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Neural processing of facial identity and emotion in infants at high-risk for autism spectrum disorders.

Frontiers in human neuroscience

Fox SE, Wagner JB, Shrock CL, Tager-Flusberg H, Nelson CA.
PMID: 23576966
Front Hum Neurosci. 2013 Apr 09;7:89. doi: 10.3389/fnhum.2013.00089. eCollection 2013.

Deficits in face processing and social impairment are core characteristics of autism spectrum disorder. The present work examined 7-month-old infants at high-risk for developing autism and typically developing controls at low-risk, using a face perception task designed to differentiate...

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Fox SE, Wagner JB, Shrock CL, et al. Neural processing of facial identity and emotion in infants at high-risk for autism spectrum disorders. Front Hum Neurosci. 2013;7:89doi: 10.3389/fnhum.2013.00089.
Fox, S. E., Wagner, J. B., Shrock, C. L., Tager-Flusberg, H., & Nelson, C. A. (2013). Neural processing of facial identity and emotion in infants at high-risk for autism spectrum disorders. Frontiers in human neuroscience, 789. https://doi.org/10.3389/fnhum.2013.00089
Fox, Sharon E, et al. "Neural processing of facial identity and emotion in infants at high-risk for autism spectrum disorders." Frontiers in human neuroscience vol. 7 (2013): 89. doi: https://doi.org/10.3389/fnhum.2013.00089
Fox SE, Wagner JB, Shrock CL, Tager-Flusberg H, Nelson CA. Neural processing of facial identity and emotion in infants at high-risk for autism spectrum disorders. Front Hum Neurosci. 2013 Apr 09;7:89. doi: 10.3389/fnhum.2013.00089. eCollection 2013. PMID: 23576966; PMCID: PMC3620489.
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The Newborn Individualized Developmental Care and Assessment Program (NIDCAP) with Kangaroo Mother Care (KMC): Comprehensive Care for Preterm Infants.

Current women's health reviews

Als H, McAnulty GB.
PMID: 25473384
Curr Womens Health Rev. 2011 Aug;7(3):288-301. doi: 10.2174/157340411796355216.

State-of-the-art Newborn Intensive Care Units (NICUs), instrumental in the survival of high-risk and ever-earlier-born preterm infants, often have costly human repercussions. The developmental sequelae of newborn intensive care are largely misunderstood. Developed countries eager to export their technologies must...

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Als H, McAnulty GB. The Newborn Individualized Developmental Care and Assessment Program (NIDCAP) with Kangaroo Mother Care (KMC): Comprehensive Care for Preterm Infants. Curr Womens Health Rev. 2011;7(3):288-301doi: 10.2174/157340411796355216.
Als, H., & McAnulty, G. B. (2011). The Newborn Individualized Developmental Care and Assessment Program (NIDCAP) with Kangaroo Mother Care (KMC): Comprehensive Care for Preterm Infants. Current women's health reviews, 7(3), 288-301. https://doi.org/10.2174/157340411796355216
Als, Heidelise, and McAnulty, Gloria B. "The Newborn Individualized Developmental Care and Assessment Program (NIDCAP) with Kangaroo Mother Care (KMC): Comprehensive Care for Preterm Infants." Current women's health reviews vol. 7,3 (2011): 288-301. doi: https://doi.org/10.2174/157340411796355216
Als H, McAnulty GB. The Newborn Individualized Developmental Care and Assessment Program (NIDCAP) with Kangaroo Mother Care (KMC): Comprehensive Care for Preterm Infants. Curr Womens Health Rev. 2011 Aug;7(3):288-301. doi: 10.2174/157340411796355216. PMID: 25473384; PMCID: PMC4248304.
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A novel gene required for male fertility and functional CATSPER channel formation in spermatozoa.

Nature communications

Chung JJ, Navarro B, Krapivinsky G, Krapivinsky L, Clapham DE.
PMID: 21224844
Nat Commun. 2011 Jan 11;2:153. doi: 10.1038/ncomms1153.

Calcium signalling is critical for successful fertilization. In spermatozoa, capacitation, hyperactivation of motility and the acrosome reaction are all mediated by increases in intracellular Ca(2+). Cation channels of sperm proteins (CATSPERS1-4) form an alkalinization-activated Ca(2+)-selective channel required for the...

