, Chalise P, Axelrod ML, Balko JM, Hunter KW, Hartman ZC, Lange CA, Hagan CR.
PMID: 33958486
J Immunother Cancer. 2021 May;9(5). doi: 10.1136/jitc-2020-001710.

BACKGROUND: Clinical studies have linked usage of progestins (synthetic progesterone [P4]) to breast cancer risk. However, little is understood regarding the role of native P4, signaling through the progesterone receptor (PR), in breast tumor formation. Recently, we reported a...

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Werner LR, Gibson KA, Goodman ML, et al. Progesterone promotes immunomodulation and tumor development in the murine mammary gland. J Immunother Cancer. 2021;9(5)doi: 10.1136/jitc-2020-001710.
Werner, L. R., Gibson, K. A., Goodman, M. L., Helm, D. E., Walter, K. R., Holloran, S. M., Trinca, G. M., Hastings, R. C., Yang, H. H., Hu, Y., Wei, J., Lei, G., Yang, X. Y., Madan, R., Molinolo, A. A., Markiewicz, M. A., Chalise, P., Axelrod, M. L., Balko, J. M., Hunter, K. W., Hartman, Z. C., Lange, C. A., & Hagan, C. R. (2021). Progesterone promotes immunomodulation and tumor development in the murine mammary gland. Journal for immunotherapy of cancer, 9(5), . https://doi.org/10.1136/jitc-2020-001710
Werner, Lauryn R, et al. "Progesterone promotes immunomodulation and tumor development in the murine mammary gland." Journal for immunotherapy of cancer vol. 9,5 (2021). doi: https://doi.org/10.1136/jitc-2020-001710
Werner LR, Gibson KA, Goodman ML, Helm DE, Walter KR, Holloran SM, Trinca GM, Hastings RC, Yang HH, Hu Y, Wei J, Lei G, Yang XY, Madan R, Molinolo AA, Markiewicz MA, Chalise P, Axelrod ML, Balko JM, Hunter KW, Hartman ZC, Lange CA, Hagan CR. Progesterone promotes immunomodulation and tumor development in the murine mammary gland. J Immunother Cancer. 2021 May;9(5). doi: 10.1136/jitc-2020-001710. PMID: 33958486; PMCID: PMC8103939.
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A Phospho-SIM in the Antiviral Protein PML is Required for Its Recruitment to HSV-1 Genomes.

Cells

Smith MC, Box AC, Haug JS, Lane WS, Davido DJ.
PMID: 25513827
Cells. 2014 Dec 10;3(4):1131-58. doi: 10.3390/cells3041131.

Herpes simplex virus type 1 (HSV-1) is a significant human pathogen that infects a large portion of the human population. Cells deploy a variety of defenses to limit the extent to which the virus can replicate. One such factor...

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Smith MC, Box AC, Haug JS, et al. A Phospho-SIM in the Antiviral Protein PML is Required for Its Recruitment to HSV-1 Genomes. Cells. 2014;3(4):1131-58doi: 10.3390/cells3041131.
Smith, M. C., Box, A. C., Haug, J. S., Lane, W. S., & Davido, D. J. (2014). A Phospho-SIM in the Antiviral Protein PML is Required for Its Recruitment to HSV-1 Genomes. Cells, 3(4), 1131-58. https://doi.org/10.3390/cells3041131
Smith, Miles C, et al. "A Phospho-SIM in the Antiviral Protein PML is Required for Its Recruitment to HSV-1 Genomes." Cells vol. 3,4 (2014): 1131-58. doi: https://doi.org/10.3390/cells3041131
Smith MC, Box AC, Haug JS, Lane WS, Davido DJ. A Phospho-SIM in the Antiviral Protein PML is Required for Its Recruitment to HSV-1 Genomes. Cells. 2014 Dec 10;3(4):1131-58. doi: 10.3390/cells3041131. PMID: 25513827; PMCID: PMC4276917.
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Effect of the p53 P72R Polymorphism on Mutant TP53 Allele Selection in Human Cancer.

Journal of the National Cancer Institute

De Souza C, Madden J, Koestler DC, Minn D, Montoya DJ, Minn K, Raetz AG, Zhu Z, Xiao WW, Tahmassebi N, Reddy H, Nelson N, Karnezis AN, Chien J.
PMID: 33555293
J Natl Cancer Inst. 2021 Sep 04;113(9):1246-1257. doi: 10.1093/jnci/djab019.

BACKGROUND: TP53 mutations occur in more than 50% of cancers. We sought to determine the effect of the intragenic P72R single nucleotide polymorphism (SNP; rs1042522) on the oncogenic properties of mutant p53.METHODS: P72R allelic selection in tumors was determined...

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De Souza C, Madden J, Koestler DC, et al. Effect of the p53 P72R Polymorphism on Mutant TP53 Allele Selection in Human Cancer. J Natl Cancer Inst. 2021;113(9):1246-1257doi: 10.1093/jnci/djab019.
De Souza, C., Madden, J., Koestler, D. C., Minn, D., Montoya, D. J., Minn, K., Raetz, A. G., Zhu, Z., Xiao, W. W., Tahmassebi, N., Reddy, H., Nelson, N., Karnezis, A. N., & Chien, J. (2021). Effect of the p53 P72R Polymorphism on Mutant TP53 Allele Selection in Human Cancer. Journal of the National Cancer Institute, 113(9), 1246-1257. https://doi.org/10.1093/jnci/djab019
De Souza, Cristabelle, et al. "Effect of the p53 P72R Polymorphism on Mutant TP53 Allele Selection in Human Cancer." Journal of the National Cancer Institute vol. 113,9 (2021): 1246-1257. doi: https://doi.org/10.1093/jnci/djab019
De Souza C, Madden J, Koestler DC, Minn D, Montoya DJ, Minn K, Raetz AG, Zhu Z, Xiao WW, Tahmassebi N, Reddy H, Nelson N, Karnezis AN, Chien J. Effect of the p53 P72R Polymorphism on Mutant TP53 Allele Selection in Human Cancer. J Natl Cancer Inst. 2021 Sep 04;113(9):1246-1257. doi: 10.1093/jnci/djab019. PMID: 33555293; PMCID: PMC8633460.
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Generation of a Stable Plasmid for .