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Chung JJ, Navarro B, Krapivinsky G, et al. A novel gene required for male fertility and functional CATSPER channel formation in spermatozoa. Nat Commun. 2011;2:153doi: 10.1038/ncomms1153.
Chung, J. J., Navarro, B., Krapivinsky, G., Krapivinsky, L., & Clapham, D. E. (2011). A novel gene required for male fertility and functional CATSPER channel formation in spermatozoa. Nature communications, 2153. https://doi.org/10.1038/ncomms1153
Chung, Jean-Ju, et al. "A novel gene required for male fertility and functional CATSPER channel formation in spermatozoa." Nature communications vol. 2 (2011): 153. doi: https://doi.org/10.1038/ncomms1153
Chung JJ, Navarro B, Krapivinsky G, Krapivinsky L, Clapham DE. A novel gene required for male fertility and functional CATSPER channel formation in spermatozoa. Nat Commun. 2011 Jan 11;2:153. doi: 10.1038/ncomms1153. PMID: 21224844; PMCID: PMC3999383.
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Developmental factors in the pathogenesis of neonatal seizures.

Journal of pediatric neurology : JPN

Jensen FE.
PMID: 20191097
J Pediatr Neurol. 2009 Jan 01;7(1):5-12. doi: 10.3233/JPN-2009-0270.

Neonatal seizures are inherently different from seizures in the child and the adult. The phenotype, often exhibiting electroclinical dissociation, is unique: neonatal seizures can be refractory to antiepileptic drugs otherwise effect for older patients. Recent experimental and human-based research...

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Jensen FE. Developmental factors in the pathogenesis of neonatal seizures. J Pediatr Neurol. 2009;7(1):5-12doi: 10.3233/JPN-2009-0270.
Jensen, F. E. (2009). Developmental factors in the pathogenesis of neonatal seizures. Journal of pediatric neurology : JPN, 7(1), 5-12. https://doi.org/10.3233/JPN-2009-0270
Jensen, Frances E. "Developmental factors in the pathogenesis of neonatal seizures." Journal of pediatric neurology : JPN vol. 7,1 (2009): 5-12. doi: https://doi.org/10.3233/JPN-2009-0270
Jensen FE. Developmental factors in the pathogenesis of neonatal seizures. J Pediatr Neurol. 2009 Jan 01;7(1):5-12. doi: 10.3233/JPN-2009-0270. PMID: 20191097; PMCID: PMC2828632.
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RIP1 kinase mediates arachidonic acid-induced oxidative death of oligodendrocyte precursors.

International journal of physiology, pathophysiology and pharmacology

Kim S, Dayani L, Rosenberg PA, Li J.
PMID: 20706550
Int J Physiol Pathophysiol Pharmacol. 2010;2(2):137-47. Epub 2010 Jun 12.

Oxidative damage is implicated in many neurological disorders including ischemic cerebral white matter injury. Oligodendrocyte precursors (preOLs) are intrinsically highly susceptible to various forms of oxidative stress. Here we report the identification of RIP1 kinase as a signaling molecule...

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Kim S, Dayani L, Rosenberg PA, et al. RIP1 kinase mediates arachidonic acid-induced oxidative death of oligodendrocyte precursors. Int J Physiol Pathophysiol Pharmacol. 2010;2(2):137-47
Kim, S., Dayani, L., Rosenberg, P. A., & Li, J. (2010). RIP1 kinase mediates arachidonic acid-induced oxidative death of oligodendrocyte precursors. International journal of physiology, pathophysiology and pharmacology, 2(2), 137-47.
Kim, Sunja, et al. "RIP1 kinase mediates arachidonic acid-induced oxidative death of oligodendrocyte precursors." International journal of physiology, pathophysiology and pharmacology vol. 2,2 (2010): 137-47.
Kim S, Dayani L, Rosenberg PA, Li J. RIP1 kinase mediates arachidonic acid-induced oxidative death of oligodendrocyte precursors. Int J Physiol Pathophysiol Pharmacol. 2010;2(2):137-47. Epub 2010 Jun 12. PMID: 20706550; PMCID: PMC2919773.
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Systems approach to the study of brain damage in the very preterm newborn.

Frontiers in systems neuroscience

Leviton A, Gressens P, Wolkenhauer O, Dammann O.
PMID: 25926780
Front Syst Neurosci. 2015 Apr 14;9:58. doi: 10.3389/fnsys.2015.00058. eCollection 2015.

BACKGROUND: A systems approach to the study of brain damage in very preterm newborns has been lacking.METHODS: In this perspective piece, we offer encephalopathy of prematurity as an example of the complexity and interrelatedness of brain-damaging molecular processes that...

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Leviton A, Gressens P, Wolkenhauer O, et al. Systems approach to the study of brain damage in the very preterm newborn. Front Syst Neurosci. 2015;9:58doi: 10.3389/fnsys.2015.00058.
Leviton, A., Gressens, P., Wolkenhauer, O., & Dammann, O. (2015). Systems approach to the study of brain damage in the very preterm newborn. Frontiers in systems neuroscience, 958. https://doi.org/10.3389/fnsys.2015.00058
Leviton, Alan, et al. "Systems approach to the study of brain damage in the very preterm newborn." Frontiers in systems neuroscience vol. 9 (2015): 58. doi: https://doi.org/10.3389/fnsys.2015.00058
Leviton A, Gressens P, Wolkenhauer O, Dammann O. Systems approach to the study of brain damage in the very preterm newborn. Front Syst Neurosci. 2015 Apr 14;9:58. doi: 10.3389/fnsys.2015.00058. eCollection 2015. PMID: 25926780; PMCID: PMC4396381.
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Neuropathic pain drives anxiety behavior in mice, results consistent with anxiety levels in diabetic neuropathy patients.

Pain reports

Sieberg CB, Taras C, Gomaa A, Nickerson C, Wong C, Ward C, Baskozos G, Bennett DLH, Ramirez JD, Themistocleous AC, Rice ASC, Shillo PR, Tesfaye S, Edwards RR, Andrews NA, Berde C, Costigan M.
PMID: 29922743
Pain Rep. 2018 May 24;3(3):e651. doi: 10.1097/PR9.0000000000000651. eCollection 2018 May.

BACKGROUND: Epidemiological studies in patients with neuropathic pain demonstrate a strong association with psychiatric conditions such as anxiety; however, the precipitating pathology between these symptoms remains unclear. To investigate this, we studied the effects of lifelong stress on levels...

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Sieberg CB, Taras C, Gomaa A, et al. Neuropathic pain drives anxiety behavior in mice, results consistent with anxiety levels in diabetic neuropathy patients. Pain Rep. 2018;3(3):e651doi: 10.1097/PR9.0000000000000651.
Sieberg, C. B., Taras, C., Gomaa, A., Nickerson, C., Wong, C., Ward, C., Baskozos, G., Bennett, D. L. H., Ramirez, J. D., Themistocleous, A. C., Rice, A. S. C., Shillo, P. R., Tesfaye, S., Edwards, R. R., Andrews, N. A., Berde, C., & Costigan, M. (2018). Neuropathic pain drives anxiety behavior in mice, results consistent with anxiety levels in diabetic neuropathy patients. Pain reports, 3(3), e651. https://doi.org/10.1097/PR9.0000000000000651
Sieberg, Christine B, et al. "Neuropathic pain drives anxiety behavior in mice, results consistent with anxiety levels in diabetic neuropathy patients." Pain reports vol. 3,3 (2018): e651. doi: https://doi.org/10.1097/PR9.0000000000000651
Sieberg CB, Taras C, Gomaa A, Nickerson C, Wong C, Ward C, Baskozos G, Bennett DLH, Ramirez JD, Themistocleous AC, Rice ASC, Shillo PR, Tesfaye S, Edwards RR, Andrews NA, Berde C, Costigan M. Neuropathic pain drives anxiety behavior in mice, results consistent with anxiety levels in diabetic neuropathy patients. Pain Rep. 2018 May 24;3(3):e651. doi: 10.1097/PR9.0000000000000651. eCollection 2018 May. PMID: 29922743; PMCID: PMC5999418.
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Understanding positive child health.