Applied and environmental microbiology

Krute CN, Krausz KL, Markiewicz MA, Joyner JA, Pokhrel S, Hall PR, Bose JL.
PMID: 27637878
Appl Environ Microbiol. 2016 Dec 01;82(23):6859-6869. doi: 10.1128/AEM.02370-16. Epub 2016 Sep 16.

A major shortcoming to plasmid-based genetic tools is the necessity of using antibiotics to ensure plasmid maintenance. While selectable markers are very powerful, their use is not always practical, such as during

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Krute CN, Krausz KL, Markiewicz MA, et al. Generation of a Stable Plasmid for . Appl Environ Microbiol. 2016;82(23):6859-6869doi: 10.1128/AEM.02370-16.
Krute, C. N., Krausz, K. L., Markiewicz, M. A., Joyner, J. A., Pokhrel, S., Hall, P. R., & Bose, J. L. (2016). Generation of a Stable Plasmid for . Applied and environmental microbiology, 82(23), 6859-6869. https://doi.org/10.1128/AEM.02370-16
Krute, Christina N, et al. "Generation of a Stable Plasmid for ." Applied and environmental microbiology vol. 82,23 (2016): 6859-6869. doi: https://doi.org/10.1128/AEM.02370-16
Krute CN, Krausz KL, Markiewicz MA, Joyner JA, Pokhrel S, Hall PR, Bose JL. Generation of a Stable Plasmid for . Appl Environ Microbiol. 2016 Dec 01;82(23):6859-6869. doi: 10.1128/AEM.02370-16. Epub 2016 Sep 16. PMID: 27637878; PMCID: PMC5103085.
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Cisplatin and Pemetrexed Activate AXL and AXL Inhibitor BGB324 Enhances Mesothelioma Cell Death from Chemotherapy.

Frontiers in pharmacology

Oien DB, Garay T, Eckstein S, Chien J.
PMID: 29375377
Front Pharmacol. 2018 Jan 11;8:970. doi: 10.3389/fphar.2017.00970. eCollection 2017.

Reactive oxygen species (ROS) can promote or inhibit tumorigenesis. In mesothelioma, asbestos exposure to serous membranes induces ROS through iron content and chronic inflammation, and ROS promote cell survival signaling in mesothelioma. Moreover, a current chemotherapy regimen for mesothelioma...

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Oien DB, Garay T, Eckstein S, et al. Cisplatin and Pemetrexed Activate AXL and AXL Inhibitor BGB324 Enhances Mesothelioma Cell Death from Chemotherapy. Front Pharmacol. 2018;8:970doi: 10.3389/fphar.2017.00970.
Oien, D. B., Garay, T., Eckstein, S., & Chien, J. (2017). Cisplatin and Pemetrexed Activate AXL and AXL Inhibitor BGB324 Enhances Mesothelioma Cell Death from Chemotherapy. Frontiers in pharmacology, 8970. https://doi.org/10.3389/fphar.2017.00970
Oien, Derek B, et al. "Cisplatin and Pemetrexed Activate AXL and AXL Inhibitor BGB324 Enhances Mesothelioma Cell Death from Chemotherapy." Frontiers in pharmacology vol. 8 (2017): 970. doi: https://doi.org/10.3389/fphar.2017.00970
Oien DB, Garay T, Eckstein S, Chien J. Cisplatin and Pemetrexed Activate AXL and AXL Inhibitor BGB324 Enhances Mesothelioma Cell Death from Chemotherapy. Front Pharmacol. 2018 Jan 11;8:970. doi: 10.3389/fphar.2017.00970. eCollection 2017. PMID: 29375377; PMCID: PMC5768913.
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Ouabain Enhances ADPKD Cell Apoptosis via the Intrinsic Pathway.

Frontiers in physiology

Venugopal J, Blanco G.
PMID: 27047392
Front Physiol. 2016 Mar 24;7:107. doi: 10.3389/fphys.2016.00107. eCollection 2016.

Progression of autosomal dominant polycystic kidney disease (ADPKD) is highly influenced by factors circulating in blood. We have shown that the hormone ouabain enhances several characteristics of the ADPKD cystic phenotype, including the rate of cell proliferation, fluid secretion...

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Venugopal J, Blanco G. Ouabain Enhances ADPKD Cell Apoptosis via the Intrinsic Pathway. Front Physiol. 2016;7:107doi: 10.3389/fphys.2016.00107.
Venugopal, J., & Blanco, G. (2016). Ouabain Enhances ADPKD Cell Apoptosis via the Intrinsic Pathway. Frontiers in physiology, 7107. https://doi.org/10.3389/fphys.2016.00107
Venugopal, Jessica, and Blanco, Gustavo. "Ouabain Enhances ADPKD Cell Apoptosis via the Intrinsic Pathway." Frontiers in physiology vol. 7 (2016): 107. doi: https://doi.org/10.3389/fphys.2016.00107
Venugopal J, Blanco G. Ouabain Enhances ADPKD Cell Apoptosis via the Intrinsic Pathway. Front Physiol. 2016 Mar 24;7:107. doi: 10.3389/fphys.2016.00107. eCollection 2016. PMID: 27047392; PMCID: PMC4805603.
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