Pediatric research

Bangma JT, Kwiatkowski E, Psioda M, Santos HP, Hooper SR, Douglass L, Joseph RM, Frazier JA, Kuban KCK, O'Shea TM, Fry RC.
PMID: 31521061
Pediatr Res. 2019 Dec;86(6):690-691. doi: 10.1038/s41390-019-0564-8. Epub 2019 Sep 14.

No abstract available.

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Bangma JT, Kwiatkowski E, Psioda M, et al. Understanding positive child health. Pediatr Res. 2019;86(6):690-691doi: 10.1038/s41390-019-0564-8.
Bangma, J. T., Kwiatkowski, E., Psioda, M., Santos, H. P., Hooper, S. R., Douglass, L., Joseph, R. M., Frazier, J. A., Kuban, K. C. K., O'Shea, T. M., Fry, R. C. (2019). Understanding positive child health. Pediatric research, 86(6), 690-691. https://doi.org/10.1038/s41390-019-0564-8
Bangma, Jacqueline T, et al. "Understanding positive child health." Pediatric research vol. 86,6 (2019): 690-691. doi: https://doi.org/10.1038/s41390-019-0564-8
Bangma JT, Kwiatkowski E, Psioda M, Santos HP, Hooper SR, Douglass L, Joseph RM, Frazier JA, Kuban KCK, O'Shea TM, Fry RC. Understanding positive child health. Pediatr Res. 2019 Dec;86(6):690-691. doi: 10.1038/s41390-019-0564-8. Epub 2019 Sep 14. PMID: 31521061; PMCID: PMC7137612.
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Sensory transduction is required for normal development and maturation of cochlear inner hair cell synapses.

eLife

Lee J, Kawai K, Holt JR, Géléoc GS.
PMID: 34734805
Elife. 2021 Nov 04;10. doi: 10.7554/eLife.69433.

Acoustic overexposure and aging can damage auditory synapses in the inner ear by a process known as synaptopathy. These insults may also damage hair bundles and the sensory transduction apparatus in auditory hair cells. However, a connection between sensory...

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Lee J, Kawai K, Holt JR, et al. Sensory transduction is required for normal development and maturation of cochlear inner hair cell synapses. Elife. 2021;10doi: 10.7554/eLife.69433.
Lee, J., Kawai, K., Holt, J. R., & Géléoc, G. S. (2021). Sensory transduction is required for normal development and maturation of cochlear inner hair cell synapses. eLife, 10. https://doi.org/10.7554/eLife.69433
Lee, John, et al. "Sensory transduction is required for normal development and maturation of cochlear inner hair cell synapses." eLife vol. 10 (2021). doi: https://doi.org/10.7554/eLife.69433
Lee J, Kawai K, Holt JR, Géléoc GS. Sensory transduction is required for normal development and maturation of cochlear inner hair cell synapses. Elife. 2021 Nov 04;10. doi: 10.7554/eLife.69433. PMID: 34734805; PMCID: PMC8598158.
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H-NS-like proteins in Pseudomonas aeruginosa coordinately silence intragenic transcription.

Molecular microbiology

Lippa AM, Gebhardt MJ, Dove SL.
PMID: 33245158
Mol Microbiol. 2021 Jun;115(6):1138-1151. doi: 10.1111/mmi.14656. Epub 2020 Dec 18.

The H-NS-like proteins MvaT and MvaU act coordinately as global repressors in Pseudomonas aeruginosa by binding to AT-rich regions of the chromosome. Although cells can tolerate loss of either protein, identifying their combined regulatory effects has been challenging because...

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Lippa AM, Gebhardt MJ, Dove SL. H-NS-like proteins in Pseudomonas aeruginosa coordinately silence intragenic transcription. Mol Microbiol. 2020;115(6):1138-1151doi: 10.1111/mmi.14656.
Lippa, A. M., Gebhardt, M. J., & Dove, S. L. (2021). H-NS-like proteins in Pseudomonas aeruginosa coordinately silence intragenic transcription. Molecular microbiology, 115(6), 1138-1151. https://doi.org/10.1111/mmi.14656
Lippa, Andrew M, et al. "H-NS-like proteins in Pseudomonas aeruginosa coordinately silence intragenic transcription." Molecular microbiology vol. 115,6 (2021): 1138-1151. doi: https://doi.org/10.1111/mmi.14656
Lippa AM, Gebhardt MJ, Dove SL. H-NS-like proteins in Pseudomonas aeruginosa coordinately silence intragenic transcription. Mol Microbiol. 2021 Jun;115(6):1138-1151. doi: 10.1111/mmi.14656. Epub 2020 Dec 18. PMID: 33245158; PMCID: PMC8156940.
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Divergent dysregulation of gene expression in murine models of fragile X syndrome and tuberous sclerosis.

Molecular autism

Kong SW, Sahin M, Collins CD, Wertz MH, Campbell MG, Leech JD, Krueger D, Bear MF, Kunkel LM, Kohane IS.
PMID: 24564913
Mol Autism. 2014 Feb 24;5(1):16. doi: 10.1186/2040-2392-5-16.

BACKGROUND: Fragile X syndrome and tuberous sclerosis are genetic syndromes that both have a high rate of comorbidity with autism spectrum disorder (ASD). Several lines of evidence suggest that these two monogenic disorders may converge at a molecular level...

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Kong SW, Sahin M, Collins CD, et al. Divergent dysregulation of gene expression in murine models of fragile X syndrome and tuberous sclerosis. Mol Autism. 2014;5(1):16doi: 10.1186/2040-2392-5-16.
Kong, S. W., Sahin, M., Collins, C. D., Wertz, M. H., Campbell, M. G., Leech, J. D., Krueger, D., Bear, M. F., Kunkel, L. M., & Kohane, I. S. (2014). Divergent dysregulation of gene expression in murine models of fragile X syndrome and tuberous sclerosis. Molecular autism, 5(1), 16. https://doi.org/10.1186/2040-2392-5-16
Kong, Sek Won, et al. "Divergent dysregulation of gene expression in murine models of fragile X syndrome and tuberous sclerosis." Molecular autism vol. 5,1 (2014): 16. doi: https://doi.org/10.1186/2040-2392-5-16
Kong SW, Sahin M, Collins CD, Wertz MH, Campbell MG, Leech JD, Krueger D, Bear MF, Kunkel LM, Kohane IS. Divergent dysregulation of gene expression in murine models of fragile X syndrome and tuberous sclerosis. Mol Autism. 2014 Feb 24;5(1):16. doi: 10.1186/2040-2392-5-16. PMID: 24564913; PMCID: PMC3940253.
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Functional connectivity in the first year of life in infants at-risk for autism: a preliminary near-infrared spectroscopy study.

Frontiers in human neuroscience

Keehn B, Wagner JB, Tager-Flusberg H, Nelson CA.
PMID: 23964223
Front Hum Neurosci. 2013 Aug 06;7:444. doi: 10.3389/fnhum.2013.00444. eCollection 2013.

BACKGROUND: Autism spectrum disorder (ASD) has been called a "developmental disconnection syndrome," however the majority of the research examining connectivity in ASD has been conducted exclusively with older children and adults. Yet, prior ASD research suggests that perturbations in...

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Keehn B, Wagner JB, Tager-Flusberg H, et al. Functional connectivity in the first year of life in infants at-risk for autism: a preliminary near-infrared spectroscopy study. Front Hum Neurosci. 2013;7:444doi: 10.3389/fnhum.2013.00444.
Keehn, B., Wagner, J. B., Tager-Flusberg, H., & Nelson, C. A. (2013). Functional connectivity in the first year of life in infants at-risk for autism: a preliminary near-infrared spectroscopy study. Frontiers in human neuroscience, 7444. https://doi.org/10.3389/fnhum.2013.00444
Keehn, Brandon, et al. "Functional connectivity in the first year of life in infants at-risk for autism: a preliminary near-infrared spectroscopy study." Frontiers in human neuroscience vol. 7 (2013): 444. doi: https://doi.org/10.3389/fnhum.2013.00444
Keehn B, Wagner JB, Tager-Flusberg H, Nelson CA. Functional connectivity in the first year of life in infants at-risk for autism: a preliminary near-infrared spectroscopy study. Front Hum Neurosci. 2013 Aug 06;7:444. doi: 10.3389/fnhum.2013.00444. eCollection 2013. PMID: 23964223; PMCID: PMC3734360.
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Showing 1 to 12 of 42 entries